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Relevant bibliographies by topics / Lymphocytes T CD4 mémoires

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Academic literature on the topic 'Lymphocytes T CD4 mémoires'

Author: Grafiati

Published: 4 June 2021

Last updated: 2 February 2022

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Journal articles on the topic "Lymphocytes T CD4 mémoires"

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Boukhedouni, Nesrine, Clément Jacquemin, Anne-Sophie Darrigade, et al. "Rôle des lymphocytes T effecteurs mémoires CD4+ et CD8+ CXCR3+ dans la perte du mélanocyte au cours du vitiligo." Annales de Dermatologie et de Vénéréologie 143, no. 12 (2016): S423. http://dx.doi.org/10.1016/j.annder.2016.09.059.

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Vocanson, M. "Lymphocytes T résidents mémoires dans les eczémas." Annales de Dermatologie et de Vénéréologie 147, no. 11 (2020): A2—A3. http://dx.doi.org/10.1016/j.annder.2020.08.002.

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Charerntantanakul, Wasin, and James A. Roth. "Biology of porcine T lymphocytes." Animal Health Research Reviews 7, no. 1-2 (2006): 81–96. http://dx.doi.org/10.1017/s1466252307001235.

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The present review concentrates on the biological aspects of porcine T lymphocytes. Their ontogeny, subpopulations, localization and trafficking, and responses to pathogens are reviewed. The development of porcine T cells begins in the liver during the first trimester of fetal life and continues in the thymus from the second trimester until after birth. Porcine T cells are divided into two lineages, based on their possession of the [@@@]\rmalpha [@@@]β or γδ T-cell receptor. Porcine [@@@]\rmalpha [@@@]β T cells recognize antigens in a major histocompatibility complex (MHC)-restricted manner, w

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Mendoza, Luis. "A Virtual Culture of CD4+ T Lymphocytes." Bulletin of Mathematical Biology 75, no. 6 (2013): 1012–29. http://dx.doi.org/10.1007/s11538-013-9814-9.

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Siri, Aurélie, Hubert de Boysson, and Guilaine Boursier. "Actualité sur les lymphocytes T régulateurs CD4+." médecine/sciences 28, no. 6-7 (2012): 646–51. http://dx.doi.org/10.1051/medsci/2012286019.

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Rudd, Christopher E. "A functional dichotomy in CD4+ T lymphocytes." Immunology Today 9, no. 12 (1988): 367–68. http://dx.doi.org/10.1016/0167-5699(88)91232-7.

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Bottomly, Kim. "A functional dichotomy in CD4+ T lymphocytes." Immunology Today 9, no. 9 (1988): 268–74. http://dx.doi.org/10.1016/0167-5699(88)91308-4.

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C.P. "Une augmentation de l'expression des cytokines du type Th2 par les lymphocytes T mémoire CD4+ et CD8+ est observée dans le sang, et est associée à une augmentation du taux des IgE sériques totales, mais non à la dermatite atopique." Revue Française d'Allergologie et d'Immunologie Clinique 40, no. 6 (2000): 660. http://dx.doi.org/10.1016/s0335-7457(00)80147-1.

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Kaur, Amitinder, Corrina L. Hale, Bradley Noren, Nadine Kassis, Meredith A. Simon, and R. Paul Johnson. "Decreased Frequency of Cytomegalovirus (CMV)-Specific CD4+ T Lymphocytes in Simian Immunodeficiency Virus-Infected Rhesus Macaques: Inverse Relationship with CMV Viremia." Journal of Virology 76, no. 8 (2002): 3646–58. http://dx.doi.org/10.1128/jvi.76.8.3646-3658.2002.

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ABSTRACT The frequency of cytomegalovirus (CMV)-specific CD4+ T lymphocytes was determined in CMV-seropositive rhesus macaques with or without simian immunodeficiency virus (SIV) infection by using the sensitive assays of intracellular cytokine staining and gamma interferon ELISPOT. Both techniques yielded 3- to 1,000-fold-higher frequencies of CMV-specific CD4+ T lymphocytes than traditional proliferative limiting dilution assays. The median frequency of CMV-specific CD4+ T lymphocytes in 23 CMV-seropositive SIV-negative macaques was 0.63% (range, 0.16 to 5.8%). The majority of CMV-specific C

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Ryu, Hoon, Chang Duk Jun, Bok Soo Lee, Byung Min Choi, Hyung Min Kim, and Hun-Taeg Chung. "Effect of Qigong Training on Proportions of T Lymphocyte Subsets in Human Peripheral Blood." American Journal of Chinese Medicine 23, no. 01 (1995): 27–36. http://dx.doi.org/10.1142/s0192415x95000055.

