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1

Maurizio, Federico, ed. Lentivirus gene engineering protocols. Humana Press, 2003.

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missing], [name. Lentivirus gene engineering protocols. Humana Press, 2003.

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3

Maurizio, Federico, ed. Lentivirus gene engineering protocols. 2nd ed. Humana Press, 2010.

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4

E, Wilcox G., Soeharsono S, Dharma D. M. N, et al., eds. Jembrana disease and the bovine lentiviruses: Proceedings of a workshop 10-13 June 1996, Bali, Indonesia. Australian Centre for International Agricultural Research in association with Direktorat Jenderal Petermakan and the Bali Cattle Disease Investigation Unit, 1997.

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Federico, Maurizio, ed. Lentivirus Gene Engineering Protocols. Humana Press, 2003. http://dx.doi.org/10.1385/1592593933.

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Federico, Maurizio, ed. Lentivirus Gene Engineering Protocols. Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-533-0.

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7

Milne, Catherine E. Maedi visna: The disease, its potential impact on the UK sheep industry and a cost benefit appraisal ofcontrol strategies. SAC, 1993.

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8

1949-, Morrow John, and Haigwood Nancy L, eds. HIV molecular organization, pathogenicity, and treatment. Elsevier, 1993.

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9

1963-, Sobo Elisa Janine, ed. The endangered self: Managing the social risk of HIV. Routledge, 2000.

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10

Nichols, Eve K. Expanding access to investigational therapies for HIV infection and AIDS: March 12-13, 1990, conference summary. National Academy Press, 1991.

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11

M, Hardy Leslie, and Institute of Medicine (U.S.). Committee on Prenatal and Newborn Screening for HIV Infection., eds. HIV screening of pregnant women and newborns. National Academy Press, 1991.

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12

Lentiviral Vectors. Springer, 2002.

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13

Lentiviruses and macrophages: Molecular and cellular interactions. Caister Academic Press, 2010.

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14

(Editor), G. E. Wilcox, S. Soeharsono (Editor), D. M. N Dharma (Editor), and J. W. Copland (Editor), eds. Jembrana Disease and the Bovine Lentiviruses (ACIAR Proceedings). Australian Centre for International Agricultural Research, 1997.

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15

Lentivirus Gene Engineering Protocols. Humana Press, 2010.

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16

Lentivirus Gene Engineering Protocols. Humana Press, 2003.

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17

Lentivirus Gene Engineering Protocols. Humana Press, 2016.

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18

Lentivirus Gene Engineering Protocols. Humana Press, 2003.

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19

Wilkerson, Melinda Joann. Immunopathology of lentivirus induced arthritis. 1994.

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20

Lentiviral Vector Systems for Gene Transfer (Medical Intelligence Unit, 31). Eurekah.com, 2001.

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21

Buchschacher, Gary L. Lentiviral Vector Systems for Gene Transfer (Medical Intelligence Unit, 31). Eurekah.com, 2001.

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22

Lentiviral vector systems for gene transfer. Eurekah.com, 2003.

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23

Phillips, M. Ian. Gene Therapy Methods. Elsevier Science & Technology Books, 2002.

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24

Letvin, Norman L. Simian Immunodeficiency Virus (Current Topics in Microbiology and Immunology). Springer-Verlag Telos, 1994.

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25

Bailey, Steven Nelson. Using novel lentivirus-infected cell microarrays and chemical genetics to study cancer cell metabolism. 2007.

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26

Lichtensteiger, Carol A. Antibody neutralization and MHC-restricted lymphocyte-mediated cytotoxicity of variants of caprine arthritis-encephalitis lentivirus. 1992.

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27

Livingston, Schuyler, Benjamin Young, Martin Markowitz, Poonam Mathur, and Bruce L. Gilliam. HIV Virology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0017.

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HIV is a member of the lentivirus subfamily of retroviruses. Two distinct groups of viruses are pathogenic in humans: HIV-1 and HIV-2. Both are transmitted sexually and known to cause immunodeficiency disease. HIV enters the cell through use of the CD4 receptor and chemokine co-receptors, primarily CCR5 and CXCR4. The viral genome is transcribed from RNA to DNA by reverse transcriptase and integrated into the host genome by integrase. The HIV genome encodes 15 proteins, comprising three categories: structural, regulatory, and accessory. After budding from the host cell, the virus matures into
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28

Avances en la deteccion de lentivirus de peque?os rumiantes. En leche de reba?os ovinos y caprinos. Editorial Academica Espa?ola, 2017.

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29

Hullinger, Gordon A. A gp135 cellular receptor mediates infection of caprine synovial membrane cells by the lentivirus caprine arthritis-encephalitis virus. 1993.

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30

Jara Orellana,, Claudia. Efectos de la proteína Tau sobre la disfunción mitocondrial y el deterioro cognitivo en el envejecimiento. Universidad Autónoma de Chile, 2018. http://dx.doi.org/10.32457/20.500.12728/87452018dcbm6.

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El envejecimiento es un proceso complejo e irreversible que afecta el funcionamiento del cerebro y se considera el principal factor de riesgo para las patologías neurodegenerativas. Las mitocondrias son organelos fundamentales para la generación de energía, el equilibrio oxidativo y la regulación del calcio. Actualmente, se considera a la disfunción mitocondrial como un factor importante que contribuye al envejecimiento cerebral y a la patogénesis de diversas enfermedades neurodegenerativas. Este daño mitocondrial se ve reflejado en una disminución del metabolismo cerebral, aumentando el daño
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31

Sex, Drugs, and HIV/AIDS in Brazil. Westview Press, 2000.

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32

Green, Gill, and Elisa Sobo. Endangered Self: Identity and Social Risk. Taylor & Francis Group, 2002.

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33

Green, Gill. The Endangered Self: Identity and Social Risk. Routledge, 2000.

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34

Green, Gill, and Elisa Sobo. Endangered Self: Identity and Social Risk. Taylor & Francis Group, 2002.

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35

Green, Gill. The Endangered Self: Identity and Social Risk. Routledge, 2000.

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36

Green, Gill, and Elisa Sobo. Endangered Self: Identity and Social Risk. Taylor & Francis Group, 2002.

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37

Endangered Self: Managing the Social Risk of HIV. Routledge, 2000.

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38

Medicine, Institute of, and Roundtable for the Development of Drugs and Vaccines Against AIDS. Expanding Access to Investigational Therapies for HIV Infection and AIDS. National Academies Press, 1991.

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39

Expanding Access to Investigational Therapies for HIV Infection and AIDS. National Academies Press, 1991.

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40

HIV Screening of Pregnant Women And Newborns. Natl Academy Pr, 1990.

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