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Relevant bibliographies by topics / Malaria research/mouse model

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Academic literature on the topic 'Malaria research/mouse model'

Author: Grafiati

Published: 4 June 2021

Last updated: 12 February 2022

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Journal articles on the topic "Malaria research/mouse model"

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Semenya, Amma A., JoAnn S. Sullivan, John W. Barnwell, and W. Evan Secor. "Schistosoma mansoni Infection Impairs Antimalaria Treatment and Immune Responses of Rhesus Macaques Infected with Mosquito-Borne Plasmodium coatneyi." Infection and Immunity 80, no. 11 (2012): 3821–27. http://dx.doi.org/10.1128/iai.00590-12.

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ABSTRACTMalaria and schistosomiasis are the world's two most important parasitic infections in terms of distribution, morbidity, and mortality. In areas wherePlasmodiumandSchistosomaspecies are both endemic, coinfections are commonplace. Mouse models demonstrate that schistosomiasis worsens a malaria infection; however, just as mice and humans differ greatly, the murine-infectingPlasmodiumspecies differ as much from those that infect humans. Research into human coinfections (Schistosoma haematobium-Plasmodium falciparumversusSchistosoma mansoni-P. falciparum) has produced conflicting results.

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Jiang, Ping, Zhishen Xu, Baiquan Xiao, et al. "Hydrogen sulfide protects against the development of experimental cerebral malaria in a C57BL/6 mouse model." Molecular Medicine Reports 16, no. 2 (2017): 2045–50. http://dx.doi.org/10.3892/mmr.2017.6854.

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Sanz, Laura M., M. Belen Jiménez-Díaz, Benigno Crespo, et al. "Cyclopropyl Carboxamides, a Chemically Novel Class of Antimalarial Agents Identified in a Phenotypic Screen." Antimicrobial Agents and Chemotherapy 55, no. 12 (2011): 5740–45. http://dx.doi.org/10.1128/aac.05188-11.

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ABSTRACTMalaria is one of the deadliest infectious diseases in the world, with the eukaryotic parasitePlasmodium falciparumcausing the most severe form of the disease. Discovery of new classes of antimalarial drugs has become an urgent task to counteract the increasing problem of drug resistance. Screening directly for compounds able to inhibit parasite growthinvitrois one of the main approaches the malaria research community is now pursuing for the identification of novel antimalarial drug leads. Very recently, thousands of compounds with potent activity against the parasiteP. falciparumhave

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Snyder, Edward L., and Roger Y. Dodd. "Reducing the Risk of Blood Transfusion." Hematology 2001, no. 1 (2001): 433–42. http://dx.doi.org/10.1182/asheducation-2001.1.433.

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Abstract There are continuing concerns over the safety of the nation's and the world's blood supply. The allogeneic blood supply is tested for antibodies to HIV1/2, HTLVI/II, hepatitis B, hepatitis C (HCV) and syphilis. Testing is also performed for donor ALT (SGOT) levels, for the presence of hepatitis B surface antigen, human immunodeficiency virus (HIV) p24 antigen and, using nucleic acid amplification testing (NAT), for HIV and HCV nucleic acids. Still, there are concerns regarding other pathogenic agents. Dr. Roger Dodd addresses a series of pathogens that are already known to be transmis

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Markus, Miles B. "Mouse-Based Research on Quiescent Primate Malaria Parasites." Trends in Parasitology 32, no. 4 (2016): 271–73. http://dx.doi.org/10.1016/j.pt.2016.02.006.

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Badell, E., V. Pasquetto, P. Druilhe, and N. Van Rooijen. "A mouse model for human malaria erythrocytic stages." Parasitology Today 11, no. 6 (1995): 235–37. http://dx.doi.org/10.1016/0169-4758(95)80088-3.

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Butcher, Geoff. "A mouse model for human malaria erythrocytic stages: Reply." Parasitology Today 11, no. 6 (1995): 224. http://dx.doi.org/10.1016/0169-4758(95)80082-4.

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Kautz, Leon, Chloe Latour, Wlodarczyk Myriam, et al. "Erythroferrone Represses Hepcidin Expression in a Mouse Model of Malaria." Blood 124, no. 21 (2014): 4022. http://dx.doi.org/10.1182/blood.v124.21.4022.4022.

