Relevant bibliographies by topics / Large artery stiffness

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'Large artery stiffness'

Author: Grafiati
Published: 9 March 2023
Last updated: 31 July 2025

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Journal articles on the topic "Large artery stiffness"

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Benetos, Athanase, Stéphane Laurent, Roland G. Asmar, and Patrick Lacolley. "Large artery stiffness in hypertension." Journal of Hypertension 15 (1997): S89—S97. http://dx.doi.org/10.1097/00004872-199715022-00009.

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Mitchell, Gary F. "Proteomics of Large Artery Stiffness." JACC: Basic to Translational Science 9, no. 10 (2024): 1192–94. http://dx.doi.org/10.1016/j.jacbts.2024.07.011.

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McEniery, C. M., and I. B. Wilkinson. "Large artery stiffness and inflammation." Journal of Human Hypertension 19, no. 7 (2005): 507–9. http://dx.doi.org/10.1038/sj.jhh.1001814.

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Blacher, J., Athanase Protogerou, and M. Safar. "Large Artery Stiffness and Antihypertensive Agents." Current Pharmaceutical Design 11, no. 25 (2005): 3317–26. http://dx.doi.org/10.2174/138161205774424654.

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Chirinos, Julio A. "Echocardiographic Assessment of Large Artery Stiffness." Journal of the American Society of Echocardiography 29, no. 11 (2016): 1117–21. http://dx.doi.org/10.1016/j.echo.2016.09.004.

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Chirinos, Julio A. "Large Artery Stiffness and New-Onset Diabetes." Circulation Research 127, no. 12 (2020): 1499–501. http://dx.doi.org/10.1161/circresaha.120.318317.

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Kingwell, Bronwyn A., Tanya L. Medley, Tamara K. Waddell, Timothy J. Cole, Anthony M. Dart, and Garry L. Jennings. "Large Artery Stiffness: Structural And Genetic Aspects." Clinical and Experimental Pharmacology and Physiology 28, no. 12 (2001): 1040–43. http://dx.doi.org/10.1046/j.1440-1681.2001.03580.x.

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Butlin, Mark, and Ahmad Qasem. "Large Artery Stiffness Assessment Using SphygmoCor Technology." Pulse 4, no. 4 (2016): 180–92. http://dx.doi.org/10.1159/000452448.

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Tan, Isabella, Bart Spronck, Hosen Kiat, et al. "Heart Rate Dependency of Large Artery Stiffness." Hypertension 68, no. 1 (2016): 236–42. http://dx.doi.org/10.1161/hypertensionaha.116.07462.

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Chirinos, Julio A., Patrick Segers, Timothy Hughes, and Raymond Townsend. "Large-Artery Stiffness in Health and Disease." Journal of the American College of Cardiology 74, no. 9 (2019): 1237–63. http://dx.doi.org/10.1016/j.jacc.2019.07.012.

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Dissertations / Theses on the topic "Large artery stiffness"

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Al, Maskari Raya. "Large artery stiffness : genes and pathways." Thesis, University of Cambridge, 2018. https://www.repository.cam.ac.uk/handle/1810/277874.

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Aortic stiffness underlies systolic hypertension, promotes heart failure and is associated with increased cardiovascular morbidity and mortality. It is regarded as a primary driver of left ventricular hypertrophy and aortic aneurysms and is linked to the pathogenesis of cognitive impairment, stroke and renal failure. Like most cardiovascular traits, aortic stiffness is a complex trait and is moderately heritable, yet the precise molecular mechanisms that underpin the stiffening process remain poorly defined. This study aimed to employ multiple approaches to further identify the genetic basis o
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Brodjeski, Alexander Lee. "Reduced SIRT3 contributes to large elastic artery stiffness with aging." Thesis, University of Iowa, 2017. https://ir.uiowa.edu/etd/5426.

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Age-related increases in arterial stiffness are mediated in part by mitochondrial dysfunction. Sirtuin 3 (SIRT3) is a mitochondrial NAD+-dependent deacetylase that regulates mitochondrial function. SIRT3 deficiency contributes to physiological dysfunction in a variety of pathological conditions. Here, we tested the hypothesis that age-associated arterial stiffness, assessed by aortic pulse wave velocity (PWV), would be accompanied with decreased renal and aortic SIRT3 expression and activity due to decreased NAD+ levels. We further tested whether boosting NAD+ concentration with nicotinamide r
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Wallace, Sharon Mary Louise. "Nitric oxide and large artery stiffness : investigation of mechanisms and clinical implications." Thesis, University of Cambridge, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.612222.

