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Relevant bibliographies by topics / Kynureniny

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Academic literature on the topic 'Kynureniny'

Author: Grafiati

Published: 28 June 2021

Last updated: 29 July 2025

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Journal articles on the topic "Kynureniny"

1

Chen, Yiquan, and Gilles J. Guillemin. "Kynurenine Pathway Metabolites in Humans: Disease and Healthy States." International Journal of Tryptophan Research 2 (January 2009): IJTR.S2097. http://dx.doi.org/10.4137/ijtr.s2097.

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Tryptophan is an essential amino acid that can be metabolised through different pathways, a major route being the kynurenine pathway. The first enzyme of the pathway, indoleamine-2,3-dioxygenase, is strongly stimulated by inflammatory molecules, particularly interferon gamma. Thus, the kynurenine pathway is often systematically up-regulated when the immune response is activated. The biological significance is that 1) the depletion of tryptophan and generation of kynurenines play a key modulatory role in the immune response; and 2) some of the kynurenines, such as quinolinic acid, 3-hydroxykynu

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Majláth, Zsófia, and László Vécsei. "A kinureninrendszer és a stressz." Orvosi Hetilap 156, no. 35 (2015): 1402–5. http://dx.doi.org/10.1556/650.2015.30246.

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The kynurenine pathway is the main route of tryptophan degradation which gives rise to several neuroactive metabolites. Kynurenic acid is an endogenous antagonist of excitatory receptors, which proved to be neuroprotective in the preclinical settings. Kynurenines have been implicated in the neuroendocrine regulatory processes. Stress induces several alterations in the kynurenine metabolism and this process may contribute to the development of stress-related pathological processes. Irritable bowel disease and gastric ulcer are well-known disorders which are related to psychiatric comorbidity an

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Zakharov, Gennady A., Alexander V. Zhuravlev, Tatyana L. Payalina, Nikolay G. Kamyshev, and Elena V. Savvateeva-Popova. "The influence of D. melanogaster mutations of the kynurenine pathway of tryptophan metabolism on locomotor behavior and expression of genes belonging to glutamatergic and cholinergic systems." Ecological genetics 9, no. 2 (2011): 65–73. http://dx.doi.org/10.17816/ecogen9265-73.

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Disbalance of kynurenines produced by Drosophila mutations of the kynurenine pathway of tryptophan metabolism influences the locomotor behavior in larvae. The most pronounced is the effect of accumulation of kynurenic acid in the mutant cinnabar manifested as sharp reduction of general level of locomotor activity. The mutations seem to act through modulatory influences of kynurenines on signal cascades governed by ionotropic glutamatergic and cholinergic receptors. Expression of receptor genes in the mutants shows age-related changes pointing to gradual evolvement of consequences of kynurenine

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Büki, Alexandra, Gabriella Kekesi, Gyongyi Horvath, and László Vécsei. "A Potential Interface between the Kynurenine Pathway and Autonomic Imbalance in Schizophrenia." International Journal of Molecular Sciences 22, no. 18 (2021): 10016. http://dx.doi.org/10.3390/ijms221810016.

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Schizophrenia is a neuropsychiatric disorder characterized by various symptoms including autonomic imbalance. These disturbances involve almost all autonomic functions and might contribute to poor medication compliance, worsened quality of life and increased mortality. Therefore, it has a great importance to find a potential therapeutic solution to improve the autonomic disturbances. The altered level of kynurenines (e.g., kynurenic acid), as tryptophan metabolites, is almost the most consistently found biochemical abnormality in schizophrenia. Kynurenic acid influences different types of rece

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Holthuijsen, Daniëlle D. B., Martijn J. L. Bours, Eline H. van Roekel, et al. "Longitudinal Associations of Adherence to the Dietary World Cancer Research Fund/American Institute for Cancer Research (WCRF/AICR) and Dutch Healthy Diet (DHD) Recommendations with Plasma Kynurenines in Colorectal Cancer Survivors after Treatment." Nutrients 14, no. 23 (2022): 5151. http://dx.doi.org/10.3390/nu14235151.

