Relevant bibliographies by topics / Lipoprotein A

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Lipoprotein A'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2025

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Journal articles on the topic "Lipoprotein A"

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Ozsavci, Derya, A. Nazli, O. Bingol Ozakpinar, G. Yanikkaya Demirel, B. Vanizor Kural, and A. Sener. "Native High-Density Lipoprotein and Melatonin Improve Platelet Response Induced by Glycated Lipoproteins." Folia Biologica 64, no. 4 (2018): 144–52. http://dx.doi.org/10.14712/fb2018064040144.

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Activated platelets and glycated lipoproteins are responsible for atherothrombosis in diabetics. Melatonin and native high-density lipoproteins are crucial in the preservation of pro/oxidant-antioxidant balance. The aim of the present study was to investigate the in vitro effects of native high-density lipoproteins and melatonin on altering the platelet response induced by glycated lipoproteins. Low-density lipoproteins and high-density lipoproteins were purified from plasma by ultracentrifugation and were glycated with glucose for three weeks. After incubation with or without melatonin/or nat
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De Sanctis, Juan B., Isaac Blanca, and Nicholas E. Bianco. "Effects of Different Lipoproteins on the Proliferative Response of Interleukin-2-Activated T Lymphocytes and Large Granular Lymphocytes." Clinical Science 89, no. 5 (1995): 511–19. http://dx.doi.org/10.1042/cs0890511.

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1. T lymphocytes and large granular lymphocytes internalized chylomicrons, very low-density lipoprotein, low-density lipoprotein, high-density lipoprotein and acetyl modified low-density lipoprotein through different receptors as assessed by flow cytometry. The observed internalization ranged from 8% to 20%. 2. All lipoproteins induced proliferative responses in T lymphocytes and large granular lymphocytes at optimum concentrations (40 μg of protein/ml for all lipoproteins except high-density lipoprotein). Chylomicrons, very low-density lipoprotein and low-density lipoprotein increased T-lymph
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Myers, D. E., W. N. Huang, and R. G. Larkins. "Lipoprotein-induced prostacyclin production in endothelial cells and effects of lipoprotein modification." American Journal of Physiology-Cell Physiology 271, no. 5 (1996): C1504—C1511. http://dx.doi.org/10.1152/ajpcell.1996.271.5.c1504.

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Although lipoprotein modification has been implicated in atherogenesis, the effect of modified forms of lipoproteins on vascular cell function has not been fully resolved. We have investigated lipoprotein-induced prostaglandin production by macrovascular endothelial cells. This study delineates early responses of endothelial cells after exposure to native and modified forms of the lipoproteins. Modification of lipoproteins by oxidation or glycation significantly affected the capacity of lipoproteins to induce prostacyclin (PGI2) production by bovine aortic endothelial cells (BAEC). Modified lo
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Yasmin, Raheela, Aashi Ahmed, Ambreen Javed, et al. "The Effect of Blood Sugar Fasting Levels on Diabetic Dyslipidemia." Pakistan Journal of Medical and Health Sciences 16, no. 4 (2022): 360–61. http://dx.doi.org/10.53350/pjmhs22164360.

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Background: Diabetic dyslipidemia is a group of lipoprotein defects described by raised triglycerides, elevated low density lipoprotein and reduced levels of high density lipoprotein. Objective: To assess the effect of blood sugar fasting levels on individual lipoproteins. Study design: Cross-sectional study Place and duration of study: Fauji Foundation Hospital Rawalpindi & POF Hospital Wah Cantt from 1st February 2014 to 31st July 2014. Methodology: Fifty patients with age from 30 to 70 years were enrolled. Patients' body mass index was calculated. Serum cholesterol, high density lipopro
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Huang, Haibin, Mingqun Lin, Xueqi Wang, et al. "Proteomic Analysis of and Immune Responses to Ehrlichia chaffeensis Lipoproteins." Infection and Immunity 76, no. 8 (2008): 3405–14. http://dx.doi.org/10.1128/iai.00056-08.

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ABSTRACT Ehrlichia chaffeensis is an obligately intracellular gram-negative bacterium and is the etiologic agent of human monocytic ehrlichiosis (HME). Although E. chaffeensis induces the generation of several cytokines and chemokines by leukocytes, E. chaffeensis lacks lipopolysaccharide and peptidoglycan. Bioinfomatic analysis of the E. chaffeensis genome, however, predicted genes encoding 15 lipoproteins and 3 posttranslational lipoprotein-processing enzymes. The present study showed that by use of multidimensional liquid chromatography followed by tandem mass spectrometry, all predicted li
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Dodds, P. F., A. Lopez-Johnston, V. A. Welch, and M. I. Gurr. "The effects of chemically modifying serum apolipoproteins on their ability to activate lipoprotein lipase." Biochemical Journal 242, no. 2 (1987): 471–78. http://dx.doi.org/10.1042/bj2420471.

