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Relevant bibliographies by topics / Lentiviruses

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Academic literature on the topic 'Lentiviruses'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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Journal articles on the topic "Lentiviruses"

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Clements, J. E., and M. C. Zink. "Molecular biology and pathogenesis of animal lentivirus infections." Clinical Microbiology Reviews 9, no. 1 (1996): 100–117. http://dx.doi.org/10.1128/cmr.9.1.100.

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Lentiviruses are a subfamily of retroviruses that are characterized by long incubation periods between infection of the host and the manifestation of clinical disease. Human immunodeficiency virus type 1, the causative agent of AIDS, is the most widely studied lentivirus. However, the lentiviruses that infect sheep, goats, and horses were identified and studied prior to the emergence of human immunodeficiency virus type 1. These and other animal lentiviruses provide important systems in which to investigate the molecular pathogenesis of this family of viruses. This review will focus on two ani

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Lairmore, M. D., S. T. Butera, G. N. Callahan, and J. C. DeMartini. "Spontaneous interferon production by pulmonary leukocytes is associated with lentivirus-induced lymphoid interstitial pneumonia." Journal of Immunology 140, no. 3 (1988): 779–85. http://dx.doi.org/10.4049/jimmunol.140.3.779.

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Abstract Ovine lentiviruses share genome sequence, structural features, and replicative mechanisms with HIV, the etiologic agent of AIDS. A lamb model of lentivirus-induced lymphoid interstitial pneumonia, comparable to lymphoid interstitial pneumonia associated with pediatric AIDS, was used to investigate production of leukocyte-soluble mediators. Lentivirus-infected lambs and adult sheep with severe lymphoid interstitial pneumonia had significantly elevated levels of spontaneous interferon (IFN) production from pulmonary leukocytes compared with ovine lentiviruses-infected animals with mild

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Hötzel, Isidro, and William P. Cheevers. "Conservation of Human Immunodeficiency Virus Type 1 gp120 Inner-Domain Sequences in Lentivirus and Type A and B Retrovirus Envelope Surface Glycoproteins." Journal of Virology 75, no. 4 (2001): 2014–18. http://dx.doi.org/10.1128/jvi.75.4.2014-2018.2001.

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ABSTRACT We recently described a sequence similarity between the small ruminant lentivirus surface unit glycoprotein (SU) gp135 and the second conserved region (C2) of the primate lentivirus gp120 which indicates a structural similarity between gp135 and the inner proximal domain of the human immunodeficiency virus type 1 gp120 (I. Hötzel and W. P. Cheevers, Virus Res. 69:47–54, 2000). Here we found that the seven-amino-acid sequence of the gp120 strand β25 in the C5 region, which is also part of the inner proximal domain, was conserved in the SU of all lentiviruses in similar or identical po

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Bose, Deepanwita, Jean Gagnon, and Yahia Chebloune. "Comparative Analysis of Tat-Dependent and Tat-Deficient Natural Lentiviruses." Veterinary Sciences 2, no. 4 (2015): 293–348. https://doi.org/10.5281/zenodo.13530659.

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(Uploaded by Plazi for the Bat Literature Project) The emergence of human immunodeficiency virus (HIV) causing acquired immunodeficiency syndrome (AIDS) in infected humans has resulted in a global pandemic that has killed millions. HIV-1 and HIV-2 belong to the lentivirus genus of the Retroviridae family. This genus also includes viruses that infect other vertebrate animals, among them caprine arthritis-encephalitis virus (CAEV) and Maedi-Visna virus (MVV), the prototypes of a heterogeneous group of viruses known as small ruminant lentiviruses (SRLVs), affecting both goat and sheep worldwide.

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Bose, Deepanwita, Jean Gagnon, and Yahia Chebloune. "Comparative Analysis of Tat-Dependent and Tat-Deficient Natural Lentiviruses." Veterinary Sciences 2, no. 4 (2015): 293–348. https://doi.org/10.5281/zenodo.13530659.

