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Journal articles on the topic 'Lambert-Eaton Myasthenic Syndrome'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Kesner, Vita G., Shin J. Oh, Mazen M. Dimachkie, and Richard J. Barohn. "Lambert-Eaton Myasthenic Syndrome." Neurologic Clinics 36, no. 2 (May 2018): 379–94. http://dx.doi.org/10.1016/j.ncl.2018.01.008.

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2

Katirji, Basfhar. "Lambert-Eaton Myasthenic Syndrome." Journal of Clinical Neuromuscular Disease 1, no. 3 (March 2000): 134–36. http://dx.doi.org/10.1097/00131402-200003000-00004.

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3

Young, Jeffery D., and Jacqueline A. Leavitt. "Lambert–Eaton Myasthenic Syndrome." Journal of Neuro-Ophthalmology 36, no. 1 (March 2016): 20–22. http://dx.doi.org/10.1097/wno.0000000000000258.

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4

D'Amour, M., G. Gariepy, and J. Braidy. "Lambert-Eaton myasthenic syndrome." Canadian Medical Association Journal 176, no. 1 (December 13, 2006): 37. http://dx.doi.org/10.1503/cmaj.060767.

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5

Mareska, Michael, and Laurie Gutmann. "Lambert-Eaton Myasthenic Syndrome." Seminars in Neurology 24, no. 02 (July 15, 2004): 149–53. http://dx.doi.org/10.1055/s-2004-830900.

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6

Newsom-Davis, J. "Lambert-Eaton Myasthenic Syndrome." Revue Neurologique 160, no. 2 (February 2004): 177–80. http://dx.doi.org/10.1016/s0035-3787(04)70888-7.

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7

Titulaer, Maarten J., and Jan J. G. M. Verschuuren. "Lambert-Eaton Myasthenic Syndrome." Annals of the New York Academy of Sciences 1132, no. 1 (June 2008): 129–34. http://dx.doi.org/10.1196/annals.1405.030.

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8

Petty, R. "Lambert-Eaton myasthenic syndrome." Case Reports 2009, jan27 1 (February 2, 2009): bcr0920080914. http://dx.doi.org/10.1136/bcr.09.2008.0914.

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9

SANDERS, DONALD B. "Lambert-Eaton Myasthenic Syndrome." Annals of the New York Academy of Sciences 998, no. 1 (September 2003): 500–508. http://dx.doi.org/10.1196/annals.1254.065.

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10

Chiang, Yi Zhen, Kian Tjon Tan, and Ian K. Hart. "Lambert–Eaton myasthenic syndrome." British Journal of Hospital Medicine 70, no. 3 (March 2009): 168–69. http://dx.doi.org/10.12968/hmed.2009.70.3.40574.

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11

Petty, R. "Lambert Eaton myasthenic syndrome." Practical Neurology 7, no. 4 (August 1, 2007): 265–67. http://dx.doi.org/10.1136/jnnp.2007.120071.

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12

Weimer, Maria B., and Joaquin Wong. "Lambert-eaton myasthenic syndrome." Current Treatment Options in Neurology 11, no. 2 (February 8, 2009): 77–84. http://dx.doi.org/10.1007/s11940-009-0010-z.

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13

Newsom-Davis, John. "Lambert-eaton myasthenic syndrome." Current Treatment Options in Neurology 3, no. 2 (March 2001): 127–31. http://dx.doi.org/10.1007/s11940-001-0047-0.

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14

Irita, Kazuo, Michiko Satoh, Takashi Akata, Takashi Yano, Shosuke Takahashi, and Junichi Yoshitake. "Lambert-Eaton myasthenic syndrome." Canadian Journal of Anaesthesia 37, no. 8 (November 1990): 944. http://dx.doi.org/10.1007/bf03006644.

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15

Zivaljevic, Milica, Svetlana Popovic, and Tamara Vujkov. "Lambert-Eaton myasthenic syndrome: A rare manifestation of paraneoplastic syndrome in ovarian cancer: Case report." Medical review 58, no. 9-10 (2005): 495–97. http://dx.doi.org/10.2298/mpns0510495z.

