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Journal articles on the topic 'Laboratorní diagnostika'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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1

Seitz, H. M. "Die mikroskopische Malaria-Diagnostik/Microscopic Malaria Diagnostics." LaboratoriumsMedizin 27, no. 9/10 (January 1, 2003): 398–400. http://dx.doi.org/10.1515/labmed.2003.055.

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2

Klingmüller, D., B. Saller, and H. J. Quabbe. "Diagnostik von Hypophysenadenomen / Diagnostics of pituitary adenomas." LaboratoriumsMedizin 28, no. 2 (January 14, 2004): 122–26. http://dx.doi.org/10.1515/labmed.2004.020.

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3

Rauer, Sebastian, Oliver Stich, and Christian Bogdan. "Mikrobiologische Liquor-Diagnostik Microbiological diagnostics in CSF." LaboratoriumsMedizin 29, no. 6 (January 1, 2005): 434–38. http://dx.doi.org/10.1515/jlm.2005.059.

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4

Lindsay, LR, K. Bernat, and A. Dibernardo. "Diagnostic en laboratoire de la maladie de Lyme." Relevé des maladies transmissibles au Canada 40, no. 11 (May 29, 2014): 232–41. http://dx.doi.org/10.14745/ccdr.v40i11a02f.

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5

Brandt, B. H. "Genetische Diagnostik hereditarer Kolonkarzinome. Genetic Diagnostics for Hereditary Colon Cancer." Laboratoriums Medizin 27, no. 3-4 (May 2003): 114–21. http://dx.doi.org/10.1046/j.1439-0477.2003.03018.x.

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6

Brandt, B. H. "Genetische Diagnostik hereditärer Kolonkarzinome/Genetic Diagnostics for Hereditary Colon Cancer." LaboratoriumsMedizin 27, no. 3/4 (January 1, 2003): 114–21. http://dx.doi.org/10.1515/labmed.2003.017.

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7

Fingerle, Volker, and Bettina Wilske. "Mikrobiologische Diagnostik der Lyme-Borreliose / Microbiological diagnostics of Lyme borreliosis." LaboratoriumsMedizin 31, no. 3 (June 2007): 141–48. http://dx.doi.org/10.1515/jlm.2007.019.

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8

Popovic, Danica, and Mirko Popovic. "Patient Safety and Rational Laboratory Diagnostic." Paripex - Indian Journal Of Research 3, no. 2 (January 15, 2012): 251–52. http://dx.doi.org/10.15373/22501991/feb2014/86.

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9

Marti, Ulrich. "Kapillarelektrophorese in der Urin-Diagnostik Urine diagnostics by capillary electrophoresis." LaboratoriumsMedizin 29, no. 5 (January 1, 2005): 325–30. http://dx.doi.org/10.1515/jlm.2005.044.

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10

Schoch, Claudia, Wolfgang Kern, Susanne Schnittger, and Torsten Haferlach. "FISH-Diagnostik bei hämatologischen Neoplasien FISH diagnostics in hematological malignancies." LaboratoriumsMedizin 29, no. 5 (January 1, 2005): 335–42. http://dx.doi.org/10.1515/jlm.2005.047.

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11

Dołęga, Boguslaw, and Tomasz Rogalski. "DIAGNOSTICS OF FLY-BY-WIRE CONTROL SYSTEM/KOMPIUTERIU REGULIUOJAMOS VALDYMO SISTEMOS DIAGNOSTIKA." Aviation 12, no. 2 (June 30, 2008): 41–45. http://dx.doi.org/10.3846/1648-7788.2008.12.41-45.

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The paper describes the diagnostics of a fly‐by‐wire control system taken during the creation of this system. Special attention is paid to laboratory tests, which should prove the system could be tested in‐flight. An important kind of verification of the system is testing and reconfiguring fault detection and localization methods. The human factor is also taken into consideration. Santrauka Straipsnyje apibūdinama kompiuteriu reguliuojamos valdymo sistemos diagnostika šios sistemos kūrimo metu. Ypatingas dėmesys skirtas laboratoriniams bandymams, kurių rezultatai turi būti patikrinti skrydžio metu. Vienas svarbiausių sistemos verifikavimo būdų yra gedimų nustatymo ir lokalizavimo metodų bandymas. Taip pat įvertinamas ir žmogiškasis faktorius.
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12

Höppner, W. "Voraussetzungen und Methoden molekulargenetischer Diagnostik. Prerequisites and Methods of Molecular Genetic Diagnostics." LaboratoriumsMedizin 25, no. 11-12 (January 2001): 453–62. http://dx.doi.org/10.1515/labm.2001.25.11-12.453.

