Relevant bibliographies by topics / L4

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'L4'

Author: Grafiati
Published: 4 June 2021
Last updated: 18 February 2022

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Journal articles on the topic "L4"

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Heiser, Gernot, and Kevin Elphinstone. "L4 Microkernels." ACM Transactions on Computer Systems 34, no. 1 (April 6, 2016): 1–29. http://dx.doi.org/10.1145/2893177.

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Scholz, Matti, Philipp Schleicher, and Frank Kandziora. "Instrumented lumbar interbody fusion L4–S1 (TLIF L4–S1)." European Spine Journal 26, S3 (January 23, 2017): 416–17. http://dx.doi.org/10.1007/s00586-016-4934-1.

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Mao, Ren, Rui-Han Tang, Yun Qiu, Bai-Li Chen, Jing Guo, Sheng-Hong Zhang, Xue-Hua Li, et al. "Different clinical outcomes in Crohn’s disease patients with esophagogastroduodenal, jejunal, and proximal ileal disease involvement: is L4 truly a single phenotype?" Therapeutic Advances in Gastroenterology 11 (January 1, 2018): 175628481877793. http://dx.doi.org/10.1177/1756284818777938.

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Background: The Montreal classification defines L4 Crohn’s disease (CD) as any disease location proximal to the terminal ileum, which anatomically includes L4-esophagogastroduodenal (EGD), L4-jejunal, and L4-proximal ileal involvement. L4-jejunal disease was established to be associated with poor prognosis. However, the outcome of patients with L4-proximal ileal disease or L4-EGD remains to be clarified. Our study aimed to investigate whether the outcome differs among CD patients with L4-EGD, L4-jejunal, and L4-proximal ileal disease. Methods: In our retrospective cohort study, 483 patients with confirmed CD were included. The primary outcome was intestinal surgery. Demographic features and outcomes were compared among L4-EGD, L4-jejunal, and L4-proximal ileal disease. Results: Thirty-nine (8.1%) patients had isolated L4 disease, whereas 146 patients had L4 as well as concomitant L1, L2, or L3 disease. During a median follow up of 5.8 years, L4 patients were more likely to have intestinal surgeries compared to non-L4 patients (31% versus 16%, p < 0.001). The percentage of L4-jejunal patients who underwent surgery was higher than that of L4-proximal ileal (66% versus 28%, p < 0.001), and both of these subtypes of L4 were at higher risk for intestinal resection compared to L4-EGD patients (66% and 28% versus 9%, respectively, p < 0.001 and p < 0.05). On multi-variable analysis, L4-jejunal (HR 3.08; 95% CI 1.30–7.31) and L4-proximal ileal disease (HR 1.83; 95% CI 1.07–3.15) were independent predictors for intestinal resection. Conclusions: L4 disease had worse prognosis compared to non-L4 disease. Within L4 disease, phenotype of L4-jejunal and L4-proximal ileal disease indicated higher risk for intestinal surgery. It might be justified to further characterize the L4 phenotype of the Montreal classification into three specific subgroups including L4-EGD, L4-jejunal, and L4-proximal ileal disease, similar to the Paris classification of pediatric patients.
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Zhou, Tian-Hua, Xun Tang, Yong-Qing Xu, and Yue-Liang Zhu. "Traumatic Spondyloptosis of L4." Spine 35, no. 17 (August 2010): E855—E859. http://dx.doi.org/10.1097/brs.0b013e3181d798f2.

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Grobler, L. J., J. E. Novotny, D. G. Wilder, J. W. Frymoyer, and M. H. Pope. "L4–5 Isthmic Spondylolisthesis." Spine 19, Supplement (January 1994): 222–27. http://dx.doi.org/10.1097/00007632-199401001-00018.

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HERRON, LARRY D., and ANTHONY C. TRIPPI. "L4-5 Degenerative Spondylolisthesis." Spine 14, no. 5 (May 1989): 534–38. http://dx.doi.org/10.1097/00007632-198905000-00013.

