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Journal articles on the topic 'Macrovascula'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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1

Bilal, Mahmod Alqudah MD JBIM *. Hamzeh Mahmoud Salman Alhusamia MD Ashraf (mohammad SH) A.odeh MD LaithTaha Al ataileh MD JBIM &. Zaid Ezzat Alawneh MD JBIM. "PREVALENCE OF HYPERTENSION AMONG DIABETIC TYPE 2 PATIENTS ATTENDING MEDICAL CLINIC AT PRINCE HASHEM BIN ABDULLAII HOSPITAL IN AQABA." Indian Journal of Medical Research and Pharmaceutical Sciences 4, no. 6 (2017): 47–54. https://doi.org/10.5281/zenodo.815699.

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<strong>Objectives:</strong> This study aimed to assess the prevalence of hypertension among adult type 2 diabetic patients and to identify risk factors that may affect the development of hypertension in those patients. Methods A cross-sectional study conducted from September 2015 to march 2016 in medical clinic at Prince Hashem bin Abdulla II hospital at Aqaba. A total of 500 diabetic type 2 patients were included in this study .Data obtained directly from patients and patients medical files. <strong>Results:</strong> In our study, prevalence of hypertension was noted in382 (76%) patients. Hy

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Yan, Michael, Zhihao Li, Pablo Munoz-Schuffenegger, et al. "1117: COMPARING RESECTION AND STEREOTACTIC BODY RADIATION THERAPY FOR HCC WITH MACROVASCULA R INVASION." Radiotherapy and Oncology 194 (May 2024): S2187—S2188. http://dx.doi.org/10.1016/s0167-8140(24)01581-0.

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3

Libby, Peter, and Jorge Plutzky. "Diabetic Macrovascular Disease." Circulation 106, no. 22 (2002): 2760–63. http://dx.doi.org/10.1161/01.cir.0000037282.92395.ae.

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4

Kraschinski, S., A. Epple, and B. Nibbio. "Macrovascular dopamine release." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 270, no. 6 (1996): R1244—R1249. http://dx.doi.org/10.1152/ajpregu.1996.270.6.r1244.

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In an animal model, the American eel, perifused elastic arteries and large veins, but not the heart and organs with extensive microvascular supply (gills and opisthonephric kidney), release spontaneously free dopamine. Only the region of the cardinal vein, which contains the adrenomedullary equivalent, also releases norepinephrine (NE) and epinephrine (E). Ca2+, KCl, and E stimulate dopamine release from the ventral aorta and caudal vein, indicating that this phenomenon is due to secretion and not to washout. E also stimulates NE release from the ventral aorta and caudal vein. In the rat, both

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5

El-Alameey, Inas R., Abeer A. Al-Mutairi, Rama D. Alamri, et al. "Predictors of macrovascular complications in Al-Madinah Al-Munawara type 2 diabetes mellitus patients, Saudi Arabia: A cross-sectional study." Journal of The Arab Society for Medical Research 19, no. 1 (2024): 55–62. http://dx.doi.org/10.4103/jasmr.jasmr_23_23.

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Background/aim Diabetes mellitus is a metabolic disease characterized by chronic hyperglycemia, which causes macrovascular complications that account for most of the morbidity, hospitalizations, and deaths. The goal of this study is to evaluate the relationship between macrovascular complications and associated risk factors in Al-Madinah Al-Munawara patients suffering from type 2 diabetes mellitus. Patients and methods This descriptive cross-sectional study involved 275 type 2 diabetes mellitus patients who resided in Al-Madinah Al-Munawara and visited the diabetic clinic at King Fahd Hospital

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Miettinen, Heikki, and Veikko Salomaa. "Diabetes and macrovascular disease." Journal of Cardiovascular Risk 4, no. 2 (1997): 76–82. http://dx.doi.org/10.1097/00043798-199704000-00003.

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7

Miettinen, Heikki, and Veikko Salomaa. "Diabetes and macrovascular disease." Coronary Artery Disease 7, no. 10 (1996): 708–14. http://dx.doi.org/10.1097/00019501-199610000-00003.

