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Journal articles on the topic 'MAG do TBC'

Author: Grafiati

Published: 4 June 2021

Last updated: 2 February 2022

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1

Pereira, João Gabriel Barrêto, Viviane Santos Salazar, and Mariana Cavalcanti Falcão de Albuquerque. "ANÁLISE DA GOVERNANÇA EM UMA EXPERIÊNCIA DE TURISMO DE BASE COMUNITÁRIA: APLICAÇÃO DO MODELO MAG DO TBC À ADESCO / GOVERNANCE ANALYSIS IN A COMMUNITY BASED TOURISM EXPERIENCE: APPLICATION OF THE TBC MAG MODEL TO ADESCO." Brazilian Journal of Development 6, no. 11 (2020): 85908–25. http://dx.doi.org/10.34117/bjdv6n11-130.

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2

Mamdoh T. Jamal and T. A. Bin Alshekh AbuBakar, Mamdoh T. Jamal and T. A. Bin Alshekh AbuBakar. "Shelf-Life of Indian White Shrimp Fenneropenaeus indicus Stored in Different Icing Conditions." journal of king abdulaziz university marine science 26, no. 2 (March 20, 2017): 45–60. http://dx.doi.org/10.4197/mar.26-2.5.

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The Shelf-life of farmed Indian white shrimp Fenneropenaeus indicus, formerly Penaeus indicus stored in ice using three different ratios of ice for 22 days was evaluated. During the storage period, changes in sensory characteristics, TMA-N levels, pH values and total bacterial count (TBC) were investigated, where the results showed a clear deterioration in the appearance, texture and odor of F. indicus from the 10th day in the samples stored at a shrimp to ice ratio of 1:1 and from the 12th day in the samples stored at a ratio of 1:2, while no such deterioration was observed in the samples stored at ratio of 1:3 until the 18th day of storage. The TMA-N levels increased with time during the icing storage. The TBC of the normal flora in the flesh of F. indicus increased with the time of storage. Changes in the sensory characteristics were associated with the changes in TMAN, pH and TBC, showing that the 1:2 ratio did not significantly extend the shrimp shelf-life compared to 1:1, whereas 1:3 significantly delayed the deterioration rate of quality, providing the longest shelf-life and the highest quality shrimp. The shelf-life extended to 9, 11 and 17 days at ratios of 1:1, 1:2 and 1:3 (shrimp to ice), respectively. Therefore, for storage purposes in ice, using a large amount of ice at a 1:3 ratio is recommended, as it extended the shelf-life to the greatest extent in this study.

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3

Lucas, Keith, and Bill Rassell. "HDB-MAC A New Proposal for High Definition TV Transmission." IEEE Transactions on Broadcasting BC-33, no. 4 (December 1987): 170–83. http://dx.doi.org/10.1109/tbc.1987.266650.

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4

Zhao Li and T. Herfet. "MAC Layer Multicast Error Control for IPTV in Wireless LANs." IEEE Transactions on Broadcasting 55, no. 2 (June 2009): 353–62. http://dx.doi.org/10.1109/tbc.2009.2016502.

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5

Kim, Chang Woo, Jae Myung Cha, and Min Seob Kwak. "Identification of Potential Biomarkers and Biological Pathways for Poor Clinical Outcome in Mucinous Colorectal Adenocarcinoma." Cancers 13, no. 13 (June 30, 2021): 3280. http://dx.doi.org/10.3390/cancers13133280.

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Colorectal cancer (CRC) comprises several histological subtypes, but the influences of the histological subtypes on prognosis remains unclear. We sought to evaluate the prognosis of mucinous adenocarcinoma (MAC), compared to that of traditional adenocarcinoma (TAC). This study used the data of patients diagnosed with CRC between 2004 and 2016, as obtained from the Surveillance, Epidemiology, and End Results database. We established a predictive model for disease-specific survival using conditional survival forest, model, non-linear Cox proportional hazards, and neural multi-task logistic regression model and identified the gene signatures for predicting poor prognosis based on the arrayexpress datasets. In total, 9096 (42.1%) patients with MAC and 12,490 (58.9%) patients with TAC were included. Those with the MAC subtype were more likely to have a poorer overall survival rate compared to those with the TAC subtype in stage II CRC (p = 0.002). The eight major genes including RPS18, RPL30, NME2, USP33, GAB2, RPS3A, RPS25, and CEP57 were found in the interacting network pathway. MAC was found to have a poorer prognosis compared to TAC, especially in Stage II CRC. In addition, our findings suggest that identifying potential biomarkers and biological pathways can be useful in CRC prognosis.