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13

Biskup, Saskia. "Hochdurchsatz-Sequenzierung in der Humangenetischen Diagnostik / Next-generation sequencing in genetic diagnostics." LaboratoriumsMedizin 34, no. 6 (January 1, 2010): 305–9. http://dx.doi.org/10.1515/jlm.2010.056.

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14

Kochanová, M., E. Prokinová, and P. Ryšánek. "Laboratory diagnostics of common bunt and dwarf bunt." Czech Journal of Genetics and Plant Breeding 42, Special Issue (August 1, 2012): 75–77. http://dx.doi.org/10.17221/6237-cjgpb.

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15

Щербо, С. Н., and Д. С. Щербо. "Лабораторная диагностика как основа медицины 5П." Laboratory diagnostics, no. 1 (February 14, 2019): 5–14. http://dx.doi.org/10.24075/vrgmu.2018.095.

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В рамках модернизации системы здравоохранения в настоящее время происходит изменение старых и становление новых направлений современной медицины: персонализированной, предиктивной, превентивной, партисипативной, прецизионной, мобильной и цифровой. Вместе с тем, в научной литературе и медицинском сообществе имеются различные мнения о целях, области применения и задачах указанных направлений здравоохранения и роли в них лабораторной диагностики. Прежде всего это относится к прецизионной медицине, которая воспринимается только как более современное название персонализированной медицины. В обзоре проанализирована и обобщена имеющаяся на сегодняшний день информация и представлены примеры применения подходов медицины 5П в клинической практике.
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16

Djurisic, Slavko, Sava Lazic, Tamas Petrovic, Sara Savic-Jevdjenic, and Diana Lupulovic. "Immunoenzyme: Elisa diagnostics in veterinary medicine." Veterinarski glasnik 57, no. 1-2 (2003): 63–72. http://dx.doi.org/10.2298/vetgl0302063d.

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The ELISA technique is one of the most commonly used laboratory-diagnostical methods for diagnosing many infective diseases in humans and animals today. Due to its high specificity and sensitivity this technique is considered a method of choice in diagnosing many viral diseases. The phases and components of the ELISA technique are constantly improved because of its mass use, as well as the high demands of laboratory diagnostics. Therefore it is necessary to continually inform the expert public of all the innovations, forms and possibilities of the ELISA technique. This paper describes the factors that caused the genesis of the ELISA technique and shows its forms. The methodological and practical aspects of the ELISA technique, when applied in laboratory diagnostics of veterinary medicine are also presented.
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17

Herrmann, B. L., K. Mann, and O. E. Janssen. "Diagnostik des Wachstumshormonmangels und des Wachstumshormonexzesses (Akromegalie) / Diagnostics of growth hormone deficiency and excess (acromegaly)." LaboratoriumsMedizin 28, no. 2 (January 14, 2004): 127–34. http://dx.doi.org/10.1515/labmed.2004.021.

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18

Klein, H. G., and U. Grau. "Arzneimittelnebenwirkungen vermeiden: Möglichkeiten der pharmakogenetischen Diagnostik. Prevention of Adverse Drug Reactions: Potential of Pharmacogenetic Diagnostics." LaboratoriumsMedizin 25, no. 11-12 (January 2001): 477–84. http://dx.doi.org/10.1515/labm.2001.25.11-12.477.

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19

Amvrosieva, Tamara, and Natallia Paklonskaya. "COVID-19: Laboratory diagnostics." Science and Innovations 7, no. 209 (July 2020): 22–27. http://dx.doi.org/10.29235/1818-9857-2020-7-22-27.

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20

Kochetov, A. G., O. V. Lyang, I. A. Zhirova, and O. O. Ivoilov. "Laboratory diagnostics in medicine." Terapevticheskii arkhiv 92, no. 4 (May 19, 2020): 4–8. http://dx.doi.org/10.26442/00403660.2020.04.000501.

