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Journal articles on the topic 'Lipotrope'

Author: Grafiati

Published: 4 May 2024

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1

Fardet, Anthony, Jean-François Martin, and Jean-Michel Chardigny. "Lipotropic capacity of raw plant-based foods: A new index that reflects their lipotrope density profile." Journal of Food Composition and Analysis 24, no. 7 (2011): 895–915. http://dx.doi.org/10.1016/j.jfca.2011.03.013.

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2

Moon, Yang S., Wanda L. Keller, and Chung S. Park. "Dietary lipotrope-mediated mammary carcinogenesis in female rats." Nutrition Research 18, no. 9 (1998): 1605–14. http://dx.doi.org/10.1016/s0271-5317(98)00134-1.

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3

Rogers, Adrianne E., Rizwan Akhtar, and Steven H. Zeisel. "Procarbazine carcinogenicity in methotrexate-treated or lipotrope-deficient male rats." Carcinogenesis 11, no. 9 (1990): 1491–95. http://dx.doi.org/10.1093/carcin/11.9.1491.

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4

Choi, Chang B., Myung G. Baik, Wanda L. Keller, and Chung S. Park. "Lipotrope‐modified diets enhance nitrosomethylurea‐induced mammary carcinogenesis in female rats." Nutrition and Cancer 20, no. 3 (1993): 215–21. http://dx.doi.org/10.1080/01635589309514289.

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5

Wainfan, Elsie, Mark Dizik, Margaret Hluboky, and M. Earl Balis. "Altered tRNA methylation in rats and mice fed lipotrope-deficient diets." Carcinogenesis 7, no. 3 (1986): 473–76. http://dx.doi.org/10.1093/carcin/7.3.473.

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6

Moon, Y. S., C. S. Park, and S. H. Kim. "Lipotrope-mediatedc-jun and ornithine decarboxylase mRNA expression in mammary acinar cells." In Vitro Cellular & Developmental Biology - Animal 32, no. 6 (1996): 313–14. http://dx.doi.org/10.1007/bf02722954.

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7

van Helden, Paul D., Albertus D. Beyers, André J. Bester, and Kasimir Jaskiewicz. "Esophageal cancer: Vitamin and lipotrope deficiencies in an at‐risk South African population." Nutrition and Cancer 10, no. 4 (1987): 247–55. http://dx.doi.org/10.1080/01635588709513962.

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8

Saito, Reisuke, Elizabeth Jahnke-Spinnenweber, Hisashi Shinozuka, and Benito Lombardi. "On the role of compensatory mitogenesis in the hepatocarcinogencity of choline and multiple-lipotrope devoid diets." Carcinogenesis 15, no. 7 (1994): 1413–19. http://dx.doi.org/10.1093/carcin/15.7.1413.

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9

Trimble, Kenneth C., Anne M. Molloy, John M. Scot, and Donald G. Weir. "The effect of ethanol on one-carbon metabolism: Increased methionine catabolism and lipotrope methyl-group wastage." Hepatology 18, no. 4 (1993): 984–89. http://dx.doi.org/10.1002/hep.1840180433.

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10

Boyd, Juanell N., Elizabeth S. Graham, Thomas C. Graham, and Bud C. Tennant. "A Comparison of the Response of Woodchucks and Rats to Variations in Dietary Lipotrope and Lipid Content." Journal of Nutrition 115, no. 9 (1985): 1136–46. http://dx.doi.org/10.1093/jn/115.9.1136.

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11

Boyd, Juanell N., Wendy K. Sherman, Elizabeth S. Graham, Thomas C. Graham, and Bud C. Tennant. "A Comparison of the Response of Woodchucks and Rats to Variations in Dietary Lipotrope and Protein Content." Journal of Nutrition 116, no. 10 (1986): 2044–54. http://dx.doi.org/10.1093/jn/116.10.2044.

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12

Wu, Dongming, Wanda L. Keller, and Chung S. Park. "Lipotrope deficiency inhibits cell growth and induces programmed cell death in human breast cancer cell line MCF‐7." Nutrition and Cancer 32, no. 1 (1998): 13–19. http://dx.doi.org/10.1080/01635589809514710.

