Relevant bibliographies by topics / Long Noncoding

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Long Noncoding'

Author: Grafiati
Published: 4 June 2021
Last updated: 11 February 2022

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Journal articles on the topic "Long Noncoding"

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Boon, Reinier A., Nicolas Jaé, Lesca Holdt, and Stefanie Dimmeler. "Long Noncoding RNAs." Journal of the American College of Cardiology 67, no. 10 (March 2016): 1214–26. http://dx.doi.org/10.1016/j.jacc.2015.12.051.

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Gough, N. R. "Painful Long Noncoding RNA." Science Signaling 6, no. 287 (August 6, 2013): ec181-ec181. http://dx.doi.org/10.1126/scisignal.2004591.

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Wu, Connie, and Pankaj Arora. "Long Noncoding Mhrt RNA." Circulation: Cardiovascular Genetics 8, no. 1 (February 2015): 213–15. http://dx.doi.org/10.1161/circgenetics.115.001019.

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Yoon, Je-Hyun, Jiyoung Kim, and Myriam Gorospe. "Long noncoding RNA turnover." Biochimie 117 (October 2015): 15–21. http://dx.doi.org/10.1016/j.biochi.2015.03.001.

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Wang, Yang-Ning-Zhi, Kun Shan, Mu-Di Yao, Jin Yao, Jia-Jian Wang, Xiang Li, Ban Liu, et al. "Long Noncoding RNA-GAS5." Hypertension 68, no. 3 (September 2016): 736–48. http://dx.doi.org/10.1161/hypertensionaha.116.07259.

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Zhang, Xu, and Wenqian Hu. "Long noncoding RNAs in hematopoiesis." F1000Research 5 (July 20, 2016): 1771. http://dx.doi.org/10.12688/f1000research.8349.1.

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Mammalian development is under tight control to ensure precise gene expression. Recent studies reveal a new layer of regulation of gene expression mediated by long noncoding RNAs. These transcripts are longer than 200nt that do not have functional protein coding capacity. Interestingly, many of these long noncoding RNAs are expressed with high specificity in different types of cells, tissues, and developmental stages in mammals, suggesting that they may have functional roles in diverse biological processes. Here, we summarize recent findings of long noncoding RNAs in hematopoiesis, which is one of the best-characterized mammalian cell differentiation processes. Then we provide our own perspectives on future studies of long noncoding RNAs in this field.
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Mueller, K. L. "Long noncoding RNAs in inflammation." Science 352, no. 6281 (March 31, 2016): 48–49. http://dx.doi.org/10.1126/science.352.6281.48-e.

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Zhang, Yang, Xiao-Ou Zhang, Tian Chen, Jian-Feng Xiang, Qing-Fei Yin, Yu-Hang Xing, Shanshan Zhu, Li Yang, and Ling-Ling Chen. "Circular Intronic Long Noncoding RNAs." Molecular Cell 51, no. 6 (September 2013): 792–806. http://dx.doi.org/10.1016/j.molcel.2013.08.017.

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Jian, Liguo, Dongdong Jian, Qishan Chen, and Li Zhang. "Long Noncoding RNAs in Atherosclerosis." Journal of Atherosclerosis and Thrombosis 23, no. 4 (2016): 376–84. http://dx.doi.org/10.5551/jat.33167.

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Atianand, Maninjay K., Daniel R. Caffrey, and Katherine A. Fitzgerald. "Immunobiology of Long Noncoding RNAs." Annual Review of Immunology 35, no. 1 (April 26, 2017): 177–98. http://dx.doi.org/10.1146/annurev-immunol-041015-055459.

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Dissertations / Theses on the topic "Long Noncoding"

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Engreitz, Jesse M. (Jesse Michael). "Genome regulation by long noncoding RNAs." Thesis, Massachusetts Institute of Technology, 2016. http://hdl.handle.net/1721.1/104616.