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Yang, Mu-Zi, Xue Hou, Ji-Bin Li, Jing-Sheng Cai, Jie Yang, Shuo Li, Hao Long, et al. "Impact of L4 lymph node dissection on long-term survival in left-side operable non-small-cell lung cancer: a propensity score matching study." European Journal of Cardio-Thoracic Surgery 57, no. 6 (February 4, 2020): 1181–88. http://dx.doi.org/10.1093/ejcts/ezaa008.

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Abstract OBJECTIVES We investigated the impact of level 4 (L4) lymph node dissection (LND) on overall survival (OS) in left-side resectable non-small-cell lung cancer (NSCLC), with the aim of guiding lymphadenectomy. METHODS A total of 1929 patients with left-side NSCLC who underwent R0 resection between 2001 and 2014 were included in the study. The patients were divided into a group with L4 LND (L4 LND+) and a group without L4 LND (L4 LND−). Propensity score matching was applied to minimize selection bias. The Kaplan–Meier method and Cox proportional hazards model were used to assess the impact of L4 LND on OS. RESULTS A total of 317 pairs were matched. Of the cohort of patients, 20.3% (391/1929) had L4 LND. Of these patients, 11.8% (46/391) presented with L4 lymph node metastasis. L4 lymph node metastasis was not associated with the primary tumour lobes (P = 0.61). Before propensity score matching, the 5-year OS was comparable between the L4 LND+ and L4 LND− groups (69.0% vs 65.2%, P = 0.091). However, after propensity score matching, the 5-year OS of the L4 LND+ group was much improved compared to that of the L4 LND− group (72.9% vs 62.3%, P = 0.002) and L4 LND was an independent factor favouring OS (hazard ratio 0.678, 95% confidence interval 0.513–0.897; P = 0.006). Subgroup analysis suggested that L4 LND was an independent factor favouring OS in left upper lobe tumours. CONCLUSIONS In patients with left-side operable NSCLC, L4 lymph node metastasis was not rare and L4 LND should be routinely performed.
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Morris, Susan J., and Keith N. Leppard. "Adenovirus Serotype 5 L4-22K and L4-33K Proteins Have Distinct Functions in Regulating Late Gene Expression." Journal of Virology 83, no. 7 (January 28, 2009): 3049–58. http://dx.doi.org/10.1128/jvi.02455-08.

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ABSTRACT Adenoviruses express up to 20 distinct mRNAs from five major late transcription unit (MLTU) regions, L1 to L5, by differential splicing and polyadenylation of the primary transcript. MLTU expression is regulated at transcriptional and posttranscriptional levels. The L4-33K protein acts as a splicing factor to upregulate several MLTU splice acceptor sites as the late phase progresses. The L4 region also expresses a 22K protein whose sequence is related to the sequence of L4-33K. L4-22K is shown here also to have an important role in regulating the pattern of MLTU gene expression. An adenovirus genome containing a stop codon in the L4-22K open reading frame expressed low levels of both structural and nonstructural late proteins compared to the wild-type (wt) adenovirus genome; a decrease in intermediate proteins, IVa2 and IX, was also observed. However, early protein synthesis and replication were unaffected by the absence of L4-22K. Intermediate and late protein expression was restored to wt levels by L4-22K expressed in trans but not by L4-33K. Increased MLTU promoter activity, resulting from stabilization of the transcriptional activator IVa2 by L4-22K, made a small contribution to this restoration of late gene expression. However, the principal effect of L4-22K was on the processing of MLTU RNA into specific cytoplasmic mRNA. L4-22K selectively increased expression of penton mRNA and protein, whereas splicing to create penton mRNA is known not to be increased by L4-33K. These results indicate that L4-22K plays a key role in the early-late switch in MLTU expression, additional to and distinct from the role of L4-33K.
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PALMANS V, VAN CALENBERGH F, and VAN LOON J. "Traumatische avulsiefractuur van de ringapofyse L4 bij een patiënt met lysis van L4." Tijdschrift voor Geneeskunde 57, no. 4 (January 1, 2001): 300–304. http://dx.doi.org/10.2143/tvg.57.4.5000977.

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Hodges, Scott D., S. Craig Humphreys, Jason C. Eck, and Laurie A. Covington. "The Surgical Treatment of Far Lateral L3–L4 and L4–L5 Disc Herniations." Spine 24, no. 12 (June 1999): 1243–46. http://dx.doi.org/10.1097/00007632-199906150-00012.

