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Books on the topic 'Kidney inflammation'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

F, Zipfel Peter, ed. Complement and kidney disease. Birkhäuser, 2005.

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2

Kidney Inflammation, Injury and Regeneration. MDPI, 2020. http://dx.doi.org/10.3390/books978-3-03928-539-6.

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3

Kidney Inflammation, Injury and Regeneration 2020. MDPI, 2021. http://dx.doi.org/10.3390/books978-3-0365-2369-9.

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4

Carrero, Juan Jesús, and Peter Stenvinkel. The role of inflammation in chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0110.

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Low-grade persistent inflammation is a common feature of chronic kidney disease. This chapter provides an overview of the pathogenesis and clinical consequences of elevated pro-inflammatory cytokines in the uraemic milieu with an emphasis on dialysis stages. It reviews the multifactorial dialysis- and non-dialysis-related causes of inflammation and its purported role in the development of protein energy wasting, vascular calcification, endocrine disorders, and depression. The chapter also discusses the use and the need of monitoring C-reactive protein levels regularly in the clinical setting a

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5

Herrington, William G., Aron Chakera, and Christopher A. O’Callaghan. The kidney in systemic disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0170.

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Many systemic diseases can affect the kidney, including autoimmune conditions, malignancies, infections, and vascular diseases. Autoimmune conditions can cause inflammation of the glomeruli or tubules, or deposition of inflammatory proteins (AA amyloidosis). Malignancy can cause infiltration of normal renal tissue, immunoglobulin deposition in the renal vessels, glomeruli or tubules, or paraneoplastic renal dysfunction as occurs in secondary focal segmental glomerulosclerosis. Infections can cause inflammation in glomeruli, in association with immune complex deposition. Vascular disease and va

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6

Dr. Sebi Cure for Kidney Disease: Through Alkaline Medicinal Herbs and Diets to Remove Kidney Stones, Inflammation; Detox, Cleanse and Revitalize Kidney and Liver of Electric Body. Independently Published, 2020.

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7

Low Oxalate Diet Cookbook: Healthy, Gluten-Free Recipes to Treat Kidney Stones, Inflammation and More. Independently Published, 2021.

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8

Malyszko, Jolanta, and Iain C. Macdougall. Iron metabolism in chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0125.

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While whole-body (‘absolute’) iron deficiency is common and probably increased in frequency in chronic kidney disease (CKD), functional iron deficiency is a particular problem in CKD. Absolute iron deficiency is likely to be present in advanced CKD when the ferritin falls below 100 ng/mL and the TSAT falls below 20%. Functional iron deficiency is characterized by the presence of adequate iron stores (as defined by conventional criteria), but with an inability to mobilize this iron rapidly enough to adequately support erythropoiesis with the administration of erythropoietin. Among such patients

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9

Prowle, John, and Rinaldo Bellomo. Acute kidney injury in severe sepsis. Edited by Norbert Lameire. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0244_update_001.

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Septic acute kidney injury (S-AKI) accounts for close to 50% of all cases of AKI in ICU and, in its various forms, affects between 15% and 20% of ICU patients. Patients typically present with clinical evidence of severe sepsis and septic shock, developing oliguria or anuria, and rapidly rising serum creatinine concentration. The pathophysiology of S-AKI is poorly understood. Although haemodynamic factors might play a role in the loss of glomerular filtration rate, this may not be through the induction of renal ischaemia. Inflammation, microvascular shunting, and changes in glomerular arteriola

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10

Cruz, Dinna N., Anna Giuliani, and Claudio Ronco. Acute kidney injury in heart failure. Edited by Norbert Lameire. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0248.

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Acute kidney injury (AKI) occurring during heart failure (HF) has been labelled cardiorenal syndrome (CRS) type 1. CRS is defined as a group of ‘disorders of the heart and kidneys whereby acute or chronic dysfunction in one organ may induce acute or chronic dysfunction of the other’. This consensus definition was proposed by the Acute Dialysis Quality Initiative, with the aim to standardize those disorders where cardiac and renal diseases coexist. Five subtypes have been proposed, according to which organ is affected first (cardiac vs renal) and whether the dysfunction is acute or chronic. Ano

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11

Low Oxalate Diet Starter's Kit: Delicious Wholesome Recipes to Treat Inflammation, Chronic Pains, and Kidney Stones. Independently Published, 2021.

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12

Avram, Morrell M. Management of Comorbidities in Kidney Disease in the 21st Century: Anemia, Bone Disease, Malnutrition, and Inflammation. Wiley & Sons, Incorporated, John, 2008.

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13

Avram, M. M. Management of Comorbidities in Kidney Disease in the 21st Century: Anemia, Bone Disease, Malnutrition and Inflammation. Blackwell Publishing Limited, 2003.

