Relevant bibliographies by topics / Leaky-gut syndrome

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Leaky-gut syndrome'

Author: Grafiati
Published: 4 June 2021
Last updated: 28 January 2023

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Journal articles on the topic "Leaky-gut syndrome"

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Obrenovich, Mark. "Leaky Gut, Leaky Brain?" Microorganisms 6, no. 4 (October 18, 2018): 107. http://dx.doi.org/10.3390/microorganisms6040107.

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‘Leaky gut’ syndrome, long-associated with celiac disease, has attracted much attention in recent years and for decades, was widely known in complementary/alternative medicine circles. It is often described as an increase in the permeability of the intestinal mucosa, which could allow bacteria, toxic digestive metabolites, bacterial toxins, and small molecules to ‘leak’ into the bloodstream. Nervous system involvement with celiac disease is know to occur even at subclinical levels. Gluten and gluten sensitivity are considered to trigger this syndrome in individuals genetically predisposed to celiac disease. However, the incidence of celiac disease in the general population is quite low. Nevertheless, increased public interest in gluten sensitivity has contributed to expanded food labels stating ‘gluten-free’ and the proliferation of gluten-free products, which further drives gluten-free lifestyle changes by individuals without frank celiac disease. Moreover, systemic inflammation is associated with celiac disease, depression, and psychiatric comorbidities. This mini-review focuses on the possible neurophysiological basis of leaky gut; leaky brain disease; and the microbiota’s contribution to inflammation, gastrointestinal, and blood-brain barrier integrity, in order to build a case for possible mechanisms that could foster further ‘leaky’ syndromes. We ask whether a gluten-free diet is important for anyone or only those with celiac disease.
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Kim, Kyu-Nam, and Kwang-Min Kim. "Leaky Gut Syndrome." Korean Journal of Family Practice 10, no. 5 (October 20, 2020): 316–23. http://dx.doi.org/10.21215/kjfp.2020.10.5.316.

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Park, Junghyun, Tae Joon Choi, Ki Sung Kang, and Seo-Hyung Choi. "The Interrelationships between Intestinal Permeability and Phlegm Syndrome and Therapeutic Potential of Some Medicinal Herbs." Biomolecules 11, no. 2 (February 15, 2021): 284. http://dx.doi.org/10.3390/biom11020284.

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The gastrointestinal (GI) tract has an intriguing and critical role beyond digestion in both modern and complementary and alternative medicine (CAM), as demonstrated by its link with the immune system. In this review, we attempted to explore the interrelationships between increased GI permeability and phlegm, an important pathological factor in CAM, syndrome, and therapeutic herbs for two disorders. The leaky gut and phlegm syndromes look considerably similar with respect to related symptoms, diseases, and suitable herbal treatment agents, including phytochemicals even though limitations to compare exist. Phlegm may be spread throughout the body along with other pathogens via the disruption of the GI barrier to cause several diseases sharing some parts of symptoms, diseases, and mechanisms with leaky gut syndrome. Both syndromes are related to inflammation and gut microbiota compositions. Well-designed future research should be conducted to verify the interrelationships for evidence based integrative medicine to contribute to the promotion of public health. In addition, systems biology approaches should be adopted to explore the complex synergistic effects of herbal medicine and phytochemicals on conditions associated with phlegm and leaky gut syndromes.
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Brom, B. "Integrative medicine and leaky gut syndrome." South African Family Practice 52, no. 4 (July 2010): 314–16. http://dx.doi.org/10.1080/20786204.2010.10873997.

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Farshchi, Masoumeh Kaboli, Farahzad Jabbari Azad, Roshanak Salari, Majid Mirsadraee, and Majid Anushiravani. "A Viewpoint on the Leaky Gut Syndrome to Treat Allergic Asthma: A Novel Opinion." Journal of Evidence-Based Complementary & Alternative Medicine 22, no. 3 (December 22, 2016): 378–80. http://dx.doi.org/10.1177/2156587216682169.

