Relevant bibliographies by topics / Long non-coding RNA

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Long non-coding RNA'

Author: Grafiati
Published: 4 June 2021
Last updated: 6 July 2024

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Journal articles on the topic "Long non-coding RNA"

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TAKAHASHI, Kenji, Yohei KITANO, Yuichi MAKINO, and Masakazu HANEDA. "Long non-coding RNAs in pancreatic cancer." Suizo 31, no. 1 (2016): 32–40. http://dx.doi.org/10.2958/suizo.31.32.

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Ma, Xiaoxia, Chaogang Shao, Yongfeng Jin, Huizhong Wang, and Yijun Meng. "Long non-coding RNAs." RNA Biology 11, no. 4 (April 2014): 373–90. http://dx.doi.org/10.4161/rna.28725.

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Kazimierczyk, Marek, and Jan Wrzesinski. "Long Non-Coding RNA Epigenetics." International Journal of Molecular Sciences 22, no. 11 (June 7, 2021): 6166. http://dx.doi.org/10.3390/ijms22116166.

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Long noncoding RNAs exceeding a length of 200 nucleotides play an important role in ensuring cell functions and proper organism development by interacting with cellular compounds such as miRNA, mRNA, DNA and proteins. However, there is an additional level of lncRNA regulation, called lncRNA epigenetics, in gene expression control. In this review, we describe the most common modified nucleosides found in lncRNA, 6-methyladenosine, 5-methylcytidine, pseudouridine and inosine. The biosynthetic pathways of these nucleosides modified by the writer, eraser and reader enzymes are important to understanding these processes. The characteristics of the individual methylases, pseudouridine synthases and adenine–inosine editing enzymes and the methods of lncRNA epigenetics for the detection of modified nucleosides, as well as the advantages and disadvantages of these methods, are discussed in detail. The final sections are devoted to the role of modifications in the most abundant lncRNAs and their functions in pathogenic processes.
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Stower, Hannah. "Long non-coding RNA stability." Nature Reviews Genetics 13, no. 5 (April 12, 2012): 298. http://dx.doi.org/10.1038/nrg3234.

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Beylerli, O. A., and I. F. Gareev. "Long non-coding RNA — perspectives?" Profilakticheskaya meditsina 23, no. 2 (2020): 124. http://dx.doi.org/10.17116/profmed202023021124.

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GAO, Yuan, Ning HUI, and Shan-rong LIU. "Long non-coding RNA: research progress." Academic Journal of Second Military Medical University 31, no. 7 (December 26, 2011): 790–94. http://dx.doi.org/10.3724/sp.j.1008.2011.00790.

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Hauptman, Nina, and Damjan Glavač. "Long Non-Coding RNA in Cancer." International Journal of Molecular Sciences 14, no. 3 (February 26, 2013): 4655–69. http://dx.doi.org/10.3390/ijms14034655.

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Zhang, J., X. Hao, M. Yin, T. Xu, and F. Guo. "Long non-coding RNA in osteogenesis." Bone & Joint Research 8, no. 2 (February 2019): 73–80. http://dx.doi.org/10.1302/2046-3758.82.bjr-2018-0074.r1.

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Saxena, Alka, and Piero Carninci. "Long non-coding RNA modifies chromatin." BioEssays 33, no. 11 (September 14, 2011): 830–39. http://dx.doi.org/10.1002/bies.201100084.

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Aurilia, Cinzia, Gaia Palmini, Simone Donati, Irene Falsetti, Teresa Iantomasi, and Maria Luisa Brandi. "Long non coding RNA in osteoporosis." International Journal of Bone Fragility 2, no. 3 (December 28, 2022): 102–5. http://dx.doi.org/10.57582/ijbf.220203.102.

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Osteoporosis (OP) is the most common skeletal disease, caused by a lack of balance between osteoclast and osteoblast activity. This results in erosion overriding the deposition of new bone matrix, consequently leading to low-quality bone and an increased risk of incurring fragility fractures. Dual energy X-ray absorptiometry is the gold standard for the diagnosis of OP, while anti-osteoporotic drugs are the gold standard for its treatment. However, due to limitations to their use, researchers have turned to epigenetics as a substantial source of molecules that could potentially be used as diagnostic, prognostic, and therapeutic biomarkers for OP. In particular, long non-coding RNAs (lncRNAs) possess special biological properties that could open new horizons in the field of personalized medicine. This mini review seeks to offer an overview of the studies carried out in the last year on the different lncRNAs that could be involved in the pathogenesis of OP and that could pave the way for the development of innovative therapeutic strategies for this disease.
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Dissertations / Theses on the topic "Long non-coding RNA"

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Ozes, Ali Rayet. "Targeting the long non coding RNA HOTAIR in cancer." Thesis, Indiana University, 2016. http://pqdtopen.proquest.com/#viewpdf?dispub=10154781.

