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1

Chemical kinetics and process dynamics in aquatic systems. Boca Raton: Lewis, 1994.

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2

Patel, Vinodkumar Hiralal. Chemical kinetic investigation of a commercial batch reactor process. Birmingham: Aston University.Department of Chemical Engineering, 1987.

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3

Cabanes, Antonio López. Pirólisis de hidrocarburos: El proceso de pirólisis con vapor , modelos cinéticos. [Murcia]: Universidad de Murcia, 1989.

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4

NATO Advanced Study Institute on the Enzyme Catalysis Process: Energetics, Mechanism, and Dynamics (1988 Barga, Italy). The enzyme catalysis process: Energetics, mechanism, and dynamics. New York: Plenum Press, 1989.

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5

Paul, Samuel John. Kinetic friction of lubricated contacts in the deep drawing process. [s.l: The Author], 1998.

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6

Verenich, Svetlana. Wet oxidation of concentrated wastewaters: Process combination and reaction kinetic modeling. Lappeenranta, Finland: Lappeenranta University of Technology, 2003.

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7

Gonawan, Fadzil Noor. Immobilized β-Galactosidase-Mediated Conversion of Lactose: Process, Kinetics and Modeling Studies. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-3468-9.

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8

Kazi, Rafiq Akhtar. A high pressure kinetic study of the in-situ combustion process for oil recovery. Salford: University of Salford, 1995.

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9

American Institute of Chemical Engineers. AICHEMI modular instruction: Series G, design of equipment. New York: American Institute of Chemical Engineers, 1986.

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10

Medhora, Hiraz Kekobad. Reaction kinetic study and determination of controlling resistances during alkaline sulfite/anthraquinone pulping. 1987.

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11

Byrnes, Ian B. Kinetic considerations and process limitations for flow injection analysis of glucose. 1995.

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12

Gonawan, Fadzil Noor. Immobilized β-Galactosidase-Mediated Conversion of Lactose: Process, Kinetics and Modeling Studies. Springer, 2019.

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13

King, Pauline M. The effect of vanadate speciation on the redox kinetics and process performance of the British Gas Stretford Process. 1998.

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14

Gentile, Luigi. Voltage-gated calcium channel kinetics implicated in the process of fast neurotransmitter release. 2005.

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15

Zhou, Yunhong. Numerical modelling of process kinetics during TLP-bonding and other diffusion-controlled processes. 1994.

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16

Gentile, Luigi. Voltage-gated calcium channel kinetics implicated in the process of fast neurotransmitter release. 2005.

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17

Li, Lichun. Fundamental kinetic and mechanistic studies of the stable free radical polymerization (SFRP) process. 2007.

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18

Wales, Douglas G. Process assessment in drawings: The replicative kinetic drawing technique : a computer-based drawing assessment technique. 1987.

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19

Microcystis Aeruginosa Removal by Dissolved Air Flotation: Options for Enchanced Process Operation and Kinetic Modelling. Taylor & Francis, 1998.

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20

S, Fetemi Navid, and United States. National Aeronautics and Space Administration., eds. An analysis of the contact sintering process in III-V solar cells. [Washington, D.C.]: NASA, 1990.

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21

S, Fetemi Navid, and United States. National Aeronautics and Space Administration., eds. An analysis of the contact sintering process in III-V solar cells. [Washington, D.C.]: NASA, 1990.

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22

Henderson, Daniel A., R. J. Boys, Carole J. Proctor, and Darren J. Wilkinson. Linking systems biology models to data: A stochastic kinetic model of p53 oscillations. Edited by Anthony O'Hagan and Mike West. Oxford University Press, 2018. http://dx.doi.org/10.1093/oxfordhb/9780198703174.013.7.

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This article discusses the use of a stochastic kinetic model to study protein level oscillations in single living cancer cells, using the p53 and Mdm2 proteins as examples. It describes the refinement of a dynamic stochastic process model of the cellular response to DNA damage and compares this model to time course data on the levels of p53 and Mdm2. The article first provides a biological background on p53 and Mdm2 before explaining how the stochastic kinetic model is constructed. It then introduces the stochastic kinetic model and links it to the data and goes on to apply sophisticated MCMC methods to compute posterior distributions. The results demonstrate that it is possible to develop computationally intensive Markov chain Monte Carlo (MCMC) methods for conducting a Bayesian analysis of an intra-cellular stochastic systems biology model using single-cell time course data.
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23

Succi, Sauro. Lattice Relaxation Schemes. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780199592357.003.0014.

