Academic literature on the topic 'Lung congesti'
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Journal articles on the topic "Lung congesti"
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Lloyd, T. C. "Breathing response to lung congestion with and without left heart distension." Journal of Applied Physiology 65, no. 1 (July 1, 1988): 131–36. http://dx.doi.org/10.1152/jappl.1988.65.1.131.
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This study compared the effect of lung congestion with and without left heart (LH) distension on breathing frequency (fr) and discriminated among responses mediated by myelinated and nonmyelinated vagal afferents. Cardiopulmonary bypass perfusion of anesthetized dogs was used to isolate reflexes. The following three groups were prepared: 1) lung vessels pressurized by pumping into the main pulmonary artery (MPA); 2) lungs and fibrillating LH pressurized by pumping into MPA while draining from LH; 3) lungs congested by occluding several pulmonary veins while holding cardiac output constant. Congestion of lungs alone in groups 1 and 3 depressed fr. Congestion of lungs and distension of LH (group 2) caused transient depression of fr but a steady-state excitation. Cooling cervical vagi to 8 degrees C prevented depression of fr by congestion in all groups. In groups 1 and 2, in which MPA pressure was higher than in group 3, congestion during vagal cooling stimulated breathing. I conclude that lung congestion may stimulate fr via C-fiber afferents, but this may be overcome by a depressor effect via myelinated afferents. Simultaneous LH distension may reflexly stimulate breathing and overcome the lung depressor reflex.
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Skorodumova, E. G., V. A. Kostenko, E. A. Skorodumova, and A. V. Siverinа. "Assessment of interstitial edema in patients with intermediate function of the left ventricle after resolving of acute decompensation of heart failure." Translational Medicine 5, no. 3 (October 1, 2018): 23–27. http://dx.doi.org/10.18705/2311-4495-2018-5-3-23-27.
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Background.According to the recommendations of the European Society of Cardiology, ultrasound examination of the lung can be used to assess congestive changes, however, this method has so far a low recommendation class IIb and a level of evidence C, which can be regarded as a consequence of its insufficient knowledge.The aim of the research.To study ultrasound characteristics of the lung tissue in patients with intermediate left ventricular function after resolving of acute decompensation of heart failure.Materials and methods. The ultrasound profile of the lung tissue was studied in 71 patients after resolution of acute heart failure decompensation under intermediate left ventricular function. The average age of patients studied was 65.2 ± 3.6 years. 64.3% of them were males ,25.7%-females. Using the ultrasonic method, B-lines were studied, the distance between them being 3 mm and 7 mm in a semi-quantitative manner, according to the method of E. Picano in 2016. The results were statistically processedResults.In persons after the acute decompensation of the heart failure residual congestion persisted with interstitial component dominated in both lungs. Appearance of interstitial lung edema to be considered as prognostic factor of re-hospitalizations rate increasing.Conclusion.Interstitial congestion in the pulmonary parenchyma considered as a factor in the deterioration of the clinical course of heart failure sign of its acute decompensation. In this connection, it is interesting to identify interstitial pulmonary edema at the early stage to prevent real clinical presentation of acute decompensation of the heart failure.
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Minnear, F. L., C. Kite, L. A. Hill, and H. van der Zee. "Endothelial injury and pulmonary congestion characterize neurogenic pulmonary edema in rabbits." Journal of Applied Physiology 63, no. 1 (July 1, 1987): 335–41. http://dx.doi.org/10.1152/jappl.1987.63.1.335.
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The objectives of the present study were to determine whether an intracisternal injection of fibrinogen-sodium citrate, a model of neurogenic pulmonary edema (NPE), produces protein-rich or protein-poor pulmonary edema, and to determine whether the edema is associated with pulmonary vascular hypertension and pulmonary congestion. Fibrinogen (6–10 mg/ml) dissolved in 0.055 M sodium citrate was injected into the cisterna magna of six New Zealand White rabbits. Six additional rabbits were injected with saline to control for the effects of intracranial hypertension and pulmonary vascular hypertension. The fibrinogen-sodium citrate solution or sodium citrate alone, as opposed to saline, produced systemic and pulmonary vascular hypertension, pulmonary edema, hypoxemia, hypercapnia, and acidosis. The lungs from fibrinogen-injected rabbits were edematous, congested, and liverlike in appearance. Tracheal froth that was blood tinged and protein rich was present in five of the six rabbits. Microscopic examination of lung biopsies revealed erythrocytes and plasma in the alveoli and focal injury to the pulmonary microvascular endothelium. Fibrinogen-sodium citrate increased (P less than 0.05) the extravascular lung water (EVLW) (10.3 +/- 2.0 vs. 5.5 +/- 0.6 g, means +/- SE), lung blood weight (9.7 +/- 1.3 vs. 3.8 +/- 0.6 g), total dry lung weight (3.2 +/- 0.4 vs. 2.0 +/- 0.1 g), and the EVLW-to-blood-free dry lung weight ratio (7.0 +/- 0.8 vs. 4.0 +/- 0.3 g) from saline-control values. There was no difference in the blood-fre dry lung weight (1.4 +/- 0.1 vs. 1.3 +/- 0.1 g) between the two groups. These findings demonstrate that pulmonary congestion, pulmonary vascular hypertension, and focal endothelial injury contribute to the development of NPE.
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P., Praveen M., Lokesh Shanmugam, and P. Arun Prasath. "A review of role of lung ultrasound and clinical congestion score in acute left ventricular failure." International Journal of Advances in Medicine 7, no. 4 (March 21, 2020): 720. http://dx.doi.org/10.18203/2349-3933.ijam20201130.
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Congestive cardiac failure (CCF) is a worldwide phenomenon and affects millions of people years and is accompanied with high mortality. The present review is undertaken to evaluate the usefulness of Lung Ultrasound Scan in diagnosis and to identify its role as a marker of clinical outcome in patients with Acute LVF. A review of literature was done to find the role of lung ultrasound and clinical congestion score in acute left ventricular failure from search engines such as PubMed, google scholar. Major exclusion criteria were the studies that included patients with Right Ventricular Failure, renal insufficiency, other respiratory causes of breathlessness like pneumonia, pulmonary embolism, pneumothorax and pleural effusion. This review concluded that lung ultrasonography is as a rapid, non-invasive, bedside tool for the diagnosis and risk assessment of pulmonary congestion in Acute LVF.