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Ovarian cancer (OC) takes the lives of nearly 14,000 US women every year. Although platinum is one of the most effective drugs in treating ovarian cancer, the development of platinum resistance is one of the biggest challenges facing patients. I have shown that the long non-coding RNA HOTAIR contributes to platinum-resistant OC and determined the regulators and targets of HOTAIR during the platinum-induced DNA damage response. My published data supports the role of HOTAIR in contributing to DNA damage induced cellular senescence and secretion of pro-inflammatory cytokines leading to cisplatin resistance. My unpublished work (under review) analyzed the interaction of HOTAIR with the PRC2, its known interacting partner. In this study, I developed a novel strategy blocking HOTAIR-PRC2 interaction and resensitized ovarian tumors to platinum in mouse studies. The results offer a pre-clinical proof of concept for targeting long non-coding RNAs as a therapeutic approach and may represent a strategy to overcome chemotherapy resistance in tumors exhibiting high expression of HOTAIR, a frequent observation in high grade serous OC.

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Ard, Ryan Anthony. "Functional long non-coding RNA transcription in Schizosaccharomyces pombe." Thesis, University of Edinburgh, 2016. http://hdl.handle.net/1842/20396.

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Eukaryotic genomes are pervasively transcribed and frequently generate long noncoding RNAs (lncRNAs). However, most lncRNAs remain uncharacterized. In this work, a set of positionally conserved intergenic lncRNAs in the fission yeast Schizosaccharomyces pombe genome are selected for further analysis. Deleting one of these lncRNA genes (ncRNA.1343) exhibited a clear phenotype: increased drug sensitivity. Further analyses revealed that deleting ncRNA.1343 also disrupted a previously unannotated lncRNA, termed nc-tgp1, transcribed in the opposite orientation of the predicted ncRNA.1343 gene and into the promoter of the phosphate-responsive permease gene tgp1+. Detailed analyses revealed that the act of transcribing nc-tgp1 into the tgp1+ promoter increases nucleosome density and prevents transcription factor access. Decreased nc-tgp1 transcription permits tgp1+ expression upon phosphate starvation, while nc-tgp1 loss induces tgp1+ in repressive phosphate-rich conditions. Notably, drug sensitivity results directly from tgp1+ expression in the absence of nc-tgp1 transcription. Similarly, lncRNA transcription upstream of pho1+, another phosphate-regulated gene, increases nucleosome density and prevents transcription factor binding to repress pho1+ in phosphate-replete cells. Importantly, the regulation of tgp1+ and pho1+ by upstream lncRNA transcription occurs in the absence of RNAi and heterochromatin components. Instead, the regulation of tgp1+ and pho1+ by upstream lncRNA transcription resembles examples of transcriptional interference reported in other organisms. Thus, tgp1+ and pho1+ are the first documented examples of genes regulated by transcriptional interference in S. pombe.
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Ottway, Charlotte Jane. "Characterisation of Nespas, a non-coding imprinted RNA." Thesis, University of Oxford, 2010. http://ora.ox.ac.uk/objects/uuid:b159c1e9-8d49-460c-a808-d920e8e17779.

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Nespas is the non-coding antisense transcript of the imprinted Gnas cluster; it is expressed from the paternal allele and is located on mouse distal chromosome 2. In this thesis new transcripts of >10 kb and 0.8 kb have been identified. The 0.8 kb transcript is a spliced variant that is retained in the nucleus and its 3’ end lies approximately 30 kb from the start site. Transcription from the Nespas promoter does not proceed beyond this point. A collection of previously known splice variants have also been detected and are exported to the cytoplasm. Nespas is expressed in the embryo during the second half of gestation and peaks at 13.5 dpc. Nespas is imprinted in the placenta at 11.5, 15.5 and 17.5 dpc. The Nespastm4Jop allele, to truncate the Nespas transcript 10.5 kb from the start site, has been transmitted through the germline and a breeding colony established. Preliminary analysis shows Nespas has a regulatory function. A second targeting construct to truncate Nespas 12.5 kb from the start site has been designed and assembled to investigate whether the 3’ end of the Nespas transcript that is transcribed upstream of the Nesp promoter is required for Nespas-mediated silencing of Nesp.
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Wijesinghe, Susanne. "Role of long non-coding RNA CCDC26 in gene regulation." Thesis, University of Birmingham, 2018. http://etheses.bham.ac.uk//id/eprint/8441/.

