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Relevant bibliographies by topics / Lupus erythematosus

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Academic literature on the topic 'Lupus erythematosus'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 February 2023

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Journal articles on the topic "Lupus erythematosus"

1

Chinnusamy, Manokaran, Ram Arvind Viswanathan, Sathiyanarayanan Janakiraman, and Roshna Elayidath. "Drug-Induced Lupus Erythematosus Associated with Proton Pump Inhibitor." Journal of Health and Allied Sciences NU 10, no. 03 (September 8, 2020): 132–34. http://dx.doi.org/10.1055/s-0040-1716601.

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AbstractDrug-induced lupus erythematosus is an autoimmune phenomenon where the drug exposure leads to the development of systemic lupus erythematous like clinical features. Drug-induced lupus erythematosus can be divided into systemic lupus erythematous, subacute cutaneous lupus erythematous, and chronic cutaneous lupus erythematous. Here, we report a case of a 29-year-old female presented with systemic lupus erythematous due to chronic use of proton pump inhibitors, which is considered to be very rare.

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Garbelini-Lima, Cleide, Gabriela Evangelista de Almeida, Sidharta Quércia Gabdelha, Andrea Cavalcante de Souza, Mara Lúcia Gomes de Souza, and Virginia Vilasboas Figueiras. "Discoid Lupus Erythematosus of the Scalp in a Patient with Systemic Lupus Erythematosus: A Case Report with Complete Hair Regrowth." Journal of the Portuguese Society of Dermatology and Venereology 79, no. 2 (June 26, 2021): 155–58. http://dx.doi.org/10.29021/spdv.79.2.1283.

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Scalp involvement with hair loss is common in systemic lupus erythematosus. Discoid lupus erythematosus may cause scarring alopecia, characterized by well-delimited erythematous plaques with scales, follicular hyperkeratosis and atrophy, which is considered a trichological emergency. Early diagnosis and treatment are necessary in order to prevent permanent hair loss. We describe a 44 years’ old female patient with systemic lupus erythematosus for 4 years, with multiple areas of occipitoparietal alopecia, erythematous plaques, atrophy, scales and some bloody crusts. Trichoscopy, histopathology and direct immunofluorescence led to the diagnosis of discoid lupus erythematosus. After 9 months treatment with thalidomide there was complete hair regrowth.

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Frey, Marcos Noronha, Ana Elisa Empinotti Ioppi, Gabriela Czarnobay Garbin, Roque Domingos Furian, and Ana Elisa Kiszewski Bau. "Congenital and neonatal lupus erythematosus: two case reports." Anais Brasileiros de Dermatologia 87, no. 4 (August 2012): 625–28. http://dx.doi.org/10.1590/s0365-05962012000400019.

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Neonatal lupus erythematosus is an autoimmune disease produced by the passage of maternal antinuclear antibodies and extractable nuclear antigen antibodies through the placenta. At the moment of the diagnosis, the mothers are asymptomatic in 40 to 60% of cases. The most common manifestations are cutaneous lesions and congenital heart block. The cutaneous findings are variable and usually begin within the first weeks or months of life. Congenital lupus erythematosus is a congenital variant of neonatal lupus erythematosus. We present one case of congenital lupus erythematosus and one case of neonatal lupus erythematous, showing the variability of this disease.

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Huzairi Sani, Nada Syazana, and Malek Faris Riza Feisal. "A different kind of skin presentation in Systemic Lupus Erythematosus (SLE): A case report." Asian Journal of Medicine and Biomedicine 4, no. 1 (April 24, 2020): 42–46. http://dx.doi.org/10.37231/ajmb.2020.4.1.329.

