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Relevant bibliographies by topics / Lung Neoplasms/chemically induced

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Academic literature on the topic 'Lung Neoplasms/chemically induced'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "Lung Neoplasms/chemically induced"

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Gunning, W. T., G. D. Stoner, and P. J. Goldblatt. "Ultrastructural observations of lung neoplasms induced in the strain A/J mouse by benzo(a)pyrene and ethylnitrosourea." Proceedings, annual meeting, Electron Microscopy Society of America 45 (August 1987): 854–55. http://dx.doi.org/10.1017/s0424820100128559.

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The current literature regarding the histogenesis of mouse lung adenomas suggests two alternative cells of origin for these tumors. The Type II pneumocyte was accepted by most investigators as the cell which proliferated either spontaneously or by chemical induction, until Kauffman and co-workers reported histologic and ultrastructural features of a Clara cell adenoma, and indicated that in Swiss mice, 63% of all chemically induced tumors appeared to be Clara cell derived. Ward et al. using immunocytochemical techniques have demonstrated that all lung adenomas they tested in B6C3F1, BALB, and

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Dixon, Darlene, and Robert R. Maronpot. "Histomorphologic Features of Spontaneous and Chemically-Induced Pulmonary Neoplasms in B6C3F1 Mice and Fischer 344 Rats." Toxicologic Pathology 19, no. 4_part_1 (1991): 540–56. http://dx.doi.org/10.1177/0192623391019004-119.

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The histomorphologic features of spontaneous and chemically-induced lung neoplasms in male and female B6C3F1 mice and Fischer 344 rats are described. Primary pulmonary neoplasms in mice and rats were classified as alveolar/bronchiolar (A/B) adenoma or carcinoma (including variants with squamous and mucinous cell differentiation), bronchial adenoma or carcinoma, squamous cell carcinoma or mesenchymal tumors. A/B adenomas and carcinomas were the most common spontaneous pulmonary neoplasms observed in both mice and rats, but were observed less frequently in rats. In the National Toxicology Progra

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Ton, Thai-Vu T., Hue-Hua L. Hong, Colleen H. Anna, et al. "Predominant K-ras Codon 12 G → A Transition in Chemically Induced Lung Neoplasms in B6C3F1 Mice." Toxicologic Pathology 32, no. 1 (2004): 16–21. http://dx.doi.org/10.1080/01926230490260682.

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Karabela, Sophia, Ioannis Psallidas, Taylor Sherrill та ін. "Opposing effects of bortezomib-induced nuclear factor-κB inhibition on chemical lung carcinogenesis." Carcinogenesis 33, № 4 (2012): 859–67. https://doi.org/10.1093/carcin/bgs024.

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Since recent evidence indicates a requirement for epithelial nuclear factor (NF)-κB signaling in lung tumorigenesis, we investigated the impact of the NF-κB inhibitor bortezomib on lung tumor promotion and growth. We used an experimental model in which wild-type mice or mice expressing an NF-κB reporter received intraperitoneal urethane (1 g/kg) followed by twice weekly bortezomib (1 mg/kg) during distinct periods of tumor initiation/progression. Mice were serially assessed for lung NF-κB activation, inflammation and carcinogenesis. Short-term proteasome inhibition with bortezomib did not impa

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Tanaka, Takuji, Takahiro Tanaka, Mayu Tanaka та Toshiya Kuno. "Cancer Chemoprevention by Citrus Pulp and Juices Containing High Amounts ofβ-Cryptoxanthin and Hesperidin". Journal of Biomedicine and Biotechnology 2012 (2012): 1–10. http://dx.doi.org/10.1155/2012/516981.

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β-Cryptoxanthin, a carotenoid, and hesperidin, a flavonoid, possess inhibitory effects on carcinogenesis in several tissues. We recently have prepared a pulp (CHRP) and citrus juices (MJ2 and MJ5) from a satsuma mandarin (Citrus unshiuMar.) juice (MJ). They contain high amounts ofβ-cryptoxanthin and hesperidin. We have demonstrated that CHRP and/or MJs inhibit chemically induced rat colon, rat tongue, and mouse lung tumorigenesis. Gavage with CHRP resulted in an increase of activities of detoxifying enzymes in the liver, colon, and tongue rats'. CHRP and MJs were also able to suppress the expr

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Ogawa, Kumiko, Katsumi Imaida, Tsuneo Masui, et al. "Chemically induced lung and forestomach neoplasias in transgenic mice carry mutant forms of the human c-Ha-ras transgene." Carcinogenesis 17, no. 2 (1996): 341–45. http://dx.doi.org/10.1093/carcin/17.2.341.

