Log in

Relevant bibliographies by topics / Malaria – immunology / Journal articles

To see the other types of publications on this topic, follow the link: Malaria – immunology.

Journal articles on the topic 'Malaria – immunology'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 January 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 50 journal articles for your research on the topic 'Malaria – immunology.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse journal articles on a wide variety of disciplines and organise your bibliography correctly.

1

PERLMANN, P., and M. TROYE-BLOMBERG. "Malaria Immunology." Revista do Instituto de Medicina Tropical de São Paulo 44, no. 4 (2002): 202. http://dx.doi.org/10.1590/s0036-46652002000400012.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Olatunde, Adesola C., Douglas H. Cornwall, Marshall Roedel, and Tracey J. Lamb. "Mouse Models for Unravelling Immunology of Blood Stage Malaria." Vaccines 10, no. 9 (2022): 1525. http://dx.doi.org/10.3390/vaccines10091525.

Full text

Abstract:

Malaria comprises a spectrum of disease syndromes and the immune system is a major participant in malarial disease. This is particularly true in relation to the immune responses elicited against blood stages of Plasmodium-parasites that are responsible for the pathogenesis of infection. Mouse models of malaria are commonly used to dissect the immune mechanisms underlying disease. While no single mouse model of Plasmodium infection completely recapitulates all the features of malaria in humans, collectively the existing models are invaluable for defining the events that lead to the immunopathog

APA, Harvard, Vancouver, ISO, and other styles

3

Roy, Arati. "Immunology of malaria." Indian Journal of Pediatrics 52, no. 3 (1985): 269–73. http://dx.doi.org/10.1007/bf02754856.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

STEVENSON, M. M., and F. ZAVALA. "Immunology of malaria infections." Parasite Immunology 28, no. 1-2 (2006): 1–4. http://dx.doi.org/10.1111/j.1365-3024.2006.00797.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Targett, G. A. T., and R. F. Anders. "Workshop 1P: Malaria immunology." International Journal for Parasitology 17, no. 5 (1987): 1013–14. http://dx.doi.org/10.1016/0020-7519(87)90208-6.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Laurens, Matthew B. "The Immunologic Complexity of Growing Up with Malaria—Is Scientific Understanding Coming of Age?" Clinical and Vaccine Immunology 23, no. 2 (2015): 80–83. http://dx.doi.org/10.1128/cvi.00697-15.

Full text

Abstract:

ABSTRACTIn the current issue ofClinical and Vaccine Immunology, Mandala et al. report changes in lymphocyte populations in children with uncomplicated malaria, severe malarial anemia, and cerebral malaria compared to controls (W. L. Mandala et al., Clin Vaccine Immunol 23:95–103, 2016,http://dx.doi.org/10.1128/CVI.00564-15). This commentary discusses the importance of understanding both helpful and detrimental aspects of the antimalarial immune response that are critical to malaria vaccine development and considers how these responses may relate to antimalarial vaccine safety and efficacy.

APA, Harvard, Vancouver, ISO, and other styles

7

Mandala, Wilson L., Chisomo L. Msefula, Esther N. Gondwe, et al. "Lymphocyte Perturbations in Malawian Children with Severe and Uncomplicated Malaria." Clinical and Vaccine Immunology 23, no. 2 (2015): 95–103. http://dx.doi.org/10.1128/cvi.00564-15.

Full text

Abstract:

ABSTRACTLymphocytes are implicated in immunity and pathogenesis of severe malaria. Since lymphocyte subsets vary with age, assessment of their contribution to different etiologies can be difficult. We immunophenotyped peripheral blood from Malawian children presenting with cerebral malaria, severe malarial anemia, and uncomplicated malaria (n= 113) and healthy aparasitemic children (n= 42) in Blantyre, Malawi, and investigated lymphocyte subset counts, activation, and memory status. Children with cerebral malaria were older than those with severe malarial anemia. We found panlymphopenia in chi