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Erythema nodosum is a septal panniculitis which is a variant of chronic cutaneous lupus erythematosus (CCLE). It is further classified in the group of Lupus Erythematous Panniculitis (LEP).[1] The most frequent cutaneous manifestations include indurated plaques, subcutaneous nodules and sometimes ulcerations. The lesions occur predominantly on the face, upper arms, upper trunk, breasts, buttocks and thighs.[2] They occur most frequently in adult females and do not typically manifest cutaneously in Systemic Lupus Erythematosus (SLE).[3] In this case report, we discuss a young gentleman who presented with erythema nodosum as a cutaneous feature of SLE. Keywords: Systemic lupus erythematosus, erythema nodosum, panniculitis, cutaneous lupus

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Guleva, D., M. Balabanova, L. Miteva, and L. Dourmishev. "Histology of Skin Alterations in Lupus Erythematosus." Acta Medica Bulgarica 49, no. 2 (June 1, 2022): 28–32. http://dx.doi.org/10.2478/amb-2022-0016.

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Abstract Lupus erythematosus is an autoimmune connective tissue disorder showing a broad spectrum of clinical manifestations. The aim of this study was to assess the correlation of skin histology and different types of lupus erythematosus. Materials and methods: Fifty-one skin specimens were assessed from 39 female and 12 male patients with acute, subcutaneous and chronic lupus erythematosus, diagnosed and treated in the Department of Dermatology and Venereology, Alexandrovska University Hospital for a 4-year period. Results: Follicular hyperkeratosis, epidermal atrophy, vacuolar degeneration and interface dermatitis were the most frequently observed lesions in chronic cutaneous lupus erythematosus while diffuse hyperkeratosis, epidermal atrophy and indistinct interface dermatitis in the dermis were predominant in subacute cutaneous lupus erythematosus. Lupus tumidus, a rare intermittent variant of cutaneous lupus erythematosus, showed almost no epidermal involvement and mucin deposition in the dermis. However, in one of our lupus tumidus patients the disease progressed to a systemic form with histological changes of acute cutaneous lupus erythematosus including atrophy, dermal-epidermal smoothing and lymphocytic infiltration in the dermis. Of note, a few patients showed histological changes of urticarial vasculitis-like and rheumatic-like patterns. Conclusion: The correlation of clinical course, histopathological findings and immunological tests are of vital importance for the correct diagnosis and follow up of patients with lupus erythematodes, thus preventing complications and improving their quality of life.

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Mrabat, Samia, Hanane aybay, Zakia Douhi, Sara Elloudi, Fatima Zahra Mernissi, and Mouna Rimani. "Cutaneous lupus tumidus: An unusual unilateral presentation." Our Dermatology Online 12, no. 2 (April 1, 2021): 151–52. http://dx.doi.org/10.7241/ourd.20212.10.

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Lupus tumidus is a rare subtype of chronic cutaneous lupus erythematosus characterized by erythema and bright urticarial erythematous and violaceous lesions on sun-exposed areas that heal without leaving scars. Lupus tumidus follows a benign and intermittent clinical course and is rarely associated with systemic lupus erythematosus. Treatment involves photoprotection, topical corticosteroids, and antimalarials. We report the case of a 42-year-old patient with an atypical unilateral form of lupus tumidus successfully treated with the administration of hydroxychloroquine in combination with photoprotection and tacrolimus.

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Kim, Esther K., Peter W. Hashim, and Saakshi Khattri. "Subacute Cutaneous Lupus Erythematosus Coexisting in a Patient with Plaque Psoriasis." Journal of Psoriasis and Psoriatic Arthritis 2, no. 2 (March 2017): 62–64. http://dx.doi.org/10.1177/247553031700200204.

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Subacute cutaneous lupus erythematosus (SCLE) is a form of cutaneous lupus erythematosus that is characterized by nonscarring, erythematous, annular eruptions occurring mainly on sun-exposed areas. The incomplete understanding of SCLE creates challenges in diagnosis and treatment. This report describes the rare case of SCLE co-existing with plaque psoriasis.

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Paul, Sujat. "Lupus With Pregnancy : Beyond the Basics." Journal of Chittagong Medical College Teachers' Association 23, no. 1 (September 22, 2012): 53–56. http://dx.doi.org/10.3329/jcmcta.v23i1.51898.