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6

Tu, Lai, and Chung-Ming Huang. "Collaborative Content Fetching Using MAC Layer Multicast in Wireless Mobile Networks." IEEE Transactions on Broadcasting 57, no. 3 (September 2011): 695–706. http://dx.doi.org/10.1109/tbc.2011.2147010.

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7

Bryksina, E. Y., V. S. Bryksin, and A. V. Pochivalov. "Incidence, pathomorphism and outcomes of the bronchopulmonary dysplasia associated with microaspiration of gastric contents." Annals of the Russian academy of medical sciences 71, no. 2 (February 6, 2016): 128–40. http://dx.doi.org/10.15690/vramn613.

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Today the influence of the digestive tract functional violations followed by microaspiration of gastric contents (MAGC) on the incidence, features and outcomes of bronchopulmonary dysplasia BPD) remains little studied. Focusing on this aspect makes the research actual.Objective: determination of the nature of influence of MAGC on the progress and course of BPD.Methods: 373 newborns exposed to artificial pulmonary ventilation (APV) in the neonatal period were examined. In a tracheobronchial aspirate (TBA) the marker of MAGC – pepsin –was determined. Its activity was measured by extinction value with subsequent analysis of the incidence and nature of the course of bronchopulmonary dysplasia (BPD) in patients against MAGC and without it. During the three years follow-up period outcomes of BPD and features of combined pathology were established.Results: it was revealed that in children suffered from MAGC the incidence of BPD was higher and grew in proportion to the increase of pepsin activity in TBA and the reduction of gestational age. The extinction increase in TBA values was followed by the increase in duration of APV and the subsequent oxygen therapy, and also the severity of BPD clinical course. By the end of 3 years clinical recovery was detected in 55,2% of children suffering from BPD without MAGC whereas in patients with BPD against MAGC this outcome occured only in 0,9% of cases.Conclusion: MAGC is clinically significant for the etiology, pathogenesis and pathomorphism of BPD. The proposed method of early detection of MAGC and algorithm of complex therapy can reduce its severity and improve the forecast accuracy of neonatal adaptation.

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8

Ghazisaidi, Navid, Martin Maier, and Martin Reisslein. "VMP: A MAC Protocol for EPON-Based Video-Dominated FiWi Access Networks." IEEE Transactions on Broadcasting 58, no. 3 (September 2012): 440–53. http://dx.doi.org/10.1109/tbc.2012.2191692.

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9

Matsuoka, M., T. Hattori, T. Chosa, H. Tsuda, S. Kuwata, M. Yoshida, T. Uchiyama, and K. Takatsuki. "T3 surface molecules on adult T cell leukemia cells are modulated in vivo." Blood 67, no. 4 (April 1, 1986): 1070–76. http://dx.doi.org/10.1182/blood.v67.4.1070.1070.

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Abstract Cells from eight patients with adult T cell leukemia (ATL) and from four patients with non-ATL were examined to see if the T3 antigen of these cells could be modulated in vitro. We found a low density of T3 antigen and the presence of Tac antigen on cells from all patients with ATL. The density of T3 antigen on non-ATL cells was normal, and Tac antigen was not detected. Modulation of T3 antigen and an increase in Tac antigen-positive cells occurred when cells from patients with T4 non-ATL were cultured with OKT3 monoclonal antibody (mAb). Those changes in T3 antigen density and the appearance of Tac antigen-bearing cells by OKT3 mAb were not so marked when ATL cells were used. But the modulation of T3 antigen and the increase in Tac antigen-bearing cells by OKT3 mAb were closely related in cells from six ATL patients. These findings suggest that T3 T cell antigen receptor complexes on ATL cells are not functionally “frozen” by leukemic changes and might be modulated in vivo. In addition, modulation of T3 surface antigen on ATL cells was not induced by cultivation with human T cell leukemia virus type I particles and envelope proteins obtained by gene technology.

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10

Matsuoka, M., T. Hattori, T. Chosa, H. Tsuda, S. Kuwata, M. Yoshida, T. Uchiyama, and K. Takatsuki. "T3 surface molecules on adult T cell leukemia cells are modulated in vivo." Blood 67, no. 4 (April 1, 1986): 1070–76. http://dx.doi.org/10.1182/blood.v67.4.1070.bloodjournal6741070.