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13

CHRISTMAN, J., M. CHEN, G. SHEIKHNEJAD, M. DIZIK, S. ABILEAH, and E. WAINFAN. "Methyl deficiency, DNA methylation, and cancer: Studies on the reversibility of the effects of a lipotrope-deficient diet☆☆☆." Journal of Nutritional Biochemistry 4, no. 12 (1993): 672–80. http://dx.doi.org/10.1016/0955-2863(93)90106-7.

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14

Andry, Christopher D., Herbert Z. Kupchik, and Adrianne E. Rogers. "L-Azaserine Induced Preneoplasia in the Rat Pancreas. A Morphometric Study of Dietary Manipulation (Lipotrope Deficiency) and Ultrastructural Differentiation." Toxicologic Pathology 18, no. 1 (1990): 10–17. http://dx.doi.org/10.1177/019262339001800102.

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15

Helman, R. G., L. G. Adams, and C. H. Bridges. "The Lesions of Hepatic Fatty Cirrhosis in Sheep." Veterinary Pathology 32, no. 6 (1995): 635–40. http://dx.doi.org/10.1177/030098589503200604.

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During a natural outbreak of hepatic fatty cirrhosis (HFC) in western Texas, 500 2-6-year-old Rambouillet ewe sheep were sequentially studied to determine the pattern of lesion development. All sheep developed lesions of HFC. Grossly, changes first began in the subcapsular hepatic parenchyma along the porta hepatis and spread peripherally until, in the final stages of the disease, approximately 80% of the liver was affected. Ascites, hydropericardium, and acquired hepatic vascular shunts were present in sheep with severe HFC. Light microscopic lesions initially appeared as accumulations of fin

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16

BEST, C. H. "The Lipotropic Factors1." Acta Medica Scandinavica 131, no. 6 (2009): 503–14. http://dx.doi.org/10.1111/j.0954-6820.1948.tb03430.x.

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17

Petraglia, F., C. Barletta, F. Facchinetti, et al. "Response of circulating adrenocorticotropin, beta-endorphin, beta-lipotropin and cortisol to athletic competition." Acta Endocrinologica 118, no. 3 (1988): 332–36. http://dx.doi.org/10.1530/acta.0.1180332.

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Abstract. Acute physical exercise stimulates the activity of the hypothalamus-pituitary-adrenal axis in man. In the present study we measured plasma adrenocorticotropin, beta-endorphin, beta-lipotropin and cortisol levels in 27 male trained athletes in basal conditions, 60 min before and immediately after an official competition. The endocrine responses were evaluated in different groups of athletes participating in races (100 m, 1500 m, 10 000 m) or in the disc throw. The athletes competing for the runs showed a statistically significant increase in plasma adrenocorticotropin, beta-endorphin,

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18

Sukhova, A. V., V. A. Kiryakov, and I. A. Bogatyreva. "Efficiency of systemic therapy of vibration disease." Occupational Health and Industrial Ecology, no. 9 (February 12, 2019): 53–58. http://dx.doi.org/10.31089/1026-9428-2018-9-53-58.

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The authors studied antioxidant therapy in complex preventive treatment of vibration disease. Findings are high therapeutic efficiency of thioctic acid — better lipid and carbohydrates metabolism parameters, endoneural circulation due to antioxidant, membrane protective, lipotropic and hypoglycemic eﬀects.

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19

Orlova, S. V., and E. A. Nikitina. "The role of lipotropic factors in the correction of metabolic disorders in women during the perimenopausal period." Medical alphabet, no. 19 (November 5, 2023): 7–15. http://dx.doi.org/10.33667/2078-5631-2023-19-7-15.

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In women during perimenopause, changes in metabolic parameters are detected, such as weight gain, which triggers a cascade of pathological reactions leading to the formation of metabolic disorders (lipid, carbohydrate metabolism) and endothelial dysfunction, during the development of which a deficiency of sex hormones plays a certain role in the onset of menopause. There is a connection between hormonal changes and an increase in the amount of abdominal and/or visceral fat, which is accompanied by a weakening of oxidative processes in adipose tissue, a decrease in energy consumption and a pred

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20

O'Connell, Yvonne, T. Joseph McKenna та Sean K. Cunningham. "β-Lipotropin-stimulated adrenal steroid production". Steroids 61, № 5 (1996): 332–36. http://dx.doi.org/10.1016/0039-128x(96)00002-5.