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Abstract Introduction: Malaria, a mosquito-borne disease caused by a parasite, represents a major global health challenge in developing countries, resulting in over half a million deaths each year. Among the many clinical complications, the multiplication of the parasites in erythrocytes leads to a severe anemia secondary to hemolysis and increased erythrophagocytosis. Malarial anemia is also characterized by insufficient erythropoiesis to compensate for the loss of red blood cells, despite high erythropoietin (EPO) levels. Iron is an essential functional component of erythrocyte hemoglobin, t

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Desruisseaux, Mahalia S., FNU Nagajyothi, Shankar Mukherjee, Dumitru A. Iacobas, Herbert B. Tanowitz, and David C. Spray. "Gene expression alterations in a mouse model of cerebral malaria." BMC Proceedings 2, Suppl 1 (2008): P15. http://dx.doi.org/10.1186/1753-6561-2-s1-p15.

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Sacci, J. B., M. E. Schriefer, J. H. Resau, et al. "Mouse model for exoerythrocytic stages of Plasmodium falciparum malaria parasite." Proceedings of the National Academy of Sciences 89, no. 9 (1992): 3701–5. http://dx.doi.org/10.1073/pnas.89.9.3701.

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Dissertations / Theses on the topic "Malaria research/mouse model"

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Gilks, C. F. "The surface of Plasmodium chabaudi infected erythrocytes." Thesis, University of Oxford, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.233501.

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Watkins, Katherine Ruth. "Investigation of pre-erythrocytic malaria vaccines in a mouse model using transgenic parasites." Thesis, University of Oxford, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.534200.

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Achtman, Ariel H. "The B cell response to Plasmodium chabaudi chabaudi : malaria in the mouse model." Thesis, Open University, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.398011.

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Min-Oo, Gundula Ellen. "The genetic basis of malaria susceptibility: uncovering novel host factors in a mouse model of blood-stage infection." Thesis, McGill University, 2009. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=66739.

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This thesis examines the genetic factors controlling host response to malaria. Recombinant congenic strains AcB55 and AcB61 were identified as uniquely resistant to malaria despite a susceptible genetic background. These mice display splenomegaly, anemia, reticulocytosis and extra-medullary erythropoiesis. Genome wide mapping led to the identification of a mutation in pyruvate kinase (PklrI90N) that is associated with increased survival and decreased peak parasite levels. We found a significant malaria-protective effect with a second Pklr mutation (PklrG338D) on a distinct ge

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Wyse, Ana Paula Pintado. "Optimal control for malaria vector for a seasonal mathematical model." Laboratório Nacional de Computação Científica, 2007. http://www.lncc.br/tdmc/tde_busca/arquivo.php?codArquivo=140.

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In the Amazonian region occurs a variation in the malaria incidence, which is related to the pluviometric variation annual. The mathematical model proposed here considers this seasonality and different treatment intensities accessible to the infected people. The numerical evidence the seasonal fluctuation and the relationship between the environment temperature and treatment efficiency, showing that the temperature increase strongly affects the extrinsic latent period,reducing the healthy care efficiency. Because malaria treatment already exists it should be import. For another hand, even the

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Komuro, Yutaro. "Altered adult neurogenesis in a mouse model of human tauopathy." Case Western Reserve University School of Graduate Studies / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=case1434743393.

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Ohashi, Taryn M. "Eradicating Malaria: Improving a Multiple-Timestep Optimization Model of Malarial Intervention Policy." Scholarship @ Claremont, 2013. http://scholarship.claremont.edu/scripps_theses/273.

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Malaria is a preventable and treatable blood-borne disease whose complications can be fatal. Although many interventions exist in order to reduce the impacts of malaria, the optimal method of distributing these interventions in a geographical area with limited resources must be determined. This thesis refines a model that uses an integer linear program and a compartmental model of epidemiology called an SIR model of ordinary differential equations. The objective of the model is to find an intervention strategy over multiple time steps and multiple geographic regions that minimizes the number o

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Huan, Xiang Quan. "Depot cytokines and chemokines for antitumor therapy in a mouse model /." [St. Lucia, Qld.], 2004. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe18435.pdf.

