Relevant bibliographies by topics / Lactogene placentaire

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Academic literature on the topic 'Lactogene placentaire'

Author: Grafiati
Published: 4 June 2021
Last updated: 10 February 2022

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Journal articles on the topic "Lactogene placentaire"

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Ingrand, Jacques. "Hormone lactogène placentaire (HPL)." EMC - Biologie Médicale 1, no. 1 (January 2006): 1–2. http://dx.doi.org/10.1016/s2211-9698(06)76080-0.

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Renegar, R. H., J. N. Southard, and F. Talamantes. "Immunohistochemical co-localization of placental lactogen II and relaxin in the golden hamster (Mesocricetus auratus)." Journal of Histochemistry & Cytochemistry 38, no. 7 (July 1990): 935–40. http://dx.doi.org/10.1177/38.7.2355175.

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Two hormones with lactogenic activity are produced by the hamster placenta during the second half of pregnancy. One of these hormones, hamster placental lactogen II (haPL-II), has been well characterized; however, its cellular source is not known. In the present study, haPL-II was localized in placental tissues using a specific antibody and the avidin-biotin-peroxidase immunohistochemical technique. Because relaxin has been localized in the hamster placenta, it was of interest to determine if haPL-II and relaxin are localized in the same cells. haPL-II immunoactivity was observed in primary and secondary giant trophoblast cells of the placenta on Days 12, 14, and 15 of pregnancy. On Day 15 positive staining was also observed in large cells located within mesometrial arteries and in eosinophilic bodies associated with degenerating sheathed arteries of the decidua basalis. haPL-II-positive staining was not observed in placentae from Days 8 or 10 of pregnancy. On Day 14, haPL-II was colocalized with relaxin in 75% of the giant trophoblast cells observed. Therefore, it is probable that these hormones are synthesized and secreted by the same cell.
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HENNEN, G., F. FRANKENNE, Marie-Louise SCIPPO, A. IGOUT, J. CLOSSET, G. PIRENS, and Françoise GOMEZ. "Hormone de croissance placentaire. Signification par rapport aux hormones de croissance et lactogène." Reproduction Nutrition Développement 28, no. 6B (1988): 1699–706. http://dx.doi.org/10.1051/rnd:19881014.

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Morrish, D. W., H. Marusyk, and D. Bhardwaj. "Ultrastructural localization of human placental lactogen in distinctive granules in human term placenta: comparison with granules containing human chorionic gonadotropin." Journal of Histochemistry & Cytochemistry 36, no. 2 (February 1988): 193–97. http://dx.doi.org/10.1177/36.2.2447154.

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Human placental lactogen (hPL) is known to originate in the syncytiotrophoblast, as demonstrated by light microscopic peroxidase and immunofluorescent staining. However, ultrastructural localization of hPL has not previously been performed. In these experiments, immunostaining of electron microscopic sections using protein A-gold and avidin-biotin complex techniques was used to study hPL and human chorionic gonadotropin (beta hCG) localization in first trimester and term placentae. HPL was localized in many small (0.12-0.25 micron) granules. In contrast, beta hCG was found in large (0.40-1.2 micron) granule complexes. The results therefore demonstrate that these two hormones are stored in two morphologically distinct types of cytoplasmic granules. Since hPL and hCG have different secretory mechanisms, this methodology will be useful in studying these differing mechanisms in human placenta.
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Lea, Richard G., Peter Wooding, Ian Stewart, Lisa T. Hannah, Stephen Morton, Karen Wallace, Raymond P. Aitken, et al. "The expression of ovine placental lactogen, StAR and progesterone-associated steroidogenic enzymes in placentae of overnourished growing adolescent ewes." Reproduction 133, no. 4 (April 2007): 785–96. http://dx.doi.org/10.1530/rep-06-0294.

