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Relevant bibliographies by topics / Leukemia and Lymphoma

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Academic literature on the topic 'Leukemia and Lymphoma'

Author: Grafiati

Published: 5 June 2025

Last updated: 2 August 2025

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Journal articles on the topic "Leukemia and Lymphoma"

1

Edlefsen, Kerstin Lara, Anneclaire De Roos, and Andrea LaCroix. "Application of the InterLymph Consortium’s Proposed Classification of Lymphoid Neoplasms for Epidemiologic Research to a Large, Nationwide Health Study: Experience in the Women’s Health Initiative." Blood 112, no. 11 (2008): 4669. http://dx.doi.org/10.1182/blood.v112.11.4669.4669.

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Abstract Large cohort studies, such as the Women’s Health Initiative, have traditionally grouped hematopoietic neoplasms (HPN) into disease categories including multiple myeloma (MM), non-Hodgkin lymphoma (NHL), Hodgkin lymphoma (HL), and leukemia, however this may not allow for optimal distinction of biologically relevant associations. The 2001 World Health Organization (WHO) classification of HPN represents the current gold standard classification scheme, and has been incorporated into the International Classification of Disease for Oncology, Third Edition (ICD-O-3). In 2007, the Internation

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Wang, Wei, Magdalena Czader, and Sa A. Wang. "Blood- and bone marrow–based mature T-cell and natural killer cell leukemias and lymphomas: a summary in the series of the 2023 SH/EAHP Workshop." American Journal of Clinical Pathology 164, no. 1 (2025): 7–25. https://doi.org/10.1093/ajcp/aqaf009.

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Abstract This session included 51 cases submitted to the workshop “Progress in T- and NK-cell Lymphomas and Leukemias” by the Society for Hematopathology and European Association for Haematopathology under “Blood/Bone Marrow–Based Mature T- and NK-Cell Leukemias/Lymphomas” or “T/NK-cell neoplasms with a Leukemic Presentation.” Entities encompassed T-cell prolymphocytic leukemia, T-cell large granular lymphocytic leukemia (LGLL), natural killer (NK)-LGLL/chronic lymphoproliferative disorder of NK cells, adult T-cell leukemia/lymphoma, aggressive NK-cell leukemia, and their mimics. Submitted cas

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Rodig, Scott J., Jeremy S. Abramson, Geraldine S. Pinkus, Steven P. Treon, Margaret A. Shipp, and Jeffery L. Kutok. "Evaluation of CD52 Expression in Hematopoietic Neoplasms by Standard Immunohistochemistry: Implications for the Expanded Use of Alemtuzumab (CAMPATH-1H) in the Treatment of Hematological Malignancies." Blood 106, no. 11 (2005): 3346. http://dx.doi.org/10.1182/blood.v106.11.3346.3346.

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Abstract CD52 is a GPI-linked glycoprotein expressed by B cells, T cells, monocytes and macrophages. The humanized monoclonal antibody alemtuzumab (CAMPATH-1H) is specific for CD52 and is FDA-approved for the treatment of relapsed or refractory chronic lymphocytic leukemia (CLL). The utility of alemtuzumab in the treatment of other lymphoid and non-lymphoid malignancies has been recently explored; however, a comprehensive survey of CD52 expression among the various classes of hematopoietic neoplasms has not been completed. In addition, most methods of detecting CD52 rely on flow cytometric tec

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Shi, Yang, and Endi Wang. "Blastic Plasmacytoid Dendritic Cell Neoplasm: A Clinicopathologic Review." Archives of Pathology & Laboratory Medicine 138, no. 4 (2014): 564–69. http://dx.doi.org/10.5858/arpa.2013-0101-rs.