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Pregnancy in patients with systemic lupus erythematosus is associated with a high risk of maternal disease exacerbation and adverse fetal outcome. This review summarizes recent published findings on lupus pregnancy. Literature review: The literature has profound agreement on thefact that, for most women with inactive and stable systemic lups erythematousus, pregnancy is safe for both mother and fetus. The main risk factors for adverse pregnancy course and outcome are active disease, nephritis with proteinuria, hypertension and maternal serum antibodies to SS-A/Ro, SS-B/La, cardiolipin, 2-glycoprotein I, and lupus anticoagulant. Recent studies have broadened our understanding of the immunological mechanism underlying congenital heart block induced by anti-Ro/La antibodies. Pregnancy in patients with systemic lupus erythematosus is safe and manageable provided the disease is stable. Patients should be closely followed up before pregnancy for pregestational risk factors and should get extra attention during gregnancy. The disease can be safely managed in some cases of lupus flare during pregnancy. JCMCTA 2012; 23(1): 53-56

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N, Habib. "Review Article: Systemic Lupus Erythematosus." Open Access Journal of Microbiology & Biotechnology 5, no. 1 (2020): 1–4. http://dx.doi.org/10.23880/oajmb-16000158.

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Systemic lupus erythematosus or SLE is a persistent heterogeneous autoimmune disease that affects multisystem of the body. It is distinguished by acute and chronic inflammation of various tissues and even organs of the body principally the skin and joints. Systemic lupus erythematosus is a multisystem disorder and hence, it can affect any tissues, organs and even systems of the body. There are few categories of lupus for instance, lupus dermatitis or cutaneous lupus erythematosus (CLE) that affects the skin and causes malar rash, discoid lupus erythematosus (DLE) as well as systemic lupus erythematosus that causes damage to single or multiple internal organs. The damage is due to the inflammation that is caused by direct antibody reaction to the body tissues as well the deposition of immune complexes. Glucocorticoids, immunosuppressant, and anti- malarial are the combination therapy used to treat SLE besides providing counseling and awareness. Lupus erythematosus in any form particularly systemic lupus erythematosus (SLE) are prevalent in women compared to men with ratio of 6:1. It has the tendency to affect all ages but most frequently attacks women of aged 20 to 45 years old compared to men. On the other hand, if lupus erythematosus causes damage to internal organs either single or multiple, it is known as systemic lupus erythematosus. The damage is due to the inflammation that is caused by direct antibody reaction to the body tissues as well the deposition of immune complexes.

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Franjic, Sinisa. "Systemic Lupus Erythematosus in Gynecology." International Journal of Reproductive Research 1, no. 1 (December 22, 2022): 01–03. http://dx.doi.org/10.58489/2836-2225/005.

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Systemic lupus erythematosus is a chronic autoimmune disease that can affect various organs and parts of the body, especially the skin, joints, blood, kidneys, and central nervous system. Systemic lupus erythematosus is not a contagious disease, it is an autoimmune disease in which the immune system loses the ability to distinguish foreign from the patient's own tissues and cells. The immune system makes mistakes and produces, among other things, autoantibodies that recognize their own cells as foreign and attack them. The result is an autoimmune reaction that causes inflammation. Inflammation means that the affected part of the body becomes warm, red, swollen and sometimes painfully sensitive. If the signs of inflammation are long-lasting, as they may be in the case of systemic lupus erythematosus, tissue damage and its normal function may occur. Therefore, the goal of treatment of systemic lupus erythematosus is to alleviate inflammation. A number of hereditary risk factors along with various environmental factors are thought to be responsible for this impaired immune response. Systemic lupus erythematosus is known to be caused by a variety of factors, including hormonal imbalances during puberty, stress, and environmental factors such as sun exposure, viral infections, and medications.

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Dissertations / Theses on the topic "Lupus erythematosus"

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Meurer, Michael. "Childhood Discoid Lupus erythematosus and Antimalarials." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-135574.

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Teh, Lee-Suan. "Neuropsychiatric systemic lupus erythematosus." Thesis, University of Aberdeen, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.240703.