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Dissertations / Theses on the topic "L4"

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Backström, Ellenor. "Regulation of Adenoviral Gene Expression by the L4-33K and L4-22K Proteins." Doctoral thesis, Uppsala universitet, Institutionen för medicinsk biokemi och mikrobiologi, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-101324.

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The splicing pattern during an adenovirus infection is shifted at the late phase towards using weaker splice sites, splicing out larger introns. Splicing of weak 3´ splice sites usually requires recognition of the 3´AG dinucleotide before the first catalytic step of splicing. Such splicing events are said to be AG-dependent and requires an interaction of both subunits of the cellular splicing factor U2AF with the 3´ splice site. We show that splicing of transcripts that are AG-dependent in uninfected nuclear extracts (NE) becomes AG-independent in nuclear extracts prepared form adenovirus late-infected HeLa cells (Ad-NE). Further we demonstrate that the first step in splicing of a model transcript, IgM, becomes completely U2AF-independent in Ad-NE. This finding supports our working model that 3´ splice site recognition in Ad-NE is altered, and in fact might be U2AF-independent. We further show that the adenovirus late protein L4-33K acts as a virus encoded alternative splicing factor. L4-33K activates splicing of both cellular and viral transcripts containing weak 3´ splice sites. This supports the hypothesis that adenovirus alter splicing during the infection to favour usage of weak, suboptimal 3´ splice sites. However, we were unable to find an alternative U2AF-related factor that could stimulate L4-33K splicing enhancer activity. Furthermore, we demonstrate that the serine residues in the C-terminal part of L4-33K are important for the splicing enhancer activity but also for its nuclear localisation. The adenovirus major late promoter is highly activated after the onset of viral genome replication. Protein complexes binding to downstream elements of the promoter are required for full enhancement of this promoter. We show that an L4-33K-related protein, L4-22K, stimulates transcription from the major late promoter. This stimulation is mainly via the downstream elements and does not require the viral IVa2 protein, which is a transcription factor of the major late promoter.
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Östberg, Sara. "Functional Characterization of the Evolutionarily Conserved Adenoviral Proteins L4-22K and L4-33K." Doctoral thesis, Uppsala universitet, Science for Life Laboratory, SciLifeLab, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-238487.

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Regulation of adenoviral gene expression is a complex process directed by viral proteins controlling a multitude of different activities at distinct phases of the virus life cycle. This thesis discusses adenoviral regulation of transcription and splicing by two proteins expressed at the late phase: L4-22K and L4-33K. These are closely related with a common N-terminus but unique C-terminal domains. The L4-33K protein is an alternative RNA splicing factor inducing L1-IIIa mRNA splicing, while L4-22K is stimulating transcription from the major late promoter (MLP). The L4-33K protein contains a tiny RS-repeat in its unique C-terminal end that is essential for the splicing enhancer function of the protein. Here we demonstrate that the tiny RS-repeat is required for localization of the protein to the nucleus and viral replication centers. Further, we describe an auto-regulatory loop where L4-33K enhances splicing of its own intron. The preliminary characterization of the responsive RNA-element suggests that it differs from the previously defined L4-33K-responsive element activating L1-IIIa mRNA splicing. L4-22K lacks the ability to enhance L1-IIIa splicing in vivo, and here we show that the protein is defective in L1-IIIa or other late pre-mRNA splicing reactions in vitro. Interestingly, we found a novel function for the L4-22K and L4-33K proteins as regulators of E1A alternative splicing. Both proteins selectively upregulated E1A-10S mRNA accumulation in transfection experiments, by a mechanism independent of the tiny RS-repeat. Although L4-22K is reported to be an MLP transcriptional enhancer protein, here we show that L4-22K also functions as a repressor of MLP transcription. This novel activity depends on the integrity of the major late first leader 5’ splice site. The model suggests that at low concentrations L4-22K activates MLP transcription while at high concentrations L4-22K represses transcription. So far, characterizations of the L4-22K and L4-33K proteins have been limited to human adenoviruses 2 or 5 (HAdV-2/5). We expanded our experiments to include HAdV-3, HAdV-4, HAdV-9, HAdV-11 and HAdV-41. The results demonstrated that the transcription- or splicing-enhancing properties of L4-22K and L4-33K, respectively, are evolutionarily conserved and non-overlapping. Thus, the sequence-based conservation is mirrored by the functions, as expected for functionally important proteins.
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Backström, Ellenor. "Regulation of Adenoviral Gene Expression by the L4-33K and L4-22K Proteins." Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-101324.

