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Relevant bibliographies by topics / Lepra. Diagnose

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Academic literature on the topic 'Lepra. Diagnose'

Author: Grafiati

Published: 4 June 2021

Last updated: 13 February 2022

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Journal articles on the topic "Lepra. Diagnose"

1

Chopra, Ajay, Debdeep Mitra, and Barnali Mitra. "812. Case–Control Trial to Evaluate the Cytokine Response to the Use of Capsule Thalidomide in Erythema Nodosum Leprosum in Leprosy Patients." Open Forum Infectious Diseases 5, suppl_1 (2018): S290—S291. http://dx.doi.org/10.1093/ofid/ofy210.819.

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Abstract Background Leprosy is a chronic granulomatous disease caused by Mycobacterium leprae. Type II lepra reaction or Erythema Nodosum Leposum is a Type III hypersentivity immune response during the chronic course of the illness. This immune response presents as systemic symptoms and neutrophilic leukocytosis, similar to sepsis. Capsule Thalidomide is considered the drug of choice, when it comes to the treatment of this acute immunological emergency. A rational study into the immunological markers involved in the pathogenesis of erythema nododsum leprosum and its successful suppression by T

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2

Fischer, Marcellus. "Lepra - Ein Überblick über Klinik, Diagnose und Therapie." JDDG: Journal der Deutschen Dermatologischen Gesellschaft 15, no. 8 (2017): 801–29. http://dx.doi.org/10.1111/ddg.13301_g.

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Besmens, Inga Swantje, Andreas Schiller, Christoph Erling, and Maurizio Calcagni. "Wenn die Handgelenkskontusion zur Lepra Diagnose führt – ein Fallbericht." Handchirurgie · Mikrochirurgie · Plastische Chirurgie 52, no. 03 (2020): 213–15. http://dx.doi.org/10.1055/a-1170-6025.

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Mitra, Debdeep. "811. A Randomized Controlled Trial of Prednisolone vs. Interleukin 17 A Inhibitor Secuinumab in the Management of Type 1 Lepra Reaction in Leprosy Patients." Open Forum Infectious Diseases 5, suppl_1 (2018): S290. http://dx.doi.org/10.1093/ofid/ofy210.818.

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Abstract Background Leprosy is a chronic granulomatous infectious disease caused by Mycobacterium leprae. Type 1 lepra reactions (T1R) are delayed hypersensitivity (Type IV) reactions which if not treated promptly leads to disability affecting eyes, hands and feet. IL-17 A which is produced mainly by inflammatory T helper 17 cells is up regulated in patients of Lepra reaction. Conventionally oral corticosteroids steroids have been the main stay in the management of Type 1 lepra reactions. This novel biologic drug is a targeted therapy which blocks the offending interleukin molecule without any

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Himstedt, Verena, Nicole Dorn, Ulrich Göggerle, Thomas Rieker, and Stephanie Krug. "Feline Lepra bei einem 5 Monate alten Kater in Deutschland." Tierärztliche Praxis Ausgabe K: Kleintiere / Heimtiere 46, no. 04 (2018): 271–75. http://dx.doi.org/10.15654/tpk-170642.

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ZusammenfassungEin 5 Monate alter, 4 kg schwerer Europäisch-Kurzhaar-Kater wurde mit ulzerierten kutanen Knoten vorgestellt, die in mehreren Arealen über den gesamten Körper verteilt waren. Die serologische Untersuchung auf das feline Immunschwächevirus und das feline Leukämievirus ergab negative Befunde. Anhand des histologischen Nachweises von säurefesten Stäbchen in drei Hautbioptaten wurde die Diagnose Mykobakteriose der Haut gestellt. Mithilfe der Polymerase-Kettenreaktion ließ sich Mycobacterium lepraemurium identifizieren. Der Kater wurde nach der operativen Entfernung aller Hautknoten

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6

Maladan, Yustinus, C. S. Whinie Lestari, Ratna Tanjung, Vatim Dwi Cahyani, and Muhammad Fajri Rokhmad. "Molecular Detection Mutation of rpoB Gene Mycobacterium leprae in Relapse and Default of Leprosy Patient in Jayapura City, Papua." Health Science Journal of Indonesia 9, no. 1 (2018): 31–36. http://dx.doi.org/10.22435/hsji.v9i1.462.

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Abstrak Latar belakang: Lepra masih menjadi masalah kesehatan di Papua terutama di Kota Jayapura.Banyaknya kasus relaps dan default juga menjadi tantangan dalam eliminasi lepra di Jayapura. Kasusrelaps dan riwayat default pada beberapa penelitian berkaitan dengan resistensi terhadap multi drugtreatment (MDT). Tujuan penelitian ini adalah mendeteksi keberadaan mutasi gen rpoB M. leprae padapasien relaps, default dan pasein yang kurang peka terhadap terapi MDT di Kota Jayapura. Metode: Sampel diperoleh dari pasien yang terdiagnosis lepra dengan kriteria pasien relaps, default danpasi

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7

Kaur, Jasleen, Jyotika Kalsy, and Riya Kaur Kalra. "Infections and reactions in leprosy: a diagnostic dilemma." International Journal of Research in Dermatology 4, no. 1 (2018): 96. http://dx.doi.org/10.18203/issn.2455-4529.intjresdermatol20180147.