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Neuropsychiatric (NP) symptoms are relatively common in patients with SLE. The diverse and dramatic clinical presentations, the unclear pathogenesis, the lack of diagnostic test/s and uncertainties about the optimal management are some problems facing a clinician. When serum anti P antibodies were claimed to be highly correlated to lupus psychosis, this needed confirmation. An ELISA for measuring anti P antibodies was developed and validated. The prevalence of anti P antibodies was determined in different patient groups in a large retrospective study. Although anti P antibodies were highly specific for SLE, there was no correlation between the presence of these antibodies and lupus psychosis or other NP symptoms. Two prospective studies were carried out to eliminate any bias in our retrospective study. In one, none of the patients developed psychosis and these antibodies were not found to be specific for lupus depression or anxiety. In the other, anti P antibodies were measured in Malaysian Chinese SLE patients. No correlation was found between these antibodies and NP-SLE but a high prevalence of these antibodies was demonstrated in this group. Genetic studies showed that there was an increase in HLA-Dr2w16X subtype allele in anti P-positive patients but this did not reach significance. The usefulness of measuring antineuronal antibodies in helping to diagnose NP-SLE was examined but these antibodies were not better indicators of NP-SLE. Although the clinical correlations of anti P antibodies remain controversial, anti P antibodies were found to selectively bind to neuroblastoma cell surfaces in vitro but the nature of the surface antigen was not determined. Finally, sera from patients with lupus psychosis were found to significantly influence the response of neuroblastoma cells to agonist-induced stimulation and if confirmed, would offer an explanation for the reversible changes in cell function associated with psychiatric lupus.

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Meurer, Michael. "Childhood Discoid Lupus erythematosus and Antimalarials." Karger, 2003. https://tud.qucosa.de/id/qucosa%3A27661.

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Brown, Aaron Clifford. "Molecular and Phenotypic Characterization of the Y-Linked Autoimmune Accelerator (YAA)." Fogler Library, University of Maine, 2007. http://www.library.umaine.edu/theses/theses.asp?highlight=1&Cmd=abstract&ID=BMB2007-011.

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Bernatsky, Sasha. "Malignancy in systemic lupus erythematosus." Thesis, McGill University, 2001. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=32761.

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Objectives. (1) To estimate cancer incidence in systemic lupus erythematosus (SLE) as compared to the general population. (2) To estimate the sensitivity and specificity of methods of cancer ascertainment. (3) To determine the prevalence of malignancy risk factors in SLE. Methods. (1) We determined the incidence of malignancy in the Montreal General Hospital (MGH) lupus cohort, through linkage with the Quebec tumor registry. Standardized incidence ratios (SIRS) were generated, using Quebec population rates. In addition, a meta-analysis was performed by pooling data from eight cohort studies of malignancy in SLE. (2) We administered a postal survey to cohort members to determine risk factors for cancer and self-report of cancer occurrence. For dead or lost-to-follow-up patients, data was abstracted from charts. We calculated the sensitivity and specificity of self-report and chart review for cancer ascertainment, compared to registry linkage results. (3) Using the data collected on self-report and chart review, we compared risk factor prevalence within the MGH cohort to that of the Quebec population. Results. (1) Observed cancers in our cohort were greater than what would be expected; for all cancers, the SIR was 1.8 (95% Confidence Interval 1.2--2.6). The meta-analysis SIR (for all malignancies) was 1.67 (1.42--1.94). Postal survey and chart review methods demonstrated high specificity. Sensitivity was imperfect, but did not greatly effect estimation of the SIR estimate. (2) Our lupus cohort had a distinct profile of risk factors for malignancy compared to the general population; differences included more prevalent nulliparity, obesity, and use of hormone replacement therapy. Conclusions. The risk of malignancy in SLE patients is increased. Risk factor profiles could influence the incidence of certain malignancies in SLE.

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Clarke, Alexander James. "Autophagy in Systemic Lupus Erythematosus." Thesis, King's College London (University of London), 2015. http://kclpure.kcl.ac.uk/portal/en/theses/autophagy-in-systemic-lupus-erythematosus(1e5a4a5e-99f7-456e-bcb4-634a4e3fd986).html.

