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Relevant bibliographies by topics / Langendorff-perfused rabbit heart

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Academic literature on the topic 'Langendorff-perfused rabbit heart'

Author: Grafiati

Published: 4 June 2021

Last updated: 17 February 2022

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Journal articles on the topic "Langendorff-perfused rabbit heart"

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Gralinski, M. R., S. C. Black, L. F. Stancato, K. S. Kilgore, P. A. Campau, J. L. Park, M. J. Ozeck, W. B. Pratt, and B. R. Lucchesi. "Heat stress protects the perfused rabbit heart from complement-mediated injury." American Journal of Physiology-Heart and Circulatory Physiology 271, no. 2 (August 1, 1996): H571—H578. http://dx.doi.org/10.1152/ajpheart.1996.271.2.h571.

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We determined if heat stress induction of heat shock protein (HSP) 70 modulates complement activation in an experimental model of xenograft rejection. Male New Zealand White rabbits were heat stressed (core body temperature to 42 degrees C for 15 min; n = 9). Control rabbits (n = 13) were not exposed to heat stress. Hearts were removed 18 h later and perfused by the Langendorff method. After equilibration, human plasma (source of human complement) was added to the perfusion medium. Hemodynamic variables recorded during perfusion with human plasma were improved in hearts from heat-stressed animals compared with control hearts. Assembly of the soluble membrane attack complex was reduced in the interstitial fluid effluent from the heat-stressed hearts. Electron microscopic evidence of ultrastructural changes was attenuated in the hearts from heat-stressed rabbits. Myocardial tissue from heat-stressed animals exhibited an increase in inducible HSP 70 that was virtually absent in the hearts of control rabbits. Previous whole body hyperthermia protects the rabbit heart against the detrimental effects of heterologous plasma, suggesting that heat-stress induction of HSP 70 limits the extent of complement activation by a discordant vascularized tissue (xenograft). Induction of heat stress proteins by the donor organ might be an important mechanism affecting the outcome of xenograft transplants.

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Kuzmin, V. S., Yu V. Egorov, and L. V. Rozenshtraukh. "Electrhopysiological Effect of the Polyamine Spermine in Normoxic and Ischemic Ventricular Myocardium." Kardiologiia 59, no. 3 (April 13, 2019): 43–51. http://dx.doi.org/10.18087/cardio.2019.3.10240.

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Cytoplasmic polyamines (PA) are involved in control of many cellular functions and are well known as regulators of so called inward-rectifier potassium ion channels. Nevertheless, functional significance of extracellular PA in the heart is poorly elucidated. Aim of this study was to study effects of endogenous PA spermine in the ventricular myocardium. Effects of the extracellular spermine were investigated in isolated multicellular preparations of rabbit and rat ventricular myocardium. Langendorff-perfused isolated rat and rabbit hearts were also used. Action potential (APs) duration and pattern of excitation in ventricular myocardium were estimated using standard microelectrode technique and optical mapping. Functional refractory periods were assessed in Langendorff perfused hearts with the help of programmedelectrical stimulation of the ventricle. In this study extracellular PA spermine (0.1–5 mM) induced shortening of the APs in multicellular preparations of rat ventricular myocardium registered using sharp microelectrode technique. However, spermine caused only weak effect in preparations of ventricular myocardium from rabbit heart: highest tested concentration of spermine (5 mM) induced 4.7 % APs shortening. Similarly, 0.1–1 mM of spermine was unable to alter substantially ventricular effective refractory periods in isolated perfused rabbit hearts. In two animal species tested (rat and rabbit) 0.1–1 mM of spermine failed to affect conduction velocity and activation pattern in ventricles of isolated Langendorff-perfused hearts under normoxia. However, in the rat no-flow model of ischemia-reperfusion extracellular spermine improved conduction of excitation in ventricles. Our results allow suggesting that extracellular spermine can prevent ischemia-induced proarrhythmic changes in ventricular myocardium probably due to reduction of calcium accumulation, but this effect is significant only when PA is applied in millimolar concentrations. Also, potential anti-ischemic effect of the PA may be species specific.