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Thesis: Ph. D. in Bioinformatics and Integrative Genomics, Harvard-MIT Program in Health Sciences and Technology, 2016.
Cataloged from PDF version of thesis.
Includes bibliographical references.
Our genomes encode the molecular information that gives rise to life, yet we are just beginning to unravel how this information is organized, interpreted, and regulated. While the human genome contains -20,000 protein-coding genes, mammalian genomes also produce thousands of long non-coding RNAs (lncRNAs), some of which are now known to play essential roles in diverse biological processes including cellular differentiation and human disease. Recent studies show that many lncRNAs localize to the nucleus and interact with chromatin regulatory complexes, suggesting that some lncRNAs may represent a crucial missing component in our understanding of genome regulation. To test whether lncRNAs localize to and regulate specific sites in the genome, we developed genome-wide approaches to map lncRNA interactions with chromatin. Through studies of three conserved lncRNAs, we demonstrate that lncRNAs can exploit the three-dimensional architecture of the genome to identify their regulatory targets and, in turn, actively manipulate genome architecture to form subcompartments containing co-regulated genes. Thus, lncRNAs have unique capabilities as dynamic regulators that can locally amplify epigenetic signals. We next explored whether this model might apply to other long noncoding RNAs, many of which are not conserved across species and thus whose functions remain unclear. Through genetic dissection of their local regulatory functions, we show that many of these genomic loci participate in the local regulation of gene expression, but that these functions do not involve the IncRNA transcripts themselves. Instead, multiple mechanisms associated with RNA production including their promoters, the process of transcription, and RNA splicing - act in local networks of regulatory connections between spatially proximal genes, both protein-coding and noncoding. These findings reveal novel mechanistic explanations for the functions and evolution of noncoding transcription in mammalian genomes. Together these studies suggest a model in which mammalian gene regulation is organized into local neighborhoods defined by the spatial architecture of the genome. Within these neighborhoods, lncRNAs and DNA regulatory elements may function cooperatively to coordinate local gene expression. Dissecting this fundamental model for genome regulation may enable manipulation of the processes that interpret our genome sequence and galvanize efforts to develop new treatments for human disease.
by Jesse M. Engreitz.
Ph. D. in Bioinformatics and Integrative Genomics
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Lagarde, Julien. "Genomic Characterization of Human Long Noncoding RNAs." Doctoral thesis, Universitat de Barcelona, 2020. http://hdl.handle.net/10803/668687.

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The human genome contains an astonishingly large fraction of noncoding DNA, which is pervasively transcribed into thousands of long noncoding RNAs (lncRNAs) -- long transcripts with no discernible protein-coding potential. However, little is known about lncRNAs' biological functions, and their genome annotations show evident signs of inadequacy: existing gene models are sketchy, and many lncRNAs remain uncatalogued. This annotation incompleteness hampers lncRNA functional characterization, notably by failing to accurately describe gene boundaries. To address this issue, the present work aims to advance towards a complete and accurate annotation of lncRNA genes in the human genome. Using a high-throughput, targeted long-read transcriptome sequencing methodology, this study uncovers thousands of novel lncRNAs, approximately doubling the annotated transcript complexity within targeted loci. The method presented vastly outperforms competing techniques in accuracy, and precisely maps many previously unknown, strongly supported lncRNA transcript boundaries. This augmented catalog provides the most definitive view of the genomic properties of lncRNAs to date, while contributing a robust foundation for future lncRNA functional characterization.
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Mohammad, Faizaan. "Long Noncoding RNA Mediated Regulation of Imprinted Genes." Doctoral thesis, Uppsala universitet, Institutionen för genetik och patologi, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-128882.