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Arystan, A. Z., Y. T. Khamzina, V. V. Benberin, D. V. Fettser, and Y. N. Belenkov. "Lung Ultrasound: new Opportunities for a Cardiologist." Kardiologiia 60, no. 1 (February 6, 2020): 81–92. http://dx.doi.org/10.18087/cardio.2020.1.n617.
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This review focused on ultrasound examination of lungs, a useful complement to transthoracic echocardiography (EchoCG), which is superior to chest X-ray in the diagnostic value. The lung acoustic window always remains open and allows obtaining high-quality images in most cases. For a cardiologist, the major points of the method application are determination of pleural effusion and lung congestion. This method has a number of advantages: it is time-saving; cost-effective; portable and accessible; can be used in a real-time mode; not associated with radiation; reproducible; and highly informative. The ultrasound finding of wet lungs would indicate threatening, acute cardiac decompensation long before appearance of clinical, auscultative, and radiological signs of lung congestion. Modern EchoCG should include examination of the heart and lungs as a part of a single, integrative ultrasound examination.
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Miah, MS, M. Asaduzzaman, A. Siddika, N. Popy, MA Sufian, and MM Hossain. "Detection of Toxic Effects of Clostridial Crude Toxin in Experimental Rats." Progressive Agriculture 21, no. 1-2 (November 1, 2013): 65–72. http://dx.doi.org/10.3329/pa.v21i1-2.16753.
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The present study was conducted for the detection of toxic effects of clostridial crude toxin in experimental rats. The crude toxin of Clostridium perfringens was prepared and the rats were injected intraperitoneally (IP) 0.5 ml, 1.0 ml and 2.0 ml of crude toxin. The rats were observed for 24 hrs. The crude toxin inoculated rats showed the dose dependent clinical signs; depression, rough hair coat, respiratory distress, diarrhea and rapid heart beats, whereas PBS inoculated rats did not show any clinical sings. Necropsy changes were variable however, highly dilated and distended whole intestine with blood stained semifluid contents and gas along with congestion in all the affected rats were found dose dependently. Liver, lung and kidney were congested, hemorrhagic and swollen. More or less hydrothorax was seen during the postmortem of all affected rats. The peritoneal fat was frequently congested in all affected rats. Histopathological changes in intestine (0.5 ml inoculated rats) involved congestion, slightly swollen goblet cells and hemorrhages. The most severe lesions comprised of profuse hemorrhages in the mucosa and submucosa with complete necrosis, desquamation and intense leukocytic infiltration in 2 ml inoculated rats. Affected liver (1 ml inoculated rats) exhibited engorgement of central veins, sinusoidal spaces with blood and fatty change. The hepatocytes revealed swelling, granulation and vacuolation of cell cytoplasm, extensive hemorrhage and congestion were seen in 2 ml toxin inoculated rats. Congestion and sometimes extravasation were observed in the subepicardial region of heart. The myocardium revealed mild degenerative changes in the form of granularity of myocardial fiber. In lungs there was congestion, hemorrhage and leukocytes infiltration in the interstitial spaces around the bronchioles in both 1 and 2 ml toxin inoculated rats. Affected kidneys of different doses of crude toxin showed hemorrhage, congestion and inflammatory cells dose dependently. From the above findings, it may be concluded that clostridial crude toxin induced clinical signs, gross and histopathological lesions dose dependently in experimental rats.DOI: http://dx.doi.org/10.3329/pa.v21i1-2.16753 Progress. Agric. 21(1 & 2): 65 - 72, 2010
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Tonu, NS, MA Sufian, S. Sarker, MM Kamal, MH Rahman, and MM Hossain. "PATHOLOGICALSTUDY ON COLIBACILLOSIS IN CHICKENS AND DETECTION OF ESCHERICHIA COLI BY PCR." Bangladesh Journal of Veterinary Medicine 9, no. 1 (July 12, 2012): 17–25. http://dx.doi.org/10.3329/bjvm.v9i1.11205.
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The aim of the present study was to detect the pathogenic Escherichia coli (E. coli) through pathological study of the colibacillosis affected birds. These isolated E. coli were further confirmed by PCR using specific primer. For this purpose, a total of 20 swabs (10 from lung and 10 from intestine of 10 dead birds) were collected in sterile nutrient broth. The histopathological samples were collected in 10% buffered neutral formalin. The used methods were histopathology, isolation and identification of E. coli by conventional methods and as well as by PCR method. A total of 10 isolates of E. coli from 20 swabs of lung and intestine was characterized by conventional routine methods of bacteriology. Gross pathological lesions of all lungs in the present investigation were congested and consolidated. Duodenum showed congestion and hemorrhages with excess mucus in the luminal surface of it. Microscopically, all the lungs showed severe congestion, infiltration of heterophils, macrophages and lymphocytes in the wall of bronchus as well as in the peribronchial alveoli. E. coli infected all the duodenum showed severe infiltration of leukocytes mainly heterophils, lymphocytes and macrophages in the submucosa of the duodenal wall. In this study, DNA of 8 isolates out of 10 isolated E. coli organisms was amplified by PCR using ECO-f and ECO-r primer targeting 16S ribosomal DNA and found 585 bp amplicon which is specific for E. coli with enteroinvasive type confirmed by histopathological lesions in duodenum. Further investigation should be focused on serotyping and detection of genes of E. coli which are responsible for pathogenicity of the organism.DOI = http://dx.doi.org/10.3329/bjvm.v9i1.11205Bangl. J. Vet. Med. (2011). 9(1): 17-25
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Li, Hong, Yi-Dan Li, Wei-Wei Zhu, Ling-Yun Kong, Xiao-Guang Ye, Qi-Zhe Cai, Lan-Lan Sun, and Xiu-Zhang Lu. "A Simplified Ultrasound Comet Tail Grading Scoring to Assess Pulmonary Congestion in Patients with Heart Failure." BioMed Research International 2018 (2018): 1–10. http://dx.doi.org/10.1155/2018/8474839.