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Kolarík, Tomáš. "Implementace protokolu ACP do operačního systému L4." Master's thesis, Vysoké učení technické v Brně. Fakulta elektrotechniky a komunikačních technologií, 2012. http://www.nusl.cz/ntk/nusl-219477.

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This thesis deals with the implementation of ACP protocol which serves to manage the access for operation system based on L4 microkernel. The theoretical part of the thesis deals with methods of access management in computer networks. It focuses primarily on AAA systems which make access management possible. Furthermore it describes in detail the ACP protocol, the types of messages and their feedback. The next theoretical part is dedicated to operation systems and in particular to their architecture and services. Then we get a closer look at L4 microkernel family, their philosophy and properties. We continue with a detailed description of the L4 application interface and its ways of expansion. The practical section deals with the implemented concept of system for ACP protocol support in computers. General concept is then applied in real implementation of ACP protocol into the L4 operation system environment based on the L4 platform. To assist, I also included a detailed tutorial explaining the modeling and compilation of software for this platform. At this point we describe the methods used at the implementation and the description of particular modules and features. The end of the thesis concludes the information about the ways of testing and the implementation properties.
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Trifa, Youssef. "Caractérisation de la protéine ribosomique L4 du plaste d'épinard." Grenoble 1, 1998. http://www.theses.fr/1998GRE10204.

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Le genome plastidial est transcrit par deux arn polymerases, l'une monomerique, codee dans le noyau (nep), l'autre multimerique codee par le genome plastidial (pep). La transcription de l'operon rrn plastidial est assuree, in vivo, par la nep. Le facteur cdf2 est implique dans la regulation de l'expression de cet operon, en inhibant sa transcription par pep. Dans le but de caracteriser des proteines impliquees dans la regulation de la transcription plastidiale, nous avons debute l'analyser des proteines qui co-purifient avec le facteur dcf2 et la nep. Une proteine ribosomique (cd2) a ainsi ete purifiee et sa sequence n-terminale determinee. Nous avons clone et identifie l'adn complementaire ainsi que le gene correspondant a cette proteine. Nous avons pu montrer que le gene identifie code pour la proteine l4 de grande sous unite 50s du ribosome plastidial d'epinard (spinacia oleracea). Il s'agit du premier gene nucleaire isole codant pour la proteine rpl4 d'une plante superieure. A l'image d'autres genes nucleaires codant pour des proteines ribosomiques plastidiales, nous avons montre que le gene rpl4 identifie est unique et s'exprime plus dans les tissus a capacite photosynthetique que dans les racines. La proteine rpl4 d'epinard est homologue aux proteines ribosomiques l4 de procaryotes mais s'y distingue par la presence d'une extension c-terminale acide. La proteine ribosomique l4 d'escherichia coli provoque une terminaison prematuree de la transcription de l'operon s10 au niveau d'un site de pause. Malgre la divergence avec celle d'escherichia coli, la proteine ribosomique l4 du chloroplaste d'epinard a conserve au cours de l'evolution la capacite de stimuler la pause de l'arn polymerase d'e. Coli au site d'attenuation de l'operon s10. Cette propriete laisse supposer que la proteine l4 joue un role dans la regulation de l'activite transcriptionnelle par la pep. Dans les plastes d'epinard. Rpl4 semble egalement moduler la transcription, in vitro, de l'operon rrn par la nep. Par son interaction avec les deux arn polymerases plastidiales, rpl4 semble etre une proteine-cle de la regulation transcriptionnelle plastidiale.
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Lan, Susan. "The Multifunctional Nature of the Adenovirus L4-22K Protein." Doctoral thesis, Uppsala universitet, Institutionen för medicinsk biokemi och mikrobiologi, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-278135.