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Lipoprotein lipase activity was measured in an acetone-dried-powder preparation from rat epididymal adipose tissue using pig serum or pig serum lipoprotein, which had been chemically modified, as activator. Modification of acidic amino acids of lipoproteins with NN-dimethyl-1,3-diamine resulted in a complete loss of ability to activate lipoprotein lipase. Modification of 34% of lipoprotein arginine groups with cyclohexanedione resulted in the loss of 75% of the activation of lipoprotein lipase; approx. 42% of the original activity was recovered after reversal of the modification. This effect w
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Giesecke, Yvonne, Samuel Soete, Katarzyna MacKinnon, et al. "Developing Electron Microscopy Tools for Profiling Plasma Lipoproteins Using Methyl Cellulose Embedment, Machine Learning and Immunodetection of Apolipoprotein B and Apolipoprotein(a)." International Journal of Molecular Sciences 21, no. 17 (2020): 6373. http://dx.doi.org/10.3390/ijms21176373.

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Plasma lipoproteins are important carriers of cholesterol and have been linked strongly to cardiovascular disease (CVD). Our study aimed to achieve fine-grained measurements of lipoprotein subpopulations such as low-density lipoprotein (LDL), lipoprotein(a) (Lp(a), or remnant lipoproteins (RLP) using electron microscopy combined with machine learning tools from microliter samples of human plasma. In the reported method, lipoproteins were absorbed onto electron microscopy (EM) support films from diluted plasma and embedded in thin films of methyl cellulose (MC) containing mixed metal stains, pr
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Faria, Eliana Cotta de, Adriana Celeste Gebrin, Wilson Nadruz Júnior, and Lucia Nassi Castilho. "Phospholipid transfer protein activity in two cholestatic patients." Sao Paulo Medical Journal 122, no. 4 (2004): 175–77. http://dx.doi.org/10.1590/s1516-31802004000400009.

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CONTEXT: Plasma phospholipid transfer protein mediates the transfer of phospholipids from triglyceride-rich lipoproteins, very low density lipoproteins and low density lipoproteins to high density lipoproteins, a process that is also efficient between high density lipoprotein particles. It promotes a net movement of phospholipids, thereby generating small lipid-poor apolipoprotein AI that contains particles and subfractions that are good acceptors for cell cholesterol efflux. CASE REPORT: We measured the activity of plasma phospholipid transfer protein in two cholestatic patients, assuming tha
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Renee Ruhaak, L., Arnoud van der Laarse, and Christa M. Cobbaert. "Apolipoprotein profiling as a personalized approach to the diagnosis and treatment of dyslipidaemia." Annals of Clinical Biochemistry: International Journal of Laboratory Medicine 56, no. 3 (2019): 338–56. http://dx.doi.org/10.1177/0004563219827620.

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An elevated low-density lipoprotein cholesterol concentration is a classical risk factor for cardiovascular disease. This has led to pharmacotherapy in patients with atherosclerotic heart disease or high heart disease risk with statins to reduce serum low-density lipoprotein cholesterol. Even in patients in whom the target levels of low-density lipoprotein cholesterol are reached, there remains a significant residual cardiovascular risk; this is due, in part, to a focus on low-density lipoprotein cholesterol alone and neglect of other important aspects of lipoprotein metabolism. A more refined
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Öörni, Katariina, Satu Lehti, Peter Sjövall, and Petri T. Kovanen. "Triglyceride-Rich Lipoproteins as a Source of Proinflammatory Lipids in the Arterial Wall." Current Medicinal Chemistry 26, no. 9 (2019): 1701–10. http://dx.doi.org/10.2174/0929867325666180530094819.

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Apolipoprotein B –containing lipoproteins include triglyceride-rich lipoproteins (chylomicrons and their remnants, and very low-density lipoproteins and their remnants) and cholesterol-rich low-density lipoprotein particles. Of these, lipoproteins having sizes below 70-80 nm may enter the arterial wall, where they accumulate and induce the formation of atherosclerotic lesions. The processes that lead to accumulation of lipoprotein-derived lipids in the arterial wall have been largely studied with a focus on the low-density lipoprotein particles. However, recent observational and genetic studie
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Dissertations / Theses on the topic "Lipoprotein A"

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Kohn, Meifania Monica. "Lipoprotein ontology: a formal representation of Lipoproteins." Thesis, Curtin University, 2013. http://hdl.handle.net/20.500.11937/1827.

