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Relevant bibliographies by topics / Macrophage

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Academic literature on the topic 'Macrophage'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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Journal articles on the topic "Macrophage"

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Rodriguez, Eric, Frederic Boudard, Michele Mallié, Jean-Marie Bastide, and Madeleine Bastide. "Murine macrophage elastolytic activity induced by Aspergillus fumigatus strains in vitro: evidence of the expression of two macrophage-induced protease genes." Canadian Journal of Microbiology 43, no. 7 (1997): 649–57. http://dx.doi.org/10.1139/m97-092.

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The interaction between Aspergillus fumigatus conidia and murine macrophages of various origins was investigated. Cocultures were carried out between A. fumigatus strains and freshly isolated murine pulmonary alveolar macrophages or two murine macrophage cell-lines: murine alveolar cell-line MALU and murine astrocytoma cell-line J774. By measuring the variation of elastolytic activity in the coculture supernatants with two elastin substrates, we demonstrated that either viable or fixed A. fumigatus or C. albicans yeasts or nonspecific particles induced significant macrophage elastolytic activi

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Stojadinović, Marija. "Macrophage polarization and infectious diseases." Biologia Serbica 45, no. 2 (2023): 38–43. https://doi.org/10.5281/zenodo.10402369.

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<strong>Summary. </strong>Macrophages are a heterogeneous cell population present in most mammalian tissues with a wide range of functions. They are an essential component of optimal tissue homeostasis and an essential first line of defense against pathogens. Activated macrophages are typically divided into two phenotypes, M1 macrophages and M2 macrophages, which are influenced by microorganisms, the tissue microenvironment, and cytokine signals from physiological conditions to infections. The management of macrophage polarity is crucial for the prevention and treatment of infections and infla

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Liu, Shuangqing, Huilei Zhang, Yanan Li та ін. "S100A4 enhances protumor macrophage polarization by control of PPAR-γ-dependent induction of fatty acid oxidation". Journal for ImmunoTherapy of Cancer 9, № 6 (2021): e002548. http://dx.doi.org/10.1136/jitc-2021-002548.

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BackgroundThe peroxisome proliferator-activated receptor γ (PPAR-γ)-dependent upregulation of fatty acid oxidation (FAO) mediates protumor (also known as M2-like) polarization of tumor-associated macrophages (TAMs). However, upstream factors determining PPAR-γ upregulation in TAM protumor polarization are not fully identified. S100A4 plays crucial roles in promotion of cancer malignancy and mitochondrial metabolism. The fact that macrophage-derived S100A4 is major source of extracellular S100A4 suggests that macrophages contain a high abundance of intracellular S100A4. However, whether intrace

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Wilson, Justin E., Bhuvana Katkere, and James R. Drake. "Francisella tularensis Induces Ubiquitin-Dependent Major Histocompatibility Complex Class II Degradation in Activated Macrophages." Infection and Immunity 77, no. 11 (2009): 4953–65. http://dx.doi.org/10.1128/iai.00844-09.

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ABSTRACT The intracellular bacterium Francisella tularensis survives and replicates within macrophages, ultimately killing the host cell. Resolution of infection requires the development of adaptive immunity through presentation of F. tularensis antigens to CD4+ and CD8+ T cells. We have previously established that F. tularensis induces macrophage prostaglandin E2 (PGE2) production, leading to skewed T-cell responses. PGE2 can also downregulate macrophage major histocompatibility complex (MHC) class II expression, suggesting that F. tularensis-elicited PGE2 may further alter T-cell responses v

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Pedicillo, Maria Carmela, Ilenia Sara De Stefano, Rosanna Zamparese, et al. "The Role of Toll-like Receptor-4 in Macrophage Imbalance in Lethal COVID-19 Lung Disease, and Its Correlation with Galectin-3." International Journal of Molecular Sciences 24, no. 17 (2023): 13259. http://dx.doi.org/10.3390/ijms241713259.