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Ultrasound lung comets (ULCs) are a nonionizing bedside approach to assess extravascular lung water. We evaluated a protocol for grading ULC score to estimate pulmonary congestion in heart failure patients and investigated clinical and echocardiographic correlates of the ULC score. Ninety-three patients with congestive heart failure, admitted to the emergency department, underwent pulmonary ultrasound and echocardiography. A ULC score was obtained by summing the ULC scores of 7 zones of anterolateral chest scans. The results of ULC score were compared with echocardiographic results, the New York Heart Association (NYHA) functional classification, radiologic score, and N-terminal pro-b-type natriuretic peptide (NT-proBNP). Positive linear correlations were found between the 7-zone ULC score and the following: E/e′, systolic pulmonary artery pressure, severity of mitral regurgitation, left ventricular global longitudinal strain, NYHA functional classification, radiologic score, and NT-proBNP. However, there was no significant correlation between ULC score and left ventricular ejection fraction, left ventricle diameter, left ventricular volume, or left atrial volume. A multivariate analysis identified the E/e′, systolic pulmonary artery pressure, and radiologic score as the only independent variables associated with ULC score increase. The simplified 7-zone ULC score is a rapid and noninvasive method to assess lung congestion. Diastolic rather than systolic performance may be the most important determinant of the degree of lung congestion in patients with heart failure.
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Rashid, MM, MJ Ferdoush, M. Dipti, P. Roy, MM Rahman, MI Hossain, and MM Hossain. "Bacteriological and pathological investigation of goat lungs in Mymensingh and determination of antibiotic sensitivity." Bangladesh Journal of Veterinary Medicine 11, no. 2 (June 13, 2014): 159–66. http://dx.doi.org/10.3329/bjvm.v11i2.19142.
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The foremost important goal of the present study was to investigate the bacteriological and pathological conditions in lungs of goats slaughtered in four different slaughter houses/places of Mymensingh Sadar, Mymensingh and in addition to it, antibiotic sensitivity test of commonly used antibiotics in Veterinary practices were performed on isolated bacteria. A total of 75 lungs of slaughtered goats were examined individually and out of which 20 affected lungs were collected for histopathology and bacterial isolation respectively from Mymensingh Sadar, Mymensingh in Bangladesh, during the period from January to May 2013.The lung lesions was grossly recorded 40% in goats (30 out of 75 lungs examined). Grossly, the lung lesions were categorized into (a) hemorrhages 35% (b) congestion 25% (c) hemorrhage and congestion 15% (d) emphysematous lung 15% and (e) hepatization in lung10%. In histopathology, lung lesions were categorized into (a) bronchopneumonia 30%, (b) pneumonia 25% (c) hemorrhagic pneumonia 20% (d) emphysema 15%, (e) purulent pneumonia 10%. Pasteurella sp. (15%) was isolated from the lung lesions of hemorrhagic pneumonia, E. coli. (25%) from bronchopneumonia and Staphylococcus sp. (40%) from purulent pneumonia, focal pneumonia and emphysema, and mixed infection (Staphylococcus sp. and E. coli) 20%. Finally antibiotics sensitivity test was performed on isolated bacteria to which ciprofloxacin was more powerful than others (penicillin, amoxicillin, streptomycin, nalidixic acid and kanamycin) tested and the second one was oxytetracyclin.DOI: http://dx.doi.org/10.3329/bjvm.v11i2.19142Bangl. J. Vet. Med. (2013).11(2): 159-166
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Samkange, Alaster, Borden Mushonga, Douglas Mudimba, Bernard A. Chiwome, Mark Jago, Erick Kandiwa, Alec S. Bishi, and Umberto Molini. "African Swine Fever Outbreak at a Farm in Central Namibia." Case Reports in Veterinary Medicine 2019 (October 29, 2019): 1–6. http://dx.doi.org/10.1155/2019/3619593.
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An outbreak of African swine fever (ASF) occurred at a farm in central Namibia in March 2018. Fourteen pigs died out of a herd of 59 animals over a period of 16 days between the first and sixteenth of March 2018. The clinical signs observed included sternal recumbency, fever, weakness, pain and reluctance to move, hyperemia of the skin and anorexia, followed by death. Necropsy findings included large amounts of unclotted blood in the pleural and peritoneal cavities, diffuse carcass congestion, splenomegaly, consolidation of both lungs, hemorrhagic and frothy airways and trachea, hepatomegaly and congestion, congestion of the gastric mucosa, enlarged and congested kidneys, ecchymotic epicardial, and endocardial hemorrhages, and very enlarged and congested urinary bladder. All the remaining pigs were euthanized, burned, and buried under state veterinary supervision. The authors concluded that the outbreak resulted from indirect transmission of the ASF virus due to lapses in biosecurity measures.
Dissertations / Theses on the topic "Lung congesti"
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Siqueira, Rafaela. "A influência do estrogênio na hipertensão arterial pulmonar : papel do estresse oxidativo." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2011. http://hdl.handle.net/10183/37037.