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Lipoproteins serve as a mode of transport for the uptake, storage and metabolism of lipids. Dysregulation in lipoprotein metabolism, known as dyslipidaemia, is strongly correlated to various diseases such as cardiovascular disease. Lipoprotein Ontology provides a formal representation of lipoprotein concepts and relationships that can be used to support the intelligent retrieval of information, faciliate collaboration between research groups, and provide the basis for the development of tools for the diagnosis and treatment of dyslipidaemia.
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Soran, Handrean. "Glycation of Lipoproteins and the Role High Density Lipoprotein and Paraoxonase -1." Thesis, University of Manchester, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.532197.

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Espinosa, Garcia Irma Leticia. "Differential density lipoprotein profiling for the characterization of Lipoprotein(a)." Texas A&M University, 2006. http://hdl.handle.net/1969.1/4359.

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Lipoprotein(a) (Lp(a)) has been described as an emerging risk factor for cardiovascular disease. The complexity of the Lp(a) molecule sets a challenge for the determination of the risk it represents for the cardiovascular system. The objective of the present study was to develop a rapid method for the separation, purification, density measurement, and characterization of Lp(a) from serum using a procedure that is isoform independent. The objective was met by linking ultracentrifugation with affinity separations for the specific separation of Lp(a) from other lipoproteins. The mean density dist
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Hacquebard, Mirjam Rebecca. "Alpha-tocopherol acquisition by plasma lipoproteins and changes in lipoprotein profile after cardiac surgery." Doctoral thesis, Universite Libre de Bruxelles, 2008. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/216586.

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Alpha-tocopherol, the most abundant form of vitamin E in man, is transported in the circulation by plasma lipoproteins. It plays important roles, not only in preventing lipid peroxidation, but also in modulating several cell functions such as cell signaling and gene expression. While chylomicrons transport dietary alpha-tocopherol after intestinal absorption, LDL and HDL are the major carriers of alpha-tocopherol in fasting plasma and largely contribute to its delivery to cells and tissues. Exchanges of alpha-tocopherol occur between plasma lipoproteins. In addition, alpha-tocopherol transfers
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Ma, Feng. "Clinical Assessment of Anti-Atherogenic Function of HighDensity Lipoprotein (HDL)." Thesis, Sorbonne université, 2018. http://www.theses.fr/2018SORUS583.

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Il a été bien établi chez l’Homme qu'il existe une forte association entre les faibles concentrations plasmatiques de cholestérol associé aux lipoprotéines de haute densité (HDL-C) et le risque accru de maladie cardiovasculaire (MCV). Augmenter le taux de HDL-C a donc été proposé comme stratégie thérapeutique visant à réduire le risque de MCV. En effet, les HDL présentent de multiples fonctions athéroprotectrices, notamment la capacité d’efflux du cholestérol, ainsi que des activités antioxydantes, anti-inflammatoires, vasodilatatrices, cytoprotectrices, anti-infectieuses et anti- thrombotique
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Ooi, Esther M. M. "Regulation of lipoprotein transport in the metabolic syndrome : impact of statin therapy." University of Western Australia. School of Medicine and Pharmacology, 2007. http://theses.library.uwa.edu.au/adt-WU2007.0125.

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[Truncated abstract] The metabolic syndrome is characterized by cardiovascular risk factors including dyslipidemia, insulin resistance, visceral obesity, hypertension and diabetes. The dyslipidemia of the metabolic syndrome includes elevated plasma triglyceride and apolipoprotein (apo) B levels, accumulation of small, dense low-density lipoprotein (LDL) particles and low high-density lipoprotein (HDL) cholesterol concentration. However, the precise mechanisms for this dyslipoproteinemia, specifically low plasma HDL cholesterol, are not well understood. This thesis therefore, focuses on HDL, it
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Toledo, Júnior Alceu de Oliveira. "MARCADORES BIOQUÍMICOS NAS DISLIPIDEMIAS E NO RISCO CARDIOVASCULAR: ANÁLISE COMPARATIVA À FÓRMULA DE MARTIN." Universidade Estadual de Ponta Grossa, 2018. http://tede2.uepg.br/jspui/handle/prefix/2712.

