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1

Josephs, Keith A., Jennifer L. Whitwell, Clifford R. Jack, Joseph E. Parisi, and Dennis W. Dickson. "Frontotemporal Lobar Degeneration Without Lobar Atrophy." Archives of Neurology 63, no. 11 (2006): 1632. http://dx.doi.org/10.1001/archneur.63.11.1632.

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2

Sirpuram Milind Joshi, Varundev. "Congenital Lobar Emphysema." International Journal of Science and Research (IJSR) 12, no. 1 (2023): 676–78. http://dx.doi.org/10.21275/mr23120200847.

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3

Huntley, James S. "Lobar Pneumonia." Family Medicine 53, no. 10 (2021): 898. http://dx.doi.org/10.22454/fammed.2021.514467.

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4

Flores-Sarnat, Laura, Harvey B. Samat, Piero Pavone, et al. "Lobar Holoprosencephaly." Journal of Child Neurology 17, no. 7 (2002): 543–44. http://dx.doi.org/10.1177/088307380201700715.

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5

Wakai, S. "Lobar hemorrhages." Stroke 25, no. 4 (1994): 914–15. http://dx.doi.org/10.1161/str.25.4.914b.

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6

Starnes, Vaughn A., Mark L. Barr, and Robbin G. Cohen. "Lobar transplantation." Journal of Thoracic and Cardiovascular Surgery 108, no. 3 (1994): 403–11. http://dx.doi.org/10.1016/s0022-5223(94)70249-7.

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7

Mintzer, Richard A., Bernard A. Sakowicz, and Jeffrey A. Blonder. "Lobar Collapse." Chest 94, no. 3 (1988): 615–20. http://dx.doi.org/10.1378/chest.94.3.615.

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8

Haddy, Steven, and Vaughn A. Starnes. "Lobar transplantation." Journal of Thoracic and Cardiovascular Surgery 126, no. 6 (2003): 2106. http://dx.doi.org/10.1016/s0022-5223(03)01011-0.

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9

Della Pona, C., G. Rocco, A. Rizzi, M. Robustellini, G. Rossi, and B. Crasti. "Lobar hypoplasia." European Respiratory Journal 4, no. 9 (1991): 1140–42. http://dx.doi.org/10.1183/09031936.93.04091140.

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Lung tissue developmental abnormalities are seldom reported. According to the classification of Schneider (1900), which was amended by Boyden (1955), they include pulmonary agenesis, aplasia and hypoplasia. Due to the early onset of symptoms, lung agenesis and aplasia are usually detected soon after birth. Conversely, lung or lobar hypoplasia may remain clinically silent for a long time. A single case of left lower lobe hypoplasia is reported. A recurring and unrelenting septic fever was the presenting symptom, whilst the radiological picture showed a left lower lobe consolidation. On the surg
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10

Li, Linxin, Ramon Luengo-Fernandez, Susanna M. Zuurbier, et al. "Ten-year risks of recurrent stroke, disability, dementia and cost in relation to site of primary intracerebral haemorrhage: population-based study." Journal of Neurology, Neurosurgery & Psychiatry 91, no. 6 (2020): 580–85. http://dx.doi.org/10.1136/jnnp-2019-322663.

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BackgroundPatients with primary intracerebral haemorrhage (ICH) are at increased long-term risks of recurrent stroke and other comorbidities. However, available estimates come predominantly from hospital-based studies with relatively short follow-up. Moreover, there are also uncertainties about the influence of ICH location on risks of recurrent stroke, disability, dementia and quality of life.MethodsIn a population-based study (Oxford Vascular Study/2002–2018) of patients with a first ICH with follow-up to 10 years, we determined the long-term risks of recurrent stroke, disability, quality of
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11

Jung, Young Hee, Hyemin Jang, Seong Beom Park, et al. "Strictly Lobar Microbleeds Reflect Amyloid Angiopathy Regardless of Cerebral and Cerebellar Compartments." Stroke 51, no. 12 (2020): 3600–3607. http://dx.doi.org/10.1161/strokeaha.119.028487.