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Introduction Paraneoplastic neurological syndromes are rare non-metastatic complications of cancer that have an immune-mediated etiology. The Lambert-Eaton myasthenic syndrome (LEMS) is a neuromuscular disorder, often associated with small cell lung carcinoma (SCLC), which is characterized by reduced quantal release of acetylcholine from the motor nerve terminals. Lambert-Eaton Myasthenic Syndrome The Lambert-Eaton Myasthenic Syndrome (LEMS) is characterized by proximal muscle weakness initially affecting gait, autonomic symptoms (dry mouth, constipation, erectile failure) and augmentation of strength during initial voluntary activation. Symptomatic treatment of the junctional disorder is based on cholinergic drugs, immunosuppression, immunomodulation and physical therapy useful in case of unsuccessful antineoplastic therapy. Case report A rare case of ovarian cancer with Eaton-Lambert syndrome is reported. A 50-year-old woman was admitted to the gynecologic department, complaining of weakness and pain in her arms and shoulders. Physical therapy resulted in partial improvement. Treatment of paraneoplastic syndrome markedly improves the quality of life of cancer patients. Patients presenting with this syndrome should undergo a careful evaluation for the presence of an occult malignancy.

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16

Sanders, Donald B., and Jeffrey T. Guptill. "Myasthenia Gravis and Lambert-Eaton Myasthenic Syndrome." CONTINUUM: Lifelong Learning in Neurology 20 (October 2014): 1413–25. http://dx.doi.org/10.1212/01.con.0000455873.30438.9b.

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17

Dineen, Jennifer M., and Steven A. Greenberg. "Myasthenis Gravis or Lambert-Eaton Myasthenic Syndrome?" American Journal of Physical Medicine & Rehabilitation 94, no. 2 (February 2015): e20-e21. http://dx.doi.org/10.1097/phm.0000000000000176.

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18

Nicolle, Michael W. "Myasthenia Gravis and Lambert-Eaton Myasthenic Syndrome." CONTINUUM: Lifelong Learning in Neurology 22, no. 6 (December 2016): 1978–2005. http://dx.doi.org/10.1212/con.0000000000000415.

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19

Palace, Jackie, and David Hilton-Jones. "Myasthenia Gravis and Lambert-Eaton Myasthenic Syndrome." Medicine 28, no. 8 (2000): 118–23. http://dx.doi.org/10.1383/medc.28.8.118.28541.

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20

Palace, Jackie, and David Hilton-Jones. "Myasthenia gravis and Lambert-Eaton myasthenic syndrome." Medicine 32, no. 11 (November 2004): 114–18. http://dx.doi.org/10.1383/medc.32.11.114.53367.

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21

Jones, R. "Lambert-Eaton Myasthenic Syndrome: LEMS." Clinical Neurophysiology 119 (October 2008): S152. http://dx.doi.org/10.1016/s1388-2457(08)60554-4.

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22

Bady, B., G. Chauplannaz, and H. Carrier. "Congenital Lambert-Eaton myasthenic syndrome." Journal of Neurology, Neurosurgery & Psychiatry 50, no. 4 (April 1, 1987): 476–78. http://dx.doi.org/10.1136/jnnp.50.4.476.

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23

O'NEILL, J. H., N. M. F. MURRAY, and J. NEWSOM-DAVIS. "THE LAMBERT-EATON MYASTHENIC SYNDROME." Brain 111, no. 3 (1988): 577–96. http://dx.doi.org/10.1093/brain/111.3.577.

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24

Costa, Mariana R., Carolina Maruta, and Mamede de Carvalho. "Lambert–Eaton Myasthenic Syndrome Plus." Journal of Clinical Neuromuscular Disease 21, no. 2 (December 2019): 119–21. http://dx.doi.org/10.1097/cnd.0000000000000260.