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LncRNAs are increasingly being recognised as functionally important for regulation of biological processes. We have identified an lncRNA which we believe is integral in lineage commitment during haematopoiesis. Here, we report that CCDC26 lncRNA is a regulator of β-globin and c-MYC gene expression. Indeed, CCDC26 silencing in erythroleukemic K562 cells led to several gene expression changes. Upon further investigation of genes linked to erythropoiesis, we observed the upregulation of β-globin expression. Our results suggest that CCDC26 regulates expression of β-globin and other genes by modulating epigenetic changes which could be important in linage-commitment. We have results to suggest the expression of β-globin and c-MYC genes may be synergistic and promotes differentiation of K562. Finally, RNAseq data further supports upregulation of differentiation specific genes further underpinning our hypothesis that this lncRNA may play an important role in lineage commitment.
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Cabili, Nataly Moran. "Integrative Characterization of Human Long Non-Coding RNAs." Thesis, Harvard University, 2014. http://dissertations.umi.com/gsas.harvard:11409.

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Since its early discovery as a messenger, RNA has been shown to play a diverse set of regulatory, structural and even catalytic roles. The more recent understanding that the genome is pervasively transcribed stimulated the discovery of a new prevalent class of long non coding RNAs (lncRNAs). While these are lower abundant and relatively less conserved than other class of functional RNAs, lncRNAs are emerging as key players in different cellular processes in development and disease.
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Hammel, Alexander John. "Evolutionary conservation of long intergenic non-coding RNA genes in Arabidopsis." Thesis, University of British Columbia, 2013. http://hdl.handle.net/2429/44974.

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Keniry, Andrew James. "H19 and miR-675 : a long noncoding RNA conceals a growth suppressing microRNA." Thesis, University of Cambridge, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.609990.

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Broadbent, Kate Mariel. "The regulatory capacity of long non-coding RNA in Plasmodium falciparum malaria." Thesis, Harvard University, 2014. http://nrs.harvard.edu/urn-3:HUL.InstRepos:13065005.

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The mechanisms underpinning gene regulation in P. falciparum malaria remain largely elusive, though mounting evidence suggests a major role for epigenetic feedback. Interestingly, long non-(protein)-coding RNAs (lncRNAs) have been found to play a dominant role in initiating and guiding the transcriptional, epigenetic, and post-transcriptional status of specific loci across a broad range of organisms. LncRNAs are uniquely poised to act co-transcriptionally on neighboring loci, and/or to remain physically tethered at their site of origin, and through sequence-specific binding activities can impart temporal and spatial specificity to ubiquitously expressed nuclear protein complexes. Proteins, on the other hand, must be translated in the cytoplasm, and hence lose memory of their transcriptional origins. Encouraged by these features of lncRNAs, we set out to investigate the regulatory capacity of P. falciparum lncRNAs on a genome-wide scale. First, we surveyed transcriptional activity across approximately one quarter of the P. falciparum genome using a custom high-density DNA tiling array. We predicted a set of 60 developmentally regulated intergenic lncRNAs, and found that many of these novel loci neighbored genes involved in parasite survival or virulence pathways. Remarkably, upon further analysis of intergenic lncRNA properties, we discovered a family of twenty-two telomere-associated lncRNAs encoded in the telomere-associated repetitive element (TARE) region of P. falciparum chromosome ends. We found that each lncRNA-TARE was encoded adjacent and divergent to a subtelomeric var virulence gene. Moreover, we found that lncRNA-TARE expression was sharply induced between the parasite DNA replication and cell division cycles, that lncRNA-TARE loci contained numerous transcription factor binding sites only otherwise found in subtelomeric var promoter regions, and that the GC content and evolutionary sequence conservation of lncRNA-TAREs was similar to that of P. falciparum ribosomal RNA. Next, we set out to assemble P. falciparum intergenic lncRNA and antisense RNA transcript structures using state-of-the-art deep sequencing and computational tools. Towards this end, we harvested an unprecedented sample set that finely maps temporal changes across 56 hours of P. falciparum blood stage development, and developed and validated strand-specific, non-polyA-selected RNA sequencing methods. This enabled the annotation of over one thousand high-confidence, bona fide lncRNA transcript models, and their comprehensive global analysis. We discovered an enrichment of negatively correlated, tail-to-tail overlapping sense-antisense transcript pairs, suggesting a conserved role for antisense-mediated transcriptional interference in P. falciparum gene regulation. We also discovered a highly correlated spliced antisense counterpart to a gene required for sexual commitment, that the expression of an intriguing subset of antisense transcripts significantly dropped during parasite invasion, and that lncRNA-TARE and 'sterile' var virulence gene transcription was markedly up-regulated during parasite invasion. Lastly, we predicted over one thousand circular RNAs (circRNAs), and validated six circRNA transcript structures. Importantly, this thesis work represents the first focused investigation of lncRNAs in P. falciparum malaria, with the characterization of a compelling family of telomere-associated lncRNAs and numerous antisense RNAs. The data, methods, and results herein offer exceptional technological advancements coupled with compelling insights into the biology of the devastating human pathogen P. falciparum malaria. It is my hope that this work will facilitate future P. falciparum lncRNA functional studies and the strand-specific profiling of additional P. falciparum samples.
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Ballantyne, Margaret. "Understanding the role of long non-coding RNA (LncRNA) in vascular pathology." Thesis, University of Glasgow, 2017. http://theses.gla.ac.uk/8101/.