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Mischke, Karl, Markus Zarse, Christian Knackstedt, and Patrick Schauerte. "Rate Control in Atrial Fibrillation by Cooling: Effect of Temperature on Dromotropy in Perfused Rabbit Hearts." Cardiology Research and Practice 2011 (2011): 1–4. http://dx.doi.org/10.4061/2011/162984.

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Background. Cooling has emerged as a therapeutic option in critically ill patients (especially after cardiac resuscitation) and might also have a negative dromotropic effect in atrial fibrillation. We sought to determine the impact of cooling on electrophysiologic properties of Langendorff-perfused rabbit hearts.Methods and Results. In 20 isolated Langendorff-perfused rabbit hearts, the temperature of the tissue bath was changed between 17 and 42°C. With decreasing temperature, significant increases of the spontaneous sinus cycle length, decreases of the mean ventricular heart rate during atrial fibrillation, and relevant increases of atrial and ventricular refractory periods were observed (ANOVAP<.01).Conclusions. Cardiac hypothermia leads to a significant drop of mean ventricular heart rate during atrial fibrillation. Negative chronotropy and dromotropy induced by moderate cardiac hypothermia might be a feasible therapeutic approach in patients with hemodynamically relevant tachyarrhythmias in a CCU/ICU setting.

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Neuber, Johanna U., Andrei G. Pakhomov, and Christian W. Zemlin. "Electroporation safety factor of 300 nanosecond and 10 millisecond defibrillation in Langendorff-perfused rabbit hearts." PLOS ONE 16, no. 9 (September 24, 2021): e0257287. http://dx.doi.org/10.1371/journal.pone.0257287.

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Aims Recently, a new defibrillation modality using nanosecond pulses was shown to be effective at much lower energies than conventional 10 millisecond monophasic shocks in ex vivo experiments. Here we compare the safety factors of 300 nanosecond and 10 millisecond shocks to assess the safety of nanosecond defibrillation. Methods and results The safety factor, i.e. the ratio of median effective doses (ED50) for electroporative damage and defibrillation, was assessed for nanosecond and conventional (millisecond) defibrillation shocks in Langendorff-perfused New Zealand white rabbit hearts. In order to allow for multiple shock applications in a single heart, a pair of needle electrodes was used to apply shocks of varying voltage. Propidium iodide (PI) staining at the surface of the heart showed that nanosecond shocks had a slightly lower safety factor (6.50) than millisecond shocks (8.69), p = 0.02; while PI staining cross-sections in the electrode plane showed no significant difference (5.38 for 300 ns shocks and 6.29 for 10 ms shocks, p = 0.22). Conclusions In Langendorff-perfused rabbit hearts, nanosecond defibrillation has a similar safety factor as millisecond defibrillation, between 5 and 9, suggesting that nanosecond defibrillation can be performed safely.

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Morgan, Eric E., Zhichuan Li, Cory Stebal, Aude Belliard, Glen Tennyson, Bijan Salari, Keith D. Garlid, and Sandrine V. Pierre. "Preconditioning by Subinotropic Doses of Ouabain in the Langendorff perfused Rabbit Heart." Journal of Cardiovascular Pharmacology 55, no. 3 (March 2010): 234–39. http://dx.doi.org/10.1097/fjc.0b013e3181ce5e14.

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Salbaş, K., G. Akgün, G. Pamīr, and T. Gürel. "Interaction between Beta-Blockers and Contrast Media during Intracoronary Administration in Rabbit Heart." Acta Radiologica 35, no. 1 (January 1994): 70–76. http://dx.doi.org/10.1177/028418519403500115.

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The effects of ionic, meglumine sodium diatrizoate (Urografin 76) and nonionic, iohexol contrast media were examined in the absence and presence of propranolol, a beta-blocker having nonspecific membrane stabilizing action and atenolol, a beta-blocker lacking nonspecific membrane stabilizing action on Langendorff-perfused rabbit hearts. Contrast medium, 0.7 ml, was injected into the aortic root in the absence and presence of 10−7 and 10−5 M beta-blocker to observe the changes in resting tension, force of contraction, rate of contraction, heart rate and PR interval. Beta-blockers and contrast media interact in affecting myocardial contractility, heart rate and atrioventricular conduction. The interaction is milder when the beta-blocker lacks membrane stabilizing activity and the contrast medium is nonionic.