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8

Fisher, Miles. "Macrovascular disease in diabetes." Medicine 34, no. 3 (2006): 101–3. http://dx.doi.org/10.1383/medc.2006.34.3.101.

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9

Valabhji, Jonathan, and Robert S. Elkeles. "Macrovascular Disease in Diabetes." Medicine 30, no. 2 (2002): 47–50. http://dx.doi.org/10.1383/medc.30.2.47.28270.

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Miettinen, H., and V. Salomaa. "Diabetes and Macrovascular Disease." European Journal of Cardiovascular Prevention & Rehabilitation 4, no. 2 (1997): 76–82. http://dx.doi.org/10.1177/174182679700400203.

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11

FISHER, M. "Prevention of macrovascular complications." European Heart Journal Supplements 5 (January 2003): B21—B26. http://dx.doi.org/10.1016/s1520-765x(03)90037-x.

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12

Vinik, Aaron, and Mark Flemmer. "Diabetes and macrovascular disease." Journal of Diabetes and its Complications 16, no. 3 (2002): 235–45. http://dx.doi.org/10.1016/s1056-8727(01)00212-4.

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13

Donaghue, Kim C., Francesco Chiarelli, Daniela Trotta, Jeremy Allgrove, and Knut Dahl-Jorgensen. "Microvascular and macrovascular complications." Pediatric Diabetes 8, no. 3 (2007): 163–70. http://dx.doi.org/10.1111/j.1399-5448.2007.00250.x.

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14

ödkvist, L. M., K. å. Thuomas, and M. Niklasson. "Macrovascular Causes Underlying Otoneurological Disturbances." Acta Oto-Laryngologica 115, no. 2 (1995): 145–48. http://dx.doi.org/10.3109/00016489509139278.

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15

Godoi, Emmanuelle Tenório Albuquerque Madruga, Alexandre Domingues Barbosa, Juannicelle Tenório Albuquerque Madruga Godoi, et al. "Envolvimento macrovascular e esclerose sistêmica." Jornal Vascular Brasileiro 8, no. 1 (2009): 65–76. http://dx.doi.org/10.1590/s1677-54492009005000008.

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Este artigo tem como objetivo revisar os aspectos descritos na literatura sobre o acometimento da macrovasculatura na esclerose sistêmica e avaliar a ocorrência e distribuição das alterações macrovasculares nos pacientes com esclerose sistêmica através do eco-Doppler e do índice tornozelo-braço, além da associação desses achados com as características demográficas, forma clínica, tempo de evolução da doença, fenômeno de Raynaud (FR), alterações digitais, ulcerações de membros, reabsorção de falange, amputação, bem como fatores de risco e antecedentes da doença ateromatosa. O estudo foi prospec

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16

Hallock, Geoffrey. "Macrovascular Surgery and the Microsurgeon." Journal of Reconstructive Microsurgery 13, no. 08 (1997): 563–70. http://dx.doi.org/10.1055/s-2007-1006438.

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17

Hsueh, Willa, Ronald E. Law, Mohammed Saad, Jacqueline Dy, Edward Feener, and George King. "Insulin resistance and macrovascular disease." Current Opinion in Endocrinology and Diabetes 3, no. 4 (1996): 346–54. http://dx.doi.org/10.1097/00060793-199608000-00010.

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18

Nuzum, Donald S., and Tonja Merz. "Macrovascular Complications of Diabetes Mellitus." Journal of Pharmacy Practice 22, no. 2 (2009): 135–48. http://dx.doi.org/10.1177/0897190008326444.

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Adults with diabetes mellitus frequently develop macrovascular complications. Vascular disease is a frequent cause of morbidity and mortality among patients with diabetes. Diabetes and vascular diseases remain among the most common causes of death in the United States. A number of clinical trials and practice guidelines have been published addressing the management of macrovascular complications. The cornerstone of preventing or delaying the progression of macrovascular complications of diabetes is aggressive management of hypertension and cholesterol. Angiotensin-converting enzyme inhibitors

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19

Alkaabi, J. K. "Rheumatoid arthritis and macrovascular disease." Rheumatology 42, no. 2 (2003): 292–97. http://dx.doi.org/10.1093/rheumatology/keg083.

