Relevant bibliographies by topics / Lymphoid blast

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Lymphoid blast'

Author: Grafiati
Published: 5 June 2025
Last updated: 2 August 2025

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Journal articles on the topic "Lymphoid blast"

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Ha, K., MH Freedman, A. Hrincu, D. Petsche, A. Poon, and EW Gelfand. "Separation of lymphoid and myeloid blasts in the mixed blast crisis of chronic myelogenous leukemia: no evidence for Ig gene rearrangement in CALLA-positive blasts." Blood 66, no. 6 (1985): 1404–8. http://dx.doi.org/10.1182/blood.v66.6.1404.1404.

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Abstract Recent studies suggest that lymphoid blast crisis cells of chronic myelogenous leukemia (CML) expressing the common acute lymphoblastic leukemia antigen (CALLA) are B precursor cells, based on the demonstration of immunoglobulin (Ig) gene rearrangement similar to common acute lymphocytic leukemia. There is little evidence to suggest whether the cells with similar lymphoid characteristics in the mixed blast crisis of CML are also committed to B cell lineage. A patient in “mixed” blast crisis of CML was studied. On the basis of morphology, cytochemistry, and immunological studies, the b
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Ha, K., MH Freedman, A. Hrincu, D. Petsche, A. Poon, and EW Gelfand. "Separation of lymphoid and myeloid blasts in the mixed blast crisis of chronic myelogenous leukemia: no evidence for Ig gene rearrangement in CALLA-positive blasts." Blood 66, no. 6 (1985): 1404–8. http://dx.doi.org/10.1182/blood.v66.6.1404.bloodjournal6661404.

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Recent studies suggest that lymphoid blast crisis cells of chronic myelogenous leukemia (CML) expressing the common acute lymphoblastic leukemia antigen (CALLA) are B precursor cells, based on the demonstration of immunoglobulin (Ig) gene rearrangement similar to common acute lymphocytic leukemia. There is little evidence to suggest whether the cells with similar lymphoid characteristics in the mixed blast crisis of CML are also committed to B cell lineage. A patient in “mixed” blast crisis of CML was studied. On the basis of morphology, cytochemistry, and immunological studies, the blasts wer
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Maslak, P. "Lymphoid Blast." ASH Image Bank 2004, no. 0630 (2004): 101139. http://dx.doi.org/10.1182/ashimagebank-2004-101139.

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Puigví, Laura, Anna Merino, Santiago Alférez, Andrea Acevedo, and José Rodellar. "New quantitative features for the morphological differentiation of abnormal lymphoid cell images from peripheral blood." Journal of Clinical Pathology 70, no. 12 (2017): 1038–48. http://dx.doi.org/10.1136/jclinpath-2017-204389.

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AimsThis work aims to propose a set of quantitative features through digital image analysis for significant morphological qualitative features of different cells for an objective discrimination among reactive, abnormal and blast lymphoid cells.MethodsAbnormal lymphoid cells circulating in peripheral blood in chronic lymphocytic leukaemia, B-prolymphocytic leukaemia, hairy cell leukaemia, splenic marginal zone lymphoma, mantle cell lymphoma, follicular lymphoma, T-prolymphocytic leukaemia, T large granular lymphocytic leukaemia and Sézary syndrome, normal, reactive and blast lymphoid cells were
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Mansurov, Alay, Michael Byrne, and Koyamangalath Krishnan. "Successful Salvage with Inotuzumab Ozogamicin in Relapsed/Refractory Lymphoid Blast Crisis of Chronic Myeloid Leukemia after Failure of Multiple Lines of Therapy Including Blinatumomab." Blood 132, Supplement 1 (2018): 5452. http://dx.doi.org/10.1182/blood-2018-99-114170.

