Relevant bibliographies by topics / Lipid Peroxidation and Phospholipid Hydroperoxides

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Reports

Academic literature on the topic 'Lipid Peroxidation and Phospholipid Hydroperoxides'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Lipid Peroxidation and Phospholipid Hydroperoxides"

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Sugihara, T., W. Rawicz, EA Evans, and RP Hebbel. "Lipid hydroperoxides permit deformation-dependent leak of monovalent cation from erythrocytes." Blood 77, no. 12 (1991): 2757–63. http://dx.doi.org/10.1182/blood.v77.12.2757.2757.

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Abstract Subtle peroxidative perturbation of normal red blood cells (RBC) using t-butylhydroperoxide creates a leak pathway for monovalent cations that is reversibly activated by cell deformation. To determine what factor promotes expression of this unique membrane defect, we have dissected “peroxidation” into components that can be evaluated separately by comparing K leak from suitably modified RBC during elliptical deformation and parallel control incubation. Selective introduction of phospholipid hydroperoxides into normal RBC membranes successfully induces a deformation-dependent leak path
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Sugihara, T., W. Rawicz, EA Evans, and RP Hebbel. "Lipid hydroperoxides permit deformation-dependent leak of monovalent cation from erythrocytes." Blood 77, no. 12 (1991): 2757–63. http://dx.doi.org/10.1182/blood.v77.12.2757.bloodjournal77122757.

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Subtle peroxidative perturbation of normal red blood cells (RBC) using t-butylhydroperoxide creates a leak pathway for monovalent cations that is reversibly activated by cell deformation. To determine what factor promotes expression of this unique membrane defect, we have dissected “peroxidation” into components that can be evaluated separately by comparing K leak from suitably modified RBC during elliptical deformation and parallel control incubation. Selective introduction of phospholipid hydroperoxides into normal RBC membranes successfully induces a deformation-dependent leak pathway havin
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SPICKETT, Corinne M., Nicola RENNIE, Helen WINTER, et al. "Detection of phospholipid oxidation in oxidatively stressed cells by reversed-phase HPLC coupled with positive-ionization electroscopy MS." Biochemical Journal 355, no. 2 (2001): 449–57. http://dx.doi.org/10.1042/bj3550449.

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Measurement of lipid peroxidation is a commonly used method of detecting oxidative damage to biological tissues, but the most frequently used methods, including MS, measure breakdown products and are therefore indirect. We have coupled reversed-phase HPLC with positive-ionization electrospray MS (LC-MS) to provide a method for separating and detecting intact oxidized phospholipids in oxidatively stressed mammalian cells without extensive sample preparation. The elution profile of phospholipid hydroperoxides and chlorohydrins was first characterized using individual phospholipids or a defined p
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Gebicki, J. M., J. Du, J. Collins, and H. Tweeddale. "Peroxidation of proteins and lipids in suspensions of liposomes, in blood serum, and in mouse myeloma cells." Acta Biochimica Polonica 47, no. 4 (2000): 901–11. http://dx.doi.org/10.18388/abp.2000_3945.

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There is growing evidence that proteins are early targets of reactive oxygen species, and that the altered proteins can in turn damage other biomolecules. In this study, we measured the effects of proteins on the oxidation of liposome phospholipid membranes, and the formation of protein hydroperoxides in serum and in cultured cells exposed to radiation-generated hydroxyl free radicals. Lysozyme, which did not affect liposome stability, gave 50% protection when present at 0.3 mg/ml, and virtually completely prevented lipid oxidation at 10 mg/ml. When human blood serum was irradiated, lipids wer
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Lane, Darius J. R., Billie Metselaar, Mark Greenough, Ashley I. Bush, and Scott J. Ayton. "Ferroptosis and NRF2: an emerging battlefield in the neurodegeneration of Alzheimer's disease." Essays in Biochemistry 65, no. 7 (2021): 925–40. http://dx.doi.org/10.1042/ebc20210017.

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Abstract Ferroptosis is an iron- and lipid peroxidation-dependent cell death modality and emerging evidence indicates that ferroptosis has great explanatory potential for neuronal loss and associated CNS dysfunction in a range of neurodegenerative diseases (e.g., Alzheimer's, Parkinson's and Huntington's diseases, Motor neuron disease, Friedreich ataxia (FRDA)). Ferroptotic death results from lethal levels of phospholipid hydroperoxides that are generated by iron-dependent peroxidation of polyunsaturated fatty acids (PUFAs), such as arachidonic and adrenic acids, which are conjugated to specif
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Pak, Jhang Ho, Yefim Manevich, Han Suk Kim, Sheldon I. Feinstein, and Aron B. Fisher. "An Antisense Oligonucleotide to 1-cys Peroxiredoxin Causes Lipid Peroxidation and Apoptosis in Lung Epithelial Cells." Journal of Biological Chemistry 277, no. 51 (2002): 49927–34. http://dx.doi.org/10.1074/jbc.m204222200.