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<p>Leprosy (Hansen’s disease) is a chronic infectious disease caused by <em>Mycobacterium leprae</em>. It continues to be a public health problem in India, which contributes about 60% to the world leprosy burden. Leprosy patients when on treatment can develop either lepra reactions or reactions due to antileprosy drugs, also they can develop other infections endemic in their areas during the course of their disease. We are presenting such two cases where in one case patient on treatment with multibacillary multidrug therapy (MBMDT) developed fever, lympadenopathy and other sy

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8

Agarwal, Archana M., Jay L. Patel, Adam Clayton, and Noel Scott Reading. "Use of Next Generation Sequencing Panel for Routine Diagnosis of Hereditary Hemolytic Anemias." Blood 132, Supplement 1 (2018): 2325. http://dx.doi.org/10.1182/blood-2018-99-112589.

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Abstract Hereditary hemolytic anemia (HHA) are a heterogeneous group of disorders due to germline mutations of the red cell cytoskeleton (e.g. hereditary spherocytosis (HS) and hereditary elliptocytosis/pyropoikilocytosis (HE/HPP)) or enzyme deficiencies (e.g. glucose 6 phosphate dehydrogenase deficiency (G6PD) and pyruvate kinase deficiency (PKD). Routine morphological and biochemical analysis may be inconclusive in neonates due to the physiological nature of erythroid cell maturation and can also be misleading in transfusion-dependent patients. Additionally, there has been increasing awarene

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Misra, Durga Prasanna, Jyoti Ranjan Parida, Abhra Chandra Chowdhury, et al. "Lepra Reaction with Lucio Phenomenon Mimicking Cutaneous Vasculitis." Case Reports in Immunology 2014 (2014): 1–4. http://dx.doi.org/10.1155/2014/641989.

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Leprosy is a disease typically found in the tropics. Patients with leprosy can have varying presentation with constitutional symptoms, joint pains, skin nodules, and rarely a vasculitis-like picture with skin ulcers and neuropathy. We present a young lady who presented with the rare manifestation of skin infarcts mimicking cutaneous vasculitis, diagnosed on histopathology to have Lucio phenomenon on a background of lepromatous leprosy. With increasing migration and widespread use of biologic response modifiers, clinicians all over the world need to be aware of various presentations of leprosy

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10

G., V. "Bacterioscopic diagnosis of leprosy." Kazan medical journal 20, no. 9 (2021): 995. http://dx.doi.org/10.17816/kazmj77094.

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Rosentul and Kruglyak (Rus. Vestn. Dormat., 1924, No. 9) applied bacterioscopic examination of the lymphatic glands according to Grenbaum and Schamberg in 38 cases of leprosy (12 cases. Leprae tuberosae, 6 - leprae mixtae and 20 - 1. maculo -anaestheticae).

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Dissertations / Theses on the topic "Lepra. Diagnose"

1

Bührer-Sékula, Samira. "A simple dipstick assay for leprosy development, evaluation and application /." [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2000. http://dare.uva.nl/document/55198.

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Reibel, Florence. "Mycobacterium leprae : de l’épidémiologie à la caractérisation de l’ADN gyrase." Electronic Thesis or Diss., Sorbonne université, 2018. http://www.theses.fr/2018SORUS560.

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Deux cent mille cas de lèpre surviennent chaque année dans le monde, surtout dans les pays en voie de développement. Le laboratoire d’accueil ayant une double activité de surveillance de la lèpre et de recherche centrée sur l’étude des mécanismes de résistance aux antituberculeux, les objectifs du travail s’inscrivent dans des enjeux médicaux et fondamentaux. Un 1er travail de génotypage de M. leprae nous a permis de montrer le caractère acquis dans des territoires hors France métropolitaine des cas de lèpre diagnostiqués sur le territoire national. L’équipement enzymatique de M. leprae est re

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Souza, Marilena Maria de. "Rastreamento da hanseníase e infecção do Mycobacterium leprae utilizando os antígenos recombinantes LID - 1 e PADL." reponame:Repositório Institucional da UFBA, 2013. http://www.repositorio.ufba.br/ri/handle/ri/13100.

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Bezerra, Janaina Miranda. "Desempenho de antígenos nativo, recombinante e sintético, em testes imunoenzimáticos, para diagnóstico e prognóstico de pacientes com diferentes formas clínicas de hanseníase." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/5/5134/tde-22012013-175521/.