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Asthma is a common respiratory disease characterized by airway inflammation, airway hyperreactivity, and reversible airflow obstruction. Despite current treatments, the prevalence of asthma has increased markedly over decades. According to the theories proposed to explain the pathophysiology of autoimmune diseases in integrative medicine, leaky gut syndrome is a phenomenon of increased intestinal permeability due to the disruption of tight junctions and is thought to be related to many chronic diseases, such as food intolerance, inflammatory bowel disease, rheumatoid arthritis, asthma, and other autoimmune disease. One of the classical approaches used by integrative physicians to treat leaky gut syndrome is to repair intestinal permeability to prevent allergic cascade. Due to several mechanisms that have been mentioned in the protective effects of plant gums and plantain family seeds on the intestinal epithelium, we can propose an effective management for leaky gut syndrome to treat asthma.
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Rodina, Jekaterina, and Aleksejs Derovs. "Establishing the cut-offs of leaky gut syndrome diagnostic: where are we now?" Proceedings of the Latvian Academy of Sciences. Section B. Natural, Exact, and Applied Sciences. 76, no. 5-6 (December 1, 2022): 569–77. http://dx.doi.org/10.2478/prolas-2022-0089.

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Abstract Gastrointestinal mucosa forms a surface that interacts with many external factors. Beside the digestion and absorption of nutrients, it also acts as a barrier to allergens, pathogens, and toxins. Leaky gut syndrome is defined as a gut mucosal barrier dysfunction, which results in abnormally increased intestinal permeability. Research shows that leaky gut syndrome (LGS) has a pathogenetic relationship with a series of gastrointestinal and extra-intestinal disorders. This review discusses the current understanding of intestinal barrier composition and pathological contribution of LGS to various diseases. The major aim of this paper is to review different methods for diagnostics and evaluation of intestinal wall permeability, identifying their priorities and disadvantages.
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Chernyavskyi, V. V., L. L. Pavlovskyi, and V. V. Tishchenko. "Leaky gut syndrome in the general clinical practice." Modern Gastroenterology, no. 3 (May 19, 2020): 91–95. http://dx.doi.org/10.30978/mg-2020-3-91.

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Twardowska, Agata, Adam Makaro, Agata Binienda, Jakub Fichna, and Maciej Salaga. "Preventing Bacterial Translocation in Patients with Leaky Gut Syndrome: Nutrition and Pharmacological Treatment Options." International Journal of Molecular Sciences 23, no. 6 (March 16, 2022): 3204. http://dx.doi.org/10.3390/ijms23063204.

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Leaky gut syndrome is a medical condition characterized by intestinal hyperpermeability. Since the intestinal barrier is one of the essential components maintaining homeostasis along the gastrointestinal tract, loss of its integrity due to changes in bacterial composition, decreased expression levels of tight junction proteins, and increased concentration of pro-inflammatory cytokines may lead to intestinal hyperpermeability followed by the development of gastrointestinal and non-gastrointestinal diseases. Translocation of microorganisms and their toxic metabolites beyond the gastrointestinal tract is one of the fallouts of the leaky gut syndrome. The presence of intestinal bacteria in sterile tissues and distant organs may cause damage due to chronic inflammation and progression of disorders, including inflammatory bowel diseases, liver cirrhosis, and acute pancreatitis. Currently, there are no medical guidelines for the treatment or prevention of bacterial translocation in patients with the leaky gut syndrome; however, several studies suggest that dietary intervention can improve barrier function and restrict bacteria invasion. This review contains current literature data concerning the influence of diet, dietary supplements, probiotics, and drugs on intestinal permeability and bacterial translocation.
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Simeonova, Denitsa, Mariya Ivanovska, Mariana Murdjeva, Andre F. Carvalho, and Michael Maes. "Recognizing the Leaky Gut as a Trans-diagnostic Target for Neuroimmune Disorders Using Clinical Chemistry and Molecular Immunology Assays." Current Topics in Medicinal Chemistry 18, no. 19 (December 21, 2018): 1641–55. http://dx.doi.org/10.2174/1568026618666181115100610.