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Overnourishing pregnant adolescent sheep promotes maternal growth but reduces placental mass, lamb birth weight and circulating progesterone. This study aimed to determine whether altered progesterone reflected transcript abundance forStAR(cholesterol transporter) and the steroidogenic enzymes (Cyp11A1,Hsd3bandCyp17). Circulating and placental expression of ovine placental lactogen (oPL) was also investigated. Adolescent ewes with singleton pregnancies were fed high (H) or moderate (M) nutrient intake diets to restrict or support placental growth. Experiment 1: peripheral progesterone and oPL concentrations were measured in H (n=7) and M (n=6) animals across gestation (days 7–140). Experiment 2: progesterone was measured to mid- (day 81; M:n=11, H:n=13) or late gestation (day 130; M:n=21, H:n=22), placental oPL,StARand steroidogenic enzymes were measured by qPCR and oPL protein by immunohistochemistry. Experiment 1: in H vs M animals, term placental (P<0.05), total cotyledon (P<0.01) and foetal size (P<0.05) were reduced. Circulating oPL and progesterone were reduced at mid- (P<0.001,P<0.01) and late gestation (P<0.01,P<0.05) and oPL detection was delayed (P<0.01). Experiment 2: placental oPL was not altered by nutrition. In day 81 H animals, progesterone levels were reduced (P<0.001) but not related to placental or foetal size. Moreover, placental steroidogenic enzymes were unaffected. Day 130 progesterone (P<0.001) andCyp11A1(P<0.05) were reduced in H animals with intrauterine growth restriction (H+IUGR). Reduced mid-gestation peripheral oPL and progesterone may reflect altered placental differentiation and/or increased hepatic clearance respectively. Restricted placental growth and reduced biosynthesis may account for reduced progesterone in day 130 H+IUGR ewes.
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Lea, Richard G., Peter Wooding, Ian Stewart, Lisa T. Hannah, Stephen Morton, Karen Wallace, Raymond P. Aitken, et al. "The expression of ovine placental lactogen, StAR and progesterone-associated steroidogenic enzymes in placentae of overnourished growing adolescent ewes." REPRODUCTION 135, no. 6 (June 2008): 889. http://dx.doi.org/10.1530/rep-06-0294e.

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Kawai, Motoyuki, Takashi Murakami, Shizuka Otani, Kenji Shima, Masaaki Yamaguchi, and Kurajiro Kishi. "Colocalization of Leptin Receptor (OB-R) mRNA and Placental Lactogen-II in Rat Trophoblast Cells: Gestational Profile of OB-R mRNA Expression in Placentae." Biochemical and Biophysical Research Communications 257, no. 2 (April 1999): 425–30. http://dx.doi.org/10.1006/bbrc.1999.0467.

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Popovych, A. I. "Immunohistochemical Study of Platcentary Lactogen and Placentary Alkaline Phosphatase in the Trophoblast of Chorionic Villi in Placental Calcnosis in Pregnant Women with Iron-deficiency Anemia." Ukraïnsʹkij žurnal medicini, bìologìï ta sportu 3, no. 3 (April 20, 2018): 39–43. http://dx.doi.org/10.26693/jmbs03.03.039.

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Kim, H. R., K. Naruse, H. R. Lee, T. Wakayama, C. S. Park, and D. I. Jin. "51 PROTEOMICS ANALYSIS OF PLACENTOMEGALY IN CLONED MICE." Reproduction, Fertility and Development 19, no. 1 (2007): 143. http://dx.doi.org/10.1071/rdv19n1ab51.

