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Relevant bibliographies by topics / Lipid deposition

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Academic literature on the topic 'Lipid deposition'

Author: Grafiati

Published: 4 June 2021

Last updated: 3 February 2022

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Journal articles on the topic "Lipid deposition"

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Bou, Marta, Xinxia Wang, Marijana Todorčević, Tone-Kari Knutsdatter Østbye, Jacob Torgersen, and Bente Ruyter. "Lipid Deposition and Mobilisation in Atlantic Salmon Adipocytes." International Journal of Molecular Sciences 21, no. 7 (2020): 2332. http://dx.doi.org/10.3390/ijms21072332.

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The present study aimed to elucidate how Atlantic salmon adipocytes pre-enriched with palmitic (16:0, PA), oleic (18:1n−9, OA), or eicosapentaenoic (20:5n−3, EPA) acid respond to a fasting condition mimicked by nutrient deprivation and glucagon. All experimental groups were supplemented with radiolabeled PA to trace secreted lipids and distribution of radioactivity in different lipid classes. There was a higher content of intracellular lipid droplets in adipocytes pre-enriched with OA than in adipocytes pre-enriched with PA or EPA. In the EPA group, the radiolabeled PA was mainly esterified in

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Crispin, Sheila. "Ocular lipid deposition and hyperlipoproteinaemia." Progress in Retinal and Eye Research 21, no. 2 (2002): 169–224. http://dx.doi.org/10.1016/s1350-9462(02)00004-6.

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Guyton, J. R., K. F. Klemp, B. L. Black, and T. M. A. Bocan. "Extracellular lipid deposition in atherosclerosis." European Heart Journal 11, suppl E (1990): 20–28. http://dx.doi.org/10.1093/eurheartj/11.suppl_e.20.

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Crispin, S. M. "Lipid deposition at the limbus." Eye 3, no. 2 (1989): 240–50. http://dx.doi.org/10.1038/eye.1989.33.

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Funai, Katsuhiko, and Clay F. Semenkovich. "Skeletal muscle lipid flux: running water carries no poison." American Journal of Physiology-Endocrinology and Metabolism 301, no. 2 (2011): E245—E251. http://dx.doi.org/10.1152/ajpendo.00152.2011.

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Lipids are the most abundant organic constituents in many humans. The rise in obesity prevalence has prompted a need for a more refined understanding of the effects of lipid molecules on cell physiology. In skeletal muscle, deposition of lipids can be associated with insulin resistance that contributes to the development of diabetes. Here, we review the evidence that muscle cells are equipped with the molecular machinery to convert and sequester lipid molecules, thus rendering them harmless. Induction of mitochondrial and lipogenic flux in the setting of elevated lipid deposition can protect m

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Tavares, G. D., M. C. de Oliveira, J. M. C. Vilela, and M. S. Andrade. "Deposition of Lipid Bilayers with Atomic Force Microscopy." Microscopy and Microanalysis 11, S03 (2005): 44–47. http://dx.doi.org/10.1017/s1431927605050853.

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Biological membranes are constituted of lipids organized as a two dimensional bilayer supporting peripheral and integral proteins, providing a barrier between the inside and the outside of a cell [1]. Similar membranes can be prepared from the lipid mixtures forming liposomes. The liposomes are multi or unilamellar spherical vesicles in which an aqueous volume is enclosed and can be used to encapsulate some drugs [2]. In order to better expose the details of their structure, these membranes are generally deposited on the surface of a flat substrate. These supported planar lipid membranes can a

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Zheng, Li-yuan, Hui Xue, Hua Yuan, Shu-xiang Liu, and Xiu-ying Zhang. "Efficacy of management for obstruction caused by precipitated medication or lipids in central venous access devices: A systematic review and meta-analysis." Journal of Vascular Access 20, no. 6 (2019): 583–91. http://dx.doi.org/10.1177/1129729819836846.

