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1

Lentiviral vector systems for gene transfer. Georgetown, TX: Eurekah.com, 2003.

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2

Trono, Didier, ed. Lentiviral Vectors. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-642-56114-6.

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3

Escors, David, Karine Breckpot, Frederick Arce, Grazyna Kochan, and Holly Stephenson. Lentiviral Vectors and Gene Therapy. Basel: Springer Basel, 2012. http://dx.doi.org/10.1007/978-3-0348-0402-8.

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Escors, David. Lentiviral Vectors and Gene Therapy. Basel: Springer Basel, 2012.

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5

Maurizio, Federico, ed. Lentivirus gene engineering protocols. Totowa, N.J: Humana Press, 2003.

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6

Federico, Maurizio, ed. Lentiviral Vectors and Exosomes as Gene and Protein Delivery Tools. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3753-0.

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7

Maurizio, Federico, ed. Lentivirus gene engineering protocols. 2nd ed. New York: Humana Press, 2010.

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8

Lentiviral Vector Systems for Gene Transfer (Medical Intelligence Unit, 31). Springer, 2003.

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9

Lentiviral Vector Systems for Gene Transfer (Medical Intelligence Unit, 31). Eurekah.com, 2001.

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10

Buchschacher, Gary L. Lentiviral Vector Systems for Gene Transfer (Medical Intelligence Unit, 31). Eurekah.com, 2001.

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11

Trono, Didier. Lentiviral Vectors. Springer, 2012.

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12

Trono, Didier. Lentiviral Vectors. Springer London, Limited, 2012.

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13

Lentiviral Vectors. Springer, 2002.

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14

Stephenson, Holly, David Escors, Karine Breckpot, Frederick Arce, and Grazyna Kochan. Lentiviral Vectors and Gene Therapy. Springer, 2012.

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15

Escors, David, Karine Breckpot, and Frederick Arce. Lentiviral Vectors and Gene Therapy. Springer, 2012.

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16

Federico, Maurizio. Lentiviral Vectors and Exosomes As Gene and Protein Delivery Tools. Springer New York, 2018.

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17

Federico, Maurizio. Lentiviral Vectors and Exosomes As Gene and Protein Delivery Tools. Springer New York, 2016.

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18

Lentivirus Gene Engineering Protocols. Humana Press, 2016.

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19

Jara Orellana,, Claudia. Efectos de la proteína Tau sobre la disfunción mitocondrial y el deterioro cognitivo en el envejecimiento. Universidad Autónoma de Chile, 2018. http://dx.doi.org/10.32457/20.500.12728/87452018dcbm6.

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Abstract:
El envejecimiento es un proceso complejo e irreversible que afecta el funcionamiento del cerebro y se considera el principal factor de riesgo para las patologías neurodegenerativas. Las mitocondrias son organelos fundamentales para la generación de energía, el equilibrio oxidativo y la regulación del calcio. Actualmente, se considera a la disfunción mitocondrial como un factor importante que contribuye al envejecimiento cerebral y a la patogénesis de diversas enfermedades neurodegenerativas. Este daño mitocondrial se ve reflejado en una disminución del metabolismo cerebral, aumentando el daño
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