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The adenovirus major late transcription unit (MLTU) encodes for most of the mRNAs that are translated into the structural proteins of the virus capsid. Transcription from the MLTU is directed by the major late promoter (MLP), which is highly activated during the late phase of infection. This thesis discusses how the adenovirus-encoded L4-22K protein regulates the MLP at both the level of transcription and pre-mRNA splicing. The study shed new light on the complex regulation of the early to late shift of adenoviral gene expression. Here we show that the L4-22K protein has opposing effects on MLP transcription, functioning both as an activator and a repressor protein. The stimulatory effect mainly depends on the direct interaction of the L4-22K protein with the downstream element (DE element) located approximately 100 nucleotides downstream of the transcription initiation site. In addition to the DE element we also show that the promoter-proximal upstream element (UPE) acts as an L4-22K responsive enhancer element in the MLP. Preliminary data suggests that the activation of MLP transcription via DE and UPE differs mechanistically. The transactivation domain of the L4-22K protein is localized to the conserved carboxy-terminus of the protein. Our results also defined a novel low affinity L4-22K binding site, the R1 region, which functions as a repressor element in MLP transcription. At high concentrations L4-22K binds to R1 and recruits the cellular transcription factor Sp1 to a DNA segment covering the major late first leader 5´ splice site that is embedded in the R1 region. Sp1 binding to R1 results in a suppression of L4-22K-mediated activation of MLP transcription. This self-limiting effect on MLP transcription might have a function to fine-tune the MLTU gene expression. Interestingly, the L4-22K protein binds with the same sequence specificity to both the R1 double-stranded DNA and R1 single-stranded RNA (ssRNA). L4-22K binds to the R1 ssRNA with the same polarity as the MLTU nascent RNA. This binding results in the recruitment of U1 snRNA to the major late first leader 5´ splice site. This enhanced U1 snRNA recruitment leads to a suppression of MLP transcription and simultaneously an increase of major late first intron splicing.
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Borowski, Stephan. "Vergleichende intrathekale Volumenmessungen an LWS Präparaten des Menschen mit degenerativer Spinalkanalstenose zwischen interlaminärer Dekompression L3/4, interlaminärer Dekompression L4/5 und Laminektomie L4." [S.l.] : [s.n.], 2003. http://deposit.ddb.de/cgi-bin/dokserv?idn=972109528.

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Sharghi-Liebeck, Sanaz [Verfasser]. "Deutsch als L4 für Muttersprachler des Persischen / Sanaz Sharghi-Liebeck." Kassel : Universitätsbibliothek Kassel, 2016. http://d-nb.info/109387029X/34.

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Arnelöv, Eric. "Design and Implementation of an EDF Scheduer for Fiasco.OC L4." Thesis, Uppsala universitet, Institutionen för informationsteknologi, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-301952.

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This paper describes the implementation of an EDF scheduler for the FIASCO microkernel. Benchmark test shows that the average case gain an improvement of 16-34%, in regards to schedule calls, running EDF over the existing fixed priority scheduler using Rate Monotonic (RM). A possible approach to mimic EDF scheduling with fixed priority is described using dynamic counters and worst case ready queue. The advantages and disadvantages are compared to the Fixed Priority with the Priority Promotion algorithm.
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Ecomard, Jean-Christophe. "Approche synthétique du L4-COOH-hexanor-LTE4 : Implication dans l'Asthme." Montpellier 2, 2004. http://www.theses.fr/2004MON20182.

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Books on the topic "L4"

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Dematra, Damien. L4 lupus: A novel. Jakarta: Gerakan Nasional Menulis, 2011.

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Chang, Albert. Identification and characterization of a novel kallikrein gene Klk-L4/KLK13. Ottawa: National Library of Canada, 2001.

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Konperensi Internasional Tentang Pengembangan Sumberdaya Manusia Dalam Kerangka Kemitraan Internasional (1994 Jakarta, Indonesia). Laporan Konperensi Internasional Tentang Pengembangan Sumberdaya Manusia Dalam Kerangka Kemitraan Internasional, Jakarta, l4-16 September 1994. Jakarta: CIDES, 1994.