APA, Harvard, Vancouver, ISO, and other styles

8

Tran, Tuan M., Babru Samal, Ewen Kirkness, and Peter D. Crompton. "Systems immunology of human malaria." Trends in Parasitology 28, no. 6 (2012): 248–57. http://dx.doi.org/10.1016/j.pt.2012.03.006.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Plebanski, Magdalena, and Adrian VS Hill. "The immunology of malaria infection." Current Opinion in Immunology 12, no. 4 (2000): 437–41. http://dx.doi.org/10.1016/s0952-7915(00)00117-5.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Budiningsih, Insani, Jaap M. Middeldorp, Yoes Prijatna Dachlan, and Usman Hadi. "Epstein-Barr Virus and Malaria Interactions: Immunology Perspective." HAYATI Journal of Biosciences 29, no. 6 (2022): 824–33. http://dx.doi.org/10.4308/hjb.29.6.824-833.

Full text

Abstract:

Epstein-Barr Virus can cause various diseases, from acute inflammatory diseases such as fatal or chronic EBV infection, infectious mononucleosis as well as lymphoid and epithelial cancer, various autoimmune diseases, and also could interact with malaria. As EBV infects 95% of the world population, and more than 30% are infected with the protozoan parasite, with more than 500,000 deaths due to malaria cases. It is important to understand how EBV dysregulates the immune system, especially when the virus is interacting with other pathogens such as malaria parasites, causing more severe conditions

APA, Harvard, Vancouver, ISO, and other styles

11

Jones, Trevor R., and Stephen L. Hoffman. "Immunology and pathogenic mechanisms of malaria." Current Opinion in Infectious Diseases 5, no. 3 (1992): 310–18. http://dx.doi.org/10.1097/00001432-199206000-00002.

Full text

APA, Harvard, Vancouver, ISO, and other styles

12

Crutcher, James M., Trevor R. Jones, and Stephen L. Hoffman. "Immunology, pathophysiology, and treatment of malaria." Current Opinion in Infectious Diseases 7, no. 5 (1994): 529–35. http://dx.doi.org/10.1097/00001432-199410000-00002.

Full text

APA, Harvard, Vancouver, ISO, and other styles

13

Antonelli, Lis R., Caroline Junqueira, Joseph M. Vinetz, Douglas T. Golenbock, Marcelo U. Ferreira, and Ricardo T. Gazzinelli. "The immunology of Plasmodium vivax malaria." Immunological Reviews 293, no. 1 (2019): 163–89. http://dx.doi.org/10.1111/imr.12816.

Full text

APA, Harvard, Vancouver, ISO, and other styles

14

Hafalla, Julius Clemence, Olivier Silvie, and Kai Matuschewski. "Cell biology and immunology of malaria." Immunological Reviews 240, no. 1 (2011): 297–316. http://dx.doi.org/10.1111/j.1600-065x.2010.00988.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

15

Tripet, Frédéric, Fred Aboagye-Antwi, and Hilary Hurd. "Ecological immunology of mosquito–malaria interactions." Trends in Parasitology 24, no. 5 (2008): 219–27. http://dx.doi.org/10.1016/j.pt.2008.02.008.

Full text

APA, Harvard, Vancouver, ISO, and other styles

16

Weiss, Walter R., and Stephen L. Hoffman. "Immunology, pathophysiology, and treatment of malaria." Current Opinion in Infectious Diseases 3, no. 3 (1990): 409–13. http://dx.doi.org/10.1097/00001432-199006000-00016.

Full text

APA, Harvard, Vancouver, ISO, and other styles

17

Jones, Trevor R., Mark J. Davis, and Stephen L. Hoffman. "Immunology, pathophysiology, and treatment of malaria." Current Opinion in Infectious Diseases 4, no. 3 (1991): 265–72. http://dx.doi.org/10.1097/00001432-199106000-00002.