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The development of clinical laboratory diagnostics is in line with the evidence-based medicine, which requires that clinical decisions have to be based on diagnostic methods with proven informativity. This creates a request for the scientific validity of the use of laboratory researches and application of probabilistic interpretation tools corresponding to the tasks. The concept of indefiniteness (analytical, biological and clinical) is at the heart of interpretation of laboratory results. The inclusion of laboratory research in clinical guidelines, the choice and appointment of this research to the patient should not be made from the position of ideas about increasing or decreasing the laboratory index in the disease, but on the basis of its scientifically proven characteristics as a laboratory biomarker sensitivity, specificity, predictive value, as well as the relationship with certain clinical events, outcomes, risks. These characteristics are probabilistic and can be defined.
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21

Grishina, M. A., E. N. Kochubeeva, N. V. V'yuchnova, V. A. Antonov, G. A. Tkachenko, V. V. Alekseev, and A. V. Lipnitsky. "Laboratory Diagnostics of Coccidioidomycosis." Problems of Particularly Dangerous Infections, no. 1(111) (February 20, 2012): 70–76. http://dx.doi.org/10.21055/0370-1069-2012-1(111)-70-76.

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22

Bujak, Renata, Wiktoria Struck-Lewicka, Michał J. Markuszewski, and Roman Kaliszan. "Metabolomics for laboratory diagnostics." Journal of Pharmaceutical and Biomedical Analysis 113 (September 2015): 108–20. http://dx.doi.org/10.1016/j.jpba.2014.12.017.

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23

Busse, Birgit, and Hanns-Georg Klein. "Pharmacogenetics in Laboratory Diagnostics." Current Pharmacogenomics and Personalized Medicine 6, no. 1 (March 1, 2008): 12–22. http://dx.doi.org/10.2174/187569208784017502.

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24

Weber, Thomas. "Laboratory diagnostics in dementia." LaboratoriumsMedizin 42, no. 4 (August 28, 2018): 121–30. http://dx.doi.org/10.1515/labmed-2018-0047.

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Abstract Although recent evidence seems to suggest a steady or even declining prevalence and incidence of dementias, these disorders pose a tremendous threat to health care and caregivers. The most common, dominant cause of dementia is Alzheimer’s disease (AD) followed by Levy body dementia (LBD) and vascular dementia (VD). Over the last 25 years, great progress has been made in understanding the pathogenesis of AD but not yet in its treatment. Advancements have been made by ever improving clinical and paraclinical definitions allowing for a continuously increasing differentiation of the various causes of dementias. Besides imaging, functional imaging using positron emission tomography (PET) is now being increasingly used to define the amyloid load in vivo in the brain. By the use of tau-specific tracers meaningful tau imaging may be achieved in the future. The discovery of the cleaving mechanisms of the amyloid precursor protein (APP) and the identification of its major products such as Aβ1−42 and Aβ1−40 as well the metabolism of tau and its phosphorylation have provided reasonably reliable markers to evaluate their usefulness for the diagnosis of AD, LBD, frontotemporal dementia (FTD), Parkinson’s disease (PD), alcohol-related dementia (ARD), traumatic brain injury (TBI), mixed dementia (MD) and others first by cerebrospinal fluid (CSF) analysis and now, due to the introduction of a digital single molecule array (Simoa), by plasma testing. This promising new technique should open avenues for the laboratory validation of other markers such as neurofilament light chains (NfL), visinin-like protein-1 (VLP-1), heart fatty acid binding protein (HFABP) and YKL-40, facilitating further differentiation of the various forms of dementia thus leading to improved treatment.
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25

Hart, Christina, and Birgit Linnemann. "Laboratory Diagnostics in Thrombophilia." Hämostaseologie 39, no. 01 (January 31, 2019): 049–61. http://dx.doi.org/10.1055/s-0039-1677840.

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AbstractA thrombophilic disorder is a hereditary or acquired condition that increases the risk of thrombosis. The most common hereditary thrombophilias that predispose to venous thrombosis in the Caucasian population are the heterozygous forms of the factor V Leiden and prothrombin G20210A mutation that are generally detected by direct DNA genotyping. Immunologic antigen assays and chromogenic or clot-based activity assays are used to identify deficiencies in the natural coagulation inhibitors antithrombin, protein C and protein S. Because pre-analytical errors and acquired causes of low antithrombin, protein C or protein S levels are considerably more common than hereditary deficiencies, all potential conditions that may lower activity levels of the natural coagulation inhibitors (e.g. concomitant liver disease, pregnancy, anticoagulant therapy) must be considered and excluded before the diagnosis of an inhibitor deficiency can be made. To avoid misclassification, the diagnosis should not be made based on a single abnormal test result. Thus, repetitive testing when the patient is not on anticoagulant therapy is mandatory to confirm the diagnosis. Screening for antiphospholipid syndrome (APS) comprises testing for lupus anticoagulants (LAs) and the presence of IgG or IgM antibodies directed against phospholipids and phospholipid-binding proteins such as β-2-glycoprotein-I. A combination of clot-based assays has been recommended to demonstrate LA activity, whereas solid-phase immunoassays allow the detection of anti-cardiolipin and anti-β-2-glycoprotein-I antibodies. The diagnosis of APS requires the persistence of antiphospholipid antibodies for at least 12 weeks together with thrombotic and/or obstetric features of APS.
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26

