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Relevant bibliographies by topics / Kinetics uptake

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Academic literature on the topic 'Kinetics uptake'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Kinetics uptake"

1

Manns, P. J., C. R. Tomczak, and R. G. Haennel. "OXYGEN UPTAKE KINETICS." Journal of Cardiopulmonary Rehabilitation and Prevention 29, no. 5 (2009): 333. http://dx.doi.org/10.1097/01.hcr.0000361192.80278.bb.

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2

Blakley, Brian W. "Kinetics of Gentamicin Uptake." Otolaryngology–Head and Neck Surgery 121, no. 4 (1999): 510. http://dx.doi.org/10.1016/s0194-5998(99)70251-2.

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de Jesus, Kelly, Ana Sousa, Karla de Jesus, et al. "The effects of intensity on V̇O2 kinetics during incremental free swimming." Applied Physiology, Nutrition, and Metabolism 40, no. 9 (2015): 918–23. http://dx.doi.org/10.1139/apnm-2015-0029.

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Swimming and training are carried out with wide variability in distances and intensities. However, oxygen uptake kinetics for the intensities seen in swimming has not been reported. The purpose of this study was to assess and compare the oxygen uptake kinetics throughout low-moderate to severe intensities during incremental swimming exercise. We hypothesized that the oxygen uptake kinetic parameters would be affected by swimming intensity. Twenty male trained swimmers completed an incremental protocol of seven 200-m crawl swims to exhaustion (0.05 m·s−1 increments and 30-s intervals). Oxygen uptake was continuously measured by a portable gas analyzer connected to a respiratory snorkel and valve system. Oxygen uptake kinetics was assessed using a double exponential regression model that yielded both fast and slow components of the response of oxygen uptake to exercise. From low-moderate to severe swimming intensities changes occurred for the first and second oxygen uptake amplitudes (P ≤ 0.04), time constants (P = 0.01), and time delays (P ≤ 0.02). At the heavy and severe intensities, a notable oxygen uptake slow component (>255 mL·min−1) occurred in all swimmers. Oxygen uptake kinetics whilst swimming at different intensities offers relevant information regarding cardiorespiratory and metabolic stress that might be useful for appropriate performance diagnosis and training prescription.

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Gao, Han, Xiaotian Zhao, Lei Zhou, Fabrizio Sabba, and George F. Wells. "Differential kinetics of nitrogen oxides reduction leads to elevated nitrous oxide production by a nitrite fed granular denitrifying EBPR bioreactor." Environmental Science: Water Research & Technology 6, no. 4 (2020): 1028–43. http://dx.doi.org/10.1039/c9ew00881k.

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Balakshin, Mikhail Yu, Chen-Loung Chen, Josef S. Gratzl, Adrianna G. Kirkman, and Harald Jakob. "Kinetic Studies on Oxidation of Veratryl Alcohol by Laccase-Mediator System. Part 2. The Kinetics of Dioxygen Uptake." Holzforschung 54, no. 2 (2000): 171–75. http://dx.doi.org/10.1515/hf.2000.029.

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Summary The kinetics of dioxygen uptake in the laccase-catalyzed oxidation of veratryl alcohol with dioxygen in the presence of ABTS, the mediator, was studied. The kinetics of dioxygen uptake consists of two phases: (1) the initial phase up to a reaction time of one hour, and (2) the second phase, after a reaction time of one hour. In the initial phase, ABTS is mainly oxidized to the corresponding cation radical. The kinetics of dioxygen uptake follows a pseudo-zero order rate law. The dioxygen uptake under the reaction condition correlates with the initial ABTS concentration according to the stoichiometric relationship of 0.25 moles dioxygen per mole ABTS. In the second phase, veratryl alcohol is mainly oxidized to veratraldehyde. The kinetics of the dioxygen uptake follows a pseudo-first order rate law. The dioxygen uptake correlates linearly with the yield of veratraldehyde. The stoichiometric ratio between the formation of veratraldehyde and the consumption of dioxygen differs slightly at different M/S ratios. On average, however, it is 0.42 moles of dioxygen per one mole of veratraldehyde formed. The reaction mechanism is discussed on the basis of the kinetic data.