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21

Kravchenko, V. M., Z. V. Shovkova, I. V. Senyuk, and O. V. Shovkova. "The study of lipotropic action of extract from fruit prunus domestica «Prunofit» byanimal models of alcoholic liver diseases." Farmatsevtychnyi zhurnal, no. 6 (December 9, 2020): 86–91. http://dx.doi.org/10.32352/0367-3057.6.20.09.

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Searchingof drugs that normalize the function of the digestive glands are important because disruption of digestion underlie the pathogenesis of many diseases such as gastroesophageal reflux disease, hepatitis, gastric ulcer, irritable bowel syndrome, cancer etc. The use of herbal objects containing fibres is a promising direction for solving of the mentioned problem. We were attracted by fruits of Prunus domestica which are rich in fibres (homo- and heteropolsaccharides) and are used in folk medicine as a laxative and hepatoprotective agent. The aim of this experimental study was to inv

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22

Moss, I. R., J. G. Inman, and C. A. Slaughter. "Posttranslational processing of opioid pro-opiomelanocortin products in piglets." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 256, no. 2 (1989): R429—R434. http://dx.doi.org/10.1152/ajpregu.1989.256.2.r429.

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We have measured levels of beta-lipotropin, beta-endorphin, and N-acetyl-beta-endorphin in the plasma, cerebrospinal fluid (CSF), and caudal medullary brain containing the respiratory-related portion of the nucleus tractus solitarius (NTS) of 2.5 +/- 1.0- (SD) and 38.2 +/- 3.7-day-old naive uninstrumented piglets. Time of day, ambient atmosphere, temperature, handling, sound, light, and nutritional status were kept constant. Experimental procedure included decapitation and rapid collection, processing, and freezing of tissues until analysis by radioimmunoassay. Young, compared with older pigle

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23

Bauduin, P., F. Testard, and Th Zemb. "Solubilization in Alkanes by Alcohols as Reverse Hydrotropes or “Lipotropes”." Journal of Physical Chemistry B 112, no. 39 (2008): 12354–60. http://dx.doi.org/10.1021/jp804668n.

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24

Wu, D. "Lipotropes alter casein gene expression in bovine mammary acinar culture." Journal of Nutritional Biochemistry 10, no. 8 (1999): 455–57. http://dx.doi.org/10.1016/s0955-2863(99)00028-5.

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25

Mabasa, L., K. Cho, S. Bae, D. M. Walsh, P. Asija, and C. S. Park. "Lipotropes (methyl nutrients) inhibit growth of feline lymphoma in vitro." Research in Veterinary Science 93, no. 1 (2012): 259–63. http://dx.doi.org/10.1016/j.rvsc.2011.05.015.

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26

Knigge, Ulrich, Steen Matzen, Flemming W. Bach, Peter Bang, and Jørgen Warberg. "Involvement of histaminergic neurons in the stress-induced release of pro-opiomelanocortin-derived peptides in rats." Acta Endocrinologica 120, no. 4 (1989): 533–39. http://dx.doi.org/10.1530/acta.0.1200533.

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Abstract. Histamine, which acts as a neurotransmitter, stimulates the release of the pro-opiomelanocortin derived peptides ACTH, β-lipotropin, and β-endorphin. Since stress affects the hypothalamic turn-over of neuronal histamine, we investigated the role of histaminergic neurons in the mediation of the stress-induced release of ACTH and β-endorphin immunoreactivity in male rats. In control animals histamine receptor antagonists had no effect on the release of ACTH or β-endorphin immunoreactivity. Restraint and ether stress increased plasma ACTH 3- and 2-fold, respectively. The responses were

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27

Chrétien, M., C. Gilardeau, and Choh Hao Li. "REVISED STRUCTURE OF SHEEP BETA-LIPOTROPIC HORMONE." International Journal of Peptide and Protein Research 4, no. 4 (2009): 263–65. http://dx.doi.org/10.1111/j.1399-3011.1972.tb03427.x.