Full text

APA, Harvard, Vancouver, ISO, and other styles

18

Burté, Florence, Biobele J. Brown, Adebola E. Orimadegun, et al. "Circulatory hepcidin is associated with the anti-inflammatory response but not with iron or anemic status in childhood malaria." Blood 121, no. 15 (2013): 3016–22. http://dx.doi.org/10.1182/blood-2012-10-461418.

Full text

APA, Harvard, Vancouver, ISO, and other styles

19

Muerhoff, A. Scott, Larry G. Birkenmeyer, Ruthie Coffey, et al. "Detection of Plasmodium falciparum, P. vivax, P. ovale, and P. malariae Merozoite Surface Protein 1-p19 Antibodies in Human Malaria Patients and Experimentally Infected Nonhuman Primates." Clinical and Vaccine Immunology 17, no. 10 (2010): 1631–38. http://dx.doi.org/10.1128/cvi.00196-10.

Full text

Abstract:

ABSTRACT Approximately 3.2 billion people live in areas where malaria is endemic, and WHO estimates that 350 to 500 million malaria cases occur each year worldwide. This high prevalence, and the high frequency of international travel, creates significant risk for the exportation of malaria to countries where malaria is not endemic and for the introduction of malaria organisms into the blood supply. Since all four human infectious Plasmodium species have been transmitted by blood transfusion, we sought to develop an enzyme-linked immunosorbent assay (ELISA) capable of detecting antibodies elici

APA, Harvard, Vancouver, ISO, and other styles

20

Lyke, Kirsten E., Robin B. Burges, Yacouba Cissoko, et al. "HLA-A2 Supertype-Restricted Cell-Mediated Immunity by Peripheral Blood Mononuclear Cells Derived from Malian Children with Severe or Uncomplicated Plasmodium falciparum Malaria and Healthy Controls." Infection and Immunity 73, no. 9 (2005): 5799–808. http://dx.doi.org/10.1128/iai.73.9.5799-5808.2005.

Full text

Abstract:

ABSTRACT Understanding HLA-restricted adaptive host immunity to defined epitopes of malarial antigens may be required for the development of successful malaria vaccines. Fourteen epitopes of preerythrocytic malarial antigens known to mediate cytotoxic T-lymphocyte responses against target cells expressing HLA-A2-restricted epitopes were synthesized and pooled based on antigen: thrombospondin-related anonymous protein (TRAP), circumsporozoite protein (CSP), and export protein 1 (Exp-1) peptides. HLA-A2 supertype (*0201, *0202, *0205, *6802) peripheral blood mononuclear cells collected from 774

APA, Harvard, Vancouver, ISO, and other styles

21

Newmark, Peter. "Colombian immunology: More plans for malaria vaccine." Nature 321, no. 6072 (1986): 721. http://dx.doi.org/10.1038/321721a0.

Full text

APA, Harvard, Vancouver, ISO, and other styles

22

HO, MAY, and H. K. WEBSTER. "Immunology of human malaria. A cellular perspective." Parasite Immunology 11, no. 2 (1989): 105–16. http://dx.doi.org/10.1111/j.1365-3024.1989.tb00652.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

23

Engwerda, Christian. "Malaria immunology: still much more to understand." Trends in Parasitology 21, no. 7 (2005): 310–11. http://dx.doi.org/10.1016/j.pt.2005.05.005.

Full text

APA, Harvard, Vancouver, ISO, and other styles

24

Akanmori, Bartholomew D. "Malaria Immunology and Pathogenesis Consortium (MIMPAC) formed." Trends in Parasitology 17, no. 5 (2001): 215. http://dx.doi.org/10.1016/s1471-4922(01)01962-6.

Full text

APA, Harvard, Vancouver, ISO, and other styles

25

Butcher, G. A. "HIV and malaria: A lesson in immunology?" Parasitology Today 8, no. 9 (1992): 307–11. http://dx.doi.org/10.1016/0169-4758(92)90104-a.