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(Uploaded by Plazi for the Bat Literature Project) The emergence of human immunodeficiency virus (HIV) causing acquired immunodeficiency syndrome (AIDS) in infected humans has resulted in a global pandemic that has killed millions. HIV-1 and HIV-2 belong to the lentivirus genus of the Retroviridae family. This genus also includes viruses that infect other vertebrate animals, among them caprine arthritis-encephalitis virus (CAEV) and Maedi-Visna virus (MVV), the prototypes of a heterogeneous group of viruses known as small ruminant lentiviruses (SRLVs), affecting both goat and sheep worldwide.

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Courgnaud, Valérie, Xavier Pourrut, Frédéric Bibollet-Ruche, et al. "Characterization of a Novel Simian Immunodeficiency Virus from Guereza Colobus Monkeys (Colobus guereza) in Cameroon: a New Lineage in the Nonhuman Primate Lentivirus Family." Journal of Virology 75, no. 2 (2001): 857–66. http://dx.doi.org/10.1128/jvi.75.2.857-866.2001.

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ABSTRACT Exploration of the diversity among primate lentiviruses is necessary to elucidate the origins and evolution of immunodeficiency viruses. During a serological survey in Cameroon, we screened 25 wild-born guereza colobus monkeys (Colobus guereza) and identified 7 with HIV/SIV cross-reactive antibodies. In this study, we describe a novel lentivirus, named SIVcol, prevalent in guereza colobus monkeys. Genetic analysis revealed that SIVcol was very distinct from all other known SIV/HIV isolates, with average amino acid identities of 40% for Gag, 50% for Pol, 28% for Env, and around 25% for

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Chen, Jianbo, Douglas Powell, and Wei-Shau Hu. "High Frequency of Genetic Recombination Is a Common Feature of Primate Lentivirus Replication." Journal of Virology 80, no. 19 (2006): 9651–58. http://dx.doi.org/10.1128/jvi.00936-06.

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ABSTRACT Recent studies indicate that human immunodeficiency virus type 1 (HIV-1) recombines at exceedingly high rates, approximately 1 order of magnitude more frequently than simple gammaretroviruses such as murine leukemia virus and spleen necrosis virus. We hypothesize that this high frequency of genetic recombination is a common feature of primate lentiviruses. Alternatively, it is possible that HIV-1 is unique among primate lentiviruses in possessing high recombination rates. Among other primate lentiviruses, only the molecular mechanisms of HIV-2 replication have been extensively studied

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de Pablo-Maiso, Lorena, Ana Doménech, Irache Echeverría, et al. "Prospects in Innate Immune Responses as Potential Control Strategies against Non-Primate Lentiviruses." Viruses 10, no. 8 (2018): 435. http://dx.doi.org/10.3390/v10080435.

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Lentiviruses are infectious agents of a number of animal species, including sheep, goats, horses, monkeys, cows, and cats, in addition to humans. As in the human case, the host immune response fails to control the establishment of chronic persistent infection that finally leads to a specific disease development. Despite intensive research on the development of lentivirus vaccines, it is still not clear which immune responses can protect against infection. Viral mutations resulting in escape from T-cell or antibody-mediated responses are the basis of the immune failure to control the infection.

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St-Louis, Marie-Claude, Mihaela Cojocariu, and Denis Archambault. "The molecular biology of bovine immunodeficiency virus: a comparison with other lentiviruses." Animal Health Research Reviews 5, no. 2 (2004): 125–43. http://dx.doi.org/10.1079/ahr200496.

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AbstractBovine immunodeficiency virus (BIV) was first isolated in 1969 from a cow, R-29, with a wasting syndrome. The virus isolated induced the formation of syncytia in cell cultures and was structurally similar to maedi-visna virus. Twenty years later, it was demonstrated that the bovine R-29 isolate was indeed a lentivirus with striking similarity to the human immunodeficiency virus. Like other lentiviruses, BIV has a complex genomic structure characterized by the presence of several regulatory/accessory genes that encode proteins, some of which are involved in the regulation of virus gene

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Narayan, O., D. Sheffer, J. E. Clements, and G. Tennekoon. "Restricted replication of lentiviruses. Visna viruses induce a unique interferon during interaction between lymphocytes and infected macrophages." Journal of Experimental Medicine 162, no. 6 (1985): 1954–69. http://dx.doi.org/10.1084/jem.162.6.1954.