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1-cys peroxiredoxin (1-cysPrx), a member of the peroxiredoxin superfamily, reduces phospholipid hydroperoxides as well as organic peroxides and H2O2. To determine the physiological function(s) of 1-cysPrx, we have used an antisense strategy to suppress endogenous 1-cysPrx in L2 cells, a rat lung epithelial cell line. A 25-base antisense morpholino oligonucleotide was designed to bind a complementary sequence overlapping the translational start site (−18 to +7) in the rat 1-cysPrx mRNA, blocking protein synthesis. Treatment with an antisense oligonucleotide for 48 h resulted in approximately 60
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Yang, Wan Seok, Katherine J. Kim, Michael M. Gaschler, Milesh Patel, Mikhail S. Shchepinov, and Brent R. Stockwell. "Peroxidation of polyunsaturated fatty acids by lipoxygenases drives ferroptosis." Proceedings of the National Academy of Sciences 113, no. 34 (2016): E4966—E4975. http://dx.doi.org/10.1073/pnas.1603244113.

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Ferroptosis is form of regulated nonapoptotic cell death that is involved in diverse disease contexts. Small molecules that inhibit glutathione peroxidase 4 (GPX4), a phospholipid peroxidase, cause lethal accumulation of lipid peroxides and induce ferroptotic cell death. Although ferroptosis has been suggested to involve accumulation of reactive oxygen species (ROS) in lipid environments, the mediators and substrates of ROS generation and the pharmacological mechanism of GPX4 inhibition that generates ROS in lipid environments are unknown. We report here the mechanism of lipid peroxidation dur
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Thomas, J. P., M. Maiorino, F. Ursini, and A. W. Girotti. "Protective action of phospholipid hydroperoxide glutathione peroxidase against membrane-damaging lipid peroxidation. In situ reduction of phospholipid and cholesterol hydroperoxides." Journal of Biological Chemistry 265, no. 1 (1990): 454–61. http://dx.doi.org/10.1016/s0021-9258(19)40252-4.

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Babizhayev, M. A., M. C. Seguin, J. Gueyne, R. P. Evstigneeva, E. A. Ageyeva та G. A. Zheltukhina. "l-carnosine (β-alanyl-l-histidine) and carcinine (β-alanylhistamine) act as natural antioxidants with hydroxyl-radical-scavenging and lipid-peroxidase activities". Biochemical Journal 304, № 2 (1994): 509–16. http://dx.doi.org/10.1042/bj3040509.

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Carnosine (beta-alanyl-L-histidine) and carcinine (beta-alanylhistamine) are natural imidazole-containing compounds found in the non-protein fraction of mammalian tissues. Carcinine was synthesized by an original procedure and characterized. Both carnosine and carcinine (10-25 mM) are capable of inhibiting the catalysis of linoleic acid and phosphatidylcholine liposomal peroxidation (LPO) by the O2(-.)-dependent iron-ascorbate and lipid-peroxyl-radical-generating linoleic acid 13-monohydroperoxide (LOOH)-activated haemoglobin systems, as measured by thiobarbituric-acid-reactive substance. Carc
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Arevalo, José, and José Vázquez-Medina. "The Role of Peroxiredoxin 6 in Cell Signaling." Antioxidants 7, no. 12 (2018): 172. http://dx.doi.org/10.3390/antiox7120172.

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Peroxiredoxin 6 (Prdx6, 1-cys peroxiredoxin) is a unique member of the peroxiredoxin family that, in contrast to other mammalian peroxiredoxins, lacks a resolving cysteine and uses glutathione and π glutathione S-transferase to complete its catalytic cycle. Prdx6 is also the only peroxiredoxin capable of reducing phospholipid hydroperoxides through its glutathione peroxidase (Gpx) activity. In addition to its peroxidase activity, Prdx6 expresses acidic calcium-independent phospholipase A2 (aiPLA2) and lysophosphatidylcholine acyl transferase (LPCAT) activities in separate catalytic sites. Prdx
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Dissertations / Theses on the topic "Lipid Peroxidation and Phospholipid Hydroperoxides"

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Södergren, Eva. "Lipid peroxidation in vivo : Evaluation and application of methods for measurement." Doctoral thesis, Uppsala universitet, Institutionen för folkhälso- och vårdvetenskap, 2000. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-1250.