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Cells from eight patients with adult T cell leukemia (ATL) and from four patients with non-ATL were examined to see if the T3 antigen of these cells could be modulated in vitro. We found a low density of T3 antigen and the presence of Tac antigen on cells from all patients with ATL. The density of T3 antigen on non-ATL cells was normal, and Tac antigen was not detected. Modulation of T3 antigen and an increase in Tac antigen-positive cells occurred when cells from patients with T4 non-ATL were cultured with OKT3 monoclonal antibody (mAb). Those changes in T3 antigen density and the appearance of Tac antigen-bearing cells by OKT3 mAb were not so marked when ATL cells were used. But the modulation of T3 antigen and the increase in Tac antigen-bearing cells by OKT3 mAb were closely related in cells from six ATL patients. These findings suggest that T3 T cell antigen receptor complexes on ATL cells are not functionally “frozen” by leukemic changes and might be modulated in vivo. In addition, modulation of T3 surface antigen on ATL cells was not induced by cultivation with human T cell leukemia virus type I particles and envelope proteins obtained by gene technology.

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11

Bousvaros, Athos. "“Tac”kling a Difficult Problem." Journal of Clinical Gastroenterology 45, no. 6 (July 2011): 479–80. http://dx.doi.org/10.1097/mcg.0b013e3182160cb5.

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12

Kang, Kyungtae, Yongwoo Cho, and Heonshik Shin. "Energy-Efficient MAC-Layer Error Recovery for Mobile Multimedia Applications in 3GPP2 BCMCS." IEEE Transactions on Broadcasting 53, no. 1 (March 2007): 338–49. http://dx.doi.org/10.1109/tbc.2007.891699.

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13

Ou Yang and W. Heinzelman. "Modeling and Performance Analysis for Duty-Cycled MAC Protocols with Applications to S-MAC and X-MAC." IEEE Transactions on Mobile Computing 11, no. 6 (June 2012): 905–21. http://dx.doi.org/10.1109/tmc.2011.121.

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14

Kuhn, Marie, Simone O. P. Rossi, John L. Plummer, and Jeremy Raftos. "Topical anaesthesia for minor lacerations: MAC versus TAC." Medical Journal of Australia 164, no. 5 (March 1996): 277–80. http://dx.doi.org/10.5694/j.1326-5377.1996.tb94188.x.

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15

Hamdaoui, B., and K. G. Shin. "OS-MAC: An Efficient MAC Protocol for Spectrum-Agile Wireless Networks." IEEE Transactions on Mobile Computing 7, no. 8 (August 2008): 915–30. http://dx.doi.org/10.1109/tmc.2007.70758.

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16

Patras, Paul, Hessan Feghhi, David Malone, and Douglas J. Leith. "Policing 802.11 MAC Misbehaviours." IEEE Transactions on Mobile Computing 15, no. 7 (July 1, 2016): 1728–42. http://dx.doi.org/10.1109/tmc.2015.2478436.

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17

Nhat Minh, Le, Vo Trong Nhan, Nguyen Thi Nga, Tran Thu Huong, Phung Thi Kim Hue, and Do Thi Thao. "Conjugation of nanomaterials containing rare-earth ion Tb3+ with CD133 monoclonal antibody and its potentials for labeling colon cancer cells (HT-29)." Vietnam Journal of Biotechnology 17, no. 3 (November 28, 2020): 427–33. http://dx.doi.org/10.15625/1811-4989/17/3/14620.

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Nanotechnology is the key technology that brings many important applications in biomedical research.Nanolantanites present high stability, easy fabrication and functionalization. Tb3+ ion-containing nanomaterial, a specific type of nanolantanites, possess great prospects. In addition, cancer stem cells (CSCs) are directlyrelated to drug resistance, metastasis, recurrent cancer, etc. Therefore, CSCs are considered as the target forcancer researching and for discovery of more effective therapies. CD133, a trans-membrane glycoprotein, isone of the typical markers that are found to appear very commonly on the surface of many types of CSCs. Inthis study, CD133 monoclonal antibody (MAb) was cojugated with nanomaterials containing Tb3+. Thecoupling between fluorescented nanomaterials containing Tb3+ ions and CD133 MAb was then incubated withhuman colon cancer cells (HT-29) to evaluate its ability to label CSCs in vitro. The results showed thatnanorods containing rare-earth based Tb3+ ions which were fabricated by hydrothermal method, present thelength of about 300 - 800 nm and the diameter in range of 40 - 50 nm. The Tb3+ nanoparticals also havehexagonal structure of terbium phosphate monohydrate and green illuminant. Tb3+ nanorods were also furthersurface silica coated and amino-silane functionalized. This nanostructure was successfully combined withmonoclonal antibodies against CD133 which labelled the surface marker of HT-29 human colon cancer cells.As a result, the combination of CD133+TbPO4@Silica-NH2 (functionalized surface) showed strongerluminescence than the CD133+TbPO4 unfunctionalized combination.