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14

Berrian, Tamara. Low-Oxalate Anti-Inflammatory Cookbook: 150 Healthy Recipes for Beginners to Manage Inflammation, Pain and Kidney Stones. Independently Published, 2021.

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15

Israel, Nicolas. Renal Diet for Dog Inflammation: The Complete Renal Diet and Cookbook Guide for Effective Natural Remedy for Dogs with Canine Kidney Disease, Pain, Anxiety, Cancer and Inflammation. Independently Published, 2020.

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16

Macdougall, Iain C. Erythropoiesis-stimulating agents in chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0124.

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The advent of recombinant human erythropoietin (epoetin) in the late 1980s transformed the management of renal anaemia, liberating many dialysis patients from lifelong regular blood transfusions, in turn causing severe iron overload and human leucocyte antigen sensitization. Epoetin can be administered either intravenously or subcutaneously, but the half-life of the drug is fairly short at around 6–8 hours, necessitating frequent injections. To circumvent this problem, two manipulations to the erythropoietin molecule were engineered. The first of these was to attach an extra two carbohydrate c

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17

Sprague, Stuart M., and James M. Pullman. Spectrum of bone pathologies in chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0122.

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Histologic bone abnormalities begin very early in the course of chronic kidney disease. The KDIGO guidelines recommend that bone disease in patients with chronic kidney disease should be diagnosed on the basis of bone biopsy examination, with bone histomorphometry. They have also proposed a new classification system (TMV), using three key features of bone histology—turnover, mineralization, and volume—to describe bone disease in these patients. However, bone biopsy is still rarely performed today, as it involves an invasive procedure and highly specialized laboratory techniques. High-turnover

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18

Dr. Sebi Alkaline Diets and Herbs Formulations for Kidney Diseases: Detoxify, Cleanse, and Remove Infections, Toxin/Poison, Stones and Inflammation; and Revitalize Kidney to Ensure Long-Lasting Recovery. Independently Published, 2022.

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19

Cook, Dorothy Dorothy. Newest Kidney Stone Diet Cookbook: An Ideal Low Oxalate Diet Guide to Eliminate Kidney Stone, Reduce Inflammation and Avoid Renal Failure with Delicious Low Sugar Recipes and Food List for Beginners. Independently Published, 2022.

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20

Low-Oxalate Diet,& Anti-Inflammatory Cookbook: This Book Includes 2 Manuscripts. Wholesome Recipes for Beginners to Manage Inflammation,Pains,and Kidney Stones. Independently Published, 2021.

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21

Kliger, Alan S., and Rita Suri. Frequent haemodialysis. Edited by Jonathan Himmelfarb. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0262_update_001.

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Haemodialysis is a renal replacement treatment, an ‘artificial kidney’ that performs some of the functions of the normal kidney. It is an inelegant device, providing only a fraction of native kidney’s ability to filter toxins from the blood, but with none of the responsiveness to volume, fine feedback control to regulate solute concentrations, or endocrine functions of the healthy organ. Conventional haemodialysis performed three times a week for 4 hours per treatment filters the blood for only 12 of 168 hours each week, and removes less than 10 per cent of small solutes like urea than does th

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22

BRANDY, Elizabeth. Low Oxalate Diet for Dummies and Beginners: Delicious Recipes for Beginners to Manage Inflammation,Pain, Kidney Food List, and a Sample Meal Plan. Independently Published, 2021.

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23

Philip D. Philip D. Carter. Newly Revised Low Oxalate Diet Cookbook: Essential Guide with Delicious Recipes to Manage Weight-Loss, Boost Energy , Reduce Inflammation and Prevent Kidney Stones. Independently Published, 2021.

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24

MASTEJ, Morris. Low Oxalate Cookbook for Beginners: The Completely New Guide to Making Low Oxalate Diet to Manage Inflammation, Pain, and Kidney Stones and Renal Diseases. Independently Published, 2022.

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25

Linder, Cynthia. New Latest 2023 Low Oxalate Diet Cookbook: 101+ Recipes to Prevent Kidney Stones,chronic Pains, Inflammation and 30 Day Meal Plan for Better Health. Independently Published, 2022.

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26

Alice, Linda. Utmost 2022 Low Oxalate Diet Cookbook: 100+ Delicious, Nourishing, Low-Oxalate Recipes, with Every Ingredient Labeled for Oxalate Content to Manage Inflammation,Pain, and Kidney. Independently Published, 2022.

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27

Richard, Haley. Low Oxalate Diet for Beginners: Comprehensive Guide to Quickly Manage and Reduce Inflammation, Prevent Kidney Stones and Renal Disease That Will Boost Your General Health. Independently Published, 2022.

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28

Low-Oxalate Diet Cookbook: An Effective Diet to Improve Your Overall Health and Energy Levels. Wholesome Recipes to Treat Inflammation,chronic Pains,and Kidney Stones. Independently Published, 2020.

