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Relevant bibliographies by topics / Male genital diseases

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Academic literature on the topic 'Male genital diseases'

Author: Grafiati

Published: 4 June 2021

Last updated: 29 July 2024

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Journal articles on the topic "Male genital diseases"

1

Jacob, Jesse T., Minh Ly T. Nguyen, and Susan M. Ray. "Male genital tuberculosis." Lancet Infectious Diseases 8, no. 5 (May 2008): 335–42. http://dx.doi.org/10.1016/s1473-3099(08)70101-4.

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Mallon, E., J. S. Ross, D. A. Hawkins, M. Dinneen, N. Francis, and C. B. Bunker. "Biopsy of male genital dermatosis." Sexually Transmitted Infections 73, no. 5 (October 1, 1997): 421. http://dx.doi.org/10.1136/sti.73.5.421-a.

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Oriel, J. D. "Male genital Chlamydia trachomatis infections." Journal of Infection 25 (July 1992): 35–37. http://dx.doi.org/10.1016/0163-4453(92)91944-7.

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Shim, T. N., D. Hawkins, A. Muneer, S. Minhas, A. Freeman, C. Jameson, N. Francis, M. Dinneen, and C. B. Bunker. "P2.182 Male Genital Dermatoses in HIV." Sexually Transmitted Infections 89, Suppl 1 (July 2013): A143.2—A143. http://dx.doi.org/10.1136/sextrans-2013-051184.0446.

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Patil C, Sridevi, Sharanbasava V, and P. S. Suman Babu. "A clinico-epidemiological study of non venereal dermatoses involving male and female genitalia." IP Indian Journal of Clinical and Experimental Dermatology 7, no. 3 (September 15, 2021): 237–42. http://dx.doi.org/10.18231/j.ijced.2021.045.

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Non venereal genital lesions may be confused with venereal diseases. This may be responsible for considerable concern to patients and may cause diagnostic dilemma to the physicians. This study was conducted to find out the hospital based prevalence and clinical profile of Non venereal dermatoses involving male and female Genitalia with or without associated lesions elsewhere.It was a descriptive study which included a series of 120 patients presenting to Dermatology department from Jan 2015 to July 2016 with non-venereal genital lesions.Among 120 patients, there were 109 males and 11 females (M:F 9.9:1). The prevalence of non-venereal genital lesion was 21.76 per 10,000 patients. The age ranged from 2 months to 65 years with the mean age of 32.94 years and majority in the age group of 21-30 years(25%). The most common disorder was fixed drug eruptions ,37 (30.83%) followed by vitiligo, 29 (24.16%) and psoriasis, 13 (10.83%). This study highlights the importance of diagnosing common non venereal genital dermatoses. It also helps in avoiding the general misconception that all genital lesions are sexually transmitted.

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Rao, A., and C. B. Bunker. "Male genital skin biopsy." International Journal of STD & AIDS 22, no. 7 (July 2011): 418–19. http://dx.doi.org/10.1258/ijsa.2011.011072.

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Mazzoni, Daniel, David Winkle, Louis Pool, Anthony Hall, and Jim Muir. "Genital premalignant and malignant diseases: a retrospective study of male genital skin biopsies." International Journal of Dermatology 60, no. 6 (February 27, 2021): 712–16. http://dx.doi.org/10.1111/ijd.15439.

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P., Mamatha, Abhijeet Vardhan, and Sheena Arora. "Incidence of non-venereal lesions of the male genitalia: a study of 248 male cases at a tertiary care centre." International Journal of Research in Dermatology 6, no. 2 (February 24, 2020): 187. http://dx.doi.org/10.18203/issn.2455-4529.intjresdermatol20200472.