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Autophagy has emerged as a critical homeostatic mechanism in lymphocytes, influencing proliferation and differentiation. I sought to explore the role of autophagy in the pathogenesis of human and murine lupus, a disease in which B cells are critical effectors of pathology. Autophagy was assessed using multiple techniques in NZB/W and control mice, and in patients with SLE compared to healthy controls. I evaluated the phenotype of the B cell compartment in Vav-Atg7-/- mice in vivo, and examined human and murine plasmablast formation following inhibition of autophagy. I found activation of autophagy in early developmental stages of B cell development in a lupus mouse model even before disease-onset, and which progressively increased with age. In human disease, again autophagy was activated compared with healthy controls, principally in naïve B cells. B cells isolated from Vav-Atg7-/- mice failed to effectively differentiate into plasma cells following stimulation in vitro. Similarly, human B cells stimulated in the presence of autophagy inhibition did not differentiate into plasmablasts. My data suggest activation of autophagy is a mechanism for survival of autoreactive B cells, and also demonstrate that it is required for plasmablast differentiation, processes that induce significant cellular stress. The implication of autophagy in two major pathogenic pathways in SLE suggests the potential to use inhibition of autophagy as a novel treatment target.

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Rupanagudi, Khader Valli. "Immunoregulatory proteases in systemic lupus erythematosus and lupus nephritis." Diss., Ludwig-Maximilians-Universität München, 2013. http://nbn-resolving.de/urn:nbn:de:bvb:19-162652.

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Victorin, Sofia. "Livskvalitet vid systemisk lupus erythematosus (SLE)." Thesis, Mid Sweden University, Department of Health Sciences, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:miun:diva-260.

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Peschken, Christine A. "Systemic lupus erythematosus in Manitoba Aboriginals." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape9/PQDD_0019/MQ55086.pdf.

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Svenungsson, Elisabet. "Cardiovascular disease in systemic lupus erythematosus /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-501-8.

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Books on the topic "Lupus erythematosus"

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Schur, Peter H., and Elena M. Massarotti, eds. Lupus Erythematosus. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-1189-5.

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1949-, Wallace Daniel J., Hahn Bevra, and Dubois Edmund L, eds. Duboisʼ lupus erythematosus. 4th ed. Philadelphia: Lea & Febiger, 1993.

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Systemic lupus erythematosus. 5th ed. Amsterdam: Elsevier Academic Press, 2011.

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Eggleton, Paul, and Frank J. Ward, eds. Systemic Lupus Erythematosus. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0326-9.

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Miescher, Peter A., ed. Systemic Lupus Erythematosus. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79622-7.

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Smolen, Josef S., and Christoph C. Zielinski. Systemic Lupus Erythematosus. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71642-3.

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Kuhn, Annegret, Percy Lehmann, and Thomas Ruzicka, eds. Cutaneous Lupus Erythematosus. Berlin, Heidelberg: Springer Berlin Heidelberg, 2005. http://dx.doi.org/10.1007/b137847.

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1945-, Lahita Robert G., ed. Systemic lupus erythematosus. New York: Wiley, 1987.

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L, Dubois Edmund, and Wallace Daniel J. 1949-, eds. Dubois' Lupus erythematosus. 3rd ed. Philadelphia: Lea & Febiger, 1987.

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Systemic lupus erythematosus. Oxford: Clinical Pub., 2012.

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Book chapters on the topic "Lupus erythematosus"

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Dooley, Mary Anne. "Lupus Nephritis." In Lupus Erythematosus, 141–52. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-1189-5_11.

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Gloster, Hugh Morris, Lauren E. Gebauer, and Rachel L. Mistur. "Lupus Erythematosus." In Absolute Dermatology Review, 105–14. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-03218-4_33.

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Kuhn, Annegret, and Aysche Landmann. "Lupus Erythematosus." In European Handbook of Dermatological Treatments, 547–60. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-45139-7_55.

