Relevant bibliographies by topics / Liver sinusoidal endothelial cell

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Liver sinusoidal endothelial cell'

Author: Grafiati
Published: 4 June 2021
Last updated: 21 February 2023

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Journal articles on the topic "Liver sinusoidal endothelial cell"

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Couvelard, A., JY Scoazec, MC Dauge, AF Bringuier, F. Potet, and G. Feldmann. "Structural and functional differentiation of sinusoidal endothelial cells during liver organogenesis in humans." Blood 87, no. 11 (June 1, 1996): 4568–80. http://dx.doi.org/10.1182/blood.v87.11.4568.bloodjournal87114568.

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During fetal life, human liver sinusoids, which differentiate between 4 and 12 weeks of gestation from capillaries of the septum transversum, must support an important hematopoietic function and acquire the structural and functional characteristics of adult sinusoids. To gain insight into their differentiation process, we studied the expression of (1) markers of continuous endothelia, absent from adult sinusoidal endothelial cells (PECAM-1, CD34, and 1F10); (2) functional markers of adult sinusoidal endothelial calls (CD4, 1CAM-1, CD32, and CD14); and (3) extracellular matrix components (lamin
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Maretti-Mira, Ana, and Laurie DeLeve. "Liver Sinusoidal Endothelial Cell: An Update." Seminars in Liver Disease 37, no. 04 (November 2017): 377–87. http://dx.doi.org/10.1055/s-0037-1617455.

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AbstractThis update focuses on two main topics. First, recent developments in our understanding of liver sinusoidal endothelial cell (LSEC) function will be reviewed, specifically elimination of blood-borne waste, immunological function of LSECs, interaction of LSECs with liver metastases, LSECs and liver regeneration, and LSECs and hepatic fibrosis. Second, given the current emphasis on rigor and transparency in biomedical research, the update discusses the need for standardization of methods to demonstrate identity and purity of isolated LSECs, pitfalls in methods that might lead to a select
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Tee, Jie, Li Ng, Hannah Koh, David Leong, and Han Ho. "Titanium Dioxide Nanoparticles Enhance Leakiness and Drug Permeability in Primary Human Hepatic Sinusoidal Endothelial Cells." International Journal of Molecular Sciences 20, no. 1 (December 21, 2018): 35. http://dx.doi.org/10.3390/ijms20010035.

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Liver sinusoidal endothelial cells (LSECs) represent the permeable interface that segregates the blood compartment from the hepatic cells, regulating hepatic vascular tone and portal pressure amidst changes in the blood flow. In the presence of pathological conditions, phenotypic changes in LSECs contribute to the progression of chronic liver diseases, including the loss of endothelial permeability. Therefore, modulating LSECs offers a possible way to restore sinusoidal permeability and thereby improve hepatic recovery. Herein, we showed that titanium dioxide nanoparticles (TiO2 NPs) could ind
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Cogger, Victoria Carroll, Mashani Mohamad, Samantha Marie Solon-Biet, Alistair M. Senior, Alessandra Warren, Jennifer Nicole O'Reilly, Bui Thanh Tung, et al. "Dietary macronutrients and the aging liver sinusoidal endothelial cell." American Journal of Physiology-Heart and Circulatory Physiology 310, no. 9 (May 1, 2016): H1064—H1070. http://dx.doi.org/10.1152/ajpheart.00949.2015.

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Fenestrations are pores within the liver sinusoidal endothelial cells (LSECs) that line the sinusoids of the highly vascularized liver. Fenestrations facilitate the transfer of substrates between blood and hepatocytes. With pseudocapillarization of the hepatic sinusoid in old age, there is a loss of fenestrations. LSECs are uniquely exposed to gut-derived dietary and microbial substrates delivered by the portal circulation to the liver. Here we studied the effect of 25 diets varying in content of macronutrients and energy on LSEC fenestrations using the Geometric Framework method in a large co
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Enomoto, Katsuhiko, Yuji Nishikawa, Yasufumi Omori, Takuo Tokairin, Masayuki Yoshida, Naoto Ohi, Takuya Nishimura, Youhei Yamamoto, and Qinchang Li. "Cell biology and pathology of liver sinusoidal endothelial cells." Medical Electron Microscopy 37, no. 4 (December 2004): 208–15. http://dx.doi.org/10.1007/s00795-004-0261-4.

