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Relevant bibliographies by topics / Kinases MAPK

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Dissertations / Theses

Academic literature on the topic 'Kinases MAPK'

Author: Grafiati

Published: 4 June 2021

Last updated: 16 February 2022

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Dissertations / Theses on the topic "Kinases MAPK"

1

Kragelj, Jaka. "Structure and dynamics of intrinsically disordered regions of MAPK signalling proteins." Thesis, Grenoble, 2014. http://www.theses.fr/2014GRENV060/document.

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Les voies de transduction du signal cellulaire permettent aux cellules de répondre aux signaux de l'environnement et de les traiter. Les voies de transduction de kinases MAP (MAPK) sont bien conservées dans toutes les cellules eucaryotes et sont impliquées dans la régulation de nombreux processus cellulaires importants. Les régions intrinsèquement désordonnées (RID), présentes dans de nombreuses MAPK, n'étaient pas encore structurellement caractérisées. Les RID de MAPK sont particulièrement importantes car elles contiennent des motifs de liaison qui contrôlent les interactions entre les protéi

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2

McCarthy, Pierre C. "Regulation of MyD88 signalling by MAPK activated kinases." Thesis, University of Dundee, 2015. https://discovery.dundee.ac.uk/en/studentTheses/ba214a96-e0d3-460e-8230-5931096601a2.

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3

Rice, Kevin M. "Effects of aging on pressure-induced MAPK activation in the rat aorta." Huntington, WV : [Marshall University Libraries], 2005. http://www.marshall.edu/etd/descript.asp?ref=526.

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4

Stead, Rebecca. "Novel roles for the MAPK-interacting serine threonine kinases (Mnks)." Thesis, University of Southampton, 2013. https://eprints.soton.ac.uk/358973/.

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The mitogen activated protein kinase (MAPK)-interacting serine threonine kinases (Mnks) are components of the MEK/ERK and p38MAPK signalling pathways. Research has focused on the role of the Mnks in tumorigenesis, inflammatory signalling cascades and translation of inflammatory cytokines. The physiological roles of these kinases are still not well understood and their functions have yet to be fully elucidated. This prompted the work presented in this study where the roles of the Mnks have been investigated in the phenomenon of rapamycin-induced eIF4E phosphorylation and the role of the Mnks in

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5

Aoidi, Rifdat. "Étude du rôle de la voie ERK/MAPK dans le développement embryonnaire chez la souris." Doctoral thesis, Université Laval, 2017. http://hdl.handle.net/20.500.11794/27476.

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Tableau d'honneur de la Faculté des études supérieures et postdoctorales, 2016-2017<br>Les mammifères possèdent deux MAP kinases kinases (MEK1 et MEK2), impliquées dans l’activation de la voie ERK/MAPK essentielle pour la différenciation, la prolifération et la survie cellulaire. Le premier objectif de cette thèse était de déterminer si les fonctions des kinases MEK1 et MEK2 sont redondantes durant le développement embryonnaire. Les souris Mek1-/- meurent à mi-gestation d’une malformation du placenta. Les souris Mek2-/- ne présentent aucun phénotype majeur, suggérant que ces deux protéines ont

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6

Pang, Wei Wei. "The role of mitochondria in regulating MAPK signalling pathways during oxidative stress." University of Western Australia. School of Biomedical, Biomolecular and Chemical Sciences, 2006. http://theses.library.uwa.edu.au/adt-WU2007.0026.

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[Truncated abstract] Reactive oxygen species (ROS) have been implicated to play a major role in many pathological conditions including heart attack and stroke. Their ability to modulate the extracellular signal-regulated protein kinase (ERK) and c-Jun Nterminal kinase (JNK) signalling pathways, thereby influencing cellular response has been well-documented. Recent studies implicate a central role for mitochondria in ERK and JNK activation by ROS although the mechanisms remained unresolved. Using Jurkat T-lymphocyte as a cell model, this study demonstrated increased mitochondrial ROS production

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7

Krayem, Mohammad. "MAPK pathway as a target for therapy in melanoma." Doctoral thesis, Universite Libre de Bruxelles, 2015. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/209067.

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8

Chen, Xi. "The role of PI3K and ERK/MAPK signal transduction cascades in long-term memory formation /." Thesis, Connect to this title online; UW restricted, 2004. http://hdl.handle.net/1773/6248.

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9

Cheung, Po Yan. "Interaction between MKK6 and p150 glued dynactin is required for microtubule-mediated p38 MAPK activation /." View Abstract or Full-Text, 2002. http://library.ust.hk/cgi/db/thesis.pl?BICH%202002%20CHEUNG.

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Thesis (M. Phil.)--Hong Kong University of Science and Technology, 2002.<br>On t.p. glued is superscript. Includes bibliographical references (leaves 84-94). Also available in electronic version. Access restricted to campus users.

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10

Gutierrez, Prat Núria. "Molecular basis of p38a MAPK signaling." Doctoral thesis, Universitat de Barcelona, 2018. http://hdl.handle.net/10803/663438.

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Cells often need to respond to damaging internal and external stimuli. One of the pathways that is frequently activated by stress stimuli involves activation of the kinase p38alpha, which can phosphorylate different substrates, including MK2. Experiments using purified proteins have shown that non-phosphorylated p38alpha and MK2 can form a tight complex, in which structural constraints impede the interaction of both kinases with effectors and regulators. It is therefore critical to understand how the interaction between p38alpha and MK2 is regulated to ensure that they can release from each

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