Relevant bibliographies by topics / Macrophage labeling

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'Macrophage labeling'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Macrophage labeling"

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Diez-Roux, G., M. Argilla, H. Makarenkova, K. Ko, and R. A. Lang. "Macrophages kill capillary cells in G1 phase of the cell cycle during programmed vascular regression." Development 126, no. 10 (1999): 2141–47. http://dx.doi.org/10.1242/dev.126.10.2141.

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Programmed capillary regression occurs during normal development of the eye and serves as a useful model for assessing the forces that drive vascular involution. Using a combination of S-phase labeling and liposome-mediated macrophage elimination, we show that during regression, macrophages induce apoptosis of both pericytes and endothelial cells in a cell cycle stage-dependent manner. Target cells are signaled to die by macrophages approximately 15 hours after S-phase labeling and this corresponds to a point in mid-G1 phase of the cell cycle. The tight correlation between the restriction poin
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Saini, Yogesh, Owais Mohammad, Brandon W. Lewis, and Razia Sultana. "Understanding macrophage recruitment to the stressed airspaces." Journal of Immunology 196, no. 1_Supplement (2016): 60.10. http://dx.doi.org/10.4049/jimmunol.196.supp.60.10.

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Abstract The presence of functionally competent macrophages in respiratory airspaces is critical to the normal host immune responses. To determine the effect of altered macrophage (MΦ) populations on macrophage recruitment, we employed LysM+/mTOM\mEGFP+/DTA+ mice, a model of macrophage depletion and mEGFP+ labeling. Since controlled by same promoter (Lysozyme M; LysM) and separate alleles of ROSA locus, the expression of mEGFP and DTA transgenes is simultaneously induced that facilitates mEGFP+ fluorescent labeling of DTA+ cells with LysM+ activity. Flow cytometric and confocal microscopic ana
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Cummings, Thomas J., Christine M. Hulette, Sandra H. Bigner, Gregory J. Riggins, and Roger E. McLendon. "HAM56-Immunoreactive Macrophages in Untreated Infiltrating Gliomas." Archives of Pathology & Laboratory Medicine 125, no. 5 (2001): 637–41. http://dx.doi.org/10.5858/2001-125-0637-himiui.

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Abstract Context.—Classic diagnostic neuropathologic teachings have cautioned against making the diagnosis of neoplasia in the presence of a macrophage population. The knowledge of macrophage distribution should prove useful when confronted with an infiltrating glioma containing macrophages. Objective.—To identify macrophages in untreated, infiltrating gliomas using the monoclonal antibody HAM56, and to confirm their presence in an untreated glioblastoma multiforme (GBM) with the serial analysis of gene expression (SAGE) method. Methods.—We evaluated the presence of macrophages in 16 cases of
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Tchernychev, Boris, Bruce Furie, and Barbara C. Furie. "Peritoneal macrophages express both P-selectin and PSGL-1." Journal of Cell Biology 163, no. 5 (2003): 1145–55. http://dx.doi.org/10.1083/jcb.200310079.

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Macrophages, phagocytic cells involved in an early phase of host defense, are known to express the P-selectin ligand, PSGL-1. Heretofore, P-selectin has only been found on platelets and endothelial cells. Here, we demonstrate that peritoneal macrophages isolated by peritoneal lavage of unchallenged mice express P-selectin on the plasma membrane. The peritoneal macrophages synthesize P-selectin, as indicated by metabolic labeling experiments. P-Selectin is constitutively expressed on the extracellular surface of macrophages but is only partially colocalized with PSGL-1. P-Selectin is rapidly tr
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Hammer, Daniel X., Anant Agrawal, Ricardo Villanueva, Osamah Saeedi, and Zhuolin Liu. "Label-free adaptive optics imaging of human retinal macrophage distribution and dynamics." Proceedings of the National Academy of Sciences 117, no. 48 (2020): 30661–69. http://dx.doi.org/10.1073/pnas.2010943117.