Full text

APA, Harvard, Vancouver, ISO, and other styles

26

Babakhanyan, Anna, Rose Leke, Freya Fowkes, and Diane Taylor. "Persistence of antibodies targeting B cell epitopes of Plasmodium falciparum VAR2CSA (P3114)." Journal of Immunology 190, no. 1_Supplement (2013): 186.4. http://dx.doi.org/10.4049/jimmunol.190.supp.186.4.

Full text

Abstract:

Abstract P. falciparum infection poses a major health risk for 85 million women during pregnancy, due to placental pathology resulting from the binding of malaria-infected erythrocytes to trophoblasts via the pregnancy-specific parasite adhesin VAR2CSA. We have demonstrated a correlation between high avidity antibodies (Ab) to VAR2CSA and protection from placental malaria. Ab to most malarial adhesins appear to be short lived, requiring exposure to antigen to sustain serum IgG. In the current study, we used serum collected longitudinally from pregnant Cameroonian women receiving antimalarial p

APA, Harvard, Vancouver, ISO, and other styles

27

Segura, Mariela, Christine Matte, Neeta Thawani, Zhong Su, and Mary M. Stevenson. "Modulation of malaria-induced immunopathology by concurrent nematode infection (46.8)." Journal of Immunology 178, no. 1_Supplement (2007): S62. http://dx.doi.org/10.4049/jimmunol.178.supp.46.8.

Full text

Abstract:

Abstract Gastrointestinal nematode infections, highly prevalent in malaria endemic areas, are known to modulate host immune responses to unrelated pathogens. However, the complex relationship between worms and malaria and the impact on malarial incidence vs. severity are unclear. Previously, we observed that C57BL/6 (B6) mice infected with the gastrointestinal nematode Heligosomoides polygyrus (Hp) for 2 wks before blood-stage Plasmodium chabaudi AS (Pc) infection developed significantly higher parasitemia compared to mice infected with Pc alone. Despite this, malaria-induced body weight loss,

APA, Harvard, Vancouver, ISO, and other styles

28

Johora, Fatema Tuj, Mohammad Golam Kibria, Hans-Peter Fuehrer, and Mohammad Shafiul Alam. "A Case of Plasmodium malariae in Bangladesh: A Representation of the Suboptimal Performance of Rapid Diagnostic Approaches in Malaria Elimination Settings." Pathogens 11, no. 10 (2022): 1072. http://dx.doi.org/10.3390/pathogens11101072.

Full text

Abstract:

Plasmodium malariae is a neglected human malaria parasite with low parasitemia that often results in the misdiagnosis and underestimation of the actual disease burden of this pathogen. Microscopy is the best diagnostic tool, despite the fact that rapid diagnostic tests (RDTs) are the best surveillance tool for malaria diagnosis in many rural areas for their ease of use in elimination settings. For parasite antigen detection other than P. falciparum, RDTs depend on essential glycolytic Plasmodium proteins, i.e., Plasmodium lactate dehydrogenase (pLDH) and Plasmodium aldolase (pAldo) antigens. T

APA, Harvard, Vancouver, ISO, and other styles

29

Hart, Geoffrey T., Jakob Theorell, Tuan Tran, et al. "Antibody-dependent NK cell control of Plasmodium falciparum infection." Journal of Immunology 198, no. 1_Supplement (2017): 68.19. http://dx.doi.org/10.4049/jimmunol.198.supp.68.19.

Full text

Abstract:

Abstract Plasmodium falciparum (P.f) infection is a major cause of morbidity and mortality world-wide where symptoms and death occur as a result of the blood stage of the P.f. life cycle. To date, there is no effective blood stage malarial vaccine. Natural Killer (NK) cells are key players in the control of hematopoietic cancers and viral infections, however their role in blood stage malaria is unknown. We undertook a comprehensive analysis of NK cell phenotype and function in a cohort of subjects from a malaria clinical study in Mali, Africa. Using an unbiased analysis of different NK cell su

APA, Harvard, Vancouver, ISO, and other styles

30

McDevitt, Michael A., Jianlin Xie, Shanmugasundaram Ganapathy-Kanniappan, et al. "A critical role for the host mediator macrophage migration inhibitory factor in the pathogenesis of malarial anemia." Journal of Experimental Medicine 203, no. 5 (2006): 1185–96. http://dx.doi.org/10.1084/jem.20052398.

