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Journal articles on the topic 'Malaria research/mouse model'

Author: Grafiati

Published: 4 June 2021

Last updated: 12 February 2022

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1

Semenya, Amma A., JoAnn S. Sullivan, John W. Barnwell, and W. Evan Secor. "Schistosoma mansoni Infection Impairs Antimalaria Treatment and Immune Responses of Rhesus Macaques Infected with Mosquito-Borne Plasmodium coatneyi." Infection and Immunity 80, no. 11 (2012): 3821–27. http://dx.doi.org/10.1128/iai.00590-12.

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ABSTRACTMalaria and schistosomiasis are the world's two most important parasitic infections in terms of distribution, morbidity, and mortality. In areas wherePlasmodiumandSchistosomaspecies are both endemic, coinfections are commonplace. Mouse models demonstrate that schistosomiasis worsens a malaria infection; however, just as mice and humans differ greatly, the murine-infectingPlasmodiumspecies differ as much from those that infect humans. Research into human coinfections (Schistosoma haematobium-Plasmodium falciparumversusSchistosoma mansoni-P. falciparum) has produced conflicting results.

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2

Jiang, Ping, Zhishen Xu, Baiquan Xiao, et al. "Hydrogen sulfide protects against the development of experimental cerebral malaria in a C57BL/6 mouse model." Molecular Medicine Reports 16, no. 2 (2017): 2045–50. http://dx.doi.org/10.3892/mmr.2017.6854.

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3

Sanz, Laura M., M. Belen Jiménez-Díaz, Benigno Crespo, et al. "Cyclopropyl Carboxamides, a Chemically Novel Class of Antimalarial Agents Identified in a Phenotypic Screen." Antimicrobial Agents and Chemotherapy 55, no. 12 (2011): 5740–45. http://dx.doi.org/10.1128/aac.05188-11.

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ABSTRACTMalaria is one of the deadliest infectious diseases in the world, with the eukaryotic parasitePlasmodium falciparumcausing the most severe form of the disease. Discovery of new classes of antimalarial drugs has become an urgent task to counteract the increasing problem of drug resistance. Screening directly for compounds able to inhibit parasite growthinvitrois one of the main approaches the malaria research community is now pursuing for the identification of novel antimalarial drug leads. Very recently, thousands of compounds with potent activity against the parasiteP. falciparumhave

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4

Snyder, Edward L., and Roger Y. Dodd. "Reducing the Risk of Blood Transfusion." Hematology 2001, no. 1 (2001): 433–42. http://dx.doi.org/10.1182/asheducation-2001.1.433.

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Abstract There are continuing concerns over the safety of the nation's and the world's blood supply. The allogeneic blood supply is tested for antibodies to HIV1/2, HTLVI/II, hepatitis B, hepatitis C (HCV) and syphilis. Testing is also performed for donor ALT (SGOT) levels, for the presence of hepatitis B surface antigen, human immunodeficiency virus (HIV) p24 antigen and, using nucleic acid amplification testing (NAT), for HIV and HCV nucleic acids. Still, there are concerns regarding other pathogenic agents. Dr. Roger Dodd addresses a series of pathogens that are already known to be transmis

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5

Markus, Miles B. "Mouse-Based Research on Quiescent Primate Malaria Parasites." Trends in Parasitology 32, no. 4 (2016): 271–73. http://dx.doi.org/10.1016/j.pt.2016.02.006.

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6

Badell, E., V. Pasquetto, P. Druilhe, and N. Van Rooijen. "A mouse model for human malaria erythrocytic stages." Parasitology Today 11, no. 6 (1995): 235–37. http://dx.doi.org/10.1016/0169-4758(95)80088-3.

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7

Butcher, Geoff. "A mouse model for human malaria erythrocytic stages: Reply." Parasitology Today 11, no. 6 (1995): 224. http://dx.doi.org/10.1016/0169-4758(95)80082-4.

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8

Kautz, Leon, Chloe Latour, Wlodarczyk Myriam, et al. "Erythroferrone Represses Hepcidin Expression in a Mouse Model of Malaria." Blood 124, no. 21 (2014): 4022. http://dx.doi.org/10.1182/blood.v124.21.4022.4022.