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Karabela, Sophia, Chrysoula Kairi, Sophia Magkouta, et al. "Neutralization of tumor necrosis factor bioactivity ameliorates urethane-induced pulmonary oncogenesis in mice." Neoplasia (New York, N.Y.) 13, no. 12 (2011): 1143–51. https://doi.org/10.1593/neo.111224.

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Tumor necrosis factor (TNF) has been implicated in inflammation-associated tumor progression. Although multiple reports identified a role for TNF signaling in established cancers, few studies have assessed the impact of TNF blockade on early tumor formation promotion. We aimed at exploring the effects of TNF neutralization in a preclinical mouse model of lung carcinogenesis. For this, Balb/c mice (n = 42) received four weekly intraperitoneal urethane injections (1 g/kg) and twice-weekly intraperitoneal soluble TNF receptor (etanercept; 10 mg/kg) administered during tumor initiation/promotion,

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Wang, Min, Theodora R. Devereux, Haris G. Vikis та ін. "Pol ι Is a Candidate for the Mouse Pulmonary Adenoma Resistance 2 Locus, a Major Modifier of Chemically Induced Lung Neoplasia". Cancer Research 64, № 6 (2004): 1924–31. http://dx.doi.org/10.1158/0008-5472.can-03-3080.

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Maydin, M. A., M. N. Yurova, E. I. Fedoros, et al. "Effect of inactivating heterozygous mutations in DNA repair genes on experimental lung carcinogenesis in mice." Russian Journal of Biotherapy 23, no. 1 (2024): 37–44. http://dx.doi.org/10.17650/1726-9784-2024-23-1-37-44.

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Background. Inactivating mutations in Chek2 and Gprc5a genes are known to be associated with cancer development. Experimental carcinogenesis studies in genetically modified mice generate new data on their influence on pathology development.Aim. In the present study in a model of lung carcinogenesis, survival parameters as well as tumor multiplicity and size in mice with Chek2 and Gprc5a heterozygous inactivating mutations were evaluated.Material and methods. F2 hybrid mice from crosses between CBAB6F1 males heterozygous for the studied mutations and wild-type BALB / c females were used: Chek2d

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Green, John. "Chemically Induced Lung Sensitisation." Indoor and Built Environment 3, no. 3 (1994): 166–67. http://dx.doi.org/10.1159/000463547.

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Dissertations / Theses on the topic "Lung Neoplasms/chemically induced"

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Prochazka, Michaela. "The risk of second primary lung carcinoma in breast cancer patients /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-649-2/.

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Kayahara, Giseli Mitsuy. "Melatonin suppression increases the incidence and progression of chemically induced oral cancer in rats /." Araçatuba, 2019. http://hdl.handle.net/11449/184152.

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Orientador: Daniel Galera Bernabé<br>Resumo: Estudos sugerem que a supressão de melatonina e disfunção circadiana em trabalhadores noturnos podem estar relacionadas ao desenvolvimento e à progressão do câncer. Pesquisas têm mostrado também que a incidência tumoral pode ser aumentada pela pinealectomia. Entretanto, nenhum estudo avaliou a influência da cirurgia de pinealectomia sobre o desenvolvimento e a progressão do câncer de boca. No presente estudo, nós investigamos os efeitos da supressão de melatonina sobre a ocorrência e a progressão tumoral um modelo pré-clínico de câncer de boca induz

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Campbell, Holly R. 1976. "Chlorine-induced lung injury and the role of iNOS." Thesis, McGill University, 2009. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=111574.

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Reactive airways dysfunction syndrome (RADS), a form of irritant-induced asthma (IIA) has been observed in humans following acute chlorine (Cl 2) gas exposure in occupational and domestic settings. Following Cl 2 injury, subepithelial fibrosis, mucous hyperplasia, and non-specific airway hyperresponsiveness have been reported. Based on the disease profile, we hypothesized that pulmonary damage may be oxidative in nature.<br>The aim of this work was to develop a murine model of irritant-induced asthma in order to investigate the pathogenic processes and potential oxidative mechanisms involved i

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Sjögren, Anna-Karin. "The importance of isoprenylation and Nf1 deficiency in K-RAS-induced cancer /." Göteborg : The Wallenberg Laboratory, Department of Molecular and Clinical Medicine, Institute of Medicine at Sahlgrenska Academy, University of Gothenburg, 2009. http://hdl.handle.net/2077/21218.

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Verza, Flávia Alves. "Social isolation stress increases the incidence and growth of chemically induced oral cancer in rats /." Araçatuba, 2017. http://hdl.handle.net/11449/151527.