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The effect of Qigong training on proportions of T lymphocyte subsets was investigated in human peripheral blood. We observed that the ratio of CD4+/CD8+ T lymphocytes was increased as much as 50% in a trainee group who practiced Qigong training more than 5 months compared to a normal healthy group who did not practice. The absolute number of CD4+ T lymphocytes was also elevated in trainee group with 100 cells/mm 3 more than in normal healthy group. The positive correlation between the ratio of CD4+/CD8+ T lymphocytes and the ratio of CD4+45RA-/CD4+ CD45RA+ T lymphocytes was shown in the traine

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Dissertations / Theses on the topic "Lymphocytes T CD4 mémoires"

1

Gondois-Rey, Françoise. "Persistance du virus HIV dans les lymphocytes T CD4 mémoires quiescents." Aix-Marseille 2, 2004. http://www.theses.fr/2004AIX22030.

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Jaafoura, Salma. "Mémoire lymphocytaire T et persistance virale." Thesis, Paris 11, 2014. http://www.theses.fr/2014PA114847.

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Au cours d’une réponse immunitaire primaire, les lymphocytes T CD8 mémoires émergent à partir d'un environnement de forte activation immunitaire. Les cellules régulatrices T CD4 FoxP3+ (LTregs) jouent un rôle clé de suppression de la réponse immunitaire. Nous montrons que les LTregs sont nécessaires pour la génération d’une réponse mémoire T CD8 fonctionnelle. En absence de LTregs lors du priming, les LT CD8 mémoires générées prolifèrent faiblement et ne parviennent pas à se différencier après une réactivation antigénique en effecteurs cytotoxiques secondaires fonctionnelles. Nous suggérons qu

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Asrir, Assia. "Caractérisation phénotypique et fonctionnelle des différentes populations de Lymphocytes T CD4 Folliculaires Mémoires." Thesis, Toulouse 3, 2015. http://www.theses.fr/2015TOU30084/document.

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Les LT CD4 folliculaires (TFH) forment un lignage distinct de LT contrôlant spécifiquement les lymphocytes B (LB) et la mise en place de la mémoire B. Alors que ces cellules étaient considérées comme des cellules effectrices uniquement, récemment il a été identifié, chez l'Homme et la souris, l'existence de TFH mémoires. Les TFH mémoires en tant que LT CD4 mémoires sont nécessaires, en cas de nouvelle rencontre avec l'antigène (Ag), à la mise en place d'une réponse Anticorps (Ac) rapide, efficace et de forte affinité. En effet, leur présence est corrélée à la génération et le maintien à long t

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Veiga, Fernandez Henrique. "Caractérisation des propriétés des cellules T CD8 mémoires." Paris 5, 2002. http://www.theses.fr/2002PA05N127.

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Nous avons démontré que les cellulles T CD8 mémoires sont plus efficaces que les cellules nai͏̈ves après stimulation antigénique in vivo. Cette caractéristique unique des cellules mémoires est due à leurs propriétés de division et de différenciation en fonctions effectrices. Après stimulation antigénique, les cellules mémoires, comparées aux cellules naives se divisent plus tôt, ont un taux de division supérieur et un taux de perte réduit. Ces propriétés expliquent l'expansion trés importante des cellules mémoires après stimulation antigénique in vivo. Pour déterminer les mécanismes responsabl

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Moukambi, Félicien. "Études de la dynamique des cellules Tfh et T CD4 mémoires au cours de l'infection au VIH." Doctoral thesis, Université Laval, 2017. http://hdl.handle.net/20.500.11794/27740.

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Depuis sa découverte, le virus de l’immunodéficience humaine de type 1 (VIH-1) a causé la mort de 33 millions de personnes, et 36,7 millions sont actuellement infectées. Malgré l’existence des thérapies antirétrovirales, celles-ci ne conduisent pas à d’éradiquer le virus. En outre, il n’existe pas de vaccin. Les lymphocytes B, dont la fonction est de produire les anticorps sont dysfonctionnels au cours du VIH-1. Or la majorité des stratégies vaccinales se basent sur la production d’anticorps dépendante des cellules T CD4. Ainsi, la première partie de mon doctorat a été consacré à la compréhens

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Jacquemont, Lola. "Lymphocytes T CD8+ mémoires et devenir du greffon en transplantation." Thesis, Nantes, 2018. http://www.theses.fr/2018NANT1010/document.