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Fry, Lionel, Fenella T. Wojnarowska, and Parvin Shahrad. "Lupus Erythematosus." In Illustrated Encyclopedia of Dermatology, 225–39. Dordrecht: Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-010-9390-3_25.

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Sönnichsen, N., H. D. Volk, F. Hiepe, and W. Jahn. "Lupus Erythematosus." In Dermatology in Five Continents, 218–21. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-83360-1_31.

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Crickx, B., and S. Belaich. "Lupus erythematosus." In European Handbook of Dermatological Treatments, 297–303. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-662-07131-1_54.

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Belmont, H. Michael, and Steven B. Abramson. "Lupus Erythematosus." In Molecular and Cellular Basis of Inflammation, 309–24. Totowa, NJ: Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-253-1_15.

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Crickx, B., and S. Belaich. "Lupus Erythematosus." In European Handbook of Dermatological Treatments, 333–39. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-662-03835-2_58.

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Sticherling, Michael, Donna M. Pellowski, and Richard D. Sontheimer. "Lupus Erythematosus." In Autoimmune Diseases of the Skin, 169–210. Vienna: Springer Vienna, 2001. http://dx.doi.org/10.1007/978-3-7091-3704-8_5.

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Sticherling, Michael, and Annegret Kuhn. "Lupus Erythematosus." In Braun-Falco´s Dermatology, 1–17. Berlin, Heidelberg: Springer Berlin Heidelberg, 2020. http://dx.doi.org/10.1007/978-3-662-58713-3_54-1.

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Conference papers on the topic "Lupus erythematosus"

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BEZERRA BRASILEIRO, ROMANO, PRISCILA GARCIA CÂMARA CABRAL TAVARES, FRANCISCO HELIALDO SOUSA DE OLIVEIRA FILHO, and PRISCILA DOURADO EVANGELISTA. "DRUG-INDUCED SYSTEMIC LUPUS ERYTHEMATOSUS." In SBR 2021 Congresso Brasileiro de Reumatologia. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2021.1795.

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DUARTE, ANNA BEATRIZ GOMES SOUZA, ELISA GUIMARÃES MOTTA, ADIB CHICRE MANSUR, GUILHERME SALLES DE ESCOBAR GONÇAVES, LARISSA BARROS DE OLIVEIRA, and GABRIELLEN VITIELLO. "FEVER IN SYSTEMIC LUPUS ERYTHEMATOSUS." In 36º Congresso Brasileiro de Reumatologia. São Paulo: Editora Blucher, 2019. http://dx.doi.org/10.5151/sbr2019-102.

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Egeli, Bugra, Asli Ece Soykut, and Serdal Ugurlu. "226 Comorbidity in systemic lupus erythematosus." In 13th International Congress on Systemic Lupus Erythematosus (LUPUS 2019), San Francisco, California, USA, April 5–8, 2019, Abstract Presentations. Lupus Foundation of America, 2019. http://dx.doi.org/10.1136/lupus-2019-lsm.226.

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FERRAÇO DALMASO, BARBARA, Ketty Lysie Libardi Lira Machado, Laura Gonçalves Aguiar, Heitor Filipe Surlo, Laís Pizzol Pasti, Paula dos Santos Athayde, Mariana de Oliveira Macabú, et al. "MULTIBACILAR LEPROSY MIMICKING SYSTEMIC LUPUS ERYTHEMATOSUS." In SBR 2021 Congresso Brasileiro de Reumatologia. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2021.2102.

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Barboza, Flavio, Tassia Caroline Beckert Viana, Eduarda Judith Dias Jacome Silva, Raquel Gerep Pereira, Bruna Luiza Oliveira Lima, Thalyne Aparecida Leite de Lima, Maitê Luise Zanette, et al. "Systemic Lupus Erythematosus mimicking Hodgkin's lymphoma." In Congresso Brasileiro de Reumatologia 2020. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2020.16937.