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A hipertensão arterial pulmonar é uma síndrome caracterizada por vasoconstrição e remodelamento vascular pulmonar, levando a um aumento progressivo na resistência vascular pulmonar, que eleva a pós-carga imposta ao ventrículo direito, gerando consequente hipertrofia e insuficiência cardíaca direita. Essa doença acomete duas vezes mais mulheres do que homens. O estresse oxidativo está envolvido na patogênese da hipertensão arterial pulmonar. O hormônio estrogênio, comportando-se como scavenger de radicais livres, é capaz de modular o estresse oxidativo. O modelo experimental de hipertensão arterial pulmonar induzido por monocrotalina vem sendo utilizado por mimetizar as alterações que decorrem desta patologia em humanos. Dessa maneira, o objetivo desse estudo foi testar a hipótese de que o estrogênio poderia atenuar a hipertrofia do ventrículo direito e sua progressão para insuficiência cardíaca, modulando o estresse oxidativo, nos animais que receberam a monocrotalina. Ratas wistar fêmeas com 60 dias foram ovariectomizadas ou sofreram simulação da mesma. Após sete dias, receberam implantação de pellets subcutâneos com 17β-estradiol ou óleo de girassol. Neste momento, foi também administrada injeção intraperitoneal de monocrotalina ou salina. Os grupos experimentais foram: sham (S) - simulação da cirurgia de ovariectomia, não submetidas ao tratamento com MCT; sham + MCT (SM) - simulação da cirurgia de ovariectomia, e tratadas com MCT; ovariectomia (O) - cirurgia de ovariectomia, não submetidas ao tratamento com MCT; ovariectomia + MCT (OM) - cirurgia de ovariectomia, e tratadas com MCT; ovariectomia + MCT + reposição estrogênio (OMR) - cirurgia de ovariectomia, tratadas com MCT e estrogênio. As medidas hemodinâmicas foram realizadas 21 dias após a administração da monocrotalina ou salina nos animais ovariectomizados e, nos outros grupos, na fase do diestro. Foi verificada a pressão diastólica final do ventrículo direito, pressão sistólica do ventrículo direito e frequência cardíaca. Após a análise, as ratas foram mortas por deslocamento cervical e o coração, pulmão, fígado e útero foram coletados. As análises morfométricas foram realizadas após a retirada dos órgãos para avaliar hipertrofia cardíaca, congestão pulmonar e hepática. Amostras de ventrículo direito foram utilizadas para analisar a concentração de peróxido de hidrogênio, a razão da glutationa reduzida/oxidada, lipoperoxidação e defesas antioxidantes enzimáticas. O imunoconteúdo de ANP (peptídeo natriurético atrial) foi também avaliado em homogeneizado cardíaco. Os dados de pressão sistólica, hipertrofia cardíaca, defesa antioxidante enzimática, concentração de peróxido de hidrogênio e lipoperoxidação não mostraram diferença entre os grupos. Houve congestão pulmonar no grupo OM, sendo esta diminuída no grupo OMR. Isto sugere que o estrogênio esteja atenuando a resistência vascular pulmonar. Também houve aumento da pressão diastólica final do ventrículo direito nos grupos OM e OMR. A razão das glutationas se mostrou diminuída nos grupos O, OM e OMR, assim como a glutationa reduzida nos grupos O e OM, sugerindo a influência do estrogênio na modulação do estado redox celular. Os dados sugerem que o estrogênio possa exercer grande influência no equilíbrio redox celular, podendo este efeito contribuir para evitar o surgimento do edema pulmonar, característico deste modelo de hipertensão arterial pulmonar e insuficiência do ventrículo direito.Pulmonary arterial hypertension is a syndrome characterized by vasoconstriction and pulmonary vascular remodeling, leading to a progressive increase in pulmonary vascular resistance, which increases the afterload imposed on the right ventricle, causing consequent hypertrophy and heart failure. This disease affects twice as many women as men. The oxidative stress is involved in pulmonary artery hypertension, as well as the estrogen hormone modulating the oxidative stress, behaving like scavenger of free radicals. The experimental model of pulmonary hypertension induced by monocrotaline has been used to mimic the changes from this pathology. Thus, the objective of this study was to test the hypothesis that estrogen could attenuate ventricular hypertrophy law and its progression to heart failure, modulating oxidative stress in animals received monocrotaline. Female Wistar rats aged 60 days were ovariectomized or underwent sham same, 7 days after implantation of pellets were subcutaneous 17β-estradiol or sunflower oil more intraperitoneal injection of monocrotaline or saline. The experimental groups (n = 9-13 per group) were: SHAM (S) – sham surgery, ovariectomy, no treated with MCT, MCT + SHAM - simulation of ovariectomy surgical and treated with MCT; OVARIECTOMY (O) – ovariectomy surgical, no treated with MCT, MCT + OVARIECTOMY (OM) –ovariectomy surgical, and treated with MCT; MCT + + OVARIECTOMY ESTROGEN REPLACEMENT (OMR) - surgery, ovariectomy, and treated with MCT estrogen. Hemodynamic measurements were performed 21 days after administration of saline or monocrotaline in ovariectomized animals and other groups in diestrus phase with anesthetized animals. The ventricular end diastolic pressure right ventricular systolic pressure and right heart rate was verified. After analyzing, the rats were killed by cervical dislocation and the heart, lung, liver and uterus were collected. Analyses morphometry were performed after withdrawal of agencies to evaluate cardiac hypertrophy, congestion lung and liver. The right ventricular mass was used to analyze the redox status (hydrogen peroxide and glutathione ratio) antioxidant enzymatic defenses and protein expression of ANP. The Data for systolic blood pressure, cardiac hypertrophy, antioxidant defense enzyme concentration hydrogen peroxide and lipid peroxidation showed no difference between the groups, which may be related with normal distribution. There was congestion pulmonary CO group and decreased in group OMR, suggesting that estrogen is attenuating the pulmonary vascular resistance, as well as increased end-diastolic pressure in the right ventricle OMR and OM groups. The glutathione ratio was shown decreased in groups O, CO and OMR, as well as reduced glutathione in groups O and CO, suggesting the influence of estrogen on modulation of cellular redox state. The data suggest that estrogen can exert great influence on the cellular redox balance and this effect may help to avoid the appearance of pulmonary edema, a characteristic of pulmonary hypertension and right ventricular failure.
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Araujo, Caroline de Souza Costa. "Ultrassonografia pulmonar na identificação da dispneia aguda cardiogênica em pacientes hospitalizados." Universidade Federal de Sergipe, 2016. https://ri.ufs.br/handle/riufs/3778.