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Lentivirus infections are characterized by a persistent, restricted type of virus replication in tissues. Using sheep and goat lentiviruses, whose target cells in vivo are macrophages, we explored virus-host cell interactions to determine whether an interferon (IFN) is produced during virus replication in vivo which causes restricted replication. We show that the lentiviruses were incapable of inducing IFN directly in any infected cell, including macrophages and lymphocytes. However, after infection with these viruses, sheep and goat macrophages acquired a factor that triggered IFN production

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Dissertations / Theses on the topic "Lentiviruses"

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Robertson, David L. "Recombination in primate lentiviruses." Thesis, University of Nottingham, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.336866.

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Vödrös, Dalma. "Receptor use of primate lentiviruses /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-497-6/.

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Bailes, Elizabeth. "Origins and evolution of primate lentiviruses." Thesis, University of Nottingham, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.246384.

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Kelly, Maureen C. "Parallels in tRNA primer acquisition by lentiviruses." Thesis, Birmingham, Ala. : University of Alabama at Birmingham, 2007. https://www.mhsl.uab.edu/dt/2009r/kelly.pdf.

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Li, Li. "Short-term and long-term evolution of lentiviruses." Thesis, University of Nottingham, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.575475.

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Lentiviruses have paradoxically fast short-term rate of evolution and slow long-term rate of evolution, which differ by several orders of magnitude. In this thesis, with a new method called truncated tree analysis, slower rates of evolution of transmitted viruses were estimated. However, the rate decline of the transmitted viruses is limited, and is not sufficient to explain the dramatic difference between the short-term and long-term evolutionary rates. These dramatically different rates were reconciled by an S shaped curve based on the new trend observed from this thesis. In the middle part

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Broughton-Neiswanger, Liam E. "Maternal transmission is the major mode of ovine lentivirus transmission in a ewe flock a molecular epidemiology study /." Pullman, Wash. : Washington State University, 2010. http://www.dissertations.wsu.edu/Thesis/Spring2010/L_Broughton_042010.pdf.

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Thesis (M.S. in veterinary science)--Washington State University, May 2010.<br>Title from PDF title page (viewed on June 29, 2010). "College of Veterinary Medicine." Includes bibliographical references (p. 20-26).

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Stewart, Meredith Ellen. "An investigation into aspects of the replication of Jembrana disease virus /." Access via Murdoch University Digital Theses Project, 2005. http://wwwlib.murdoch.edu.au/adt/browse/view/adt-MU20051222.104106.

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Bichel, Katsiaryna. "Understanding post-entry pre-integration lentiviral biology." Thesis, University of Cambridge, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648287.

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Harris, Matthew E. "Analysis of post-transcriptional regulation of lentiviruses and mammalian hepadnaviruses /." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 1999. http://wwwlib.umi.com/cr/ucsd/fullcit?p9935471.

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Ditcham, William. "The development of recombinant vaccines against Jembrana disease." Thesis, Ditcham, William (2007) The development of recombinant vaccines against Jembrana disease. PhD thesis, Murdoch University, 2007. https://researchrepository.murdoch.edu.au/id/eprint/438/.

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Jembrana disease virus (JDV) is a lentivirus causing an acute infection with a 17% case fatality rate in Bali cattle in Indonesia. Control of the disease is currently achieved by identification of infected areas and restriction of cattle movement. A detergent-inactivated whole virus tissue-derived vaccine is sometimes employed in affected areas. This thesis reports initial attempts to produce genetically engineered vaccines to replace the inactivated tissue-derived vaccine, which as it is made from homogenised spleen of infected animals, is expensive to produce and could contain adventitio

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Books on the topic "Lentiviruses"

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Maurizio, Federico, ed. Lentivirus gene engineering protocols. Humana Press, 2003.

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missing], [name. Lentivirus gene engineering protocols. Humana Press, 2003.

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Maurizio, Federico, ed. Lentivirus gene engineering protocols. 2nd ed. Humana Press, 2010.