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25

Carpentier, Antoine F., and Jean-Yves Delattre. "The Lambert-Eaton Myasthenic Syndrome." Clinical Reviews in Allergy & Immunology 20, no. 1 (2001): 155–58. http://dx.doi.org/10.1385/criai:20:1:155.

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26

Bady, B., G. Chauplannaz, and H. Carrier. "Congenital Lambert-Eaton myasthenic syndrome." Electroencephalography and Clinical Neurophysiology 61, no. 3 (September 1985): S85. http://dx.doi.org/10.1016/0013-4694(85)90344-x.

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27

Kohler, Siegfried, and Andreas Meisel. "The Lambert-Eaton Myasthenic Syndrome — an Overview." Neurology International Open 02, no. 01 (January 2018): E40—E45. http://dx.doi.org/10.1055/s-0043-118274.

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AbstractThe Lambert Eaton myasthenic syndrome (LEMS) has a prevalence of around 5/100 0000 and is around 10–20 times rarer than myasthenia gravis (MG). Although LEMS does have a number of similarities to MG, there are important differences. The syndrome is characterized by a mostly proximally localised exercise induced muscle weakness that can lead to respiratory failure often accompanied by autonomous dysfunction. Disease symptoms are caused by autoantibodies directed against P/Q type voltage gated calcium channels (VGCC) that are expressed in the presynaptic motoric nerve terminals. The diagnosis of LEMS is based on the detection of the pathogenic anti-VGCC antibodies as well as the observation of an increment of at least 60% in the electrophysiological examination of an affected muscle. An increment is defined by an increase of the at rest reduced compound muscle action potential (CMAP) either after voluntary maximal innervation or after high frequent (≥20 Hz) stimulation. In almost one third LEMS is of paraneoplastic origin. Therefore an intensive tumor screening is necessary after diagnosis.There are some differences in the clinical presentation between paraneoplastic (pLEMS) and the exclusively autoimmune (aiLEMS) form of LEMS. With respect to this the DELTA-P-Score and the detection of SOX1-antibody are important. The most frequent tumor associated with LEMS is small cell lung carcinoma (SCLC). Therapy is based on the initial distinction between paraneoplastic and autoimmune ethiology. pLEMS necessitates therapy of underlying neoplasia. Usually, aiLEMS- as well as pLEMS patients respond well to 3,4 diaminopyridine (3,4 DAP) often augmented by pyridostigmine. Similar to treatment of myasthenia gravis long-term immunosuppressive treatment is usually required to control symptoms effectively. Myasthenic crisis in LEMS can be controlled by intensive care and immunoglobulins, plasmaphereses or immunoadsorption. Based on case reports more specific immunomodulatory treatment approaches such as the B-cell depleting therapeutic antibody rituximab should be considered in therapy refractory courses of LEMS. Long-term prognosis of autoimmune LEMS with respect to clinical stabilization with (pharmacological) remission is good, although in around 75% of patients significant reductions in quality of life remain. Prognosis of tumor-associated LEMS is largely determined by the tumor and its effective therapy. Curative treatment of the tumour as well as complete remission of pLEMS are possible.

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28

Lorenzoni, Paulo José, Cláudia Suemi Kamoi Kay, Lineu Cesar Werneck, and Rosana Herminia Scola. "Lambert-Eaton myasthenic syndrome: the 60th anniversary of Eaton and Lambert's pioneering article." Arquivos de Neuro-Psiquiatria 76, no. 2 (February 2018): 124–26. http://dx.doi.org/10.1590/0004-282x20170194.

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ABSTRACT This historical review describes the contribution of Drs. Lee M. Eaton and Edward H. Lambert to the diagnosis of myasthenic syndrome on the 60th anniversary of their pioneering article (JAMA 1957) on the disease. There are important landmarks in their article on a disorder of the neuromuscular junction associated with thoracic neoplasm and the electrophysiological criteria for Lambert-Eaton myasthenic syndrome (LEMS). After 60 years, the main electrophysiological criteria described in Eaton and Lambert's pioneering article are still currently useful in the diagnosis of LEMS.