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Wang, Lin, Xiangdong Wang, Guanhua Xie, Lei Wang, Colin K. Hill, and Laurie D. DeLeve. "Liver sinusoidal endothelial cell progenitor cells promote liver regeneration in rats." Journal of Clinical Investigation 122, no. 4 (April 2, 2012): 1567–73. http://dx.doi.org/10.1172/jci58789.

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Pitas, R. E., J. Boyles, R. W. Mahley, and D. M. Bissell. "Uptake of chemically modified low density lipoproteins in vivo is mediated by specific endothelial cells." Journal of Cell Biology 100, no. 1 (January 1, 1985): 103–17. http://dx.doi.org/10.1083/jcb.100.1.103.

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Acetoacetylated (AcAc) and acetylated (Ac) low density lipoproteins (LDL) are rapidly cleared from the plasma (t1/2 approximately equal to 1 min). Because macrophages, Kupffer cells, and to a lesser extent, endothelial cells metabolize these modified lipoproteins in vitro, it was of interest to determine whether endothelial cells or macrophages could be responsible for the in vivo uptake of these lipoproteins. As previously reported, the liver is the predominant site of the uptake of AcAc LDL; however, we have found that the spleen, bone marrow, adrenal, and ovary also participate in this rapi
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Koch, Philipp-Sebastian, Ki Hong Lee, Sergij Goerdt, and Hellmut G. Augustin. "Angiodiversity and organotypic functions of sinusoidal endothelial cells." Angiogenesis 24, no. 2 (March 21, 2021): 289–310. http://dx.doi.org/10.1007/s10456-021-09780-y.

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Abstract‘Angiodiversity’ refers to the structural and functional heterogeneity of endothelial cells (EC) along the segments of the vascular tree and especially within the microvascular beds of different organs. Organotypically differentiated EC ranging from continuous, barrier-forming endothelium to discontinuous, fenestrated endothelium perform organ-specific functions such as the maintenance of the tightly sealed blood–brain barrier or the clearance of macromolecular waste products from the peripheral blood by liver EC-expressed scavenger receptors. The microvascular bed of the liver, compos
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Gibert-Ramos, Albert, David Sanfeliu-Redondo, Peio Aristu-Zabalza, Ana Martínez-Alcocer, Jordi Gracia-Sancho, Sergi Guixé-Muntet, and Anabel Fernández-Iglesias. "The Hepatic Sinusoid in Chronic Liver Disease: The Optimal Milieu for Cancer." Cancers 13, no. 22 (November 15, 2021): 5719. http://dx.doi.org/10.3390/cancers13225719.

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The liver sinusoids are a unique type of microvascular beds. The specialized phenotype of sinusoidal cells is essential for their communication, and for the function of all hepatic cell types, including hepatocytes. Liver sinusoidal endothelial cells (LSECs) conform the inner layer of the sinusoids, which is permeable due to the fenestrae across the cytoplasm; hepatic stellate cells (HSCs) surround LSECs, regulate the vascular tone, and synthetize the extracellular matrix, and Kupffer cells (KCs) are the liver-resident macrophages. Upon injury, the harmonic equilibrium in sinusoidal communicat
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Zhang, Xingqin, Yi Chen, Liqun Tang, Yunhai Zhang, Pengkai Duan, Lei Su, and Huasheng Tong. "The liver sinusoidal endothelial cell damage in rats caused by heatstroke." European Journal of Inflammation 16 (January 2018): 205873921879432. http://dx.doi.org/10.1177/2058739218794328.

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This study was designed to explore whether liver sinusoidal endothelial cells (SECs) play a pathological role in liver injury of heatstroke (HS) in rats. An HS rat model was prepared in a pre-warmed incubator. Rats were randomized into four groups: HS-sham group (SHAM group), the 39°C group, the 42°C group, and the HS group. The serum concentrations of SEC injury biomarkers including hyaluronic acid (HA), von Willebrand factor (vWF), thrombomodulin (TM), were measured. Plasma alanine aminotransferase (ALT) and aspartate aminotransferase (AST) activities and endothelium-derived vasoactive subst
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Dissertations / Theses on the topic "Liver sinusoidal endothelial cell"

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Cheluvappa, Rajkumar. "Pathophysiology of Liver Sinusoidal Endothelial Cells." Thesis, The University of Sydney, 2008. http://hdl.handle.net/2123/2802.