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18

Tom Hao Luan, Xinhua Ling, and Xuemin Shen. "MAC in Motion: Impact of Mobility on the MAC of Drive-Thru Internet." IEEE Transactions on Mobile Computing 11, no. 2 (February 2012): 305–19. http://dx.doi.org/10.1109/tmc.2011.36.

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19

Nhat Minh, Le, Vo Trong Nhan, Tran Thu Huong, Nguyen Thi Nga, Le Tri Vien, and Phung Thi Kim Hue. "Labeling efficiency of Tb3+ conjugated CD133 monoclonal antibody nanocomplex targeting in vitro metastatic cancer cells." Vietnam Journal of Biotechnology 17, no. 3 (November 28, 2020): 435–40. http://dx.doi.org/10.15625/1811-4989/17/3/14775.

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Efficient cancer treatment remains a huge challenge worldwide. As reported by the World HealthOrganization, the number of cancer patients is estimated to be 24 million in 2035, which is a 70% increasecompared to 14.1 million in 2012. The severity of cancer is due to the presence of cancer stem cells (CSCs),which are directly related to drug resistance, metastasis, and tumor relapse. Because of the unknown location ofthe primary tumor and/or the residency of CSCs, standard therapies deliver a high dose of drugs to the whole body,which can have negative effects and deadly consequences for patients undergoing treatment. Therefore, efficientluminescent materials for labeling and tracking CSCs are urgently needed to determine their distribution andtarget treatment. Herein, a fluorescent Tb3+ nano-ion and CD133 monoclonal antibody (mAb) were conjugatedinto a nano probe-complex (ET2). Tb3+ nano-ion is a rare-earth element and the CD133 mAb targets CD133,which is a CSC surface marker. The Tb3+ nanorods were surface treated with silica and activated with -NH2 forfunctioning before being coupled with CD133 mAb. Strong fluorescent Tb3+ nanorods were used to decrease thetoxicity of high-dose medicines, and the purpose of the CD133 mAb was to increase the specific binding capacityof CSCs to the ET2 nanocomplex. The luminescent properties of this coupled ET2 complex were determinedand its ability to target and label CSCs was determined using the pluripotent human embryonic carcinoma cellline, NTERA-2. Fluorescence microscopy showed strong luminescent signals from ET2-exposed NTERA-2cells. It was also demonstrated that the ET2 nanocomplex effectively labeled up to 97.74% of the tested NTERA-2 cells, but only 2.35% of CCD-18Co human colon normal cells. Therefore, these results show that the ET2luminescent nanocomplex specifically targeted and labeled CSCs, and may be used for further applications infundamental and clinical research.

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20

Mohd Ghazali, Sheikh Ahmad Izaddin Sheikh, Mohd Zobir Hussein, Ri Hanum Yahaya Subhan, and Siti Halimah Sarijo. "Formation of Zinc-Aluminium Layered Double Hydroxide-2,4,5-Tricholorophenoxybutyrate Nanocomposites by Ion Exchange Method." Advanced Materials Research 832 (November 2013): 374–78. http://dx.doi.org/10.4028/www.scientific.net/amr.832.374.

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The intercalation of herbicide, 2,4,5-tricholorophenoxybutyrate (TBA) , into zinc-aluminium-layered double hydroxide (ZAL) for the formation of a new nanocomposite ZAT, was accomplished via anion exchange method. Due to the intercalation of TBA with ZAL interlayer domain, basal spacing expanded from 8.9Å in the ZAL to 23.3 Å in the ZAT. The percentage loading of TBA in the ZAT is 45.5 % (w/w). The FTIR spectra of the nanocomposite shows resemblance peaks of the TBA and Zn-Al-layered double hydroxide indicating the inclusion of TBA into the layered double hydroxide. Surface area of the resulting nanocomposite increased from 1.3 to 15.6 m2g-1with the nitrogen adsorption-desorption of type IV, indicating the mesopore type of material.

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21

Nelson, Rebecca, Janelle Perkins, Jamie Shapiro, Jongphil Kim, Binglin Yue, Claudio Anasetti, and Mohamed A. Kharfan-Dabaja. "Sirolimus, Tacrolimus and Antithymocyte Globulin Are Associated with Improved Overall Survival When Compared to Methotrexate, Tacrolimus and Antithymocyte Globulin in Mismatched Unrelated Allogeneic Hematopoietic Cell Transplantation." Blood 124, no. 21 (December 6, 2014): 2553. http://dx.doi.org/10.1182/blood.v124.21.2553.2553.