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Abstract Introduction: Malaria, a mosquito-borne disease caused by a parasite, represents a major global health challenge in developing countries, resulting in over half a million deaths each year. Among the many clinical complications, the multiplication of the parasites in erythrocytes leads to a severe anemia secondary to hemolysis and increased erythrophagocytosis. Malarial anemia is also characterized by insufficient erythropoiesis to compensate for the loss of red blood cells, despite high erythropoietin (EPO) levels. Iron is an essential functional component of erythrocyte hemoglobin, t

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9

Desruisseaux, Mahalia S., FNU Nagajyothi, Shankar Mukherjee, Dumitru A. Iacobas, Herbert B. Tanowitz, and David C. Spray. "Gene expression alterations in a mouse model of cerebral malaria." BMC Proceedings 2, Suppl 1 (2008): P15. http://dx.doi.org/10.1186/1753-6561-2-s1-p15.

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10

Sacci, J. B., M. E. Schriefer, J. H. Resau, et al. "Mouse model for exoerythrocytic stages of Plasmodium falciparum malaria parasite." Proceedings of the National Academy of Sciences 89, no. 9 (1992): 3701–5. http://dx.doi.org/10.1073/pnas.89.9.3701.

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11

Vercellotti, Gregory M., and John D. Belcher. "Heme and the Vasculature: How the Endothelium Protects Itself Against Toxic Iron." Blood 116, no. 21 (2010): SCI—25—SCI—25. http://dx.doi.org/10.1182/blood.v116.21.sci-25.sci-25.

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Abstract Abstract SCI-25 Iron-derived reactive oxygen species (ROS) are involved in the pathogenesis of numerous vascular disorders. Heme-derived iron plays an instrumental role in the pathology of intravascular hemolytic diseases including malaria, sickle cell anemia, transfusion reactions, DIC and PNH. Heme catalyzed oxidative stress promotes a pro-inflammatory/prothrombogenic endothelium, diminution of bio-available nitric oxide (NO) and attraction of leukocytes and platelets. The vasculature is protected against heme-catalyzed injury by plasma proteins including haptoglobin, hemopexin, alb

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12

Touitou, Elka, Judith H. Waknine, Biana Godin, and Jacob Golenser. "Treatment of malaria in a mouse model by intranasal drug administration." International Journal for Parasitology 36, no. 14 (2006): 1493–98. http://dx.doi.org/10.1016/j.ijpara.2006.07.006.

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13

Couto, Joana, Miray Tonk, Joana Ferrolho, et al. "Antiplasmodial activity of tick defensins in a mouse model of malaria." Ticks and Tick-borne Diseases 9, no. 4 (2018): 844–49. http://dx.doi.org/10.1016/j.ttbdis.2018.03.011.

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14

Mohmmed, Asif, Palakodeti V. N. Dasaradhi, Raj K. Bhatnagar, Virander S. Chauhan, and Pawan Malhotra. "In vivo gene silencing in Plasmodium berghei—a mouse malaria model." Biochemical and Biophysical Research Communications 309, no. 3 (2003): 506–11. http://dx.doi.org/10.1016/j.bbrc.2003.08.027.

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15

Anderson, John W., Dimitri Sarantakis, Jacek Terpinski, et al. "Novel diaryl ureas with efficacy in a mouse model of malaria." Bioorganic & Medicinal Chemistry Letters 23, no. 4 (2013): 1022–25. http://dx.doi.org/10.1016/j.bmcl.2012.12.022.

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16

Le Bras, Alexandra. "A mouse model for IPF research." Lab Animal 49, no. 6 (2020): 171. http://dx.doi.org/10.1038/s41684-020-0568-3.

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17

Yamamoto, Kouichi, Misuzu Matsui, Tomoko Morimoto, Yumiko Yamamoto, Noriaki Takeda, and Atsushi Yamatodani. "A mouse model for emesis research." Japanese Journal of Pharmacology 79 (1999): 75. http://dx.doi.org/10.1016/s0021-5198(19)34323-9.

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18

Won, Jungyeon, Lan Ying Shi, Wanda Hicks, et al. "Mouse Model Resources for Vision Research." Journal of Ophthalmology 2011 (2011): 1–12. http://dx.doi.org/10.1155/2011/391384.