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Genomic imprinting is an epigenetic phenomenon that causes a subset of mammalian genes to be expressed from only one allele in a parent-of-origin manner. The defects in the imprinting regulation result in disorders that affect development, growth and metabolism. We have used the Kcnq1 imprinted cluster as a model to understand the mechanism of imprinted gene regulation. The imprinting at the Kcnq1 locus is regulated by a long noncoding RNA, Kcnq1ot1, whose transcription on the paternal chromosome is associated with the silencing of at least eight neighboring genes. By destabilizing Kcnq1ot1 in an episomal system, we have conclusively shown that it is the RNA and not the process of transcription that is required for the gene silencing in cis. Kcnq1ot1 RNA interacts with the chromatin modifying enzymes such as G9a and Ezh2 and recruits them to imprinted genes to establish repressive chromatin compartment and gene silencing. Using the episomal system, we have identified an 890 bp silencing domain (SD) at the 5’ end of Kcnq1ot1 RNA, which is required for silencing of neighboring reporter genes. The deletion of the SD in the mouse resulted in the relaxation of imprinting of ubiquitously imprinted genes (Cdkn1c, Kcnq1, Slc22a18, and Phlda2) as well as reduced DNA methylation over the somatic DMRs associated with the ubiquitously imprinted genes. Moreover, Kcnq1ot1 RNA interacts with Dnmt1 and recruits to the somatic DMRs and this recruitment was significantly affected in the SD mutant mice. By using a transgenic mouse, we have conditionally deleted Kcnq1ot1 promoter at different developmental stages and demonstrated that Kcnq1ot1 maintains imprinting of the ubiquitously imprinted genes by regulating DNA methylation over the somatic DMRs. Kcnq1ot1 is dispensable for the maintenance of repressive histone marks and the imprinting of placental-specific imprinted genes (Tssc4 and Osbpl5). In conclusion, we have described the mechanisms by which Kcnq1ot1 RNA establishes and maintains expression of multiple imprinted genes in cis.
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Richards, Edward J. "Function of Long Noncoding RNAs in Breast Cancer." Scholar Commons, 2015. http://scholarcommons.usf.edu/etd/5767.

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Breast cancer is a disease that will be diagnosed in about 1 in 10 women throughout their lifetime. The majority of breast cancers are originated from the epithelial cells of the mammary ducts, and this occurrence can be due to several factors including hereditary and acquired mutation. There are several major breast cancer subtypes, including estrogen receptor-α (ERα)-positive, HER2-enriched and triple-negative (TNBC). Patients diagnosed with ER+ tumors are generally treated with estrogen blockers (e.g., tamoxifen, letrozole and fulvestrant). Patients with HER2+ tumors are commonly administered with drugs that block HER2 signaling (e.g., trastuzumab) or inhibit HER2’s tyrosine kinase activity (e.g., lapatinib). For patients with TNBC, chemotherapies such as taxanes and anthracyclines are standard of care therapies. However, for each breast cancer subtype, a significant number of patients develop resistance to these therapies and eventually die from metastasis, a process which accounts for ~90% of breast cancer mortality. Currently, metastatic breast cancer is incurable, and the short median survival of 3 years for patients with metastatic breast cancer has not significantly changed in over 20 years. Therefore identification of new molecules that are involved in breast cancer metastasis and development of more precisely targeted therapeutic strategies are urgently needed to improve the clinical outcome for this disease. The transforming growth factor pathway beta (TGFβ) pathway has been show to play a key role in metastasis through induction of epithelial-mesenchymal transition (EMT), cell migration and invasion. Over more than a decade, this pathway has been studied across several cancers and it is now better established that it has context-dependent tumor suppressive and oncogenic qualities. In the early stages of breast cancer, TGFβ pathway is a suppressor of benign and early stage tumor growth. However, as disease progresses and corresponding levels of TGFβ ligands become elevated, a “switch” will take place and promote oncogenic phenotypes like EMT and cancer cell stemness which drive metastasis. Long noncoding RNAs (lncRNAs) are an emerging subclass of RNA molecules in cancer biology. LncRNAs are >200nt and can influence target gene expression locally in “cis”, or along a distant chromosome in “trans”, through various mechanisms and interactions with other biological molecules. The contribution of TGFβ-regulated lncRNAs to associated phenotypes like EMT and cancer cell stemness has not been very well studied. The aim of this doctoral dissertation is to address the functional and mechanistic roles of lncRNAs in these processes. Using a well-established TGFβ-induced EMT model (e.g., mouse mammary epithelial cell NMuMG treated with TGFβ, we have identified 3 conserved lncRNAs (lncRNA-HIT, WDFY3-AS2 and TIL) that are significantly upregulated upon TGFβ-induced EMT. They all mediate TGFβ-induced EMT, cell migration and invasion. Overexpression of these lncRNAs is frequently detected during the breast cancer progression and is associated with high grade and late stage of breast cancer as well as metastatic lesion. We have also demonstrated that lncRNA-HIT positively regulates HOXA13 through “cis” mechanism and that WDFY3-AS2 induces WDFY3 and STAT3 expression at mRNA level by direct interaction with hnRNP-R. Interestingly, TIL stimulates C-MYC protein but not mRNA expression by promoting Akt phosphorylation of NF90 leading to its translation from the nucleus to cytosol where NF90 binds to C-MYC mRNA and enhances C-MYC translation. Importantly, we have shown that knockdown of lncRNA-HIT and WDFY3-AS2 significantly reduces breast cancer growth and lung metastasis in orthotopic breast cancer model. These findings indicate that these TGF-induced lncRNAs play critical role in EMT, metastasis, and are relevant in human patient tumors. Therefore, it is important to consider utilizing these molecules for clinical applications like diagnosis, monitoring recurrence, predicting a response to therapy, and even as a direct target for therapeutic intervention.
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Geisler, Sarah J. "Decapping of Long Noncoding RNAs Regulates Inducible Genes." Case Western Reserve University School of Graduate Studies / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=case1340141951.