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In Chapter 13, it was shown that the complexity of the LBE collision operator can be cut down dramatically by formulating discrete versions with prescribed local equilibria. In this chapter, the process is taken one step further by presenting a minimal formulation whereby the collision matrix is reduced to the identity, upfronted by a single relaxation parameter, fixing the viscosity of the lattice fluid. The idea is patterned after the celebrated Bhatnagar–Gross–Krook (BGK) model Boltzmann introduced in continuum kinetic theory as early as 1954. The second part of the chapter describes the comeback of the early LBE in optimized multi-relaxation form, as well as few recent variants hereof.
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24

Fozdar, Manish A., and Jacob C. Holzer. Psychopharmacological Agents, Side Effects, and Black Box Warnings. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199374656.003.0015.

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From a medical-legal viewpoint, psychopharmacological management in the elderly population requires a multifaceted, comprehensive approach, involving a detailed clinical diagnostic assessment, an awareness of a patient’s current medical status, risk factors, and an understanding of the risks and benefits of various treatment options. Several risk management strategies can be employed as part of the treatment process, which are reviewed in this chapter. Special attention is needed in the pharmacological management of the elderly population with respect to efficacy and risk as well as polypharmacy. It is important that clinicians have a broad understanding of the different medication classes, including antidepressants, antipsychotics, mood stabilizers, cognitive-enhancing medications, benzodiazepines, sedative-hypnotics, and stimulants; specific agents; indications; kinetics; and side effects in both the clinical management of elderly patients and in medical-legal consultation.
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25

Nail, Thomas. Marx in Motion. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780197526477.001.0001.

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Socialism is back, and with it comes a renewed interest in Marx’s critique of capitalism. After the 2008 financial crash, international book sales of Capital exploded for the first time in decades. In a world of rising income inequality, right-wing nationalisms, and global climate change, people are looking to the father of modern socialism for answers. This book has been written to help those returning to Marx get answers to their pressing questions about the nature of wealth, ecological crisis, gender inequality, colonialism, migration, and the possibility of socialism. This book also offers readers a new perspective on several major ideas in Marx’s work. It argues that Marx, contrary to conventional wisdom, did not think history was deterministic or that reality could be reduced to classical materialism. Marx was not an anthropocentric humanist, nor did he have a labor theory of value. The unique contribution of this book is that it begins with Marx’s earliest and most neglected book on ancient naturalism in order to show its lasting methodological effect on his “process materialism,” defined by the primacy of motion. This “kinetic Marxism” offers a new way to reread Capital that bears directly on a number of contemporary issues. This also makes Marx in Motion the first book to offer a new materialist reading of Marx. The result is a fresh new view on the important theories of primitive accumulation, metabolism, value, fetishism, dialectics, and the possibility of a kinetic communism for the twenty-first century.
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26

Hendrickx, Jan F. A., André van Zundert, and Andre De Wolf. Inhaled anaesthetics. Edited by Michel M. R. F. Struys. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0014.

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Inhaled anaesthetic drugs are administered via the lungs to provide ‘general anaesthesia’. They are considered complete anaesthetics because they in and by themselves can in most patients ensure all clinical end-points that are required for ‘general anaesthesia’ (unconsciousness, immobility, and haemodynamic stability). The dose–response curve of each clinical end-point is conveniently defined by its mid-point, the end-expired concentration Fa that ensures response suppression in 50 % of the patients (MACawake, MAC, and MACBAR). By understanding the dose–response curves and the factors that influence them (pharmacodynamics), the target Fa and the dose of other drugs can be selected in each individual patient. This target Fa is achieved by adjusting the carrier fresh gas flow (O2, air, N2O) and agent vaporizer setting Fd. ‘Pharmacokinetics’ is the study of the factors that affect the partial pressure cascade from the vaporizer down to the site of action. Because IADs are transported down a partial pressure gradient, Fa will always try to approach the inspired concentration Fi, a process that is described by the Fa/Fi ratio over time. Both Fa and Fi are routinely measured. N2O remains widely used, with scientific scrutiny rather than belief finally delineating its advantages and disadvantages. Xenon, the near-ideal agent, is discussed briefly because it may enter clinical practice despite its cost because of its potential advantages in a yet to be defined subgroup of high-risk patients. The carrier gas N2 is often overlooked, but deserves careful analysis to help the reader understand how rebreathing affects its kinetics in a circle breathing system.
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27

