Relevant bibliographies by topics / LN229

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Academic literature on the topic 'LN229'

Author: Grafiati
Published: 24 April 2022
Last updated: 30 July 2025

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Journal articles on the topic "LN229"

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Kumar, Vivek, Mohit Vashishta, Lin Kong, et al. "Carbon Ion Irradiation Downregulates Notch Signaling in Glioma Cell Lines, Impacting Cell Migration and Spheroid Formation." Cells 11, no. 21 (2022): 3354. http://dx.doi.org/10.3390/cells11213354.

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Photon-based radiotherapy upregulates Notch signaling in cancer, leading to the acquisition of the stem cell phenotype and induction of invasion/migration, which contributes to the development of resistance to therapy. However, the effect of carbon ion radiotherapy (CIRT) on Notch signaling in glioma and its impact on stemness and migration is not explored yet. Human glioma cell lines (LN229 and U251), stable Notch1 intracellular domain (N1ICD) overexpressing phenotype of LN229 cells, and Notch inhibitor resistant LN229 cells (LN229R) were irradiated with either photon (X-rays) or (carbon ion
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Liu, Qian Rong, Ju Mei Liu, Yong Chen, et al. "Piperlongumine Inhibits Migration of Glioblastoma Cells via Activation of ROS-Dependent p38 and JNK Signaling Pathways." Oxidative Medicine and Cellular Longevity 2014 (2014): 1–12. http://dx.doi.org/10.1155/2014/653732.

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Piperlongumine (PL) is recently found to kill cancer cells selectively and effectively via targeting reactive oxygen species (ROS) responses. To further explore the therapeutic effects of PL in cancers, we investigated the role and mechanisms of PL in cancer cell migration. PL effectively inhibited the migration of human glioma (LN229 or U87 MG) cells but not normal astrocytes in the scratch-wound culture model. PL did not alter EdU+-cells and cdc2, cdc25c, or cyclin D1 expression in our model. PL increased ROS (measured by DCFH-DA), reduced glutathione, activated p38 and JNK, increased IκBα,
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Galicka, Anna, Łukasz Szoka, Iwona Radziejewska, and Cezary Marcinkiewicz. "Effect of Dimeric Disintegrins Isolated from Vipera lebetina obtusa Venom on Glioblastoma Cellular Responses." Cancers 15, no. 19 (2023): 4805. http://dx.doi.org/10.3390/cancers15194805.

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Integrins play a fundamental role in the migration and invasiveness of glioblastoma (GBM) cells, making them suitable targets for innovative cancer therapy. The aim of this study was to evaluate the effect of the RGD homodimeric disintegrin VLO4, isolated from Vipera lebetina obtusa venom, on the adhesion, spreading, migration, and survival of LBC3, LN18, and LN229 cell lines. This disintegrin, as a potent antagonist for α5β1 integrin, showed pro-adhesive properties for these cell lines, the highest for LN229 and the lowest for LBC3. Glioblastoma cells displayed significant differences in the
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Han, Ji-Hun, Da-Young Chang, Young-Joon Lee, Haeyoung Suh-Kim, and Sung-Soo Kim. "STEM-01. CHARACTERIZATION OF ORTHOTOPIC GLIOBLASTOMA MODEL FOR THE GENE-AND CELL THERAPY." Neuro-Oncology 21, Supplement_6 (2019): vi234. http://dx.doi.org/10.1093/neuonc/noz175.975.

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Abstract Intrinsic heterogeneous and infiltrative nature of glioblastoma cells to the adjacent normal brain parenchyma are the main obstacles to the treatment of glioblastoma multiforme (GBM). The infiltrative nature of GBM increases following various treatments, which often leads to tumor recurrence. U87 cell line, a commonly used as a GBM cell line till recently, has appeared inappropriate for GBM model. Thus, we established an orthotopic GBM model using another cell line, LN229. Here, we discuss the infiltrative nature of LN229 in the mouse brain and their interaction with adjacent brain mi
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Chen, J., C. Pellegrino, P. Roth, et al. "P18.01.A ADAPTER FITC CAR T-CELLS PLUS OCTOFLUO FOR THE TREATMENT OF GLIOMAS VIA SSTR2." Neuro-Oncology 26, Supplement_5 (2024): v95. http://dx.doi.org/10.1093/neuonc/noae144.317.

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Abstract BACKGROUND Somatostatin receptor (SSTR) 2 has been proposed as a target to deliver therapeutic agents to gliomas. The SSTR2 antagonist peptide octofluo was developed to redirect fluorescein-5-isothiocyanate (FITC)-specific adapter chimeric antigen receptor (AdCAR)-T cells towards SSTR2-expressing tumor cells. MATERIAL AND METHODS A tissue microarray of glioblastoma patient samples (N=161) was stained for SSTR2. Octofluo was used as an adapter to direct FITC-specific human CAR-T cells against glioma cells. The SSTR2- glioma cell line LN229 was lentivirally transduced to express SSTR2 a
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He, Xiu-Ying, Liu-Lin Xiong, Qing-Jie Xia, et al. "C18H17NO6and Its Combination with Scutellarin Suppress the Proliferation and Induce the Apoptosis of Human Glioma Cells via Upregulation of Fas-Associated Factor 1 Expression." BioMed Research International 2019 (February 20, 2019): 1–20. http://dx.doi.org/10.1155/2019/6821219.