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29

Török, M. Estée, Fiona J. Cooke, and Ed Moran. Urinary tract infections. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199671328.003.0017.

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This chapter covers cystitis (an infection of the bladder, characterized by dysuria), acute pyelonephritis (an infection of the kidney), chronic pyelonephritis (which is a chronic diffuse interstitial inflammation), renal abscesses (such as perinephric abscess, renal corticomedullary abscess, and renal cortical abscess), catheter-associated urinary tract infections, prostatitis (including granulomatous prostatitis and prostatic abscess), epididymitis, and orchitis.

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30

Complete Guide to Oxalate Diet Cookbook for Beginners: Essential Guide with 100 Recipes and 30 Day Meal Plan to Treat Inflammation Chronic Pains and Kidney Stones. Independently Published, 2022.

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31

Afzali, Behdad, and Claudia Kemper. Immunity. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0128.

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Immunological health relies on a balance between immune responsiveness to foreign pathogens and tolerance to self-components, commensals, food-derived components, and semi-allogeneic fetal antigens. Disruptions of this balance are hallmarks of immunodeficiency diseases, autoimmune diseases, and pregnancy failure. Patients with chronic kidney disease are immunologically unique in demonstrating features of both chronic inflammation and acquired immunodeficiency—predisposing these individuals to the two commonest causes of death, namely cardiovascular disease and sepsis. Defects and abnormalities

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32

Radović, Milan, and Adalbert Schiller. Balkan endemic nephropathy. Edited by Adrian Covic. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0090_update_001.

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Balkan endemic nephropathy (BEN) is a chronic, slowly progressive tubulointerstitial nephritis, with familial clustering, occurring in several endemic rural regions in countries of the Balkan Peninsula. BEN is characterized by anaemia, tubular proteinuria, renal shrinkage, and slowly declining glomerular filtration rate (GFR). Up to one-third of patients may also develop upper urothelial tumours. The aetiology of BEN is unclear; chronic exposure to aristolochic acid and a polygenic predisposition are the most likely contributing factors. The major pathological characteristics of BEN are symmet

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33

Eliana, Scarlett. New 2022 Low Oxalate Diet Cookbook: Homemade Effortless 100+ and 30 Day Meal Plan Healthy Recipes to Quickly Manage and Reduce Inflammation, Prevent Kidney Stones and Renal. Independently Published, 2022.

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34

Aiden, Jessica. Low Oxalate Diet Cookbook 101: Nutricious and Delicious Healthy Low Oxalate Recipes to Help Manage and Reduce Inflammation, Prevent Kidney Stones and Renal and Imrove Overall Health. Independently Published, 2022.

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35

Gartman, Karen. Dexamethasone: One Medicine for the Treatment of Blood or Bone Marrow Problems, Inflammation, Severe Allergies, Arthritis, Kidney Problems, Asthma, Flare-Ups of Multiple Sclerosis, Skin Conditions, and Adrenal Problems. Independently Published, 2019.

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36

Harvey, Mitch. Kidney Stone Diet Cookbook: An Ideal Low Oxalate Diet Guide with Food Lists and Tips to Increase Your Energy and General Wellness. Easy Recipes to Cure Renal Issues, Severe Pain, and Inflammation. Independently Published, 2022.

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37

BRITTANY, Cynthia. Low Oxalate Diet : Reviews, Pros, Cons, Food List and How It Works : : A Step-By-Step Guide for Prevent Kidney Stones, Reduce Inflammation and Renal Disease , a Low Oxalate Food List, and a Sample Meal Plan. Independently Published, 2022.

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38

Speer, Thimoteus, and Danilo Fliser. Abnormal endothelial vasomotor and secretory function. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0113.

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The endothelium plays a crucial role in the maintenance of vascular integrity and function. Nitric oxide produced by endothelial cells is a key player, inducing relaxation of vascular smooth muscle cells, inhibition of vascular inflammation, and prevention of coagulatory activation. Chronic kidney disease (CKD) is characterized by deterioration of different protective endothelial properties, collectively described as endothelial dysfunction. Several factors such as methylarginines, modified lipoproteins, and other substances that accumulate may be involved in the pathogenesis of endothelial dy

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39

Neary, John, and Neil Turner. The patient with haematuria. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0046.

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Haematuria is a common presenting feature of diseases of the kidney or the renal tract. It is also common in screening tests, single dipstick tests being positive in perhaps 5% of individuals. Age and whether the blood is visible (macroscopic) or non-visible (microscopic) impact largely on whether the explanation is likely to be broadly urological or nephrological. Origins are most commonly simple or urological. Macroscopic bleeding is rare in renal disease, and urine colour is then usually more rather smoky than red except when there is very acute inflammation. The chief urological causes are

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40

Keum, NaNa, Mingyang Song, Edward L. Giovannucci, and A. Heather Eliassen. Obesity and Body Composition. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0020.