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Krishnamurthy, Varun K. "Biomechanical and Molecular Approaches to Aortic Valve Disease in a Mouse Model." University of Cincinnati / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1354297165.

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Quiggle, David Douglas. "The effects of R-flurbiprofen in reducing tumors in a multiple intestinal neoplasia mouse model." CSUSB ScholarWorks, 2001. https://scholarworks.lib.csusb.edu/etd-project/2009.

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The design of the proposed study was to administer R-FB to 72-day old Min/+ mice for up to 42 days. In order to capture the process of tumor reduction, animals were necropsied at various time points. At each time point animals were evaluated for tumor loads and presence of apoptotic cells along the small intestine. Studies have shown that when R-flurbiprofen (R-FB) is administered in the Min/+ mouse model it can cause the prevention and regression on intestinal tumors.

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Books on the topic "Malaria research/mouse model"

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Brakebusch, Cord. Mouse as a Model Organism: From Animals to Cells. Springer Science+Business Media B.V., 2011.

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Pihlajaniemi, Taina, and Cord Brakebusch. Mouse as a Model Organism: From Animals to Cells. Springer, 2014.

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Bentham, James R. The genetics of congenital heart disease. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, et al. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0022.

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Congenital heart disease (CHD) is defined as a structural cardiac malformation resulting from an abnormality of development; 8% of CHD is inherited in a Mendelian fashion and 12% results from chromosomal imbalance. Recurrence risk and new research suggest that even the remaining 80% of patients without an identifiable familial or syndromic basis for disease may have an identifiable genetic cause. The potential to understand these mechanisms is increasing with the advent of new sequencing techniques which have identified multiple or single rare variants and/or copy number variants clustering in

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Book chapters on the topic "Malaria research/mouse model"

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Peng, Cong, and Shaoguang Li. "CML Mouse Model in Translational Research." In Methods in Molecular Biology. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60761-058-8_15.

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Monach, Paul, Kimie Hattori, Haochu Huang, et al. "The K/BxN Mouse Model of Inflammatory Arthritis." In Arthritis Research. Humana Press, 2007. http://dx.doi.org/10.1007/978-1-59745-402-5_20.

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de Oca, Marcela Montes, Christian Engwerda, and Ashraful Haque. "Plasmodium berghei ANKA (PbA) Infection of C57BL/6J Mice: A Model of Severe Malaria." In Mouse Models of Innate Immunity. Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-481-4_23.

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Kamii, Hideyuki, and Teiji Tominaga. "Filament Perforation Subarachnoid Hemorrhage Mouse Model." In Springer Series in Translational Stroke Research. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-16082-1_16.

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Belayev, Ludmila. "Blood Injection Intracerebral Hemorrhage Mouse Model." In Springer Series in Translational Stroke Research. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-16082-1_21.

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Tada, Yoshiteru, Yasuhisa Kanematsu, Miyuki Kanematsu, et al. "A Mouse Model of Intracranial Aneurysm: Technical Considerations." In Intracerebral Hemorrhage Research. Springer Vienna, 2011. http://dx.doi.org/10.1007/978-3-7091-0693-8_6.

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Kazuki, Y., T. C. Schulz, T. Shinohara, et al. "A new mouse model for Down syndrome." In Advances in Down Syndrome Research. Springer Vienna, 2003. http://dx.doi.org/10.1007/978-3-7091-6721-2_1.

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Brayton, Cory. "Nature and Nurture: Impacts on Mouse Phenotypes and Translational Research." In Mouse as a Model Organism. Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-0750-4_3.

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Manaenko, Anatol, Nancy Fathali, Shammah Williams, Tim Lekic, John H. Zhang, and Jiping Tang. "Geldanamycin Reduced Brain Injury in Mouse Model of Intracerebral Hemorrhage." In Intracerebral Hemorrhage Research. Springer Vienna, 2011. http://dx.doi.org/10.1007/978-3-7091-0693-8_27.

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Cristina, Carolina, Isabel García-Tornadú, Graciela Díaz-Torga, Marcelo Rubinstein, Malcolm J. Low, and Damasia Becú-Villalobos. "Dopaminergic D2 Receptor Knockout Mouse: An Animal Model of Prolactinoma." In Frontiers of Hormone Research. KARGER, 2006. http://dx.doi.org/10.1159/000094308.