Full text

Abstract:

The pathogenesis of malarial anemia is multifactorial, and the mechanisms responsible for its high mortality are poorly understood. Studies indicate that host mediators produced during malaria infection may suppress erythroid progenitor development (Miller, K.L., J.C. Schooley, K.L. Smith, B. Kullgren, L.J. Mahlmann, and P.H. Silverman. 1989. Exp. Hematol. 17:379–385; Yap, G.S., and M.M. Stevenson. 1991. Ann. NY Acad. Sci. 628:279–281). We describe an intrinsic role for macrophage migration inhibitory factor (MIF) in the development of the anemic complications and bone marrow suppression that

APA, Harvard, Vancouver, ISO, and other styles

31

Lamikanra, Abigail A., Douglas Brown, Alexandre Potocnik, Climent Casals-Pascual, Jean Langhorne, and David J. Roberts. "Malarial anemia: of mice and men." Blood 110, no. 1 (2007): 18–28. http://dx.doi.org/10.1182/blood-2006-09-018069.

Full text

Abstract:

Severe malaria is manifest by a variety of clinical syndromes dependent on properties of both the host and the parasite. In young infants, severe malarial anemia (SMA) is the most common syndrome of severe disease and contributes substantially to the considerable mortality and morbidity from malaria. There is now growing evidence, from both human and mouse studies of malaria, to show that anemia is due not only to increased hemolysis of infected and clearance of uninfected red blood cells (RBCs) but also to an inability of the infected host to produce an adequate erythroid response. In this re

APA, Harvard, Vancouver, ISO, and other styles

32

Ashraf, Shoaib, Areeba Khalid, Arend L. de Vos, Yanfang Feng, Petra Rohrbach, and Tayyaba Hasan. "Malaria Detection Accelerated: Combing a High-Throughput NanoZoomer Platform with a ParasiteMacro Algorithm." Pathogens 11, no. 10 (2022): 1182. http://dx.doi.org/10.3390/pathogens11101182.

Full text

Abstract:

Eradication of malaria, a mosquito-borne parasitic disease that hijacks human red blood cells, is a global priority. Microscopy remains the gold standard hallmark for diagnosis and estimation of parasitemia for malaria, to date. However, this approach is time-consuming and requires much expertise especially in malaria-endemic countries or in areas with low-density malaria infection. Thus, there is a need for accurate malaria diagnosis/parasitemia estimation with standardized, fast, and more reliable methods. To this end, we performed a proof-of-concept study using the automated imaging (NanoZo

APA, Harvard, Vancouver, ISO, and other styles

33

Gordeuk, Victor R., Ishmael Kasvosve, Janneke van Dijk, et al. "Altered Immune Response in Severe Malaria Anemia in Children." Blood 108, no. 11 (2006): 1303. http://dx.doi.org/10.1182/blood.v108.11.1303.1303.

Full text

Abstract:

Abstract We prospectively assessed immune markers in children &lt;6 years with severe malarial anemia (hemoglobin &lt;5.0 g/dL; n = 72) and uncomplicated malaria (n = 69) who presented to Macha Mission Hospital in Zambia’s Southern Province. We also studied 70 children &lt;6 years who presented to well child clinics in Harare, Zimbabwe as controls. Compared to controls, children with uncomplicated malaria had significantly higher temperatures and parasite counts, lower hemoglobin and platelet concentrations, higher plasma levels of interferon-gamma, tumor necrosis factor alpha, and

APA, Harvard, Vancouver, ISO, and other styles

34

Chaisavaneeyakorn, Sujittra, Caroline Othoro, Ya Ping Shi, et al. "Relationship between Plasma Interleukin-12 (IL-12) and IL-18 Levels and Severe Malarial Anemia in an Area of Holoendemicity in Western Kenya." Clinical Diagnostic Laboratory Immunology 10, no. 3 (2003): 362–66. http://dx.doi.org/10.1128/cdli.10.3.362-366.2003.