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Young, Rob. "Evolution and function of long noncoding RNAs in Drosophila." Thesis, University of Oxford, 2011. http://ora.ox.ac.uk/objects/uuid:0eee0960-fe31-41ca-a6f9-0b29e0b9fed9.

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Not all transcribed DNA encodes protein, and some of these noncoding RNAs (ncRNAs), such as roX1 and roX2, may play important roles in the cell. The functional roles of the majority of these, however, remain largely unknown. In this thesis, I first used EST and mRNA evidence to define 2,788 lincRNA loci within the Drosophila melanogaster genome. I suggest that up to 1,652 of these are functional, as 1,411 show evidence for significant evolutionary constraint while 241 fast-evolving loci are enriched in short RNA species. A distinct set of 1,119 lincRNA loci were defined by RNA-seq, the vast majority of which show clear primary sequence constraint. Their expression profiles and enrichment in particular chromatin domains indicate that these lincRNAs are likely involved in developmental regulation. I also identified 42 potential analogous lincRNAs with shared genomic locations between Drosophila and mouse. Constrained, non-embryonic lincRNAs defined by ESTs are transcribed preferentially in the vicinity of protein-coding genes encoding transcription factors and I demonstrated that one of these, which I name dEvf-2, positively regulates the expression of its genomically adjacent transcription factor, Dll, in cell culture. Finally, I used a reverse genetics approach to search for lincRNA promoter mutations and examined the effect of these on lincRNA expression. My findings suggest that many, previously unknown, functional lincRNAs exist within the Drosophila genome and are worthy of further in-depth experimental investigation.
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Liapis, Stephen Constantine. "Discovery and In Vivo Characterization of Long Noncoding RNAs." Thesis, Harvard University, 2016. http://nrs.harvard.edu/urn-3:HUL.InstRepos:33493297.

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The noncoding genome, or the portion of the genome that does not encode for proteins, encompasses >95% of the human genome. It has been found that the majority of disease-associated genetic variants identified by genome-wide association studies (GWAS) are located in this noncoding 95%, where they have the potential to affect regions that control transcription (promoters, enhancers) and noncoding RNAs that also can influence gene expression. The discovery of these alterations has already contributed to a better understanding of the etiology of human diseases and has begun to yield insight into the function of these noncoding loci I am interested in studying how the noncoding genome functions and contributes to human development and disease pathology, especially when it is considered that our understanding of human disease is almost entirely contained within the realm of the <5% of the genome that is protein coding. Toward this end, I have focused my studies on one part of the noncoding genome, long noncoding RNAs. In order to identify whether long noncoding RNAs are important for mammalian development and disease, our lab created a set of lincRNA knockout animal models in which a cassette expressing beta-galactosidase (lacZ) replaces the lincRNA DNA sequence. I have used these models for the in vivo characterization of several lincRNAs, including Fendrr in the lungs, Brn1b in the brain, Tug1 in the testes, and Cox2 in the innate immune system. Each of these studies reveals perturbations in development induced by loss of function of the respective lincRNA locus, and demonstrates promising potential for further examination of the role these molecules play in human disease.
Biology, Molecular and Cellular
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Jacob, Mathieu. "Functional Remodelling of the Nucleolus by Long Noncoding RNA." Thesis, Université d'Ottawa / University of Ottawa, 2013. http://hdl.handle.net/10393/30288.

