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Relevant bibliographies by topics / Kölliker-Fuse (KF)

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Academic literature on the topic 'Kölliker-Fuse (KF)'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Kölliker-Fuse (KF)"

1

Damasceno, Rosélia S., Ana C. Takakura, and Thiago S. Moreira. "Regulation of the chemosensory control of breathing by Kölliker-Fuse neurons." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 307, no. 1 (2014): R57—R67. http://dx.doi.org/10.1152/ajpregu.00024.2014.

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The Kölliker-Fuse region (KF) and the lateral parabrachial nucleus (LPBN) have been implicated in the maintenance of cardiorespiratory control. Here, we evaluated the involvement of the KF region and the LPBN in cardiorespiratory responses elicited by chemoreceptor activation in unanesthetized rats. Male Wistar rats (280–330 g; n = 5–9/group) with bilateral stainless-steel guide cannulas implanted in the KF region or the LPBN were used. Injection of muscimol (100 and 200 pmol/100 nl) in the KF region decreased resting ventilation (1,140 ± 68 and 978 ± 100 vs. saline: 1,436 ± 155 ml·kg−1·min−1)

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2

Barnett, William H., Sarah E. M. Jenkin, William K. Milsom, et al. "The Kölliker-Fuse nucleus orchestrates the timing of expiratory abdominal nerve bursting." Journal of Neurophysiology 119, no. 2 (2018): 401–12. http://dx.doi.org/10.1152/jn.00499.2017.

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Coordination of respiratory pump and valve muscle activity is essential for normal breathing. A hallmark respiratory response to hypercapnia and hypoxia is the emergence of active exhalation, characterized by abdominal muscle pumping during the late one-third of expiration (late-E phase). Late-E abdominal activity during hypercapnia has been attributed to the activation of expiratory neurons located within the parafacial respiratory group (pFRG). However, the mechanisms that control emergence of active exhalation, and its silencing in restful breathing, are not completely understood. We hypoth

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3

Hodge, Charles J., A. Vania Apkarian, and Richard T. Stevens. "Inhibition of dorsal-horn cell responses by stimulation of the Kölliker-Fuse nucleus." Journal of Neurosurgery 65, no. 6 (1986): 825–33. http://dx.doi.org/10.3171/jns.1986.65.6.0825.

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✓ The Kölliker-Fuse nucleus (KF) in the dorsolateral pons has been shown to be the major source of catecholamine innervation of the spinal cord. This has important implications in terms of pain control mechanisms, since catecholamine-mediated mechanisms are essential for the expression of opiate and other varieties of antinociception. This study examines the effects of KF stimulation on responses of dorsal-horn cells to innocuous and noxious cutaneous stimuli in anesthetized cats. Stimulation of the KF potently inhibits the responses of dorsal-horn cells to both noxious and innocuous stimuli.

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4

Mirzaei-Damabi, Nafiseh, Bahar Rostami, and Masoumeh Hatam. "Role of the Kölliker-Fuse nucleus in cardiovascular responses to hypoxia and baroreceptor activation in anesthetized rats." BioImpacts 10, no. 1 (2019): 55–61. http://dx.doi.org/10.15171/bi.2020.07.

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<span style="color: #1f497d;">Introduction: Parabrachial Kölliker-Fuse (KF) complex, located in dorsolateral part of the pons, is involved in the respiratory control, however, its role in the baroreflex and chemoreflex responses has not been established yet. This study was performed to test the contribution of the KF to chemoreflex and baroreflex and the effect of microinjection of a reversible synaptic blocker (Cocl2) into the KF in urethane anesthetized rats. <br /> <span style="color: #1f497d;">Methods: Activation of chemoreflex was induced by systemic hypoxia caused by N2

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Balaban, Carey D., David M. McGee, Jianxun Zhou, and Charles A. Scudder. "Responses of Primate Caudal Parabrachial Nucleus and Kölliker-Fuse Nucleus Neurons to Whole Body Rotation." Journal of Neurophysiology 88, no. 6 (2002): 3175–93. http://dx.doi.org/10.1152/jn.00499.2002.

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The caudal aspect of the parabrachial (PBN) and Kölliker-Fuse (KF) nuclei receive vestibular nuclear and visceral afferent information and are connected reciprocally with the spinal cord, hypothalamus, amygdala, and limbic cortex. Hence, they may be important sites of vestibulo-visceral integration, particularly for the development of affective responses to gravitoinertial challenges. Extracellular recordings were made from caudal PBN cells in three alert, adult female Macaca nemestrina through an implanted chamber. Sinusoidal and position trapezoid angular whole body rotation was delivered in

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6

Suzuki, Takeshi, Yoichiro Sugiyama, and Bill J. Yates. "Integrative responses of neurons in parabrachial nuclei to a nauseogenic gastrointestinal stimulus and vestibular stimulation in vertical planes." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 302, no. 8 (2012): R965—R975. http://dx.doi.org/10.1152/ajpregu.00680.2011.