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Background and Purpose: We aimed to determine whether lobar cerebellar microbleeds or concomitant lobar cerebellar and deep microbleeds, in the presence of lobar cerebral microbleeds, attribute to underlying advanced cerebral amyloid angiopathy pathology or hypertensive arteriopathy. Methods: We categorized 71 patients with suspected cerebral amyloid angiopathy markers (regardless of the presence of deep and cerebellar microbleeds) into 4 groups according to microbleed distribution: L (strictly lobar cerebral, n=33), L/LCbll (strictly lobar cerebral and strictly lobar cerebellar microbleeds, n
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12

Sawyer, Russell P., Padmini Sekar, Jennifer Osborne, et al. "Racial/ethnic variation of APOE alleles for lobar intracerebral hemorrhage." Neurology 91, no. 5 (2018): e410-e420. http://dx.doi.org/10.1212/wnl.0000000000005908.

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ObjectiveAPOE ε2 and ε4 alleles have been associated with lobar intracerebral hemorrhage (ICH) in predominately white populations; we sought to evaluate whether this held true among black and Hispanic populations.MethodsThe Ethnic/Racial Variations of Intracerebral Hemorrhage study is a prospective, multicenter case-control study of ICH among white, black, and Hispanic participants. Controls were recruited to match cases based on age, ethnicity/race, sex, and geographic location. APOE genotyping and ICH location was determined blinded to clinical data.ResultsThere were 907 cases of lobar ICH a
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13

Nakase, Taizen, Shotaroh Yoshioka, Masahiro Sasaki, and Akifumi Suzuki. "Clinical features of recurrent stroke after intracerebral hemorrhage." Neurology International 4, no. 2 (2012): 10. http://dx.doi.org/10.4081/ni.2012.e10.

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There have been many reports about the prognosis and risk factors of stroke recurrence following brain infarction (BI). However, little is known about the stroke recurrence after primary intracerebral hemorrhage (PICH). Therefore, we explored the recurrent stroke patients after initial PICH retrospectively, to reveal the critical factors of stroke recurrence. Acute BI (n=4013) and acute PICH patients (n=1067) admitted to the hospital between April 2000 and March 2009 were consecutively screened. PICH patients with a history of ICH and BI patients with a history of ICH were then classified into
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14

Matsukawa, H., M. Shinoda, M. Fujii, et al. "Factors associated with lobar vs. non-lobar intracerebral hemorrhage." Acta Neurologica Scandinavica 126, no. 2 (2011): 116–21. http://dx.doi.org/10.1111/j.1600-0404.2011.01615.x.

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15

Rajeshwari, M. S., and Priya Ranganath. "Variations in Draining Patterns of Right Pulmonary Veins at the Hilum and an Anatomical Classification." ISRN Pulmonology 2012 (June 19, 2012): 1–4. http://dx.doi.org/10.5402/2012/786549.

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Pulmonary veins carry oxygenated blood from the lungs to the left atrium. Variations are quite common in the pattern of drainage. The present study was undertaken to evaluate the incidence of different draining patterns of the right pulmonary veins at the hilum by dissecting the human fixed cadaveric lungs. Clinically, pulmonary veins have been demonstrated to often play an important role in generating atrial fibrillation. Hence, it is important to look into the anatomy of the veins during MR imaging and CT angiography. In 53.8% of cases, the right superior lobar vein and right middle lobar ve
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16

Gilmetdinov, A. F., and V. P. Potanin. "Our experience of ipsilateral lobar lymph node dissection in lung cancer." Kazan medical journal 99, no. 4 (2018): 717–21. http://dx.doi.org/10.17816/kmj2018-717.

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Aim. Development of the technique for performing ipsilateral lobar lymph node dissection.
 Methods. Ipsilateral lobar lymph node dissection was performed in 40 patients diagnosed with non-small cell lung cancer when performing radical surgical treatment (lung resection with systematic lymph node dissection). First of all, ipsilateral lobar lymph node dissection was performed on the lung sample after pneumonectomy. After having developed the technique, dissection was performed during lobectomy through thoracotomy access. After having mastered the technique of performing dissection through
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17

Noone, Imelda, Mary Kate Meagher, Naomi Davey, Diarmuid O'Shea, and Tim Cassidy. "86 Haemorrhagic Stroke: What's in a Bleed?" Age and Ageing 48, Supplement_3 (2019): iii17—iii65. http://dx.doi.org/10.1093/ageing/afz103.48.