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Blastic plasmacytoid dendritic cell neoplasm is a rare entity grouped with the acute myeloid leukemia–related precursor neoplasms in the 2008 World Health Organization classification. It was previously postulated to originate from natural killer cells, T cells, or monocytes but is now believed to arise from the plasmacytoid dendritic cell. The pathogenesis of blastic plasmacytoid dendritic cell neoplasm is not well understood, although the neoplasm demonstrates frequent deletion of tumor suppressor genes, including RB1, CDKN1B, CDKN2A, and TP53. Blastic plasmacytoid dendritic cell neoplasm is

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Dunphy, Cherie H. "Gene Expression Profiling Data in Lymphoma and Leukemia: Review of the Literature and Extrapolation of Pertinent Clinical Applications." Archives of Pathology & Laboratory Medicine 130, no. 4 (2006): 483–520. http://dx.doi.org/10.5858/2006-130-483-gepdil.

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Abstract Context.—Gene expression (GE) analyses using microarrays have become an important part of biomedical and clinical research in hematolymphoid malignancies. However, the methods are time-consuming and costly for routine clinical practice. Objectives.—To review the literature regarding GE data that may provide important information regarding pathogenesis and that may be extrapolated for use in diagnosing and prognosticating lymphomas and leukemias; to present GE findings in Hodgkin and non-Hodgkin lymphomas, acute leukemias, and chronic myeloid leukemia in detail; and to summarize the pr

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Pals, S. T., M. Zijstra, T. Radaszkiewicz, et al. "Immunologic induction of malignant lymphoma: graft-vs-host reaction-induced B cell lymphomas contain integrations of predominantly ecotropic murine leukemia proviruses." Journal of Immunology 136, no. 1 (1986): 331–39. http://dx.doi.org/10.4049/jimmunol.136.1.331.

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Abstract The induction of a graft-vs-host reaction in (BALB/c X A)F1 mice by i.v. injection with BALB/c lymphoid cells leads to a lymphoid hyperplasia that may progress to malignant lymphoma. In the present paper, the following aspects of graft-vs-host-reaction lymphomagenesis were studied: 1) the cellular requirements for the induction of lymphomas, 2) their cellular origin, and 3) the role of murine leukemia viruses. The development of graft-vs-host-reaction lymphomas was found to be mediated by donor T cells and to require the presence of histoincompatibility between donor and host. Histolo

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Rateesh, Sareen, Garima Agarwal, and Gajendra Nath Gupta. "Lymphoglandular bodies as useful morphological clue in diagnosis of Lymphoid malignancies- A Case Report." International Journal of Clinicopathological Correlation 8, no. 1 (2024): 12–16. http://dx.doi.org/10.56501/intjclinicopatholcorrel.v8i1.1036.

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Lymphoglandular bodies, observed as round basophilic cytoplasmic fragments on Giemsa stain, are linked with lymphoid malignancies, aiding in distinguishing lymphomas from other small round cell tumors. This case report underscores the diagnostic significance of Lymphoglandular bodies in Acute lymphoid leukemia diagnosis through bone marrow biopsy. We present a case of a 21-year-old male with chest pain and weakness. The bone marrow biopsy revealed a monomorphic cell population with a high nuclear cytoplasmic ratio, prompting immunomarker analysis that confirmed the presence of blasts as lympho

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Longe, Harold, Douglas V. Faller, and Gerald V. Denis. "Telomere-Based Pre-Clinical Therapy in a Murine Model of Non-Hodgkin’s Lymphoma of the Diffuse Large B Cell (DLCL)Type." Blood 106, no. 11 (2005): 607. http://dx.doi.org/10.1182/blood.v106.11.607.607.

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Abstract The dual bromodomain Brd2 is closely related to the basal transcription factor TAFII250, which is essential for cyclin A transactivation and mammalian cell cycle progression. Constitutive expression of BRD2 (under Eμ control) in the lymphoid lineage of transgenic mice elevates basal transcription of cyclin A, destabilizes the cell cycle and leads to B cell leukemias and lymphomas that are monoclonal, morphologically uniform, transplantable and highly malignant. The surface immunophenotype of the lymphoma cells is: B220+, CD19+, sIgM+, CD5+, CD9+; B7-1 and B7-2 elevated, CD23low; CD11b

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Taylor, Justin, Wenbin Xiao, and Omar Abdel-Wahab. "Diagnosis and classification of hematologic malignancies on the basis of genetics." Blood 130, no. 4 (2017): 410–23. http://dx.doi.org/10.1182/blood-2017-02-734541.