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29

Sanadze, A. G., D. V. Sidnev, and D. A. Tumurov. "Seronegative nonparaneoplastic Lambert—Eaton myasthenic syndrome." Zhurnal nevrologii i psikhiatrii im. S.S. Korsakova 117, no. 5 (2017): 77. http://dx.doi.org/10.17116/jnevro20171175177-80.

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30

Oh, S. J. "WS6-3 Lambert-Eaton myasthenic syndrome." Clinical Neurophysiology 121 (October 2010): S80. http://dx.doi.org/10.1016/s1388-2457(10)60337-9.

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31

Tsao, Chang-Yong, Jerry R. Mendell, Miriam L. Friemer, and John T. Kissel. "Lambert-Eaton Myasthenic Syndrome in Children." Journal of Child Neurology 17, no. 1 (January 2002): 74–76. http://dx.doi.org/10.1177/088307380201700123.

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32

Matsumoto, Hideyuki, and Yoshikazu Ugawa. "Lambert-Eaton Myasthenic Syndrome: A Review." Journal of General and Family Medicine 17, no. 2 (June 2016): 138–43. http://dx.doi.org/10.14442/jgfm.17.2_138.

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33

Motomura, Masakatsu, Ruka Nakata, and Hirokazu Shiraishi. "Lambert-Eaton myasthenic syndrome: Clinical review." Clinical and Experimental Neuroimmunology 7, no. 3 (August 2016): 238–45. http://dx.doi.org/10.1111/cen3.12326.

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34

Weingarten, Toby N., Cadmar N. Araka, Matthew E. Mogensen, Joseph P. Sorenson, Mary E. Marienau, James C. Watson, and Juraj Sprung. "Lambert-Eaton Myasthenic Syndrome During Anesthesia." Survey of Anesthesiology 59, no. 2 (April 2015): 71. http://dx.doi.org/10.1097/sa.0000000000000119.

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35

Maddison, Paul. "Treatment in Lambert-Eaton myasthenic syndrome." Annals of the New York Academy of Sciences 1275, no. 1 (December 2012): 78–84. http://dx.doi.org/10.1111/j.1749-6632.2012.06769.x.

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36

Morimoto, Mitsuaki, Toshihiro Osaki, Yuko Nagara, Mantaro Kodate, Masakatsu Motomura, and Hiroyuki Murai. "Thymoma With Lambert-Eaton Myasthenic Syndrome." Annals of Thoracic Surgery 89, no. 6 (June 2010): 2001–3. http://dx.doi.org/10.1016/j.athoracsur.2009.11.041.

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37

Kostera-Pruszczyk, A., B. Ryniewicz, K. Rowinska-Marcinska, M. Dutkiewicz, and A. Kamińska. "Lambert–Eaton myasthenic syndrome in childhood." European Journal of Paediatric Neurology 13, no. 2 (March 2009): 194–96. http://dx.doi.org/10.1016/j.ejpn.2008.03.006.

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38

Denys, E. H., and V. A. Lennon. "101. Asymptomatic Lambert–Eaton myasthenic syndrome." Clinical Neurophysiology 120, no. 2 (February 2009): e118. http://dx.doi.org/10.1016/j.clinph.2008.10.119.

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39

Souza, Paulo Victor Sgobbi de, Gabriel Novaes de Rezende Batistella, Valéria Cavalcante Lino, Wladimir Bocca Vieira de Rezende Pinto, Marcelo Annes, and Acary Souza Bulle Oliveira. "Clinical and genetic basis of congenital myasthenic syndromes." Arquivos de Neuro-Psiquiatria 74, no. 9 (September 2016): 750–60. http://dx.doi.org/10.1590/0004-282x20160106.