Azar, Marwan M., and Chadi A. Hage. "Laboratory Diagnostics for Histoplasmosis." Journal of Clinical Microbiology 55, no. 6 (March 8, 2017): 1612–20. http://dx.doi.org/10.1128/jcm.02430-16.

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ABSTRACTThe diagnosis of histoplasmosis is based on a multifaceted approach that includes clinical, radiographic, and laboratory evidence of disease. The gold standards for laboratory diagnosis include demonstration of yeast on pathological examination of tissue and isolation of the mold in the culture of clinical specimens; however, antigen detection has provided a rapid, noninvasive, and highly sensitive method for diagnosis and is a useful marker of treatment response. Molecular methods with improved sensitivity on clinical specimens are being developed but are not yet ready for widespread clinical use. This review synthesizes currently available laboratory diagnostics for histoplasmosis, with an emphasis on complexities of testing and performance in various clinical contexts.
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27

Siahaan, L. "Laboratory diagnostics of malaria." IOP Conference Series: Earth and Environmental Science 125 (March 2018): 012090. http://dx.doi.org/10.1088/1755-1315/125/1/012090.

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28

Zolotarev, P. N., O. V. Lyang, and A. G. Kochetov. "Healthcare in laboratory diagnostics." Laboratornaya sluzhba 4, no. 3 (2015): 30. http://dx.doi.org/10.17116/labs20154330-34.

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29

Lindström, Miia, and Hannu Korkeala. "Laboratory Diagnostics of Botulism." Clinical Microbiology Reviews 19, no. 2 (April 2006): 298–314. http://dx.doi.org/10.1128/cmr.19.2.298-314.2006.

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SUMMARY Botulism is a potentially lethal paralytic disease caused by botulinum neurotoxin. Human pathogenic neurotoxins of types A, B, E, and F are produced by a diverse group of anaerobic spore-forming bacteria, including Clostridium botulinum groups I and II, Clostridium butyricum, and Clostridium baratii. The routine laboratory diagnostics of botulism is based on the detection of botulinum neurotoxin in the patient. Detection of toxin-producing clostridia in the patient and/or the vehicle confirms the diagnosis. The neurotoxin detection is based on the mouse lethality assay. Sensitive and rapid in vitro assays have been developed, but they have not yet been appropriately validated on clinical and food matrices. Culture methods for C. botulinum are poorly developed, and efficient isolation and identification tools are lacking. Molecular techniques targeted to the neurotoxin genes are ideal for the detection and identification of C. botulinum, but they do not detect biologically active neurotoxin and should not be used alone. Apart from rapid diagnosis, the laboratory diagnostics of botulism should aim at increasing our understanding of the epidemiology and prevention of the disease. Therefore, the toxin-producing organisms should be routinely isolated from the patient and the vehicle. The physiological group and genetic traits of the isolates should be determined.
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30

Troshina, E. A., D. G. Bel'tsevich, and M. Iu Iukina. "Laboratory diagnostics of pheochromocytoma." Problems of Endocrinology 56, no. 4 (August 15, 2010): 39–43. http://dx.doi.org/10.14341/probl201056439-43.

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Biochemical diagnosis of pheochromocytoma is based on the measurement of altered levels of normetanephrine and metanephrine in plasma or 24-hour urine samples. Plasma normetanephrine and metanephrine levels four times the upper limit of the normal value or metanephrine and normetanephrine excretion in urine above 700 and 1500 mcg/24 hours respectively makes further testing unnecessary and subsequent examination must be focused on the determination of tumour localization. In patients presenting with the above parameters elevated within the "grey zone", effect of medicinal products needs to be excluded and the diagnosis confirmed in the clonidine test or by the measurement of chromogranin A level.
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31

Cuny, Christa, G. Werner, Christine Braulke, I. Klare, and W. Witte. "Diagnostics of Staphylococci with Special Reference to MRSA/Diagnostik von Staphylokokken unter besonderer Berücksichtigung von MRSA." LaboratoriumsMedizin 26, no. 3/4 (January 1, 2002): 165–73. http://dx.doi.org/10.1515/labmed.2002.022.