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Orientador: Daniel Galera Bernabé<br>Coorientadora: Sandra Helena Penha de Oliveira<br>Banca: Ana Maria Pires Soubhia<br>Banca: André Caroli Rocha<br>Resumo: Estudos têm mostrado que o estresse crônico pode influenciar a progressão do câncer, porém sua influência sobre o início da doença é pouco compreendida. Dentre os eventos estressores, o estresse por isolamento social (EIS) afeta grande número de indivíduos, podendo induzir alterações neuro-hormonais e desordens emocionais. Embora estudos mostrem que o EIS pode aumentar a progressão de alguns tipos de neoplasias malignas, sua influência so

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Gonçalves, Bianca Facchim 1986. "Carcinogênese prostática quimicamente induzida por N-metil N-nitrosuréia (MNU) em gerbilos da Mongólia = associação com promotores esteróides ou dieta hiperlipídica = Prostate carcinogenesis chemically induced by N-methyl-N Prostate carcinogenesis chemically induced by N-methyl-Nnitrosourea: association with steroids promoters or high-fat diet." [s.n.], 2013. http://repositorio.unicamp.br/jspui/handle/REPOSIP/317938.

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Orientadores: Sebastião Roberto Taboga, Silvana Gisele Pegorin de Campos<br>Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia<br>Made available in DSpace on 2018-08-23T14:53:15Z (GMT). No. of bitstreams: 1 Goncalves_BiancaFacchim_D.pdf: 8580892 bytes, checksum: 1e3927b5ad73e189b4b4245aa141895e (MD5) Previous issue date: 2013<br>Resumo: Um dos principais desafios no campo de pesquisa do câncer prostático é a busca por sistemas modelo que permitam a investigação dos aspectos patológicos, bioquímicos e genéticos desta doença. O gerbilo Meriones unguiculatus tem possibi

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Reis, Rafael de Almeida dos. "Efeito do inibidor de proteinase de origem vegetal BbCI, sobre a lesão pulmonar induzida pela elastase em camundongos C57BI6." Universidade de São Paulo, 2014. http://www.teses.usp.br/teses/disponiveis/5/5165/tde-02062014-095433/.

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Introdução: As proteinases tem um papel importante no desenvolvimento, na destruição tecidual e na produção de muco causada pela DPOC. O inibidor de proteinase de origem vegetal Bauhinia bauhinioides Cruzipain Inhibitor (BbCI) inibe tanto as proteinases da classe serina quanto da classe cisteína. Objetivos: Deste modo consideramos relevante estudar os efeitos do tratamento com BbCI nas alterações pulmonares induzidas pela elastase em camundongos. Métodos: Camundongos C57Bl6 receberam elastase via intratraqueal (50 uL/animal, grupo ELA) ou salina (grupo SAL). Os camundongos foram tratados com B

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Fló, Claudia Marina. "Efeito da atividade física sobre a evolução do enfisema pulmonar: um estudo experimental em ratos wistar." Universidade de São Paulo, 2003. http://www.teses.usp.br/teses/disponiveis/5/5160/tde-05092014-161209/.

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O propósito da presente investigação foi avaliar o papel da atividade física no desenvolvimento de enfisema induzido por papaína em ratos. Para tanto, ratos Wistar foram randomicamente divididos em quatro grupos (n = 10 para cada grupo) que receberam, respectivamente, infusão intra-traqueal de papaína (6 mg em 1 mL de NaCl 0,9%) ou veículo e foram submetidos ou não ao protocolo de exercício em uma esteira ergométrica. Os ratos exercitaram-se a 13,3 m/min, 6 dias por semana, durante 9 semanas (o tempo de exercício foi aumentado gradualmente, de 10 a 35 min). Foram medidas a elastância e a resis

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Junior, Osmar Aparecido Theodoro. "Efeito do inibidor de proteinase de origem vegetal EcTI, sobre a lesão pulmonar induzida pela elastase em camundongos C57BI6." Universidade de São Paulo, 2014. http://www.teses.usp.br/teses/disponiveis/5/5165/tde-04082014-090830/.

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Introdução: As proteinases tem um papel importante no desenvolvimento, na destruição tecidual e na produção de muco causada pela DPOC. O inibidor de proteinase de origem vegetal Enterolobium contortisiliquum Tripsin Inhibitor (EcTI) inibe tanto as proteinases da classe serina quanto da classe cisteína. Objetivos: Avaliar os efeitos do tratamento com EcTI nas alterações pulmonares induzidas pela elastase em camundongos. Métodos: Camundongos C57Bl6 receberam elastase via intratraqueal (50 uL/animal, grupo ELA) ou salina (grupo SAL). Os camundongos foram tratados com EcTI (2mg/kg) nos dias 1, 15

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Capelozzi, Marco Antonio. ""Determinantes funcionais e morfológicos de ação de droga sobre os pulmões utilizando um modelo experimental em cobaias sob uso do cloridrato de fluoxetina"." Universidade de São Paulo, 2005. http://www.teses.usp.br/teses/disponiveis/5/5144/tde-29092005-155733/.