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Background: Increased intestinal permeability with heightened translocation of Gramnegative bacteria, also known as “leaky gut”, is associated with the pathophysiology of neuroimmune disorders, such as Major Depressive Disorder (MDD), Chronic Fatigue Syndrome (CSF) and (deficit) schizophrenia, as well as with general medical disorders, including irritable bowel syndrome. This review aims to summarize clinical biochemistry and molecular immunology tests that may aid in the recognition of leaky gut in clinical practice. <p> Methods: We searched online libraries, including PubMed/MEDLINE, Google Scholar and Scopus, with the key words “diagnosis” or “biomarkers” and “leaky gut”, “bacterial translocation”, and “intestinal permeability” and focused on papers describing tests that may aid in the clinical recognition of leaky gut. <p> Results: To evaluate tight junction barrier integrity, serum IgG/IgA/IgM responses to occludin and zonulin and IgA responses to actomyosin should be evaluated. The presence of cytotoxic bacterial products in serum can be evaluated using IgA/IgM responses to sonicated samples of common Gram-negative gut commensal bacteria and assays of serum lipopolysaccharides (LPSs) and other bacterial toxins, including cytolethal distenting toxin, subunit B. Major factors associated with increased gut permeability, including gut dysbiosis and yeast overgrowth, use of NSAIDs and alcohol, food hypersensitivities (IgE-mediated), food intolerances (IgG-mediated), small bacterial overgrowth (SIBO), systemic inflammation, psychosocial stressors, some infections (e.g., HIV) and dietary patterns, should be assessed. Stool samples can be used to assay gut dysbiosis, gut inflammation and decreased mucosal defenses using assays of fecal growth of bacteria, yeast and fungi and stool assays of calprotectin, secretory IgA, β-defensin, α- antitrypsin, lysozyme and lactoferrin. Blood and breath tests should be used to exclude common causes of increased gut permeability, namely, food hypersensitivities and intolerances, SIBO, lactose intolerance and fructose malabsorption. <p> Discussion: Here, we propose strategies to recognize “leaky gut” in a clinical setting using the most adequate clinical chemistry and molecular immunology assays.
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Roorda, Andrew K., S. Gill Kanwar Rupinder, Terrie Mendelson, Daniel L. Peterson, Margaret Allen, and Kenneth F. Binmoeller. "Wireless Capsule Endoscopy in Patients with Leaky Gut Syndrome." Gastrointestinal Endoscopy 63, no. 5 (April 2006): AB172. http://dx.doi.org/10.1016/j.gie.2006.03.365.

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Dissertations / Theses on the topic "Leaky-gut syndrome"

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Ducloy, Cédric. "Le "Leaky-Gut" syndrome: bases physiopathologiques, éthiopathogénie, diagnostic et modalités de prise en charge." Antilles-Guyane, 2010. http://www.theses.fr/2010AGUY0321.

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L'intestin grêle possède une dualité fonctionnelle:d'une part il doit assurer l'absorption des nutriments essentiels à la survie de l' organisme; et d'autre part il joue le rôle de barrière contre la pénétration d'éléments nuisibles. Le Leaky-gut syndrome est définit par la rupture de la barrière physique assurée par les Tight-junctions secondaire à de multiples situations physiopathologiques. L' altération de cette fonction de barrière entraine l'augmentation de la perméabilité intestinale aux macromolécules, antigènes, microorganismes et est impliquée dans de nombreuses pathologies comme les allergies, les maladies inflammatoires intestinales, l'autisme. La prise en charge du LGS implique une bonne connaissance de la barrière intestinale assurée par l' épithélium,le microbiote et le système immunitaire et les résultats des thérapeutiques par la glutamine et les probiotiques sont prometteurs. Depuis quelques années l'intérêt de la communauté scientifique pour le LGS est grandissant,et la meilleure connaissance des processus physiopathologiques pourra probablement permettre une meilleure approche thérapeutique de pathologies dont le LGS est la pierre angulaire
The smail intestine has a dual function: first it must ensure absorption of nutrients essential to the survival of the organism, and secondly it acts as a barrier against penetration of harmful components. The Leaky-gut syndrome is sets by breaking the physical barrier provided by Tight junclions of secondary Multiple pathophysiological situations. The alteration of this barrier function leads the increase in intestinal permeability to macromolecules, antigens, microorganisms and is involved in many diseases such as allergies, inflammatory bowel disease, autism. The management of LGS implies good knowledge of the intestinal barrier provided by the epithelium, the microbiota and immune system and results of treatment by glutamine and probiotics are promising. Ln recent years the interest of the scientific community for LGS is growing, and better knowledge of pathophysiological processes may probably allow a better therapeutic approach to disorders whose LGS is the cornerstone
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Books on the topic "Leaky-gut syndrome"