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Coronary heart disease is a major cause of morbidity and mortality in the Western society. In the case of severe atherosclerosis, percutaneous intervention and coronary bypass grafting remain the preferred form of surgical treatment. However, the patency of both these treatments is limited and several bypass grafts and stents fail due to neointimal formation and in stent restenosis attributable to the proliferation of VSMCs. The resultant luminal renarrowing may manifest clinically with the return of symptoms such as chest pain or shortness of breath and ultimately requires further surgical intervention. Unfortunately, current antiproliferative therapies to inhibit VSMC proliferation have off target effects and can inhibit vessel re-endothelialisation resulting in thrombus formation. As such, novel therapies that specifically target VSMC proliferation but do not affect endothelial growth are urgently required. Long non-coding RNA (lncRNA) are transcripts >200 nucleotides that have been shown to bind DNA, RNA and proteins in order to exert their function. To date a few lncRNAs have been identified that control key aspects of VSMC phenotype, including contraction, proliferation, migration and apoptosis, however, very little is known as to the role of lncRNA in the proliferative and inflammatory phenotype associated with this phenotypic switching. It was therefore hypothesised that lncRNA may be dysregulated in the setting of inflammatory and proliferative vascular pathology and may provide novel therapies to counteract VSMC proliferation and hence vascular disease. The project sought to identify lncRNA expression in quiescent, non-proliferating human saphenous vein (HSV) SMCs, and HSVSMCs that had been treated with the IL1α cytokine and PDGF growth factor. This cytokine and growth factor pair have been implicated in the synergistic activation of the NF-κB transcription factor and in the control of vascular diseases including in stent restenosis, neo intimal formation and atherosclerosis. Using RNA-sequencing, >300 lncRNAs were identified whose expression was altered in HSVSMCs following stimulation with IL1α and PDGF. These lncRNA exhibited distinct expression patterns in a tissue cohort and all showed enrichment in vascular SMCs from either an arterial or venous lineages. Experiments focused on a novel lncRNA (Ensembl: RP11-94A24.1) which showed specific expression in HSVSMCs following treatment but no expression in endothelial cells. This lncRNA was termed smooth muscle induced lncRNA enhances replication (SMILR). Following stimulation, SMILR expression was increased in both the nucleus and cytoplasm, and was detected in conditioned media from dual stimulated HSVSMCs. Furthermore, knockdown of SMILR markedly reduced cell proliferation. Mechanistically, it was noted that expression of genes proximal to SMILR were also altered by IL1α/PDGF treatment possibly indicating that these two genes are under the same promoter control, and HAS2 expression was reduced by SMILR knockdown. Additionally the proliferation of HSVSMCs was increased in a dose dependent manner following administration of a lentivirus containing the full SMILR transcript, confirming the knockdown data. In human samples, increased expression of SMILR was detected in plaque compared to adjacent non-plaque sections by qRT-PCR and following on from the detection of SMILR in conditioned media, SMILR was also detected in plasma samples from patients with inflammatory CVD. Interestingly, the levels of SMILR correlated with plasma C-reactive protein, a current biomarker capable of detecting atherosclerosis progression in patients. These results identify SMILR as a driver of VSMC proliferation and suggest that modulation of SMILR may be a novel therapeutic strategy to reduce vascular pathologies. Additionally the detection of SMILR in plasma highlights the possibility that this lncRNA may have the potential as a biomarker of vascular disease. However, further large cohort studies are required to identify this potential clinical role.
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Pettini, Tom. "The role of novel long non-coding RNAs in Hox gene regulation." Thesis, University of Manchester, 2013. https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-novel-long-noncoding-rnas-in-hox-gene-regulation(c8e44900-3ac0-40be-8ec6-b50179381d17).html.