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Abstract Background Intracerebral haemorrhage (ICH) accounts for approximately 20% of all strokes and is a leading cause of disability and mortality. The distribution of the ICH(Lobar v Deep) may be explained by the two major aetiologies of ICH, Cerebral Amyloid Angiopathy (CAA) versus hypertensive (HBP) respectively. The aim of our study was to look at outcomes of lobar versus deep ICH stroke patients. Methods We analyzed data of all ICH’s admitted in 2018 using SPSS. Demography, classification of haemorrhage, hypertension, atrial fibrillation (AF) and anticoagulation history, pre and post st
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18

Yogendrakumar, Vignan, Andrew M. Demchuk, Richard I. Aviv, et al. "Location of intracerebral haemorrhage predicts haematoma expansion." European Stroke Journal 2, no. 3 (2017): 257–63. http://dx.doi.org/10.1177/2396987317715836.

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Introduction The role of intracerebral haemorrhage location in haematoma expansion remains unclear. Our objective was to assess the effect of lobar versus non-lobar haemorrhage on haematoma expansion and clinical outcome. Patients and methods We analysed data from the prospective PREDICT study where patients with intracerebral haemorrhage presenting to hospital under 6 h of symptom onset received baseline computed tomography (CT), CT angiogram, 24 h follow-up CT, and 90-day mRS. Intracerebral haemorrhage location was categorised as lobar versus non-lobar, and primary outcomes were significant
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19

Marín Gómez, Lizeth, and Juan Carlos Jiménez Salazar. "Enfisema lobar congénito." Pediatría 53, no. 3 (2020): 107–10. http://dx.doi.org/10.14295/rp.v53i3.212.

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El enfisema lobar congénito es una malformación pulmonar infrecuente caracterizada por hiperinsuflación del lóbulo pulmonar afectado, el 50 % de los pacientes presentan síntomas al nacer, los cuales pueden ser confundidos con otras entidades como infecciones respiratorias, atelectasias, neumotórax, entre otras. La radiografía de tórax es útil para su diagnóstico, sin embargo, es necesaria una tomografía de tórax para diferenciarla de otras malformaciones. En la mayoría de los casos el tratamiento de elección es la lobectomía y el neumotórax la complicación mas frecuente. Presentamos un pacient
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20

Choh, Suhil A., Naseer A. Choh, and Majid Jehangir. "Congenital lobar emphysema." Annals of Saudi Medicine 28, no. 4 (2008): 310. http://dx.doi.org/10.5144/0256-4947.2008.310.

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21

Celiksoy, Mehmet Halil, Ayhan Sogut, Recep Sancak, and Ozlem Koken. "Congenital lobar emphysema." Abant Medical Journal 3, no. 3 (2014): 289–92. http://dx.doi.org/10.5505/abantmedj.2014.24186.

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22

Seltman, Rachel E., and Brandy R. Matthews. "Frontotemporal Lobar Degeneration." CNS Drugs 26, no. 10 (2012): 841–70. http://dx.doi.org/10.2165/11640070-000000000-00000.

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23

Altınel Acoglu, Esma, Umit Murat Sahiner, Ozge Meral, Ali Koc, Mehmet Kose, and Yasemin Altuner Torun. "Congenital Lobar Emphysema." Erciyes Tıp Dergisi/Erciyes Medical Journal 37, no. 3 (2015): 122–24. http://dx.doi.org/10.5152/etd.2015.8528.

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24

Mutlu, Pınar, and Merve Ilcın Guven. "Congenital Lobar Emphysema." Respiratory Case Reports 7, no. 1 (2018): 50–53. http://dx.doi.org/10.5505/respircase.2018.55707.

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25

Korzhynskyy, Yu S., D. I. Hrytsak, V. A. Chuyko, and O. Ya Shadrin. "INFANTILE LOBAR EMPHYSEMA." Neonatology, surgery and perinatal medicine 5, no. 1(15) (2015): 108–12. http://dx.doi.org/10.24061/2413-4260.v.1.15.2015.22.