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Conference papers on the topic "Malaria research/mouse model"

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Batista, Camila, Helena D’Anunciação De Oliveira, Ana Maria Garcia-Darze, et al. "The effect of combined therapy using rosuvastatin and dihydroarteminin in a pulmonary malaria mouse model." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa4290.

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Keller, C. "Imaging Tumor Vasculature By Computed Tomography in vivo For Mouse Tumour Models." In 2nd International University of Malaya Research Imaging Symposium (UMRIS) 2005: Fundamentals of Molecular Imaging. Department of Biomedical Imaging, University of Malaya, 2005. http://dx.doi.org/10.2349/biij.1.1.e7-52.

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Keller, C. "The Next Generation Of Preclinical Mouse Models Of Human Cancer: A Primer For Radiologists." In 2nd International University of Malaya Research Imaging Symposium (UMRIS) 2005: Fundamentals of Molecular Imaging. Department of Biomedical Imaging, University of Malaya, 2005. http://dx.doi.org/10.2349/biij.1.1.e7-47.

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Miermont, Anne, and Priscilla Furth. "Abstract A6: Stat5a and pregnancy protection in the CERM mouse model." In Abstracts: Frontiers in Cancer Prevention Research 2008. American Association for Cancer Research, 2008. http://dx.doi.org/10.1158/1940-6207.prev-08-a6.

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Melkamu, Tamene, Xuemin Qian, Gerry O'Sullivan, and Fekadu Kassie. "Abstract B41: A mouse model for inflammation-driven lung tumorigenesis." In Abstracts: AACR International Conference on Frontiers in Cancer Prevention Research‐‐ Oct 22-25, 2011; Boston, MA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1940-6207.prev-11-b41.

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Kon, Cynthia Mui Lian, and Jane Labadin. "Simulating the spread of malaria using a generic transmission model for mosquito-borne infectious diseases." In INNOVATIONS THROUGH MATHEMATICAL AND STATISTICAL RESEARCH: Proceedings of the 2nd International Conference on Mathematical Sciences and Statistics (ICMSS2016). Author(s), 2016. http://dx.doi.org/10.1063/1.4952532.

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Shahabi, Shahram, Ebrahim Mazloomi, Behrooz Ilkhanizadeh, and Ahad Zare. "Subcutaneous Isopathic Immunotherapy of Allergic Asthma in a Mouse Model of Allergic Asthma." In HRI London 2019—Cutting Edge Research in Homeopathy: Presentation Abstracts. The Faculty of Homeopathy, 2020. http://dx.doi.org/10.1055/s-0040-1702082.

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Gonzalez-Silva, L., L. Quevedo, T. Moreno, et al. "PO-339 Intratumour heterogeneity in a pancreatic cancer mouse model." In Abstracts of the 25th Biennial Congress of the European Association for Cancer Research, Amsterdam, The Netherlands, 30 June – 3 July 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/esmoopen-2018-eacr25.851.

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Kulkarni, P. V., C. R. Roney, V. Arora, et al. "Radiolabled Polymeric Nanoparticles for Imaging Alzheimer’s Plaques in a Mouse Model of Alzheimer’s Disease (AD)." In APPLICATION OF ACCELERATORS IN RESEARCH AND INDUSTRY: Twentieth International Conference. AIP, 2009. http://dx.doi.org/10.1063/1.3120081.

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Gadeau, Alain-pierre, Laura Cetran, Isabelle Belloc, Marie-ange Renault, Larry Fliegel, and Fatima Mraiche. "In Vivo Characterization Of A New Mouse Model Expressing The Na+/h+ Exchanger Isoform 1 Of Dilated Cardiomyopathy." In Qatar Foundation Annual Research Conference Proceedings. Hamad bin Khalifa University Press (HBKU Press), 2014. http://dx.doi.org/10.5339/qfarc.2014.hbpp0193.

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Reports on the topic "Malaria research/mouse model"

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Rich, Tyvin A., and Sarah Parsons. The Transgenic TGF-Alpha or EGFR1 Overexpression Mouse Model for Symptom Complex Research. Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada533932.

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