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Microglia are resident central nervous system macrophages and the first responders to neural injury. Until recently, microglia have been studied only in animal models with exogenous or transgenic labeling. While these studies provided a wealth of information on the delicate balance between neuroprotection and neurotoxicity within which these cells operate, extrapolation to human immune function has remained an open question. Here we examine key characteristics of retinal macrophage cells in live human eyes, both healthy and diseased, with the unique capabilities of our adaptive optics–optical
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Mercurio, A. M., and P. W. Robbins. "Activation of mouse peritoneal macrophages alters the structure and surface expression of protein-bound lactosaminoglycans." Journal of Immunology 135, no. 2 (1985): 1305–12. http://dx.doi.org/10.4049/jimmunol.135.2.1305.

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Abstract We have begun to analyze and compare the surface carbohydrates present on populations of resident and activated mouse peritoneal macrophages. The activated macrophage populations studied include TG-elicited macrophages, BCG-activated macrophages, and resident macrophages cultured for 24 hr in the presence of lymphokines or heterologous serum. Analysis of glycopeptides generated by pronase digestion of surface glycoproteins labeled by the neuraminidase/galactose oxidase/NaB3H4 method indicates that the macrophage surface contains a class of high m.w. carbohydrates susceptible to degrad
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Bassiouni, Mohamed, Philipp Arens, Samira Ira Zabaneh, Heidi Olze, David Horst, and Florian Roßner. "The Relationship between the M1/M2 Macrophage Polarization and the Degree of Ossicular Erosion in Human Acquired Cholesteatoma: An Immunohistochemical Study." Journal of Clinical Medicine 11, no. 16 (2022): 4826. http://dx.doi.org/10.3390/jcm11164826.

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The differential involvement of the macrophage activation phenotypes (M1 vs. M2) has been linked to disease severity in various chronic inflammatory disorders. Pharmacologic manipulation of the M1/M2 macrophage polarization has shown therapeutic potential. Cholesteatoma is a destructive chronic middle ear disease with potentially life-threatening complications. The distribution of macrophage polarization phenotypes in middle ear cholesteatoma has not been described. In the present study, human cholesteatoma specimens acquired during tympanomastoidectomy were retrospectively retrieved and immun
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Mecham, J. O., M. M. Soong, C. A. Cain, S. Koehm, J. Goff, and W. A. Tompkins. "Binding of calmodulin to the microfilament network correlates with induction of a macrophage tumoricidal response." Journal of Immunology 134, no. 5 (1985): 3516–23. http://dx.doi.org/10.4049/jimmunol.134.5.3516.

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Abstract Induction of mouse peritoneal macrophage cytotoxicity against SV3T3, a line of virally transformed mouse cells correlated with the distribution of cytoplasmic calmodulin in the macrophages. The organization of the cytoskeleton was examined by fluorescent microscopy and by transmission electron microscopy, using immunogold tagging after Triton-X-100 (TX-100) extraction of the macrophages. Macrophages that had been activated to a tumoricidal state in vivo by vaccinia virus or in vitro by lymphokine stimulation displayed cytoskeletal networks that were more extended and weblike than did
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Metcalf, D., M. J. Elliott, and N. A. Nicola. "The excess numbers of peritoneal macrophages in granulocyte-macrophage colony-stimulating factor transgenic mice are generated by local proliferation." Journal of Experimental Medicine 175, no. 4 (1992): 877–84. http://dx.doi.org/10.1084/jem.175.4.877.

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Mice transgenic for the hemopoietic growth factor, granulocyte-macrophage colony-stimulating factor (GM-CSF), exhibit a sustained elevation of GM-CSF levels and a 50-100-fold elevation of peritoneal macrophage cell numbers. The excess cell numbers were found to be generated in pre-adult life, with numbers remaining relatively constant thereafter. In the pre-adult period, no abnormalities were noted in the number or composition of blood, bone marrow, or spleen cells, the type or number of GM progenitor cells in the marrow or spleen, or the rate of appearance of newly formed monocytes in the per
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Morrissette, N. S., E. S. Gold, J. Guo, et al. "Isolation and characterization of monoclonal antibodies directed against novel components of macrophage phagosomes." Journal of Cell Science 112, no. 24 (1999): 4705–13. http://dx.doi.org/10.1242/jcs.112.24.4705.