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Owing to its strategic position in the liver sinusoid, pathologic and morphologic alterations of the Liver Sinusoidal Endothelial Cell (LSEC) have far-reaching repercussions for the whole liver and systemic metabolism. LSECs are perforated with fenestrations, which are pores that facilitate the transfer of lipoproteins and macromolecules between blood and hepatocytes. Loss of LSEC porosity is termed defenestration, which can result from loss of fenestrations and/ or decreases in fenestration diameter. Gram negative bacterial endotoxin (Lipopolysaccharide, LPS) has marked effects on LSEC morpho
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Cheluvappa, Rajkumar. "Pathophysiology of Liver Sinusoidal Endothelial Cells." University of Sydney, 2008. http://hdl.handle.net/2123/2802.

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Doctor of Philosophy(PhD)<br>Owing to its strategic position in the liver sinusoid, pathologic and morphologic alterations of the Liver Sinusoidal Endothelial Cell (LSEC) have far-reaching repercussions for the whole liver and systemic metabolism. LSECs are perforated with fenestrations, which are pores that facilitate the transfer of lipoproteins and macromolecules between blood and hepatocytes. Loss of LSEC porosity is termed defenestration, which can result from loss of fenestrations and/ or decreases in fenestration diameter. Gram negative bacterial endotoxin (Lipopolysaccharide, LPS) has
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Akingbasote, J. A. "The potential role of liver sinusoidal endothelial cells in drug-induced liver injury." Thesis, University of Liverpool, 2016. http://livrepository.liverpool.ac.uk/3005113/.

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Liver sinusoidal endothelial cells (LSEC) constitute a unique population of endothelial cells with specialised liver-specific morphologic features and functions. LSEC are the only endothelial cells with fenestrations and which lack an organised basement membrane. They are involved in hepatic stellate cell (HSC) quiescence, endocytosis of small particles, selective transfer of substances from the blood, in the hepatic sinusoid, to the parenchymal cells and in liver regeneration. As the group of cells that form the inner lining of the capillaries of the liver sinusoids, and being the first to be
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O'REILLY, Jennifer. "The role and ultrastructure of the liver sinusoidal endothelial cell in fasting, hepatoxicity, and ageing." Thesis, The University of Sydney, 2014. http://hdl.handle.net/2123/10550.

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The majority of liver studies focus on the hepatocyte however the work of this thesis investigates the vital role of the liver sinusoidal endothelial cell (LSEC). LSECs line the liver sinusoids forming a protective barrier between the blood and hepatocytes. The LSEC cytoplasm resembles a sieve, perforated with thousands of transcellular pores of approximately 50-150 nm in diameter called fenestrations, and is underlined by a very sparse extracellular matrix. This facilitates the virtually unimpeded passage of fluid and substances smaller than fenestrations from the blood such as drugs and nutr
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Rowe, Ian Alston Cooper. "The role of liver sinusoidal endothelial cells in hepatitis C virus infection." Thesis, University of Birmingham, 2013. http://etheses.bham.ac.uk//id/eprint/4123/.

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Hepatitis C virus (HCV) infection is a major cause of global morbidity, causing chronic liver injury that can progress to cirrhosis and hepatocellular carcinoma. The liver is a large and complex organ containing multiple cell types, including hepatocytes, sinusoidal endothelial cells (LSEC), stellate cells, Kupffer cells and biliary epithelial cells. Hepatocytes are the major reservoir supporting HCV replication, however, the role of non-­‐parenchymal cells in the viral lifecycle remain largely unexplored. Endothelial cell hepatocyte co-­‐cultures were established to study the role of LSEC in
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Mohamad, Mashani. "The Role of the Liver Sinusoidal Endothelial Cells in the Pathophysiology of Insulin Resistance." Thesis, The University of Sydney, 2016. http://hdl.handle.net/2123/15716.