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The tryptophan-kynurenine pathway has been linked to cancer aetiology and survivorship, and diet potentially affects metabolites of this pathway, but evidence to date is scarce. Among 247 stage I-III CRC survivors, repeated measurements were performed at 6 weeks, 6 months, and 1 year post-treatment. Adherence to the World Cancer Research Fund/ American Institute for Cancer Research (WCRF) and Dutch Healthy Diet (DHD) recommendations was operationalized using seven-day dietary records. Plasma kynurenines of nine metabolites were analysed. Longitudinal associations of adherence to these dietary

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Hafstad Solvang, Stein-Erik, Jan Erik Nordrehaug, Dag Aarsland, et al. "Kynurenines, Neuropsychiatric Symptoms, and Cognitive Prognosis in Patients with Mild Dementia." International Journal of Tryptophan Research 12 (January 2019): 117864691987788. http://dx.doi.org/10.1177/1178646919877883.

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Introduction: Circulating tryptophan (Trp) and its downstream metabolites, the kynurenines, are potentially neuroactive. Consequently, they could be associated with neuropsychiatric symptoms and cognitive prognosis in patients with dementia. Objective: The objective of this study was to assess associations between circulating kynurenines, cognitive prognosis, and neuropsychiatric symptoms. Methods: We measured baseline serum Trp, neopterin, pyridoxal 5′-phosphate (PLP), and 9 kynurenines in 155 patients with mild dementia (90 with Alzheimer’s disease, 65 with Lewy body dementia). The ratios be

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Szűcs, Edina, Azzurra Stefanucci, Marilisa Pia Dimmito, et al. "Discovery of Kynurenines Containing Oligopeptides as Potent Opioid Receptor Agonists." Biomolecules 10, no. 2 (2020): 284. http://dx.doi.org/10.3390/biom10020284.

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Kynurenine (kyn) and kynurenic acid (kyna) are well-defined metabolites of tryptophan catabolism collectively known as “kynurenines”, which exert regulatory functions in host-microbiome signaling, immune cell response, and neuronal excitability. Kynurenine containing peptides endowed with opioid receptor activity have been isolated from natural organisms; thus, in this work, novel opioid peptide analogs incorporating L-kynurenine (L-kyn) and kynurenic acid (kyna) in place of native amino acids have been designed and synthesized with the aim to investigate the biological effect of these modific

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Gawel, Kinga. "A Review on the Role and Function of Cinnabarinic Acid, a “Forgotten” Metabolite of the Kynurenine Pathway." Cells 13, no. 5 (2024): 453. http://dx.doi.org/10.3390/cells13050453.

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In the human body, the majority of tryptophan is metabolized through the kynurenine pathway. This consists of several metabolites collectively called the kynurenines and includes, among others, kynurenic acid, L-kynurenine, or quinolinic acid. The wealth of metabolites, as well as the associated molecular targets and biological pathways, bring about a situation wherein even a slight imbalance in the kynurenine levels, both in the periphery and central nervous system, have broad consequences regarding general health. Cinnabarinic acid (CA) is the least known trace kynurenine, and its physiologi

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Theofylaktopoulou, Despoina, Arve Ulvik, Øivind Midttun та ін. "Vitamins B2and B6as determinants of kynurenines and related markers of interferon-γ-mediated immune activation in the community-based Hordaland Health Study". British Journal of Nutrition 112, № 7 (2014): 1065–72. http://dx.doi.org/10.1017/s0007114514001858.

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Vitamins B2and B6are cofactors in the kynurenine pathway. Many of the kynurenines are neuroactive compounds with immunomodulatory effects. In the present study, we aimed to investigate plasma concentrations of vitamins B2and B6as determinants of kynurenines and two markers of interferon-γ-mediated immune activation (kynurenine:tryptophan ratio (KTR) and neopterin). We measured the concentrations of vitamins B2and B6vitamers, neopterin, tryptophan and six kynurenines (i.e. kynurenine, anthranilic acid, kynurenic acid, 3-hydroxykynurenine, 3-hydroxyanthranilic acid and xanthurenic acid) in plasm

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Mieszkowski, Jan, Paulina Brzezińska, Błażej Stankiewicz, et al. "Direct Effects of Vitamin D Supplementation on Ultramarathon-Induced Changes in Kynurenine Metabolism." Nutrients 14, no. 21 (2022): 4485. http://dx.doi.org/10.3390/nu14214485.

