Books on the topic 'Lungs Inflammation'

To see the other types of publications on this topic, follow the link: Lungs Inflammation.

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 50 books for your research on the topic 'Lungs Inflammation.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse books on a wide variety of disciplines and organise your bibliography correctly.

1

Symposium, on Airway Obstruction and Inflammation (1988 Florence Italy). Airway obstruction and inflammation: Present status and perspectives. Basel: Karger, 1990.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
2

Criner, Gerard J., William D. Cornwell, and Thomas J. Rogers. Smoking and lung inflammation: Basic, pre-clinical, and clinical research advances. New York: Springer, 2013.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
3

L, Kradin Richard, and Robinson Bruce W. S, eds. Immunopathology of lung disease. Boston: Butterworth-Heinemann, 1996.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
4

Bertolini, Renzo. Animal and vegetable dusts as a cause of deep lung inflammation. Hamilton: CCOHS, 1988.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
5

Michel, Chignard, and Conference on Cytokines and Adhesion Molecules in Lung Inflammation (1995 : Paris, France), eds. Cytokines and adhesion molecules in lung inflammation. New York: New York Academy of Sciences, 1996.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
6

F, Donner C., ed. COPD is/is not a systemic disease? Hauppauge, NY: Nova Science, 2009.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
7

1952-, Strieter Robert M., Kunkel S. L, and Standiford Theodore J, eds. Chemokines in the lung. New York: Marcel Dekker, Inc., 2003.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
8

Rogers, Thomas J., Gerard J. Criner, and William D. Cornwell, eds. Smoking and Lung Inflammation. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-7351-0.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Dubinett, Steven M., ed. Inflammation and Lung Cancer. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2724-1.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Alper, Scott, and William J. Janssen, eds. Lung Innate Immunity and Inflammation. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8570-8.

Full text
APA, Harvard, Vancouver, ISO, and other styles
11

Wang, Yong-Xiao, ed. Lung Inflammation in Health and Disease, Volume I. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-63046-1.

Full text
APA, Harvard, Vancouver, ISO, and other styles
12

Wang, Yong-Xiao, ed. Lung Inflammation in Health and Disease, Volume II. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-68748-9.

Full text
APA, Harvard, Vancouver, ISO, and other styles
13

Pokorski, Mieczyslaw. Pulmonary Infection and Inflammation. Springer London, Limited, 2016.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
14

Pokorski, Mieczyslaw. Pulmonary Infection and Inflammation. Springer, 2018.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
15

Pokorski, Mieczyslaw. Pulmonary Infection and Inflammation. Springer, 2016.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
16

Dubinett, Steven M. Inflammation and Lung Cancer. Springer, 2015.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
17

Dubinett, Steven M. Inflammation and Lung Cancer. Springer, 2016.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
18

Dubinett, Steven M. Inflammation and Lung Cancer. Springer, 2015.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
19

Rogers, Thomas J., Gerard J. Criner, and William D. Cornwell. Smoking and Lung Inflammation: Basic, Pre-Clinical and Clinical Research Advances. Springer, 2016.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
20

Martin, Thomas. Cytokines in Pulmonary Disease: Infection and Inflammation (Lung Biology in Health & Disease). Informa Healthcare, 2000.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
21

Strieter, Robert M., Steven L. Kunkel, and Theodore J. Standiford. Chemokines in the Lung. Taylor & Francis Group, 2003.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
22

Michel, Chignard, ed. Cells and cytokines in lung inflammation. New York, NY: New York Academy of Sciences, 1994.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
23

(Editor), Duncan F. Rogers, and Louise E. Donnelly (Editor), eds. Human Airway Inflammation: Sampling Techniques and Analytical Protocols (Methods in Molecular Medicine). Humana Press, 2001.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
24

(Editor), Trevor T. Hansel, and Peter J. Barnes (Editor), eds. Recent Advances in the Pathophysiology of COPD (Progress in Inflammation Research). Birkhäuser Basel, 2004.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
25

(Editor), Johan Zaagsma, Herman Meurs (Editor), and Ad F. Roffel (Editor), eds. Muscarinic Receptors in Airways Diseases (Progress in Inflammation Research). Birkhauser, 2001.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
26