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Lipid peroxidation is thought to be an important factor in the pathophysiology of a number of diseases and in the process of ageing, but its measurement in vivo has been difficult. The aim of this thesis was to evaluate methods for measurement of lipid peroxidation in vivo that are suitable for clinical investigations, and to apply these methods in animal and human studies investigating basal conditions and situations associated with increased lipid peroxidation. The ferrous oxidation in xylenol orange assay for quantification of total plasma lipid hydroperoxides was re-evaluated regarding sam
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Mattos, Thiago Cardoso Genaro de. "Modificação de proteínas por produtos de oxidação do colesterol: mecanismos e implicações biológicas." Universidade de São Paulo, 2014. http://www.teses.usp.br/teses/disponiveis/46/46131/tde-01102014-075856/.

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O colesterol é um importante componente das membranas celulares em eucariotos superiores, desempenhando papéis estruturais e funcionais. O colesterol possui uma insaturação em sua estrutura sendo, portanto, alvo de oxidação mediada por espécies reativas de oxigênio e/ou nitrogênio. A oxidação não enzimática do colesterol gera, como produtos primários, os hidroperóxidos de colesterol. Tais moléculas, por sua vez, são altamente reativas e podem reagir com metais livres e/ou metaloproteínas, trazendo consequências à celula. Neste sentido, o primeiro capítulo deste trabalho tem como objetivo estud
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Calhoon, Elisabeth A. "Lipid class and phospholipid species composition associated with life history variation in north temperate and neotropical birds." The Ohio State University, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=osu1450091613.

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Grim, Jeffrey Matthew. "The Effects of Acclimation Temperature on the Susceptibility of Biological Membranes in Fish Muscle to Lipid Peroxidation and the Role of Phospholipid Composition on Antioxidant Defenses in Vertebrates." Ohio University / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1282594590.

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Scalfo, Alexsandra Cristina. "Geração de oxigênio molecular singlete: termólise de endoperóxidos naftalênicos e reações de hidroperóxidos lipídicos com íon nitrônio." Universidade de São Paulo, 2014. http://www.teses.usp.br/teses/disponiveis/46/46131/tde-01102014-103728/.

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Oxigênio molecular singlete [O2(1Δg)], uma espécie excitada, desempenha um papel importante em sistemas químicos e biológicos. É um poderoso eletrófilo, que reage com moléculas ricas em elétrons através de cicloadições [2+2], [4+2] e reações tipo ene. Ácidos graxos poliinsaturados, proteínas e DNA são alvos vulneráveis para o ataque de O2(1Δg). Os endoperóxidos de derivados de naftaleno são muito úteis e versáteis como fontes limpas de O2(1Δg), uma vez que são quase quimicamente inertes. Por outro lado, o desenvolvimento de novas fontes de O2(1Δg) ainda é uma tarefa desafia
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Book chapters on the topic "Lipid Peroxidation and Phospholipid Hydroperoxides"

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Weiss, R. H., J. L. Arnold, and R. W. Estabrook. "Cytochrome P-450 Interaction with Arachidonic Acid Hydroperoxides: Role in Lipid Peroxidation." In Prostaglandin and Lipid Metabolism in Radiation Injury. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5457-4_12.

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Coudray, C., M. J. Richard, and A. E. Favier. "Determination of primary and secondary lipid peroxidation products: Plasma lipid hydroperoxides and thiobarbituric acid reactive substances." In Analysis of Free Radicals in Biological Systems. Birkhäuser Basel, 1995. http://dx.doi.org/10.1007/978-3-0348-9074-8_13.

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Onur Yaman, Suzan, and Adnan Ayhanci. "Lipid Peroxidation." In Lipid Peroxidation [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.95802.

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Lipid peroxidation (LPO) is initiated by the attack of free radicals (eg OH ·, O2- and H2O2) on cellular or organelle membranes phospholipids or polyunsaturated fatty acids (PUFA), and with the formation of various types of aldehydes, ketones, alkanes, carboxylic acids and polymerization products. It is an autoxidation process that results. These products are highly reactive with other cellular components and serve as biological markers of LPO. Malondialdehyde (MDA), a toxic aldehyde end product of LPO, causes structural changes that mediate its oxidation, such as fragmentation, modification,
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Flohé, Leopold. "The Discovery of Glutathione Peroxidases: Milestones in Understanding the Biological Role of Selenium und Sulfur." In Chalcogen Chemistry: Fundamentals and Applications. The Royal Society of Chemistry, 2023. http://dx.doi.org/10.1039/bk9781839167386-00603.