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In order to identify novel proteins associated with various stages of macrophage phagocytosis, we have generated monoclonal antibodies that recognize phagosomes. Purified Fc receptor-mediated phagosomes, isolated by feeding IgG-conjugated magnetic beads to LPS-primed murine peritoneal macrophages, were used as the immunogen. An immunofluorescence screen was used to isolate and single-cell clone approximately 150 monoclonal antibodies that recognize mouse macrophage phagosomes as well as labeling other cellular components in patterns which are frequently distinct from those observed with previo
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Dissertations / Theses on the topic "Macrophage labeling"

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Bessaad, Mohamed El Amine. "Détection et suivi en IRM du macrophage polarisé et marqué aux nanoparticules de fer dans l’athérosclérose et l’imagerie de l’inflammation." Thesis, Lyon 1, 2010. http://www.theses.fr/2010LYO10041.

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L’athérosclérose est une maladie inflammatoire, caractérisée par une accumulation lipidique et cellulaire, dont le macrophage est l’acteur principal. Dans l’athérosclérose, le macrophage sécrète des cytokines qui favorisent le chimiotactisme et donc la migration et l’internalisation des cellules immunitaires, accentuant le processus pro-inflammatoire et accélérant l’évolution des plaques athéromateuses et leur instabilité jusqu’à éventuellement la rupture et/ou la formation de thrombus. Il est donc important de développer une technique d’imagerie cellulaire permettant de visualiser les macroph
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Sun, Xing [Verfasser], and Wolfgang [Akademischer Betreuer] Hansen. "Labeling Stem Cells and Macrophages with Gold and Iron Oxide Nanoparticles for Tracking Applications / Xing Sun ; Betreuer: Wolfgang Hansen." Hamburg : Staats- und Universitätsbibliothek Hamburg, 2019. http://d-nb.info/1195709587/34.

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Selt, Marion [Verfasser], Hermann [Akademischer Betreuer] Wagner, and Mathias [Akademischer Betreuer] Hoehn. "Observing the blood police : iron labelling of murine monocytes and macrophages for in vivo noninvasive multimodal imaging in neurodegenerative diseases / Marion Selt ; Hermann Wagner, Mathias Hoehn." Aachen : Universitätsbibliothek der RWTH Aachen, 2016. http://d-nb.info/1125911298/34.

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Yang, Chung-Yi, and 楊中宜. "Labeling of Iron Oxide Nanoparticles on Macrophage and Mesenchymal Stem Cells: Study of Labeling Efficacy, Mechanism of Cellular Uptake and Impact on Cellular Physiology." Thesis, 2012. http://ndltd.ncl.edu.tw/handle/62423880444417185555.

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博士<br>國立臺灣大學<br>醫學工程學研究所<br>100<br>Various magnetic resonance (MR) contrast agents, including paramagnetic and superparamagnetic substances, have been used for cell labeling in cellular imaging. Superparamagnetic iron oxide (SPIO) nanoparticles are the most commonly used superparamagnetic contrast agents used for magnetic resonance imaging while Gadolinium (Gd) is the most common clinically used paramagnetic contrast agents. Macrophages, one kind of specialized blood cells that are capable of phagocytosis, would ingest SPIOs and produces signals on MR imaging. To improve the labeling efficacy
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Swain, Lija. "Prolyl-4-hydroxylase domain 3 (PHD3) is a critical terminator for cell survival of macrophages under stress conditions." Doctoral thesis, 2014. http://hdl.handle.net/11858/00-1735-0000-0022-5F15-A.