Macdougall, Iain C. Iron management in renal anaemia. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0126.

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Although erythropoiesis-stimulating agent therapy is the mainstay of renal anaemia management, maintenance of an adequate iron supply to the bone marrow is also pivotal in the process of erythropoiesis. Thus, it is important to be able to detect iron insufficiency, and to treat this appropriately. Iron deficiency may be absolute (when the total body iron stores are exhausted) or functional (when the total body iron stores are normal or increased, but there is an inability to release iron from the stores rapidly enough to provide a ready supply of iron to the bone marrow). Several markers of iron status have been tested, but those of the greatest utility are the serum ferritin, transferrin saturation, and percentage of hypochromic red cells. Measurement of serum hepcidin, which is the master regulator of iron homoeostasis, has to date proved disappointing as a means of detecting iron insufficiency, and none of the available iron markers reliably exclude the need for supplemental iron. Iron may be replaced by either the oral or the intravenous route. In the advanced stages of chronic kidney disease, however, hepcidin is upregulated, and this powerfully inhibits the absorption of iron from the gut. Thus, such patients often require intravenous iron, particularly those on dialysis. Several intravenous (IV) iron preparations are available, and they have in common a core containing an iron salt, surrounded by a carbohydrate shell. The IV iron preparations differ in their kinetics of iron release from the iron–carbohydrate complex. In recent times, several new IV iron preparations have become available, and these allow a greater amount of iron to be given more rapidly as a single administration, without the need for a test dose.
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28

Kirchman, David L. Microbial primary production and phototrophy. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198789406.003.0006.

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This chapter is focused on the most important process in the biosphere, primary production, the turning of carbon dioxide into organic material by higher plants, algae, and cyanobacteria. Photosynthetic microbes account for roughly 50% of global primary production while the other half is by large, terrestrial plants. After reviewing the basic physiology of photosynthesis, the chapter discusses approaches to measuring gross and net primary production and how these processes affect fluxes of oxygen and carbon dioxide into and out of aquatic ecosystems. It then points out that terrestrial plants have high biomass but relatively low growth, while the opposite is the case for aquatic algae and cyanobacteria. Primary production varies greatly with the seasons in temperate ecosystems, punctuated by the spring bloom when the biomass of one algal type, diatoms, reaches a maximum. Other abundant algal types include coccolithophorids in the oceans and filamentous cyanobacteria in freshwaters. After the bloom, small algae take over and out-compete larger forms for limiting nutrients because of superior uptake kinetics. Abundant types of small algae include two coccoid cyanobacteria, Synechococcus and Prochlorococcus, the latter said to be the most abundant photoautotroph on the planet because of its large numbers in oligotrophic oceans. Other algae, often dinoflagellates, are toxic. Many algae can also graze on other microbes, probably to obtain limiting nitrogen or phosphorus. Still other microbes are mainly heterotrophic but are capable of harvesting light energy. Primary production in oxic environments is carried out by oxygenic photosynthetic organisms, whereas in anoxic environments with sufficient light, it is anaerobic anoxygenic photosynthesis in which oxygen is not produced. Although its contribution to global primary production is small, anoxygenic photosynthesis helps us understand the biophysics and biochemistry of photosynthesis and its evolution on early Earth. These microbes as well as aerobic phototrophic and heterotrophic microbes make up microbial mats. These mats can provide insights into early life on the planet when a type of mat, “stromatolites,” covered vast areas of primordial seas in the Proterozoic.
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