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To the current data, there have been 6,955,141 COVID-19-related deaths worldwide, reported to WHO. Toll-like receptors (TLRs) implicated in bacterial and virus sensing could be a crosstalk between activation of persistent innate-immune inflammation, and macrophage’s sub-population alterations, implicated in cytokine storm, macrophage over-activation syndrome, unresolved Acute Respiratory Disease Syndrome (ARDS), and death. The aim of this study is to demonstrate the association between Toll-like-receptor-4 (TLR-4)-induced inflammation and macrophage imbalance in the lung inflammatory infiltrat

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Careau, Éric, Léa-Isabelle Proulx, Philippe Pouliot, Annie Spahr, Véronique Turmel, and Élyse Y. Bissonnette. "Antigen sensitization modulates alveolar macrophage functions in an asthma model." American Journal of Physiology-Lung Cellular and Molecular Physiology 290, no. 5 (2006): L871—L879. http://dx.doi.org/10.1152/ajplung.00219.2005.

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We have previously demonstrated that adoptive transfer of alveolar macrophages from allergy-resistant rats to alveolar macrophage-depleted allergic rats prevents airway hyperresponsiveness development, suggesting an important role for alveolar macrophages in asthma pathogenesis. Given that ovalbumin sensitization can modulate alveolar macrophage cytokine production, we investigated the role of sensitized and unsensitized alveolar macrophages in an asthma model. Alveolar macrophages from unsensitized or sensitized Brown Norway rats were transferred to alveolar macrophage-depleted sensitized rat

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Xu, Jiawei, Lanya Fu, Junyao Deng, et al. "miR-301a Deficiency Attenuates the Macrophage Migration and Phagocytosis through YY1/CXCR4 Pathway." Cells 11, no. 24 (2022): 3952. http://dx.doi.org/10.3390/cells11243952.

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(1) Background: the miR-301a is well known involving the proliferation and migration of tumor cells. However, the role of miR-301a in the migration and phagocytosis of macrophages is still unclear. (2) Methods: sciatic nerve injury, liver injury models, as well as primary macrophage cultures were prepared from the miR-301a knockout (KO) and wild type (WT) mice to assess the macrophage’s migration and phagocytosis capabilities. Targetscan database analysis, Western blotting, siRNA transfection, and CXCR4 inhibition or activation were performed to reveal miR301a’s potential mechanism. (3) Result

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McKenzie, C. G. J., U. Koser, L. E. Lewis, et al. "Contribution of Candida albicans Cell Wall Components to Recognition by and Escape from Murine Macrophages." Infection and Immunity 78, no. 4 (2010): 1650–58. http://dx.doi.org/10.1128/iai.00001-10.

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ABSTRACT The pathogenicity of the opportunistic human fungal pathogen Candida albicans depends on its ability to escape destruction by the host immune system. Using mutant strains that are defective in cell surface glycosylation, cell wall protein synthesis, and yeast-hypha morphogenesis, we have investigated three important aspects of C. albicans innate immune interactions: phagocytosis by primary macrophages and macrophage cell lines, hyphal formation within macrophage phagosomes, and the ability to escape from and kill macrophages. We show that cell wall glycosylation is critically importan

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Fahey, T. J., K. J. Tracey, P. Tekamp-Olson, et al. "Macrophage inflammatory protein 1 modulates macrophage function." Journal of Immunology 148, no. 9 (1992): 2764–69. http://dx.doi.org/10.4049/jimmunol.148.9.2764.

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Abstract Macrophage inflammatory protein 1 (MIP 1), initially purified from the conditioned medium of endotoxin-stimulated macrophages, is a low m.w. heparin-binding protein doublet comprising two peptides, MIP 1 alpha and MIP 1 beta. Although native doublet MIP 1 has previously been shown to exert pyrogenic, mitogenic, and proinflammatory effects on other cell types, its actions on its cell of origin, the macrophage, have not been well catalogued. Our study reports several aspects of macrophage function that are modulated by MIP 1. MIP 1 was not directly cytotoxic for WEHI tumor cells, but MI

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Dende, Chaitanya, Mihir Pendse, Daniel Propheter, Gabriella Quinn, and Lora V. Hooper. "Vitamin A regulates phagocytosis by resident macrophages of the small intestine." Journal of Immunology 208, no. 1_Supplement (2022): 113.23. http://dx.doi.org/10.4049/jimmunol.208.supp.113.23.