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Acute dyspnea (AD) is a common complaint in a hospital unit and frequently, differential diagnosis is a challenge despite acquirement of diverse methodologies. Lung ultrasonography (LU) has shown useful identifying AD of cardiogenic origin. However, there is a shortage of national studies confirming such quality. Objectives: Evaluate LU’s contribution on identification of cardiogenic origin AD. Methodology: This is a prospective, longitudinal cohort of patients in emergency room and admitted into hospital unit, with AD complaint. They were evaluated by Framingham risk score (FRS) for cardiac insufficiency (CI) and subjected to the following additional tests: electrocardiogram (ECG), chest radiography (CR), transthoracic ecodoppler cardiogram (TTE) and LU. Excluded pulmonary fibrosis and lung neoplasm patients. Calculated: sensitivity, specificity, positive and negative predictive value, as well as ROC curve of LU for AD diagnosis. Interobserver agreement among methods was estimated by kappa statistic. Results: From February to October of 2015, 118 patients (9 excluded) were classified according to presence or absence of CI by FRS. Of 109 cases, 60 (55%) had CI. These were elder (average age 76±16.7, p=0.01), men (60%, p=0.004), hypertensive (83%, p=0.01), diabetic (50.8%, p=0.03), with chronic renal insufficiency (CRI) (32.2%, p=0.03) and with higher adapted Killip classification (p=0.00) and functional class (p=0.003). Presented more pulmonary interstitial edema(IPE) on LU, CI to CR criteria (p=0.009) and, on TTE, higher E/e’ ratio (p=0.002) and left ventricle diastolic dysfunction (p=0.04), besides lower ejection fraction (p=0.00). Importants predictors on IPE detection by US in these patients were male gender, diabetics and with ejection fraction of reduced(p=0.01; 0.02 and 0.03, respectively). There was modest kappa agreement (k) between LU with FRS (k=0.25) and CR (k=0.22) for CI and moderate between LU (k=0,48) and CR and interobserver CR (kappa=0,44) LU sensibility for IEP in patients with CI was 90.91%, 65% specificity, 85.1% positive predictive value and 76.4% negative. Conclusion: therefore, concludes that LU proved to be a useful tool and with reproducibility when identifying AD of cardiogenic.A dispneia aguda (DA) é uma queixa frequente em unidade hospitalar e o diagnóstico diferencial torna-se desafiador a despeito da aquisição de diferentes metodologias aplicadas. A ultrassonografia pulmonar (UP) tem-se demonstrado útil na identificação de DA de origem cardiogênica. Todavia, há poucos estudos nacionais ratificando esse benefício. Objetivos: Avaliar a contribuição da UP na identificação de DA de origem cardiogênica. Metodologia: Trata-se de uma coorte, prospectiva, longitudinal, de pacientes em pronto-socorro e internos em unidade hospitalar, com queixa principal de DA. Foram avaliados pelo escore de Framingham (EF) para insuficiência cardíaca(IC) e submetidos aos seguintes exames complementares: eletrocardiograma(ECG), radiografia de tórax (RT), ecodopplercardiograma transtorácico(ETT) e UP. Excluídos os portadores de fibrose ou neoplasia pulmonar. Foram calculados os parâmetros de sensibilidade, especificidade, valor preditivo positivo e negativo, assim como a curva ROC da UP para o diagnóstico de DA. Concordância inter-observador entre métodos foi estimada pelo método kappa. Resultados: De fevereiro a outubro/2015, 118 pacientes (9 excluídos) foram classificados quanto a presença ou não de IC pelo EF. Dos 109 casos, 60(55%) tinham IC. Esses eram mais idosos (idade média 76±16.7, p=0.01), homens (60%, p=0.004), hipertensos (83%, p=0.01), diabéticos (50.8%, p=0.03), com insuficiência renal crônica (IRC)(32.2%, p=0.03), maior graduação de Killip adaptada(p=0.00) e classe funcional(p=0.003). Apresentaram mais edema intersticial pulmonar(EIP) à UP (p=0.00), critérios de IC à RT (p=0.009) e, ao ETT, maiores relação E/e’(p=0.002) e disfunção diastólica do ventrículo esquerdo(p=0.04), além de menor fração de ejeção(p=0.00). Os preditores independentes na detecção de EIP pela US nesses pacientes foram o sexo masculino, diabéticos e com fração de ejeção reduzida(p=0.01; 0.02 e 0.03, respectivamente). Houve razoável concordância kappa (k) entre UP com EF(k=0.25) e RT(k=0.22) para IC e moderada da UP(k: 0.48) e RT(k:0.44) inter-observador. A sensibilidade da UP para EIP em pacientes com IC foi 90.91%, especificidade 65%, valor preditivo positivo 85.1% e preditivo negativo de 76.4%. Conclusão: Conclui-se que a UP demonstrou ser ferramenta útil e com reprodutibilidade na identificação de DA de origem cardiogênica.
Books on the topic "Lung congesti"
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Subcommittee, United States Congress House Committee on Government Operations Employment and Housing. Delays in processing and adjudicating black lung claims: Hearing before a subcommittee of the Committee on Government Operations, House of Representatives, Ninety-ninth Congress, first session, June 24, 1985. Washington: U.S. G.P.O., 1986.
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Standards, United States Congress House Committee on Education and Labor Subcommittee on Labor. Investigation of the backlog in black lung cases: Hearings before the Subcommittee on Labor Standards of the Committee on Education and Labor, House of Representatives, Ninety-ninth Congress, first session, hearings held in Uniontown, PA, May 3; Wise, VA, June 7; Washington, DC, July 11; and Wilkes-Barre, PA, September 9, 1985. Washington: U.S. G.P.O., 1986.
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Gargani, Luna, and Marcelo-Haertel Miglioranza. Lung ultrasound. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0016.
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The lung is a relatively new site for the application of ultrasound. Beyond the more established assessment of pleural effusion, this organ has been traditionally considered off limits for sonographic investigation, since air is a well-known foe of the ultrasound beam. However, in recent years it has been shown that this apparent physical limitation can be overcome when the air content decreases, as happens in a diseased pulmonary parenchyma. The most useful lung ultrasound sign for cardiologists is the presence of B-lines, the sonographic hallmark of pulmonary interstitial syndrome, including interstitial pulmonary oedema. Bilateral multiple B-lines are present in patients with pulmonary congestion and may help assess and semiquantify the extent of extravascular lung water in patients with heart failure. This sign is low cost, easy to perform, can be repeated at bedside, and does not employ ionizing radiation. Lung ultrasound is also useful for detecting other pulmonary conditions such as pneumothorax, and lung consolidations such as pneumonia or pulmonary infarction.
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Congestion of the lungs and its dangers. Montreal: Montreal Homoeopathic Pharmacy, 1993.
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Swanson, Karen L. Neoplastic and Vascular Diseases. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0618.
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Neoplastic and vascular disorders are reviewed. Lung cancer is the most common malignancy and cause of cancer death in both men and women worldwide. The incidence of new lung cancers has continued to decrease in men and increase in women. The risk factors include cigarette smoking, other carcinogens, cocarcinogens, radon exposure, arsenic, asbestos, coal dust, chromium, vinyl chloride, chloromethyl ether, and chronic lung injury. Genetic and nutritional factors have been implicated. Among vascular disorders, pulmonary embolism is most common. Pulmonary embolism (PE) is the cause of death in 5% to 15% of hospitalized patients who die in the United States. In a multicenter study of PE, the mortality rate at 3 months was 15% and important prognostic factors included age older than 70 years, cancer, congestive heart failure, COPD, systolic arterial hypotension, tachypnea, and right ventricular hypokinesis.
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Sleep Apnea: Implications in Cardiovascular and Cerebrovascular Disease (Lung Biology in Health and Disease). Informa Healthcare, 2000.
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Awan, Kanwal, and Martin Steinberg. Medical Conditions That May Cause Cognitive Impairment and Depression. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199959549.003.0005.