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The need for mouse models, with their well-developed genetics and similarity to human physiology and anatomy, is clear and their central role in furthering our understanding of human disease is readily apparent in the literature. Mice carrying mutations that alter developmental pathways or cellular function provide model systems for analyzing defects in comparable human disorders and for testing therapeutic strategies. Mutant mice also provide reproducible, experimental systems for elucidating pathways of normal development and function. Two programs, the Eye Mutant Resource and the Translatio

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19

Barker, P. M. "Mouse model for cystic fibrosis research." BMJ 305, no. 6863 (1992): 1229. http://dx.doi.org/10.1136/bmj.305.6863.1229-c.

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20

Willimann, K., H. Matile, N. A. Weiss, and B. A. Imhof. "In vivo sequestration of Plasmodium falciparum-infected human erythrocytes: a severe combined immunodeficiency mouse model for cerebral malaria." Journal of Experimental Medicine 182, no. 3 (1995): 643–53. http://dx.doi.org/10.1084/jem.182.3.643.

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Cerebral malaria is a fatal complication of infection by Plasmodium falciparum in man. The neurological symptoms that characterize this form of malarial disease are accompanied by the adhesion of infected erythrocytes to the vasculature of the brain. To study this phenomenon in vivo, an acute phase severe combined immunodeficiency (SCID) mouse model was developed in which sequestration of P. falciparum-infected human erythrocytes took place. During acute cerebral malaria in humans, the expression of intercellular adhesion molecule-1 (ICAM-1) is induced in vascular endothelium by inflammatory r

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21

Min, Byung Soh. "Mouse Model for Colorectal Cancer Metastasis Research." Korean Journal of Clinical Oncology 8, no. 1 (2012): 4–8. http://dx.doi.org/10.14216/kjco.12001.

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22

Comfort, Nathaniel. "The prisoner as model organism: malaria research at Stateville Penitentiary." Studies in History and Philosophy of Science Part C: Studies in History and Philosophy of Biological and Biomedical Sciences 40, no. 3 (2009): 190–203. http://dx.doi.org/10.1016/j.shpsc.2009.06.007.

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23

Torre, Sabrina, David Langlais, and Philippe Gros. "Genetic analysis of cerebral malaria in the mouse model infected with Plasmodium berghei." Mammalian Genome 29, no. 7-8 (2018): 488–506. http://dx.doi.org/10.1007/s00335-018-9752-9.

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24

Badell, Edgar, Claude Oeuvray, Alicia Moreno, et al. "Human Malaria in Immunocompromised Mice." Journal of Experimental Medicine 192, no. 11 (2000): 1653–60. http://dx.doi.org/10.1084/jem.192.11.1653.

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We have recently described that sustained Plasmodium falciparum growth could be obtained in immunodeficient mice. We now report the potential of this new mouse model by assaying the effect of the passive transfer of antibodies (Abs) which in humans have had a well-established effect. Our results show that the total African adult hyperimmune immunoglobulin Gs (HI-IgGs) strongly reduce P. falciparum parasitemia similarly to that reported in humans, but only when mice are concomitantly reconstituted with human monocytes (HuMNs). In contrast, neither HI-IgGs nor HuMNs alone had any direct effect u

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25

Butala, Parag, Caroline Szpalski, Marc Soares, Edward H. Davidson, Denis Knobel, and Stephen M. Warren. "Zmpste24−/− Mouse Model for Senescent Wound Healing Research." Plastic and Reconstructive Surgery 130, no. 6 (2012): 788e—798e. http://dx.doi.org/10.1097/prs.0b013e31826d102b.

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26

Ramezani, A., J. Noronha, V. W. K. Tsoi, et al. "A simple SCID mouse model for HIV research." Transfusion Science 17, no. 1 (1996): 99–108. http://dx.doi.org/10.1016/0955-3886(95)00063-1.

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27

Jumani, R. S., K. Bessoff, M. S. Love, et al. "A Novel Piperazine-Based Drug Lead for Cryptosporidiosis from the Medicines for Malaria Venture Open-Access Malaria Box." Antimicrobial Agents and Chemotherapy 62, no. 4 (2018): e01505-17. http://dx.doi.org/10.1128/aac.01505-17.