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26

Wakai, Susumu, Nahomi Kumakura, and Masakatsu Nagai. "Lobar intracerebral hemorrhage." Journal of Neurosurgery 76, no. 2 (1992): 231–38. http://dx.doi.org/10.3171/jns.1992.76.2.0231.

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✓ The authors operated consecutively on 50 patients with lobar intracerebral hemorrhage during a prospectively designed study period from January, 1986, to March, 1990. They investigated the correlations between the underlying causes and the clinicoradiographic features in 29 patients who showed no angiographic vascular abnormalities, in order to elucidate the operative indication for such cases. Patients with ruptured saccular aneurysm or trauma were not included in this study. There were 15 males and 14 females, ranging in age from 7 to 76 years (mean 52.4 years). Histological diagnoses of t
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27

Zhurylo, I. P., G. A. Sopov, A. M. Moiseev, et al. "CONGENITAL LOBAR EMPHYSEMA." Neonatology, surgery and perinatal medicine 3, no. 2(8) (2013): 44–49. http://dx.doi.org/10.24061/2413-4260.iii.2.8.2013.6.

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The experience of diagnosis andtreatment of congenital lobar emphysema (CLE) wassummarized in 5 children aged from 3 weeks old to10 months. Male patients was 3, female - 2. In fourobservations noted lesion of the upper lobe of the leftlung, and in one - the middle lobe of the right lung. CLEdiagnosed in all children after birth. The indicationfor surgery was a progressive increase of the affectedlobe. In all cases lobectomy was performed. Therewere no complications. The possibility of prenataldetection of EDF and differential diagnosis with othercongenital anomalies of the lungs was discussed.
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28

Donahoe, Laura L., and Marcelo Cypel. "Lobar Lung Transplantation." Operative Techniques in Thoracic and Cardiovascular Surgery 26, no. 4 (2021): 658–68. http://dx.doi.org/10.1053/j.optechstcvs.2021.10.003.

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29

Alghamdi, Hassan. "Recurrent Lobar Torsion." Open Journal of Thoracic Surgery 03, no. 04 (2013): 103–6. http://dx.doi.org/10.4236/ojts.2013.34021.

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30

Rabinovici, Gil D., and Bruce L. Miller. "Frontotemporal Lobar Degeneration." CNS Drugs 24, no. 5 (2010): 375–98. http://dx.doi.org/10.2165/11533100-000000000-00000.

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31

Lee, Theodore J., Jessica W. T. Leung, Gautham P. Reddy, and Michael B. Gotway. "Persistent Lobar Consolidation." Clinical Pulmonary Medicine 13, no. 4 (2006): 258–61. http://dx.doi.org/10.1097/01.cpm.0000227380.12348.80.

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32

Powers, James E., and Francis L. Counselman. "Congenital Lobar Emphysema." Pediatric Emergency Care 21, no. 11 (2005): 760–62. http://dx.doi.org/10.1097/01.pec.0000186432.82085.bf.

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33

Greene, Reginald. "Acute lobar collapse." Critical Care Medicine 27, no. 8 (1999): 1677–79. http://dx.doi.org/10.1097/00003246-199908000-00064.

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34

Sacco, Ralph L. "Lobar Intracerebral Hemorrhage." New England Journal of Medicine 342, no. 4 (2000): 276–79. http://dx.doi.org/10.1056/nejm200001273420410.

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35

Tack, D., O. Alkeilani, D. Lauwers, and C. Delcour. "Lobar Mucus Plugging." Clinical Pulmonary Medicine 8, no. 2 (2001): 115–16. http://dx.doi.org/10.1097/00045413-200103000-00009.

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36

JAFFEY, PAMELA B., PAUL W. ENGLISH, GERALD A. CAMPBELL, SANFORD A. RUBIN, and ABIDA K. HAQUE. "Escherichia Lobar Pneumonia." Southern Medical Journal 89, no. 6 (1996): 628–30. http://dx.doi.org/10.1097/00007611-199606000-00017.