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Ruling out medical conditions that can cause depression or cognitive impairment is essential in effectively caring for elderly patients. Case examples illustrate how these may present. Diabetes can cause confusion due to either hyper- or hypoglycemia. Congestive heart failure and chronic obstructive lung disease can cause hypoxia and resulting confusion. Sleep apnea can present with amnesia, apathy, and depression. Physiological changes make elderly patients especially susceptible to adverse drug effects, including hyponatremia and anticholinergic symptoms. Depression and cognitive changes have been associated with both hyper- and hypothyroidism, as well as with hyperparathyroidism. Elderly patients are at risk for developing subdural hematomas which can present with cognitive deficits and blunting of mood, and some patients may not have taken notice of the traumatic incident. Vitamin B12 deficiency can present with neurological symptoms including dementia, and cancer may present with fatigue and weight loss, which may be interpreted as depression.
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Galiè, Nazzareno, Alessandra Manes, and Massimiliano Palazzini. Pulmonary hypertension. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0065.
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Pulmonary hypertension is a haemodynamic and pathophysiological condition defined as an increase in the mean pulmonary arterial pressure of ≥25 mmHg at rest, as assessed by right heart catheterization. In fact, while transthoracic echocardiography may provide clues on the presence of pulmonary hypertension, the haemodynamic evaluation offers a more precise and comprehensive assessment. Pulmonary hypertension is heterogeneous from a pathophysiological point of view, and the diversity is reflected in the haemodynamic definitions. The different haemodynamic forms of pulmonary hypertension can be found in multiple clinical conditions which have been classified into six main groups and at least twenty-six subgroups. Each main clinical group shows specific pathological changes in the lung distal arteries, capillaries, and small veins. If we combine the haemodynamic and clinical heterogeneity, we understand the complexity of an accurate diagnosis in the individual patient which is crucial for the prognostic assessment and treatment strategy. In addition, the concomitant presence of different haemodynamic and clinical mechanisms cannot be excluded in individual cases (e.g. in patients with congestive heart failure and associated lung diseases). The presence of pulmonary hypertension, as defined above, is always an ominous prognostic sign, even if the severity may differ according to the haemodynamic changes and underlying clinical condition. The therapeutic approach also is markedly different, according to the clinical group, and symptomatic and haemodynamic severity. For these reasons, the four more frequent clinical groups are discussed individually, while the classifications are described in the Introduction section.
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Chiumello, Davide, and Silvia Coppola. Management of pleural effusion and haemothorax. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0125.
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The main goal of management of pleural effusion is to provide symptomatic relief removing fluid from the pleural space. The options depend on type, stage, and underlying disease. The first diagnostic instrument is the chest radiography, while ultrasound can be very useful to guide thoracentesis. Pleural effusion can be a transudate or an exudate. Generally, a transudate is uncomplicated effusion treated by medical therapy, while an exudative effusion is considered complicated effusion and should be managed by drainage. Refractory non-malignant effusions can be transudative (congestive heart failure, cirrhosis, nephrosis) or exudative (pancreatitis, connective tissue disease, endocrine dysfunction), and the management options include repeated therapeutic thoracentesis, in-dwelling pleural catheter for intermittent external drainage, pleuroperitoneal shunts for internal drainage, or surgical pleurectomy. Parapneumonic pleural effusions can be classified as complicated when there is persistent bacterial invasion of the pleural space, uncomplicated and empyema with specific indications for pleural fluid drainage. Malignancy is the most common cause of exudative pleural effusions in patients aged >60 years and the decision to treat depends upon the presence of symptoms and the underlying tumour type. Options include in-dwelling pleural catheter drainage, pleurodesis, pleurectomy, and pleuroperitoneal shunt. Haemothorax needs to be differentiated from a haemorrhagic pleural effusion and, when suspected, the essential management is intercostal drainage. It achieves two objectives to drain the pleural space allowing expansion of the lung and to allow assessment of rates of blood loss to evaluate the need for emergency or urgent thoracotomy.
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Blasi, Francesco, and Paolo Tarsia. Pathophysiology and causes of haemoptysis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0126.
Full textAbstract:
The main goal of management of pleural effusion is to provide symptomatic relief removing fluid from pleural space and the options depend on type, stage and underlying disease. The first diagnostic instrument is the chest radiography while ultrasound can be very useful to guide thoracentesis. Pleural effusion can be a transudate or an exudate. Generally a transudate is uncomplicated effusion treated by medical therapy, while an exudative effusion is considered complicated effusion and should be managed by drainage. Refractory non-malignant effusions can be transudative (congestive heart failure, cirrhosis, nephrosis) or exudative (pancreatitis, connective tissue disease, endocrine dysfunction), and the management options include repeated therapeutic thoracentesis, indwelling pleural catheter for intermittent external drainage, pleuroperitoneal shunts for internal drainage, or surgical pleurectomy. Parapneumonic pleural effusions can be divided in complicated when there is persistent bacterial invasion of the pleural space, uncomplicated and empyema with specific indications for pleural fluid drainage. Malignancy is the most common cause of exudative pleural effusions in patients aged >60 years and the decision to treat depends upon the presence of symptoms and the underlying tumour type. Options include indwelling pleural catheter drainage, pleurodesis, pleurectomy and pleuroperitoneal shunt. Hemothorax needs to be differentiated from a haemorrhagic pleural effusion and when is suspected the essential management is the intercostal drainage. It achieves two objectives to drain the pleural space allowing expansion of the lung and to allow assessment of rates of blood loss to evaluate the need for emergency or urgent thoracotomy.
Book chapters on the topic "Lung congesti"
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Martin, Cindy M., and James H. Moller. "Heart and Heart–Lung Transplantation in Adults with Congenital Heart Disease." In Congestive Heart Failure and Cardiac Transplantation, 539–47. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-44577-9_33.
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Gissing, George. "Chapter XXVII The Lonely Man." In New Grub Street. Oxford University Press, 2016. http://dx.doi.org/10.1093/owc/9780198729181.003.0028.
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A touch of congestion in the right lung was a warning to Reardon that his half-year of insufficient food and general waste of strength would make the coming winter a hard time for him, worse probably than the last. Biffen, responding in person...
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Tkachenko, Oleksiy, and Olena Havrylina. "PECULIARITIES OF PATHOMORPHOLOGICAL DIAGNOSIS OF THE MOST COMMON RESPIRATORY INFECTIONS OF A PIG." In Integration of traditional and innovative scientific researches: global trends and regional aspect. Publishing House “Baltija Publishing”, 2020. http://dx.doi.org/10.30525/978-9934-26-001-8-3-13.