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Abstract Genomic analysis has greatly influenced the diagnosis and clinical management of patients affected by diverse forms of hematologic malignancies. Here, we review how genetic alterations define subclasses of patients with acute leukemias, myelodysplastic syndromes (MDS), myeloproliferative neoplasms (MPNs), non-Hodgkin lymphomas, and classical Hodgkin lymphoma. These include new subtypes of acute myeloid leukemia defined by mutations in RUNX1 or BCR-ABL1 translocations as well as a constellation of somatic structural DNA alterations in acute lymphoblastic leukemia. Among patients with M

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Ginsberg, AM, M. Raffeld, and J. Cossman. "Inactivation of the retinoblastoma gene in human lymphoid neoplasms." Blood 77, no. 4 (1991): 833–40. http://dx.doi.org/10.1182/blood.v77.4.833.833.

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Abstract The absence of wild type retinoblastoma (Rb) gene expression in a wide variety of human solid tumors suggests an etiologic role for this tumor suppressor gene in human cancer. We have evaluated the involvement of Rb gene inactivation in the pathogenesis and progression of human lymphoma and leukemia. We examined the genomic configuration and transcription of the Rb gene in cultured cell lines and primary cases of T- and B-cell lymphomas and leukemias. By Southern analysis, abnormalities of the Rb locus were identified in 1 of 5 T-cell acute lymphoblastic lymphoma (T-ALL) cell lines, 1

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Dissertations / Theses on the topic "Leukemia and Lymphoma"

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Kwok, Suet-kei Gladys. "The effectiveness of a chemotherapy educational programme (CEP) for Leukaemia and Lymphoma patients." Click to view the E-thesis via HKUTO, 2004. http://sunzi.lib.hku.hk/hkuto/record/B31972937.

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黃傑煇 and Kit-fai Wong. "CD56-positive: natural killer cell lymphoma/leukaemia." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hub.hku.hk/bib/B3198177X.

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Wong, Kit-fai. "CD56-positive natural killer cell lymphoma/leukaemia /." Hong Kong : University of Hong Kong, 2001. http://sunzi.lib.hku.hk/hkuto/record.jsp?B23736197.

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Cho, Candice. "Factors affecting stem cell transplantation for leukemia and lymphoma." CONNECT TO ELECTRONIC THESIS, 2006. http://hdl.handle.net/1961/3595.

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Adamson, Penelope Jane. "Engineering antibodies for use in leukemia and lymphoma therapy /." Title page, contents and summary only, 2000. http://web4.library.adelaide.edu.au/theses/09PH/09pha221.pdf.

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Lundin, Jeanette. "Targeted CD52 therapy in lymphoid malignancies : a clinical and immunological study /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-441-0/.

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Johansson, Ann-Sofie. "Establishment and characterization of a murine T-cell lymphoma/leukemia model." Doctoral thesis, Umeå universitet, Institutionen för strålningsvetenskaper, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-35195.

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Mouse models of human disease are valuable tools for studying pathogenesis and for evaluating novel therapies. T-cell lymphoma is a relatively rare disease in humans, affecting 100-150 persons yearly in Sweden. It exists in both aggressive and more indolent forms. We have established a mouse model for an aggressive T-cell lymphoma, the T-cell lymphoma/leukemia (TLL) mouse. In the present thesis, the TLL mouse model was characterized and used for experimental therapeutic and primary prevention studies. The TLL mouse was established unintentionally in our laboratory during work on VH-gene replac

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Thörn, Ingrid. "Minimal Residual Disease Assessment in Childhood Acute Lymphoblastic Leukemia." Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-101028.