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Abstract Intestinal Tim4+ CD4+ macrophages are a distinctive macrophage subset that express Tim4, a receptor for phosphatidylserine on dying apoptotic cells, Unlike other macrophage subsets, they do not depend on blood monocytes for their turnover, instead self-maintained in the small intestine. The signal(s) responsible for the self-maintenance and function of Tim4+ CD4+ macrophages is not known. We have discovered that maintenance of the gut resident Tim4+ CD4+ macrophage population depends on dietary vitamin A and its derivative retinoic acid (RA). Retinoic acid receptors, which direct RA-d

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Dissertations / Theses on the topic "Macrophage"

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Svensson, Ulf. "Macrophage activation by bacteria signalling to prostaglandin and cytokine responses /." Lund : Dept. of Medical & Physiological Chemistry, Lund University, 1994. http://books.google.com/books?id=sAhrAAAAMAAJ.

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Higuera, González Laura 1993. "Novel transcription regulators of tissue macrophages and alternative macrophage polarization." Doctoral thesis, TDX (Tesis Doctorals en Xarxa), 2021. http://hdl.handle.net/10803/672702.

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Macrophages play crucial roles in the defense of the organism against a wide range of pathogens. Macrophages can rapidly adapt to perturbations in the microenvironment due to the existence of a network of transcription factors that modulates their responses. While transcription factors that regulate macrophage identity have been widely described in the past decades, the role of transcription regulators that fine-tune tissue macrophage responses in homeostasis and infection is starting to be elucidated. Our group has previously identified transcription regulators of pro-inflammatory macrophag

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Tabata, Yasuhiko. "Macrophage phagocytosis of polymer microspheres and antitumor activation of macrophages." Kyoto University, 1987. http://hdl.handle.net/2433/74704.

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Raborn, Erinn Shenee. "Cannabinoid Modulation of Chemotaxis of Macrophages and Macrophage-like Cells." VCU Scholars Compass, 2007. http://hdl.handle.net/10156/1333.

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Adler, Heiko. "Fetal bovine bone marrow-derived macrophages : a model for studying basic aspects of macrophage biology and pathogen-macrophage interaction in cattle /." [S.l.] : [s.n.], 1994. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.

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Grand-Perret, Thierry A. R. "Induction d'une activité anti-tumorale chez les macrophages péritonéaux murins." Paris 11, 1986. http://www.theses.fr/1986PA112301.

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Di, Maggio Paula. "Dietary lipids and inflammation : chylomicron remnants suppress pro-inflammatory pathways and activate antioxidant defence mechanisms in human macrophages." Thesis, Royal Veterinary College (University of London), 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618287.

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Soe-Lin, Shan. "Macrophage iron recycling." Thesis, McGill University, 2009. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=66717.

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In an absurd twist of nature, the physiological role of iron is paradoxical. Iron is the most abundant element found on Earth and yet is insoluble under physiological conditions. Furthermore, life is not possible without iron; iron is indispensible for life, as it is a vital co-factor for essential enzymes due to its unique redox abilities. And yet, high concentrations of iron lead to the formation of reactive oxygen species and are toxic. Consequently, living creatures have evolved ingenious strategies for acquiring and managing otherwise insoluble iron atoms, and for tightl

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Georges, George Tharwat. "Novel Characteristics of Murine Bone Marrow-Derived Macrophages and Human Macrophage-Like Cells." VCU Scholars Compass, 2006. http://scholarscompass.vcu.edu/etd/932.

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These studies provide evidence for novel properties of macrophages derived from bone marrow stem cells. In study 1, treatment of activated mouse bone marrow-derived macrophages (BMM) with either catecholamine synthesis inhibitors (α-methyl-para-tyrosine and fusaric acid) or the β2 adrenergic receptor antagonist ICI 118,551 demonstrated that BMM produce catecholamines. The catecholamines modulated macrophage cytokine production through autocrine actions on adrenergic receptors. In study II, undifferentiated human bone marrow cells were incubated in 30% mouse L929 fibroblast conditioned medium

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Sobhani, Kimia. "Proteomic analysis of macrophage proinflammatory programmed cell death and macrophage activation /." Thesis, Connect to this title online; UW restricted, 2006. http://hdl.handle.net/1773/8688.

