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Relevant bibliographies by topics / Lympholytes T

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Academic literature on the topic 'Lympholytes T'

Author: Grafiati

Published: 4 June 2021

Last updated: 8 February 2022

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Journal articles on the topic "Lympholytes T"

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Morris, Emma C., Peppy Rebello, Kirsty J. Thomson, et al. "Pharmacokinetics of alemtuzumab used for in vivo and in vitro T-cell depletion in allogeneic transplantations: relevance for early adoptive immunotherapy and infectious complications." Blood 102, no. 1 (2003): 404–6. http://dx.doi.org/10.1182/blood-2002-09-2687.

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Abstract Persistence of alemtuzumab at lympholytic concentrations after reduced-intensity conditioning allogeneic stem cell transplantations (RITs) could impair immune reconstitution and reduce donor T-cell–mediated graft-versus-leukemia/lymphoma (GVL) effects, derived from the graft or subsequent adoptive immunotherapy. We have studied the pharmacokinetics of alemtuzumab in 2 different groups: RIT (100 mg alemtuzumab in vivo over 5 days) and myeloablative allografts (20 mg alemtuzumab added in vitro to the stem cells prior to return). Alemtuzumab concentrations in RIT patients were in excess

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Schirren, CA, H. Volpel, and SC Meuer. "Adhesion molecules on freshly recovered T leukemias promote tumor- directed lympholysis." Blood 79, no. 1 (1992): 138–43. http://dx.doi.org/10.1182/blood.v79.1.138.138.

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Abstract Besides facilitating cell to cell adhesion, the molecular interactions between CD2 and its ligand CD58 (lymphocyte function-associated antigen- 3 [LFA-3]), as well as between CD11a/18 (LFA-1) and CD54 (intercellular adhesion molecule-1) have recently been recognized to participate in lymphocyte activation, recirculation, and effector function, including cytolytic activity towards tumor cells. We have investigated the role of CD2/CD58 and CD11a/18/CD54 interactions in cellular immune responses directed towards freshly recovered human T-cell leukemias. The data support the notion that d

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Schirren, CA, H. Volpel, and SC Meuer. "Adhesion molecules on freshly recovered T leukemias promote tumor- directed lympholysis." Blood 79, no. 1 (1992): 138–43. http://dx.doi.org/10.1182/blood.v79.1.138.bloodjournal791138.

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Besides facilitating cell to cell adhesion, the molecular interactions between CD2 and its ligand CD58 (lymphocyte function-associated antigen- 3 [LFA-3]), as well as between CD11a/18 (LFA-1) and CD54 (intercellular adhesion molecule-1) have recently been recognized to participate in lymphocyte activation, recirculation, and effector function, including cytolytic activity towards tumor cells. We have investigated the role of CD2/CD58 and CD11a/18/CD54 interactions in cellular immune responses directed towards freshly recovered human T-cell leukemias. The data support the notion that downregula

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4

Kristensen, Tom. "Human Histocompatibility Testing by T Cell-Mediated Lympholysis: A European Standard CML Technique." Tissue Antigens 16, no. 5 (2008): 335–67. http://dx.doi.org/10.1111/j.1399-0039.1980.tb00316.x.

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5

Owens, William J., Thomas C. Cesario, and Edward Shanbrom. "Selective Lympholysis: A Unique Method Using the Double-Dye Technique." Blood 108, no. 11 (2006): 3884. http://dx.doi.org/10.1182/blood.v108.11.3884.3884.

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Abstract Many methods are utilized to destroy mononuclear cells (primarily lymphocytes) either for neoplasm or immunosuppression. Neither radiation nor chemotherapy are truly selective or completely successful. The concept of “Double-Dye” treatment of blood for transfusion has been developed in order to inactivate parasites, bacteria and viruses (J Thromb Haemost2003; 1 Supplement 1 July: P1114). In recent studies, it has been observed that this same “Double-Dye” concept presents the possibility of very selectively eliminating lymphocytes (mononuclear cells) without affecting neutrophils in wh

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and, Adriana Zeevi, and Rene J. Duquesnoy. "SPECIFICITY OF ALLOACTIVATED HUMAN T LYMPHOCYTE CLONES IN SECONDARY PROLIFERATION, CELL-MEDIATED LYMPHOLYSIS AND INTERLEUKIN-2 RELEASE." European Journal of Immunogenetics 12, no. 1 (1985): 17–31. http://dx.doi.org/10.1111/j.1744-313x.1985.tb00826.x.