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ABSTRACT Neuromuscular junction disorders represent a wide group of neurological diseases characterized by weakness, fatigability and variable degrees of appendicular, ocular and bulbar musculature involvement. Its main group of disorders includes autoimmune conditions, such as autoimmune acquired myasthenia gravis and Lambert-Eaton syndrome. However, an important group of diseases include congenital myasthenic syndromes with a genetic and sometimes hereditary basis that resemble and mimick many of the classic myasthenia neurological manifestations, but also have different presentations, which makes them a complex clinical, therapeutic and diagnostic challenge for most clinicians. We conducted a wide review of congenital myasthenic syndromes in their clinical, genetic and therapeutic aspects.

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40

Oh, S. J., D. S. Dwyer, and R. J. Bradley. "Overlap myasthenic syndrome: Combined myasthenia gravis and Eaton-Lambert syndrome." Neurology 37, no. 8 (August 1, 1987): 1411. http://dx.doi.org/10.1212/wnl.37.8.1411.

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41

Prommer, Eric. "Neuromuscular Paraneoplastic Syndromes: The Lambert-Eaton Myasthenic Syndrome." Journal of Palliative Medicine 13, no. 9 (September 2010): 1159–62. http://dx.doi.org/10.1089/jpm.2009.0418.

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42

van Sonderen, Agnes, Paul W. Wirtz, Jan JGM Verschuuren, and Maarten J. Titulaer. "Treatment options for Lambert–Eaton myasthenic syndrome." Expert Opinion on Orphan Drugs 2, no. 2 (January 10, 2014): 159–67. http://dx.doi.org/10.1517/21678707.2014.872559.

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43

Warren, N. M., M. Bennett, M. Lai, J. Forty, and T. J. Walls. "Lambert-Eaton myasthenic syndrome associated with thymitis." Neurology 64, no. 1 (January 10, 2005): 168–69. http://dx.doi.org/10.1212/01.wnl.0000148722.40575.27.

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44

Gracey, Douglas R., and Peter A. Southorn. "Respiratory Failure in Lambert-Eaton Myasthenic Syndrome." Chest 91, no. 5 (May 1987): 716–18. http://dx.doi.org/10.1378/chest.91.5.716.

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45

Wilcox, Pearce G., Nancy J. Morrison, Andre R. A. Anzarut, and Richard L. Pardy. "Lambert-Eaton Myasthenic Syndrome Involving the Diaphragm." Chest 93, no. 3 (March 1988): 604–6. http://dx.doi.org/10.1378/chest.93.3.604.

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46

Erlington, Giles. "Review paper: The Lambert-Eaton myasthenic syndrome." Palliative Medicine 6, no. 1 (January 1992): 9–17. http://dx.doi.org/10.1177/026921639200600103.

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47

Robinson, Richard. "In the Pipeline-Lambert-Eaton Myasthenic Syndrome." Neurology Today 18, no. 3 (February 2018): 6–7. http://dx.doi.org/10.1097/01.nt.0000530607.25180.73.

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48

LoMonaco, M. "Decremental pattern in Lambert-Eaton myasthenic syndrome." Electroencephalography and Clinical Neurophysiology 103, no. 1 (July 1997): 126. http://dx.doi.org/10.1016/s0013-4694(97)88560-8.

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49

LANG, BETHAN, IRENE JOHNSTON, KATHERINE LEYS, GILES ELRINGTON, JOHN NEWSOM-DAVIS, BEATRICE MARQUEZE, CHRISTIAN LEVEQUE, et al. "Autoantibody Specificities in Lambert-Eaton Myasthenic Syndrome." Annals of the New York Academy of Sciences 681, no. 1 Myasthenia Gr (June 1993): 382–93. http://dx.doi.org/10.1111/j.1749-6632.1993.tb22917.x.

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50

Sanders, Donald. "Lambert-Eaton Myasthenic Syndrome: Pathogenesis and Treatment." Seminars in Neurology 14, no. 02 (June 1994): 111–17. http://dx.doi.org/10.1055/s-2008-1041067.

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