Montuschi, Paolo. New Perspectives in Monitoring Lung Inflammation: Analysis of Exhaled Breath Condensate. Taylor & Francis Group, 2004.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
27

Montuschi, Paolo. New Perspectives in Monitoring Lung Inflammation: Analysis of Exhaled Breath Condensate. Taylor & Francis Group, 2004.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
28

Montuschi, Paolo. New Perspectives in Monitoring Lung Inflammation: Analysis of Exhaled Breath Condensate. Taylor & Francis Group, 2004.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
29

New perspectives in monitoring lung inflammation: Analysis of exhaled breath condensate. Boca Raton, FL: CRC Press, 2006.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
30

Montuschi, Paolo. New Perspectives in Monitoring Lung Inflammation: Analysis of Exhaled Breath Condensate. Taylor & Francis Group, 2004.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
31

Montuschi, Paolo. New Perspectives in Monitoring Lung Inflammation: Analysis of Exhaled Breath Condensate. Taylor & Francis Group, 2004.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
32

Acute Lung Injury: From Inflammation to Repair (Biomedical and Health Research). Ios Pr Inc, 2001.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
33

A, Persson C. G., ed. Inflammatory indices in chronic bronchitis. Basel: Birkhäuser, 1990.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
34

1942-, Church Martin, and Robinson Clive 1958-, eds. Eicosanoids in inflammatory conditions of the lung, skin, and joints. Lancaster: MTP Press, 1988.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
35

Ray, Abhijit, and Punit Kumar Srivastave. Obstructive Airway Diseases. Taylor & Francis Group, 2011.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
36

Ray, Abhijit, and Punit Kumar Srivastava. Obstructive Airway Diseases: Role of Lipid Mediators. Taylor & Francis Group, 2016.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
37

Ray, Abhijit. Obstructive Airway Diseases: Role of Lipid Mediators. Taylor & Francis Group, 2011.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
38

Ray, Abhijit, and Punit Kumar Srivastava. Obstructive Airway Diseases: Role of Lipid Mediators. Taylor & Francis Group, 2016.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
39

Ray, Abhijit, and Punit Kumar Srivastava. Obstructive Airway Diseases: Role of Lipid Mediators. Taylor & Francis Group, 2018.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
40

Ong, Kian Chung, ed. Lung Inflammation. InTech, 2014. http://dx.doi.org/10.5772/57068.

Full text
APA, Harvard, Vancouver, ISO, and other styles
41

Adlam, David. Pericardial disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0109.

Full text
APA, Harvard, Vancouver, ISO, and other styles
Abstract:
The pericardium forms a continuous sac around the heart, analogous to the pleura surrounding the lungs, and the peritoneum surrounding the abdominal viscera. Between the parietal and visceral layers of the serous pericardium is the pericardial space, which normally contains a small volume of pericardial fluid. The clinical spectrum of pericardial diseases can be divided into: pericarditis, caused by acute inflammation; pericardial effusion, or fluid accumulation in the pericardial space, leading to tamponade; and constrictive pericarditis, caused by chronic infiltration or inflammation leading to pericardial constriction.
42

Wick, Mark R., and Henry D. Tazelaar. Neoplastic Mimics in Thoracic and Cardiovascular Pathology. Springer Publishing Company, Incorporated, 2013.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
43

Wick, Mark R., and Henry D. Tazelaar. Neoplastic Mimics in Thoracic and Cardiovascular Pathology. Springer Publishing Company, Incorporated, 2013.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
44

Wiffen, Philip, Marc Mitchell, Melanie Snelling, and Nicola Stoner. Therapy-related issues: musculoskeletal diseases. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199603640.003.0025.