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Paul, Rik. Chevrolet citation owners workshop manual: Models covered, Chevrolet Citation Hatchback Sedan, Hatchback Coupe, Club Coupe and XII models with 2.5 liter (151 cu in) L4 and 2.8 liter (173 cu in) V6 engines 1980 thru 1985. Sparkford: Haynes, 1985.

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Ganeri, Anita. Kingfisher Readers L4: Sharks. Kingfisher, 2012.

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Ganeri, Anita. Kingfisher Readers L4: Weather. Kingfisher, 2012.

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Oxlade, Chris. Kingfisher Readers L4: Flight. Kingfisher, 2012.

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Oxlade, Chris. Kingfisher Readers L4: Flight. Kingfisher, 2012.

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Ganeri, Anita. Kingfisher Readers L4: Sharks. Kingfisher, 2012.

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Huggins-Cooper, Lynn. DK Readers L4 Robot Universe. DK Children, 2017.

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Book chapters on the topic "L4"

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Stoll, Kurt E., Daniel A. Marchwiany, Daniel L. Cavanaugh, and Gurvinder S. Deol. "Lateral Lumbar Interbody Fusion L3–L4, L4–L5." In The Resident's Guide to Spine Surgery, 163–70. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-20847-9_19.

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Wiese, Harald. "Lerneffekte bei Mengenwettbewerb (L4)." In Lern- und Netzeffekte im asymmetrischen Duopol, 226–45. Heidelberg: Physica-Verlag HD, 1993. http://dx.doi.org/10.1007/978-3-642-95896-0_12.

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Katkhouda, Namir, Sanjay Ghosh, and Srinath Samudrala. "Laparoscopic Approach to the L3–L4 and L4–L5 Intervertebral Discs." In Surgical Approaches to the Spine, 185–90. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2465-3_30.

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Katkhouda, Namir, Sanjay Ghosh, and Srinath Samudrala. "Laparoscopic Approach to the L3-L4 and L4-L5 Intervertebral Discs." In Surgical Approaches to the Spine, 337–44. New York, NY: Springer New York, 2003. http://dx.doi.org/10.1007/978-1-4613-0009-0_42.

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Klein, Gerwin. "The L4.verified Project — Next Steps." In Verified Software: Theories, Tools, Experiments, 86–96. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-15057-9_6.

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Watkins, Robert G. "Transperitoneal Midline Approach to L4–S1." In Surgical Approaches to the Spine, 179–84. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2465-3_29.

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Watkins, Robert G. "Transperitoneal Midline Approach to L4-51." In Surgical Approaches to the Spine, 195–201. New York, NY: Springer New York, 2003. http://dx.doi.org/10.1007/978-1-4613-0009-0_27.

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Räbiger, Norbert, Michael Schlüter, Alfons Mersmann, Hans Detlef Dahl, Andrea Luke, Peter Walzel, and Emir Musemic. "L4 Blasen und Tropfen in technischen Apparaten." In VDI-Wärmeatlas, 1413–58. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-19981-3_92.

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Schmidt, D. S. "The Stability of the Lagrangian Point L4." In Applications of Computer Technology to Dynamical Astronomy, 201–6. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-0985-4_34.

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Räbiger, Norbert, Michael Schlüter, Alfons Mersmann, and Hans Detlef Dahl. "L4 Bubble and Drops in Technical Equipment." In VDI Heat Atlas, 1239–70. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-77877-6_89.

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Conference papers on the topic "L4"

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Armoni, Roy, Amnon Ta-Shma, Avi Wigderson, and Shiyu Zhou. "SL ⊆L4/3." In the twenty-ninth annual ACM symposium. New York, New York, USA: ACM Press, 1997. http://dx.doi.org/10.1145/258533.258593.

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Ravindu, G. U., G. Kosalishkwaran, S. Parasuraman, and I. Elamvazuthi. "Spine (L3-L4) Mechanics." In 2018 IEEE 4th International Symposium in Robotics and Manufacturing Automation (ROMA). IEEE, 2018. http://dx.doi.org/10.1109/roma46407.2018.8986715.