Full text

Abstract:

ABSTRACT In this study, we investigated whether levels of interleukin-12 (IL-12) and IL-18 in plasma are associated with severe malarial anemia outcomes in an area of holoendemicity in western Kenya. We compared plasma IL-12 and IL-18 levels in six groups of children grouped into the categories aparasitemic, asymptomatic, mild malaria, high-density uncomplicated malaria (UC), moderate malarial anemia (MMA), or severe malarial anemia (SMA). IL-12 levels were significantly reduced in children with SMA (P < 0.05) but not in other groups compared to children in the aparasitemic control group. I

APA, Harvard, Vancouver, ISO, and other styles

35

Chau, Jennifer Y., Caitlin M. Tiffany, Shilpa Nimishakavi, et al. "Malaria-Associated l-Arginine Deficiency Induces Mast Cell-Associated Disruption to Intestinal Barrier Defenses against Nontyphoidal Salmonella Bacteremia." Infection and Immunity 81, no. 10 (2013): 3515–26. http://dx.doi.org/10.1128/iai.00380-13.

Full text

Abstract:

ABSTRACTCoinfection with malaria and nontyphoidalSalmonellaserotypes (NTS) can cause life-threatening bacteremia in humans. Coinfection with malaria is a recognized risk factor for invasive NTS, suggesting that malaria impairs intestinal barrier function. Here, we investigated mechanisms and strategies for prevention of coinfection pathology in a mouse model. Our findings reveal that malarial-parasite-infected mice, like humans, developl-arginine deficiency, which is associated with intestinal mastocytosis, elevated levels of histamine, and enhanced intestinal permeability. Prevention or rever

APA, Harvard, Vancouver, ISO, and other styles

36

Willimann, K., H. Matile, N. A. Weiss, and B. A. Imhof. "In vivo sequestration of Plasmodium falciparum-infected human erythrocytes: a severe combined immunodeficiency mouse model for cerebral malaria." Journal of Experimental Medicine 182, no. 3 (1995): 643–53. http://dx.doi.org/10.1084/jem.182.3.643.

Full text

Abstract:

Cerebral malaria is a fatal complication of infection by Plasmodium falciparum in man. The neurological symptoms that characterize this form of malarial disease are accompanied by the adhesion of infected erythrocytes to the vasculature of the brain. To study this phenomenon in vivo, an acute phase severe combined immunodeficiency (SCID) mouse model was developed in which sequestration of P. falciparum-infected human erythrocytes took place. During acute cerebral malaria in humans, the expression of intercellular adhesion molecule-1 (ICAM-1) is induced in vascular endothelium by inflammatory r

APA, Harvard, Vancouver, ISO, and other styles

37

Cornwall, Douglas, Mellina Srey, Franklin Maloba, et al. "Using wild mice to interrogate immunological mechanisms of asymptomatic malaria." Journal of Immunology 204, no. 1_Supplement (2020): 231.27. http://dx.doi.org/10.4049/jimmunol.204.supp.231.27.

Full text

Abstract:

Abstract Malaria is a significant world health problem, with billions of people at risk and over 200 million clinical cases per year that result in over 200,000 deaths. However, the majority of the population in malaria endemic areas (&gt;60%) is asymptomatic (without overt symptoms), even in high transmission areas. Although identified by Plasmodium-infected red blood cells in the circulation, the term asymptomatic malaria is a misnomer with individuals experiencing mild anemia, vascular activation and increased susceptibility to co-morbidities such as non-typhoidal Salmonella infections.

APA, Harvard, Vancouver, ISO, and other styles

38

Fonjungo, Peter N., Ibrahim M. Elhassan, David R. Cavanagh, et al. "A Longitudinal Study of Human Antibody Responses toPlasmodium falciparum Rhoptry-Associated Protein 1 in a Region of Seasonal and Unstable Malaria Transmission." Infection and Immunity 67, no. 6 (1999): 2975–85. http://dx.doi.org/10.1128/iai.67.6.2975-2985.1999.