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Is it leaky gut or leaky gut syndrome?: Clean gut, allergies, fatty liver, autoimmune diseases, fibromyalgia, multiple sclerosis, autism, psoriasis, diabetes, cancer, Parkinson's, thyroiditis, & more. Shreveport, La: Logos Enterprises LLC, 2014.

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One cause-- many ailments: The leaky gut syndrome : what it is and how it may be affecting your health. Virginia Beach, Va: A.R.E. Press, 2008.

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Leaky gut syndrome: What to do about a health threat that can cause arthrities, allergies and a host of other illnesses. New Canaan, Conn: Keats Pub., 1998.

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I Was Poisoned By My Body: The Odyssey of a Doctor Who Reversed Fibromyalgia, Leaky Gut Syndrome, and Multiple Chemical Sensitivity, Naturally! Lancaster, OH: Lucky Press, 2001.

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Invisible illnesses: Multiple chemical sensitivities, fibromyalgia, allergic/inflammatory arthritis, chronic fatigue, chemically induced immune system disorders, leaky gut/irritable bowel/colon disorders, prescription drug withdrawal syndrome. Topanga, Calif: Freedom Press, 2002.

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I was poisoned by my body--I have a gut feeling you could be, too!: The odyssey of a doctor who reversed fibromyalgia, leaky gut syndrome, multiple allergic responses, naturally and her life 10 years after recovery. 2nd ed. Lancaster, OH: Lucky Press, 2007.

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Publications, Healthful. No-Nonsense Guide to Leaky Gut Syndrome. Independently Published, 2017.

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A Simple Bone Broth Recipe to Heal Leaky Gut Syndrome. CreateSpace Independent Publishing Platform, 2017.

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Bush, Leah. Leaky Gut Syndrome Explained: A Syndrome That Is Highly Overlooked But Extremely Dangerous. Createspace Independent Publishing Platform, 2015.

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Naturopath, Case Adams. The Science of Leaky Gut Syndrome: Intestinal Permeability and Digestive Health. Logical Books, 2014.

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Book chapters on the topic "Leaky-gut syndrome"

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Lewis, Moira, Courtenay Norbury, Rhiannon Luyster, Lauren Schmitt, Andrea McDuffie, Eileen Haebig, Donna S. Murray, et al. "Leaky Gut Syndrome." In Encyclopedia of Autism Spectrum Disorders, 1706–12. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1698-3_29.

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Johnson, Kimberly. "Leaky Gut Syndrome." In Encyclopedia of Autism Spectrum Disorders, 2653–58. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-91280-6_29.

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Wallace, Daniel J., and Janice Brock Wallace. "Controversial Syndromes and Their Relationship to Fibromyalgia." In All About Fibromyalgia. Oxford University Press, 2002. http://dx.doi.org/10.1093/oso/9780195147537.003.0022.