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Xu, Fan, and Edward C. Rhodes. "Oxygen Uptake Kinetics During Exercise." Sports Medicine 27, no. 5 (1999): 313–27. http://dx.doi.org/10.2165/00007256-199927050-00003.

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Zhou, Jin, Yves Desjardins, and Line Lapointe. "NUTRIENT UPTAKE KINETICS OF CLOUDBERRY." Journal of Plant Nutrition 36, no. 8 (2013): 1219–33. http://dx.doi.org/10.1080/01904167.2013.780610.

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Pauli, Anneli S. L., and Seppo Kaitala. "Phosphate uptake kinetics byAcinetobacter isolates." Biotechnology and Bioengineering 53, no. 3 (1997): 304–9. http://dx.doi.org/10.1002/(sici)1097-0290(19970205)53:3<304::aid-bit9>3.0.co;2-m.

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9

Scheuermann, Barry W., and Thomas J. Barstow. "O2 uptake kinetics during exercise at peak O2 uptake." Journal of Applied Physiology 95, no. 5 (2003): 2014–22. http://dx.doi.org/10.1152/japplphysiol.00590.2002.

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Compared with moderate- and heavy-intensity exercise, the adjustment of O2 uptake (V̇o2) to exercise intensities that elicit peak V̇o2 has received relatively little attention. This study examined the V̇o2 response of 21 young, healthy subjects (25 ± 6 yr; mean ± SD) during cycle ergometer exercise to step transitions in work rate (WR) corresponding to 90, 100, and 110% of the peak WR achieved during a preliminary ramp protocol (15-30 W/min). Gas exchange was measured breath by breath and interpolated to 1-s values. V̇o2 kinetics were determined by use of a two- or three-component exponential model to isolate the time constant (τ2) as representative of V̇o2 kinetics and the amplitude (Amp) of the primary fast component independent of the appearance of any V̇o2 slow component. No difference in V̇o2 kinetics was observed between WRs (τ90 = 24.7 ± 9.0; τ100 = 22.8 ± 6.7; τ110 = 21.5 ± 9.2 s, where subscripts denote percent of peak WR; P > 0.05); nor in a subgroup of eight subjects was τ2 different from the value for moderate-intensity (<lactate threshold) exercise (τ2 = 25 ± 12 s, P > 0.05). As expected, the Amp increased with increasing WRs (Amp90 = 2,089 ± 548; Amp100 = 2,165 ± 517; Amp110 = 2,225 ± 559 ml/min; Amp90 vs. Amp110, P < 0.05). However, the gain (G) of the V̇o2 response (ΔV̇o2/ΔWR) decreased with increasing WRs (G90 = 8.5 ± 0.6; G100 = 7.9 ± 0.6; G110 = 7.3 ± 0.6 ml·min-1·W-1; P < 0.05). The Amp of the primary component approximated 85, 88, and 89% of peak V̇o2 during 90, 100, and 110% WR transitions, respectively. The results of the present study demonstrate that, compared with moderate- and heavy-intensity exercise, the gain of the V̇o2 response (as ΔV̇o2/ΔWR) is reduced for exercise transitions in the severe-intensity domain, but the approach to this gain is well described by a common time constant that is invariant across work intensities. The lower ΔV̇o2/ΔWR may be due to an insufficient adjustment of the cardiovascular and/or pulmonary systems that determine O2 delivery to the exercising muscles or due to recruitment of motor units with lower oxidative capacity, after the onset of exercise in the severe-intensity domain.

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Postlethwait, E. M., S. D. Langford, and A. Bidani. "Kinetics of NO2 air space absorption in isolated rat lungs." Journal of Applied Physiology 73, no. 5 (1992): 1939–45. http://dx.doi.org/10.1152/jappl.1992.73.5.1939.