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7

Vasilyeva, Vera, Elena N. Parovichnikova, Larisa A. Kuzmina, et al. "Reconstitution of Memory T -Cells after Allogeneic Stem Cell Transplantation in Patients with Post-Transplantation Cyclophosphamide As Tolerance Induction." Blood 126, no. 23 (2015): 5475. http://dx.doi.org/10.1182/blood.v126.23.5475.5475.

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Abstract Introduction. Nowadays high-dose post-transplantation cyclophosphamide (CY) replace standard immunosuppression (IST). Thereby, the investigation of T-cells reconstitution after post-transplant-CY doesn't reach appropriate level, and probably it's very different from what we see after standard IST. We studied the reconstitution of memory T-cells on day of engraftment (WBC>1000 cells\us) after allogenic hematopoietic stem cell transplantation (allo-HSCT) with post-transplant-CY and standard immunosuppression therapy. Patients and methods. During 2 years, 29 patients with different he

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Klingemann, HG, MS Tsoi, and R. Storb. "Inhibition of prostaglandin E2 restores defective lymphocyte proliferation and cell-mediated lympholysis in recipients after allogeneic marrow grafting." Blood 68, no. 1 (1986): 102–7. http://dx.doi.org/10.1182/blood.v68.1.102.102.

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Abstract Prostaglandins are said to influence T and B cell function by inhibiting the generation of interleukin 2 (IL 2) and the formation of suppressor lymphocytes. After bone marrow transplantation, patients usually have a profound immunodeficiency that persists in recipients with chronic graft-v-host disease (GVHD) and generally resolves in long- term survivors without GVHD. In vitro tests of lymphocyte function such as allogeneic mixed lymphocyte culture (MLC) and cell-mediated lympholysis (CML) have been shown to be impaired in many patients. We postulated that prostaglandin E2 (PGE2) pla

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9

Klingemann, HG, MS Tsoi, and R. Storb. "Inhibition of prostaglandin E2 restores defective lymphocyte proliferation and cell-mediated lympholysis in recipients after allogeneic marrow grafting." Blood 68, no. 1 (1986): 102–7. http://dx.doi.org/10.1182/blood.v68.1.102.bloodjournal681102.

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Prostaglandins are said to influence T and B cell function by inhibiting the generation of interleukin 2 (IL 2) and the formation of suppressor lymphocytes. After bone marrow transplantation, patients usually have a profound immunodeficiency that persists in recipients with chronic graft-v-host disease (GVHD) and generally resolves in long- term survivors without GVHD. In vitro tests of lymphocyte function such as allogeneic mixed lymphocyte culture (MLC) and cell-mediated lympholysis (CML) have been shown to be impaired in many patients. We postulated that prostaglandin E2 (PGE2) plays a role

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10

Sanchez., Diana, Rosana Pelayo, Rosa Elena Sarmiento, et al. "In Vitro and in Vivo oncolytic Activity of Lasota Strain of Newcastle Disease Virus on a Lymphoma B-Cell Line and a Canine Cutaneous T-Cell Lymphoma." Blood 124, no. 21 (2014): 5504. http://dx.doi.org/10.1182/blood.v124.21.5504.5504.

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Abstract Oncolytic viruses, either naturally or genetically modified, possess the ability to kill cancer cells. Newcastle Disease Virus (NDV), a type I avian paramyxovirus, has demonstrated a selective ability to kill cancer cells directly as well as through immunostimulation. NDV is able to infect more than 250 species of birds, particularly poultry and is considered a zoonosis, primarily causing conjunctivitis. Importantly, no infections have been reported in mammalian species nor is there evidence of human to human transmission. We use the LaSota strain which is lentogenic (less pathogenic)

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Dissertations / Theses on the topic "Lympholytes T"

1

Schiffer, Cécile. "Etude de la phase afférente du cycle réplicatif du virus de l'immunodéficience humaine (VIH) - 1 dans les cellules du système immunitaire." Paris 7, 2004. http://www.theses.fr/2004PA077162.

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Book chapters on the topic "Lympholytes T"

1

Rakha, Aruna, Marta Todeschini, and Federica Casiraghi. "Assessment of Anti-donor T Cell Proliferation and Cytotoxic T Lymphocyte-Mediated Lympholysis in Living Donor Kidney Transplant Patients." In Methods in Molecular Biology. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1453-1_29.

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