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A variety of mammalian species have been cloned during the past few years. However, the success rate of somatic cell nuclear transfer in animals has been extremely low with many problems. Particularly, placentomegaly is a frequent finding in cloned mice and cattle (Wakayma et al. 1999 PNAS USA 96, 14 984–14 989; Niemann et al. 2000 Theriogenology 53, 21–34). To assess protein expression profile in the placentomagaly of cloned mice produced by nuclear transfer of embryonic stem cells, we have used global proteomics approach by 2-D gel electrophoresis and mass-spectrometry with the differential protein patterns using the 3 placentae of cloned mice and 4 normal mouse placentae. Proteins within isoelectric point range pH 4.0~9.0 and molecular weight range of 20–100 kDa separately were analyzed by 2-D gel electrophoresis with 3 replications of each sample. A total of approximately 3500 spots were detected 2-D gel. In the comparison of normal and cloned placenta samples, a total of 49 protein spots were expressed differentially, of which 28 spots were up-regulated proteins including alpha-fetoprotein, aspartyl aminopeptidase, placental lactogen 2, tissue inhibitor of metalloproteinase 2 (TIMP-2), etc., and 21 spots were down-regulated proteins including peroxiredoxin 6, creatine kinase, pre-B-cell colony-enhancing factor 1 (PBEF), etc. Eight spots could not be identified. One of differentially up-regulated proteins in cloned mouse placenta was identified as TIMP-2 protein that is related to extracellular matrix degradation and tissue remodeling processes. Western blot was performed with placental sample used in the 2-D gel electrophoresis analysis and normal mouse placenta samples on Days 11.5 to 18.5. Indeed, Western blot analysis confirmed a significant increase of TIMP-2 protein level in cloned mouse placenta compared with normal. The expression levels of TIMP-2 in normal mouse placenta were highest at the normal mid gestation (Day 13.5) and exhibited prominent decrease at late gestation period during normal pregnancy. However, the expression levels of TIMP-2 in placenta of cloned mice appeared to be similar to levels of mid gestation normal mouse placenta. And one of down-regulated protein in NT placenta was identified as PBEF protein that is known to be related to induction of spontaneous labor. PBEF protein level in cloned mice placenta was revealed to decrease remarkably compared with normal. The expression levels of PBEF in normal mice placenta exhibited a gradual decrease between Day 11.5 and Day 18.5 without complete depletion. However, the expression levels of PBEF in placenta of cloned mice appeared to be markedly lower than those of the same day normal placenta. In conclusion, abnormal expression of placental proteins associated with tissue remodeling and labor induction may be the cases of placentomegaly in cloned mice.
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PERIER, A., F. BOCQUIER, G. KANN, and J. MARTINET. "Influence de la photopériode et des apports énergétiques pendant la gestation sur les taux plasmatiques de la prolactine, de l'hormone de croissance et de l'hormone placentaire lactogène puis sur la production laitière de la brebis traite." Reproduction Nutrition Développement 26, no. 1B (1986): 391–92. http://dx.doi.org/10.1051/rnd:19860272.

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Dissertations / Theses on the topic "Lactogene placentaire"

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POLLIOTTI, BRUNO. "Contribution a l'etude de la regulation de la secretion placentaire de l'hormone chorionique gonadotrope et placentaire lactogene in vitro." Paris 6, 1991. http://www.theses.fr/1991PA066284.

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La regulation de la secretion d'hcg et d'hpl par le placenta est peu connue lorsqu'on la compare a celle de la secretion des hormones hypophysaires. Le but de cette etude etait de mettre en evidence les mecanismes ioniques susceptibles d'intervenir dans la secretion placentaire de ces deux hormones. Un systeme d'incubation permettant une secretion hormonale stable dans un milieu minimum a ete mis au point. Dans ces conditions, l'addition de calcium extracellulaire stimule de maniere similaire et dose-dependante les secretions d'hcg et d'hpl. Cet effet peut etre bloque par le cobalt ou mine par l'ionomycine et le barium. Le retrait progressif de sodium extracellulaire provoque egalement une augmentation dose-dependante des secretions d'hcg et d'hpl. L'effet secretoire du retrait de sodium est aboli en presence de cobalt ou en l'absence de calcium extra-cellulaire. Il est egalement inhibe par l'amiloride, le magnesium et le strontium. Cet effet est potentialise en presence d'ouabaine mais non affecte en presence de nifedipine, de d600 et de tetrodotoxine. L'addition de potassium dans le milieu induit une augmentation de la secretion d'hcg et d'hpl. Cette riposte secretoire est passagere et dose-dependante. Elle est abolie en presence de cobalt, de nifedipine et de d600 ainsi qu'en l'absence de calcium extracellulaire. Enfin, des granules de secretion contenant de l'hpl immunoreactive mais pas d'hcg ont pu etre mis en evidence dans le syncytiotrophoblaste. En conclusion, ces observations demontrent pour la premiere fois que les secretions d'hcg et d'hpl dependent de l'entree de calcium et que cette entree peut etre mediee par un processus de sodium-calcium contre-transport et/ou par des canaux calcium voltage-dependant. Cette augmentation cytosolique du calcium a pour resultat ultime, une riposte secretoire par un mecanisme d'exocytose. La fonction secretoire du placenta humain semble donc bien adherer au principe du stimulus secretion coupling
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Chiffoleau, Pascal. "Doppler obstetrical et hormone placentaire lactogene : leur rapport dans le suivi des grossesses a risque ; a propos de 146 dossiers." Nantes, 1989. http://www.theses.fr/1989NANT057M.