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The nucleolus is a plurifunctional organelle in which structure and function are intimately linked. Though it is primarily known as the site of ribosomal biogenesis, the nucleolus is also capable of orchestrating the immobilization of a broad range of proteins under specific environmental conditions. This process, known as nucleolar sequestration, contributes to cell viability under stress. Despite the importance of this post-translational regulatory pathway, very little is known about the mechanisms that govern it. Here, we show that heat shock and acidosis, two physiological stimuli associated with nucleolar sequestration, induce the expression of long noncoding RNA (lncRNA) from stimulus-specific loci of the ribosomal intergenic spacer (IGS). These lncRNAs, in turn, immobilize proteins encoding a nucleolar detention sequence (NoDS) within a compartment of the nucleolus termed the detention centre (DC). The DC is a spatially and dynamically distinct region, characterized by an 8-anilino-1-naphthalenesulfonate (ANS)-positive hydrophobic signature. Its formation is accompanied by a redistribution of nucleolar factors and an arrest in ribosomal biogenesis. Silencing of regulatory IGS lncRNA prevents the creation of this structure and allows the nucleolus to retain its tripartite organization and transcriptional activity. Signal termination causes a decrease in IGS transcript levels and a return to the active nucleolar conformation. We propose that the induction of IGS lncRNA, by environmental signals, operates as a molecular switch that regulates the structure and function of the nucleolus.
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Chodroff, Rebecca A. "Characterization of long noncoding RNAs in vertebrate brain development and evolution." Thesis, University of Oxford, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558292.

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Background: Long considered to be the building block of life, it is now apparent that protein is only one of many functional products generated by the eukaryotic genome. Indeed, more of the mammalian genome is transcribed into noncoding than into protein-coding sequence. This extensive and interleaved network of noncoding transcripts partially emerges from evolutionarily conserved genomic sequence, emphasizing its potential biological relevance. Nevertheless, the biological function of the vast majority of eutherian long intergenic noncoding RNAs (lincRNAs) has not been experimentally validated and the relationship between evolutionary sequence constraint and the role of non coding transcribed sequence remains unclear. To clarify the implications of evolutionary sequence conservation for biological function of lincRNAs, this thesis aims to (l) determine whether evolutionary sequence conservation is more often directed towards particular regions involved in the transcription of lincRNA loci, such as promoter and/or intron-exon boundaries; (2) identify and characterise the tissue expression patterns of lincRNA orthologs across diverse amniotes, ranging from mouse to chicken; and (3) describe the biological functions (if any) of four highly conserved brain-expressed lincRNAs. Results: Here, we performed a multi-disciplinary study of four highly conserved and brain-expressed transcripts selected from a list of mouse long intergenic noncoding RNA (lincRNA) loci that generally show pronounced evolutionary constraint within their putative promoter regions and across exon-intron boundaries. We identified some of the first lincRNA orthologs present in birds (chicken), marsupial (opossum) and eutherian mammals (mouse), and investigated whether they exhibit conservation of brain expression. In contrast to conventional protein-coding genes, the sequences, transcriptional start sites, exon structures and lengths for these noncoding genes are all highly variable. In a series of preliminary experiments, we found that these lincRNAs do not significantly contribute to global transcriptional regulation within a mouse cell line. Furthermore, a transgenic mouse model with a targeted deletion of one of these lincRNAs did not present a noticeable phenotype, suggesting that this lincRNA is not critical for survival. Conclusions: We identified four lincRNAs with evolutionary conservation in ex on structure and transcription, and similarities in brain expression pattern during embryonic and early postnatal stages across diverse amniotes. While tissue-specific expression patterns and evolutionary sequence constraint are suggestive of function, preliminary experiments investigating each transcripts' role did not provide significant proof for biological function. Nevertheless, the high levels of evolutionary sequence conservation and specific brain expression patterns among these four lincRNAs warrant further experimental inquiry.
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Keniry, Andrew James. "H19 and miR-675 : a long noncoding RNA conceals a growth suppressing microRNA." Thesis, University of Cambridge, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.609990.