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Books on the topic "Macrophage"

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Bernard, Burke, and Lewis Claire E, eds. The macrophage. 2nd ed. Oxford University Press, 2002.

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E, Lewis Claire, and McGee J. O'D, eds. The Macrophage. IRL Press at Oxford University Press, 1992.

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Röszer, Tamás. The M2 Macrophage. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-50480-9.

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Clemens, Sorg, and Immunologische Tage. (1987 : Münster, West Germany), eds. The Alveolar macrophage. Regensberg & Biermann Wissenschaftliche Verlagsgellschaft, 1988.

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1943-, Zwilling Bruce S., and Eisenstein Toby K, eds. Macrophage-pathogen interactions. M. Dekker, 1994.

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Gupta, Swati, and Yashwant V. Pathak, eds. Macrophage Targeted Delivery Systems. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-84164-5.

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Russell, Stephen W., and Siamon Gordon, eds. Macrophage Biology and Activation. Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-77377-8.

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Harris, James, and Eric F. Morand, eds. Macrophage Migration Inhibitory Factor. Springer US, 2020. http://dx.doi.org/10.1007/978-1-4939-9936-1.

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1929-, Staub Norman C., ed. The Pulmonary intravascular macrophage. Futura Pub. Co., 1989.

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W, Russell Stephen, and Gordon Siamon, eds. Macrophage biology and activation. Springer-Verlag, 1992.

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Book chapters on the topic "Macrophage"

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Kawauchi, Hideyuki. "Macrophage." In Nasal Physiology and Pathophysiology of Nasal Disorders. Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-37250-6_6.

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Chazaud, Bénédicte. "Macrophage." In Encyclopedia of Exercise Medicine in Health and Disease. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_245.

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Gooch, Jan W. "Macrophage." In Encyclopedic Dictionary of Polymers. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_14168.

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Aribi, Mourad. "Macrophage Bactericidal Assays." In Macrophages. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_14.

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Guidi-Rontani, C., and M. Mock. "Macrophage Interactions." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-662-05767-4_6.

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Vignery, Agnès. "Macrophage Fusion." In Cell Fusion. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-250-2_9.

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Verschoor, Chris P., Alicja Puchta, and Dawn M. E. Bowdish. "The Macrophage." In Methods in Molecular Biology. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-527-5_10.

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Schomburg, Dietmar, and Dörte Stephan. "Macrophage elastase." In Enzyme Handbook 16. Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-58903-4_87.

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Lewis, J. G. "Macrophage Activation." In Encyclopedia of Immunotoxicology. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-54596-2_936.

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Hartwig, J. H., and T. P. Stossel. "Macrophage movements." In Mononuclear Phagocytes. Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-5020-7_34.

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Conference papers on the topic "Macrophage"

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Alhammadi, Jawaher, Nabila Yasmeen, Nicholas Hallfors, et al. "Bioelectronic Monitoring of Monocyte-to-Macrophage Differentiation." In 2024 International Conference on Engineering and Emerging Technologies (ICEET). IEEE, 2024. https://doi.org/10.1109/iceet65156.2024.10913575.

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Mahgoub, Yasmine, Rida Arif, and Susu Zughaier. "Pyocyanin pigment from Pseudomonas aeruginosa modulates innate immune defenses in macrophages." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2021. http://dx.doi.org/10.29117/quarfe.2021.0137.

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Background: Pseudomonas aeruginosa is a well-known opportunistic pathogen. The gram-negative bacillus, commonly associated with hospital-acquired infections, utilizes the host’s impaired immune responses to establish infection. Of its many virulence factors, pyocyanin is essential for P. aeruginosa to establish its full infectivity. Macrophages act as sentinels of the innate immune system, as well as play other roles in homeostasis, tissue remodeling, and bridging between the innate and adaptive immune systems. Aim: This study aimed to investigate the effects of pyocyanin on macrophage innate

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van Dam-Mieras, M. C. E., A. D. Muller, and G. Hornstra. "DIETARY LIPIDS, INFECTION AND MACROPHAGE PROCOAGULANT ACTIVITY." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643398.