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Whole genome transcriptome analysis has revealed that a large proportion of the genome in higher metazoa is transcribed, yet only a small proportion of this transcription is protein-coding. One possible function of non-coding transcription is that it enables complex and diverse body plans to evolve through variation in deployment of a relatively common set of protein-coding genes. Functional studies suggest that long non-coding RNAs (lncRNAs) regulate gene expression via diverse mechanisms, operating in both cis and trans to activate or repress target genes. An emerging theme common to lncRNA function is interaction with proteins that modify chromatin and mediate epigenetic regulation. The Hox gene complexes are particularly rich in lncRNAs and require precise and fine-tuned expression to deploy Hox transcription factors throughout development. Here we identify and functionally characterize two novel lncRNAs within the D. melanogaster Hox complex, in the interval between Scr and Antp. We use nascent transcript fluorescent in-situ hybridization (ntFISH) to characterize the embryonic expression patterns of each lncRNA with respect to flanking Hox genes, and to analyze co-transcription within individual nuclei. We find that the transcription of one lncRNA, ncX, is an initial response to early transcription factors and may activate Scr expression, while transcription of the other lncRNA, ncPRE is consistent with activation and/or maintenance of Scr expression. ntFISH performed in D.virilis embryos revealed the presence of a lncRNA ortholog with highly similar expression to ncX, indicating functional conservation of lncRNA transcription across ~60 million years of evolution. We identify the ncPRE lncRNA locus as a binding site for multiple proteins associated with Polycomb/Trithorax response elements (PREs/TREs) and show that DNA encoding the ncPRE lncRNA functions as a bona fide PRE, mediating trans-interactions between chromosomes and silencing of nearby genes. We find that transcription through the ncPRE DNA relieves silencing, suggesting a role for endogenous transcription of the ncPRE lncRNA in relieving Polycomb-silencing and enabling Scr activation. We demonstrate that both lncRNA transcripts are required for proper Scr expression, and over-expression of either lncRNAs from ectopic genomic loci has no effect on Scr expression, but ectopic expression at the endogenous locus is associated with ectopic Scr activation, indicating that the lncRNA-mediated regulation functions locally at the site of transcription on the chromosome. ncX may mediate transvection effects previously observed at the Scr locus, independent of the protein Zeste. Together our results support a model of competing mechanisms in the regulation of Scr expression - a background of Polycomb repression acting from the ncPRE locus, which in the first thoracic segment is counteracted by lncRNA transcription and Trithorax binding to ncPRE, enabling activation and maintenance of Scr expression. This work provides a functional insight into the complex regulatory interactions between lncRNAs and epigenetic mechanisms, essential to establish and maintain the precise expression pattern of Hox genes through development.
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Books on the topic "Long non-coding RNA"

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Rao, M. R. S., ed. Long Non Coding RNA Biology. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-5203-3.

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Zhang, Lin, and Xiaowen Hu, eds. Long Non-Coding RNAs. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1697-0.

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Ugarkovic, Durdica, ed. Long Non-Coding RNAs. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16502-3.

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Feng, Yi, and Lin Zhang, eds. Long Non-Coding RNAs. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3378-5.

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Chekanova, Julia A., and Hsiao-Lin V. Wang, eds. Plant Long Non-Coding RNAs. New York, NY: Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9045-0.

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Navarro, Alfons, ed. Long Non-Coding RNAs in Cancer. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1581-2.

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Morris, Kevin V., ed. Long Non-coding RNAs in Human Disease. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-23907-1.