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We previously showed, during quasi-steady-state exposures, that the rate of inhaled NO2 uptake displays reaction-mediated characteristics (J. Appl. Physiol. 68: 594–603, 1990). In vitro kinetic studies of pulmonary epithelial lining fluid (ELF) demonstrated that NO2 interfacial transfer into ELF exhibits first-order kinetics with respect to NO2, attains [NO2]-dependent rate saturation, and is aqueous substrate dependent (J. Appl. Physiol. 71: 1502–1510, 1991). We have extended these observations by evaluating the kinetics of NO2 gas phase disappearance in isolated ventilating rat lungs. Transient exposures (2–3/lung at 25 degrees C) employed rebreathing (NO2-air) from a non-compliant continuously stirred closed chamber. We observed that 1) NO2 uptake rate is independent of exposure period, 2) NO2 gas phase disappearance exhibited first-order kinetics [initial rate (r*) saturation occurred when [NO2] > 11 ppm], 3) the mean effective rate constant (k*) for NO2 gas phase disappearance ([NO2] < or = 11 ppm, tidal volume = 2.3 ml, functional residual capacity = 4 ml, ventilation frequency = 50/min) was 83 +/- 5 ml/min, 4) with [NO2] < or = 11 ppm, k* and r* were proportional to tidal volume, and 5) NO2 fractional uptakes were constant across [NO2] (< or = 11 ppm) and tidal volumes but exceeded quasi-steady-state observations. Preliminary data indicate that this divergence may be related to the inspired PCO2. These results suggest that NO2 reactive uptake within rebreathing isolated lungs follows first-order kinetics and displays initial rate saturation, similar to isolated ELF.(ABSTRACT TRUNCATED AT 250 WORDS)

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Dissertations / Theses on the topic "Kinetics uptake"

1

Fawkner, Samantha Gieva. "Oxygen uptake kinetics in children." Thesis, University of Exeter, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.393144.

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Baker, Mark. "Kinetics determinants of hepatic uptake." Thesis, University of Sheffield, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.699811.

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Bell, Christopher. "Control and modelling of oxygen uptake kinetics." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp02/NQ42497.pdf.

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Bauer, Timothy Alan. "Oxygen uptake kinetics in peripheral arterial disease." Diss., Manhattan, Kan. : Kansas State University, 2005. http://hdl.handle.net/2097/125.

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Blumoff, Sonja. "Oxygen Uptake Kinetics in Severe Intensity Exercise." Thesis, University of North Texas, 2000. https://digital.library.unt.edu/ark:/67531/metadc2539/.

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The purpose of this study was to describe mathematically the oxygen uptake kinetics during cycle ergometry, and to examine the effect of intensity on the kinetic responses within the severe domain. Sixteen volunteers performed a series of exercise tests at a range of intensities selected to elicit fatigue in ~3 to 10 min. A simple mono-exponential model effectively described the response across all intensities. There was a positive correlation between the response time and the time to fatigue, demonstrating that the maximal oxygen uptake was achieved faster at higher intensities within the severe domain. Models incorporating two components effectively described the responses only in tests lasting 8 min or more. It was concluded that there is a second, slow component in the oxygen uptake response only at the lower intensities within the severe domain.

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Wilkerson, Daryl P. "Oxygen uptake kinetics during supra-maximal intensity exercise." Thesis, Manchester Metropolitan University, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.424750.

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7

Claxton, David B. "The measurement of oxygen uptake kinetics in children." Thesis, Sheffield Hallam University, 1999. http://shura.shu.ac.uk/3152/.