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Guillon, Jean Michel, Michel Roche, Veronique Ballet, and Michel Doubovetzky. "Electrocardiographic effects of moxifloxacin, quinidine and quinimax on a langendorff-perfused rabbit heart model." Journal of Pharmacological and Toxicological Methods 62, no. 2 (September 2010): e37. http://dx.doi.org/10.1016/j.vascn.2010.11.126.

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Laycock, Sarra, Neal Appleton, Anouska Godier, and Kirsty Macfarlane. "FREQUENCY-DEPENDENT EFFECTS OF PRO-ARRHYTHMIC COMPOUNDS IN THE LANGENDORFF-PERFUSED ISOLATED RABBIT HEART." Journal of Pharmacological and Toxicological Methods 56, no. 2 (September 2007): e52. http://dx.doi.org/10.1016/j.vascn.2007.02.104.

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Klair, Sarbjit S., James B. Louttit, and Peter A. Charlton. "Atrial natriuretic factor in the Langendorff perfused coronary vasculature of the rabbit isolated heart." Life Sciences 45, no. 25 (January 1989): 2477–83. http://dx.doi.org/10.1016/0024-3205(89)90014-3.

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Vosyliute, Ruta, Arvydas Matiukas, Irma Martišienė, Jonas Jurevičius, Antanas Navalinskas, Rimantas Treinys, Regina Mačianskienė, and Arkady M. Pertsov. "Illumination-Induced Changes in Action Potential during Optical Mapping in Langendorff-Perfused Rabbit Heart." Biophysical Journal 104, no. 2 (January 2013): 337a. http://dx.doi.org/10.1016/j.bpj.2012.11.1875.

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Dissertations / Theses on the topic "Langendorff-perfused rabbit heart"

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Kappadan, Vineesh [Verfasser]. "Ratiometric fluorescence imaging and marker-free motion tracking of Langendorff perfused beating rabbit hearts / Vineesh Kappadan." Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2021. http://d-nb.info/1224681673/34.

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Berg, Sebastian Stephan. "Characterization and Control of Wave Propagation in the Heart." Doctoral thesis, 2018. http://hdl.handle.net/11858/00-1735-0000-002E-E607-5.

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Kappadan, Vineesh. "Ratiometric fluorescence imaging and marker-free motion tracking of Langendorff perfused beating rabbit hearts." Doctoral thesis, 2020. http://hdl.handle.net/21.11130/00-1735-0000-0005-1538-5.

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Book chapters on the topic "Langendorff-perfused rabbit heart"

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Khatib, Said Y., and Mark R. Boyett. "Effects of glyburide (glibenclamide) on myocardial function in Langendorff perfused rabbit heart and on myocardial contractility and slow calcium current in guinea-pig single myocytes." In Cardiac Cell Biology, 81–87. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4757-4712-6_11.

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Kolářová, J., O. Janoušek, M. Nováková, K. Fialová, and I. Provazník. "Influence of ischemia on left ventricular depolarization in isolated rabbit hearts perfused according to Langendorff." In IFMBE Proceedings, 1419–22. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-03882-2_376.

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Conference papers on the topic "Langendorff-perfused rabbit heart"

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Janousek, Oto, Marina Ronzhina, Jakub Hejc, Veronika Olejnickova, Tibor Stracina, Katerina Fialova, Marie Novakova, Ivo Provaznik, and Jana Kolarova. "The effect of voltage-sensitive dye di-4-ANEPPS on heart rate variability in Langendorff-perfused isolated rabbit heart." In 2015 Computing in Cardiology Conference (CinC). IEEE, 2015. http://dx.doi.org/10.1109/cic.2015.7411094.

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Salameh, A., I. Dähnert, and S. Dhein. "Olesoxime Inhibits Cardioplegia-Induced Ischemia/Reperfusion Injury: A Study in Langendorff-Perfused Rabbit Hearts." In 50th Annual Meeting of the German Society for Pediatric Cardiology (DGPK). Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1628325.

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