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Relevant bibliographies by topics / Lipoprotein(a)-cholesterol / Dissertations / Theses

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Dissertations / Theses on the topic 'Lipoprotein(a)-cholesterol'

Author: Grafiati

Published: 4 June 2025

Last updated: 1 August 2025

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1

Wilson, Diane Lilias. "Lipoprotein oxidation and the cholesterol esterification in a human monocytic cell line." Thesis, University College London (University of London), 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.309473.

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2

Cooke, Allison L. B. A. "The Molecular Interaction of Apolipoprotein A-I and Lecithin: Cholesterol Acyl Transferase." University of Cincinnati / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1543580636448357.

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3

Coghill, Nicola. "The effect of a walking programme on the total cholesterol to high-density lipoprotein cholesterol ratio and components of the metabolic syndrome in hyperlipidaemic men aged 45-65." Thesis, University of Bristol, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.430156.

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4

Sun, Cathy J. "Concordance and Discordance Between Non-High-Density Lipoprotein Cholesterol and Apolipoprotein B as Cardiovascular Disease Risk Markers over the Full Spectrum of Hypertriglyceridemia: A Cross-sectional Analysis of Lipid Clinic Data." Thesis, Université d'Ottawa / University of Ottawa, 2021. http://hdl.handle.net/10393/41980.

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Cardiovascular disease is a leading cause of morbidity and mortality worldwide. Lipid biomarkers are frequently used for prediction of cardiovascular disease risk. Triglycerides are routinely checked in blood work, and triglycerides are a key component of lipoproteins that contribute to atherogenic plaques, which cause cardiovascular disease. High triglycerides are a common condition in the general population. The relative effect of high triglycerides on the lipid biomarkers (non-high-density lipoprotein cholesterol, and apolipoprotein B) for cardiovascular disease risk prediction is the focus

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5

Lee, Dong-Young. "Modification of high-density lipoprotein 3 by secretory sphingomyelinase, its effects on cholesterol trafficking/ transport, and secretory sphingomyelinase as a protein biomarker for inflammatory disease." Thesis, McGill University, 2008. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=21990.

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Secretory sphingomyelinase (S-SMase), one of the products of acid sphingomyelinase (ASM) or sphingomyelin phosphodiesterase-1 (SMPD-1) gene, hydrolyses sphingomyelin (SM) present in plasma membrane and lipoprotein particles. A number of studies have indicated that S-SMase plays an important role in intracellular or paracrine ceramide second messenger signaling pathways and development of subendothelial atherosclerotic plaque. However, a well-defined function of this enzyme in reverse cholesterol pathway (RCT) in vivo is yet to be elucidated. In this dissertation, we investigated modification

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6

Alghamdi, Rasha Hassen. "Development of Inhibitors of Human PCSK9 as Potential Regulators of LDL-Receptor and Cholesterol." Thesis, Université d'Ottawa / University of Ottawa, 2014. http://hdl.handle.net/10393/30492.

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Proprotein Convertase Subtilisin/Kexin 9 (PCSK9) is the ninth member of the Ca+2-dependent mammalian proprotein convertase super family of serine endoproteases that is structurally related to the bacterial subtilisin and yeast kexin enzymes. It plays a critical role in the regulation of lipid metabolism and cholesterol homeostasis by binding to and degrading low-density lipoprotein-receptor (LDL-R) which is responsible for the clearance of circulatory LDL-cholesterol from the blood. Owing to this functional property, there is plenty of research interest in the development of functional inhibit

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7

Gibran, Luciano. "A expressão de receptores de LDL em membrana celular de focos de endometriose profunda para viabilização do uso de nanoemulsão lipídica carreadora de droga antiproliferativa." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/5/5139/tde-04112016-111940/.

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Objetivo: O objetivo desse estudo foi avaliar a expressão gênica e a determinação proteica de receptores de LDL (LDL-R e LRP-1) na lesão de endometriose profunda e comparar com o endométrio de mulheres com e sem endometriose, assim como determinar o perfil lipídico de pacientes com e sem endometriose profunda. Pacientes e métodos: Realizamos estudo transversal, caso-controle, exploratório com 39 pacientes, sendo 20 pacientes com diagnóstico histológico de endometriose profunda com comprometimento intestinal e 19 pacientes sem endometriose submetidas à laqueadura tubária laparoscópica. Foram co

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8

Hayat, Shaista. "The effect of serum amyloid A on high density lipoprotein function." Thesis, London School of Hygiene and Tropical Medicine (University of London), 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.287948.

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9

Almeida, Cristina Pio de. "Captação celular de uma nanoemulsão semelhante a LDL (LDE): efeito da variação na composição química e expressão de receptores de lipoproteínas." Universidade de São Paulo, 2010. http://www.teses.usp.br/teses/disponiveis/5/5166/tde-22092010-132936/.