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Abstract Background: Lymphoid blast crisis, one of the two major forms of chronic myeloid leukemia (CML) blast crisis (BC), is comprised of lymphoblasts and occurs in about 30% of BC patients. These individuals often respond to the same treatment strategies used in Philadelphia chromosome positive acute lymphoblastic leukemia (Ph+ ALL). Blinatumomab, a bispecific T engager, or BiTE, is an antibody with dual specificity against both CD3 and CD19, and is approved for use in relapsed/refractory ALL. We present the first known case of relapsed/refractory lymphoid blast phase CML, including treatme
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Padhi, Parikshit, Margarita Topalovski, Radwa El Behery, Eduardo S. Cantu, and Ramadevi Medavarapu. "A Rare Case of Chronic Myelogenous Leukemia Presenting as T-Cell Lymphoblastic Crisis." Case Reports in Oncological Medicine 2018 (November 18, 2018): 1–4. http://dx.doi.org/10.1155/2018/7276128.

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Chronic Myelogenous Leukemia in blast crisis can manifest as either myeloid (more common) or lymphoid blast crisis. Most lymphoblastic crises are of B-cell lineage. T-cell blast crisis is extremely rare, with only a few reported cases. We present a case of a middle-aged man who was diagnosed with CML on peripheral blood and bone marrow biopsy. Because of a generalized lymphadenopathy noted at the time of diagnosis, a lymph node biopsy was also performed, which revealed a T-cell lymphoblastic leukemia/lymphoma, BCR/ABL1 positive, with clonal evolution. This is a very rare manifestation of CML i
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Pui, CH, FG Behm, DK Kalwinsky, et al. "Clinical significance of low levels of myeloperoxidase positivity in childhood acute nonlymphoblastic leukemia." Blood 70, no. 1 (1987): 51–54. http://dx.doi.org/10.1182/blood.v70.1.51.51.

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Abstract The clinical significance of a low percentage of myeloperoxidase- positive blast cells in childhood acute nonlymphoblastic leukemia was determined. Of 155 consecutive cases studied by cytochemical staining methods, 14 were characterized by 4% to 15% (median 6%) myeloperoxidase- positive blasts. All 14 cases showed reactivity to Sudan black B stain, and 7 had Auer rods. The morphological subtypes of leukemia were M1 (8 cases), M2 (3), M4 (1), and M5 (2). Immunological marker studies disclosed the lymphoid-associated T11 antigen on cells from 8 of the 11 cases tested. Other lymphoid-rel
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Pui, CH, FG Behm, DK Kalwinsky, et al. "Clinical significance of low levels of myeloperoxidase positivity in childhood acute nonlymphoblastic leukemia." Blood 70, no. 1 (1987): 51–54. http://dx.doi.org/10.1182/blood.v70.1.51.bloodjournal70151.

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The clinical significance of a low percentage of myeloperoxidase- positive blast cells in childhood acute nonlymphoblastic leukemia was determined. Of 155 consecutive cases studied by cytochemical staining methods, 14 were characterized by 4% to 15% (median 6%) myeloperoxidase- positive blasts. All 14 cases showed reactivity to Sudan black B stain, and 7 had Auer rods. The morphological subtypes of leukemia were M1 (8 cases), M2 (3), M4 (1), and M5 (2). Immunological marker studies disclosed the lymphoid-associated T11 antigen on cells from 8 of the 11 cases tested. Other lymphoid-related find
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Kim, Miyoung, Dae Young Zang, Jiwon Lee, Ji-Young Park, Yousun Chung, and Young Kyung Lee. "Mixed Phenotype Acute Leukemia that Evolved from Myelodysplastic Syndrome with Excess Blasts." Laboratory Medicine 51, no. 3 (2019): 288–95. http://dx.doi.org/10.1093/labmed/lmz054.

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Abstract Myelodysplastic syndrome (MDS) that evolves into acute leukemia with blasts of mixed phenotypes has rarely been reported and has no distinct diagnostic category. Herein, we describe a 79-year-old Korean female patient with MDS–excess blasts (MDS-EB) that evolved into acute leukemia; the blasts simultaneously expressed B-lymphoid and myeloid antigens. The patient was diagnosed with MDS-EB with blasts of myeloid lineage coexpressing a few B-lymphoid antigens with 7q and 20q abnormalities. The disease progressed to acute leukemia with blasts carrying more B-lymphoid antigens, which was i
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Estrov, Z., A. Tawa, XH Wang, et al. "In vitro and in vivo effects of deferoxamine in neonatal acute leukemia." Blood 69, no. 3 (1987): 757–61. http://dx.doi.org/10.1182/blood.v69.3.757.bloodjournal693757.