Full text
APA, Harvard, Vancouver, ISO, and other styles
Abstract:
Rheumatoid arthritis 548Gout 556Rheumatoid arthritis (RA) is an autoimmune disease which causes joints lined with synovium to become inflamed, swollen, stiff, and painful, and leads to joint erosion. It is a multisystem disorder which can affect many organs including the eyes, lungs, heart, and blood vessels. The aim of treatment is to decrease pain and inflammation, prevent joint damage, and ultimately induce remission of disease....
45

Blend, Doctor Herry. Ketoconazole: Perfect Guide for Most Effective Antifungal Treatment That Completely Cures Jock Itch, Athlete's Foot, Ringworm, Painful Inflammation, Lungs or Skin Diseases and Other Fungal Diseases. Lulu Press, Inc., 2018.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
46

Klepikov, Igor. Didactics of Acute Lung Inflammation. Cambridge Scholars Publishing, 2022.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
47

Kriemler, Susi, Thomas Radtke, and Helge Hebestreit. Exercise, physical activity, and cystic fibrosis. Edited by Neil Armstrong and Willem van Mechelen. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198757672.003.0027.

Full text
APA, Harvard, Vancouver, ISO, and other styles
Abstract:
Cystic fibrosis (CF) is a genetic disease resulting in an impaired mucociliary clearance, chronic bacterial airway infection, and inflammation. The progressive destruction of the lungs is the main cause of morbidity and premature death. Diverse other organ systems such as heart, muscles, bones, gastro-intestinal tract, and sweat glands are often also affected and interfere with exercise capacity. Hence, exercise capacity is reduced as the disease progresses mainly due to reduced functioning of the muscles, heart, and/or lungs. Although there is still growing evidence of positive effects of exercise training in CF on exercise capacity, decline of pulmonary function, and health-related quality of life, the observed effects are encouraging and exercise should be implemented in all patient care. More research is needed to understand pathophysiological mechanisms of exercise limitations and to find optimal exercise modalities to slow down disease progression, predict long-term adherence, and improve health-related quality of life.
48

Balhara, Kamna S., Basem F. Khishfe, and Jamil D. Bayram. Sepsis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0004.

Full text
APA, Harvard, Vancouver, ISO, and other styles
Abstract:
Sepsis is a clinical syndrome characterized by systemic inflammation in the presence of infection. The source of infection may be occult. One must be aware of the epidemiology, presenting features and complications, diagnostic considerations and tests, and the organisms involved. Bacteria (gram positive and negative) are most commonly associated with sepsis, although fungi, viruses, and parasites can cause sepsis. Infections in the lungs, urinary tract, abdomen, skin, brain, and other areas can cause bacteremia and lead to sepsis. Treatment includes airway, breathing, and circulation (ABCs) management; aggressive fluid resuscitation; early administration of broad-spectrum antibiotics; and early goal-directed therapy and severe sepsis resuscitation bundle. Diagnosis can be challenging in pediatric and geriatric populations.
49

Lynn, William S. Inflammatory Cells & Lung Disease. Taylor & Francis Group, 2020.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
50

Kriemler, Susi. Exercise, physical activity, and cystic fibrosis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199232482.003.0033.

Full text
APA, Harvard, Vancouver, ISO, and other styles
Abstract:
Cystic fibrosis (CF) is the most common genetic autosomal recessive disease of the Caucasian race, generally leading to death in early adulthood.1 The frequency of the gene carrier (heterozygote) is 1:20–25 in Caucasian populations, 1:2000 in African-Americans, and practically non-existent in Asian populations. The disease occurs in about 1 in every 2500 life births of the white population. Mean survival has risen from 8.4 years in 1969 to 32 years in 2000 due to improvements in treatment. The genetic defect causes a pathological electrolyte transport through the cell membranes by a defective chloride channel membrane transport protein [cystic fibrosis transmembrane conductance regulator (CFTR)]. With respect to the function, this affects mainly the exocrine glands of secretory cells, sinuses, lungs, pancreas, liver, and the reproductive tract of the human body leading to a highly viscous, water-depleted secretion. The secretion cannot leave the glands and in consequence causes local inflammation and destruction of various organs. The main symptoms include chronic inflammatory pulmonary disease with a progressive loss of lung function, exocrine and sometimes endocrine pancreas insufficiency, and an excessive salt loss through the sweat glands.1 A summary of the signs and symptoms of CF will be given with a special emphasis on the effect of exercise performance and capacity.

To the bibliography