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Abstract Background: Sirolimus, an inhibitor of the mammalian target of rapamycin (mTOR), is used in combination with tacrolimus as prophylaxis for acute graft-versus-host-disease (GVHD). Two published randomized trials (one single institution and a multicenter one) failed to demonstrate a survival advantage of sirolimus plus tacrolimus (SIR-TAC) vis-à-vis methotrexate plus tacrolimus (MTX-TAC) when used for acute GVHD prophylaxis in HLA-matched related or unrelated allogeneic hematopoietic cell transplantation. No data exists comparing these two regimens when combined with antithymocyte globulin (ATG) in mismatched unrelated donor (MMUD) allografting. This study aims at comparing the efficacy of SIR-TAC-ATG vs. MTX-TAC-ATG in MMUD allografting. Patients and methods: We retrospectively analyzed 122 patients allografted between 01/2006 and 08/2013 after acute GVHD prophylaxis using SIR-TAC-ATG (n=28) or MTX-TAC-ATG (n=94). All patients received 7.5 mg/kg of rabbit ATG. Conditioning regimens were myeloablative (n=94) or reduced-intensity (n=28). Patient-, disease-, and treatment-related characteristics are summarized in Table 1. Results: Patients receiving SIR-TAC-ATG were older (54 vs. 47 years, p=0.015). The median follow-up from day of cell infusion for all patients was 11.2 (0.5-84.4) months; and for patients in the sirolimus-group or methotrexate-group were 11.5 (1.8-54.9) months and 10.4 (0.5-84.4) months, respectively. The 1-year survival was superior in patients who received SIR-TAC-ATG (83% vs. 57%, p=0.012). The 100-day cumulative incidences of grade 2-4 acute GVHD were 57% in the sirolimus- and 71% in the methotrexate-group, p=0.14. Patients who received methotrexate had over two-fold higher cumulative incidence of NRM (3-year), albeit not statistically significant (37% vs. 16%, p=0.1). There were no difference in median relapse-free survival (RFS) (MTX-TAC-ATG=17.8 months vs. SIR-TAC-ATG=not reached, p=0.21) or cumulative incidence of relapse at 1-year (MTX-TAC-ATG =19% vs. 19%, p= 0.94). Use of rituximab for preemptive treatment of EBV reactivation was higher in the group treated with sirolimus (79% vs. 51%, p= 0.01). Multivariate analyses using Cox proportional-hazard model identified use of sirolimus (HR=0.28 (95%CI=0.10, 0.78), p=0.015) and low CIBMTR disease risk (Hazard ratio (HR) =0.49 (95%CI=0.27, 0.88), p=0.018) as favorable predictors of overall survival (OS); but only low CIBMTR disease risk (HR=0.47 (95%CI=0.22, 0.98), p=0.045) as predictor for lower NRM. Conclusion: This study suggests that combining SIR-TAC-ATG results in improved OS in MMUD allogeneic hematopoietic cell transplantation when compared to MTX-TAC-ATG. These findings need to be confirmed in a prospective randomized controlled trial. Table 1. Variables Sirolimus-based Methotrexate-based p-value Recipient median (range) age (years) 54 (24-67) 47(20-69) 0.015 Recipient gender Male (M) Female (F) M=46% F=54% M=63% F=37% 0.13 Conditioning regimen MAC RIC MAC=75% RIC=25% MAC=78% RIC=22% 0.80 Recipient CMVserology seropositive 75% 70% 0.81 Primary disease Myeloid Lymphoid Others 68% 11% 21% 55% 17% 28% 0.55 CIBMTR disease risk High=18% Int=36% Low=47% High=23% Int=33% Low=44% 0.78 Median CD34 cell dose (x106/kg) 8.5 (3.2-23) 8.6 (1.8-25) 0.53 Rituximab Yes 79% 51% 0.01 MAC regimens: FLU-BU (AUC5300); RIC regimens: FLU-BU (AUC3500), FLU-CY, FLU-MEL, FLU-2GyTBI; Myeloid: AML, CML, MDS; Lymphoid: ALL, CLL Disclosures Off Label Use: sirolimus for prophylaxis for acute Graft-versus-host disease; Antithymocyte globulin for prophylaxis for acute Graft-versus-host disease.

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22

Jeonghoon Mo, H. S. W. So, and J. Walrand. "Comparison of Multichannel MAC Protocols." IEEE Transactions on Mobile Computing 7, no. 1 (January 2008): 50–65. http://dx.doi.org/10.1109/tmc.2007.1075.

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23

Moulik, Soumen, Sudip Misra, and Debayan Das. "AT-MAC: Adaptive MAC-Frame Payload Tuning for Reliable Communication in Wireless Body Area Networks." IEEE Transactions on Mobile Computing 16, no. 6 (June 1, 2017): 1516–29. http://dx.doi.org/10.1109/tmc.2016.2598166.