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Traditional approaches to exercise testing in children may not provide the most appropriate measures of a child's physiological responses to exercise, partly because they do not reflect children's normal intermittent activity patterns. The measurement of the rate and magnitude of change of oxygen uptake to dynamic exercise, oxygen uptake kinetics (V02 KINETICS provides an alternative approach to exercise testing. A submaximal, intermittent, pseudo-random binary sequence (PRBS) exercise test to measure V02 KINETICS may provide a useful method of measuring the metabolic responses of children to exercise. Traditional methods used in the analysis of V02 KINETICS require the fitting of explicit models in order to characterise the data. These models have not however been validated for use in children. As the responses to the PRBS protocol are analysed in the frequency domain, explicit models and their physiological correlates are not required to characterise the data. Another potential problem in the measurement of V02 KINETICS in children are the small work rate changes that can be employed to stimulate the exercise response whilst constraining the test to the aerobic range. In respiratory gas measurement, breath-by-breath variability (noise) can be large in comparison to the magnitude of the metabolic response and this signal noise can obscure some characteristics of the response. The aim of the study was to develop appropriate measurement techniques to reduce the effects of breath-by-breath variability and to apply the techniques to the measurement of V02 KINETICS in children. The main experimental study compared the V02 KINETICS of children with those of adults. Ten children (3 females) in the age range 8 to 13 and twenty adults (10 females) in the age range 20 to 28 years completed a PRBS test to measure V02 KINETICS and an incremental ramp protocol on a cycle ergometer (Bosch 550 ERG) to establish V02 MAX, T VENT and delta efficiency. Breath-by-breath respiratory gas analysis was undertaken using a respiratory mass spectrometer (MGA1100). Estimates of alveolar gas exchange were made using the algorithm of Beaver et al. (1981) and a post hoc value of an effective lung volume was calculated to minimise the breath-by-breath variability. A cross-correlation technique (CC) was used to filter out the effects of anomalous (nonphysiologic) V02 responses recorded during the PRBS protocol. Subsequent Fourier analysis of the auto-correlation and CC functions provided a description of V02 KINETICS in the frequency domain in terms of amplitude ratio and phase delay over the frequency range of 2.2-8.9mHz. At each of the frequencies assessed amplitude ratio was higher in children (P<0.001) than in either of the adult groups. Phase delay was also significantly shorter in children compared to adults males (P<0.01) and adult females (P<0.001) but this effect was not identifiable at any specific frequency. Maximal oxygen uptake was not significantly different in adult males (42.5 ml"kg "min) and children (44.7 ml-kg'-min') but was lower in adult females (36.9 ml"kg "min) than adult males (P<0.01) and children (P<0.001). Ventilatory threshold (% V02 MAX) was not different between groups. Delta efficiency was significantly lower in children than adult males (P<0.05) and adult females (P<0.01). These results support the contention that there are maturational differences between adults and children in the metabolic processes involved in the utilisation of oxygen during physical activity. It has been argued, theoretically, that in adults the control of V02 KINETICS is driven by ATP demand in the skeletal muscle. As the mitochondria] capacity and the concentration of oxidative enzymes is higher in children than in adults it is likely that the controlling factor(s) for V02 KINETICS in children also relates to some aspect of peripheral metabolism. It is suggested that the PRBS protocol, with appropriate noise reduction techniques, is considered a suitable method for investigating the metabolic responses of children to dynamic exercise.

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Grant, Crystelle Kiyoko. "Influence of cardiac output on oxygen uptake kinetics /." Diss., CLICK HERE for online access, 2010. http://contentdm.lib.byu.edu/ETD/image/etd3341.pdf.

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Grant, Crystelle Kiyoko. "Influence of Cardiac Output on Oxygen Uptake Kinetics." BYU ScholarsArchive, 2009. https://scholarsarchive.byu.edu/etd/1989.

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The purpose of this study was to evaluate increased cardiac output (Q) on oxygen kinetics at exercise intensities above and below the lactate threshold (LT). We hypothesized the increase in Q using head-out water immersion (HOI) while treadmill running would reduce the rate constant of the fast component and reduce the amplitude of the slow component of oxygen kinetics compared with land treadmill running. Subjects (n=10) performed two 6 min exercise bouts at a 15% below and above the LT on a land and underwater treadmill following rest. A single exponential equation [VO2(t) = VO2(b) + A1•(1-e-t/TC1] was used to evaluate VO2. The slow component at the end of exercise was estimated by subtracting (VO2(b) + A1) from the plateau. The mean LT for HOI running 1.80 ± .09 L • min-1 was significantly lower (p < 0.05) than 2.15 ± 1.03 L • min-1 while running on the land. The Q during HOI exercise below and above the LT (16.5 ± 0.6 L • min-1, 18.0 ± 1.2 L • min-1) was significantly higher (p < 0.05) than the Q during exercise below and above the LT on land (11.5 ± 0.8 L • min-1, 13.0 ± 0.7 L • min-1). During HOI exercise below LT time to reach steady-state was delayed (8 ± 2 s). Exercise above LT showed similar phase one time constants for all exercise trials. The amplitude of the slow component was not influenced by HOI. As such, the increase in during HOI exercise did not hastening uptake kinetics.