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32

Gatermann, Sören, and Martin Kaase. "Erkennung von bakteriellen Resistenzmechanismen in der täglichen Diagnostik / Recognition of bacterial resistance mechanisms in routine diagnostics." LaboratoriumsMedizin 32, no. 4 (January 1, 2008): 235–52. http://dx.doi.org/10.1515/jlm.2008.042.

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33

Toader, Mioriţa, Olivia Ştefan, Andreea Şerbănică, Daniela Neacşu, Mircea Drăghici, and Corneliu Toader. "HIV infection in newborns – laboratory diagnosis." Romanian Journal of Infectious Diseases 19, no. 1 (March 31, 2016): 22–25. http://dx.doi.org/10.37897/rjid.2016.1.4.

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AIDS was reported in the pediatric population for the first time in 1982 in the USA by pediatricians who hardly managed to convince those responsible for childcare, because it can also occur in this population segment. Thanks to efforts for disease recognition in the pediatric population, scientists have been able to provide support and children infected with human immunodeficiency virus (HIV) receive the same treatment protocol as adults. Early detection of HIV infection in pregnant women leads to the possibility for starting a treatment to prevent HIV transmission from mother to child. The ELISA test has a high sensitivity and is indicated for HIV screening and Western Blot test has highly specificity and confirming the HIV infection.
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34

Aleksandrova, E. H., and A. A. Novikov. "Sovremennye standarty laboratornoy diagnostiki revmaticheskikh zabolevaniy." Rheumatology Science and Practice, no. 2s (April 15, 2006): 47. http://dx.doi.org/10.14412/1995-4484-2006-1430.

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35

Galzigna, Lauro. "Medicina di laboratorio. Fondamenti di Diagnostica." Clinica Chimica Acta 210, no. 3 (September 1992): 241–42. http://dx.doi.org/10.1016/0009-8981(92)90212-9.

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36

Klouche, Mariam. "Editorial: Current Status and Future Development of HLA Diagnostics/Gegenwärtiger Stand und zukünftige Entwicklung der HLA-Diagnostik." LaboratoriumsMedizin 27, no. 9/10 (January 1, 2003): 349–50. http://dx.doi.org/10.1515/labmed.2003.048.

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37

Haferlach, T., A. Kohlmann, S. Schnittger, M. Dugas, W. Kern, and C. Schoch. "Genexpressionsanalysen bei akuten Leukämien: Diagnostik der Zukunft? / Gene expression profiling in acute leukemia: diagnostics of the future?" LaboratoriumsMedizin 28, no. 3 (January 17, 2004): 225–32. http://dx.doi.org/10.1515/labmed.2004.032.

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38

Großmann, R., U. Geisen, S. Schwender, and F. Keller. "Diagnostik und Therapiesteuerung bei erworbenen Faktoreninhibitoren. Diagnostics and Therapy Monitoring in Patients with Acquired Coagulation Factor Inhibitors." LaboratoriumsMedizin 23, no. 2 (January 1999): 70–79. http://dx.doi.org/10.1515/labm.1999.23.2.70.

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39

Walochnik, Julia, and Horst Aspöck. "Die Diagnostik von Infektionen mit freilebenden Amöben (FLA) Diagnostics of Infections with free-living amoebae (FLA)." LaboratoriumsMedizin 29, no. 6 (January 1, 2005): 446–56. http://dx.doi.org/10.1515/jlm.2005.061.

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40

Zajkowska, Joanna M., and Justyna Dunaj. "Lyme borreliosis. Challenges and difficulties of laboratory diagnosis." Forum Zakażeń 4, no. 4 (October 21, 2013): 241–49. http://dx.doi.org/10.15374/fz2013042.

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41

Reinheimer, Claudia, and Hans Wilhelm Doerr. "Spezifische Immuntherapie und Vakzination als Störfaktoren in der serologischen Diagnostik / Immunotherapy and vaccination: interfering factors in serological diagnostics." LaboratoriumsMedizin 34, no. 5 (January 1, 2010): 257–60. http://dx.doi.org/10.1515/jlm.2010.045.