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At present, central venous access devices (CVADs) are widely used in clinical practice. The reasons for CVAD obstruction caused by precipitated medication or lipids are increasingly complex. However, there is no clear treatment program for CVAD obstruction caused by precipitated medication or lipids. The target of this study was to analyze data regarding obstruction caused by precipitated medication or lipids in CVADs and to calculate the efficacy of different treatment methods. A systematic review with meta-analysis was conducted in alignment with the Preferred Reporting Items for Systematic

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Rattenbury, J. M., C. J. Taylor, and S. Ganapathy. "LIPID DEPOSITION IN PARENTERAL INFUSION LINES." Lancet 331, no. 8587 (1988): 701. http://dx.doi.org/10.1016/s0140-6736(88)91493-6.

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Wu, Zhuanchang, Hongxin Ma, Liyuan Wang, et al. "Tumor suppressor ZHX2 inhibits NAFLD–HCC progression via blocking LPL-mediated lipid uptake." Cell Death & Differentiation 27, no. 5 (2019): 1693–708. http://dx.doi.org/10.1038/s41418-019-0453-z.

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AbstractNon-alcoholic fatty liver disease (NAFLD) leads to hepatocellular carcinoma (HCC). However, the underlying mechanism remains largely unclear. Here, we investigated the role of the tumor suppressor Zinc fingers and homeoboxes 2 (ZHX2) in the progression of NAFLD to HCC. ZHX2 expression was significantly decreased in fatty liver tissues, especially in the liver with NAFLD–HCC. ZHX2 overexpression disturbed lipid homeostasis of cultured HCC cells, and inhibited lipid deposition in hepatocytes both in vitro and in vivo. Moreover, ZHX2 inhibited uptake of exogenous lipids through transcript

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Feng, Lei, Chengwu Gu, Yanxia Li та Jiasui Huang. "High Glucose Promotes CD36 Expression by Upregulating Peroxisome Proliferator-Activated Receptor γ Levels to Exacerbate Lipid Deposition in Renal Tubular Cells". BioMed Research International 2017 (2017): 1–10. http://dx.doi.org/10.1155/2017/1414070.

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Diabetic kidney disease (DKD) appears to be closely related to lipid deposition in kidney. The aim of this study was to determine whether high glucose (HG) exacerbated lipid deposition by increasing CD36 expression via AKT-PPARγ signaling pathway. Our results showed that HG activated AKT signaling pathway, followed by an increase in PPARγ that induced CD36 overexpression, ultimately causing lipid deposition in HK-2 cells. We also found that inhibition of AKT-PPARγ signaling pathway or knockdown of CD36 could reduce HG-induced lipid accumulation in HK-2 cells. These results indicated that AKT-P

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Dissertations / Theses on the topic "Lipid deposition"

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Shilton, Catherine Margaret. "Corneal lipid deposition in anurans." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0020/NQ55639.pdf.

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Lorentz, Holly. "Lipid Deposition on Hydrogel Contact Lenses." Thesis, University of Waterloo, 2006. http://hdl.handle.net/10012/2963.

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The primary objective of this study was to quantify and characterise lipid deposition on soft (hydrogel) contact lenses, particularly those containing siloxane components. Studies involving a variety of <em>in vitro</em> doping and <em>in vivo</em> worn contact lenses were undertaken, in which lipid deposition was analyzed by either TLC or HPLC. Specific experiments were completed to optimize a method to extract the lipid from the lens materials, to compare the total lipid deposition on nine different hydrogel lenses and to analyze the effect that lipid deposition had on wettability. A m

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Silva, Amélia Cláudia Figueiredo. "Nutritional Regulation of Lipid Deposition in Blackspot Seabream (Pagellus bogaraveo)." Tese, Instituto de Ciências Biomédicas Abel Salazar, 2008. http://hdl.handle.net/10216/19972.

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Silva, Amélia Cláudia Figueiredo. "Nutritional Regulation of Lipid Deposition in Blackspot Seabream (Pagellus bogaraveo)." Doctoral thesis, Instituto de Ciências Biomédicas Abel Salazar, 2008. http://hdl.handle.net/10216/19972.