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A six week old infant with acute leukemia failed to attain remission with chemotherapy. Because we previously demonstrated that the iron chelator deferoxamine (DFO) has antiproliferative properties and modulatory effects on cell differentiation, a protocol was designed for in vitro study and for clinical use in the patient. At diagnosis, blast cells were morphologically undifferentiated, had nondiagnostic cytochemistry, showed an abnormal karyotype (t[4;11]), expressed markers of B cell lineage, and demonstrated C mu gene rearrangement. Tissue culture of marrow or blood cells yielded colonies
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Dissertations / Theses on the topic "Lymphoid blast"

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Abrahamsson, Elina. "Utvärdering av felmeddelande i eMM Software Version 00-06 till SysmexXE-5000." Thesis, Malmö högskola, Fakulteten för hälsa och samhälle (HS), 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:mau:diva-25564.

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Sysmex XE-5000 är en automatiserad cellräknare som utför mätningar enligt olikamätprinciper, de två som tillämpats i projektet är RF/DC(Radio Frequency/DirectCurrent) samt Flödescytometri med halvledarlaser. RF/DC bygger på förändringari radiofrekventa resistansen och likspänningsresistansen. Förändringar i denradiofrekventa resistansen (RF) ger information om densiteten i cellernas inre(exempelvis kärnans storlek) och förändringar i likspänningsresistensen (DC) gerinformation om blodcellernas storlek. Flödescytometri definierar ett mått påcellers fysiologiska och kemiska egenskaper. Detektion
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Ndikung, Bonaventure Soh Bejeng [Verfasser]. "Lymphoid specific gene rearrangement and mutation mechanisms in chronic myeloid leukemia blast crisis / vorgelegt von Bonaventure Soh Bejeng Ndikung." 2006. http://d-nb.info/982963629/34.

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Books on the topic "Lymphoid blast"

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Collins, Graham, and Chris Bunch. Acute leukaemia. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0286.

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Acute leukaemias are rapidly progressive, clonal haematopoietic stem cell disorders resulting in the accumulation of immature blood cell precursors (known as blasts) in the bone marrow. There are two main types, defined by the presence of myeloid lineage or lymphoid markers on the blast cells: acute myeloid leukaemia and acute lymphoblastic leukaemia. This chapter addresses the causes, diagnosis, and management of the acute leukaemias.
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Book chapters on the topic "Lymphoid blast"

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Miljković, Ana K., Katarina Isaković, Tatjana Matejić, and Branka Stojanović. "Ia+T10+ Blast Cells in Acute Lymphoblastic Leukaemia are Precursors of T+ and B+ Cells." In Microenvironments in the Lymphoid System. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4613-2463-8_123.

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Pallis, M., J. Turzanski, S. Langabeer, and N. H. Russell. "Reproducible Flow Cytometric Methodology for Measuring Multidrug Resistance in Leukaemic Blasts." In Drug Resistance in Leukemia and Lymphoma III. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4811-9_10.

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Frost, Britt-Marie, Rolf Larsson, Peter Nygren, and Gudmar Lönnerholm. "Is In Vitro Sensitivity of Blast Cells Correlated to Therapeutic Effect in Childhood Acute Lymphoblastic Leukemia?" In Drug Resistance in Leukemia and Lymphoma III. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4811-9_45.

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Sargent, J. M., A. W. Elgie, C. J. Williamson, and C. G. Taylor. "Aphidicolin Markedly Increases the In Vitro Sensitivity to ARA-C of Blast Cells From Patients with AML." In Drug Resistance in Leukemia and Lymphoma III. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4811-9_62.

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Baryshnikov, A. Yu, E. R. Polosukhina, N. N. Tupitsin, et al. "CD95 (FAS/APO-1) Antigen is a New Prognostic Marker of Blast Cells of Acute Lymphoblastic Leukaemia Patients." In Drug Resistance in Leukemia and Lymphoma III. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4811-9_27.