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42

Kotschote, Stefan, Carola Wagner, Christoph Marschall, Karin Mayer, Kaimo Hirv, Martin Kerick, Bernd Timmermann, and Hanns-Georg Klein. "Translation of next-generation sequencing (NGS) into molecular diagnostics / Umsetzung von Next Generation Sequencing in der molekularen Diagnostik." LaboratoriumsMedizin 34, no. 6 (January 1, 2010): 311–18. http://dx.doi.org/10.1515/jlm.2010.054.

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43

Dziatkiewicz-Warkocz, Paulina, Lidia Gil, and Maria Kozłowska-Skrzypczak. "Laboratory diagnostics of iron overload." Diagnostyka Laboratoryjna 56, no. 1 (August 21, 2020): 35–42. http://dx.doi.org/10.5604/01.3001.0014.3611.

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Iron is a fundamental trace element, essential to maintain homeostasis in living organisms. Iron overload can result from increased iron absorption in the gastrointestinal system, repeated blood transfusions or liver diseases. The iron turnover is regulated by a group of specialized proteins, responsible for its absorption, transport, oxidation and preventing tissue damage that can be caused by the ferrous ions Fe2+. Diagnosis of iron overload states, as well the course of treatment oversight, is based on continuous monitoring of the iron concentration in the body. The use of numerous laboratory tests is the first stage of diagnostics. This work describes primary mechanisms responsible for the regulation of iron metabolism and the consequences of iron overload. Furthermore, it presents an analysis of the currently applied laboratory parameters and other methods used to evaluate iron overload conditions.
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44

Makarova, M. A., Z. N. Matveeva, and L. A. Kaftyreva. "MODERN LABORATORY DIAGNOSTICS OF ESСHERICHIOSIS." Russian Journal of Infection and Immunity 8, no. 4 (January 16, 2019): 514–15. http://dx.doi.org/10.15789/2220-7619-2018-4-2.10.

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45

Flesch, Brigitte. "Laboratory Diagnostics of HLA Antibodies." Laboratoriums Medizin 27, no. 9-10 (October 2003): 351–58. http://dx.doi.org/10.1046/j.1439-0477.2003.03067.x.

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46

Wobeser, Bruce K., and Colleen Duncan. "Equine Pathology and Laboratory Diagnostics." Veterinary Clinics of North America: Equine Practice 31, no. 2 (August 2015): i. http://dx.doi.org/10.1016/s0749-0739(15)00039-5.

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47

Lindsay, LR, K. Bernat, and A. Dibernardo. "Laboratory diagnostics for Lyme disease." Canada Communicable Disease Report 40, no. 11 (May 29, 2014): 209–17. http://dx.doi.org/10.14745/ccdr.v40i11a02.

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48

Bison, Elisa, Gentian Denas, Seena Jose, Giacomo Zoppellaro, Alessandra Banzato, and Vittorio Pengo. "Laboratory Diagnostics of Antiphospholipid Syndrome." Seminars in Thrombosis and Hemostasis 44, no. 05 (May 3, 2017): 439–44. http://dx.doi.org/10.1055/s-0037-1601331.

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AbstractDiagnosis of antiphospholipid syndrome (APS) lies in the recognition of antiphospholipid antibodies (aPL). As standardization of tests for the detection of aPL is far from being optimal and reference material is not available, inappropriate diagnoses of APS are not unusual. In the last few years, the concept of triple test positivity has emerged as a useful tool to identify patients with APS. Clinical studies on patients and carriers of triple positivity clearly show that these individuals are at high risk of thromboembolic events and pregnancy loss. Moreover, triple positivity arises from a single (probably pathogenic) antibody directed to domain 1 of β2-glycoprotein I, a protein whose function is still unknown. Studies on homogenous group of patients with single or double positivity are scant, and uncertainties arise on their association with clinical events. Promising but undetermined results come also from the determination of antibodies directed to phosphatidylserine/prothrombin complex. Interpretation of laboratory profile in APS is challenging, and the collaboration between clinical pathologists and clinicians is highly desirable.
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49

Karpova, T. I., and I. S. Tartakovskiy. "Laboratory diagnostics of legionellae infection." Laboratornaya sluzhba 4, no. 1 (2015): 30. http://dx.doi.org/10.17116/labs20154130-34.

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50

Philippidis, Alex. "Clinical Laboratory, Meet Molecular Diagnostics." Clinical OMICs 1, no. 7 (July 16, 2014): 18–21. http://dx.doi.org/10.1089/clinomi.01.07.07.

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