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Ageing is associated with increased prevalence of metabolic syndrome, as well as impaired glucose metabolism, hyperinsulinemia and insulin resistance. The mechanism underlying these associations is poorly understood and is likely to be complex and multifactorial. The liver is the key target for insulin action and while the endothelium has been shown to influence insulin activity in muscle and fat, the role of the liver sinusoidal endothelium on the action of insulin in the liver is unknown. The liver sinusoidal endothelium is unique: it is perforated with transcellular pores called fenestratio
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Banga, Neal Roop. "Effects of Ischaemia-Reperfusion Injury and Ischaemic Preconditioning on Human Liver Sinusoidal Endothelial Cells." Thesis, University of Leeds, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.515298.

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Kojima, Hidenobu. "Establishment of practical recellularized liver graft for blood perfusion using primary rat hepatocytes and liver sinusoidal endothelial cells." Kyoto University, 2018. http://hdl.handle.net/2433/233836.

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Yagi, Toshikazu. "The protective effects of prostaglandin E1 on sinusoidal endothelial cells in xenogeneic pig liver perfusion." Kyoto University, 1998. http://hdl.handle.net/2433/182254.

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Miyachi, Yosuke. "Causes of liver steatosis influence the severity of ischemia reperfusion injury and survival after liver transplantation in rats." Doctoral thesis, Kyoto University, 2021. http://hdl.handle.net/2433/263516.

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Books on the topic "Liver sinusoidal endothelial cell"

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Morgan, Glyn. Correlation of donor nutritional status with sinusoidal lining cell viability and liver function in the rat. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1992.

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Lalor, Patricia, Leo A. van Grunsven, and Thomas Huser, eds. Roles of Liver Sinusoidal Endothelial Cells in Liver Homeostasis and Disease. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88974-794-8.

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Charles, Balabaud, and Bioulac-Sage Paulette, eds. Sinusoids in human liver: Health and disease : editors, Paulette Bioulac-Sage, Charles Balabaud. Rijswijk, The Netherlands: Kupffer Cell Foundation, 1988.

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Claire Maureen Barbara.* Holloway. Sinusoidal lining cell damage: the critical injury in cold preservation of liver allografts in the rat. 1991.

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Bhopal, Raj S. Epidemic of Cardiovascular Disease and Diabetes. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780198833246.001.0001.

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Coronary heart disease (CHD) and stroke, collectively cardiovascular disease (CVD), are caused by narrowing and blockage of the arteries supplying the heart and brain, respectively. In type 2 diabetes (DM<sub>2</sub>) insulin is insufficient to maintain normal blood glucose. South Asians have high susceptibility to these diseases. Drawing upon the scientific literature and discussions with 22 internationally recognized scholars, this book focuses on causal explanations and their implications for prevention and research. Genetically based hypotheses are considered together with the developmenta
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Book chapters on the topic "Liver sinusoidal endothelial cell"

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Kmieć, Zbigniew. "Sinusoidal Endothelial Cells." In Cooperation of Liver Cells in Health and Disease, 13–19. Berlin, Heidelberg: Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-642-56553-3_3.

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Hilscher, Moira B., Robert C. Huebert, and Vijay H. Shah. "Hepatic sinusoidal endothelial cells." In Signaling Pathways in Liver Diseases, 73–84. Chichester, UK: John Wiley & Sons, Ltd, 2015. http://dx.doi.org/10.1002/9781118663387.ch5.

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Huebert, Robert C., and Vijay H. Shah. "Hepatic Sinusoidal Endothelial Cells." In Signaling Pathways in Liver Diseases, 79–91. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-00150-5_5.

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DeLeve, Laurie D. "Vascular Liver Disease and the Liver Sinusoidal Endothelial Cell." In Vascular Liver Disease, 25–40. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-8327-5_2.

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Fujiwara, Kenji, and Satoshi Mochida. "Sinusoidal Endothelial Cells in Liver Regeneration." In Liver Diseases and Hepatic Sinusoidal Cells, 114–23. Tokyo: Springer Japan, 1999. http://dx.doi.org/10.1007/978-4-431-67935-6_8.