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ABSTRACTCryptosporidiosis causes life-threatening diarrhea in children under the age of 5 years and prolonged diarrhea in immunodeficient people, especially AIDS patients. The standard of care, nitazoxanide, is modestly effective in children and ineffective in immunocompromised individuals. In addition to the need for new drugs, better knowledge of drug properties that drivein vivoefficacy is needed to facilitate drug development. We report the identification of a piperazine-based lead compound forCryptosporidiumdrug development, MMV665917, and a new pharmacodynamic method used for its charact

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28

Ohno, Tamio, Yuki Miyasaka, Masako Kuga, et al. "Mouse NC/Jic strain provides novel insights into host genetic factors for malaria research." Experimental Animals 68, no. 3 (2019): 243–55. http://dx.doi.org/10.1538/expanim.18-0185.

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29

Ntim, Stephanie B., and Katherine J. Johnson. "The art of malaria education: an arts-based malaria education model, Pepease-Kwahu, Ghana." International Journal Of Community Medicine And Public Health 6, no. 12 (2019): 5042. http://dx.doi.org/10.18203/2394-6040.ijcmph20195443.

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Background: Malaria is a major health concern in Ghana as well as other countries in West Africa, where it is estimated that more than 300 million people are at risk of malaria infection. While prior research has highlighted promising school-based interventions often facilitated through textbook information or teacher-based lectures to promote awareness about the disease, less is known as to how well such interventions are able to actively involve and engage students in learning about malaria in their schools.Methods: This research examines the role of the performing arts as a heuristic for st

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30

Montosi, E., S. Manzoni, A. Porporato, and A. Montanari. "An ecohydrological model of malaria outbreaks." Hydrology and Earth System Sciences 16, no. 8 (2012): 2759–69. http://dx.doi.org/10.5194/hess-16-2759-2012.

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Abstract. Malaria is a geographically widespread infectious disease that is well known to be affected by climate variability at both seasonal and interannual timescales. In an effort to identify climatic factors that impact malaria dynamics, there has been considerable research focused on the development of appropriate disease models for malaria transmission driven by climatic time series. These analyses have focused largely on variation in temperature and rainfall as direct climatic drivers of malaria dynamics. Here, we further these efforts by considering additionally the role that soil wate

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31

Lauterwasser, Erica M. W., Shaun D. Fontaine, Hao Li, et al. "Trioxolane-Mediated Delivery of Mefloquine Limits Brain Exposure in a Mouse Model of Malaria." ACS Medicinal Chemistry Letters 6, no. 11 (2015): 1145–49. http://dx.doi.org/10.1021/acsmedchemlett.5b00296.

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32

Min-Oo, G., A. Fortin, M.-F. Tam, P. Gros, and MM Stevenson. "Phenotypic expression of pyruvate kinase deficiency and protection against malaria in a mouse model." Genes & Immunity 5, no. 3 (2004): 168–75. http://dx.doi.org/10.1038/sj.gene.6364069.

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33

Ngo-Thanh, Ha, Trang Dam Thuy, Kazutomo Suzue, et al. "Long-term acrylamide exposure exacerbates brain and lung pathology in a mouse malaria model." Food and Chemical Toxicology 151 (May 2021): 112132. http://dx.doi.org/10.1016/j.fct.2021.112132.

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34

Kuhen, Kelli L., Arnab K. Chatterjee, Matthias Rottmann, et al. "KAF156 Is an Antimalarial Clinical Candidate with Potential for Use in Prophylaxis, Treatment, and Prevention of Disease Transmission." Antimicrobial Agents and Chemotherapy 58, no. 9 (2014): 5060–67. http://dx.doi.org/10.1128/aac.02727-13.

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ABSTRACTRenewed global efforts toward malaria eradication have highlighted the need for novel antimalarial agents with activity against multiple stages of the parasite life cycle. We have previously reported the discovery of a novel class of antimalarial compounds in the imidazolopiperazine series that have activity in the prevention and treatment of blood stage infection in a mouse model of malaria. Consistent with the previously reported activity profile of this series, the clinical candidate KAF156 shows blood schizonticidal activity with 50% inhibitory concentrations of 6 to 17.4 nM agains

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35

Hausmann Muela, Susanna, Joan Muela Ribera, Elizabeth Toomer, and Koen Peeters Grietens. "The PASS-model: a model for guiding health-seeking behavior and access to care research." Malaria Reports 2, no. 1 (2012): 3. http://dx.doi.org/10.4081/malaria.2012.e3.