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Westerbacka, Jukka. "Insulin action on large artery stiffness in normal and insulin resistant subjects." Helsinki : University of Helsinki, 2001. http://ethesis.helsinki.fi/julkaisut/laa/kliin/vk/westerbacka/.

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Butlin, Mark Graduate School of Biomedical Engineering Faculty of Engineering UNSW. "Structural and functional effects on large artery stiffness: an in-vivo experimental investigation." Awarded by:University of New South Wales. Graduate School of Biomedical Engineering, 2007. http://handle.unsw.edu.au/1959.4/29479.

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Large artery stiffness is predictive of adverse cardiovascular events and all cause mortality. Artery structure and function are determinants of artery stiffness. This thesis presents a series of in-vivo experimental studies of effect of structural and functional changes on large artery stiffness. Improved analysis methods were developed for measurement of arterial stiffness indexes, Pulse Wave Velocity (PWV) and pressure wave re ection. These were applied in studies of acute in ammation, active and passive changes in systemic pressures, aortic elastic laminae defects, and aortic calcification
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Werner, Timothy Jason. "Effect of Nebivolol and Lifestyle Modification on Large Artery Stiffness in Middle-Aged and Older Hypertensive Adults." Diss., Virginia Tech, 2013. http://hdl.handle.net/10919/23316.

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For more than half a century cardiovascular disease has been the leading cause of death in the United States.  Aging, hypertension, and obesity are major risk factors for cardiovascular disease and clearly associated with arterial stiffness.  Arterial stiffness generates higher afterloads and diminishes coronary perfusion thereby causing ventricular hypertrophy and ischemia.  Importantly, arterial stiffness is an independent predictor of cardiovascular disease risk and all-cause mortality.   Current strategies such as inhibition of angiotensin II or angiotensin converting enzyme, reduction of
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DuBose, Lyndsey Elisabeth. "Role of aging and aerobic fitness on large elastic artery stiffness, brain structure and cognitive performance in humans." Thesis, University of Iowa, 2015. https://ir.uiowa.edu/etd/1590.

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Older age is a primary risk factor for the development of cardiovascular disease in part through the stiffening of the large cardiothoracic elastic arteries (e.g., aorta, carotid arteries). Aging is also associated with reduced cognitive function, cerebrovascular reactivity and brain white matter integrity, but whether these changes in brain structure and function are associated with age-related large artery stiffness remains unclear. In contrast, older adults who have high aerobic fitness demonstrate attenuated large artery stiffness and better cognitive performance compared to their sedentar
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Ajibewa, Tiwaloluwa Adedamola. "Role of anxiety on vascular dysfunction." Thesis, University of Iowa, 2016. https://ir.uiowa.edu/etd/3035.

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High anxiety is associated with an increased risk of developing cardiovascular disease (CVD), in particular, atherosclerotic coronary artery disease. However, the mechanisms by which anxiety contributes to the development of CVD are unclear. Unlike other common psychiatric disorders such as depression, anxiety and its effects on CVD risk has not been studied extensively. Moreover, whether elevated anxiety is associated with arterial stiffness and vascular endothelial dysfunction, biomarkers of CVD risk, in healthy adults and whether a psychological intervention designed to lower anxiety levels
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OCHOA, MUNERA JUAN EUGENIO. "Effects of insulin resistance on systemic haemodynamics and autonomic cardiovascular regulation in normotensive healthy adults." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2013. http://hdl.handle.net/10281/46090.

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Hemodynamic effects of insulin resistance (IR) are thought to be largely dependent on its relationship with body mass index (BMI) and blood pressure (BP) levels. The first part of the present thesis was aimed at exploring whether IR is associated with hemodynamic indices of cardiovascular function in a large sample of non-diabetic individuals from the general population (n=731) and if so, to explore if such relationship is continuous across different categories of BMI (lean, overweight and obese), and BP (normal BP, high-normal BP and hypertension). IR was assessed with the homeostasis model a
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Maritz, Melissa. "Vascular and metabolic profile of 5-year sustained hypertensive versus normotensive black South Africans / Melissa Maritz." Thesis, 2014. http://hdl.handle.net/10394/15399.

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Motivation A close association exists between hypertension and arterial stiffness. Whether the increased arterial stiffness seen in hypertensives are due to structural or functional adaptations in the vasculature is uncertain. Hypertension is more common in blacks and they have an increased arterial stiffness and higer stroke prevalence than white populations. Arterial stiffening, or a loss of arterial distensibility, increases the risk for cardiovascular events, including stroke and heart failure, as it increases the afterload on the heart, as well as creating a higher pulsatile load on the m
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Book chapters on the topic "Large artery stiffness"

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Climie, Rachel E. D. "Endothelial Dysfunction and Large Artery Stiffness." In Blood Pressure Disorders in Diabetes Mellitus. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-13009-0_12.