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Sade, Slim. "Recherche d'une methodologie pour l'etude du role physiologique de l'hormone placentaire lactogene ovine (opl) dans la croissance embryonnaire." Paris 6, 1987. http://www.theses.fr/1987PA066611.

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Maazouzi, Hafid. "Hormone lactogène placentaire (hPL) : fabrication d'anticorps anti-hPL et recherche des épitopes : l'hPL est-elle une kinase ?" Nancy 1, 1993. http://docnum.univ-lorraine.fr/public/SCD_T_1993_0426_MAAZOUZI.pdf.

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Charpigny, Gilles. "Purification par HPLC de deux messages embryonnaires de nature protéique l'hormone lactogène placentaire et la trophoblastine." Grenoble 2 : ANRT, 1986. http://catalogue.bnf.fr/ark:/12148/cb37596620s.

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Charpigny, Gilles. "Purification par HPLC de deux messages embryonnaires de nature protéique : l'hormone lactogène placentaire et la trophoblastine." Paris 6, 1986. http://www.theses.fr/1986PA066361.

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Sade, Slim. "Recherche d'une méthodologie pour l'étude du rôle physiologique de l'hormone placentaire lactogène ovine (oPL) dans la croissance embryonnaire." Grenoble 2 : ANRT, 1987. http://catalogue.bnf.fr/ark:/12148/cb376095405.

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Lagrandcourt-Hess, Ketsia. "Mise au point de systèmes de transcription et de traduction acellulaires à partir de placenta humain application à l'hormone lactogène placentaire." Grenoble 2 : ANRT, 1986. http://catalogue.bnf.fr/ark:/12148/cb37598871q.

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Iliou, Jean-Pierre. "Contribution a l'étude des mécanismes de régulation de la lipolyse du tissu adipeux au cours de la gestation et de la lactation : étude bibliographique, étude expérimentale "in vitroe sur adipocytes isolés de brebis." Paris 7, 1985. http://www.theses.fr/1985PA077129.

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Chez la Brebis, au cours de la gestation et de la lactation, l'évolution du métabolisme lipidique du tissu adipeux est caractérisée par 2 phases métaboliques successives et opposées. La prmière est une phase d'accumulation des réserves lipidiques (lipogénèse élevée, lipolyse aibl), elle correspond aux 2 premiers tiers de la gestation. La seconde est une phase de mobilisation des réserves lipidiques (lipogénèse faible, lipolyse élvée), elle englobe les périodes du dernier tiers de la gestation et du début de la lactation. Ce profil d'évolution du métabolism lipidique doit permettre à l'organisme maternel de subvenir d'une part aux besoins énergétiques importants du foetus en fin de gestation et d'autre part, de préparer et de aire face au coût énergétique très élevé de la lactation. En utilisant un système d'incubation d'adipocytes isolés maintenus en survie, ces derniers étant prélevés sur des Brebis en cours de gestation et de lactation, l'étude expérimental a porté sur l'évolution de la réponse cellulaire à l'action de différents effecteurs de la lipolyse. La sensibilité des adipocytes aux stimuli lipolytiques de l'isoprénaline (caté¬cholamine de synthèse) et de la théophylline (méthylxanthine) croît au cours de la gestation pour tendre vers un maximum à quelques jours "pré-partum". Pendant la 3ème semaine de lactation, les cellules demeurent à un niveau élevé de sensibilité. Le niveau de l'effet antilipolytique de l'adénosine est proportionnel à la sensibilité des cellules isolées au stimulus lipolytique de l'isoprénaline. Cet effet rétro-régulateur a été mis en évidence de manière directe en présence d'adé-nosine et d'isoprénaline et de manière indirecte en présence d'adénosine désaminase. Les actions sur la lipolyse de 2 hormones, l'hormone placentaire lactogène ovine (oPL) et l'hormone de croissance bovine (bGh) ont été testées. L'oPL ne présente aucun effet, la bGh présente en début de gestation un effet de potentialisation sur l'action lipolytique du couple isoprénaline-théophylline. La réorientation complète du métabolisme lipidique à partir de la fin du second tiers de la gestation se reflète dans l'évolution de la sensibilité cellulaire aux stimuli lipolytiques de l'isoprénaline et de la théophylline. Les rôles suggérés dans la littérature pour la bGh et l'oPL au sujet de leur action lipolytique demeurent très controversés.
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Sollero, Celina de Paula Azevedo. "Condições maternas interferentes nas concentrações plasmaticas da gonadotrofina corionica e lactogenio placentario no primeiro trimestre de gravidez." [s.n.], 1996. http://repositorio.unicamp.br/jspui/handle/REPOSIP/309794.