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The mobilization of affordable measures and treatments has brought health services and health care tools closer to the poor. This is particularly the case in the context of malaria control and elimination efforts. Still, the other side of delivery is use: the targeted populations have to access and accept these resources. Although the need to better align the delivery and user sides is increasingly recognised, there still is a gap between this awareness and researchers&rsquo; response to adequately address the community side in a way that actionable results can be achieved. In order to avo

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36

Darling, Thayer K., Michael P. Schenk, Chengjing C. Zhou та ін. "Platelet α-granules contribute to organ-specific pathologies in a mouse model of severe malaria". Blood Advances 4, № 1 (2019): 1–8. http://dx.doi.org/10.1182/bloodadvances.2019000773.

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Key Points Nbeal2 deficiency leads to significantly reduced lung and brain pathology and enhanced survival in a mouse model of malaria. Both antibody-dependent and antibody-independent platelet depletion in mice recapitulate the findings observed in Nbeal2−/− mice.

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37

Buskbjerg Jager, Sara, Giulia Ronchi, Christian Bjerggaard Vaegter, and Stefano Geuna. "The Mouse Median Nerve Experimental Model in Regenerative Research." BioMed Research International 2014 (2014): 1–6. http://dx.doi.org/10.1155/2014/701682.

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Sciatic nerve crush injury in rat animal model is one of the most common experimental models used in regenerative research. However, the availability of transgenic mouse for nerve regeneration studies is constantly increasing and, therefore, the shift from rat model to mouse model is, in some cases, necessary. Moreover, since most of the human nerve lesions occur in the upper limb, it is also advantageous to shift from sciatic nerve to median nerve. In this study we described an experimental model which involves lesions of the median nerve in the mouse. Data showed that the finger flexor muscl

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38

Wada, Kosuke, Hiroshi Makino, Kenji Shimada, Fumiaki Shikata, Atsushi Kuwabara, and Tomoki Hashimoto. "Translational Research Using a Mouse Model of Intracranial Aneurysm." Translational Stroke Research 5, no. 2 (2013): 248–51. http://dx.doi.org/10.1007/s12975-013-0296-8.

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39

Wang, Bailing, Hongri Liu, Kaixin Wang, Guodong Xin, and Jia Song. "Research Notes: User Identification Model Based on Mouse Behaviors." International Journal of Software Engineering and Knowledge Engineering 28, no. 02 (2018): 175–92. http://dx.doi.org/10.1142/s0218194018400028.

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This study investigates the user identification method based on the computer mouse dynamic behaviors. One of the purposes is to refine seven types of mouse actions, and about 110 dimensional features have been employed from user session statistics and operating characteristics. And then, two basic techniques, principal component analysis (PCA) and weighted multiclassifier, are used to lay out the mouse behavior. Combing PCA and the new proposed classifier, two experiments on identification and authentication have been carried out. By validating the selected mouse data, the accuracy rate is as

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40

Klotz, F. W., J. D. Chulay, W. Daniel, and L. H. Miller. "Invasion of mouse erythrocytes by the human malaria parasite, Plasmodium falciparum." Journal of Experimental Medicine 165, no. 6 (1987): 1713–18. http://dx.doi.org/10.1084/jem.165.6.1713.

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Plasmodium falciparum malaria merozoites require erythrocyte sialic acid for optimal invasion of human erythrocytes. Since mouse erythrocytes have the form of sialic acid found on human erythrocytes (N-acetyl neuraminic acid), mouse erythrocytes were tested for invasion in vitro. The Camp and 7G8 strains of P. falciparum invaded mouse erythrocytes at 17-45% of the invasion rate of human erythrocytes. Newly invaded mouse erythrocytes morphologically resembled parasitized human erythrocytes as shown on Giemsa-stained blood films and by electron microscopy. The rim of parasitized mouse erythrocyt

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41

Stelzer, Marie K., Henry C. Pitot, Amy Liem, Johannes Schweizer, Charles Mahoney, and Paul F. Lambert. "A Mouse Model for Human Anal Cancer." Cancer Prevention Research 3, no. 12 (2010): 1534–41. http://dx.doi.org/10.1158/1940-6207.capr-10-0086.