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En transplantation rénale, malgré une amélioration de la prise en charge des rejets aigus, les patients restent exposés au risque de dysfonction chronique secondaire à la néphrotoxicité des immunosuppresseurs ainsi qu’à des phénomènes de rejet chronique cellulaire et/ou humoral. Identifier précocement les patients à haut risque de perte de greffon et plus particulièrement ceux présentant un sur risque immunologique demeure un défi. Les T CD8+ sont des acteurs majeurs de l’alloréactivité et une augmentation des T CD8+ mémoires de type TEMRA (CD45RA+CCR7-) est associée à un risque deux fois plus

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Grau, Morgan. "Identification de nouveaux biomarqueurs permettant la caractérisation des lymphocytes T CD8 mémoires innés." Thesis, Lyon, 2016. http://www.theses.fr/2016LYSE1023/document.

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Deux grandes classes de cellules composent le pool de lymphocytes T (LT) CD8 mémoires. D'une part, les LT CD8 mémoires conventionnels sont générés via la reconnaissance spécifique d'antigènes dérivés de pathogènes ou de tumeurs. D'autre part, les LT CD8 mémoires innés sont générés via différents mécanismes impliquant de fortes stimulations par des cytokines γc indépendamment de la reconnaissance d'antigènes du non soi. Le phénotype extrêmement similaire de ces deux populations cellulaires ne permet pas de les distinguer in vivo. En conséquence, la population de LT CD8 mémoires innés est relati

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Rapetti, Vachieri Laëtitia. "Les mécanismes de différenciation des lymphocytes T CD8 en cellules mémoires." Paris 5, 2008. http://www.theses.fr/2008PA05T004.

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Dans cette étude, nous avons montré la multiplicité de l'effet helper des cellules TCD4 sur les différents aspects des réponses CD8: prolifération, survie, fonctions effectrices et différenciation en cellule mémoire. Ces effets helper impliquent la signalisation CD40 ainsi que d'autres signaux. L'expression de CD40 par les CFA et les cellules TCD8 joue un rôle direct et complémentaire: l'expression de CD40 par les CFA est importante pour l'expansion des cellules TCD8; l'expression de CD40 par les cellules TCD8 est cruciale pour leur différenciation en cellule mémoire. En réponse secondaire, la

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Hani, Lylia. "Caractérisation et rôle des lymphocytes T CD4+ mémoires SAMHD1low au cours de l'infection par le VIH-1." Thesis, Paris Est, 2018. http://www.theses.fr/2018PESC0087/document.

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La mise en évidence du rôle de la molécule SAMHD1 dans l’infection par le VIH-1 en tant que facteur de restriction a ouvert de nouvelles perspectives dans la compréhension de la pathogénicité du virus.En effet, il a été clairement démontré que dans les cellules myéloïdes comme les monocytes/macrophages et les cellules dendritiques ainsi que les lymphocytes T CD4+ quiescents, SAMHD1 jouait un rôle important dans la protection de ces cellules de l’infection. En revanche, le rôle de cette molécule dans l’infection des lymphocytes activés, qui sont souvent la cible préférentielle du virus, n’est p

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Stubbe, Muriel. "Lymphocytes T CD4 et réponses vaccinales: du processus de différenciation à la mémoire immunologique." Doctoral thesis, Universite Libre de Bruxelles, 2007. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/210593.

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Les lymphocytes T CD4 (LT CD4) jouent un rôle central dans la régulation des réponses immunitaires vis-à-vis des agents infectieux et des vaccins. Cependant, leur différenciation in vivo est encore mal comprise et les caractéristiques des LT CD4 capables de persister à long terme tout en assurant une réponse immunitaire protectrice sont mal définies. L’approfondissement de ces connaissances est indispensable pour le développement de nouveaux vaccins. <p>Pour approcher cette question, nous avons utilisé deux approches expérimentales. La première est un suivi de la différenciation des LT CD4 au

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Books on the topic "Lymphocytes T CD4 mémoires"

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Na, Songqing, and Chandrasekar Venkataraman Iyer. Effector CD4+ T cells in health and disease 2007. Transworld Research Network, 2007.

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2

Rahelu, Manjit. Characterisation of human CD4[superior plus] cytolytic T lymphocytes, with special reference to mycobacterial antigens. University of Birmingham, 1992.

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Littman, D. R. Cd4 Molecule: Roles in T Lymphocytes & in HIV Disease. Edited by D. R. Littman. SPRINGER-VERLAG, 1996.

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Gravelle, Micheline Louise *. The targeting of CD4 r T lymphocytes to a B cell lymphoma: acomparison of two in vitro immunotherapeutic strategies. 1989.