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DiStefano, Marina T. "A Role for the Lipid Droplet Protein HIG2 in Promoting Lipid Deposition in Liver and Adipose Tissue: A Dissertation." eScholarship@UMMS, 2016. http://escholarship.umassmed.edu/gsbs_diss/830.

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Chronic exposure of humans or rodents to high calorie diets leads to hypertriglyceridemia and ectopic lipid deposition throughout the body, resulting in metabolic disease. Cellular lipids are stored in organelles termed lipid droplets (LDs) that are regulated by tissue-specific LD proteins. These proteins are critical for lipid homeostasis, as humans with LD protein mutations manifest metabolic dysfunction. Identification of novel components of the LD machinery could shed light on human disease mechanisms and suggest potential therapeutics for Type 2 Diabetes. Microarray analyses pinpointed th

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DeBono, Allan. "The role and behavior of Arabidopsis thaliana lipid transfer proteins during cuticular wax deposition." Thesis, University of British Columbia, 2011. http://hdl.handle.net/2429/39381.

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The primary aerial surfaces of terrestrial plants are coated with a protective hydrophobic layer comprising insoluble and soluble lipids. The lipids are known collectively as cuticular wax. To generate the waxy cuticle during elongative growth, plants dedicate half of the fatty acid metabolism of their epidermal cells. It is unknown how cuticular wax is exported from the plasma membrane into the cell wall, and eventually, to the cuticle at the cell surface. I hypothesized that lipid transfer proteins (LTPs) were responsible for plasma membrane to cell wall transport of cuticular lipids. Using

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Lewis, Kristen Oblad. "COMPARATIVE LIPIDOMICS OF HYDROGEL CONTACT LENSES IN-VITRO AND IN-VIVO." The Ohio State University, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=osu1243889772.

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Castellana, Edward Thomas. "Investigation of the photocatalytic lithographic deposition of metals in sealed microfluidic devices on TiO2 surfaces." [College Station, Tex. : Texas A&M University, 2005. http://hdl.handle.net/1969.1/ETD-TAMU-1009.

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Levelt, Eylem. "The role of rest and stress cardiac energy metabolism and ectopic lipid deposition in diabetic cardiomyopathy." Thesis, University of Oxford, 2015. https://ora.ox.ac.uk/objects/uuid:54ffde14-5e1c-4484-83e5-6e6fbe784df0.

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Type 2 diabetes mellitus (T2DM) is associated with an increased risk of heart failure and cardiovascular mortality even in the absence of coronary artery disease. Although the reasons for this are not clear, impaired cardiac high energy phosphate metabolism, coronary microvascular dysfunction and ectopic lipid deposition have emerged among the candidate mechanisms. Cardiac magnetic resonance imaging (CMR) and magnetic resonance spectroscopy (MRS) are powerful tools to characterise these conditions. The findings here suggest that, in diabetes, coronary microvascular dysfunction may potentially

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Yamashita, Yui. "Brain-specific natriuretic peptide receptor-B deletion attenuates high-fat diet-induced visceral and hepatic lipid deposition in mice." Kyoto University, 2016. http://hdl.handle.net/2433/217139.

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Books on the topic "Lipid deposition"

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McCaffrey, Mark A. Sedimentary lipids as indicators of depositional conditions in the coastal Peruvian upwelling regime. 1990.

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Book chapters on the topic "Lipid deposition"

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Teira, E. "Catalyzed Reporter Deposition- Fluorescence In Situ Hybridization (CARD-FISH) and Abundance of Cycloclasticus." In Handbook of Hydrocarbon and Lipid Microbiology. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-77587-4_318.

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Small, Donald M. "Evolution of the Atherosclerotic Plaque—A Physico—chemical Approach to Lipid Deposition." In Pathobiology of the Human Atherosclerotic Plaque. Springer New York, 1990. http://dx.doi.org/10.1007/978-1-4612-3326-8_11.