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Over the years, a variety of health professionals have developed terms or phrases to denote seemingly unique clinical combinations of symptoms and signs. A disorder or syndrome does not necessarily exist simply because it has been described in the medical literature. Some have stood the test of time, others overlap with syndromes described by different specialists, and additional terms may be favored by a single practitioner advocating a “cause.” This chapter reviews conditions that have overlapping features with fibromyalgia but are not yet regarded as full-blown, legitimate disorders by organized medicine. When Dr. Fine first met Wanda, she was a basket case. Wanda had canceled three prior appointments because smells from a new carpet had made her sick, Med fly agricultural spraying 30 miles away prevented her from getting out of bed, and she developed a severe headache when her neighbors’ house was being painted. She almost passed out in the elevator going to Dr. Fine’s office because somebody was smoking. Wanda had been to three allergists, who obtained normal skin tests and blood tests. Desperate, she traveled to Mexico, where “immune rejuvenating” injections were administered, and to Texas, where a clinical ecologist sequestered her in a pollution-free, environmentally safe quonset hut for a month. There she received daily colonies, antiyeast medication, and vitamin shots, to no avail. Dr. Fine elicited a history of aching, sleep disorder, a “leaky gut,” muscle pains, fatigue, and a spastic colon. His physical examination and mental status examination revealed evidence of anxiety, obsessive-compulsive tendencies, and fibromyalgia tender points. Wanda was treated with fluoxetine (Prozac) for pain and obsessive behavior, buspirone (Buspar), for anxiety during the day, and trazodone (Desyrel), a tricyclic, to help her sleep at night. She was referred to a psychologist who worked to improve Wanda’s socialization skills and encouraged her to go out rather than be a prisoner in her own home. Wanda is slowly improving but will need many months of therapy. Self-reported environmental sensitivities are observed in 15 percent of Americans.
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Conference papers on the topic "Leaky-gut syndrome"

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Oliveira, Nathália Brígida de, Anna Clara Silva Fonseca, Lailla Luísa Silva Gomes, Aline Balducci Ferreira Dos Santos, and Beatriz Martins Borelli. "ANÁLISE DA RELAÇÃO ENTRE A SÍNDROME DE LEAKY GUT, A MICROBIOTA INTESTINAL E DOENÇAS AUTOIMUNES: UMA REVISÃO BIBLIOGRÁFICA." In I Congresso Nacional de Microbiologia Clínica On-Line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/1215.

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Introdução: Às células epiteliais do trato gastrointestinal participam da absorção de nutrientes e conferem proteção. Essa barreira é composta por junções estreitas, formando uma proteção física e conferindo a capacidade permeativa do intestino, e por uma camada de muco. Quando essas junções são agredidas por fatores imunológicos, citocinas ou patógenos, elas perdem a sua capacidade de selecionar o conteúdo intestinal e, assim, desenvolve a Síndrome do Intestino Permeável. Está disfunção se relaciona com inflamações sistêmicas e doenças autoimunes, o que ocorre, também pela alteração na composição da flora intestinal. Objetivos: Realizar revisão bibliográfica da literatura a respeito da correlação da Síndrome de Leaky gut e doenças autoimunes, bem como a importância da microbiota intestinal neste processo. Material e Métodos: A pesquisa foi realizada nas bases de dados SCIELO e PUBMED. Os descritores utilizados foram “autoimmune diseases”, “autoimmune disorders”, “gut microbiome”, “leaky gut” e “leaky gut syndrome”. Foram selecionados artigos acadêmicos originais, escritos na língua inglesa, que foram publicados entre 2014 e 2020. Tais artigos foram analisados de acordo com o ano de publicação, método de avaliação, objetivos e principais resultados. Resultados: A produção de moléculas pró-inflamatórias capazes de comprometer a integridade da barreira intestinal pode ser provocada por antígenos bacterianos intestinais. Esse quadro pode levar a síndrome de Leaky gut, o que explica a presença de bactérias intestinais no Sistema Imune, acarretando doenças autoimunes como: artrite reumatoide (AR), doença celíaca, Diabetes Mellitus tipo 1 (DM1). Na relação da AR e a permeabilidade intestinal, observou-se que indivíduos mais propensos a doenças tendem a ter um desequilíbrio da microbiota, gerando disbiose e contra regulação do sistema imune local e sistêmico à longo prazo e esse mesmo mecanismo ocorre com pacientes com AR. Conclusão: Considerando os dados obtidos, observou-se que há uma relação entre Leaky gut e a microbiota reduzida, como também a alteração funcional, tendo efeito na resposta imune principalmente em doenças autoimunes (AR, DM1 e doença celíaca). Percebe-se também que pela importância do tema há poucos estudos em humanos, o que demonstra a necessidade de mais pesquisas na área.
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Freire, Isabela Franco, and Silvia Fernandes Ribeiro da Silva. "RELAÇÃO DA SÍNDROME DO INTESTINO PERMEÁVEL NO DESENVOLVIMENTO DA ARTRITE REUMATÓIDE: REVISÃO DE LITERATURA." In I Congresso Brasileiro de Estudos Patológicos On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/conbesp/40.