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Muito se tem pesquisado sobre os efeitos adversos dos antidepressivos tricíclicos (p.e. imipramina) sobre o sistema respiratório, embora pouco ou quase nada se encontre com relação a tal aspecto na literatura médica sobre a fluoxetina (Prozac®) um inibidor seletivo da recaptação de serotonina, até porque esta droga começou a ser utilizada somente há cerca de quinze anos. Ambas substâncias (fluoxetina e imipramina, ou seus congêneres) agem basicamente sobre quadros depressivos, fóbicos e obsessivo-compulsivos, obesidade, anorexia, e outras indicações de várias naturezas, indo desde a ansiedade

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Books on the topic "Lung Neoplasms/chemically induced"

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L, Mauderly J., and McCunney Robert J, eds. Particle overload in the rat lung and lung cancer: Implications for human risk assessment : proceedings of a conference held at the Massachusetts Institute of Technology on March 29 and 30, 1995. Taylor & Francis, 1996.

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International Agency for Research on Cancer and World Health Organization, eds. Carbon black, titanium dioxide, and talc. International Agency for Research on Cancer, 2010.

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National Research Council (U.S.). Committee on Health Effects of Exposure to Radon., ed. Health effects of exposure to radon: Time for reassessment? National Academy Press, 1994.

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NATO Advanced Research Workshop on Mechanisms in Fibre Carcinogenesis (1990 Albuquerque, N.M.). Mechanisms in fibre carcinogenesis. Plenum Press, 1991.

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D, Cooper J. Allen, ed. Drug-induced pulmonary disease. W. B. Saunders, 1990.

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Imperial Cancer Research Fund (Great Britain). Hormones and cancer: 90 years after Beatson. Edited by Bulbrook R. D. Published for the Imperial Cancer Research Fund by Oxford University Press, 1986.

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Andrew, Churg, and Green Francis H. Y, eds. Pathology of occupational lung disease. 2nd ed. Williams & Wilkins, 1998.

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Sardinian, International Meeting on Models and Mechanisms in Chemical Carcinogenesis (4th 1987 Alghero Italy). Chemical carcinogenesis: Models and mechanisms. Plenum Press, 1988.

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Imperial Cancer Research Fund (Great Britain). Nitrate, nitrite and nitroso compounds in human cancer. Edited by Forman David, Shuker David, and Shuker David E. G. Published for the Imperial Cancer Research Fund by Oxford University Press, 1989.

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National Toxicology Program (U.S.). NTP technical report on the toxicology and carcinogenesis studies of 1,3-butadiene: (CAS no. 106-99-0) in B6C3F mice (inhalation studies). U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, National Toxicology Program ; Springfield, VA : Available for sale from the National Technical Information Service, U.S. Dept. of Commerce, 1993.

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Book chapters on the topic "Lung Neoplasms/chemically induced"

1

"Drug-Induced Lung Disease." In Specialty Imaging: Thoracic Neoplasms. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-323-37706-5.50156-6.

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Ajaykumar, Chittipolu, K. Swathi, and Pavithra Dasari. "Effect of Ocimum sanctum on Cancer Progression and Its Effective Therapeutic Activity." In Advances in Medical Diagnosis, Treatment, and Care. IGI Global, 2023. http://dx.doi.org/10.4018/979-8-3693-0703-8.ch001.

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Ocimum sanctum L., sometimes referred to as Tulsi in various Indian languages or Holy Basil in English, is a significant medicinal herb used in Southeast Asian traditional and folk medicine. It has been demonstrated via scientific studies to have immunomodulatory, anti-inflammatory, antidiabetic, hypolipidemic, antipyretic, analgesic, hepatoprotective, and stress-relieving properties. Studies have also demonstrated that Tulsi and some of its phytochemicals—including eugenol, apigenin, myretenal, sitosterol, rosmarinic acid, carnosic acid and luteolin—prevent chemically induced oral, gastric, c

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Conference papers on the topic "Lung Neoplasms/chemically induced"

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Sargent, Linda M., Michael L. Kashon, Ann F. Hubbs, et al. "Abstract 2345: Amplification of mouse chromosome 4 in chemically induced and invasive mouse lung adenocarcinoma." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-2345.

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Utembe, Wells, and Arox Wadson Kamng’ona. "The Knowns and Unknowns of Chemically Induced Lower Respiratory Tract Microbiota Dysbiosis and Lung Disease." In ECAS 2023. MDPI, 2023. http://dx.doi.org/10.3390/ecas2023-1634.

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Lu, Gang, Hang Xiao, GuangXun Li та ін. "Abstract 2874: A γ-tocopherol-rich mixture of tocopherols inhibits chemically induced lung tumorigenesis in A/J mice and xenograft tumor growth". У Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-2874.

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