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Jin, X., and H. S. Kruth. "4. Macrophage deposition of extracellular lipid particles with microdomains of ordered cholesterol arrays." In Handbook of cholesterol. Wageningen Academic Publishers, 2016. http://dx.doi.org/10.3920/978-90-8686-821-6_4.

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Bell, David R., Hani N. Sabbah, and Paul D. Stein. "Areas of High Shear Show Sparing of Lipid Deposition in Hypercholesterolemic Rabbits with Constricted Aortas." In Biofluid Mechanics. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-52338-0_28.

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Rosenfeld, Michael E. "Lipid Deposition and Oxidation in the Evolution of the Atherosclerotic Lesion: Lessons Learned from Hypercholesterolemic Animal Models." In Medical Science Symposia Series. Springer Netherlands, 1998. http://dx.doi.org/10.1007/978-94-011-5022-4_15.

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Rongioletti, Franco. "Lipoid Proteinosis." In Clinical and Pathological Aspects of Skin Diseases in Endocrine, Metabolic, Nutritional and Deposition Disease. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-60761-181-3_19.

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Walton, K. W. "Non-Arterial Deposition of Lipids and Lipoproteins: Significance in Relation to Atherogenesis." In Expanding Horizons in Atherosclerosis Research. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71753-6_5.

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Puu, G., I. Gustafson, P. Å. Ohlsson, G. Olofsson, and Å. Sellström. "Deposition of Biological Lipids on Solid Planar Substrates by Langmuir Blodgett Technique." In Progress in Membrane Biotechnology. Birkhäuser Basel, 1991. http://dx.doi.org/10.1007/978-3-0348-7454-0_20.

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Berzina, Tatiana S., Vladimir I. Troitsky, Sergey Vakula, et al. "Application of Langmuir-Blodgett Technique for Depositing Thin Films of Lipids from Archaebacterium." In Molecular Manufacturing. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4899-0215-3_4.

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Sirtori, Cesare R., and Massimiliano Ruscica. "Lipid-lowering drugs." In ESC CardioMed. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0039.

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Hyperlipidaemias, multifactorial conditions partly genetically and partly life habit induced, are the most important underlying risk factors for cardiovascular disease. They can lead to arterial lipid deposition with a consequent increased risk of coronary events. The primary effort in hypolipidaemic drug therapy is focused on the lowering of the primary carriers of cholesterol, the low-density lipoproteins (LDLs), but more recent efforts have been placed on the lowering of triglycerides. Reduced levels of the protective high-density lipoproteins (HDLs) are generally considered a primary risk factor, ‘dysfunctional’ HDL may probably be a more important factor. Among drugs primarily reducing LDL cholesterol the most important systemic agents are statins. Non-systemic agents, such as resins, have a lesser use, whereas ezetimibe is frequently given in combination with statins. A new series of systemic compounds, the inhibitors of proprotein convertase subtilisin/kexin type 9 (PCSK9), recently available, have a higher activity on LDL cholesterol. Drugs indicated for the treatment of the homozygous forms of hypercholesterolaemia are lomitapide, an inhibitor of the microsomal transfer protein, and the antisense nucleotide mipomersen, designed to inhibit synthesis of apolipoprotein B. Treatment of hypertriglyceridaemias mainly relies on fibrates, activating the peroxisomal proliferator-activated receptor-α‎. They treat particularly the atherogenic dyslipidaemias (elevated triglycerides with low HDL cholesterol). Nicotinic acid is less frequently used and the omega-3 fatty acids have an as yet unclear cardiovascular protective activity.

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Conference papers on the topic "Lipid deposition"

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Mostaço-Guidolin, Leila B., Alex C. T. Ko, Fei Wang, et al. "Evaluating collagen morphology and pathological lipid deposition using multiphoton image statistics." In SPIE BiOS, edited by Ammasi Periasamy, Peter T. C. So, and Karsten König. SPIE, 2014. http://dx.doi.org/10.1117/12.2038232.