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20

Nassabeh, Negin. "Microvascular clues to macrovascular disease." Nature Reviews Rheumatology 5, no. 12 (2009): 655. http://dx.doi.org/10.1038/nrrheum.2009.222.

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21

Ho, M. "Macrovascular disease and systemic sclerosis." Annals of the Rheumatic Diseases 59, no. 1 (2000): 39–43. http://dx.doi.org/10.1136/ard.59.1.39.

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22

Ledet, Thomas, Lene Heickendorff, and Lars M. Rasmussen. "7 Pathology of macrovascular disease." Baillière's Clinical Endocrinology and Metabolism 2, no. 2 (1988): 391–405. http://dx.doi.org/10.1016/s0950-351x(88)80039-9.

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23

Robertson, D. A., P. J. Hale, and M. Nattrass. "8 Macrovascular disease and hyperinsulinaemia." Baillière's Clinical Endocrinology and Metabolism 2, no. 2 (1988): 407–24. http://dx.doi.org/10.1016/s0950-351x(88)80040-5.

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24

Putri, Rr Erni Kusuma, Endang Darmawan, and Dyah Aryani Perwitasari. "Cost of Illness Diabetes Melitus Tipe 2 dan Komplikasinya pada Peserta Jaminan Kesehatan Nasional (JKN) di Rawat Jalan Rumah Sakit Condong Catur Yogyakarta." Pharmacon: Jurnal Farmasi Indonesia 16, no. 2 (2019): 89–101. http://dx.doi.org/10.23917/pharmacon.v16i2.8915.

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Type 2 diabetes mellitus is a chronic disease that requires lifelong therapy and high service costs so that the use of antidiabetic drugs affects the patient's therapeutic costs. The purpose of this study was to determine the cost of outpatient type 2 diabetes mellitus therapy for National Health Insurance (JKN) participants in Condong Catur Hospital Yogyakarta. The research method used is observational. The parameters measured are calculating the average direct medical costs (drug costs, doctor's examination and consultation fees, laboratory costs, medical treatment costs), and comparison bet

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Viigimaa, Margus, Alexandros Sachinidis, Maria Toumpourleka, Konstantinos Koutsampasopoulos, Signe Alliksoo, and Tiina Titma. "Macrovascular Complications of Type 2 Diabetes Mellitus." Current Vascular Pharmacology 18, no. 2 (2020): 110–16. http://dx.doi.org/10.2174/1570161117666190405165151.

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Background: Type 2 diabetes mellitus (T2DM) has emerged as a pandemic. It has different complications, both microvascular and macrovascular. Objective: The purpose of this review is to summarize the different types of macrovascular complications associated with T2DM. Methods: A comprehensive review of the literature was performed to identify clinical studies, which determine the macrovascular complications associated with T2DM. Results: Macrovascular complications of T2DM include coronary heart disease, cardiomyopathy, arrhythmias and sudden death, cerebrovascular disease and peripheral artery

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Celik, Mustafa, Serkan Cerrah, Mahmut Arabul, and Aysen Akalin. "Relation of Asymmetric Dimethylarginine Levels to Macrovascular Disease and Inflammation Markers in Type 2 Diabetic Patients." Journal of Diabetes Research 2014 (2014): 1–6. http://dx.doi.org/10.1155/2014/139215.

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Aim. We aimed to determine the relation of asymmetric dimethyl arginine (ADMA) levels to atherosclerotic vascular disease and inflammation markers in type 2 diabetes.Methods. We recruited 50 type 2 diabetic patients with atherosclerosis, 50 type 2 diabetic patients without atherosclerosis, and 31 healthy control patients into our study. We obtained fasting serum and plasma samples and measured HbA1c, fasting blood glucose, C-peptide, creatinine, total cholesterol, triglycerides, HDL cholesterol, LDL cholesterol, hsCRP, fibrinogen, erythrocyte sedimentation rate, total homocysteine, and ADMA le

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Mohr, David C., Libin Zhang, Julia C. Prentice, et al. "Association of hemoglobin A1c time in range with risk for diabetes complications." BMJ Open Diabetes Research & Care 10, no. 4 (2022): e002738. http://dx.doi.org/10.1136/bmjdrc-2021-002738.