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It is generally accepted that the type of dietary fat influences arterial thrombosis and atherosclerosis. Although it is still largely unknown how the dietary lipid composition influences the process of atherogenesis, it is evident that several cell types are involved. Morphological evidence for the involvement of monocyte/macropages has been given.We described before that the dietary lipid composition has striking effects on the procoagulant activity of macrophages. When macrophages were isolated from the spleens of healthy rats the procoagulant activity slightly decreased during the first fe

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Hobro, Alison J., Takeshi Sugiyama, Nicolas Pavillon, Takayuki Umakoshi, Prabhat Verma, and Nicholas Smith. "Label-free Raman imaging of saturated and unsaturated fatty acid uptake, storage, and return toward baseline levels in macrophages." In JSAP-Optica Joint Symposia. Optica Publishing Group, 2023. http://dx.doi.org/10.1364/jsapo.2023.19a_a602_1.

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Lipids play many important roles in the body including cell signaling and energy storage. The presence of excessive lipids, or disruption of normal lipid metabolic processes in the cell, has been linked to lifestyle diseases such as atherosclerosis and obesity. Where the affected cells are part of the innate immune system such dysregulation of lipids has also been implicated in impaired immune responses to infection. Therefore, understanding how macrophages are affected by the presence of fatty acids in their local environment is an important step in understanding lifestyle disease development

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Reinhard, Björn M., Hongyun Wang, and Linxi Wu. "Monitoring Cellular Trafficking of Nanoparticle Cargo in Murine Macrophages Through Plasmon Coupling Microscopy." In ASME 2013 2nd Global Congress on NanoEngineering for Medicine and Biology. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/nemb2013-93078.

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A detailed analysis of silver nanoparticle (NP) uptake and trafficking in the murine macrophage cell line J774A.1 through spectral analysis of the resonance wavelength of the metal NP cargo is presented. The NP spectra reveal a strong phenotypic variability in the NP uptake and processing on the single cell level. Cells containing non- or low-agglomerated NPs are found to coexist with cells containing NPs of varying degrees of NP agglomeration, clearly indicated by a spectral red-shift in the resonance wavelength. Pharmacological inhibition studies indicate that the observed differences in the

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Tenorio, Elizabeth, Del Donehoo, Sanjeevani Sahu, Achu Byju, Shreya A. Raghavan, and Balakrishna Haridas. "Evaluating Macrophage Immune Response to Degradation Byproducts from Magnesium Alloy Wire." In 2025 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2025. https://doi.org/10.1115/dmd2025-1062.

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Abstract Magnesium alloys have recently gained increasing attention for biomedical applications due to their unique biodegradation and biocompatibility profiles. However, investigations of the interactions between magnesium alloy degradation byproducts and host immune cells (particularly macrophages) are still relatively unexplored. Here, we study the interaction between degradation byproducts and macrophage response in vitro. An extract-based in vitro study was developed to examine macrophage response to different degradation byproducts formed at various time points of the corrosion process w

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Gijsen, Frank, Anna Ten Have, Jolanda Wentzel, and Antonius Van Der Steen. "Temperature Measurement of Advanced Murine Atherosclerotic Plaques." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176307.

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Ischaemic heart disease is most frequently caused by coronary atherosclerosis, of which the vulnerable plaque is one of the developmental stages. Rupture of a vulnerable plaque with superimposed thrombosis frequently leads to acute coronary syndromes. The major components of a vulnerable plaque are a lipid-rich, atheromatous core, and a thin fibrous cap with macrophage and macrophage infiltration (Schaar et al., 2004). After the first paper suggesting the possibility of thermographic detection of vulnerable plaques (Casscells et al., 1996), intracoronary thermography as a vulnerable plaque det

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McGee, Maria, and Henry Rothberger. "MECHANISMS OF PROCOAGULANT GENERATION BY ALVEOLAR MACROPHAGES DURING MATURATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643168.