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The active spread of respiratory infections in pig farms raises the issue of differential pathomorphological diagnosis of diseases related to a single syndrome of respiratory pathologies. Pathological autopsy and histopathological examinations of organs from 72 carcasses of pigs during the fattening period were performed. Pathological autopsy of pigs was performed by complete evisceration. Histologically examined 360 lung samples with regional lymph nodes. The presence of bacterial and viral infections was confirmed by bacteriological and PCR studies. The aim of the study is to establish the characteristic differential features at the macro- and micro-level in the lungs of domestic pigs for viral and bacterial pathogens. The main tasks of the work are to determine the morphofunctional features and dynamics of pathomorphological changes in the parenchyma and immune formations of the lungs in respiratory pathology. As a result of complex pathomorphological studies of the lungs in respiratory infections of pigs found that structural and functional changes in the body have different localization, stage and nature of the pathological process, which depend on the direct action of the etiological factor. Acute catarrhal bronchopneumonia is registered in respiratory mycoplasmosis (enzootic pneumonia), which in a prolonged course turns into chronic catarrhal or catarrhal-purulent pneumonia. Hemorrhagic necrotizing pneumonia is a manifestation of actinobacillary pleuropneumonia. Interstitial (diffuse proliferative) pneumonia develops with viral pathogens - circovirus infection and reproductive and respiratory syndrome of pigs (PRRS). Serous fibrinous and fibrinous pleurisy develop in hemophilic polyserositis and actinobacillary pleuropneumonia. Pathomorphological changes of the lungs in the reproductive and respiratory syndrome of pigs are polymorphic and are manifested by the gradual progressive development of the inflammatory process from congestive hyperemia, acute catarrh to diffuse interstitial pneumonia. Pathomorphological changes of the lungs in mycoplasmosis (enzootic pleuropneumonia) of pigs are polymorphic and are manifested by the gradual progressive development of the inflammatory process, which is localized in the cranial, middle and peripheral parts of the diaphragmatic particles and is characterized by acute catarrhal bronchopneumonia. In actinobacillary pleuropneumonia pathohistological studies revealed a pronounced stage of morphological changes in the lungs and regional lymph nodes in actinobacillary pleuropneumonia. Depending on the form of the disease, serous-hemorrhagic exudation is exacerbated by fibrinogen exudation and increased migration of lymphocytes and mononuclear cells. In subacute and chronic forms of the disease, necrotic phenomena prevail in combination with areas of serous-fibrinous inflammation. In the future, further studies of immunohistochemical analysis to establish the tropism of the pathogen and the study of markers of lymphoid cells.
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Roversi, Sara, and Nathaniel Hawkins. "Co-morbidity (HFrEF and HFpEF): lung disease." In ESC CardioMed, 1825–29. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0419.
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Approximately one in four patients with heart failure (HF) has a clinical diagnosis of pulmonary disease, predominantly chronic obstructive pulmonary disease (COPD). A further 40–70% may have unrecognized airflow obstruction. Clinical diagnosis is challenging given the cardinal symptom of both conditions is dyspnoea. Objective documentation of airflow obstruction by pulmonary function tests is essential, performed during clinical stability to avoid obstruction associated with congestion. In patients with pulmonary disease, natriuretic peptides retain high negative predictive values for excluding heart failure. The treatment of both HF and COPD should not deviate from international guidelines. The risk of bronchoconstriction due to beta blockers, and adverse cardiovascular events attributable to bronchodilators is controversial. The balance of evidence strongly favours prescribing beta blockers irrespective of pulmonary disease, recommending caution only in very severe COPD. Similarly, bronchodilators exhibit a reassuring safety profile, particularly long-acting muscarinic antagonists. Caution is advised when commencing new bronchodilator therapy, using high-dose, short-acting compounds, or when treating patients with severe heart failure. Concurrent pulmonary disease in patients with HF further impairs symptoms, quality of life, and exercise capacity. It also increases the risk of HF hospitalization, non-cardiovascular adverse outcomes, and long-term mortality. All efforts should be made to correctly identify and treat lung disease in clinical practice.
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Voors, Adriaan A., and Piotr Ponikowski. "Acute heart failure: diagnosis." In ESC CardioMed, 1911–17. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0439.
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Acute heart failure is a life-threatening medical condition typically leading to urgent hospital admission. Early diagnosis is of great importance, since it will lead to earlier and better targeted treatment, leading to a decrease in length of hospital stay, and most importantly to improved clinical outcome. The initial diagnostic work-up includes a clinical history, evaluation of symptoms and signs, an electrocardiogram, chest X-ray, natriuretic peptide levels, echocardiography and perhaps lung ultrasound. After the initial work-up, a clinical classification according to blood pressure, congestion, and peripheral perfusion should be performed, since it will guide treatment. During the diagnostic work-up, treatable and life-threatening conditions always need to be considered since they need immediate and case-specific treatment.
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Farmakis, Dimitrios, and Gerasimos Filippatos. "Acute heart failure: epidemiology, classification, and pathophysiology." In The ESC Textbook of Intensive and Acute Cardiovascular Care, edited by Marco Tubaro, Pascal Vranckx, Eric Bonnefoy-Cudraz, Susanna Price, and Christiaan Vrints, 603–16. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198849346.003.0046.
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Acute heart failure (AHF) is defined as the rapid development or change of symptoms and signs of heart failure that requires urgent medical attention and usually hospitalization. it represents the first reason for hospital admission in individuals aged 65 or more and accounts for nearly 70% of the total healthcare expenditure for heart failure. It is generally characterized by adverse prognosis, with an in-hospital mortality rate of 4-7%, a 2 to 3-month post-discharge mortality of 7-11% and a 2 to 3-month readmission rate of 25-30%. The majority of patients have a previous history of heart failure and present with symptoms and/or signs of congestion and normal or increased blood pressure, while about half of them have preserved left ventricular ejection fraction. A high prevalence of cardiovascular or non-cardiovascular comorbidities is further observed, including coronary artery disease, arterial hypertension, atrial fibrillation, diabetes mellitus, renal dysfunction, chronic lung disease, anemia and iron deficiency. Different classification criteria have been proposed for AHF, reflecting the clinical heterogeneity of the syndrome. Classifications according to the past history of heart failure (acutely decompensated chronic or de novo), the systolic blood pressure upon presentation (hypertensive, normotensive or hypotensive) and the presence or absence of congestion and peripheral hypoperfusion are among the most widely used. The pathophysiology of AHF involves several mechanisms, including volume overload, pressure overload, myocardial loss and restrictive filling, while several cardiovascular and non-cardiovascular precipitating factors lead to AHF. Regardless of the underlying mechanism, peripheral and/or pulmonary congestion is present in the vast majority of AHF, resulting from fluid retention and/or fluid redistribution, while a marked reduction in cardiac output with peripheral hypoperfusion occurs in a minority of cases. Myocardial injury and renal dysfunction are important factor involved in the precipitation and progression of the syndrome.