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IntroductionWe assessed the association between hemoglobin A1c time in range (A1c TIR), based on unique patient-level A1c target ranges, with risks of developing microvascular and macrovascular complications in older adults with diabetes.Research design and methodsWe used a retrospective observational study design and identified patients with diabetes from the Department of Veterans Affairs (n=397 634). Patients were 65 years and older and enrolled in Medicare during the period 2004–2016. Patients were assigned to individualized A1c target ranges based on estimated life expectancy and the pres

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Krzyzanowska, Katarzyna, Friedrich Mittermayer, Gerit H. Schernthaner, et al. "Renal Function but Not Asymmetric Dimethylarginine Is Independently Associated with Retinopathy in Type 2 Diabetes." Cardiology Research and Practice 2011 (2011): 1–6. http://dx.doi.org/10.4061/2011/260191.

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Background. Asymmetric dimethylarginine (ADMA) is associated with macrovascular disease and possibly with microangiopathy in type 2 diabetes (T2DM). We tested the hypothesis that ADMA is related to diabetic retinopathy (DR) independently of macrovascular disease.Methods. This cross-sectional study included 127 T2DM patients selected to achieve equal distributions of patients with and without macrovascular disease in the groups with and without DR.Results. Patients with DR had increased ADMA, longer diabetes duration, and reduced glomerular filtration rate (GFR). ADMA correlated with GFR (; ),

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29

Dormandy, John. "The need for outcome studies in type 2 diabetes." British Journal of Diabetes & Vascular Disease 2, no. 1_suppl (2002): S32—S36. http://dx.doi.org/10.1177/1474651402002001s0801.

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Outcome studies use primary end points such as overall mortality or major morbidity to demonstrate that treatments deliver meaningful clinical benefits. Historically it was thought that most of the cardiovascular morbidity due to diabetes was related to microvascular disease, providing a marker for macrovascular disease. In diabetes an outcome study would measure all-cause death, cardiac death and cardiovascular morbidity (end points related to macrovascular disease), whereas conventional trials in diabetes have used surrogate end points, such as blood pressure, microvascular disease (retinopa

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30

Shwetha, T., B. Sri vidya, B. Deekshitha, T. Srihari, Amatul Ali Sameera, and Nazia Lateef Amrohi. "Epidemiological Studies of Microvascular and Macrovasacular Complications in Diabetes Mellitus Type." Journal of Neonatal Surgery 14, no. 12S (2025): 982–88. https://doi.org/10.52783/jns.v14.3451.

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Aim: To perform an observational study on epidemiology of micro and macrovascular complications in diabetes mellitus type II. Objectives: To assess the occurrence and prevalence of micro and macrovascular complications in type 2 diabetes mellitus. Methodology: The study's methodology will be carried out in the internal medicine department of Gleneagles Aware Hospital in Bairamalguda, Saroornagar, Hyderabad, Telangana state. Over 6 month period with 100 patients individuals with microvascular problems. Patients aged 20 to 80 years, included with comorbid conditions in both genders. Patient demo

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Hilkens, Nina A., Charlotte J. J. van Asch, David J. Werring, et al. "Predicting the presence of macrovascular causes in non-traumatic intracerebral haemorrhage: the DIAGRAM prediction score." Journal of Neurology, Neurosurgery & Psychiatry 89, no. 7 (2018): 674–79. http://dx.doi.org/10.1136/jnnp-2017-317262.

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ObjectiveA substantial part of non-traumatic intracerebral haemorrhages (ICH) arises from a macrovascular cause, but there is little guidance on selection of patients for additional diagnostic work-up. We aimed to develop and externally validate a model for predicting the probability of a macrovascular cause in patients with non-traumatic ICH.MethodsThe DIagnostic AngioGRAphy to find vascular Malformations (DIAGRAM) study (n=298; 69 macrovascular cause; 23%) is a prospective, multicentre study assessing yield and accuracy of CT angiography (CTA), MRI/ magnetic resonance angiography (MRA) and i

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Wilson, Duncan, Ayokunle Ogungbemi, Gareth Ambler, Ifan Jones, David J. Werring, and Hans R. Jäger. "Developing an algorithm to identify patients with intracerebral haemorrhage secondary to a macrovascular cause." European Stroke Journal 2, no. 4 (2017): 369–76. http://dx.doi.org/10.1177/2396987317732874.