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28

Rao, A. Jagannadha, та Choh Hao Li. "IMMUNOCHEMICAL INVESTIGATIONS OF HUMAN PITUITARY β-LIPOTROPIN". International Journal of Peptide and Protein Research 10, № 2 (2009): 167–71. http://dx.doi.org/10.1111/j.1399-3011.1977.tb02791.x.

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29

IZDEBSKI, JAN, DONALD YAMASHIRO, CHOH HAO LI та GIOVANNI VITI. "Synthesis and properties of human γ-lipotropin". International Journal of Peptide and Protein Research 20, № 1 (2009): 87–92. http://dx.doi.org/10.1111/j.1399-3011.1982.tb02657.x.

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30

Matthews, John. "β-Endorphin/β-Lipotropin Immunoreactivity in Endogenous Depression". Archives of General Psychiatry 43, № 4 (1986): 374. http://dx.doi.org/10.1001/archpsyc.1986.01800040084012.

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31

Orlov, Ye V., P. Ye Konnov, and Ye V. Kotelnikova. "Assessment of the clinical efficacy of the administration of Phosphogliv in complex treatment of psoriasis patients." Vestnik dermatologii i venerologii 86, no. 4 (2010): 69–72. http://dx.doi.org/10.25208/vdv926.

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The article presents the results of a study of the clinical efficacy of the administration of Phosphogliv for psoriasis and its effect on the functional and biochemical parameters as well as patients quality of life. The authors established that the administration of Phosphogliv results in a considerable improvement of the course of extensive psoriasis and reduction in the activity of the inflammatory process contributing to the normalization of the lipid composition of the blood serum at the expense of the lipotropic and membrane stabilizing action.

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32

Semenenko, Marina P., Tatyana A. Sakhno, Elena V. Kuzminova, and Aleksey V. Savinkov. "Endogenic intoxication as a reflection of metabolic disorders in cows and possibilities of its pharmacological correction." BIO Web of Conferences 27 (2020): 00029. http://dx.doi.org/10.1051/bioconf/20202700029.

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The article presents the results of studies of medium mass molecules concentration in the blood serum of cows with different physiological conditions and correction of this state with livazen, which has an antioxidant and lipotropic effect on liver cells. It was determined that the use of livazen contributed to a decrease in cholesterol in the group of pregnant cows by 21.0 % and in the group of fresh cows by 15.9 %, with a decrease in the concentration of average molecular peptides by 4.0-11.8 % depending on the length of exposure.

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33

PANOUSIS (Ν. ΠΑΝΟΥΣΗΣ), N., and H. KARATZIAS (Χ. ΚΑΡΑΤΖΙΑΣ). "Postparturient liver diseases of dairy cattle (Ketosis, fatty liver). II. Management principles." Journal of the Hellenic Veterinary Medical Society 51, no. 4 (2018): 249. http://dx.doi.org/10.12681/jhvms.15682.

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Two of the commonest in practice postparturient liver diseases of dairy cows, ketosis and fatty liver, are discussed in this paper. It is not uncommon for both diseases to occur simultaneously, or each to act as a predisposing factor for the other. Clinical findings are nonspecific and certain biochemical parameters can be used as diagnostic tools. Treatment is focused in restoring glucose and energy balance of the affected animals with the administration of glucose, glucogenic substances, corticosteroids, insulin and lipotropic agents. Prevention aims in proper herd management, especially in

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34

RICHTER, W. O., та P. SCHWANDT. "Cellular mechanisms of β-lipotropin-mediated stimulation of lipolysis". Acta Endocrinologica 113, № 1_Suppl (1986): S158—S159. http://dx.doi.org/10.1530/acta.0.111s158.

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35

Chrétien, Michel, and Majambu Mbikay. "60 YEARS OF POMC: From the prohormone theory to pro-opiomelanocortin and to proprotein convertases (PCSK1 to PCSK9)." Journal of Molecular Endocrinology 56, no. 4 (2016): T49—T62. http://dx.doi.org/10.1530/jme-15-0261.