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Khalil, Ahmad M., and Jeff Coller, eds. Molecular Biology of Long Non-coding RNAs. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-8621-3.

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Khalil, Ahmad M., ed. Molecular Biology of Long Non-coding RNAs. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-17086-8.

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Cao, Haiming, ed. Functional Analysis of Long Non-Coding RNAs. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1158-6.

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Book chapters on the topic "Long non-coding RNA"

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Gullerova, Monika. "Long Non-coding RNA." In Genomic Elements in Health, Disease and Evolution, 83–108. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-3070-8_4.

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Zhang, Youyou, Yi Feng, Zhongyi Hu, Xiaowen Hu, Chao-Xing Yuan, Yi Fan, and Lin Zhang. "Characterization of Long Noncoding RNA-Associated Proteins by RNA-Immunoprecipitation." In Long Non-Coding RNAs, 19–26. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3378-5_3.

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Wu, Mengshi, Dan Peng, and Xiaomin Zhong. "Exploration of Circular RNA Interactomes by RNA Pull-Down Method." In Long Non-Coding RNAs, 203–8. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1697-0_18.

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Lai, Lan-Tian, Zhenyu Meng, Fangwei Shao, and Li-Feng Zhang. "Simultaneous RNA–DNA FISH." In Long Non-Coding RNAs, 135–45. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3378-5_11.

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Lai, Lan-Tian, Zhenyu Meng, Fangwei Shao, and Li-Feng Zhang. "Simultaneous RNA-DNA FISH." In Long Non-Coding RNAs, 111–21. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1697-0_11.

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Wijesinghe, Susanne N., Mark A. Lindsay, and Simon W. Jones. "Long Non-coding RNAs in Rheumatology." In Long Noncoding RNA, 35–70. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-92034-0_4.

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Jiang, Junjie, Tianli Zhang, Yutian Pan, Zhongyi Hu, Jiao Yuan, Xiaowen Hu, Lin Zhang, and Youyou Zhang. "Characterization of Long Non-coding RNA Associated Proteins by RNA-Immunoprecipitation." In Long Non-Coding RNAs, 19–26. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1697-0_3.

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Dhaliwal, Navroop K., and Jennifer A. Mitchell. "Nuclear RNA Isolation and Sequencing." In Long Non-Coding RNAs, 63–71. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3378-5_7.

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Dhaliwal, Navroop K., and Jennifer A. Mitchell. "Nuclear RNA Isolation and Sequencing." In Long Non-Coding RNAs, 75–83. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1697-0_8.

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Harvey, Samuel E., and Chonghui Cheng. "Methods for Characterization of Alternative RNA Splicing." In Long Non-Coding RNAs, 229–41. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3378-5_18.

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Conference papers on the topic "Long non-coding RNA"

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Cristiano, Francesca, Pierangelo Veltri, Mattia Prosperi, and Giuseppe Tradigo. "On the identification of long non-coding RNAs from RNA-seq." In 2016 IEEE International Conference on Bioinformatics and Biomedicine (BIBM). IEEE, 2016. http://dx.doi.org/10.1109/bibm.2016.7822675.

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Manchon, Laurent, Audrey Vautrin, Jamal Tazi, Aude Garcel, and Noelie Campos. "Targeting Long Non-Coding RNA splicing by novel candidate drug." In 2019 IEEE International Conference on Bioinformatics and Biomedicine (BIBM). IEEE, 2019. http://dx.doi.org/10.1109/bibm47256.2019.8982977.

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Chakrabortty, Sudipto K., Lisa Bedford, Hidefumi Uchiyama, Vasisht Tadigotla, Michael D. Valentino, Dominik Grimm, Dalin Chan, Sunita Badola, Graham Brock, and Johan Skog. "Abstract 5686: Long RNA sequencing of human plasma exosomes reveals full coverage of diverse protein coding and long non coding RNA." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-5686.

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Chiyomaru, Takeshi, Soichiro Yamamura, Shinichiro Fukuhara, Takashi Kinoshita, Shahana Majid, Sharanjot Saini, Inik Chang, et al. "Abstract 4374: Genistein suppresses prostate cancer regulating long non-coding RNA." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-4374.

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Omura, J., K. Habbout, T. Shimauchi, S. Breuils-Bonnet, E. Tremblay, S. Martineau, V. Nadeau, et al. "Long Non-Coding RNA H19 Promotes Right Ventricular Failure in PAH." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a2496.