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Introdução: A Síndrome do Intestino Permeável (SIP) consiste no aumento da permeabilidade do epitélio gastrointestinal, que possibilita a translocação bacteriana e de seus metabólitos para a corrente sanguínea. Essa transposição ocorre a partir de uma microbiota intestinal patológica (disbiose), que estimula a liberação de citocinas pró-inflamatórias (TNF-alfa, IL-6, IFN-gama) e ativação de linfócitos T, que podem estar envolvidos na etiopatôgenese e no agravamento da Artrite Reumatóide (AR). Objetivo: Avaliar a relação da SIP com o desenvolvimento da AR. Material e métodos: O presente resumo foi baseado em pesquisa bibliográfica feita na base de dados PubMed utilizando Leaky Gut Syndrome, Autoimmunity e Rheumatoid Arthritis como descritores. Foram incluídos 7 artigos publicados entre 2016 e 2021. Resultados: A SIP é uma condição presente em indivíduos com disbiose em decorrência da idade, dieta, sedentarismo, tabagismo e medicamentos. A descontinuidade da parede intestinal na SIP ocorre devido ao desbalanço das Junções Oclusivas Paracelulares (JOP), que mantêm o epitélio intestinal seletivamente permeável, devido à proteína Zonulina, que fosforila a proteína oclusiva ZO-1 da JOP. Estudos mostram que na SIP há diminuição de bactérias que contribuem para a integridade das JOPs e aumento de outras que induzem respostas inflamatórias com produção das citocinas pró-inflamatórias. Por outro lado, sabe-se que pacientes com AR apresentam diminuição da diversidade microbiana e aumento de algumas bactérias, tais como a Collinsella aerofaciens, que induzem a diminuição da expressão de ZO-1 no epitélio, alterando a permeabilidade intestinal. Além disso, pacientes com AR apresentam níveis elevados de marcadores de danos na barreira intestinal, tais como LPS de bactérias GRAM negativas, LBP, proteína produzida pelo epitélio em resposta a translocação de LPS e I-FABP, um biomarcador específico da integridade epitelial intestinal. Conclusão: A microbiota é um fator ambiental que desempenha um papel importante na progressão da AR. Porém, estudos são necessários para se compreender melhor os mecanismos da associação da SIP no desenvolvimento da AR.
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Meining, A., T. Malzacher, and D. Hose. "Nahrungsmittelallergen-getriggertes Leaky-gut-Syndrom - Versuch einer objektivierbaren Auswertung laser-endomikroskopischer Befunde." In Viszeralmedizin 2021 Gemeinsame Jahrestagung Deutsche Gesellschaft für Gastroenterologie, Verdauungs- und Stoffwechselkrankheiten (DGVS), Sektion Endoskopie der DGVS, Deutsche Gesellschaft für Allgemein und Viszeralchirurgie (DGAV). Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1734072.

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Meining, A., T. Malzacher, and D. Hose. "Nahrungsmittelallergen-getriggertes Leaky-gut-Syndrom - Versuch einer objektivierbaren Auswertung laser-endomikroskopischer Befunde." In Viszeralmedizin 2021 Gemeinsame Jahrestagung Deutsche Gesellschaft für Gastroenterologie, Verdauungs- und Stoffwechselkrankheiten (DGVS), Sektion Endoskopie der DGVS, Deutsche Gesellschaft für Allgemein und Viszeralchirurgie (DGAV). Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1734072.

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