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Books on the topic "Long Noncoding"

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Kurokawa, Riki, ed. Long Noncoding RNAs. Tokyo: Springer Japan, 2015. http://dx.doi.org/10.1007/978-4-431-55576-6.

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Jurga, Stefan, and Jan Barciszewski, eds. The Chemical Biology of Long Noncoding RNAs. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-44743-4.

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Long Noncoding Rnas. Springer, 2011.

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Long Noncoding RNAs in Plants. Elsevier, 2021. http://dx.doi.org/10.1016/c2019-0-03200-1.

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Kurokawa, Riki. Long Noncoding RNAs: Structures and Functions. Springer, 2015.

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Jurga, Stefan, and Jan Barciszewski. The Chemical Biology of Long Noncoding RNAs. Springer, 2020.

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Post-transcriptional Regulation through Long Noncoding RNAs (lncRNAs). MDPI, 2021. http://dx.doi.org/10.3390/books978-3-0365-1217-4.

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Huang, Yingqun, Romano Regazzi, and William Cho, eds. Emerging Roles of Long Noncoding RNAs in Neurological Diseases and Metabolic Disorders. Frontiers Media SA, 2015. http://dx.doi.org/10.3389/978-2-88919-571-8.

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Khalil, Ahmad M., and Jeff Coller. Molecular Biology of Long Non-coding RNAs. Springer, 2013.

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Khalil, Ahmad M. Molecular Biology of Long Non-coding RNAs. Springer, 2019.

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Book chapters on the topic "Long Noncoding"

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Schein, Aleks, and Piero Carninci. "Complexity of Mammalian Transcriptome Analyzed by RNA Deep Sequencing." In Long Noncoding RNAs, 3–22. Tokyo: Springer Japan, 2015. http://dx.doi.org/10.1007/978-4-431-55576-6_1.

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Kotake, Yojiro, and Masatoshi Kitagawa. "Regulation of pRB and p53 Pathways by the Long Noncoding RNAs ANRIL, lincRNA-p21, lincRNA-RoR, and PANDA." In Long Noncoding RNAs, 175–89. Tokyo: Springer Japan, 2015. http://dx.doi.org/10.1007/978-4-431-55576-6_10.

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Takayama, Ken-ichi, and Satoshi Inoue. "The Role of Androgen-Regulated Long Noncoding RNAs in Prostate Cancer." In Long Noncoding RNAs, 191–210. Tokyo: Springer Japan, 2015. http://dx.doi.org/10.1007/978-4-431-55576-6_11.

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Allison, Karmel A., and Christopher K. Glass. "Macrophage Activation as a Model System for Understanding Enhancer Transcription and eRNA Function." In Long Noncoding RNAs, 211–29. Tokyo: Springer Japan, 2015. http://dx.doi.org/10.1007/978-4-431-55576-6_12.

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Jin, Chunyu, and Michael G. Rosenfeld. "Long Noncoding RNA Functions as a Regulator for Steroid Hormone Receptor-Related Breast and Prostate Cancers." In Long Noncoding RNAs, 231–49. Tokyo: Springer Japan, 2015. http://dx.doi.org/10.1007/978-4-431-55576-6_13.

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Pandian, Ganesh N., Junetha Syed, and Hiroshi Sugiyama. "Synthetic Strategies to Identify and Regulate Noncoding RNAs." In Long Noncoding RNAs, 23–43. Tokyo: Springer Japan, 2015. http://dx.doi.org/10.1007/978-4-431-55576-6_2.