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Introduction Determining the cause of spontaneous (non-traumatic) intracerebral haemorrhage (ICH) is critical to guide treatment and prognosis. We investigated whether small vessel disease (SVD) in addition to clinical and other radiological findings on acute neuroimaging predicts a low risk of a macrovascular cause (e.g. an arterio-venous malformation, aneurysm or dural arteriovenous fistula). Patients and methods We identified patients with acute spontaneous ICH who underwent acute non-contrast CT, CT angiography (CTA) and intra-arterial digital subtraction angiography (IADSA) at our institu

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Lee, Wen-Chin, Wei-Hung Kuo, Sin-Hua Moi, Barry Chiu, Jin-Bor Chen, and Cheng-Hong Yang. "Associations between Circulating Markers of Cholesterol Homeostasis and Macrovascular Events among Patients Undergoing Hemodialysis." Nutrients 13, no. 3 (2021): 1014. http://dx.doi.org/10.3390/nu13031014.

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Current strategies targeting serum cholesterol bring limited benefits to mortality and macrovascular events prevention among hemodialysis patients. Direct measurements and analysis on circulating markers of cholesterol homeostasis could be promising solutions to this bottleneck. We prospectively enrolled 90 maintenance hemodialysis patients and 9 healthy controls in 2019 for 1 year. We measured circulating desmosterol and lathosterol as markers for cholesterol synthesis and campesterol and sitosterol for cholesterol absorption. At baseline, hemodialysis patients showed higher levels of campest

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34

Çakmak, Güzin, Sencer Ganidağlı, Eyyüp Efendioğlu, Ercüment Öztürk, and Zeynel Öztürk. "Do Long-Term Complications of Type 2 Diabetes Increase Susceptibility to Geriatric Syndromes in Older Adults?" Medicina 57, no. 9 (2021): 968. http://dx.doi.org/10.3390/medicina57090968.

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Background and Objectives: Type 2 diabetes is one of the common chronic diseases in the elderly. It is thought that long-term complications of type 2 diabetes will negatively affect the quality of life in elderly individuals. It is possible that geriatric syndromes, especially frailty syndrome, are associated with diabetic complications, too. In this study, we aimed to evaluate the effect of macrovascular and microvascular complications of type 2 diabetes on frailty and other geriatric syndromes. In addition, the effect of these complications on quality of life was also reviewed. Materials and

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Lunder, Mojca, Miodrag Janić, and Mišo Šabovič. "Prevention of Vascular Complications in Diabetes Mellitus Patients: Focus on the Arterial Wall." Current Vascular Pharmacology 17, no. 1 (2018): 6–15. http://dx.doi.org/10.2174/1570161116666180206113755.

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In Diabetes Mellitus (DM), hyperglycaemia and insulin resistance progressively lead to both microvascular and macrovascular complications. Whereas the incidence of microvascular complications is closely related to tight glycaemic control, this does not apply to macrovascular complications. Hyperglycaemia influences many interweaving molecular pathways that initially lead to increased oxidative stress, increased inflammation and endothelial dysfunction. The latter represents the initial in both types of vascular complications; it represents the “obligatory damage” in microvascular complications

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36

Geskin, Gennady, Michael D. Mulock, Nicole L. Tomko, Anna D’Asta, and Sandeep Gopalakrishnan. "Effects of Lower Limb Revascularization on the Microcirculation of the Foot: A Retrospective Cohort Study." Diagnostics 12, no. 6 (2022): 1320. http://dx.doi.org/10.3390/diagnostics12061320.

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Background: Current assessment standards in chronic limb-threatening ischemia (CLTI) focus on macrovascular function while neglecting the microcirculation. Multispectral near-infrared spectroscopy (NIRS) provides hemodynamic characteristics of the microcirculation (i.e., capillaries) and may be a powerful tool for monitoring CLTI and preventing extremity loss. The aims of this study were to (1) investigate the effects of lower limb revascularization on the microcirculation and (2) determine if macrovascular and microvascular assessments correlate. Methods: An observational, retrospective cohor

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37

Fowler, M. J. "Microvascular and Macrovascular Complications of Diabetes." Clinical Diabetes 26, no. 2 (2008): 77–82. http://dx.doi.org/10.2337/diaclin.26.2.77.