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(Editor), B. Kyewski, and Elisabeth Suri-Payer (Editor), eds. CD4+CD25+ Regulatory T Cells: Origin, Function and Therapeutic Potential (Current Topics in Microbiology and Immunology). Springer, 2005.

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National AIDS Control Organization (India), ed. National guidelines for the enumeration of CD4⁺ T-Lymphocytes with single platform technology for initiation and monitoring of art in HIV infected individuals. National Aids Control Organisation, Ministry of Health and Family Welfare, 2007.

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National AIDS Control Organization (India), ed. National guidelines for the enumeration of CD4⁺ T-Lymphocytes with single platform technology for initiation and monitoring of art in HIV infected individuals. National Aids Control Organisation, Ministry of Health and Family Welfare, 2007.

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National guidelines for the enumeration of CD4+ T-Lymphocytes with single platform technology for initiation and monitoring of art in HIV infected individuals. National Aids Control Organisation, 2007.

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Hartigan-O’Connor, Dennis J., and Christian Brander. Immunology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0005.

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The key factor in HIV pathogenesis is the decline in CD4+ T cells with resultant immunodeficiency and chronic inflammation. Depletion of CD4+ T cells from the gastrointestinal mucosa followed by microbial translocation and subsequent immune activation are components of disease progression in untreated patients. Symptomatic and occult opportunistic infections including cytomegalovirus contribute to chronic inflammation in persons infected with HIV. Antiretroviral therapy (ART) results in immune reconstitution, with increases in peripheral CD4+ T cell lymphocytes in most persons infected with HI

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Piccio, Laura, and Anne H. Cross. Immunology of Multiple Sclerosis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199341016.003.0004.

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Multiple sclerosis (MS) is considered to be an autoimmune disease of the central nervous system that targets myelin but affects both white matter and gray matter. Multiple sclerosis is thought to be mediated by cells of the adaptive and innate immune systems. CD4+ T lymphocytes of the Th1 and Th17 subtypes are believed to be critical for the initiation of multiple sclerosis. Treatment with monoclonal antibodies that deplete B lymphocytes has proven that B cells are critical to relapse development in multiple sclerosis. While immunopathophysiology is clearly important in MS, whether multiple sc

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Book chapters on the topic "Lymphocytes T CD4 mémoires"

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Abbas, Abul K., Victor L. Perez, Luk van Parijs, and Richard C. K. Wong. "Differentiation and Tolerance of CD4+ T Lymphocytes." In Novartis Foundation Symposia. John Wiley & Sons, Ltd., 2007. http://dx.doi.org/10.1002/9780470514849.ch2.

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Tanaka, Hirokazu, and Ichiro Taniuchi. "The CD4/CD8 Lineages: Central Decisions and Peripheral Modifications for T Lymphocytes." In Thymic Development and Selection of T Lymphocytes. Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/82_2013_323.

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Withers-Ward, Elizabeth S., and Irvin S. Y. Chen. "Gene Therapy in CD4+ T Lymphocytes in SCID-hu Mice." In The DNA Provirus. ASM Press, 2014. http://dx.doi.org/10.1128/9781555818302.ch19.

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Flaherty, Stephanie, and Joseph M. Reynolds. "TLR Function in Murine CD4+ T Lymphocytes and Their Role in Inflammation." In Methods in Molecular Biology. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3335-8_14.

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Pinchuk, Lesya M., Patricia S. Polacino, Michael B. Agy, Stephen J. Klaus, and Edward A. Clark. "Cell-Cell Interactions Regulate Dendritic Cell-Dependent HIV-1 Production in CD4+ T Lymphocytes." In Advances in Experimental Medicine and Biology. Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-1971-3_103.

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Arbour, Nathalie, and Alexandre Prat. "Roles of CD4 and CD8 T Lymphocytes in Multiple Sclerosis and Experimental Autoimmune Encephalomyelitis." In Neuroinflammation. John Wiley & Sons, Inc, 2015. http://dx.doi.org/10.1002/9781118732748.ch3.

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Fowlkes, B. J., D. Pardoll, T. Lantz, and F. Ramsdell. "Participation of CD4 and CD8 Accessory Molecules in the Development and Selection of T Lymphocytes." In Progress in Immunology. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-83755-5_38.

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Dumitriu, Ingrid E., and Juan Carlos Kaski. "The Role of Lymphocytes in the Pathogenesis of Atherosclerosis: Focus on CD4+ T Cell Subsets." In Inflammatory Response in Cardiovascular Surgery. Springer London, 2013. http://dx.doi.org/10.1007/978-1-4471-4429-8_2.