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<p class="abstract"><strong>Background:</strong> Non-venereal dermatoses of external genitalia refers to those dermatoses involving external genitalia which are not sexually transmitted. These are associated with mental distress and guilt feelings in affected patients. The aim was to study the incidence of non-venereal lesions of the genitalia.</p><p class="abstract"><strong>Methods:</strong> This was a cross-sectional clinical observational study in 248 male patients attending dermatovenereology OPD of Vydehi Institute of Medical Sciences and Research Center with non-venereal genital lesions for a period of 18 months. Cases having venereal diseases were excluded from the study.<strong></strong></p><p class="abstract"><strong>Results:</strong> The study included 248 male patients with non-venereal genital lesions. A total of twenty-five different types of non-venereal dermatoses were noted in our study. The most common non-venereal dermatoses were Non-venereal infections, seen in 107 patients (43.14%), which includes scabies in 45 patients (18.14%), tinea cruris 25 patients (11.29%), candidiasis 28 (11.29%) followed by lichen simplex chronicus 23 cases (9.27%) and scrotal dermatitis (6.45%) other dermatoses include psoriasis, lichen planus, fixed drug eruptions, scrotal horn, histoid hansens, squamous cell carcinoma, Zoon’s balanitis.</p><p class="abstract"><strong>Conclusions:</strong> This study reflected the importance of diagnosis of non-Venereal dermatoses and refutes the general misconception that all genital lesions are of venereal origin.</p><p> </p>

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Ramoni, Stefano, Susanna Benardon, Cristina Beatrice Spigariolo, Luciano Süss, and Stefano Veraldi. "Ticks on the external male genital structures." Parasitology International 83 (August 2021): 102336. http://dx.doi.org/10.1016/j.parint.2021.102336.

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Bachaspatimayum, Romita, Zamzachin Guite, and Thangjam Bijayanti Devi. "Clinical and laboratory profiles of genital ulcers (sexually transmitted diseases) in a tertiary care center in northeastern India." Our Dermatology Online 12, no. 2 (April 1, 2021): 120–29. http://dx.doi.org/10.7241/ourd.20212.4.

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Background: Genital ulcers are defined as breaches in the continuity of the genital mucosa and/or skin. Sexually transmitted infections (STIs) that cause genital ulcer disease (GUD) are syphilis, chancroid, donovanosis, lymphogranuloma venereum (LGV), and herpes genitalis. This study aimed to investigate the clinical and laboratory profiles of STI-related genital ulcers. Materials and Methods: A cross-sectional two-year study was conducted on patients attending the Outpatient Department of Dermatology, Venereology and Leprosy in a tertiary care center in northeastern India. Selected were 95 patients who presented themselves with STI-related genital ulcers. Detailed history taking and examination were conducted with basic tests to assist the diagnosis. Results: The male-to-female ratio was 3.32:1, and the most common site was the glans and prepuce in males (28.77%) and the labia majora and minora in females (36.36%). 96.84% of patients had superficial ulcers. The KOH mount was positive in 26 patients. The Tzanck smear was positive in 31 patients. RPR was positive in four. HIV was positive in eleven. Herpes genitalis (96.84%) was the most common GUD. Mixed STIs were attested in 41.05% of patients. Conclusion: GUD can take various forms of presentation. The available laboratory tests should be utilized. The possibility of mixed infections should always be kept in mind.

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Dissertations / Theses on the topic "Male genital diseases"

1

Partridge, Jeffrey M. "Genital human papillomavirus infection in men : incidence, duration, and risk factors in a cohort of young male university students /." Thesis, Connect to this title online; UW restricted, 2006. http://hdl.handle.net/1773/10868.

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Okonofua, Friday Ebhodaghe. "Female and male infertility in Nigeria : studies on the epidemiology of infertility in Nigeria with special reference to the role of genital tract infections and sexual and reproductive risk factors /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-354-X/.

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GRIZARD, DELORME GENEVIEVE. "Fonction leydigienne et cryptorchidie experimentale chez le rat." Clermont-Ferrand 2, 1988. http://www.theses.fr/1988CLF2E408.