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Katahira, Masato. "Structure and Interaction with Protein of Noncoding RNA: A Case for an RNA Aptamer Against Prion Protein." In Long Noncoding RNAs, 47–56. Tokyo: Springer Japan, 2015. http://dx.doi.org/10.1007/978-4-431-55576-6_3.

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Oyoshi, Takanori. "Characterization of G-Quadruplex DNA- and RNA-Binding Protein." In Long Noncoding RNAs, 57–65. Tokyo: Springer Japan, 2015. http://dx.doi.org/10.1007/978-4-431-55576-6_4.

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Kurokawa, Riki. "Initiation of Transcription Generates Divergence of Long Noncoding RNAs." In Long Noncoding RNAs, 69–91. Tokyo: Springer Japan, 2015. http://dx.doi.org/10.1007/978-4-431-55576-6_5.

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Kumon, Tomohiro, and Kunihiro Ohta. "Beneath the Veil of Biological Complexity There Lies Long Noncoding RNA: Diverse Utilization of lncRNA in Yeast Genomes." In Long Noncoding RNAs, 93–110. Tokyo: Springer Japan, 2015. http://dx.doi.org/10.1007/978-4-431-55576-6_6.

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Conference papers on the topic "Long Noncoding"

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Singh, Vikram, and Ramakrishna Ramaswamy. "Spectral analysis of long noncoding RNAs." In Annual International Conference on BioInformatics and Computational Biology & Annual International Conference on Advances in Biotechnology. Global Science and Technology Forum, 2011. http://dx.doi.org/10.5176/978-981-08-8119-1_bicb27.

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Chang, Howard Y. "Abstract IA02: Genome regulation by long noncoding RNAs." In Abstracts: AACR Special Conference on Noncoding RNAs and Cancer: Mechanisms to Medicines; December 4-7, 2015; Boston, MA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.nonrna15-ia02.

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Chang, Howard Y. "Abstract SY31-03: Genome regulation by long noncoding RNAs." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-sy31-03.

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Diederichs, Sven. "Abstract IA08: Functional long noncoding RNAs in lung cancer." In Abstracts: AACR Special Conference on Noncoding RNAs and Cancer: Mechanisms to Medicines; December 4-7, 2015; Boston, MA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.nonrna15-ia08.

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Chang, Howard Y. "Abstract SY25-01: Genome regulation by long noncoding RNAs." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-sy25-01.

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Wang, Bixuan. "Aberrant expression of long noncoding RNAs in bladder cancer." In 3RD INTERNATIONAL CONFERENCE ON FRONTIERS OF BIOLOGICAL SCIENCES AND ENGINEERING (FBSE 2020). AIP Publishing, 2021. http://dx.doi.org/10.1063/5.0048430.

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"Identification and structural features analysis of long noncoding RNAs." In Plant Genetics, Genomics, Bioinformatics, and Biotechnology. Novosibirsk ICG SB RAS 2021, 2021. http://dx.doi.org/10.18699/plantgen2021-163.

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HY, Chang. "Abstract MS1-2: Long Noncoding RNAs in Breast Cancer Progression." In Abstracts: Thirty-Third Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 8‐12, 2010; San Antonio, TX. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/0008-5472.sabcs10-ms1-2.

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Alanisi, Entkhab M., Jiuhui Wang, Yande Guo, and Daotai Nie. "Abstract 2463: Long noncoding RNAs in tumor responses toward lovastatin." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-2463.

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Yang, J., L. Kaphalia, W. J. Calhoun, and A. Brasier. "Long Noncoding RNA Regulates IRF1/IFNL Responses to Respiratory Viruses." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6168.

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Reports on the topic "Long Noncoding"

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Xu, Jianhao, Fang Cao, Yongwei Hu, and Zaichang Chen. Circulating long noncoding RNAs as potential biomarkers for stomach cancer: A systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, February 2021. http://dx.doi.org/10.37766/inplasy2021.2.0079.

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Liang, Xuejun, Hanlong Zhu, Guoxing Tang, Wencai LI, Lei Ye, and Shaopei Shi. Clinicopathological significance and prognostic value of long noncoding RNA MIAT in human Cancers: a meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2021. http://dx.doi.org/10.37766/inplasy2021.9.0076.

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