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Fowler, M. J. "Microvascular and Macrovascular Complications of Diabetes." Clinical Diabetes 29, no. 3 (2011): 116–22. http://dx.doi.org/10.2337/diaclin.29.3.116.

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39

Arora, S., and FW LoGerfo. "Lower extremity macrovascular disease in diabetes." Journal of the American Podiatric Medical Association 87, no. 7 (1997): 327–31. http://dx.doi.org/10.7547/87507315-87-7-327.

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Lower extremity macrovascular disease is more common and progresses more rapidly in the presence of diabetes and has a characteristic peritibial distribution with sparing of the foot arteries. The biology of the diabetic foot is compromised, thereby making it more susceptible to injury. Hence, compromises in perfusion have a greater significance, warranting an aggressive approach to revascularization.

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40

Jarrett, R. J. "Macrovascular disease in Caucasoid diabetic patients." Diabetologia 28, no. 10 (1985): 794. http://dx.doi.org/10.1007/bf00265031.

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41

Nankivell, Brian J., Siew-Gek Lau, Jeremy R. Chapman, Philip J. O???Connell, John P. Fletcher, and Richard D. M. Allen. "PROGRESSION OF MACROVASCULAR DISEASE AFTER TRANSPLANTATION1." Transplantation 69, no. 4 (2000): 574–81. http://dx.doi.org/10.1097/00007890-200002270-00019.

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Roesen, P., P. Ferber, and D. Tschoepe. "Macrovascular disease in diabetes: current status." Experimental and Clinical Endocrinology & Diabetes 109, Suppl 2 (2001): S474—S486. http://dx.doi.org/10.1055/s-2001-18603.

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Stafford, L., H. Englert, J. Gover, and J. Bertouch. "Distribution of macrovascular disease in scleroderma." Annals of the Rheumatic Diseases 57, no. 8 (1998): 476–79. http://dx.doi.org/10.1136/ard.57.8.476.

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Vella, Sandro, and John R. Petrie. "Macrovascular disease: pathogenesis and risk assessment." Medicine 38, no. 11 (2010): 626–31. http://dx.doi.org/10.1016/j.mpmed.2010.08.019.

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Vella, Sandro, and John R. Petrie. "Macrovascular disease: pathogenesis and risk assessment." Medicine 43, no. 1 (2015): 1–6. http://dx.doi.org/10.1016/j.mpmed.2014.10.012.

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Vella, Sandro, and John R. Petrie. "Macrovascular disease: pathogenesis and risk assessment." Medicine 47, no. 2 (2019): 65–71. http://dx.doi.org/10.1016/j.mpmed.2018.11.011.

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Hettema, M. E., H. Bootsma, and C. G. M. Kallenberg. "Macrovascular disease and atherosclerosis in SSc." Rheumatology 47, no. 5 (2008): 578–83. http://dx.doi.org/10.1093/rheumatology/ken078.

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Yeung, Jacky, Branden Cord, Timothy O'Rourke, Renee Maina, Samuel Sommaruga, and Charles Matouk. "Macrovascular Lesions Underlying Spontaneous Intracerebral Hemorrhage." Seminars in Neurology 36, no. 03 (2016): 244–53. http://dx.doi.org/10.1055/s-0036-1581994.

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Kucukgul, Can, Burce Ozler, H. Ezgi Karakas, Devrim Gozuacik, and Bahattin Koc. "3D Hybrid Bioprinting of Macrovascular Structures." Procedia Engineering 59 (2013): 183–92. http://dx.doi.org/10.1016/j.proeng.2013.05.109.

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Cianflone, Domenico, Ali A. Rizvi, and Manfredi Rizzo. "Microvascular and macrovascular effects of liraglutide." International Journal of Cardiology 286 (July 2019): 17–18. http://dx.doi.org/10.1016/j.ijcard.2019.02.041.

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