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Horgan, Kevin J., Yoshiya Tanaka, and Stephen Shaw. "Postthymic Differentiation of CD4 T Lymphocytes: Naive Versus Memory Subsets and Further Specialization among Memory Cells (Part 1 of 2)." In Regulation and Functional Significance of T-Cell Subsets. KARGER, 1992. http://dx.doi.org/10.1159/000319115.

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Horgan, Kevin J., Yoshiya Tanaka, and Stephen Shaw. "Postthymic Differentiation of CD4 T Lymphocytes: Naive Versus Memory Subsets and Further Specialization among Memory Cells (Part 2 of 2)." In Regulation and Functional Significance of T-Cell Subsets. KARGER, 1992. http://dx.doi.org/10.1159/000319116.

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Conference papers on the topic "Lymphocytes T CD4 mémoires"

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Lo Re, Sandra, Marylène Lecocq, Francine Uwambayinema, et al. "PDGF-Producing CD4+ Foxp3+ Regulatory T Lymphocytes Promote Lung Fibrosis." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a5552.

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Jiang, W., H. Hong, R. Juskevicius, et al. "Study of 3D Structural Differences between CD4+ and CD8+ T lymphocytes." In Biomedical Optics. OSA, 2014. http://dx.doi.org/10.1364/biomed.2014.bs3a.78.

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RODRIGUES COSTA, TABATA, Alessandro Farias, Carolina Francelin Rovarotto, et al. "Analysis of CD4+RUNX3+ T lymphocytes during clinical course of experimental autoimmune encephalomyelitis." In XXIV Congresso de Iniciação Científica da UNICAMP - 2016. Galoa, 2016. http://dx.doi.org/10.19146/pibic-2016-51128.

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Fogel, O., A. Bugge Tingaard, M. Fagny, et al. "FRI0165 Micrornas dysregulation in monocytes and t cd4 lymphocytes from patients with axial spondyloarthritis." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.7073.

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Gohring, John T., and Xudong Fan. "Detection of CD4+ and CD8 + T-lymphocytes with the optofluidic ring resonator (OFRR) biosensor." In SPIE Defense, Security, and Sensing, edited by Hai Xiao, Xudong Fan, and Anbo Wang. SPIE, 2009. http://dx.doi.org/10.1117/12.818255.

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Guo, Zhijun, David Owen, Pamela Rosato, David Masopust, Michael A. Farrar, and David A. Potter. "Abstract 3525: N1-hexyl-N5-benzyl-biguanide promotes proliferation of CD4+ and CD8+ T lymphocytes." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-3525.

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Prince Poveda, Nathalie, Alessandro Farias, Vinícius de Boldrini, Raphael Patrício Silva Quintiliano, Verônica Almeida de Paula Galdino da Silva, and Leonilda Maria Barbosa dos Santos. "Study of cytotoxic activity by T CD4+ lymphocytes in patients with Secondary Progressive Multiple Sclerosis." In XXV Congresso de Iniciação Cientifica da Unicamp. Galoa, 2017. http://dx.doi.org/10.19146/pibic-2017-78013.

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Zhang, Yaqing, Wei Yan, Wei Yan, et al. "Abstract B23: CD4+ T lymphocytes promote pancreatic cancer progression by suppressing anti-tumor immune responses." In Abstracts: AACR Special Conference on RAS Oncogenes: From Biology to Therapy; February 24-27, 2014; Lake Buena Vista, FL. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1557-3125.rasonc14-b23.

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Amara, Suneetha, Pooja Yenuga, Roy Zent, and Venkataswarup Tiriveedhi. "Abstract B45: NFAT5/TonEBP mediated anti-tumor efficiency of high salt activated CD4+T lymphocytes." In Abstracts: AACR Special Conference on Tumor Immunology and Immunotherapy; November 27-30, 2018; Miami Beach, FL. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/2326-6074.tumimm18-b45.

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Shellito, Judd E., and Sanbao Ruan. "Treatment With IL-7 Restores Host Defense Against Pneumocystis In Mice Depleted Of CD4+ T-Lymphocytes." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a2499.

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Reports on the topic "Lymphocytes T CD4 mémoires"

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Mekova, Ralitsa V., Spaska S. Lesichkova, Adelina D. Tsakova, Julieta Z. Bakalova, Deniz Bakalov, and Mihail Boyanov. Circulating CD3(+)CD4(+)CD28(‒) T Lymphocytes in Patients with Autoimmune Thyroiditis. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, 2020. http://dx.doi.org/10.7546/crabs.2020.05.14.

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Mastro, Andrea M. The Use of Exercise to Increase CD4 (+) T Lymphocytes Following Chemotheraphy Treatment for Breast Cancer. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada398031.

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