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Al Mamun, Abdullah, and Ananda Mohan Mondal. "Long Non-coding RNA Based Cancer Classification using Deep Neural Networks." In BCB '19: 10th ACM International Conference on Bioinformatics, Computational Biology and Health Informatics. New York, NY, USA: ACM, 2019. http://dx.doi.org/10.1145/3307339.3343249.

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Pedrini, Fabiola, Fabian Rose, Nada Ekiaby, Sofia Weiler, Marcell Toth, Bruno Köhler, Anna Saborowski, et al. "Oncogene-induced long non-coding RNA (lncRNA) signatures in liver cancer." In 40. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag, 2024. http://dx.doi.org/10.1055/s-0043-1777604.

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Chiquitto, Alisson G., Lucas Otavio L. Silva, Liliane S. Oliveira, Douglas S. Domingues, and Alexandre R. Paschoal. "Impact of sequencing technologies on long non-coding RNA computational identification." In 2022 IEEE International Conference on Bioinformatics and Biomedicine (BIBM). IEEE, 2022. http://dx.doi.org/10.1109/bibm55620.2022.9995443.

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Button, A. C., R. Z. Blumhagen, I. V. Yang, and D. A. Schwartz. "Long Non-coding RNA in IPF: Regulatory Players in Lung Fibrosis." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a2583.

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O'Brien, Stephen J., Theodore Kalbfleisch, Sudhir Srivastava, Shesh Rai, and Susan Galandiuk. "Abstract 1817: Differential expression of long non-coding RNA in colon adenocarcinoma RNA-sequence data set." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-1817.

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Reports on the topic "Long non-coding RNA"

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Liao, Jianhua, Jingting Liu, Baoqing Liu, Chunyan Meng, and Peiwen Yuan. Effect of OIP5-AS1 on clinicopathological characteristics and prognosis of cancer patients: a meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2022. http://dx.doi.org/10.37766/inplasy2022.10.0118.

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Abstract:
Review question / Objective: According to recent studies, long non-coding RNA (lncRNAs) i.e., OPA-interacting protein 5 antisense RNA 1 (OIP5-AS1) has an important role in various carcinomas. However, its role in the cancer is contradictory. Therefore, we aimed to evaluate the link between OIP5-AS1 and cancer patients' clinicopathological characteristics and prognosis to better understand OIP5-AS1's role in cancer. Condition being studied: Reported studies have revealed that long non-coding RNA (lncRNAs) are considerably involved in crucial physiological events in several carcinomas, it can inhibit or promote the occurrence and development of tumors by changing the sequence and spatial structure, modulating epigenetic, regulating the expression level and interacting with binding proteins. However, the mechanism of cancer regulation via lncRNAs was incompletely understood. Hence, clarifying the application value of lncRNAs in preclinical and clinical disease diagnosis and treatment was therefore the prime objective in the field of cancer research at the time.
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Liu, Miao, Tihong Liang, Fengyan Wang, Hua Yang, Xu Ning, and Hong Sun. Prognostic value of long non-coding RNA PVT1 as a potential biomarker in osteosarcoma: A protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2020. http://dx.doi.org/10.37766/inplasy2020.9.0050.

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Tianzi, Zhang. The Emerging Roles of Long Non-coding RNAs in the Pathogenesis of Breast Cancer. Envirarxiv, November 2022. http://dx.doi.org/10.55800/envirarxiv488.

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Zhong, Xiaoling, Qin Guo, Jing Zhao, Yinyue Li, Xue Li, Min Ren, and Min Shu. Diagnostic significance of long non-coding RNAs expression in TB patients: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, July 2020. http://dx.doi.org/10.37766/inplasy2020.7.0043.

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Zhou, Xuefeng, Wenjing Liu, Zhenhuan Yang, Wei'e Zhou, and Ping Li. Long non-coding RNAs, one of candidate biomarkers in diabetic kidney disease A systematic review protocol of profiling studies. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2020. http://dx.doi.org/10.37766/inplasy2020.11.0136.

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Wu, Zilong, Zihao Xu, Boyao Yu, Jing tao Zhang, and Bentong Yu. The potential diagnostic value of exosomal long non-coding RNAs in solid tumours: a meta-analysis and systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2020. http://dx.doi.org/10.37766/inplasy2020.6.0083.

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You might also be interested in the extended bibliographies on the topic 'Long non-coding RNA' for particular source types:
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