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During maturation in vivo and in vitro alveolar macrophages generate procoagulant(s) capable of activating the extrinsic pathway. It is generally agreed that at least part of the activity is due to TF (tissue factor). However, whether or not macrophages also generate functional factor VII or X is controversial. To characterize procoagulant activity increases, we measured kinetic parameters defining interactions between components of the TF-VII complex on membranes of alveolar macrophages either freshly isolated or cultured in serum free medium. In incubation mixtures with fixed concentrations

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Nikishina, M. A., S. P. Fedotov, E. V. Fedotov, and V. Fedotov. "STATISTICAL ANALYSIS OF NANOPARTICLE UPTAKE BY CELLS." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-31.

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Experimental data on nanoparticle uptake by rat alveolar macrophages were analyzed. The distribution of pits on the surface of a single macrophage was statistically analyzed based on a limited sample of 100 and 200 subregions. It was shown that the negative binomial distribution provides a more accurate model for experimentally observed pits on the surface of a single cell.

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DUBOR, F., A. M. DOSNE, and L. CHEDID. "Effect of dexamethasone and endothelial cell supernatant on u-PA produced by human promyelocyte cells treated with phorbol myristate acetate (PMA)." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643191.

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After treatment with PMA the human promyelocytic HL60 cells were induced to differentiate into a monocyte-macrophage population and to produce a high amount of plasminogen activator in the supernatant. This response was detected from 0,5 ng/ml of PMA and culminated at 5 ng. The plasminogen activator appeared of urokinase-type as showed by fibrinenzymographic analysis : the enzymatic profile of cell supernatant showed 2 lysis band (Mr 33.000 and 55.000) corresponding to those of urokinase of low and high mol. weight. Dexamethasone (100 pM) suppressed the production of this macrophage u-PA witho

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Reports on the topic "Macrophage"

1

Hanna, Philip C. Macrophage Responses to B. Anthracis. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada456287.

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Peterson, Scott N. Macrophage Responses to B. Anthracis. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada428855.

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Broaddus, V. C. Role of Macrophage-induced Inflammation in Mesothelioma. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada582550.

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Abrass, Itamar B., and Christine K. Abrass. Influence of Stress-Induced Catecholamines on Macrophage Phagocytosis. Defense Technical Information Center, 1989. http://dx.doi.org/10.21236/ada206608.

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Blystone, Robert V. Image Analysis of Viral-Expressing Mouse Macrophage Cells. Defense Technical Information Center, 1991. http://dx.doi.org/10.21236/ada238230.

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Groopman, Jerome E. Pathobiology of HTLV-III/LAV In Human Monocyte-Macrophage. Defense Technical Information Center, 1990. http://dx.doi.org/10.21236/ada221724.

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Laouar, A., C. B. H. Chubb, F. Collart, and E. Huberman. Human macrophage differentiation involves an interaction between integrins and fibronectin. Office of Scientific and Technical Information (OSTI), 1996. http://dx.doi.org/10.2172/495739.

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Laouar, A., C. B. H. Chubb, F. Collart, and E. Huberman. Human macrophage differentiation involves an interaction between integrins and fibronectin. Office of Scientific and Technical Information (OSTI), 1997. http://dx.doi.org/10.2172/515532.

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Cioffi, William G., Burleson Jr, Jordan David G., Becker Bryan S., McManus William K., and William F. Effects of Granulocyte-Macrophage Colony-Stimulating Factor in Burn Patients. Defense Technical Information Center, 1991. http://dx.doi.org/10.21236/ada245115.

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Ciraci, Ceren, Christopher K. Tuggle, Michael J. Wannemuehler, Daniel S. Nettleton, and Susan J. Lamont. Kinetic Profile of Chicken Macrophage Immune Response upon exposure to Salmonella-derived Endotoxin. Iowa State University, 2010. http://dx.doi.org/10.31274/ans_air-180814-134.

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