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Souza da Paz, Adriane, Gustavo Luiz Behrens Pinto, and Mittermayer Barreto Santiago. "COLLAPSING GLOMERULOPATHY IN SYSTEMIC LUPUS ERYTHEMATOSUS." In Congresso Brasileiro de Reumatologia 2020. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2020.17616.

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dos Santos Cavalcante, Anauá Fernanda, Sabrina Longo, Lucas P. G., Julia Goginski, Aline Neppel, Macleise A. Kemes, Thiago Alberto G. dos Santos, and Thelma L. Skare. "DRY EYE IN SYSTEMIC LUPUS ERYTHEMATOSUS." In Congresso Brasileiro de Reumatologia 2020. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2020.17297.

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Voulgari, PV, P. Katsimbri, Y. Alamanos, and AA Drosos. "FRI0115 Systemic lupus erythematosus in men." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.150.

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Moszkorzova, L., C. Dostal, J. Marek, Z. Lacinova, and L. Musilova. "FRI0137 Prolactin in systemic lupus erythematosus." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.172.

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Guma, M., A. Olive, J. Forcada, JC Duró, J. Roca, S. Holgado, E. Casado, and X. Tena. "FRI0116 Hypermobility in sistemic lupus erythematosus." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.151.

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Reports on the topic "Lupus erythematosus"

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Harley, John. Genome-Wide Association Study in African-Americans with Systemic Lupus Erythematosus. Fort Belvoir, VA: Defense Technical Information Center, September 2012. http://dx.doi.org/10.21236/ada574794.

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Harley, John. Genome-Wide Association Study in African-Americans with Systemic Lupus Erythematosus. Fort Belvoir, VA: Defense Technical Information Center, September 2013. http://dx.doi.org/10.21236/ada592038.

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Harley, John. Genome-Wide Association Study in African-Americans With Systemic Lupus Erythematosus. Fort Belvoir, VA: Defense Technical Information Center, September 2011. http://dx.doi.org/10.21236/ada554417.

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Xie, Juan, Dan Zhao, Lei Pan, Wenfang Xu, Beibei Wang, and Yan Yang. Experiences of Patients Living with Systemic Lupus Erythematosus: A Qualitative Meta-synthesis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, January 2022. http://dx.doi.org/10.37766/inplasy2022.1.0033.

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Pfeifer, Maria. Self-help Support Groups: Choices in Participation Among Women Facing Systemic Lupus Erythematosus (SLE). Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.6685.

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liu, zhihui, ruijuan cheng, and yi liu. Evaluation of anifrolumab safety in systemic lupus erythematosus: A meta-analysis and systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2022. http://dx.doi.org/10.37766/inplasy2022.3.0054.

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Gontar, I. P., O. A. Rusanova, O. I. Emelyanova, and O. V. Paramonova. CLINICLA FEATURES OF SYSTEMIC LUPUS ERYTHEMATOSUS ASSOCIATED WITH CHANGES IN ANTIBODIES TO THYROXIN AND TRIIODOTHYRONINE. Планета, 2018. http://dx.doi.org/10.18411/978-5-907109-24-7-2018-xxxv-96-100.

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Gontar, I. P., O. A. Rusanova, O. I. Emelyanova, S. S. Khortieva, and E. G. Cherkesova. AUTOIMMUNITY ISSUES IN PATIENTS WITH SYSTEMIC SCLERODERMA AND SYSTEMIC LUPUS ERYTHEMATOSUS WITH PREDOMINANT PULMONARY INVOLVEMENT. "PLANET", 2019. http://dx.doi.org/10.18411/978-5-907192-54-6-2019-xxxvi-73-81.

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Cai, Tiantian, Guofei Wang, and Jin-an Zhang. Hydroxychloroquine use reduces mortality risk in systemic lupus erythematosus: a meta-analysis of cohort studies. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2022. http://dx.doi.org/10.37766/inplasy2022.3.0157.

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Luo, Shuaihantian, Ying Zhou, and Guiying Zhang. Association between complement 4 copy number variation and systemic lupus erythematosus: a protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2020. http://dx.doi.org/10.37766/inplasy2020.8.0076.

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