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With the discovery of glutathione peroxidase (GPx1), the role of glutathione in counteracting oxidative challenge became clear. GPx1 was the first selenoprotein discovered in mammals. It contains a selenocysteine residue integrated into the peptide chain. The phospholipid hydroperoxide glutathione peroxidase (GPx4) also proved to be a selenoprotein. In the cytosol, it inhibits lipid peroxidation and ferroptosis; in the nucleus, it supports protamine compaction; its mitochondrial expression form builds the sheath surrounding the mitochondria in spermatozoa and is essential for male fertility. I
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Ursini, Fulvio, Matilde Maiorino, Antonella Roveri, et al. "PHOSPHOLIPID HYDROPEROXIDE GLUTATHIONE PEROXIDASE: FROM THE INHIBITION OF LIPID PEROXIDATION TO THE CONTROL OF CELLULAR FUNCTIONS?" In Oxidative Damage & Repair. Elsevier, 1991. http://dx.doi.org/10.1016/b978-0-08-041749-3.50111-x.

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Niki, Etsuo. "Lipid peroxides." In Experimental protocols for reactive oxygen and nitrogen species. Oxford University PressOxford, 2000. http://dx.doi.org/10.1093/oso/9780198506683.003.0043.

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Abstract Lipid peroxides are major primary products of lipid peroxidation. Measurement of lipid peroxides in in vitro experiments or biological samples is often required to assess the extent both of oxidative damage and of changes in antioxidant activity. Various methods have been proposed and used, but each method has both merits and demerits. There still is no single method which is specific, quantitative and sufficiently accurate that can be applied widely. Numerous kinds of lipid peroxide can be formed during lipid peroxidation and these subsequently undergo secondary reactions Hence, meas
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Shimasaki, Hiroyuki. "Diene conjugation." In Experimental protocols for reactive oxygen and nitrogen species. Oxford University PressOxford, 2000. http://dx.doi.org/10.1093/oso/9780198506683.003.0041.

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Abstract Lipid peroxidation begins with the abstraction of a hydrogen atom from an unsaturated lipid, and involves the formation and propagation of lipid radicals, uptake of oxygen, and rearrangement of double bonds in the polyunsaturated fatty acid (PUFA). Rearrangement of the double bonds results in the formation of conjugated dienes (lipid hydroperoxides), which are characterized by intense absorption near 233 nm (1).
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Brown, R. K., and F. J. Kelly. "Peroxides and other products." In Free Radicals. Oxford University PressOxford, 1996. http://dx.doi.org/10.1093/oso/9780199635603.003.0008.

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Abstract The only definitive way to demonstrate excessive free radical activity in vivo is by electron spin resonance as already discussed in Chapter 2, but clearly this is currently inapplicable in clinical practice due to the nature of the spin traps that must be used. Instead investigators must rely upon the measurement of established markers of free radical activity in biological fluids and tissues. Traditionally, most markers of oxidative injury utilized reflect free radical attack on polyunsaturated fatty acids, with the classical route of attack involving lipid peroxidation, generating
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Reports on the topic "Lipid Peroxidation and Phospholipid Hydroperoxides"

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Handa, Avtar K., Yuval Eshdat, Avichai Perl, Bruce A. Watkins, Doron Holland, and David Levy. Enhancing Quality Attributes of Potato and Tomato by Modifying and Controlling their Oxidative Stress Outcome. United States Department of Agriculture, 2004. http://dx.doi.org/10.32747/2004.7586532.bard.

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General The final goal and overall objective of the current research has been to modify lipid hydroperoxidation in order to create desirable phenotypes in two important crops, potato and tomato, which normally are exposed to abiotic stress associated with such oxidation. The specific original objectives were: (i) the roles of lipoxygenase (LOX) and phospholipids hydroperoxide glutathione peroxidase (PHGPx) in regulating endogenous levels of lipid peroxidation in plant tissues; (ii) the effect of modified lipid peroxidation on fruit ripening, tuber quality, crop productivity and abiotic stress
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Kanner, Joseph, Edwin Frankel, Stella Harel, and Bruce German. Grapes, Wines and By-products as Potential Sources of Antioxidants. United States Department of Agriculture, 1995. http://dx.doi.org/10.32747/1995.7568767.bard.

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Several grape varieties and red wines were found to contain large concentration of phenolic compounds which work as antioxidant in-vitro and in-vivo. Wastes from wine production contain antioxidants in large amounts, between 2-6% on dry material basis. Red wines but also white wines were found to prevent lipid peroxidation of turkey muscle tissues stored at 5oC. The antioxidant reaction of flavonoids found in red wines against lipid peroxidation were found to depend on the structure of the molecule. Red wine flavonoids containing an orthodihydroxy structure around the B ring were found highly
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