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Bailey, Stephen John. "O2 uptake kinetics as a determinant of exercise tolerance." Thesis, University of Exeter, 2011. http://hdl.handle.net/10036/3078.

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Oxygen uptake ( O2) kinetics determine the magnitude of the O2 deficit and the degree of metabolic perturbation and is considered to be an important determinant of exercise tolerance; however, there is limited empirical evidence to demonstrate that O2 kinetics is a direct determinant of exercise tolerance. The purpose of this thesis was to investigate O2 kinetics as a determinant of exercise tolerance and to consider its potential interaction with the maximum O2 ( O2max) and the W′ (the curvature constant of the hyperbolic power-duration relationship) in setting the tolerable duration of exercise. Recreationally-active adult humans volunteered to participate in the investigations presented in this thesis. Pulmonary O2 kinetics was assessed on a breath-by-breath basis and exercise tolerance was assessed by a time-to-exhaustion trial, with exhaustion taken as the inability to maintain the required cadence. A period of repeated sprint training (RST) resulted in faster phase II O2 kinetics (Pre: 29 ± 5, Post: 23 ± 5 s), a reduced O2 slow component (Pre: 0.52 ± 0.19, Post: 0.40 ± 0.17 L•min-1), an increased O2max (Pre: 3.06 ± 0.62, Post: 3.29 ± 0.77 L•min-1) and a 53% improvement in severe exercise tolerance. A reduced O2 slow component and enhanced exercise tolerance was also observed following inspiratory muscle training (Pre: 0.60 ± 0.20, Post: 0.53 ± 0.24 L•min-1; Pre: 765 ± 249, Post: 1061 ± 304 s, respectively), L-arginine (ARG) administration (Placebo: 0.76 ± 0.29 L•min-1 vs. ARG: 0.58 ± 0.23; Placebo: 562 ± 145 s vs. ARG: 707 ± 232 s, respectively) and dietary nitrate supplementation administered as nitrate-rich beetroot juice (BR) (Placebo: 0.74 ± 0.24 vs. BR: 0.57 ± 0.20 L•min-1; Placebo: 583 ± 145 s vs. BR: 675 ± 203, respectively). However, compared to a control condition without prior exercise, the completion of a prior exercise bout at 70% Δ (70% of the difference between the work rate at the gas exchange threshold [GET] and the work rate at the O2max + the work rate at the GET) with 3 minutes recovery (70-3-80) speeded overall O2 kinetics by 41% (Control: 88 ± 22 s, 70-3-80: 52 ± 13 s), but impaired exercise tolerance by 16% (Control: 437 ± 79 s, 70-3-80: 368 ± 48 s) during a subsequent exercise bout. When the recovery duration was extended to 20 minutes (70-20-80) to allow a more complete replenishment of the W′, overall kinetics was speeded to a lesser extent (by 23%; 70-20-80: 68 ± 19 s) whereas exercise performance was enhanced by 15% (70-20-80: 567 ± 125 s) compared to the control condition. In addition, the faster O2 kinetics observed when exercise was initiated with a fast start (FS; 35 ± 6 s), compared to an even start (ES; 41 ± 10 s) and slow start (SS; 55 ± 14 s) pacing strategy, allowed the achievement of O2max in a 3 minute trial and exercise performance was enhanced. Exercise performance was unaffected in a 6 minute trial with a FS, despite faster O2 kinetics, as the O2max was attained in all the variously paced trials. Therefore, the results of this thesis demonstrate that changes in exercise performance cannot be accounted for, purely, by changes in O2 kinetics. Instead, enhanced exercise performance appears to be contingent on the interaction between the factors underpinning O2 kinetics, the O2max and the W′, in support of the proposed ‘triad model’ of exercise performance.