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Background. Glioma is the most common malignant brain tumor and the patients are prone to poor prognosis. Due to limited treatments, new drug exploration has become a general trend. Therefore, the objective of this study is to investigate the effect of the new drugs C18H17NO6and its combination with Scutellarin on glioma cells and the underlying mechanism.Method. U251 and LN229 cells were administrated with C18H17NO6and its combination with Scutellarin. The proliferation ability of glioma cells was determined by cell counting kit-8, plate clone formation assay, and EdU incorporation assay. The
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Sravya, Palavalasa, Vidya Nimbalkar, Nandaki Kanuri, et al. "RDNA-11. LOW MITOCHONDRIAL DNA CONTENT INDUCES TREATMENT RESISTANCE AND CONFERS POOR PROGNOSIS IN GLIOBLASTOMA." Neuro-Oncology 21, Supplement_6 (2019): vi209. http://dx.doi.org/10.1093/neuonc/noz175.870.

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Abstract BACKGROUND Investigation of mitochondrial DNA (mtDNA) content in glioblastoma has gathered speed in recent years. Our study focusses on the effect of mtDNA content depletion on treatment response in glioblastoma. MATERIALS AND METHODS The study consists of a survival cohort (n=130), a recurrent cohort (32 pairs) and 35 non neoplastic control brain tissues. Malignant glioma cell lines, U87 and LN229 were utilized for radiation biology experiments. Patient tumor tissue was molecularly characterized for IDH, ATRX and TERT promoter mutations, MGMT promoter methylation and EGFR amplificati
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Sravya, P., V. P. Nimbalkar, N. N. Kanuri, et al. "P14.94 Mitochondrial DNA copy number in new onset and recurrent glioblastoma and its effect on radiation resistance and patient survival." Neuro-Oncology 21, Supplement_3 (2019): iii90. http://dx.doi.org/10.1093/neuonc/noz126.329.

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Abstract BACKGROUND Recent evidence shows that mitochondrial DNA (mtDNA) content is responsible for radiation resistance in various cancers, but not evaluated in glioblastoma(GBM).Hence,we studied the role of mtDNA content in GBM pathogenesis and treatment response. MATERIAL AND METHODS Archived FFPE tissues of newly diagnosed GBM(n=130), recurrent GBM (n=32 pairs) and non-neoplastic control brain(n=30) with available clinical details were utilized for the study. Immunohistochemistry, Sanger’s sequencing, methylation specific PCR and fluorescent in-situ hybridization were used to study IDH, AT
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Lee, Eun Jeong, Jee Young Sung, Kyung Hee Koo, et al. "Anti-Tumor Effects of Sodium Meta-Arsenite in Glioblastoma Cells with Higher Akt Activities." International Journal of Molecular Sciences 21, no. 23 (2020): 8982. http://dx.doi.org/10.3390/ijms21238982.

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Glioblastoma is a type of aggressive brain tumor that grows very fast and evades surrounding normal brain, lead to treatment failure. Glioblastomas are associated with Akt activation due to somatic alterations in PI3 kinase/Akt pathway and/or PTEN tumor suppressor. Sodium meta-arsenite, KML001 is an orally bioavailable, water-soluble, and trivalent arsenical and it shows antitumoral effects in several solid tumor cells via inhibiting oncogenic signaling, including Akt and MAPK. Here, we evaluated the effect of sodium meta-arsenite, KML001, on the growth of human glioblastoma cell lines with di
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Li, Tian-Yi, Yang Du, Min-Chang Wang, et al. "Cytotoxic Steroidal Saponins Containing a Rare Fructosyl from the Rhizomes of Paris polyphylla var. latifolia." International Journal of Molecular Sciences 24, no. 8 (2023): 7149. http://dx.doi.org/10.3390/ijms24087149.

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A phytochemical investigation of the steroidal saponins from the rhizomes of Paris polyohylla var. latifolia led to the discovery and characterization of three new spirostanol saponins, papolatiosides A–C (1–3), and nine known compounds (4–12). Their structures were established via extensive spectroscopic data analysis and chemical methods. Interestingly, compounds 1 and 2 possessed a fructosyl in their oligosaccharide moiety, which is rare in natural product and was firstly reported in family Melanthiaceae. The cytotoxicity of these saponins against several human cancer cell lines was evaluat
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Books on the topic "LN229"

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Aubrey, David G., and Lee Weishar, eds. Hydrodynamics and Sediment Dynamics of Tidal Inlets. American Geophysical Union, 1988. http://dx.doi.org/10.1029/ln029.

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Conference papers on the topic "LN229"

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Dong, Junhong, Ping Wang, Qingluan Xu, Kunpeng Lv, Yu Wang, and Junhong Dong. "Expression of Beta-Adrenergic Receptor in Glioma LN229 Cells and Its Effect on Cell Proliferation." In 2016 7th International Conference on Education, Management, Computer and Medicine (EMCM 2016). Atlantis Press, 2017. http://dx.doi.org/10.2991/emcm-16.2017.140.

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Kim, Sun Jin, Seung Wook Kim, Hyun Jin Choi, et al. "Abstract 5481: Therapy of human LN229 glioblastoma growing in the brain of nude mice by Macitentan, a dual endothelin receptor A and B antagonist, combined with Temozolomide." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-5481.

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