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In humans, most free tryptophan is degraded via kynurenine pathways into kynurenines. Kynurenines modulate the immune system, central nervous system, and skeletal muscle bioenergetics. Consequently, kynurenine pathway metabolites (KPMs) have been studied in the context of exercise. However, the effect of vitamin D supplementation on exercise-induced changes in KPMs has not been investigated. Here, we analyzed the effect of a single high-dose vitamin D supplementation on KPMs and tryptophan levels in runners after an ultramarathon. In the study, 35 amateur runners were assigned into two groups:

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Dissertations / Theses on the topic "Kynureniny"

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Urenjak, Jutta A., and Tihomir P. Obrenovitch. "Accumulation of quinolinic acid with euro-inflammation: does it mean excitotoxicity?" Thesis, Kluwer Academic, Plenum Publishers, New York, 2003. http://hdl.handle.net/10454/2833.

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Tutakhail, Abdulkarim. "Potential muscular doping effects of anti-depressants." Thesis, Université Paris-Saclay (ComUE), 2019. http://www.theses.fr/2019SACLS513.

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Bien que l’effet psychotrope des antidépresseurs soit bien connu, afin de corriger les conséquences du stress et de renforcer la confiance en soi, de nombreux autres effets pharmacologiques, notamment périphériques, doivent encore être approfondis. Les antidépresseurs inhibiteurs de la recapture de la sérotonine (ISRS) peuvent avoir un effet bénéfique sur la performance physique en participant à une réparation et à une croissance plus rapides des muscles. Il a récemment été démontré que la sérotonine était impliquée dans la récupération de la force musculaire chez un modèle murin de myopathie

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Pershing, Michelle. "Acute elevations in kynurenic acid result in cognitive inflexibility in an attentinal set-shfiting task via an alpha 7-mediated mechanism." The Ohio State University, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=osu1354032404.

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Milne, Gavin D. S. "Inhibition studies of kynurenine 3-monooxygenase." Thesis, University of St Andrews, 2013. http://hdl.handle.net/10023/4101.

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Kynurenine 3-monooxygenase (K3MO) lies on the kynurenine pathway, the major pathway for the catabolism of L-tryptophan. It converts kynurenine to 3-hydroxy kynurenine. Inhibition of K3MO is important in several neurological diseases and there is evidence that inhibition of K3MO could also be targeted for the prevention of multiple organ failure, secondary to acute pancreatitis. A structure activity relationship based upon the 1,2,4-oxadiazoles motif was carried out which revealed amide 207 as an inhibitor of P. fluorescens K3MO. Further structure activity relationships were developed based upo

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Thevandavakkam, Mathuravani Aaditiyaa. "Deciphering the kynurenine-3-monooxygenase interactome." Thesis, University of Leicester, 2011. http://hdl.handle.net/2381/10070.

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Kynurenine-3-monooxygenase (KMO) is a mitochondrial enzyme in the kynurenine pathway (KP) through which tryptophan is degraded to NAD+. The central KP is altered in neurodegenerative diseases and other CNS disorders. The causative role of KP metabolites has been particularly well studied in the neurodegenerative disorder Huntington’s disease (HD), a fatal adult onset condition inherited in an autosomal dominant manner. In HD, flux in the KP is perturbed such that neurotoxic metabolites (3-hydroxykynurenine and quinolinic acid) of the pathway are increased relative to a neuroprotective metaboli

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Mackay, Gillian Moira. "Kynurenines in neurological disorders." Thesis, University of Glasgow, 2007. http://theses.gla.ac.uk/39/.

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The kynurenine pathway is thought to be involved in neurological disorders but its precise role and the mechanisms involved have yet to be established. Tryptophan can be metabolised via this pathway to produce the neurotoxic N-methyl-D-aspartate (NMDA) receptor agonist, quinolinic acid (QUIN), and the direct generators of reactive oxygen species, 3-hydroxykynurenine (3HKYN) and 3-hydroxyanthranilic acid (3HANA), as well as the neuroprotective NMDA receptor antagonist, kynurenic acid (KYNA). High performance liquid chromatography (HPLC) methods were successfully developed and validated for meas

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Bell, Helen Barbara. "Characterisation of the active site of kynurenine 3-monooxygenase." Thesis, University of Edinburgh, 2016. http://hdl.handle.net/1842/20397.

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Kynurenine 3-monooxygenase (KMO) is a flavoprotein which has been implicated in Huntington’s disease, Alzheimer’s disease and acute pancreatitis. Recently there has been important research published about this enzyme including the structure of a truncated Saccharomyces cerevisiae KMO enzyme and KMO inhibition studies in animal models of disease. In previous work from this research group the complete Pseudomonas fluorescens KMO enzyme has been successfully crystallised both with and without the substrate, L-kynurenine, from which significant insights were gained into function and the potential

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Wilkinson, Martin. "Structural dynamics and ligand binding in kynurenine-3-monooxygenase." Thesis, University of Edinburgh, 2013. http://hdl.handle.net/1842/7965.