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Halldórsdóttir, Anna Margrét. "Genetic and Epigenetic Profiling of Mantle Cell Lymphoma and Chronic Lymphocytic Leukemia." Doctoral thesis, Uppsala universitet, Hematologi och immunologi, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-156786.

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Mantle cell lymphoma (MCL) and chronic lymphocytic leukemia (CLL) both belong to the group of mature B-cell malignancies. However, MCL is typically clinically aggressive while the clinical course of CLL varies. CLL can be divided into prognostic subgroups based on IGHV mutational status and into multiple subsets based on closely homologous (stereotyped) B-cell receptors. In paper I we investigated 31 MCL cases using high-density 250K single-nucleotide polymorphism arrays and gene expression arrays. Although most copy-number aberrations (CNAs) were previously reported in MCL, a novel deletion w

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Kohart, Nicole Ann Kohart. "Models, Mechanisms, and Treatment of Adult T-cell Leukemia/Lymphoma Bone Metastasis." The Ohio State University, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=osu1503248777003095.

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Books on the topic "Leukemia and Lymphoma"

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Kaspers, G. J. L., 1963-, ed. Innovative leukemia and lymphoma therapy. Informa Healthcare, 2008.

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Zipf, Theodore F., and Dennis A. Johnston. Leukemia and Lymphoma. Humana Press, 2002. http://dx.doi.org/10.1385/1592593186.

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Zipf, Theodore F., and Dennis A. Johnston, eds. Leukemia and Lymphoma. Humana Press, 2003. https://doi.org/10.1007/978-1-59259-318-7.

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A, Scheinberg David, and Jurcic Joseph G, eds. Treatment of leukemia and lymphoma. Elsevier, 2004.

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Mughal, Tariq I. Understanding leukemias, lymphomas, and myelomas. Taylor & Francis, 2006.

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Leukemia Research Fund International Workshop (1984 Oxford). Epidemiology of leukemia and lymphoma. Edited by Greaves M. F and Chan L. C. Pergamon, 1985.

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Watanabe, Toshiki, and Takuya Fukushima, eds. Adult T-cell Leukemia/Lymphoma. Springer Japan, 2017. http://dx.doi.org/10.1007/978-4-431-56523-9.

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F, Zipf Theodore, and Johnston Dennis A, eds. Leukemia and lymphoma: Detection of minimal residual disease. Humana Press, 2003.

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Leong, Anthony S.-Y. 1945-, ed. Essential oncology of the lymphocyte. Springer-Verlag, 1987.

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Kaspers, G. J. L., 1963-, Pieters R, Veerman A. J. P, and International Symposium on Drug Resistance in Leukemia and Lymphoma (3rd : 1998 : Amsterdam, Netherlands), eds. Drug resistance in leukemia and lymphoma III. Kluwer Academic/Plenum Publishers, 1999.

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Book chapters on the topic "Leukemia and Lymphoma"

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Montserrat, Emili. "Small Lymphocytic Lymphoma/Chronic Lymphocytic Leukemia." In Lymphoma. Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-408-1_4.

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Cross, Nicholas C. P., and Andreas Hochhaus. "Minimal Residual Disease in Chronic Myelogenous Leukemia." In Leukemia and Lymphoma. Humana Press, 2003. https://doi.org/10.1007/978-1-59259-318-7_11.

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Zipf, Theodore F., and Dennis A. Johnston. "The Application of Minimal Residual Disease Detection Methodology to the Clinical Decision-Making Process." In Leukemia and Lymphoma. Humana Press, 2003. https://doi.org/10.1007/978-1-59259-318-7_5.

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Ratei, Richard, and Wolf-Dieter Ludwig. "Flow-Cytometry Methods for the Detection of Residual Leukemia." In Leukemia and Lymphoma. Humana Press, 2003. https://doi.org/10.1007/978-1-59259-318-7_1.