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Books on the topic "Kinetics uptake"

1

Ingham, Stephen A. Oxygen uptake kinetics and performance in rowing. University of Surrey Roehampton, 2003.

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2

Jones, Andrew M. Oxygen uptake kinetics in sport, exercise and medicine: A practical handbook. Routledge, 2004.

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Fischer, Astrid Carolien. A compartmental analysis of the kinetics of iron uptake by two Antarctic diatoms. IOS Press, 2009.

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4

Fawkner, Samantha G., and Neil Armstrong. Oxygen uptake kinetics. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199232482.003.0022.

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The two main purposes of Chapter 22 are to (i) explore the methodological issues involved in assessing the O2 kinetic response to exercise in children, and (ii) explain the O2 kinetic response to exercise in children and review the literature regarding changes with age and sex and with respect to conventional markers of aerobic fitness.

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Barker, Alan R., and Neil Armstrong. Pulmonary oxygen uptake kinetics. Edited by Neil Armstrong and Willem van Mechelen. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198757672.003.0013.

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The pulmonary oxygen uptake (pV̇O2) kinetic response to exercise provides valuable non-invasive insight into the control of oxidative phosphorylation and determinants of exercise tolerance in children and adolescents. Few methodologically robust studies have investigated pV̇O2 kinetics in children and adolescents, but age- and sex-related differences have been identified. There is a clear age-related slowing of phase II pV̇O2 kinetics during heavy and very heavy exercise, with a trend showing during moderate intensity exercise. During heavy and very heavy exercise the oxygen cost is higher for phase II and the pV̇O2 component is truncated in children. Sex-related differences occur during heavy, but not moderate, intensity exercise, with boys having faster phase II pV̇O2 kinetics and a smaller pV̇O2 slow component compared to girls. The mechanisms underlying these differences are likely related to changes in phosphate feedback controllers of oxidative phosphorylation, muscle oxygen delivery, and/or muscle fibre recruitment strategies.

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1970-, Jones Andrew M., and Poole David C. 1959-, eds. Oxygen uptake kinetics in sport, exercise and medicine. Routledge, 2005.

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Jones, Andrew M., and David C. Poole. Oxygen Uptake Kinetics in Sport, Exercise and Medicine. Routledge, 2013. http://dx.doi.org/10.4324/9780203613771.

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Taillefer, Raymond, and Frans J. Th Wackers. Kinetics of Conventional and New Cardiac Radiotracers. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199392094.003.0004.

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The kinetics of radiotracers, that is the mode of uptake, retention and release from the myocardium, are relevant for designing and implementing optimized nuclear cardiac imaging protocols. This chapter addresses the kinetics of commonly used radiotracers for imaging myocardial perfusion, sympathetic neuronal function and cardiac metabolism, both with SPECT and PET cardiac imaging. The optimal timing of imaging after injection either at stress or at rest is determined by rate of uptake in the heart and adjacent organs, as well as the residence time of radiotracers within the myocytes. The efficiency of myocardial extraction over a wide range myocardial blood flows is relevant for reliable detection of obstructive coronary artery disease and absolute quantification of regional myocardial blood flow. For each cardiac imaging agent the cellular mechanism of uptake and its release or retention are discussed with an emphasis on the clinical impact of these parameters.

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Burnley, Mark. Effects of prior exercise on the on-transient oxygen uptake kinetics of constant-load exercise. 2002.

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Armstrong, Neil, and Alison M. McManus. Aerobic fitness. Edited by Neil Armstrong and Willem van Mechelen. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198757672.003.0012.

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Peak oxygen uptake (V̇O2) is the criterion measure of young people's aerobic fitness, and blood lactate accumulation (BLA) is a useful indicator of aerobic fitness with reference to the ability to sustain submaximal exercise. In sport and in everyday life it is the pulmonary (p)V̇O2 kinetics of the non-steady state which best assess the integrated responses of the oxygen delivery system and the metabolic demands of the exercising muscle. Data analysis using sophisticated modelling techniques has enhanced understanding of sexual dimorphism and the independent effects of chronological age, body size, and biological maturity on peak V̇O2 and BLA. The extant data on young people's pV̇O2 kinetic responses to step changes in exercise intensity are sparse, but describe intriguing chronological age and sex differences across exercise domains. However, independent effects of biological maturation are yet to be revealed.