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24

Kim, Daewoo, Jinhwan Jung, Yoonpyo Koo, and Yung Yi. "Bird-MAC: Energy-Efficient MAC for Quasi-Periodic IoT Applications by Avoiding Early Wake-up." IEEE Transactions on Mobile Computing 19, no. 4 (April 1, 2020): 788–802. http://dx.doi.org/10.1109/tmc.2019.2899572.

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25

Lee, Chang Ho, So Young Kim, Hyoung-Mi Kim, Young Ju Kim, Ji Yoon Kim, and Mi Kyung Kim. "Cochleariform Process Abutment on TBCT in Early Congenital Cholesteatoma." Otology & Neurotology 38, no. 1 (January 2017): 79–85. http://dx.doi.org/10.1097/mao.0000000000001240.

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26

Kyasanur, P., and N. H. Vaidya. "Selfish MAC layer misbehavior in wireless networks." IEEE Transactions on Mobile Computing 4, no. 5 (September 2005): 502–16. http://dx.doi.org/10.1109/tmc.2005.71.

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27

Xue Yang and N. H. Vaidya. "A wireless MAC protocol using implicit pipelining." IEEE Transactions on Mobile Computing 5, no. 3 (March 2006): 258–73. http://dx.doi.org/10.1109/tmc.2006.27.

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28

Peng, Jun, Liang Cheng, and Biplab Sikdar. "A Wireless MAC Protocol with Collision Detection." IEEE Transactions on Mobile Computing 6, no. 12 (December 2007): 1357–69. http://dx.doi.org/10.1109/tmc.2007.1073.

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29

Takeshita, T., Y. Goto, K. Tada, K. Nagata, H. Asao, and K. Sugamura. "Monoclonal antibody defining a molecule possibly identical to the p75 subunit of interleukin 2 receptor." Journal of Experimental Medicine 169, no. 4 (April 1, 1989): 1323–32. http://dx.doi.org/10.1084/jem.169.4.1323.

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A mouse hybridoma cell line, TU27, producing an mAb was established. TU27 mAb reacted with various human and Gibbon ape T cell lines bearing the IL-2R p75 (IL-2Rp75), but not with cell lines expressing only Tac antigen, IL-2Rp55, and numbers of its binding sites on cell surfaces were similar to those of high-affinity IL-2R. Radioimmunoprecipitation with TU27 mAb defined a molecule with a molecular mass of 75 kD on the surface of IL-2Rp75 bearing cells. TU27 mAb completely blocked IL-2 binding to IL-2Rp75 and to the high-affinity IL-2R but not to IL-2Rp55 composing the low-affinity IL-2R. The IL-2-dependent growth of a human T cell line, ILT-Mat, was significantly inhibited by TU27 mAb only at low concentrations of IL-2, and combination of TU27 mAb and H-31 mAb specific for IL-2Rp55 completely inhibited the cell growth even at high concentrations of IL-2. These data strongly suggest that TU27 mAb is specific for the human IL-2Rp75.

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30

Luo, T., M. Motani, and V. Srinivasan. "Cooperative Asynchronous Multichannel MAC: Design, Analysis, and Implementation." IEEE Transactions on Mobile Computing 8, no. 3 (March 2009): 338–52. http://dx.doi.org/10.1109/tmc.2008.109.

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31

Tie Luo, Mehul Motani, and Vikram Srinivasan. "Energy-Efficient Strategies for Cooperative Multichannel MAC Protocols." IEEE Transactions on Mobile Computing 11, no. 4 (April 2012): 553–66. http://dx.doi.org/10.1109/tmc.2011.60.

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32

Sen, S., N. Santhapuri, R. R. Choudhury, and S. Nelakuditi. "Successive Interference Cancellation: Carving Out MAC Layer Opportunities." IEEE Transactions on Mobile Computing 12, no. 2 (February 2013): 346–57. http://dx.doi.org/10.1109/tmc.2012.17.

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33

Shiqiang Wang, Qingyang Song, Xingwei Wang, and A. Jamalipour. "Distributed MAC Protocol Supporting Physical-Layer Network Coding." IEEE Transactions on Mobile Computing 12, no. 5 (May 2013): 1023–36. http://dx.doi.org/10.1109/tmc.2012.69.

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34

Zhu, Yibo, Zheng Peng, Jun-Hong Cui, and Huifang Chen. "Toward Practical MAC Design for Underwater Acoustic Networks." IEEE Transactions on Mobile Computing 14, no. 4 (April 1, 2015): 872–86. http://dx.doi.org/10.1109/tmc.2014.2330299.