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Pro-opiomelanocortin (POMC), is a polyprotein expressed in the pituitary and the brain where it is proteolytically processed into peptide hormones and neuropeptides with distinct biological activities. It is the prototype of multipotent prohormones. The prohormone theory was first suggested in 1967 when Chrétien and Li discovered γ-lipotropin and observed that (i) it was part of β-lipotropin (β-LPH), a larger polypeptide characterized 2 years earlier and (ii) its C-terminus was β-melanocyte-stimulating hormone (β-MSH). This discovery led them to propose that the lipotropins might be related bi

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36

Knigge, Ulrich, Flemming W. Bach, Steen Matzen, Peter Bang, and Jørgen Warberg. "Effect of histamine on the secretion of pro-opiomelanocortin derived peptides in rats." Acta Endocrinologica 119, no. 2 (1988): 312–19. http://dx.doi.org/10.1530/acta.0.1190312.

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Abstract. In conscious male rats intracerebroventricular infusion of histamine increased the plasma concentrations of ACTH and β-endorphin immunoreactivity 2.5-fold (P < 0.01). Gel filtration of plasma revealed two peaks of β-endorphin immunoreactivity corresponding to β-endorphin and β-lipotropin. The two fractions increased almost equally in histamine-stimulated animals, whereas most of the circulating β-endorphin immunoreactivity in control animals corresponded to β-endorphin. Central infusion of the H1-receptor agonist 2-thiazolylethylamine and of the H2-receptor agonists dimaprit or 4-

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37

Anugrah, Dian, Andi Magfira Satya Apada, Fedri Rell, Rian Hari Suharto, and Muhammad Fadhlullah Mursalim. "Treatment Leptospira Infection In Dog At Pet Vet Clinic, Center of Jakarta." Media Kedokteran Hewan 34, no. 3 (2023): 189–97. http://dx.doi.org/10.20473/mkh.v34i3.2023.189-197.

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Leptospirosis is a disease caused by Leptospira spp., zoonotic Gram-negative bacteria. This study aimed to determine the handling of a leptospirosis case in a dog at the Pet Vet Clinic, Jakarta. Handling began with anamnesis and continued by examining the patient's physical condition, followed by serological, microscopic, and polymerase chain reaction examinations. An examination revealed that the dog had Leptospirosis. The therapy given was the administration of crystalline procaine and penicillin-G antibiotics. Supportive treatment was given with hepatoprotective, lipotropic, and diuretic su

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38

Facchinetti, Fabio, Grazia Sances, Charissula Bakalakis, et al. "Neuroendocrine Effects of Flunarizine Treatment in Postmenopausal Women." Cephalalgia 5, no. 2_suppl (1985): 187–90. http://dx.doi.org/10.1177/03331024850050s237.

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Flunarizine (10 mg/day for 60 days) was given to eight postmenopausal women with common migraine. Plasma LH pulsatility fluctuation, peripheral concentrations of prolactin (PRL), cortisol, β-endorphin (β-EP), β-lipotropin (β-LPH) and Pain Total Index (PTI) were evaluated before and after treatment. PTI was significantly reduced by flunarizine, which did not affect β-LPH, β-EP and cortisol plasma levels. On the contrary, both PRL values and amplitude, and length of LH pulses had increased at the end of treatment. Flunarizine reduced head pain in postmenopausal women. However, the enhancement of

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39

Smyth, D. G. "60 YEARS OF POMC: Lipotropin and beta-endorphin: a perspective." Journal of Molecular Endocrinology 56, no. 4 (2016): T13—T25. http://dx.doi.org/10.1530/jme-16-0033.

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Many important fields of research had a humble origin. In the distant past, A J P Martin’s discovery that amino acids could be separated by paper chromatography and Moore and Stein’s use of columns for quantitative amino acid analysis provided the first steps towards the determination of structure in complex biologically active molecules. They opened the door to reveal the essential relationship that exists between structure and function. In molecular endocrinology, for example, striking advances have been made by chemists with their expertise in the identification of structure working with bi

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40

LOBSTEIN, DENNIS D., and A. H. ISMAIL. "Decreases in resting plasma beta-endorphin/-lipotropin after endurance training." Medicine & Science in Sports & Exercise 21, no. 2 (1989): 161???166. http://dx.doi.org/10.1249/00005768-198904000-00008.

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41

RICHTER, W. O., та P. SCHWANDT. "Degradation of β-lipotropin and β-endorphin by human plasma". Acta Endocrinologica 116, № 3_Suppl (1987): S69—S70. http://dx.doi.org/10.1530/acta.0.114s069.