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Kynurenine 3-monooxygenase is a FAD-dependent aromatic hydroxylase (FAH) which is a widely suggested therapeutic target for controlling the balance of bioactive metabolite levels produced by the mammalian kynurenine pathway (KP). Prior to starting this work no structural information was known for the enzyme, with studies of the human form complicated by the presence of a C-terminal transmembrane helix. The bacterial Pseudomonas fluorescens enzyme (PfKMO) lacks the transmembrane region and has been previously characterised by Crozier-Reabe and Moran [1, 2]. Therefore PfKMO, which shares 32 % se

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Owe-Young, Robert School of Medicine UNSW. "Kynurenine pathway metabolism at the blood-brain barrier." Awarded by:University of New South Wales. School of Medicine, 2006. http://handle.unsw.edu.au/1959.4/26183.

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A major product of HIV-infected and cytokine-stimulated monocytic-lineage cells is quinolinic acid (QUIN), a neurotoxic metabolite of the kynurenine pathway (KP) of L-tryptophan (L-Trp) metabolism. Despite the large number of neurotoxins found in HIV patients with AIDS Dementia Complex (ADC), only QUIN correlates with both the presence and severity of ADC. With treatment, cerebrospinal fluid (CSF) QUIN concentrations decrease proportionate to the degree of clinical and neuropsychological improvement. As endothelial cells (EC) of the blood-brain barrier (BBB) are the first brain-associated cell

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Taylor, Mark Robert Duncan. "High-resolution structural studies of kynurenine 3-monooxygenase." Thesis, University of Edinburgh, 2018. http://hdl.handle.net/1842/28913.

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The kynurenine pathway produces NAD+ from L-tryptophan. Metabolites known as the kynurenines are produced within the pathway. The effects of the kynurenines have been associated with a number of diseases including cancer, Alzheimer’s disease, Huntington’s disease, and acute pancreatitis. Kynurenine monooxygenase (KMO) is an enzyme that catalyses the conversion of L-kynurenine to 3-hydroxy-L-kynurenine, the downstream product of which is the neurotoxic quinolinic acid. L-kynurenine is positioned at a branching point within the pathway. Metabolism via KMO leads to quinolinic acid production wher

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Books on the topic "Kynureniny"

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Schwarcz, Robert, Simon N. Young, and Raymond R. Brown, eds. Kynurenine and Serotonin Pathways. Springer New York, 1991. http://dx.doi.org/10.1007/978-1-4684-5952-4.

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Mittal, Sandeep, ed. Targeting the Broadly Pathogenic Kynurenine Pathway. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-11870-3.

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W, Stone T., ed. Quinolinic acid and the kynurenines. CRC Press, 1989.

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Robert, Schwarcz, Young Simon N, and Brown Raymond R, eds. Kynurenine and serotonin pathways: Progress in tryptophan research. Plenum Press, 1991.

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Lázló, Vécsei, ed. Kynurenines in the brain: From experiments to clinics. Nova Science Publishers, 2005.

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Rickards, Edward Hugh Galbraith. Plasma kynurenine tryptophan metabolites and associated substances in Gilles de la Tourette's Syndrome. University of Birmingham, 1999.

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Lapin, Izjaslav P. The neuroactivities of kynurenines: Stress, anxiety, depression, alcoholism, epilepsy : the 2000 Oswald Schmiedeberg lecture. Tartu Ülikool, 2000.

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Mirza, Sarwarbeg. The hepatic and the peripheral metabolism of tryptophan via the kynurenine pathway in children with biliary atresiaand with orthotopic liver transplant: The assessment of the relationship between the levels of the kynurenine metabolites, neopterin, biopterin and liver function tests. University of Surrey, 1995.

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Naleem, Wazeer A. A study of urinary kynurenine metabolites in pre-pubertal, pubertal and post-pubertal male offsprings of families with family history negative and family history positive of alcoholism. University of Surrey, 1995.

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Young, Simon N., and Raymond R. Brown. Kynurenine and Serotonin Pathways. Island Press, 1991.

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Book chapters on the topic "Kynureniny"

1

Sewell, A. C. "Kynurenin." In Lexikon der Medizinischen Laboratoriumsdiagnostik. Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-49054-9_1799-1.