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35

Frasao, Beatriz Da Silva, Marion Pereira da Costa, Bruna Leal Rodrigues, Bruno Reis Costa Lima, and Carlos Adam Conte-Junior. "Natural Antioxidant Activity and Compounds Content from Wastes of Euterpe edulis Berries." Journal of Agricultural Science 9, no. 3 (February 13, 2017): 178. http://dx.doi.org/10.5539/jas.v9n3p178.

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The Euterpe edulis (Juçara) is native to Brazil, which berries and wastes present high antioxidant content. Therefore, in this study, microwave-assisted extraction (MAE) was investigated for antioxidant compounds extraction from E. edulis waste and maximized antioxidant activities using response surface methodology coupled with a central composite design. Three factors were observed: microwave power (400/500/600 W), exposition time (30/60/90 sec) and ethanol concentration solvent (40/60/80%). The extracts were characterized by determination of total phenolic (TPC), flavonoids (TFC), monomeric anthocyanins (TAC), tannins content (TTC), and in vitro antioxidant assay (AA%). The yield of TPC, TFC, TAC, and TTC varied at 595.43-2171.34 mg GAE.100 g DM-1, 137.36-251.24 mg QE.100 g DM-1, 179.32-354.38 mg C-3-GE.100 g DM-1 and 0.23-1.00 µg TAE.100 g DM-1, respectively. The optimal MAE parameters for TPC was microwave power 668.18 W, exposition time 110.45 s and aqueous ethanol concentration 93.64%, for TFC same parameters observed; though for TAC the different parameters were 532.28 W, and for TTC 9.55 s. However, for antioxidant activity, the parameters were 668.18 W, 110.45 s time and 64.41% of aqueous ethanol solvent. Therefore, this methodology was successfully applied for optimal extraction of total phenolics, flavonoids, monomeric anthocyanins and tannins from juçara waste and obtain optimal antioxidant activity.

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Espinoza-Delgado, I., J. R. Ortaldo, R. Winkler-Pickett, K. Sugamura, L. Varesio, and D. L. Longo. "Expression and role of p75 interleukin 2 receptor on human monocytes." Journal of Experimental Medicine 171, no. 5 (May 1, 1990): 1821–26. http://dx.doi.org/10.1084/jem.171.5.1821.

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We investigated the expression of IL-2R subunits in human monocytes using the TU27 mAb, which recognizes the p75 chain, and anti-Tac mAb, which recognizes the p55 moiety of the IL-2R. We found that p75 but not p55 is constitutively expressed in more than 90% of fresh human monocytes. Antibody to p75, but not to p55, inhibited the activation of monocytes to a cytotoxic stage induced by IL-2 but did not block IFN-gamma-induced cytotoxicity. Our data demonstrate that the p75 chain is expressed on human monocytes and is involved in the activation of monocytes by IL-2.

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Merlin, Christophe J., and Wendi B. Heinzelman. "Duty Cycle Control for Low-Power-Listening MAC Protocols." IEEE Transactions on Mobile Computing 9, no. 11 (November 2010): 1508–21. http://dx.doi.org/10.1109/tmc.2010.116.

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38

Broustis, Ioannis, Angelos Vlavianos, Prashant Krishnamurthy, and Srikanth V. Krishnamurthy. "MAC Layer Throughput Estimation in Impulse-Radio UWB Networks." IEEE Transactions on Mobile Computing 10, no. 5 (May 2011): 700–715. http://dx.doi.org/10.1109/tmc.2010.203.

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39

Azarfar, Arash, Jean-Francois Frigon, and Brunilde Sanso. "Delay Analysis of Multichannel Opportunistic Spectrum Access MAC Protocols." IEEE Transactions on Mobile Computing 15, no. 1 (January 1, 2016): 92–106. http://dx.doi.org/10.1109/tmc.2015.2409882.

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40

Mach, Jennifer. "Making Connections: MAC Function in Splicing and MicroRNA Biogenesis." Plant Cell 29, no. 10 (October 2017): 2316–17. http://dx.doi.org/10.1105/tpc.17.00786.

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41

Wang, Zijie, Ming Zheng, Haiwei Yang, Zhijian Han, Jun Tao, Hao Chen, Li Sun, et al. "Association of Genetic Variants in CYP3A4, CYP3A5, CYP2C8, and CYP2C19 with Tacrolimus Pharmacokinetics in Renal Transplant Recipients." Current Drug Metabolism 20, no. 7 (August 7, 2019): 609–18. http://dx.doi.org/10.2174/1389200220666190627101927.