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Book chapters on the topic "Kinetics uptake"

1

Langel, Ülo. "Kinetics of CPPs Cellular Uptake." In CPP, Cell-Penetrating Peptides. Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-8747-0_8.

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Justice, J. B., M. D. Bailey, E. L. Barker, and R. D. Blakely. "Voltammetric Studies on the Kinetics and Mechanism of Catecholamine Transporters." In Neutrotransmitter Release and Uptake. Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-60704-2_19.

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Florén, Anders, Imre Mäger, and Ülo Langel. "Uptake Kinetics of Cell-Penetrating Peptides." In Methods in Molecular Biology. Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-919-2_9.

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Cochrane, J. E., R. L. Hughson, and P. C. Murphy. "On Modelling Alveolar Oxygen Uptake Kinetics." In Respiratory Control. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-0529-3_16.

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Uusna, Julia, Kent Langel, and Ülo Langel. "Toxicity, Immunogenicity, Uptake, and Kinetics Methods for CPPs." In Methods in Molecular Biology. Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2806-4_9.

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Mano, D. M. S., K. Buff, and T. Langenbach. "Kinetics of Kelthane Uptake and Distribution in Azospirillum Lipoferum." In Nitrogen Fixation. Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3486-6_15.

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Small, Donald M., Susanne Bennett Clark, Anna Tercyak, John Steiner, Donald Gantz, and Arie Derksen. "The Lipid Surface of Triglyceride-Rich Particles Can Modulate (Apo)Protein Binding and Tissue Uptake." In Hypercholesterolemia, Hypocholesterolemia, Hypertriglyceridemia, in Vivo Kinetics. Springer US, 1990. http://dx.doi.org/10.1007/978-1-4684-5904-3_33.

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Weisiger, Richard A. "Impact of Extracellular and Intracellular Diffusion on Hepatic Uptake Kinetics." In Whole Organ Approaches to Cellular Metabolism. Springer New York, 1998. http://dx.doi.org/10.1007/978-1-4612-2184-5_16.

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Sultan, Fabrice, Dominique Lagrange, and Sabine Griglio. "In Vitro Binding and in Vivo Uptake of Chylomicron Remnants after their Hydrolysis by Hepatic Lipase." In Hypercholesterolemia, Hypocholesterolemia, Hypertriglyceridemia, in Vivo Kinetics. Springer US, 1990. http://dx.doi.org/10.1007/978-1-4684-5904-3_37.

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Portielje, R., and L. Lijklema. "Kinetics of luxury uptake of phosphate by algae-dominated benthic communities." In Nutrient Dynamics and Biological Structure in Shallow Freshwater and Brackish Lakes. Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-017-2460-9_31.

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Conference papers on the topic "Kinetics uptake"

1

Miller, Gerald G., Kevin Brown, Ronald B. Moore, et al. "Intracellular uptake kinetics of hypocrellin photosensitizers for photodynamic therapy." In Fifth International Photodynamic Association Biennial Meeting, edited by Denis A. Cortese. SPIE, 1994. http://dx.doi.org/10.1117/12.203432.

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Combret, Yann, Clément Médrinal, Guillaume Prieur, Aurora Robledo Quesada, Pascal Le Roux, and Grégory Reychler. "Oxygen uptake kinetics in walking children with cystic fibrosis." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa4618.

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Rezzoug, Hadjira, Jean-Louis Merlin, Nadia Zeghari, et al. "Cellular uptake kinetics and photodynamic activity of meso-tetrahydroxyphenylchlorin (mTHPC)." In BiOS Europe '95, edited by Benjamin Ehrenberg, Giulio Jori, and Johan Moan. SPIE, 1996. http://dx.doi.org/10.1117/12.230967.