Full text

Abstract:

ABSTRACT Rhoptry-associated protein 1 (RAP1) of Plasmodium falciparum is a nonpolymorphic merozoite antigen that is considered a potential candidate for a malaria vaccine against asexual blood stages. In this longitudinal study, recombinant RAP1 (rRAP1) proteins with antigenicity similar to that of P. falciparum-derived RAP1 were used to analyze antibody responses to RAP1 over a period of 4 years (1991 to 1995) of 53 individuals naturally exposed to P. falciparum malaria. In any 1 year during the study, between 23 and 39% of individuals who had malaria developed immunoglobulin G (IgG) antibodi

APA, Harvard, Vancouver, ISO, and other styles

39

Loiseau, Claire, Martha M. Cooper, and Denise L. Doolan. "Deciphering host immunity to malaria using systems immunology." Immunological Reviews 293, no. 1 (2019): 115–43. http://dx.doi.org/10.1111/imr.12814.

Full text

APA, Harvard, Vancouver, ISO, and other styles

40

Kopecky, Jan. "P. Perlmann, M. Troye-Blomberg (Eds.): Malaria Immunology." Folia Parasitologica 49, no. 4 (2002): 304. http://dx.doi.org/10.14411/fp.2002.056.

Full text

APA, Harvard, Vancouver, ISO, and other styles

41

de Souza, J. Brian, Manohursingh Runglall, Patrick H. Corran, et al. "Neutralization of Malaria Glycosylphosphatidylinositol In Vitro by Serum IgG from Malaria-Exposed Individuals." Infection and Immunity 78, no. 9 (2010): 3920–29. http://dx.doi.org/10.1128/iai.00359-10.

Full text

Abstract:

ABSTRACT Parasite-derived glycosylphosphatidylinositol (GPI) is believed to be a major inducer of the pathways leading to pathology and morbidity during Plasmodium falciparum infection and has been termed a malaria “toxin.” The generation of neutralizing anti-GPI (“antitoxic”) antibodies has therefore been hypothesized to be an important step in the acquisition of antidisease immunity to malaria; however, to date the GPI-neutralizing capacity of antibodies induced during natural Plasmodium falciparum infection has not been evaluated. Here we describe the development of an in vitro macrophage-b

APA, Harvard, Vancouver, ISO, and other styles

42

Wångdahl, Andreas, Katja Wyss, Dashti Saduddin, et al. "Severity of Plasmodium falciparum and Non-falciparum Malaria in Travelers and Migrants: A Nationwide Observational Study Over 2 Decades in Sweden." Journal of Infectious Diseases 220, no. 8 (2019): 1335–45. http://dx.doi.org/10.1093/infdis/jiz292.

Full text

Abstract:

Abstract Background The aim was to assess factors affecting disease severity in imported P. falciparum and non-falciparum malaria. Methods We reviewed medical records from 2793/3260 (85.7%) of all episodes notified in Sweden between 1995 and 2015 and performed multivariable logistic regression. Results Severe malaria according to WHO 2015 criteria was found in P. falciparum (9.4%), P. vivax (7.7%), P. ovale (5.3%), P. malariae (3.3%), and mixed P. falciparum episodes (21.1%). Factors associated with severe P. falciparum malaria were age <5 years and >40 years, origin in nonendemic countr

APA, Harvard, Vancouver, ISO, and other styles

43

Hart, Geoffrey T., Tuan Tran, Jakob Theorell, et al. "A new malaria killer: Fc receptor gamma chain and PLZF identify NK cell subsets that correlate with reduced Plasmodium falciparum parasitemia and increased antibody dependent cellular cytotoxicity against opsonized infected RBCs." Journal of Immunology 200, no. 1_Supplement (2018): 168.17. http://dx.doi.org/10.4049/jimmunol.200.supp.168.17.