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Litvinova, E., K. Gurova, K. Chimishkian, and G. Mentkevich. "Effects of CSFS and their Combinations with Chemotherapeutic Agents (CH) on Leukemic Blasts (LB) in Children (MTT-Assay)." In Drug Resistance in Leukemia and Lymphoma III. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4811-9_65.

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Anilkumar, K. K., V. J. Manoj, and T. M. Sagi. "Automated Segregation of Lymphoid and Myeloid Blasts in Acute Leukemia Cases Using a Deep Convolutional Neural Network." In Applied Intelligence for Medical Image Analysis. Apple Academic Press, 2024. http://dx.doi.org/10.1201/9781003461852-10.

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Look, A. Thomas. "Aberrant Control of Cell Fate in the Leukemias and Lymphomas." In Hematopoiesis. Oxford University PressNew York, NY, 2001. http://dx.doi.org/10.1093/oso/9780195124507.003.0057.

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Abstract In the human acute leukemias and lymphomas, the normal sequence of biochemical events contributing to lymphoid or myeloid cell development is interrupted by genetic lesions that can lead to arrested differentiation, dysregulated proliferation, or aberrant survival, followed by clonal expansion of the affected cells. Cytogenetic analysis has revealed that blast cells from patients with leukemia or lymphoma almost invariably harbor clonal chromosomal abnormalities.
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Reiter, Andreas, and Nicholas C. P. Cross. "Eosinophilia-associated myeloproliferative neoplasms." In Oxford Specialist Handbook: Myeloproliferative Neoplasms. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198744214.003.0013.

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Accurate diagnosis of eosinophilia-associated disorders remains problematic. The World Health Organization (WHO) 2008 classification defines a rare subgroup: myeloid and lymphoid neoplasms with eosinophilia and abnormalities of PDGFRA, PDGFRB, or FGFR1 (MLN-eo), of which by far the most common is FIP1L1-PDGFRA. It is likely that other tyrosine kinase (TK) fusions will be incorporated into this category in due course. For other cases, the finding of increased numbers of blasts and/or proof of clonality is the basis for chronic eosinophilic leukaemia, not otherwise specified (CEL-NOS); however,
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Vormoor, H. Josef, Tobias F. Menne, and Anthony V. Moorman. "Acute lymphoblastic leukaemia." In Oxford Textbook of Medicine, edited by Chris Hatton and Deborah Hay. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0523.

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Acute lymphoblastic leukaemia (ALL) is a malignant proliferation of lymphoid blasts, most commonly of B-lineage origin. The clinical symptoms and signs are either a consequence of bone marrow failure (infections, bruising, petechiae, pallor, and tiredness) or a consequence of the uncontrolled proliferation of the blasts (lymphadenopathy, hepatosplenomegaly, and cranial nerve palsies). Its peak incidence is in young children but ALL occurs at all ages. More than 80% of all affected children are cured with modern chemotherapy, but unfortunately the outcome of adults is much worse despite some im
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Conference papers on the topic "Lymphoid blast"

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Falang, A., G. M. Alessio, M. Donati, and T. A. Barbui. "DISSEMINATED INTRAVASCULAR COAGULATION (DIC) AND ACUTE LEUKEMIA:IDENTIFICATION OF A NEW CELLULAR PROCOAGULANT." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643661.

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There is an enhanced incidence (>50%) of severe coagulopathy in association with several types of acute leukemias. Cell associated procoagulants are considered important in this context. So far only a Tissue Factor (TF)-type procoagulant has been described in leukemic cells. We have set up here the experimentalconditions to identify other possible cellular procoagulants in leukemia. We have tested blast cell extracts from 21 patients with 5 different cytological subtypes (from Ml to M5 of acute non lymphoid leukemia (ANLL), according to theFAB classification, in order to assay whether they
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Reports on the topic "Lymphoid blast"

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Jordan, Craig T. Properties of Leukemia Stem Cells in a Novel Model of Cml Progression to Lymphoid Blast Crisis. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada484102.

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Jordan, Craig T. Properties of Leukemia Stem Cells in a Novel Model of CML Progression to Lymphoid Blast Crisis. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada471560.

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