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A nanoemulsão LDE tem composição lipídica semelhante à da LDL natural e é utilizada para estudos do metabolismo da LDL. Estudos anteriores mostraram que a LDE é captada pelas células pelo LDL-r, porém outros receptores podem estar envolvidos nesta captação, como LRP-1, CD36 e CD68. Os objetivos deste estudo foram: investigar a captação da LDE por células endoteliais, fibroblastos, monócitos, macrófagos e H292, identificar os receptores envolvidos na captação da LDE pelas mesmas células e avaliar os efeitos da modificação química da LDE sobre a estabilidade, captação celular, lipoperoxidação ce

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10

Yang, Kai-Sheng, and 楊凱盛. "Specific Detection of High Density Lipoprotein (HDL) Cholesterol in Whole Blood on a Dry-phase Test Strip." Thesis, 2015. http://ndltd.ncl.edu.tw/handle/s93ezt.

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11

Van, Zyl Tertia. "The association of LDLR and PCSK9 variants with LDL-c levels in a black South African population in epidemiological transition / Tertia van Zyl." Thesis, 2013. http://hdl.handle.net/10394/15738.

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Background Elevated concentrations of low-density lipoprotein cholesterol (LDL-c) are a major risk factor for the development of coronary artery disease (CAD) because of their role in the progression of atherosclerosis. The black South African population is known to have had historically low LDL-c and in the past there was almost no CAD in the population. However, as this population moves through the nutrition transition, LDL-c levels are increasing. LDL-c levels are regulated by the LDL receptors, which is the major protein involved with transporting cholesterol across cell membranes in human

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12

Adams, Thaddeus Hunter. "Effects of a High Oleic Acid Beef Diet on Cardiovascular Disease Risk Factors of Human Subjects." Thesis, 2012. http://hdl.handle.net/1969.1/ETD-TAMU-2012-08-11871.

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The consumption of high-fat hamburger enriched with saturated fatty acids (SFA) and trans-fatty acids (TFA) may increase risk factors for cardiovascular disease, whereas hamburger enriched with monounsaturated fatty acids (MUFA) may have the opposite effect. Ten mildly hypercholesterolemic men consumed five, 114-g hamburger patties per week for two consecutive phases. Participants consumed low-MUFA (high SFA) hamburger (MUFA:SFA = 0.95; produced from pasture-fed cattle) for 5 wk, consumed their habitual diets for 3 wk, and then consumed high-MUFA hamburger (MUFA:SFA = 1.31; produced from gra

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13

Matos, Carla Manuela Silva. "Utilização terapêutica das estatinas: indicações, novas perspetivas e efeitos laterais a curto e longo prazo." Master's thesis, 2019. http://hdl.handle.net/10284/8723.

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As doenças cardiovasculares são a principal causa de morte a nível mundial. Estas habitualmente estão associadas a um conjunto de condições que se designam por fatores de risco. Estes fatores podem ser modificáveis como o estilo de vida, a diabetes, a dislipidemia e hipertensão arterial ou não modificáveis como a hereditariedade, o sexo e a idade. Depois de terem sido descobertas as estatinas, no ano de 1978 estas passaram a ser os fármacos de eleição para o tratamento da dislipidemia quer na prevenção primária quer na prevenção secundária. As estatinas, atuam através da redução da HMG-CoA

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14

Létourneau, Jason. "Les bactéries exprimant AIDA-I interagissent avec l'apolipoprotéine A-I cellulaire." Thèse, 2012. http://hdl.handle.net/1866/9741.

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AIDA-I (adhesin involved in diffuse adherence) est une importante adhésine autotransporteur exprimée par certaines souches de Escherichia. coli impliquée dans la colonisation des porcelets sevrés causant la diarrhée post-sevrage et la maladie de l’œdème. Une précédente étude de notre laboratoire a identifié l’apolipoprotéine AI (ApoAI) du sérum porcin, la protéine structurale des lipoprotéines à haute densité, comme récepteur cellulaire putatif de AIDA-I. L’interaction entre ces deux protéines doit être caractérisée. Ici, nous montrons par ELISA que AIDA-I purifiée est capable d’interagir avec

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15

Zatu, Mandlenkosi Caswell. "Alcohol intake and cardiovascular function of black South Africans : a 5-year prospective study / Mandlenkosi Caswell Zatu." Thesis, 2015. http://hdl.handle.net/10394/15827.

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Motivation Alcohol consumption is one of the major risk factors of cardiovascular disease (CVD). Excessive alcohol drinking is the fifth leading cause of death worldwide and the prevalence of alcohol abuse continues to increase especially in low-income areas of sub-Saharan Africa. The alarming rate of urbanisation seems to be the driving force for excessive alcohol intake in the developing world. In addition to its influence on CVD, heavy drinking also results in a number of non-cardiovascular consequences that include injury, risky sexual behaviour, violent crime and family dysfunction among

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