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Verschoor, Pola, Megan Fraser, Mirela Delibegovic, Graeme F. Nixon, and Justin J. Rochford. "125 A novel protein target to reduce plaque formation and lipid deposition in experimental atherosclerosis." In British Cardiovascular Society Annual Conference ‘High Performing Teams’, 4–6 June 2018, Manchester, UK. BMJ Publishing Group Ltd and British Cardiovascular Society, 2018. http://dx.doi.org/10.1136/heartjnl-2018-bcs.124.

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Verschoor, Pola J., Megan L. Fraser, Justin J. Rochford, Mirela Delibegovic, and Graeme F. Nixon. "RW1 A novel protein target to reduce plaque formation and lipid deposition in experimental atherosclerosis." In The Scottish Cardiovascular Forum 2019, Saturday 2nd February 2019, The Centre for Health Science, Old Perth Road, Inverness, Scotland. BMJ Publishing Group Ltd and British Cardiovascular Society, 2019. http://dx.doi.org/10.1136/heartjnl-2019-scf.6.

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Hallow, K. Melissa, Peter C. Carnell, W. Robert Taylor, and Raymond P. Vito. "Collocation of Macrophages and Mechanical Stress in Human Coronary Atherosclerotic Plaque." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176528.

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Cardiovascular disease (CVD) is the leading cause of death in the United States. One of the primary causes of CVD is atherosclerosis, a progressive disease that begins with the deposition of lipid on arterial walls to form plaque. Over time, some plaques progress to an unstable configuration that is vulnerable to rupture, while others remodel to more stable configurations. The factors directing plaque progression toward instability are poorly understood.

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Chu, Benjamin, Dean Ho, Hyeseung Lee, Karen Kuo, and Carlo Montemagno. "Protein-Functionalized Proton Exchange Membranes." In ASME 2004 3rd Integrated Nanosystems Conference. ASMEDC, 2004. http://dx.doi.org/10.1115/nano2004-46018.

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Protein-functionalized biomimetic membranes, based upon a triblock copolymer simulating a natural lipid bilayer in a single chain, serves as a core technology for applications in bioenergetics. Monolayers of block copolymer, which simulates the hydrophilic-hydrophobic-hydrophilic chain of a natural cell membrane, can be formed by Langmuir-Blodgett (LB) deposition and provides a favorable environment for protein refolding. Large-scale membrane formation is achieved using LB deposition on a variety of substrates, such as gold, quartz, silicon, and Nafion®. We have successfully inserted membrane

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Sun, Hyung Jin, Yunjie Wang, and Katherine Yanhang Zhang. "Changes in the Mechanical Properties of Arterial Elastin With Cholesterol Effect." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14591.

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Elastin is a protein in the extracellular matrix that provides critical mechanical properties of elasticity and extensibility to many connective tissues, including arteries. Such properties of elastin allow arteries to accommodate deformations encountered during physiological functions. Elastin is subjected to changes in mechanical properties upon exposure to various chemical environments. Elastin is a hydrophobic protein, which makes it an attractive site for the deposition of hydrophobic ligands such as cholesterol [1]. Cholesterol is a type of lipid that gradually builds up along arterial w

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Ho, D., B. Chu, H. Lee, K. Kuo, and C. D. Montemagno. "Fabrication of Hybrid Bionanodevices Based on Coupled Protein Functionality." In ASME 2004 3rd Integrated Nanosystems Conference. ASMEDC, 2004. http://dx.doi.org/10.1115/nano2004-46012.

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Block copolymer-based membrane technology represents a versatile class of nanoscale materials in which biomolecules, such as membrane proteins, can be reconstituted. Among its many advantages over conventional lipid-based membrane systems, block copolymers can mimic natural cell biomembrane environments in a single chain, enabling large-area membrane fabrication using methods like Langmuir-Blodgett deposition, or spontaneous protein-functionalized nanovesicle formation. Based on this unique membrane property, a wide variety of membrane proteins possessing unique functionalities including pH/vo

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