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42

JACOB, B. G., W. O. RICHTER та P. SCHWANDT. "Degradation of β-lipotropin by subcellular fractions of rabbit adipocytes". Acta Endocrinologica 117, № 4_Suppl (1988): S110. http://dx.doi.org/10.1530/acta.0.117s110.

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43

LI, CHOH HAO, M. M. J. OOSTHUIZEN та DAVID CHUNG. "Isolation and primary structures of elephant adrenocorticotropin and β-lipotropin". International Journal of Peptide and Protein Research 32, № 6 (2009): 573–78. http://dx.doi.org/10.1111/j.1399-3011.1988.tb01389.x.

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44

Richter, W. O., та P. Schwandt. "Physiologic concentrations of β-lipotropin stimulate lipolysis in rabbit adipocytes". Metabolism 34, № 6 (1985): 539–43. http://dx.doi.org/10.1016/0026-0495(85)90191-x.

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45

Rhew, Tae H., and Dileep S. Sachan. "Dose-Dependent Lipotropic Effect of Carnitine in Chronic Alcoholic Rats." Journal of Nutrition 116, no. 11 (1986): 2263–69. http://dx.doi.org/10.1093/jn/116.11.2263.

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46

Kumar, Neeraj, Subodh Gupta, Nitish Kumar Chandan, Md Aklakur, Asim Kumar Pal, and Sanjay Balkrishna Jadhao. "Lipotropes Protect against Pathogen-Aggravated Stress and Mortality in Low Dose Pesticide-Exposed Fish." PLoS ONE 9, no. 4 (2014): e93499. http://dx.doi.org/10.1371/journal.pone.0093499.

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47

Mabasa, Lawrence, Kyongshin Cho, Woo-Sik Choi, et al. "Suppression of Mammary Carcinogenesis Through Early Exposure to Dietary Lipotropes Occurs Primarily In Utero." Nutrition and Cancer 67, no. 8 (2015): 1278–84. http://dx.doi.org/10.1080/01635581.2015.1087039.

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48

Muthappa, N. A., Subodh Gupta, Sona Yengkokpam, et al. "Lipotropes promote immunobiochemical plasticity and protect fish against low-dose pesticide-induced oxidative stress." Cell Stress and Chaperones 19, no. 1 (2013): 61–81. http://dx.doi.org/10.1007/s12192-013-0434-y.

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49

Milgram, S. L., and R. E. Mains. "Differential effects of temperature blockade on the proteolytic processing of three secretory granule-associated proteins." Journal of Cell Science 107, no. 3 (1994): 737–45. http://dx.doi.org/10.1242/jcs.107.3.737.

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Vesicular transport within the secretory pathway can be arrested by incubating cells at 15 degrees C or 20 degrees C to block exit from the endoplasmic reticulum or trans-Golgi network, respectively. Using this powerful tool we have compared the intracellular sites of endoproteolytic processing of proopiomelanocortin and two prohormone processing enzymes in AtT-20 mouse pituitary corticotrope tumor cells. For comparison, proopiomelanocortin processing was also evaluated in primary neurointermediate pituitary cultures. AtT-20 cells synthesize and store endogenous proopiomelanocortin and prohorm

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50

Pavlovskyi, S. "EFFICIENCY OF COMPLEX PATHOGENIC APPROACH FOR TREATMENT OF PATIENTS WITH NONALCOHOLIC STEATOHEPATITIS COMBINED WITH TYPE 2 DIABETES MELLITUS." Ukrainian Scientific Medical Youth Journal 106, no. 2 (2018): 32–36. http://dx.doi.org/10.32345/usmyj.2(106).2018.32-36.

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Aim is to optimize treatment of non-alcoholic fatty liver disease (NAFLD) combined with type 2 diabetes mellitus. 100 patients with NAFLD in stage of non-alcoholic steatohepatitis (NASH) were enrolled into study. Application of S-adenosylmethionine during the treatment of patients with non-alcoholic fatty liver disease (NAFLD) had high efficacy in the normalization of the functional, biochemical and cytokine profile of the blood. Combination of gliclizide and pioglitazone led to the decrease in the levels of cytolysis and mesenchymal-inflammatory syndromes, stabilization of lipid metabolism. T

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