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In 2014, an estimated 1.9 billion adults worldwide were either overweight (BMI 25–29.9) or obese (BMI ≥30). The so-called obesity epidemic began in high-income, English-speaking countries in the early 1970s, but soon spread globally; more than one-third (38%) of all adults and 600,000 children under age five are overweight or obese, as are two-thirds (69%) of adults in the United States. Excessive body fat is a major cause of type 2 diabetes, hypertension, cardiovascular and liver disease, among other disorders, and has been designated a definite cause of at least fourteen cancer sites: breast

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41

Izzedine, Hassan, and Victor Gueutin. Drug-induced acute tubulointerstitial nephritis. Edited by Adrian Covic. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0084.

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Drug-induced acute tubulointerstitial nephritis (ATIN) is the most common aetiology of ATIN and a potentially correctable cause of acute kidney injury (AKI). An interval of 7–10 days typically exists between drug exposure and development of AKI, but this interval can be considerably shorter following re-challenge or markedly longer with certain drugs. It occurs in an idiosyncratic and non-dose-dependent manner. Antibiotics, NSAIDs, and proton pump inhibitors are the most frequently involved agents, but the list of drugs that can induce ATIN is continuously increasing. The mechanism of renal in

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42

Meroni, Mietta, and A. Sessa. Renal Involvement in Systemic Vasculitis: First Seminar on Renal Involvement in Systemic Vasculitis, Vimercate, September 22, 1990 (Contributions to Nephrology). S Karger Pub, 1991.

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43

Dalbeth, Nicola. Gout. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0141.

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Gout is a common and treatable disorder of purine metabolism. Gout typically presents as recurrent self-limiting episodes of severe inflammatory arthritis affecting the foot. In the presence of persistent hyperuricaemia, tophi, chronic synovitis, and joint damage may develop. Diagnosis of gout is confirmed by identification of monosodium urate (MSU) crystals using polarizing light microscopy. Hyperuricaemia is the central biochemical cause of gout. Genetic variants in certain renal tubular urate transporters including SLC2A9 and ABCG2, and dietary factors including intake of high-purine meats

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44

Dalbeth, Nicola. Gout. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0141_update_003.

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Gout is a common and treatable disorder of purine metabolism. Gout typically presents as recurrent self-limiting episodes of severe inflammatory arthritis affecting the foot. In the presence of persistent hyperuricaemia, tophi, chronic synovitis, and joint damage may develop. Diagnosis of gout is confirmed by identification of monosodium urate (MSU) crystals using polarizing light microscopy. Hyperuricaemia is the central biochemical cause of gout. Genetic variants in certain renal tubular urate transporters including SLC2A9 and ABCG2, and dietary factors including intake of high-purine meats

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45

Keogh, Karina A. Vasculitis in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0277.

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The vasculitic syndromes are a heterogeneous group of rare disorders characterized by degrees of inflammation and necrosis of blood vessels with a wide variety of clinical manifestations. Intensive care treatment is most commonly required for vasculitis involving small blood vessels, including capillaries. Involvement of these vessels in the lung causes alveolar haemorrhage, which may lead to respiratory failure. In the kidneys it may cause glomerulonephritis leading to renal failure. Severe cardiac, neurological, and gastrointestinal manifestions can also be seen. Non-vasculitic manifestation

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46

Hughes, Jeremy. Proteinuria as a direct cause of progression. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0137.

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Proximal tubular cells reabsorb any filtered proteins during health via cell surface receptors such as megalin and cubulin so that very low levels of protein are present in the excreted urine. Significant proteinuria is a common finding in patients with many renal diseases. Proteinuria is a marker of glomerular damage and podocyte loss and injury in particular. The degree of proteinuria at presentation or during the course of the disease correlates with long-term outcome in many renal diseases. Proteinuria per se may be nephrotoxic and thus directly relevant to the progression of renal disease

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47

Renal Diet for Cats : Renal Diet for Cats: Effective Natural Remedies for Cats, with Kidney Disease Anxiety, Pain and Inflammations. Independently Published, 2020.

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48

Schiller, Adalbert, Adrian Covic, and Liviu Segall. Chronic tubulointerstitial nephritis. Edited by Adrian Covic. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0086_update_001.

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Chronic tubulointerstitial nephropathies (CTINs) are a group of renal diseases, characterized by variable interstitial inflammation and fibrosis and tubular atrophy, and a slow course towards end-stage renal disease (ESRD). The causes of CTIN are numerous, including nephrotoxic drugs and chemicals, infections, autoimmune diseases, obstructive uropathies, and metabolic disorders. Taken together, CTIN are responsible for less than 10% of all ESRD cases requiring renal replacement therapy. The clinical manifestations of CTIN typically comprise low-grade proteinuria, leucocyturia, and variably red

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