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E, Wilcox G., Soeharsono S, Dharma D. M. N, et al., eds. Jembrana disease and the bovine lentiviruses: Proceedings of a workshop 10-13 June 1996, Bali, Indonesia. Australian Centre for International Agricultural Research in association with Direktorat Jenderal Petermakan and the Bali Cattle Disease Investigation Unit, 1997.

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Federico, Maurizio, ed. Lentivirus Gene Engineering Protocols. Humana Press, 2003. http://dx.doi.org/10.1385/1592593933.

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Federico, Maurizio, ed. Lentivirus Gene Engineering Protocols. Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-533-0.

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Milne, Catherine E. Maedi visna: The disease, its potential impact on the UK sheep industry and a cost benefit appraisal ofcontrol strategies. SAC, 1993.

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1949-, Morrow John, and Haigwood Nancy L, eds. HIV molecular organization, pathogenicity, and treatment. Elsevier, 1993.

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1963-, Sobo Elisa Janine, ed. The endangered self: Managing the social risk of HIV. Routledge, 2000.

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Nichols, Eve K. Expanding access to investigational therapies for HIV infection and AIDS: March 12-13, 1990, conference summary. National Academy Press, 1991.

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Book chapters on the topic "Lentiviruses"

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Federico, Maurizio. "From Lentiviruses to Lentivirus Vectors." In Lentivirus Gene Engineering Protocols. Humana Press, 2003. http://dx.doi.org/10.1385/1-59259-393-3:3.

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Gonda, Matthew A. "The Lentiviruses of Cattle." In The Retroviridae. Springer US, 1994. http://dx.doi.org/10.1007/978-1-4899-1730-0_3.

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Déglon, N., and P. Aebischer. "Lentiviruses as Vectors for CNS Diseases." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-642-56114-6_10.

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Huso, David L., and Opendra Narayan. "Escape of Lentiviruses from Immune Surveillance." In Virus Variability, Epidemiology and Control. Springer US, 1990. http://dx.doi.org/10.1007/978-1-4757-9271-3_5.

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Lin, Yuan, Amar Desai, and Stanton L. Gerson. "Lentiviruses: Vectors for Cancer Gene Therapy." In Gene-Based Therapies for Cancer. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-6102-0_10.

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Narayan, Opendra, Mary C. Zink, Mark Gorrell, et al. "The Lentiviruses of Sheep and Goats." In The Retroviridae. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4899-1627-3_4.

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Kaur, Amitinder, Marie-Claire Gauduin, and R. Paul Johnson. "Immune Responses to Nonhuman Primate Lentiviruses." In Retroviral Immunology. Humana Press, 2001. https://doi.org/10.1007/978-1-59259-110-7_11.

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Pancino, G., H. Ellerbrok, M. Sitbon, and P. Sonigo. "Conserved Framework of Envelope Glycoproteins Among Lentiviruses." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-78536-8_5.

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Jeang, K. T., and A. Gatignol. "Comparison of Regulatory Features Among Primate Lentiviruses." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-78536-8_7.

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Gelderblom, Hans R., Preston A. Marx, Muhsin Özel, et al. "Morphogenesis, Maturation and Fine Structure of Lentiviruses." In Retroviral Proteases. Macmillan Education UK, 1990. http://dx.doi.org/10.1007/978-1-349-11907-3_17.

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Conference papers on the topic "Lentiviruses"

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Xiao, Yun-Feng. "Label-free Detection of Single Nanoparticles and Lentiviruses Using an Optical Microcavity." In Optical Sensors. OSA, 2013. http://dx.doi.org/10.1364/sensors.2013.st2b.3.

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Hoffman, Robert M., Hiroyuki Kishimoto, and Toshiyoshi Fujiwara. "Specific in vivo labeling with GFP retroviruses, lentiviruses, and adenoviruses for imaging." In Biomedical Optics (BiOS) 2008, edited by Alexander P. Savitsky, Robert E. Campbell, and Robert M. Hoffman. SPIE, 2008. http://dx.doi.org/10.1117/12.773308.