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Sewell, A. C. "Kynurenin." In Springer Reference Medizin. Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_1799.

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Schomburg, Dietmar, and Dörte Stephan. "Kynurenine 3-monooxygenase." In Enzyme Handbook. Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-57942-4_91.

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Schomburg, Dietmar, and Dörte Stephan. "Kynurenine-oxoglutarate transaminase." In Enzyme Handbook 13. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-59176-1_46.

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Schomburg, Dietmar, and Dörte Stephan. "Kynurenine-glyoxylate transaminase." In Enzyme Handbook 13. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-59176-1_98.

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Minatogawa, Y., C. Kawai, S. Hatada, and M. Sato. "Liver Specific Kynurenine (Alanine)." In Advances in Experimental Medicine and Biology. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4613-0381-7_73.

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Schomburg, Dietmar, and Dörte Stephan. "Kynurenine 7, 8-hydroxylase." In Enzyme Handbook. Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-57942-4_154.

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Majláth, Zsófia, Levente Szalárdy, Dénes Zádori, et al. "Neuroprotection by Kynurenine Metabolites." In Handbook of Neurotoxicity. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-15080-7_165.

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Rudzite, V., and E. Jurika. "Kynurenine and Lipid Metabolism." In Advances in Experimental Medicine and Biology. Springer New York, 1991. http://dx.doi.org/10.1007/978-1-4684-5952-4_45.

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Majláth, Zsófia, Levente Szalárdy, Dénes Zádori, et al. "Neuroprotection by Kynurenine Metabolites." In Handbook of Neurotoxicity. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-5836-4_165.

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Conference papers on the topic "Kynureniny"

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Sordillo, Laura A., Peter P. Sordillo, Lin Zhang, and Robert R. Alfano. "Tryptophan and kynurenines in neurodegenerative disease." In Bio-Optics: Design and Application. OSA, 2019. http://dx.doi.org/10.1364/boda.2019.jt4a.8.

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Gosker, Harry R., Gerard Clarke, John F. Cryan, and Annemie M. Schols. "Impaired skeletal muscle kynurenine metabolism in patients with COPD." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa940.

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Tanaka, Masaru, Ágnes Szabó, Bálint Lőrinczi, István Szatmári, Ferenc Fülöp, and László Vécsei. "Antidepressant-like Effects of Kynurenic Acid Analogues." In 1st International Electronic Conference on Biomedicine. MDPI, 2021. http://dx.doi.org/10.3390/ecb2021-10301.

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Tharawadeephimuk, Waranan, Chaiyavat Chaiyasut, Sasithorn Sirilun, and Phakkharawat Sittiprapaporn. "Preliminary Study of Probiotics and Kynurenine Pathway in Autism Spectrum Disorder." In 2019 16th International Conference on Electrical Engineering/Electronics, Computer, Telecommunications and Information Technology (ECTI-CON). IEEE, 2019. http://dx.doi.org/10.1109/ecti-con47248.2019.8955380.

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Triplett, Todd A., Kendra Triplett, Everett Stone, et al. "Abstract 5571: Immune-checkpoint inhibition via enzyme-mediated degradation of kynurenine." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-5571.

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Pinto, Sheena, Christoph Steeneck, Michael Albers, et al. "Abstract 1210: Targeting the IDO1-Kynurenine-AhR pathway for cancer immunotherapy." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-1210.

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Pinto, Sheena, Christoph Steeneck, Michael Albers, et al. "Abstract 1210: Targeting the IDO1-Kynurenine-AhR pathway for cancer immunotherapy." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-1210.

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Botticelli, Andrea, Bruna Cerbelli, Luana Lionetto, et al. "Abstract 5705: The key role of kynurenine in anti-PD-1 failure." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-5705.

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Breda, Carlo, Aisha M. Swaih, Mariaelena Repici, and Flaviano Giorgini. "A29 Kynurenine 3-monooxygenase interacts with huntingtin at the outer mitochondrial membrane." In EHDN 2018 Plenary Meeting, Vienna, Austria, Programme and Abstracts. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/jnnp-2018-ehdn.27.

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Poulain-Godefroy, Odile, Mélina Le Roux, Gwenola Kervoaze, Anais Ollivier, Muriel Pichavant, and Philippe Gosset. "Kynurenine pathway as a modulator of inflammation in COPD and its exacerbations." In ERS Lung Science Conference 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/23120541.lsc-2022.61.

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