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Gielen, Stephan, M. Harold Laughlin, and Dirk J. Duncker. "Vascular remodelling." In The ESC Textbook of Sports Cardiology, edited by Antonio Pelliccia, Hein Heidbuchel, Domenico Corrado, Mats Börjesson, and Sanjay Sharma. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780198779742.003.0005.

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Vascular remodelling plays an important role in the adaptation of the athlete to increased exercise duration and intensity. Endurance exercise improves endothelium-dependent flow-mediated vasodilation and leads to increases in conduit artery lumen diameter after regular exercise, typically in the trained limb. These changes result in a reduced vascular stiffnes. On the contrary strength training (e.g. for weight-lifting) produces increased vascular stiffness and enlarged central vessels (e.g. aortic root diameters), while the diameters of peripheral vessels are unchanged. In the skeletal muscl
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Capric, Violeta, Harshith Priyan Chandrakumar, Jessica Celenza-Salvatore, and Amgad N. Makaryus. "The Role of the Renin-Angiotensin-Aldosterone System in Cardiovascular Disease: Pathogenetic Insights and Clinical Implications." In Renin-Angiotensin Aldosterone System [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.96415.

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Increased attention has been placed on the activation of the renin-angiotensin-aldosterone system (RAAS) and pathogenetic mechanisms in cardiovascular disease. Multiple studies have presented data to suggest that cardiac and arterial stiffness leading to adverse remodeling of both the heart and vasculature leads to the various pathological changes seen in coronary artery disease, heart failure (with preserved and reduced ejection fractions), hypertension and renal disease. Over-activation of the RAAS is felt to contribute to these structural and endocrinological changes through its control of
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Silman, Alan J., and Marc C. Hochberg. "Polymyalgia rheumatica and giant cell arteritis." In Epidemiology of the Rheumatic Diseases. Oxford University PressNew York, NY, 1993. http://dx.doi.org/10.1093/oso/9780192623560.003.0010.

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Abstract Polymyalgia rheumatica (PMR) and giant cell (syn. temporal) artent1s (GCA) are two related disorders which, for epidemiological purposes, are best considered together. PMR is a relatively ill-defined clinical syndrome of limb-girdle pain and stiffness in association with systemic features such as pyrexia and a raised erythrocyte sedimentation rate (ESR). Diagnosis depends on the presence of combinations of the relevant features in the absence of another pathology such as rheumatoid arthritis or multiple myeloma. By contrast GCA is a vasculitic disorder, which can be widely disseminate
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K. Day, Thomas. "Role of Arterial Pressure, Wall Stiffness, Pulse Pressure and Waveform in Arterial Wall Stress/Strain and Its Clinical Implications." In Risk Factors for Cardiovascular Disease. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.100048.

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Biomechanical stress applied to the intima of arteries has long been suspected as a factor in the initiation and localisation of atherosclerotic plaque, and it is implicated in the separation of plaque from the underlying arterial wall giving rise to the acute clinical consequences of thrombosis, dissection and embolism. The factors underlying transmural stress were investigated in-vitro using fresh porcine abdominal aortas on an experimental rig in which pulse pressure, pulse waveform, fluid viscosity, pulse rate, vessel wall compliance and systolic and diastolic blood pressure could be varie
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Conference papers on the topic "Large artery stiffness"

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Ooi, Chen Yen, and Naomi C. Chesler. "The Role of Collagen in Pulmonary Hypertension-Induced Large Artery Stiffening." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192951.

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Hypoxic pulmonary hypertension (HPH) leads to stiffening of large pulmonary arteries, which affects right ventricular afterload. We hypothesized that vascular collagen accumulation is the major cause of large pulmonary artery (PA) stiffening in HPH. We tested this hypothesis with transgenic mice that produce collagen type I resistant to degradation (Col1a1R/R) and wild type littermate controls (Col1a1+/+) exposed to hypoxia and allowed to recover. Pressure-diameter testing on left PAs demonstrated that stiffness in control mice increased with hypoxia and decreased with recovery (p < 0.05).
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Desai, Vaidehi S., Alberto P. Avolio, Isabella Tan, Karen C. Peebles, and Mark Butlin. "Interaction of large artery stiffness and baroreceptor function explored through multiple measurement techniques - a pilot study." In 2023 45th Annual International Conference of the IEEE Engineering in Medicine & Biology Society (EMBC). IEEE, 2023. http://dx.doi.org/10.1109/embc40787.2023.10340723.