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Book chapters on the topic "Macrophage labeling"

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Williams, Kevin J., and Steven J. Bensinger. "Cellular Fatty Acid Analysis in Macrophage Using Stable Isotope Labeling." In Methods in Molecular Biology. Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0802-9_4.

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Soesatyo, Marsetyawan, Theo Thepen, Muhammad Ghufron, Jeike Biewenga, and Taede Sminia. "Peritoneal Cell Labelling: A Study on the Migration of Macrophages and Dendritic Cells Towards the GUT." In Advances in Experimental Medicine and Biology. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2930-9_54.

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Conference papers on the topic "Macrophage labeling"

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Adany, R., A. Kiss, J. Kappelmayer, R. J. Ablin, and L. Muszbek. "EXPRESSION OF FACTOR XIII SUBUNIT A IN DIFFERENT TYPES OF HUMAN MACROPHAGES." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644651.

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In addition to plasma the presence of subunit a of blood coagulation Factor XIII (FXIIl) has been verified in platelets and megakariocytes. Most recently, we demonstrated that human peripheral blood monocytes also contain FXIII subunit a. The present study was designed 1/ to determine the stage in the maturation sequence of bone marrow monocytopoesis in which FXIII appears 2/ to establish if FXIII is retained during differentiation into macrophages 3/ to assess how general is the presence of FXIII subunit a in different types of macrophages. FXIII subunit a was immunomorphologically detected i
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Adày, R., A. Szegedi, Z. Nemes, and L. Muszbek. "EXTRAVASAL FIBRIN STABILIZATION BY FACTOR XIII IN LYMPH NODES WITH HODGKIN’S DISEASE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643667.

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The formation of extravasal fibrin deposits in various tumors has been recognized a long time ago and it has been implicated in various aspects of tumor growth. However, no adequate information is available on the nature of intratumoral fibrin. In this study we attempted to find out if fibrin deposit in human lymph nodes with Hodgkin’s disease is stabilized and made resistant to fibrinolysis by factor XIII /FXIII/ of blood coagulation. The two main tasks for FXIII in fibrin stabilization is to attach a^antiplas-min the main phyiological inhibitor of fibrinolysis to fibrin strands and crosslink
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Bryckaert, M. C., A. Wasteson, M. Lindroth, and G. C. Tobelem. "PLATELET DERIVED GROWTH FACTOR (PDGF) BINDS TO HUMAN BONE MARROW FIBROBLASTS AND STIMULATES THEIR PROLIFERATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643494.

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A role for the Platelet Derived Growth Factor (PDGF) has been suggested in the abnormal proliferation of bone marrow fibroblasts occuring during myelofibrosis. To investigate this hypothesis, human bone marrow fibroblasts were isolated, and the cultures were characterized by immunofluorescent staining and electron microscopy. Electron microscopy eliminated the presence of endothelial cells by the absence of Weibel-Palade-Bodies. A positive intra and extra cellular antifibro-nectin staining was observed by immunofluorescent staining. The cultured cells didn’t show any labeling with specific ant
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Xia, Ruihua, Ping Wang, Rongling Chen, and Fei Guo. "One Kind of Macrophages Images Segmentation and Labeling Method." In 2009 2nd International Congress on Image and Signal Processing (CISP). IEEE, 2009. http://dx.doi.org/10.1109/cisp.2009.5303515.

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Kieffer, N., M. Titeux, A. Henri, J. Breton-Gorius, and W. Vainchenker. "MEGAKARYOCYTIC ORIGIN OF PLATELET HLA CLASS I ANTIGEN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643546.

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The existence of HLA class I antigens on human platelets is well established. However, several authors have suggested that platelet HLA antigens are not integral membrane components but are acquired from soluble plasma sources and adsorbed to the platelet surface.In the present study, we used the monoclonal antibody W6/32, directed against a monomorphic epitope of the HLA class I antigen for the immunochemical characterization of platelet HLA. Immunoprecipitation experiments, performed after in vitro metabolic radiolabeling of human platelets revealed a band of molecular weight 44,000 identica
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