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The parabrachial and adjacent Kölliker-Fuse (PBN/KF) nuclei play a key role in relaying visceral afferent inputs to the hypothalamus and limbic system and are, thus, believed to participate in generating nausea and affective responses elicited by gastrointestinal (GI) signals. In addition, the PBN/KF region receives inputs from the vestibular system and likely mediates the malaise associated with motion sickness. However, previous studies have not considered whether GI and vestibular inputs converge on the same PBN/KF neurons, and if so, whether the GI signals alter the responses of the cells

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7

Mörschel, Michael, and Mathias Dutschmann. "Pontine respiratory activity involved in inspiratory/expiratory phase transition." Philosophical Transactions of the Royal Society B: Biological Sciences 364, no. 1529 (2009): 2517–26. http://dx.doi.org/10.1098/rstb.2009.0074.

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Control of the timing of the inspiratory/expiratory (IE) phase transition is a hallmark of respiratory pattern formation. In principle, sensory feedback from pulmonary stretch receptors (Breuer–Hering reflex, BHR) is seen as the major controller for the IE phase transition, while pontine-based control of IE phase transition by both the pontine Kölliker–Fuse nucleus (KF) and parabrachial complex is seen as a secondary or backup mechanism. However, previous studies have shown that the BHR can habituate in vivo . Thus, habituation reduces sensory feedback, so the role of the pons, and specificall

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8

Gang, Song, Yoshimi Nakazono, and Namoru Aoki. "Differential projections to the raphe nuclei from the medial parabrachial-Kölliker-Fuse (NPBM-KF) nuclear complex and the retrofacial nucleus in cats: retrograde WGA-HRP tracing." Journal of the Autonomic Nervous System 45, no. 3 (1993): 241–44. http://dx.doi.org/10.1016/0165-1838(93)90056-z.

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9

Gang, Song, Yoshimi Nakazono, and Mamoru Aoki. "Differential projections to the raphe nuclei from the medial parabrachial-Kölliker-Fuse (NPBM-KF) nuclear complex and the retrofacial nucleus in cats: retrograde WGA-HRP tracing." Journal of the Autonomic Nervous System 47, no. 3 (1994): 263. http://dx.doi.org/10.1016/0165-1838(94)90187-2.

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10

Zuperku, Edward J., Astrid G. Stucke, Francis A. Hopp, and Eckehard A. E. Stuth. "Characteristics of breathing rate control mediated by a subregion within the pontine parabrachial complex." Journal of Neurophysiology 117, no. 3 (2017): 1030–42. http://dx.doi.org/10.1152/jn.00591.2016.

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The role of the dorsolateral pons in the control of expiratory duration (Te) and breathing frequency is incompletely understood. A subregion of the pontine parabrachial-Kölliker-Fuse (PB-KF) complex of dogs was identified via microinjections, in which localized pharmacologically induced increases in neuronal activity produced increases in breathing rate while decreases in neuronal activity produced decreases in breathing rate. This subregion is also very sensitive to local and systemic opioids. The purpose of this study was to precisely characterize the relationship between the PB-KF subregion

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Dissertations / Theses on the topic "Kölliker-Fuse (KF)"

1

Damasceno, Rosélia dos Santos. "Envolvimento do núcleo Kölliker-Fuse e do núcleo parabraquial lateral no controle cardiorrespiratório promovido pela ativação dos quimiorreceptores centrais e periféricos." Universidade de São Paulo, 2014. http://www.teses.usp.br/teses/disponiveis/42/42137/tde-10072014-162705/.

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No presente trabalho, avaliamos o envolvimento da região Kölliker Fuse (KF) e núcleo parabraquial lateral (NPBL) nas respostas cardiorrespiratórias induzidas pela ativação dos quimiorreceptores centrais e periféricos em ratos não anestesiados. A injeção bilateral de muscimol (200 pmol/100 nl) no KF reduziu a ventilação basal (978 ± 100, vs. salina: 1436 ± 155 ml/kg/min). Injeção de muscimol no KF reduziu a hiperventilação (1827 ± 61, vs. salina: 3179 ± 325 ml/kg/min) e a taquicardia (380 ± 9, vs. salina: 423 ± 12 bpm), produzidos pela hipóxia (8% O2 - 10 min). Muscimol no KF reduziu a hiper

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