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Demoux, AL, K. Aissi, B. Chaudier, et al. "FRI0696 Impact of adalimumab therapy on brachial endothelial function and large artery stiffness in patients with rheumatoid arthritis." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.6128.

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Dodson, Reuben Blair, Paul J. Rozance, Kendall S. Hunter, and Virginia L. Ferguson. "Increased Stiffness of the Abdominal Aorta With Intrauterine Growth Restriction in the Near-Term Fetal Sheep." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80634.

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Fetal intrauterine growth restriction (IUGR) results in increased placental resistance to blood flow, fetal hypertension and increased pulsatility [1]. These hemodynamic changes have been shown to lead to vascular remodeling in adolescents and adults [2, 3] but have received little study of its effect during this critical period of vascular formation. Epidemiological studies link IUGR to cardiovascular disease in adulthood [4], but the reason for this is not clearly understood. Here, we examine a large elastic artery for developmental alterations under hypertensive conditions. We hypothesize t
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Wang, Zhijie, Roderic S. Lakes, and Naomi C. Chesler. "Changes in Conduit Pulmonary Arterial Static and Dynamic Mechanical Properties During Severe Hypoxic Pulmonary Hypertension." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80382.

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Pulmonary hypertension (PH) is a complex disorder that manifests as abnormally high blood pressure in the vasculature of the lungs. The chronic structural and mechanical changes in the proximal pulmonary artery (PA) associated with PH include smooth muscle cell hypertrophy and proliferation, accumulation of extracellular matrix (ECM) protein and increased stiffness1–4. Recent evidence has shown that conduit PA stiffness is a strong predictor of mortality in pulmonary arterial hypertension5,6. This suggests a potential association between large PA biomechanics and right ventricle failure.
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Liu, Aiping, Lian Tian, Diana M. Tabima, and Naomi C. Chesler. "Sex Differences in Right Ventricular-Vascular Coupling and Pulmonary Artery Impedance in Response to Chronic Hypoxia and Recovery." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80835.

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Pulmonary artery hypertension (PAH) is a female dominant disease (the female-to-male ratio is 4:1), characterized by small distal pulmonary arterial narrowing and large proximal arterial stiffening, which increase right ventricle (RV) afterload and ultimately lead to RV failure [1,2]. Our recent studies have shown that collagen accumulation induced by chronic hypoxia increases the stiffness of the large extralobar pulmonary arteries (PAs) [3], and affects pulmonary vascular impedance (PVZ) [4]. The role of collagen in the female predominance in developing PAH has not been explored to date.
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Gourisankaran, Vijay. "Effects of Stenosis Severity and Balloon Characteristics on Stresses in Balloon Angioplasty — A FEM Study." In ASME 1999 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1999. http://dx.doi.org/10.1115/imece1999-0465.

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Abstract Studies of stresses in an artery under normal physiological pressure loads have been made to gain insight into the possible causes of plaque rupture and progression of atherosclerosis. This study addresses the effects of stenosis severity, and balloon stiffness on the peak stresses experienced in the system during angioplasty. It is proposed that the effectiveness and therefore success of an angioplasty procedure can be measured by the non-propensity for restenosis to occur, and the absence of high stresses in the diseased artery at peak balloon dilation pressures. The models used in
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Hansen, Laura, Manu Platt, Roy L. Sutliff, and Rudolph L. Gleason. "The Mechanical and Structural Effects of HIV Proteins on Murine Carotid Arteries." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53693.

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Acquired immunodeficiency syndrome (AIDS) is considered a global epidemic with over 65 million people worldwide infected with the HIV-1 virus, the causative agent [1]. The development of highly active antiretroviral therapy (HAART) has significantly increased the life expectancy of people infected with the virus by slowing the progression to the development of AIDS. However, the treatment has also led to the emergence of early onset cardiovascular complications including myocardial infarction [2] and atherosclerotic lesions [3], as well as subclinical markers of atherosclerosis including incre
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Delgado, Ana, Pengfei Dong, Mahyar Sameti, et al. "Mechanical Characterization of Calcificaiton in Diseased Coronary Artery with Atomic Force Microscope." In 2022 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2022. http://dx.doi.org/10.1115/dmd2022-1055.

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Abstract In this work, the mechanical propertied of calcification in diseased coronary artery was evaluated with atomic force microscope (AFM). The heavily calcified coronary artery was harvest from a cadaver’s heart. The artery slices with thickness of 10 um were prepared with cryosectioning. Staining with Alizarin Red has been performed to highlight the calcification region. Results have shown that the calcified areas have a significant larger stiffness compared with the surrounding plaque and the media layer of a healthy artery. The calcification showed a heterogeneous property with larger
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