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Neale, Geoffrey. "PCR Methods for the Detection of Minimal Residual Disease in Childhood Acute Lymphoblastic Leukemia." In Leukemia and Lymphoma. Humana Press, 2003. https://doi.org/10.1007/978-1-59259-318-7_3.

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Johnston, Dennis A., and Theodore F. Zipf. "Statistical Considerations in the Analysis of Minimal Residual Disease." In Leukemia and Lymphoma. Humana Press, 2003. https://doi.org/10.1007/978-1-59259-318-7_4.

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Coco, Francesco Lo, and Daniela Diverio. "Minimal Residual Disease in Acute Promyelocytic Leukemia." In Leukemia and Lymphoma. Humana Press, 2003. https://doi.org/10.1007/978-1-59259-318-7_10.

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Campana, Dario. "Flow-Cytometry—Based Studies of Minimal Residual Disease in Children with Acute Lymphoblastic Leukemia." In Leukemia and Lymphoma. Humana Press, 2003. https://doi.org/10.1007/978-1-59259-318-7_2.

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Moppett, John, Amos Burke, Christopher Knechtli, Anthony Oakhill, Colin Steward, and Nicholas J. Goulden. "Measurement of Minimal Residual Disease in Children Undergoing Allogeneic Stem Cell Transplant for Acute Lymphoblastic Leukemia." In Leukemia and Lymphoma. Humana Press, 2003. https://doi.org/10.1007/978-1-59259-318-7_6.

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Lee, Ming-Sheng, and Fernando Cabanillas. "Monitoring Follicular Lymphoma by Polymerase Chain Reaction." In Leukemia and Lymphoma. Humana Press, 2003. https://doi.org/10.1007/978-1-59259-318-7_14.

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Conference papers on the topic "Leukemia and Lymphoma"

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Babrah, Jaspreet, Keith P. McCarthy, Richard Lush, Adam D. Rye, Conrad Bessant, and Nicholas Stone. "FT-infrared spectroscopic studies of lymphoma, lymphoid, and myeloid leukemia cell lines." In European Conference on Biomedical Optics, edited by Dietrich Schweitzer and Maryann Fitzmaurice. SPIE, 2007. http://dx.doi.org/10.1117/12.728220.

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Klekawka, T., A. Moryl-Bujakowska, K. Smalisz, et al. "Nodular Lymphocyte Predominant Hodgkin Lymphoma - experience of Polish Pediatric Leukemia/Lymphoma Study Group." In ISCAYAHL 2020. © Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1701854.

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Zaouk, Salimar Al, Tala Matar, Mohamad Abou Ali, Abdallah Kassem, and Lara Hamawy. "Classification of Leukemia and Lymphoma using Faster R-CNN." In 2022 International Conference on Microelectronics (ICM). IEEE, 2022. http://dx.doi.org/10.1109/icm56065.2022.10005371.

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Dražić, Branka, Mirjana Antonijević Nikolić, Vojislav Stanić, Vesna Stojiljković, and Slađana Tanasković. "CYTOTOXIC ACTIVITY OF AMINOCARBOXYLATEMACROCYCLIC Cu(II) COMPLEXES." In 17th International Conference on Fundamental and Applied Aspects of Physical Chemistry. Society of Physical Chemists of Serbia, 2024. https://doi.org/10.46793/phys.chem24ii.577d.

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Five binuclear complexes with general formula [Cu2(L)tpmc](ClO4)4, L= valine (1), norvaline (2) leucine (3), norleucine (4) and isoleucine (5) were tested in vitro against human tumor cell lines: THP-1 (human acute monocytic leukemic cell line), Jurkat (human acute T cell leukemia) and Ramos (B cells, Burkitt’s lymphoma cell line). The values of IC50 were calculated. All tested complexes showed significant activity against the tested lines. Complex 2 has promoted significant decreases in the metabolic activity of all three cell lines, especially according to Ramos (IC50 value is 40.21 ± 1.45 µ

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Kanda, Hiroaki, Kimie Nomura, Toshihiko Iizuka, Mutsunori Fujiwara, Yuichi Ishikawa, and Toshiro Migita. "Abstract 1637: ATP-citrate lyase transgenic mice frequently develop lymphoma/leukemia spontaneously." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-1637.