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Background: Our study aimed to investigate the pharmacogenetics of cytochrome P3A4 (CYP3A4), CYP3A5, CYP2C8, and CYP2C19 and their influence on TAC Pharmacokinetics (PKs) in short-term renal transplant recipients. Method: A total of 105 renal transplant recipients were enrolled. Target Sequencing (TS) based on next-generation sequencing technology was used to detect all exons, exon/intron boundaries, and flanking regions of CYP3A4, CYP3A5, CYP2C8, and CYP2C19. After adjustment of Minor Allele Frequencies (MAF) and Hardy-Weinberg Equilibrium (HWE) analysis, tagger Single-nucleotide Polymorphisms (SNPs) and haplotypes were identified. Influence of tagger SNPs on TAC concentrations was analyzed. Results: A total of 94 SNPs were identified in TS analysis. Nine tagger SNPs were selected, and two SNPs (rs15524 and rs4646453) were noted to be significantly associated with TAC PKs in short-term post-transplant follow-up. Measurement time points of TAC, body mass index (BMI), usage of sirolimus, and incidence of Delayed Graft Function (DGF) were observed to be significantly associated with TAC PKs. Three haplotypes were identified, and rs15524-rs4646453 was found to remarkably contribute to TAC PKs. Recipients carrying H2/H2 (GG-AA) haplotype also showed significantly high weight- and dose-adjusted TAC concentrations in posttransplant periods of 7, 14, and 30 days and 3 and 6 months. Conclusions: Two tagger SNPs, namely, rs15524 and rs4646453, are significantly related to the variability of TAC disposition, and TAC measurement time points, BMI, usage of sirolimus, and incidence of DGF contribute to this influence. Recipients carrying H2/H2 (GG-AA) haplotype in rs15524–rs4646453 may require a low dosage of TAC during 1-year follow-up posttransplant.

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Egan, C. A., M. Brown, J. D. White, and K. B. Yancey. "Treatment of Epidermolysis Bullosa Acquisita with the Humanized Anti-Tac mAb Daclizumab." Clinical Immunology 101, no. 2 (November 2001): 146–51. http://dx.doi.org/10.1006/clim.2001.5113.

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43

Raya, M., I. Aad, J. P. Hubaux, and A. El Fawal. "DOMINO: Detecting MAC Layer Greedy Behavior in IEEE 802.11 Hotspots." IEEE Transactions on Mobile Computing 5, no. 12 (December 2006): 1691–705. http://dx.doi.org/10.1109/tmc.2006.183.

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Choudhury, R. R., Xue Yang, R. Ramanathan, and N. H. Vaidya. "On designing MAC protocols for wireless networks using directional antennas." IEEE Transactions on Mobile Computing 5, no. 5 (May 2006): 477–91. http://dx.doi.org/10.1109/tmc.2006.69.

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Cicconetti, Claudio, Alessandro Erta, Luciano Lenzini, and Enzo Mingozzi. "Performance Evaluation of the IEEE 802.16 MAC for QoS Support." IEEE Transactions on Mobile Computing 6, no. 1 (January 2007): 26–38. http://dx.doi.org/10.1109/tmc.2007.250669.

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Salameh, H. A. B., M. M. Krunz, and O. Younis. "MAC Protocol for Opportunistic Cognitive Radio Networks with Soft Guarantees." IEEE Transactions on Mobile Computing 8, no. 10 (October 2009): 1339–52. http://dx.doi.org/10.1109/tmc.2009.19.

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Debroy, Saptarshi, Swades De, and Mainak Chatterjee. "Contention Based Multichannel MAC Protocol for Distributed Cognitive Radio Networks." IEEE Transactions on Mobile Computing 13, no. 12 (December 1, 2014): 2749–62. http://dx.doi.org/10.1109/tmc.2014.2352260.

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Johnson, Matthew P., Brian Phelan, Amotz Bar-Noy, Prithwish Basu, and Ram Ramanathan. "Minimum-Cost Network-Wide Broadcast over Reliable MAC-Layer Multicast." IEEE Transactions on Mobile Computing 16, no. 12 (December 1, 2017): 3390–402. http://dx.doi.org/10.1109/tmc.2016.2627035.

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Shi, Xiaolei, and Guido Stromberg. "SyncWUF: An Ultra Low-Power MAC Protocol for Wireless Sensor Networks." IEEE Transactions on Mobile Computing 6, no. 1 (January 2007): 115–25. http://dx.doi.org/10.1109/tmc.2007.250675.

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I-Sheng Liu, F. Takawira, and Hong-Jun Xu. "A Hybrid Token-CDMA MAC Protocol for Wireless Ad Hoc Networks." IEEE Transactions on Mobile Computing 7, no. 5 (May 2008): 557–69. http://dx.doi.org/10.1109/tmc.2007.70744.

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