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Submitted by Angela Maria de Oliveira (amolivei@uepg.br) on 2018-12-19T13:31:11Z No. of bitstreams: 2 license_rdf: 811 bytes, checksum: e39d27027a6cc9cb039ad269a5db8e34 (MD5) Alceu de Oliveira Toledo Junior.pdf: 3822777 bytes, checksum: 8621dbda843628c32379a4d6c54e0ac2 (MD5)<br>Made available in DSpace on 2018-12-19T13:31:11Z (GMT). No. of bitstreams: 2 license_rdf: 811 bytes, checksum: e39d27027a6cc9cb039ad269a5db8e34 (MD5) Alceu de Oliveira Toledo Junior.pdf: 3822777 bytes, checksum: 8621dbda843628c32379a4d6c54e0ac2 (MD5) Previous issue date: 2018-12-17<br>Este estudo tem como objetivo ava
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Gordon, Scott M. "The role of high density lipoprotein compositional and functional heterogeneity in metabolic disease." University of Cincinnati / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1353100684.

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Sledziecka, Anna Katarzyna. "Covalent lipoprotein(a) assembly : characterization of oxidase activity responsible for catalyzing covalent lipoprotein(a)." Kingston, Ont. : [s.n.], 2008. http://hdl.handle.net/1974/1596.

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Palm, Wilhelm, Marta M. Swierczynska, Veena Kumari, Monika Ehrhart-Bornstein, Stefan R. Bornstein, and Suzanne Eaton. "Secretion and Signaling Activities of Lipoprotein-Associated Hedgehog and Non-Sterol-Modified Hedgehog in Flies and Mammals." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2015. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-180911.

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Hedgehog (Hh) proteins control animal development and tissue homeostasis. They activate gene expression by regulating processing, stability, and activation of Gli/Cubitus interruptus (Ci) transcription factors. Hh proteins are secreted and spread through tissue, despite becoming covalently linked to sterol during processing. Multiple mechanisms have been proposed to release Hh proteins in distinct forms; in Drosophila, lipoproteins facilitate long-range Hh mobilization but also contain lipids that repress the pathway. Here, we show that mammalian lipoproteins have conserved roles in Sonic Hedg
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Books on the topic "Lipoprotein A"

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Jayme, Borensztajn, ed. Lipoprotein lipase. Evener Publishers, 1987.

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Hendriks, Wilhelmina Leonie. Lipoprotein lipase-mediated interactions of lipoproteins with macrophages. University of Leiden, 1998.

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1924-, Scanu Angelo M., ed. Lipoprotein (a). Academic Press, 1990.

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Ordovas, Jose M. Lipoprotein Protocols. Humana Press, 1998. http://dx.doi.org/10.1385/1592595820.

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A, Converse Carolyn, and Skinner E. Roy, eds. Lipoprotein analysis. IRL Press at Oxford University Press, 1992.

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M, Ordovas J., ed. Lipoprotein protocols. Humana Press, 1998.

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James, Shepherd, ed. Lipoprotein metabolism. Baillie re, 1987.

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Kostner, Karam, Gerhard M. Kostner, and Peter P. Toth, eds. Lipoprotein(a). Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-24575-6.

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Angel, Aubie, and Jiri Frohlich, eds. Lipoprotein Deficiency Syndromes. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-1262-8.

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International Conference on Lipoprotein Deficiency Syndromes (1985 Vancouver, B.C.). Lipoprotein deficiency syndromes. Plenum Press, 1986.

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Book chapters on the topic "Lipoprotein A"

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Bährle-Rapp, Marina. "Lipoprotein." In Springer Lexikon Kosmetik und Körperpflege. Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-71095-0_6064.

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Harlapur, Manjunath, and Daichi Shimbo. "Lipoprotein." In Encyclopedia of Behavioral Medicine. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39903-0_1276.

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Malik, Jamil A., Theresa A. Morgan, Falk Kiefer, et al. "Lipoprotein." In Encyclopedia of Behavioral Medicine. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_1276.

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Harlapur, Manjunath, and Daichi Shimbo. "Lipoprotein." In Encyclopedia of Behavioral Medicine. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4614-6439-6_1276-2.

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Fruchart, J. C., and G. Luc. "Lipoprotein Oxidation." In Lipid-Soluble Antioxidants: Biochemistry and Clinical Applications. Birkhäuser Basel, 1992. http://dx.doi.org/10.1007/978-3-0348-7432-8_43.