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42

Stephens, Robin, and Jean Langhorne. "Effector Memory Th1 CD4 T Cells Are Maintained in a Mouse Model of Chronic Malaria." PLoS Pathogens 6, no. 11 (2010): e1001208. http://dx.doi.org/10.1371/journal.ppat.1001208.

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43

Falanga, Pierre B., and Eugene C. Butcher. "Late treatment with anti-LFA-1 (CD11a) antibody prevents cerebral malaria in a mouse model." European Journal of Immunology 21, no. 9 (1991): 2259–63. http://dx.doi.org/10.1002/eji.1830210938.

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44

Montosi, E., S. Manzoni, A. Porporato, and A. Montanari. "An eco-hydrologic model of malaria outbreaks." Hydrology and Earth System Sciences Discussions 9, no. 3 (2012): 2831–54. http://dx.doi.org/10.5194/hessd-9-2831-2012.

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Abstract. Malaria is a geographically widespread infectious disease that is well known to be affected by climate variability at both seasonal and interannual timescales. In an effort to identify climatic factors that impact malaria dynamics, there has been considerable research focused on the development of appropriate disease models for malaria transmission and their consideration alongside climatic datasets. These analyses have focused largely on variation in temperature and rainfall as direct climatic drivers of malaria dynamics. Here, we further these efforts by considering additionally th

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45

Swanson, Phillip A., Aron E. Lukacher, and Eva Szomolanyi-Tsuda. "Immunity to polyomavirus infection: The polyomavirus–mouse model." Seminars in Cancer Biology 19, no. 4 (2009): 244–51. http://dx.doi.org/10.1016/j.semcancer.2009.02.003.

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46

Mandt, Rebecca E. K., Maria Jose Lafuente-Monasterio, Tomoyo Sakata-Kato, et al. "In vitro selection predicts malaria parasite resistance to dihydroorotate dehydrogenase inhibitors in a mouse infection model." Science Translational Medicine 11, no. 521 (2019): eaav1636. http://dx.doi.org/10.1126/scitranslmed.aav1636.

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Resistance has developed in Plasmodium malaria parasites to every antimalarial drug in clinical use, prompting the need to characterize the pathways mediating resistance. Here, we report a framework for assessing development of resistance of Plasmodium falciparum to new antimalarial therapeutics. We investigated development of resistance by P. falciparum to the dihydroorotate dehydrogenase (DHODH) inhibitors DSM265 and DSM267 in tissue culture and in a mouse model of P. falciparum infection. We found that resistance to these drugs arose rapidly both in vitro and in vivo. We identified 13 point

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47

Et. al., Yusuf Aliyu Adamu,. "Malaria Prediction Model Using Machine Learning Algorithms." Turkish Journal of Computer and Mathematics Education (TURCOMAT) 12, no. 10 (2021): 7488–96. http://dx.doi.org/10.17762/turcomat.v12i10.5655.

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Measures have been taking to ensure the safety of individuals from the burden of vector-borne disease but it remains the causative agent of death than any other diseases in Africa. Many human lives are lost particularly of children below five years regardless of the efforts made. The effect of malaria is much more challenging mostly in developing countries. In 2019, 51% of malaria fatality happen in Africa which it increased by 20% in 2020 due to the covid-19 pandemic. The majority of African countries lack a proper or a sound health care system, proper environmental settlement, economic hards

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48

Groffen, John, Jan-Willem Voncken, Vesa Kaartinen, Christine Morris, and Nora Heisterkamp. "Ph-positive Leukemia: A Transgenic Mouse Model." Leukemia & Lymphoma 11, sup1 (1993): 19–24. http://dx.doi.org/10.3109/10428199309047857.

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49

Opitz, Oliver G., Michael Quante, Alexander von Werder, Steffen Heeg, and Hubert E. Blum. "A Mouse Model of Oral-Esophageal Carcinogenesis." Oncology Research and Treatment 28, no. 1 (2004): 44–48. http://dx.doi.org/10.1159/000082039.

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50

Nair, D., G. Gallos, W. L. Yang, and T. S. Ravikumar. "A nude mouse model for studying radiofrequency ablation." European Journal of Cancer 37 (April 2001): S298. http://dx.doi.org/10.1016/s0959-8049(01)81592-6.

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