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Apesar da Organização Mundial de Saúde (OMS) ter adotado medidas de controle, a hanseníase permanece como um problema social e de saúde pública em muitos países. O diagnóstico clínico e a baciloscopia permanecem como as principais ferramentas utilizadas para a classificação dos pacientes e definição da terapêutica a ser instituída. A identificação de novos antígenos de M. leprae, o desenvolvimento de novos métodos laboratoriais e melhorar o desempenho de testes sorológicos já existentes são prioridades para tentar atingir as metas da OMS programadas para o período de 2011-2015. Neste estudo av

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Fernandes, Victor Hugo Damasceno. "Reação em cadeia da polimerase em amostras de linfa coletadas em papel filtro no diagnóstico da hanseníase." Universidade de São Paulo, 2018. http://www.teses.usp.br/teses/disponiveis/17/17138/tde-13092018-140429/.

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O diagnóstico etiológico da hanseníase, causada pelo bacilo Mycobacterium leprae, pode ser difícil na forma paucibacilar (PB), na qual os exames têm baixa sensibilidade na detecção do bacilo. A reação em cadeia da polimerase (PCR) tem se mostrado sensível para o diagnóstico da hanseníase. Amostras de linfa são rapidamente obtidas em campo e pouco invasivas. O objetivo principal deste trabalho foi comparar os resultados da PCR com 4 pares de primers (Ag85B, MntH, Pra-36kDa e RLEP), descritos na literatura para o diagnóstico da hanseníase, utilizando-se DNA extraído de amostras de linfa de orelh

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6

Medeiros, Mildred Ferreira. "Hanseníase neural, aspectos diagnósticos da forma neural pura e mecanismos imunopatogênicos da lesão do nervo na doença. Participação de quimiocinas CCL2 e CXCL10 e metaloproteinases 2 e 9." Universidade do Estado do Rio de Janeiro, 2014. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=7356.

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Coordenação de Aperfeiçoamento de Pessoal de Nível Superior<br>O diagnóstico da hanseníase neural pura baseia-se em dados clínicos e laboratoriais do paciente, incluindo a histopatologia de espécimes de biópsia de nervo e detecção de DNA de Mycobacterium leprae (M. leprae) pelo PCR. Como o exame histopatológico e a técnica PCR podem não ser suficientes para confirmar o diagnóstico, a imunomarcação de lipoarabinomanana (LAM) e/ou Glicolipídio fenólico 1 (PGL1) - componentes de parede celular de M. leprae foi utilizada na primeira etapa deste estudo, na tentativa de detectar qualquer presença

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Books on the topic "Lepra. Diagnose"

1

Vries, René R. P. de., ed. Recognition of M. leprae antigens. Nijhoff, 1987.

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2

World Health Organization. Working Group on Rapid Diagnostic Methods for M. leprae Infection. Report: Meeting of the Working Group on Rapid Diagnostic Methods for M. leprae Infection. The Office, 1986.

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3

World Health Organization. Scientific Working Group on the Development of Rapid Diagnostic Methods for M. leprae Infection. Meeting of the Scientific Working Group on the Development of Rapid Diagnostic Methods for M. leprae Infection: Tokyo, Japan, 18-21 June 1985. The Office, 1986.

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4

International, Neonatal Screening Symposium (8th 1991 Leura N. S. W. ). Neonatal screening in the nineties: Proceedings of the 8th International Neonatal Screening Symposium, Leura, New South Wales, Australia, 12th-15th November, 1991, and the Arthur Veale Memorial Meeting, Auckland, New Zealand, 5th-9th November, 1991. The Symposium, 1991.

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5

Marcato, Paolo Stefano. Patologia del coniglio e della lepre: Atlante a colori e compendio = pathology of the rabbit and hare : a color atlas and compendium. Esculapio, 1986.

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6

Matthews, Philippa C. Infections caused by mycobacteria. Edited by Philippa C. Matthews. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198737773.003.0003.

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This chapter consists of short notes, diagrams, and tables to summarize infections caused by Mycobacterium tuberculosis (MTB) complex and Mycobacterium leprae. For ease of reference, each topic is broken down into sections, including classification, epidemiology, microbiology, pathophysiology, clinical syndromes, diagnosis, treatment, and prevention.

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7

Abdulkader, Rita, and Richard A. Watts. Mycobacterial diseases. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0103.

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The main diseases caused by mycobacterial infection are tuberculosis (TB) and leprosy. Despite a fall in the prevalence of these diseases over the last decade, they are still significant causes of morbidity and mortality worldwide. Atypical mycobacterial infections are encountered less frequently. Immigration patterns, the frequency of human immunodeficiency infection, and the increased numbers of patients on immunosuppressive treatments render mycobacterial infections relevant not only to physicians in the developing world where they traditionally occurred but also in the developed world. Ske

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Book chapters on the topic "Lepra. Diagnose"

1

Schliack, H. "Die Diagnose der Lepra aus neurologischer Sicht Nonne-Gedächtnisvorlesung in Darmstadt 1990." In Multiple Sklerose Neuroonkologie Konstitutionelle Dyslexie. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-84478-2_3.