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37

Berlinger, Norman T., Dennis P. Porto, and Theodore R. Thompson. "Infantile Lobar Emphysema." Annals of Otology, Rhinology & Laryngology 96, no. 1 (1987): 106–11. http://dx.doi.org/10.1177/000348948709600124.

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Infantile lobar emphysema is a symptom complex representing a spectrum of diseases characterized by overdistention of a pulmonary lobe by a check valve mechanism. The earlier in life infantile lobar emphysema presents, the more severe are the symptoms. Half of the cases appear in the first 4 weeks of life. The chest radiograph is the best diagnostic tool but can be misinterpreted. Computed tomography sometimes discloses the cause, which appears to be bronchial obstruction in 25% of cases. The bronchial obstruction may be due to intrinsic defects or to extrinsic compression. Bronchoscopy should
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38

Markowitz, Richard I., Mark R. Mercurio, Glen A. Vahjen, Ian Gross, and Robert J. Touloukian. "Congenital Lobar Emphysema." Clinical Pediatrics 28, no. 1 (1989): 19–23. http://dx.doi.org/10.1177/000992288902800104.

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39

Stiller, Kathy, Tim Geake, James Taylor, Ruth Grant, and Bob Hall. "Acute Lobar Atelectasis." Chest 98, no. 6 (1990): 1336–40. http://dx.doi.org/10.1378/chest.98.6.1336.

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40

Hussen, Waleed Mustafa, Shatha Abdulameir Alkawaz, and Laith Saleh Abood. "CONGENITAL LOBAR EMPHYSEMA." Egyptian Journal of Surgery 32, no. 2 (2013): 116–20. http://dx.doi.org/10.21608/ejsur.2013.366744.

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41

Shepherd, Ray W. "Lobar Airway Stents." Journal of Bronchology & Interventional Pulmonology 25, no. 1 (2018): 3–5. http://dx.doi.org/10.1097/lbr.0000000000000466.

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42

Bowdish, Michael E., Mark L. Barr, and Vaughn A. Starnes. "Living lobar transplantation." Chest Surgery Clinics of North America 13, no. 3 (2003): 505–24. http://dx.doi.org/10.1016/s1052-3359(03)00058-9.

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43

Silverman, Stuart G., Richard C. Pfister, Nicholas Papanicolaou, and Isabel C. Yoder. "Migratory lobar nephronia." Urologic Radiology 11, no. 1 (1989): 16–19. http://dx.doi.org/10.1007/bf02926466.

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44

Tolnay, M., and A. Probst. "Frontotemporal Lobar Degeneration." Gerontology 47, no. 1 (2001): 1–8. http://dx.doi.org/10.1159/000052763.

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45

Josephs, Keith A. "Frontotemporal Lobar Degeneration." Neurologic Clinics 25, no. 3 (2007): 683–96. http://dx.doi.org/10.1016/j.ncl.2007.03.005.

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46

Rajan, R. S., and OP Yadava. "Congenital lobar emphysema." Indian Journal of Thoracic and Cardiovascular Surgery 4, no. 1 (1985): 89–91. http://dx.doi.org/10.1007/bf02664097.

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47

McKinstry, C. S. "Acute lobar nephronia." British Journal of Radiology 58, no. 696 (1985): 1217–19. http://dx.doi.org/10.1259/0007-1285-58-696-1217.

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48

Ranke, Felipe Mussi von, Heloisa Maria Pereira Freitas, Vanessa Dinoá, Fernanda Miraldi, and Edson Marchiori. "Congenital lobar emphysema." Radiologia Brasileira 51, no. 3 (2018): 205–6. http://dx.doi.org/10.1590/0100-3984.2016.0224.

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49

Tibana, Tiago Kojun, Denise Maria Rissato Camilo, Thiago Franchi Nunes, and Edson Marchiori. "Congenital lobar emphysema." Radiologia Brasileira 52, no. 1 (2019): 62–63. http://dx.doi.org/10.1590/0100-3984.2017.0138.

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50

Glüer, Sylvia, Marc Reismann, and Benno M. Ure. "Congenital Lobar Emphysema." Annals of Thoracic Surgery 85, no. 2 (2008): 665. http://dx.doi.org/10.1016/j.athoracsur.2007.06.021.

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