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Nallamshetty, Shriram, Jorge Plutzky, and Jorge Plutzky. "Lipoprotein Disorders." In MGH Cardiology Board Review. Springer London, 2013. http://dx.doi.org/10.1007/978-1-4471-4483-0_6.

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Sorrentino, Matthew J. "Lipoprotein(a)." In Hyperlipidemia in Primary Care. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-60327-502-6_10.

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Rader, Daniel J., and Sumeet A. Khetarpal. "Lipoprotein Physiology." In Dyslipidemias. Humana Press, 2015. http://dx.doi.org/10.1007/978-1-60761-424-1_1.

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Enkhmaa, Byambaa, Erdembileg Anuurad, Wei Zhang, and Lars Berglund. "Lipoprotein(a)." In Dyslipidemias. Humana Press, 2015. http://dx.doi.org/10.1007/978-1-60761-424-1_3.

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Bock, H. H., P. May, and J. Herz. "Lipoprotein Transport." In Transgenic Models in Pharmacology. Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-642-18934-0_14.

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Conference papers on the topic "Lipoprotein A"

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Abu-Safe, Husam H. "Probing the Oxidation levels of Low-Density Lipoprotein Using the Z-scan Technique." In Frontiers in Optics. Optica Publishing Group, 2024. https://doi.org/10.1364/fio.2024.jtu5a.65.

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The oxidation levels of low-density lipoprotein (LDL) using the z-scan method was investigated. The LDL samples were oxidized using copper sulfate. The amplitude of the z-scan signal modeled the changes in the oxidization levels.
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Chen, Kaili, Le Gao, Yue Hou, Ling Liu, Qingyuan Huang, and Yunxuan Guo. "Study on the Detection Method of Lipoprotein Phospholipase A2 Based on Nanomicrospheres." In 2024 IEEE International Conference on Manipulation, Manufacturing and Measurement on the Nanoscale (3M-NANO). IEEE, 2024. https://doi.org/10.1109/3m-nano61605.2024.10769659.

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Yu, Tingting, Weiru Bao, and Guiyin Li. "A colourimetric sensing based on MXene@Hemin nanoenzyme for the rapid detection of low-density lipoprotein." In 6th International Conference on Optoelectronic Science and Materials (ICOSM 2024), edited by Yuan Lu, Yonghong Wang, and Chongwen Zou. SPIE, 2024. https://doi.org/10.1117/12.3052640.

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Koller, E., and F. Koller. "LIPOPROTEIN BINDING TOHUMAN PLATELETS IS LOCATED AT GPIIb/IIIa COMPLEX." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643702.

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Human Platelets possess specific binding sites for low density lipoproteins (LDL) and high density lipoproteins(HDL)(1). Binding of both classes of plasma lipoproteins, though competitive, has been shown by several groups to facilitate platelet activation.Isolated washed platelets occasionally aggregate upon addition of high concentrations of LDL even in the absence of known platelet activators. The proteins responsible for this binding have been visualized by ligand blotting (2). Both types of ligand specifically bind to two glycoproteins with molecular weights of 135 and 115 kD, respectively
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FATHIL, Noor Mahdi. "EFFECT OF THE ENERGY DRINK (TIGER) ON THE PARAMETERS OF LIPID PROFILEIN THE FEMALE ALBINO MICE." In III.International Scientific Congress of Pure,Appliedand Technological Sciences. Rimar Academy, 2021. http://dx.doi.org/10.47832/minarcongress3-5.

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The aim of research is to clarify effect of an energy drink (Tiger) on Lipid profile physiological parameters in female mice for a period of four weeks. Adults female mice were used in research and divide to two groups. The first group is the control group that give distilled water (D.w.) for four weeks and the second group was treated group with tiger concentration dose 1.5 ml/mg for period of four weeks. After the end of the dosing period, sacrifices animals and the blood samples are collected without anticoagulant, and blood serum is obtained and kept at− 20 °C for biochemical tests. The re
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Damirchi, Behzad, Amir Rouhollahi, Salman Sohrabi, and Seyyed Mahdi Nemati Mehr. "Modeling and Stability Analysis of Truncated High Density Lipoprotein (HDL) System Using Martini Coarse Grain Technique." In ASME 2013 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/imece2013-64808.

