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Books on the topic 'Lipophilie'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Baker, Neil David. Lipophilic antifolates as potential antipsoriatic agents. Birmingham: Aston University. Department of Pharmaceutical Sciences, 1989.

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2

Pashley, Stephen George Henry. Pharmacokinetics and metabolism of MZPES, a novel lipophilic antifolate. Birmingham: Aston University. Department of Pharmaceutical Sciences, 1987.

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3

I, Glushenkova Anna, and SpringerLink (Online service), eds. Lipids, Lipophilic Components and Essential Oils from Plant Sources. London: Springer London, 2012.

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4

Krugli͡akov, P. M. Hydrophile-lipophile balance of surfactants and solid particles: Physicochemical aspects and applications. Amsterdam: Elsevier Science B. V., 2000.

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5

Borg-Neczak, Kathleen. The disposition of some metals in mammals and fish: Effects of lipophilic chelation. Uppsala: Sveriges Lantbruksuniversitet, 1994.

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6

Gottofrey, James. Disposition of cadmium, nickel, mercury and methylmercury in fish and effects of lipophilic metal chelation. Uppsala: Sveriges Lantbruksuniversitet, 1990.

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7

R, Nagarajan. Amphiphiles: Molecular assembly and applications. Washington, DC: American Chemical Society, 2011.

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8

(Editor), Vladimir Pliska, Bernard Testa (Editor), Han Van De Waterbeemd (Editor), and Han Van De Waterbeemd (Editor), eds. Lipophilicity in Drug Action and Toxicology. John Wiley & Sons, 1996.

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9

Lipophilicity in drug action and toxicology /edited by Vladimir Pliška, Bernard Testa, and Han van de Waterbeemd. Weinheim: VCH, 1996.

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10

Lipids Lipophilic Components And Essential Oils From Plant Sources. Springer, 2011.

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11

Kahan, Ileana. Studies on the integration of lipophilin in the human myelin membrane. 1987.

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12

Hydrophile-Lipophile Balance of Surfactants and Solid Particles - Physicochemical Aspects and Applications. Elsevier, 2000. http://dx.doi.org/10.1016/s1383-7303(00)x8012-x.

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13

Smith, Glynda A. The synthesis and examination of lipophilic crown ethers as selective reagents for lithium. 1990.

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14

Persaud, Rajenda. The interaction of the major glycosylation site of human myelin basic protein with lipids and lipophilin. 1987.

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15

M. Englmaierová, M. Marounek*, M. Skřivan, and D. Dušková. Effect of the alga Chlorella vulgaris alone and in combination with rapeseed oil on carotenoids and lipophilic vitamins in eggs. Verlag Eugen Ulmer, 2020. http://dx.doi.org/10.1399/eps.2020.298.

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16

Sia, Alex Tiong Heng, Ban Leong Sng, and Serene Leo. Maintenance of neuraxial labour analgesia. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713333.003.0015.

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Labour pain may be treated with epidural analgesia providing effective pain relief with no maternal sedation to allow maternal participation in the labouring process. Epidural analgesia is commonly initiated using an epidural or combined spinal–epidural technique. Most epidural maintenance regimens would include a long-acting amide anaesthetic in low concentration together with a lipophilic opioid to maximize analgesia whilst reducing motor blockade. With the advent of advanced infusion delivery systems, maintenance of epidural analgesia may be individualized through the use of patient-controlled epidural analgesia systems. Patient self-administered bolus is used as a feedback to increase local anaesthetic use when labour pain intensifies. Recent developments in pump technology and innovations have enabled novel epidural delivery systems such as automated mandatory boluses, variable frequency automated mandatory boluses, and computer integrated patient controlled epidural analgesia.

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17

Columb, Malachy O. Local anaesthetic agents. Edited by Michel M. R. F. Struys. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0017.

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Local anaesthetic agents cause a pharmacologically induced reversible neuropathy characterized by axonal conduction blockade. They act by blocking the sodium ionophore and exhibit membrane stabilizing activity by inhibiting initiation and propagation of action potentials. They are weak bases consisting of three components: a lipophilic aromatic ring, a link, and a hydrophilic amine. The chemical link classifies them as esters or amides. Local anaesthetics diffuse through the axolemma as unionized free-base and block the ionophore in the quaternary ammonium ionized form. The speed of onset of block is therefore dependent on the pKa of the agent and the ambient tissue pH. Esters undergo hydrolysis by plasma esterases and amides are metabolized by hepatic microsomal mixed-function oxidases. Local anaesthetics are bound in the blood to α‎1-acid glycoproteins. Pharmacological potency is dependent on the lipid solubility of the drug as is the potential for systemic toxicity. The blood concentrations required to cause cardiovascular system (CVS) collapse and early central nervous system (CNS) toxicity are used to quantify the CVS:CNS toxicity ratio. Local anaesthetics also have the potential to induce direct neuronal damage. Intravenous lipid emulsion is used for the treatment of systemic toxicity but the scientific evidence is inconsistent. With regard to the pipecoloxylidine local anaesthetics, early evidence indicated that the S- was less toxic than the R-enantiomer. However, clinical research using minimum local analgesic concentration designs suggests that reduced systemic toxicity and motor block sparing is mainly explained by potency rather than enantiomerism.

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18

Servin, Frédérique S., and Valérie Billard. Anaesthesia for the obese patient. Edited by Philip M. Hopkins. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0087.

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Obesity is becoming an epidemic health problem, and the number of surgical patients with a body mass index of more than 50 kg m−2 requiring anaesthesia is increasing. Obesity is associated with physiopathological changes such as metabolic syndrome, cardiovascular disorders, or sleep apnoea syndrome, most of which improve with weight loss. Regarding pharmacokinetics, volumes of distribution are increased for both lipophilic and hydrophilic drugs. Consequently, doses should be adjusted to total body weight (propofol for maintenance, succinylcholine, vancomycin), or lean body mass (remifentanil, non-depolarizing neuromuscular blocking agent). For all drugs, titration based on monitoring of effects is recommended. To minimize recovery delays, drugs with a rapid offset of action such as remifentanil and desflurane are preferable. Poor tolerance to apnoea with early hypoxaemia and atelectasis warrant rapid sequence induction and protective ventilation. Careful positioning will prevent pressure injuries and minimize rhabdomyolysis which are frequent. Because of an increased risk of pulmonary embolism, multimodal prevention is mandatory. Regional anaesthesia, albeit technically difficult, is beneficial in obese patients to treat postoperative pain and improve rehabilitation. Maximizing the safety of anaesthesia for morbidly obese patients requires a good knowledge of the physiopathology of obesity and great attention to detail in planning and executing anaesthetic management. Even in elective surgery, many cases can be technical challenges and only a step-by-step approach to the avoidance of potential adverse events will result in the optimal outcome.

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