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Vo, Thanh-Trang, Jong-Hoon Scott Lee, Lomon So, Brandon Beagle, Matthew R. Janes, and David A. Fruman. "Abstract IA16: Mechanisms of resistance to mTOR inhibitors in leukemia and lymphoma." In Abstracts: AACR Special Conference: Targeting the PI3K-mTOR Network in Cancer; September 14-17, 2014; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-8514.pi3k14-ia16.

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Thor, Danielle C., Rohan Umrani, Jack Bergal, Charles Yang, and Richard Gordon. "Diagnosis and Management of a Patient with Chronic Lymphocytic Leukemia and a Concurrent Plasmacytoma: An Overview of a Case Report." In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. http://dx.doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.32_2024.

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Chronic lymphocytic leukemia (CLL) typically presents as an indolent disease with a benign disposition in most patients. In select patients, CLL can progress into a more aggressive disease via its original morphology, following a Richter transformation to an alternative non-Hodgkin lymphoma, or with the concomitant development of multiple myeloma. In an extremely rare subset of individuals with CLL, an extramedullary plasmacytoma may coexist. This presentation seeks to describe the diagnosis and treatment of a patient with concurrent CLL and a plasmacytoma.

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Klekawka, T., A. Moryl-Bujakowska, K. Smalisz, et al. "Brentuximab vedotin (BV) in recurrent and refractory Hodgkin Lymphoma (HL) in children. Experience of Polish Pediatric Leukemia/Lymphoma Study Group." In ISCAYAHL 2020. © Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1701899.

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Garnica, Taismara, Jéssika Lesbon, Arina Rochetti, et al. "Abstract PO-21: Investigating the role of exosomes derived from chemotherapy-resistant leukemia cells as mediators of cellular plasticity." In Abstracts: AACR Virtual Meeting: Advances in Malignant Lymphoma; August 17-19, 2020. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/2643-3249.lymphoma20-po-21.

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Williams, Christopher K. "Abstract 5484: Childhood leukemia and lymphoma: African experience supports a role for environmental factors." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-5484.

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Reports on the topic "Leukemia and Lymphoma"

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Cooper, Laurence, and Rita Young. Development of Augmented Leukemia/Lymphoma-Specific T-Cell Immunotherapy for Deployment with Haploidentical, Hematompoietic Progenitor-Cell Transplant. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada487262.

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Cooper, Laurence. Development of Augmented Leukemia/Lymphoma-Specific T-Cell Immunotherapy for Deployment with Haploidentical, Hematompoietic Progenitor-Cell Transplant. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada560655.

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Iqbal, Fatiha. Chimeric Antigen Receptor (CAR) T Cell Immunotherapies for Leukemias and Lymphomas. Iowa State University, 2019. http://dx.doi.org/10.31274/cc-20240624-354.

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Garland, Cedric F., Margaret A. Ryan, Edward D. Gorham, E. K. Gunderson, and Tyler Smith. Incidence Rates of Lymphoid Leukemia in Children of Active-Duty Navy Servicemembers. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada419667.

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Jordan, Craig T. Properties of Leukemia Stem Cells in a Novel Model of Cml Progression to Lymphoid Blast Crisis. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada484102.

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Jordan, Craig T. Properties of Leukemia Stem Cells in a Novel Model of CML Progression to Lymphoid Blast Crisis. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada471560.

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Gorham, Edward D., Frank Groves, Cedric F. Garland, and Frank C. Garland. Incidence of Lymphoid and Myeloid Leukemia in US Active-Duty Military Men According to Occupation and Comparisons with Incidence from the US Surveillance Epidemiology and End Results (SEER) Populations. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada419383.

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