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Roberts, Charlotte A. "The Bioarchaeology of Leprosy." In Leprosy. University Press of Florida, 2020. http://dx.doi.org/10.5744/florida/9781683401841.003.0005.

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This chapter explores the bone changes in the skeleton related to leprosy (paleopathology). Diagnosing leprosy in skeletons ideally requires a complete well-preserved skeleton. The facial, hand, and foot bones are affected, but only a few percent of untreated people will develop bone lesions, and the type of leprosy depends on the resistance of their immune system to M. leprae. Most skeletons diagnosed will display lesions due to the low-resistant form: lepromatous leprosy. Damage to the peripheral nerves by M. leprae is responsible for the alterations to the hand and foot bones. The bone changes of the skull represent the direct effects of M. leprae being inhaled into the mouth and nose. Beyond visual/macroscopic analysis, imaging and microscopy have been used for diagnosis. Biomolecular analysis is the main advance in analytical methods (mainly focusing on ancient DNA). Sequencing of the modern M. leprae and M. lepromatosis genomes have provided data for comparative analyses and testable hypotheses regarding the origin, evolution, and history of leprosy. These data are enabling paleogeneticists, historians, and bioarchaeologists to re-evaluate the long history of leprosy in relation to historical accounts of the drivers for the migration of people with leprosy across the globe.

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Roberts, Charlotte A. "Past and Present Diagnosis, Treatment, and Prognosis." In Leprosy. University Press of Florida, 2020. http://dx.doi.org/10.5744/florida/9781683401841.003.0004.

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This chapter considers diagnosis of leprosy both today and in the past. Recently, molecular methods have contributed to diagnosis, detecting drug resistant M. leprae strains and bacterial strain-specific markers, assessing exposure to M. leprae, and tracing transmission patterns. Treatment of leprosy is focused on drug therapy, but a holistic approach is needed from both medical and social perspectives. Since the 1940s drug therapy has contributed to leprosy’s decline, free for over twenty years. However, access can be a challenge. Effective vaccines for preventing leprosy need development. Equally important is the prevention and treatment of damage to the hands and feet due to loss of skin sensation. Historically, diagnosis of leprosy in the past could be done by inexperienced people and often seemed inappropriate. However, rational tests are described, such as looking at urine and blood, and focusing on the skin lesions. Treatments were varied, such as bathing, dressing skin lesions, cautery of body parts, and herbal remedies. The most widespread “treatment” was segregation into leprosy hospitals (leprosaria). It is unclear how many people in the past were segregated in this way, compared to the number more readily accepted within their communities.

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4

Abdulkader, Rita, and Richard A. Watts. "Mycobacterial diseases." In Oxford Textbook of Rheumatology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0103_update_001.

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The main diseases caused by mycobacterial infection are tuberculosis (TB) and leprosy. Despite a fall in the prevalence of these diseases over the last decade, they are still significant causes of morbidity and mortality worldwide. Atypical mycobacterial infections are encountered less frequently. Immigration patterns, the frequency of human immunodeficiency infection, and the increased numbers of patients on immunosuppressive treatments render mycobacterial infections relevant not only to physicians in the developing world where they traditionally occurred but also in the developed world. Skeletal TB occurs in 1–3% of cases of TB infection, and is more frequently encountered in the immunocompromised. A high index of suspicion is required, diagnosis relies on a combination of clinical features and radiological, histological, and microbiological tests. Multidrug regimens are required for treatment with surgery in selected cases. Leprosy is caused by M. leprae infection. The disease is still a leading cause of disability worldwide. Diagnosis is usually clinical. The course of the disease is indolent but may be interrupted by acute inflammatory reactions, which contribute to nerve damage and disability. Treatment aims at eliminating the mycobacteria using multidrug regimens, and management of complications including leprosy reactions and long-term nerve damage. Atypical mycobacterial infections affecting bone and joints are uncommon; they usually follow direct inoculation of the pathogen. Haematogenous dissemination is encountered in immunocompromised patients. These microorganisms are not usually susceptible to the same drug regimens used in the treatment of tuberculosis.

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Conference papers on the topic "Lepra. Diagnose"

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Gomez-Bañuelos, E., E. Chavarria-Avila, K. Arrona-Rios, et al. "AB0083 Single nucleotide polymorphisms in lep – 2548 g>a and lepr + 668 a>g in rheumatoid arthritis mexican mestizos are associated with age at diagnosis, disease activity and anti-ccp antibodies." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.6729.

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