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Shakado, Satoshi, Shotaro Sakisaka, Kazunori Noguchi, Michio Sata, and Kyuichi Tanikawa. "Angiogenesis of Cultured Rat Sinusoidal Endothelial Cells." In Liver Diseases and Hepatic Sinusoidal Cells, 124–27. Tokyo: Springer Japan, 1999. http://dx.doi.org/10.1007/978-4-431-67935-6_9.

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Wisse, E., F. Braet, D. Luo, D. Vermijlen, M. Eddouks, M. Konstandoulaki, C. Empsen, and R. B. de Zanger. "Endothelial Cells of the Hepatic Sinusoids: A Review." In Liver Diseases and Hepatic Sinusoidal Cells, 17–53. Tokyo: Springer Japan, 1999. http://dx.doi.org/10.1007/978-4-431-67935-6_2.

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Okanoue, Takeshi, Shinichi Sakamoto, Takashi Mori, Yoshihiko Sawa, Hikoharu Kanaoka, Kenichi Nishioji, and Yoshito Itoh. "Role of Sinusoidal Endothelial Cells in Alcoholic Liver Disease." In Liver Diseases and Hepatic Sinusoidal Cells, 190–98. Tokyo: Springer Japan, 1999. http://dx.doi.org/10.1007/978-4-431-67935-6_15.

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Ohira, Hiromasa, Takato Ueno, Kyuichi Tanikawa, and Reiji Kasukawa. "Changes in Adhesion Molecules of Sinusoidal Endothelial Cells in Liver Injury." In Liver Diseases and Hepatic Sinusoidal Cells, 91–100. Tokyo: Springer Japan, 1999. http://dx.doi.org/10.1007/978-4-431-67935-6_6.

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Oda, Masaya, Hiroaki Yokomori, and Yoshitaka Kamegaya. "Roles of Sinusoidal Endothelial Cells in the Local Regulation of Hepatic Sinusoidal Blood Flow—Involvement of Endothelins and Nitric Oxide." In Liver Diseases and Hepatic Sinusoidal Cells, 141–55. Tokyo: Springer Japan, 1999. http://dx.doi.org/10.1007/978-4-431-67935-6_11.

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Conference papers on the topic "Liver sinusoidal endothelial cell"

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Kang, Young Bok (Abraham), Joseph Cirillo, Siddhartha Rawat, Michael Bouchard, and Hongseok (Moses) Noh. "Layered Hepatocytes and Endothelial Cells on a Transwell Membrane: Toward Engineering the Liver Sinusoid." In ASME 2012 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/imece2012-89413.

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This paper presents a novel liver model platform that mimics the liver sinusoid, a functional unit of the liver where most liver activities occur. A key component of the current liver model is a layered co-culture of primary rat hepatocytes (PRH) and primary rat liver sinusoidal endothelial cells (LSEC) or a bovine aortic endothelial cells (BAEC) as an alternative. Poly-dimethylsiloxane (PDMS) microchannels were fabricated and attached to transwell membranes that contain submicroscale pores. Cells were cultured either on one side or on both sides of the transwell membrane, and in both cases ce
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El-Maarri, Osman, Muhammad Ahmer Jamil, Heike Singer, Rawya Al-Rifai, and Johannes Oldenburg. "Molecular Profiling of Fetal and Adult Liver Sinusoidal Endothelial Cells: A F8 Secreting Cell." In Hamburger Hämophilie Symposion Hamburg, Germany. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1721572.

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McNerney, G. P., W. Hubner, V. C. Cogger, D. L. Thompson, C. I. Oie, L. D. DeLeve, P. McCourt, et al. "Structured illumination microscopy applications towards liver sinusoidal endothelial cell fenestrations and HIV-1 cell-to-cell transmission." In 2010 Asia Communications and Photonics Conference and Exhibition (ACP 2010). IEEE, 2010. http://dx.doi.org/10.1109/acp.2010.5682515.

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Gottwick, Cornelia, Daria Krzikalla, LígiaMargaridaMarques Mesquita, Federica Mancini, Marco Fanzutti, Reinaldo Digigow, AnsgarW Lohse, Antonella Carambia, and Johannes Herkel. "Liver fibrosis does not impair tolerance induction by liver sinusoidal endothelial cells in vivo." In 38. Jahrestagung der Deutsche Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag, 2022. http://dx.doi.org/10.1055/s-0041-1740793.