Full text

Abstract:

Abstract Plasmodium falciparum (P.f) infection is a major cause of morbidity and mortality world-wide where symptoms and death occur as a result of the blood stage of the P.f. life cycle. To date, there is no effective blood stage malarial vaccine. Natural Killer (NK) cells are key players in the control of hematopoietic cancers and viral infections, however their role in blood stage malaria is unknown. We undertook a comprehensive analysis of NK cell phenotype and function in a cohort of subjects from a malaria clinical study in Mali, Africa. Using an unbiased analysis of different NK cell su

APA, Harvard, Vancouver, ISO, and other styles

44

Kusi, Kwadwo A., Ben A. Gyan, Bamenla Q. Goka, et al. "Levels of Soluble CD163 and Severity of Malaria in Children in Ghana." Clinical and Vaccine Immunology 15, no. 9 (2008): 1456–60. http://dx.doi.org/10.1128/cvi.00506-07.

Full text

Abstract:

ABSTRACT CD163 is an acute-phase-regulated monocyte/macrophage membrane receptor expressed late in inflammation. It is involved in the haptoglobin-mediated removal of free hemoglobin from plasma, has been identified as a naturally soluble plasma glycoprotein with potential anti-inflammatory properties, and is possibly linked to an individual's haptoglobin phenotype. High levels of soluble CD163 (sCD163) in a malaria episode may therefore downregulate inflammation and curb disease severity. In order to verify this, the relationships between sCD163 levels, malaria severity, and selected inflamma

APA, Harvard, Vancouver, ISO, and other styles

45

MITRA, S., B. RAVINDRAN, B. K. DAS, R. K. DAS, P. K. DAS, and R. N. RATH. "Human cerebral malaria: characterization of malarial antibodies in cerebrospinal fluid." Clinical & Experimental Immunology 86, no. 1 (2008): 19–21. http://dx.doi.org/10.1111/j.1365-2249.1991.tb05767.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

46

Dempsey, Laurie A. "Neutralizing malaria." Nature Immunology 17, no. 3 (2016): 229. http://dx.doi.org/10.1038/ni.3404.

Full text

APA, Harvard, Vancouver, ISO, and other styles

47

Romemro, Pedro. "Malaria vaccines." Current Opinion in Immunology 4, no. 4 (1992): 432–41. http://dx.doi.org/10.1016/s0952-7915(06)80035-x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

48

Amador, Roberto, and Manuel E. Patarroyo. "Malaria vaccines." Journal of Clinical Immunology 16, no. 4 (1996): 183–89. http://dx.doi.org/10.1007/bf01541223.

Full text

APA, Harvard, Vancouver, ISO, and other styles

49

Moreno, Alberto, and Manuel E. Patarroyo. "Malaria vaccines." Current Opinion in Immunology 7, no. 5 (1995): 607–11. http://dx.doi.org/10.1016/0952-7915(95)80064-6.

Full text

APA, Harvard, Vancouver, ISO, and other styles

50

Su, Zhong, Mariela Segura, and Mary M. Stevenson. "Reduced Protective Efficacy of a Blood-Stage Malaria Vaccine by Concurrent Nematode Infection." Infection and Immunity 74, no. 4 (2006): 2138–44. http://dx.doi.org/10.1128/iai.74.4.2138-2144.2006.

Full text

Abstract:

ABSTRACT Helminth infections, which are prevalent in areas where malaria is endemic, have been shown to modulate immune responses to unrelated pathogens and have been implicated in poor efficacy of malaria vaccines in humans. We established a murine coinfection model involving blood-stage Plasmodium chabaudi AS malaria and a gastrointestinal nematode, Heligmosomoides polygyrus, to investigate the impact of nematode infection on the protective efficacy of a malaria vaccine. C57BL/6 mice immunized with crude blood-stage P. chabaudi AS antigen in TiterMax adjuvant developed strong protection agai

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!