Relevant bibliographies by topics / M1dG DNA Adduct

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  1. Journal articles
  2. Dissertations / Theses

Academic literature on the topic 'M1dG DNA Adduct'

Author: Grafiati
Published: 4 June 2021
Last updated: 5 February 2022

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Journal articles on the topic "M1dG DNA Adduct"

1

Moore, S. A., E. Humphreys, M. D. Friesen, D. E. G. Shuker, and S. A. Bingham. "The Effect of n-6 Polyunsaturated Fatty Acid on Blood Levels of Malondialdehyde-Deoxyguanosine Adducts in Human Subjects." Open Biomarkers Journal 1, no. 1 (2008): 28–35. http://dx.doi.org/10.2174/1875318300801010028.

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The role of n-6 polyunsaturated fats upon the formation of the mutagenic DNA adduct malondialdehydedeoxyguanosine (M1dG) in blood was investigated in male volunteers (n = 13) who consumed diets high in saturated and polyunsaturated fats, and polyunsaturated fat plus a-tocopherol supplemention (400 IU per day). On day 14 there was a significant difference in adduct levels between diets with saturated fats giving higher levels than polyunsaturated fats but this effect had disappeared by day 20 indicating that there is a relatively rapid adjustment to the effects on DNA damage of changes in dieta
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Cellai, Filippo, Fabio Capacci, Carla Sgarrella та ін. "A Cross-Sectional Study on 3-(2-Deoxy-β-D-Erythro-Pentafuranosyl)Pyrimido[1,2-α]Purin-10(3H)-One Deoxyguanosine Adducts among Woodworkers in Tuscany, Italy". International Journal of Molecular Sciences 20, № 11 (2019): 2763. http://dx.doi.org/10.3390/ijms20112763.

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Occupational exposure to wood dust has been estimated to affect 3.6 million workers within the European Union (EU). The most serious health effect caused by wood dust is the nasal and sinonasal cancer (SNC), which has been observed predominantly among woodworkers. Free radicals produced by inflammatory reactions as a consequence of wood dust could play a major role in SNC development. Therefore, we investigated the association between wood dust and oxidative DNA damage in the cells of nasal epithelia, the target site of SNC. We have analyzed oxidative DNA damage by determining the levels of 3-
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3

Wauchope, Orrette R., Michelle M. Mitchener, William N. Beavers, et al. "Oxidative stress increases M1dG, a major peroxidation-derived DNA adduct, in mitochondrial DNA." Nucleic Acids Research 46, no. 7 (2018): 3458–67. http://dx.doi.org/10.1093/nar/gky089.

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Wauchope, Orrette R., William N. Beavers, James J. Galligan, Michelle M. Mitchener, Philip J. Kingsley, and Lawrence J. Marnett. "Nuclear Oxidation of a Major Peroxidation DNA Adduct, M1dG, in the Genome." Chemical Research in Toxicology 28, no. 12 (2015): 2334–42. http://dx.doi.org/10.1021/acs.chemrestox.5b00340.

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Otteneder, M. B., C. G. Knutson, J. S. Daniels, et al. "In vivo oxidative metabolism of a major peroxidation-derived DNA adduct, M1dG." Proceedings of the National Academy of Sciences 103, no. 17 (2006): 6665–69. http://dx.doi.org/10.1073/pnas.0602017103.

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Knutson, Charles G., Paul L. Skipper, Rosa G. Liberman, Steven R. Tannenbaum та Lawrence J. Marnett. "Monitoring in Vivo Metabolism and Elimination of the Endogenous DNA Adduct, M1dG {3-(2-Deoxy-β-d-erythro-pentofuranosyl)pyrimido[1,2-α]purin-10(3H)-one}, by Accelerator Mass Spectrometry†". Chemical Research in Toxicology 21, № 6 (2008): 1290–94. http://dx.doi.org/10.1021/tx800049v.

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7

Riggins, James N., Derek A. Pratt, Markus Voehler, J. Scott Daniels та Lawrence J. Marnett. "Kinetics and Mechanism of the General-Acid-Catalyzed Ring-Closure of the Malondialdehyde−DNA Adduct,N2-(3-Oxo-1-propenyl)deoxyguanosine (N2OPdG-), to 3-(2‘-Deoxy-β-d-erythro-pentofuranosyl)pyrimido[1,2-α]purin- 10(3H)-one (M1dG)". Journal of the American Chemical Society 126, № 34 (2004): 10571–81. http://dx.doi.org/10.1021/ja040010q.

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Knutson, Charles G., Dapo Akingbade, Brenda C. Crews, Markus Voehler, Donald F. Stec, and Lawrence J. Marnett. "Metabolism in Vitro and in Vivo of the DNA Base Adduct, M1G." Chemical Research in Toxicology 20, no. 3 (2007): 550–57. http://dx.doi.org/10.1021/tx600334x.

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9

Jeong, Yo-Chan, Nigel J. Walker, Deborah E. Burgin, et al. "Accumulation of M1dG DNA adducts after chronic exposure to PCBs, but not from acute exposure to polychlorinated aromatic hydrocarbons." Free Radical Biology and Medicine 45, no. 5 (2008): 585–91. http://dx.doi.org/10.1016/j.freeradbiomed.2008.04.043.

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10

Schnetz-Boutaud, Nathalie C., Sam Saleh, Lawrence J. Marnett, and Michael P. Stone. "The Exocyclic 1,N2-Deoxyguanosine Pyrimidopurinone M1G Is a Chemically Stable DNA Adduct When Placed Opposite a Two-Base Deletion in the (CpG)3Frameshift Hotspot of theSalmonellatyphimurium hisD3052Gene†." Biochemistry 40, no. 51 (2001): 15638–49. http://dx.doi.org/10.1021/bi011242u.

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Dissertations / Theses on the topic "M1dG DNA Adduct"

1

Yates, Sally A. "The cytotoxic effects of malondialdehyde on human lung fibroblast cells." Thesis, Liverpool John Moores University, 2015. http://researchonline.ljmu.ac.uk/4529/.

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Malondialdehyde (MDA) is a mutagenic and carcinogenic product of lipid peroxidation which has also been found at elevated levels in smokers. MDA reacts with nucleic acid bases to form pyrimidopurinone DNA adducts, of which 3-(2-deoxy-β-D-erythro-pentofuranosyl)pyrimidol[1,2-α]purin-10(3H)-one (M1dG) is the most abundant and has been linked to smoking. Mutations in the TP53 tumour suppressor gene are associated with half of all cancers. This research applied a multidisciplinary approach to investigate the toxic effects of MDA on the human lung fibroblasts MRC5, which have an intact p53 response
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