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Gottwick, C., A. Carambia, R. Digigow, M. Şeleci, D. Mungalpara, M. Heine, FA Schuran, et al. "Nanoparticle-mediated peptide delivery to liver sinusoidal endothelial cells protects from CD8 T cell-driven cholangitis." In 36. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0039-3402201.

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Harrison, R. "HUMAN HEPATIC ENDOTHELIAL CELLS AND HEPATOCYTES IN CULTURE: MORPHOLOGICAL FEATURES, AND PRODUCTION OF VON WILLEBRAND FACTOR AND FIBRINOGEN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643350.

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Liver cells were derived from cadaveric organ donors. Pieces of human liver 5 to 50 grams were minced, washed, and incubated in collagenase at 37 degrees C. After washing, the cell suspension was plated into culture vessels that had been briefly pre-treated with an extract derived from human liver. A mixed population of liver cells, including endothelial cells, hepatocytes, and Kupffer cells, attached within hours. At the end of 2 to 3 weeks there developed clusters of densely packed cells of two types. The most numerous cells were initially fusiform but grew as a monolayer even when densely p
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Colucci, S., S. Hammad, S. Altamura, O. Marques, A. Dropmann, NK Horvat, K. Müdder, K. Gould, S. Dooley та MU Muckenthaler. "Liver sinusoidal endothelial cells suppress BMP2 production in response to TGFβ pathway activation". У Viszeralmedizin 2021 Gemeinsame Jahrestagung Deutsche Gesellschaft für Gastroenterologie, Verdauungs- und Stoffwechselkrankheiten (DGVS), Sektion Endoskopie der DGVS, Deutsche Gesellschaft für Allgemein und Viszeralchirurgie (DGAV). Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1733619.

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Colucci, S., S. Hammad, S. Altamura, O. Marques, A. Dropmann, NK Horvat, K. Müdder, K. Gould, S. Dooley та MU Muckenthaler. "Liver sinusoidal endothelial cells suppress BMP2 production in response to TGFβ pathway activation". У Viszeralmedizin 2021 Gemeinsame Jahrestagung Deutsche Gesellschaft für Gastroenterologie, Verdauungs- und Stoffwechselkrankheiten (DGVS), Sektion Endoskopie der DGVS, Deutsche Gesellschaft für Allgemein und Viszeralchirurgie (DGAV). Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1733619.

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Slater, John H., Shailendra Jain, Robin N. Coger, and Charles Y. Lee. "The Effects of Shear Stress on Endothelial Cells at Hypothermic Temperatures." In ASME 2002 International Mechanical Engineering Congress and Exposition. ASMEDC, 2002. http://dx.doi.org/10.1115/imece2002-33705.

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Hypothermic machine perfusion preservation (MPP) has proven to be a successful technique for hypothermic kidney storage, however this technology has not successfully been applied to the liver. Recent research has indicated that the endothelial cells lining the liver sinusoids display rounding phenomena during MPP that is not fully understood. In order to gain a better understanding of endothelial cell shear stress response and the factors that induce rounding, a temperature-controlled micro-shear chamber has been designed and fabricated. The micro-shear chamber has been used to apply shear str
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Butola, Ankit, David A. Coucheron, Karolina Szafranska, Azeem Ahmad, Hong Mao, Jean-Claude Tinguely, Peter McCourt, et al. "Quantitative phase imaging and on-chip nanoscopy for 3D imaging of liver sinusoidal endothelial cells." In Digital Holography and Three-Dimensional Imaging. Washington, D.C.: Optica Publishing Group, 2022. http://dx.doi.org/10.1364/dh.2022.w4a.2.

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We present a highly spatially sensitive quantitative phase microscopy system integrated with on-chip nanoscopy to visualize 3D morphology of liver sinusoidal endothelial cells (LSECs). We used the system to obtain 3D morphology of LSEC by using chip-based nanoscopy for lateral super-resolution, and QPM for mapping nanoscale thickness.
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