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Tapanes-Castillo, Alexis, Derek Dykxhoorn, Leana Ramos, Milagros Mulero, Deliabell Hernandez, and Vadym Trokhymchuk. "Culturing Human Neural Stem Cells and Quantifying Lentiviruses to Study Autism." In MOL2NET 2016, International Conference on Multidisciplinary Sciences, 2nd edition. MDPI, 2016. http://dx.doi.org/10.3390/mol2net-02-07008.

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Zhang, Dongming, Frederic Vigant, Qun He, et al. "Abstract 1511: Subcutaneous injection of total nucleated cells rapidly isolated following four-hour peripheral whole blood exposure to CD3-directed CAR-T lentiviruses with a synthetic driver results in robust CAR-T proliferation and anti-tumor immunity." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-1511.

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Tulio, Robertha, and Rômulo Machado Balmant. "Dental care for HIV positive patients - care and importance - case report." In II INTERNATIONAL SEVEN MULTIDISCIPLINARY CONGRESS. Seven Congress, 2023. http://dx.doi.org/10.56238/homeinternationalanais-087.

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Abstract Acquired immunodeficiency syndrome (AIDS) is caused by the "Lentivirus" family of retroviruses, called HIV-1. This syndrome is defined as an infectious disease of viral origin, with its manifestation interspersed in peaks and troughs, with a pathophysiology involving the compromising of the immune system, causing the defense system to not operate correctly, leaving the patient susceptible to the development of infections.

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Alton, EWFW, AC Boyd, JC Davies, et al. "S68 Towards a first-in-human trial with a pseudotyped lentivirus." In British Thoracic Society Winter Meeting, Wednesday 17 to Friday 19 February 2021, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2021. http://dx.doi.org/10.1136/thorax-2020-btsabstracts.73.

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Lund-Palau, H., C. Meng, A. Pilou, et al. "T2 Lentivirus GM-CSF gene therapy ameliorates autoimmune pulmonary alveolar proteinosis." In British Thoracic Society Winter Meeting 2018, QEII Centre, Broad Sanctuary, Westminster, London SW1P 3EE, 5 to 7 December 2018, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2018. http://dx.doi.org/10.1136/thorax-2018-212555.2.

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Burke, David, Kristine Drafahl, Clark Fjeld, Chad Galderisi, and Cindy Spittle. "Abstract 896: Enhanced sensitivity detection of replication competent lentivirus by qPCR." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-896.

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Wilson, AA, GJ Murphy, H. Hamakawa, et al. "Lentivirus-Based Expression of Human Alpha-1 Antitrypsin Ameliorates Emphysema in Mice." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3509.

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Marcotte, Richard, Azin Sayad, Maliha Haider, et al. "Abstract PR01: Functional characterization of breast cancer using pooled lentivirus shRNA screens." In Abstracts: AACR Precision Medicine Series: Synthetic Lethal Approaches to Cancer Vulnerabilities - May 17-20, 2013; Bellevue, WA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1535-7163.pms-pr01.

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Reports on the topic "Lentiviruses"

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Lindner, Daniel. Complementation of Myelodysplastic Syndrome Clones with Lentivirus Expression Libraries. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada566912.

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Lindner, Daniel J. Complementation of Myelodysplastic Syndrome Clones with Lentivirus Expression Libraries. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada581503.

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Lindner, Daniel. Complementation of Myelodysplastic Syndrome Clones with Lentivirus Expression Libraries. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada581646.

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DeMartini, James C., Abraham Yaniv, Jonathan O. Carlson, et al. Evaluation of Naked Proviral DNA as a Vaccine for Ovine Lentivirus Infection. United States Department of Agriculture, 1994. http://dx.doi.org/10.32747/1994.7570553.bard.

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Ovine lentivirus (OvLV) infection is widespread in sheep of the United States and Israel and is responsible for substantial economic losses. The primary goal of this project was to evaluate naked proviral DNA as a vaccine to induce protective immunity in sheep in endemic areas. Contrary to expectations, inoculation of sheep with proviral DNA derived from the full length OvLV molecular clone pkv72 did not result in detectable OvLV infection, but infectious virus was recovered from transfected ovine cells. Kv72 virus produced by these cells infected sheep and induced antibody responses, and was

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