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Palazzini, Massimiliano, Nazzareno Galiè, and Alessandra Manes. "Pulmonary hypertension." In The ESC Textbook of Intensive and Acute Cardiovascular Care, edited by Marco Tubaro, Pascal Vranckx, Eric Bonnefoy-Cudraz, Susanna Price, and Christiaan Vrints, 839–48. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198849346.003.0063.
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Pulmonary hypertension is a haemodynamic and pathophysiological condition defined as an increase in the mean pulmonary arterial pressure of ?25 mmHg at rest, as assessed by right heart catheterization. Recently, a new definition has been proposed as mean pulmonary arterial pressure >20 mmHg combined with pulmonary vascular resistance ? 3 Wood units. While transthoracic echocardiography may provide clues on the presence of pulmonary hypertension, the haemodynamic evaluation offers a more precise and comprehensive assessment. Pulmonary hypertension is heterogeneous from a pathophysiological point of view, and the diversity is reflected in the haemodynamic definitions. The different haemodynamic and clinical forms of pulmonary hypertension can be found in multiple clinical conditions which have been classified into five main groups and at least twenty-six subgroups. Each main clinical group shows specific pathological changes in the lung distal arteries, capillaries, and small veins. If we combine the haemodynamic and clinical heterogeneity, we understand the complexity of an accurate diagnosis in the individual patient which is crucial for the prognostic assessment and treatment strategy. In addition, the concomitant presence of different haemodynamic and clinical mechanisms cannot be excluded in individual cases (e.g. in patients with congestive heart failure and associated lung diseases). The presence of pulmonary hypertension, as defined above, is always an ominous prognostic sign, even if the severity may differ according to the haemodynamic changes and underlying clinical condition. The therapeutic approach also is markedly different, according to the clinical group, and symptomatic and haemodynamic severity. For these reasons, the four more frequent clinical groups are discussed individually, while the classifications are described in the Introduction section.
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O’Halpin, Eunan, and Daithí Ó Corráin. "1917." In The Dead of the Irish Revolution, 102–3. Yale University Press, 2020. http://dx.doi.org/10.12987/yale/9780300123821.003.0003.
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This chapter focuses on the deaths of the people who died in Ireland in 1917. Some of these deaths were those of the released 1916 Rising prisoners, including packaging porter Christopher Brady, who was released due to ill-health and died at home from pneumonia. Other 1916 Rising prisoners, like carpenter Bernard Ward, died from prison-related illness. Trade unionist engineer William Partridge, who died two months after release from Lewes on medical grounds and whose 'death was due to prison treatment', became a union official after losing his railway job for protesting at the preferential promotion of Protestants. Meanwhile, schoolteacher Thomas Ashe was jailed in Mountjoy for a seditious speech, during which he and others went on hunger strike for political status. Ashe died due to 'heart failure and congestion of the lungs caused by being left to lie on the cold floor for fifty hours and then subjected to forcible feeding in his weak condition after hunger strike'. Police reported that Ashe's death 'evoked demonstrations of sympathy on the part of Nationalists' across Ireland and gave a fresh impetus to the Sinn Féin movement.
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Neuberger, James. "The liver in systemic disease." In Oxford Textbook of Medicine, edited by Jack Satsangi, 3169–78. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0331.
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The liver is affected in many systemic diseases, with important examples being cardiovascular diseases—raised venous pressure can lead to hepatic congestion. Hepatomegaly is frequent in moderately severe heart failure. Cardiac cirrhosis is a rare complication. Pulmonary diseases—conditions that involve the liver as well as the lungs include cystic fibrosis, sarcoidosis, and α1-antitrypsin deficiency. Gastrointestinal diseases—inflammatory bowel disease is associated with a range of hepatic pathology including fatty change, pericholangitis, sclerosing cholangitis, autoimmune hepatitis, cirrhosis, and (rarely) amyloidosis. Hepatobiliary disease associated with total parenteral nutrition varies from a mild, asymptomatic disease to jaundice, cirrhosis, and liver failure. Coeliac disease may rarely present with abnormal liver tests. Obesity, especially in association with the metabolic syndrome, may be associated with nonalcoholic hepatitis and steatohepatitis. Endocrine diseases—autoimmune hepatitis and primary biliary cholangitis may be associated with autoimmune endocrine disorders. Both hypothyroidism and hyperthyroidism can cause abnormalities of liver function, which are usually mild. Haematological diseases—conditions associated with abnormal blood clotting, such as protein C or S deficiency and paroxysmal nocturnal haemoglobinuria, may lead to Budd–Chiari syndrome (hepatic vein thrombosis). The liver may be involved in both non-Hodgkin’s lymphoma and leukaemia. Infectious diseases—agents that particularly affect the liver (e.g. viral hepatitis) are discussed elsewhere although many systemic infections also infect the liver. Abnormal liver function may occur during many systemic infections, but it is rare for patients with sepsis to present primarily with liver symptoms, although jaundice, abnormal liver function tests, or (very rarely) fulminant hepatic failure may be the principal presenting feature. Rheumatological diseases—hepatic disease may either be a consequence of treatment or occur in association with other autoimmune diseases.
Conference papers on the topic "Lung congesti"
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Omer, Noam, Shimon Abboud, and Marina Arad. "Classifying lung congestion in congestive heart failure using electrical impedance - a 3D model." In 2015 Computing in Cardiology Conference (CinC). IEEE, 2015. http://dx.doi.org/10.1109/cic.2015.7408663.
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Saint-Pierre, M. D., J. Abdulnour, and R. Sabbagh. "Interstitial Lung Disease and Congestive Heart Failure Overlap: Impact of this Presentation on Hospitalization Rates." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a1587.
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Saint-Pierre, M. D., J. Abdulnour, and R. Sabbagh. "Inhaler Prescription in Congestive Heart Failure Patients Without Evidence of Obstructive Lung Disease: A Single-Center Review." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a1599.
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