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Stoffer, Peter, Gilberto Colangelo, and Emilie Passemar. "A dispersive treatment of $K_{l4}$ decays." In The 8th International Workshop on Chiral Dynamics. Trieste, Italy: Sissa Medialab, 2016. http://dx.doi.org/10.22323/1.253.0067.

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Oguchi, K., N. Mochizuki, A. Matsumoto, and S. Date. "New Mitsubishi L4 5-Liter DI Diesel Engine." In International Truck & Bus Meeting & Exposition. 400 Commonwealth Drive, Warrendale, PA, United States: SAE International, 1998. http://dx.doi.org/10.4271/982800.

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Bang, Ki-Seok, Su-Young Lee, Ki-Hyuk Nam, Wan-Yeon Lee, and Young-Woong Ko. "Verifying Behavior of L4 Microkernel based Mobile Phone." In The 9th International Conference on Advanced Communication Technology. IEEE, 2007. http://dx.doi.org/10.1109/icact.2007.358317.

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Zhou, Qingguo, Canyu Li, Ying Ding, Guanghui Cheng, and Bin Hu. "The analysis of L4 Linux implementation for education." In Education (ITIME). IEEE, 2009. http://dx.doi.org/10.1109/itime.2009.5236446.

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Kosalishkwaran, G., S. Parasuraman, I. Elamvazuthi, and J. George. "Imaging and Modelling of L3-L4 Spine: Simulation." In 2018 International Conference on Intelligent and Advanced System (ICIAS). IEEE, 2018. http://dx.doi.org/10.1109/icias.2018.8540611.

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Benjamin, Namitha, Robert Boutin, Abhijit Chaudhari, and Kwan-Liu Ma. "Genetic Algorithm based L4 Identification and Psoas Segmentation." In 8th International Conference on Bioimaging. SCITEPRESS - Science and Technology Publications, 2021. http://dx.doi.org/10.5220/0010207701200127.

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Moreno, Richard, and Francois Bermudo. "SMOS L3 and L4 Data Processing, Archiving and Operations." In SpaceOps 2008 Conference. Reston, Virigina: American Institute of Aeronautics and Astronautics, 2008. http://dx.doi.org/10.2514/6.2008-3534.

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Kim, Dong-Guen, Sang-Min Lee, and Dong-Ryeol Shin. "Design of the Operating System Virtualization on L4 Microkernel." In 2008 Fourth International Conference on Networked Computing and Advanced Information Management (NCM). IEEE, 2008. http://dx.doi.org/10.1109/ncm.2008.165.

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Reports on the topic "L4"

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Boszcyk, Bronek. L4-L5 Disc Replacement. Touch Surgery Simulations, May 2018. http://dx.doi.org/10.18556/touchsurgery/2018.s0082.

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Vialle, Emiliano. AO L4/5 Degenerative Spondylolisthesis Open TLIF. Touch Surgery Simulations, 2018. http://dx.doi.org/10.18556/touchsurgery/2018.s0092.

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Touch Surgery. AO L4/5 Degenerative Spondylolisthesis Open TLIF. Touch Surgery Publications, July 2018. http://dx.doi.org/10.18556/touchsurgery/2018.s0135.

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Gebhard, Harry, and Cristiano Magalhães Menezes. AO Lateral Lumbar Interbody Fusion (LLIF) L3/L4. Touch Surgery Simulations, February 2021. http://dx.doi.org/10.18556/touchsurgery/2021.s0178.

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Loomis, Eric Nicholas, Paul Andrew Bradley, Elizabeth Catherine Merritt, Joyce Ann Guzik, and Patrick Hagen Denne. L4 Milestone Report for MixEOS 2016 experiments and simulations. Office of Scientific and Technical Information (OSTI), August 2016. http://dx.doi.org/10.2172/1296705.

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Parajon, Avelino, and Roger Härtl. AO L4/5–Degenerative spondylolisthesis MIS TLIF using AP/lateral fluoroscopy. Touch Surgery Simulations, 2018. http://dx.doi.org/10.18556/touchsurgery/2018.s0090.

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You might also be interested in the extended bibliographies on the topic 'L4' for particular source types:
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