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Lipoproteins are biochemical compounds containing both proteins and lipids. These particles carry chemicals like cholesterol and triglycerides that are not soluble in aqueous solutions. This paper presents modeling of lipoprotein system using coarse grain molecular dynamics technique and stability analysis of this system in a water solution like blood. A high density lipoprotein (HDL) that consists of two annular monomers is modeled. Also there are lipid bilayers located in center of the rings, so the whole HDL and lipid bilayers are called lipoprotein system. First, all atom model is provided
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Hubbard, A. R., and C. A. Jennings. "TISSUE FACTOR-FACTOR VII INHIBITION REQUIRES FACTOR Xa AND PLASMA LIPOPROTEINS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643291.

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Tissue factor rapidly loses procoagulant activity when incubated with defibrmated normal plasma and calcium ions. Inhibition is apparently directed against the tissue factor-Factor VII complex (TF-EVII) and requires Factor Xa and a component(s) found in A1 (OH)3-adsorbed plasma (AP). We have developed a two stage assay for the inhibitor which involves first, the incubation of a TF-FVII complex with test material in the presence of Factor Xa, followed by the amidolytic assay of residual TF-EVII activity.Our studies have indicated that the component of AP responsible for this effect is lipoprote
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Palvinskaya, Tatsiana, Christopher Lenox, MaryEllen Antkowiak, et al. "Low Density Lipoprotein Activate Neutrophils." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4349.

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Ittrich, H., O. Bruns, A. Bartelt, et al. "In-vivo MR imaging of lipoprotein distribution and metabolism using spio-labeled lipoproteins at 3T." In 2013 International Workshop on Magnetic Particle Imaging (IWMPI). IEEE, 2013. http://dx.doi.org/10.1109/iwmpi.2013.6528361.

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Kuemmerle, Nancy Benton, Leslie E. Lupien, Nicole C. Smits, Wilson L. Davis, and William B. Kinlaw. "Abstract 5607: Lipoprotein lipase binds to the surface of cancer cells and facilitates uptake of lipoproteins." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-5607.

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Reports on the topic "Lipoprotein A"

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Kahl, S. B. Low density lipoprotein development and evaluation. Office of Scientific and Technical Information (OSTI), 1995. http://dx.doi.org/10.2172/421327.

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Hinshaw, Jerald C. Synthesis of Lipoprotein Immunostimulants for Treating Prostate Cancer. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada434134.

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Hinshaw, Jerald C. Synthesis of Lipoprotein Immunostimulants for Treating Prostate Cancer. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada423265.

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Pang, Ying, and Chi Yi. Lipoprotein(a) and the risk of diabetic retinopathy. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2024. http://dx.doi.org/10.37766/inplasy2024.8.0114.

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Hung, Hsuan-Yu, Hui-Hsiung Lai, Hui-Chuan Lin, and Chung-Yu Chen. Impact of interferon-free antivirus therapy on lipid profiles in patients with chronic hepatitis C: A network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.7.0055.

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Abstract:
Review question / Objective: P: ("Hepatitis C"[Mesh] AND "Hepacivirus"[Mesh] AND "Hepatitis C, Chronic”[Mesh]) I: (direct acting antiviral OR asunaprevir OR boceprevir OR daclatasvir OR dasabuvir OR elbasvir OR glecaprevir OR grazoprevir OR ledipasvir OR ombitasvir OR paritaprevir OR pibrentasvir OR simeprevir OR sofosbuvir OR telaprevir OR velpatasvir OR voxilaprevir) C: placebo O: ( "Cholesterol, VLDL"[Mesh] OR "Cholesterol, LDL"[Mesh] OR "Cholesterol, HDL"[Mesh] OR "Dyslipidemias"[Mesh] OR "lipoprotein cholesterol ester, human" [Supplementary Concept] OR "lipoprotein cholesterol" [Supplemen
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Rubin, E. M., and A. S. Plump. The use of transgenic animals to study lipoprotein metabolism. Office of Scientific and Technical Information (OSTI), 1993. http://dx.doi.org/10.2172/102282.

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Lacko, Andras G. High Density Lipoprotein Complexes as Delivery Vehicles for Breast Cancer. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada416984.

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Lacko, Andras G. High Density Lipoprotein Complexes as Delivery Vehicles for Breast Cancer Chemotherapy. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada408103.

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Zhang, Wei-Yang. Studies on the Formation of Murein-Bound Lipoprotein in Escherichia coli. Defense Technical Information Center, 1992. http://dx.doi.org/10.21236/ad1011159.

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Krauss, R. M., and D. M. Dreon. Low density lipoprotein subclasses and response to a low-fat diet in healthy men. Office of Scientific and Technical Information (OSTI), 1994. http://dx.doi.org/10.2172/41265.

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