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Jain, R., C. S. McCool, D. W. Green, G. P. Willhite, and M. J. Michnick. "Reaction Kinetics of the Uptake of Chromium(III) Acetate by Polyacrylamide." In SPE/DOE Symposium on Improved Oil Recovery. Society of Petroleum Engineers, 2004. http://dx.doi.org/10.2118/89399-ms.

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Sinha, Lagnojita, Jonathan T. Elliott, Tayyaba Hasan, Brian W. Pogue, Kimberley S. Samkoe, and Kenneth M. Tichauer. "Early photosensitizer uptake kinetics predict optimum drug-light interval for photodynamic therapy." In SPIE BiOS, edited by David H. Kessel and Tayyaba Hasan. SPIE, 2015. http://dx.doi.org/10.1117/12.2077842.

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Han, ZhaoXiang, and GuanDong He. "Toxicity and Uptake Kinetics of Pentachlorophenol on the Eisenia fetida Exposed to Test Soils." In 2011 International Conference on Computer Distributed Control and Intelligent Environmental Monitoring (CDCIEM). IEEE, 2011. http://dx.doi.org/10.1109/cdciem.2011.444.

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Roman, Michael A., Janos Porszasz, Robert Cao, et al. "An Efficient New Method For Determining Oxygen Uptake Kinetics During Exercise: The Chirp Waveform." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a2394.

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Zhou, Xiao-Hong, and Guo-Xiang Wang. "Notice of Retraction: Phosphorus Uptake Kinetics of Three Plant Grown in the Ecological Floating Bed." In 2011 5th International Conference on Bioinformatics and Biomedical Engineering. IEEE, 2011. http://dx.doi.org/10.1109/icbbe.2011.5781538.

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Richter, Anna M. "Kinetics of cellular uptake and retention of the benzoporphyrin derivative (BPD): relevance to photodynamic therapy." In Photodynamic Therapy of Cancer II. SPIE, 1995. http://dx.doi.org/10.1117/12.199144.

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Akguen, Nermin, Reinhard Sailer, Karin Kunzi-Rapp, Herbert Schneckenburger, Gerd C. Beck, and Angelika C. Rueck. "Phototoxicity, dark-toxicity, and uptake-kinetics of natural hydrophilic and hydrophobic porphyrins in endothelial cells." In BiOS Europe '95, edited by Benjamin Ehrenberg, Giulio Jori, and Johan Moan. SPIE, 1996. http://dx.doi.org/10.1117/12.231003.

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Reports on the topic "Kinetics uptake"

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Safarik, Douglas, Michael Aloi, and Arthur Nobile, Jr. Semi-Empirical Material Model for Hydrogen Uptake Kinetics by 1,4-bis(phenylethynyl)benzene (DEB)-based Getters. Office of Scientific and Technical Information (OSTI), 2020. http://dx.doi.org/10.2172/1673346.

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Vidic, Radisav D. COMBINED THEORETICAL AND EXPERIMENTAL INVESTIGATION OF MECHANISMS AND KINETICS OF VAPOR-PHASE MERCURY UPTAKE BY CARBONACOUES SURFACES. Office of Scientific and Technical Information (OSTI), 2002. http://dx.doi.org/10.2172/800758.

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Vidic, Radisav D., Eric V. Borguet, and Karl J. Johnson. COMBINED THEORETICAL AND EXPERIMENTAL INVESTIGATION OF MECHANISMS AND KINETICS OF VAPOR-PHASE MERCURY UPTAKE BY CARBONACEOUS SURFACES. Office of Scientific and Technical Information (OSTI), 2000. http://dx.doi.org/10.2172/786363.

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Bruland, Kenneth W. Trace Metal Speciation: Equilibrium and Kinetic Considerations on Biological Effects, Phytoplankton Uptake and Sorption Processes in Coastal Waters (Field and Laboratory Studies). Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada627883.

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Bruland, Kenneth W. Trace Metal Speciation: Equilibrium and Kinetic Considerations on Biological Effects, Phytoplankton Uptake and Sorption Processes in Coastal Waters (Field and Laboratory Studies). Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada630785.

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