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Orientador: Jesse de Paula Neves Jorge
Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas
Made available in DSpace on 2018-07-21T21:35:04Z (GMT). No. of bitstreams: 1 Sollero_CelinadePaulaAzevedo_D.pdf: 3949620 bytes, checksum: 6f7cdfc8912d5039d8dbe3c676068889 (MD5) Previous issue date: 1996
Resumo: Partindo de estudos epidemiológicos que demonstraram haver associações entre baixo peso ao nascer, hipertrofia placentária e hipertensão arterial na vida adulta, nós estudamos um grupo de mulheres no primeiro trimestre da gestação, para verificar se existem indícios de interferências entre as condições maternas e a função placentária. Analisamos as relações entre o hormônio gonadotrófico coriônico e o hormônio lactogênio placentário e idade materna, paridade, índice de massa corpórea, pressão arterial, hemoglobina materna, hematócrito, volume corpuscular médio, contagem de glóbulos vermelhos, ferro, ferritina, capacidade total de ligação do ferro, 2,3-difosfoglicerato, carbóxihemoglobina, pressão parcial do oxigênio para a hemoglobina saturada em 50%, pressão parcial do hidrogênio, pressão parcial do oxigênio, pressão parcial do dióxido de carbono e saturação do oxigênio. Encontramos diferenças significativas entre fumantes e não fumantes com relação ao hormônio gonadotrófico coriônico, hematócrito, volume corpuscular médio, ferro e carbóxihemoglobina. Encontramos ainda correlações entre o hormônio gonadotrófico coriônico, e o índice de massa corpórea, a hemoglobina, o hematócrito e a carbóxihemoglobina, para o grupo total de gestantes. Entre as não fumantes o hormônio gonadotrófico coriônico se correlacionou negativamente com a pressão sistólica. O hormônio lactogênio placentário se correlacionou com a pressão sistólica. Entre as não fumantes o hormônio lactogênio placentário se correlacionou com a pressão parcial do oxigênio e a saturação do oxigênio. Concluímos que as condições maternas tem interferência na concentração plasmática dos hormônios placentários já no primeiro trimestre da gestação.
Abstract: Several studies have shown a relationship between low birth weight, large placental size and high blood pressure in adult life. Large placentas, were reported to be associated with maternal anaemia at the time of delivery. We studied a group of pregnant women during the first trimester of gestation, with the aim of investigating the influences of the maternal environment into the levels of placental hormones in maternal blood at this stage of pregnancy. We studied maternal blood levels of hCG and hPL and its relations to maternal age, parity, gestation age, maternal high and weight, Body Mass Index, blood pressure, Hb, Ht, MCV, RBCC, iron, ferritin, TIBC, 2,3-DPG, carboxi Hb, P50, PH, PC02, P02, 02sat and smoking habits. We found significant differences between smokers and non smokers in relation to hCG, Ht, MCV, Iron and carboxi Hb. We also found a relationship between hCG, and maternal weight, BMI, Hb, Ht, and carboxi Hb. Among the non smokers there was a significant negative correlation between the sistolic blood pressure and the levels of hCG. In hPL, there were significant correlation with maternal weight, BMI, and systolic blood pressure. Among the non smokers hPL correlated positively with P02 and 02sat. We concluded that maternal environment has influence in maternal blood levels of hCG and hPL in the early stages of pregnancy
Doutorado
Tocoginecologia
Doutor em Ciências Médicas
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