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Steele, Matthew Stephen. "Male microbicides, genital hygiene, and HIV risk behavior among Kenyan men /." Thesis, Connect to this title online; UW restricted, 2004. http://hdl.handle.net/1773/6434.

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El-Demiry, Mostafa Ibrahim Metwally. "Lymphocyte subsets and macrophages in the male genital tract in health and disease." Thesis, University of Edinburgh, 1987. http://hdl.handle.net/1842/18860.

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The ability of sperm cells to induce specific auto- and iso-immunity was reported as early as the turn of the century. However the mechanism controlling autoreactivity to sperm is not well known. As lymphocytes constitute the major cellular components of the immune system, determination of their anatomical location within the tissues of the male genital tract may be of considerable importance in understanding immunological infertility and other urogenital disorders. A series of monoclonal antibodies that react with human lymphoreticular cells was therefore used in an indirect immunoperoxidase technique to study their distribution throughout the male genital tract. The normal human tissues investigated were: testis, epididymis, vas deferens, prostate and seminal vesicles obtained from multiorgan transplant donors. The clinical specimens examined included surgical biopsies of testis, epididymis and prostate obtained during surgical procedures directed at the investigation and treatment of subfertile males and other patients. All normal tissues, apart from the testis, were found to contain appreciable numbers of T-lymphocytes (leu 4⁺). T-cells of the suppressor/cytotoxic phenotype (leu 2a⁺) were more abundant in the intraepithelial compartment while T-cells of the helper/inducer phenotye (leu 3a⁺) were more common in the interstitial areas. With the exception of the prostate, very few B-cells were observed. Macrophages (leu M3⁺) were identified within normal testicular tissues as well as the rest of the male genital tract. HLA-DR⁺ cells were also identified and the HLA-DR antigens were normally expressed on the lining epithelium of the rete testis, epididymis and vas deferens. Derangement of this pattern was observed in clinical specimens. Testicular biopsies from patients with testicular obstruction showed marked infiltration with lymphocytes mainly of the T-cell type. Biopsies from patients with benign prostatic hyperplasia showed increased infiltration with the helper/inducer T-cells and other cell types such as natural killer cells (leu llb⁺) and activated T-cells (IL₂-r⁺). These patterns of lymphoid cells distribution could provide an insight into both normal immunohomeostatic mechanisms and pathological events within the male genital tract. The presence and distribution of lymphocyte subpopulations and macrophages within human urothelium in health and disease was also examined. T-lymphocyte subsets and macrophages were identified in normal urothelium and shown to have a similar pattern of distribution to that seen in normal epithelium of the genital tract. The existence of these cell populations may contribute to the health and protection of urothelium, particularly in resistance to infection and tumour surviellance. The presence of leucocytes and their subpopulations was also studied in the ejaculate from 69 men with an infertile marriage and 12 fertile men. Leucocytes were found in large numbers in the fertile men compared with the patients. Lymphocytes were found in 20% of the patients. There was no correlation between leucocyte counts and growth of micro-organisms. These results cast doubt on the conventional criteria of subclinical genital tract infection, namely positive culture and excess leucocyte counts.

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Gomes, Camila Richieri. "Análise clínica e molecular de pacientes com distúrbios do desenvolvimento gonadal." Universidade de São Paulo, 2009. http://www.teses.usp.br/teses/disponiveis/5/5135/tde-05032010-110358/.

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Introdução: O termo distúrbios do desenvolvimento gonadal (DDG) inclui condições congênitas nas quais o desenvolvimento gonadal é atípico. Estudos feitos em camundongos observaram que alguns genes como o Cbx2 e o Tcf21 interferem na fase inicial do desenvolvimento gonadal, afetando tanto gônadas XX quanto XY. O gene Dhh, por sua vez, codifica o fator de transcrição Dhh, produzido pelas células de Sertoli, que é fundamental para a diferenciação das células de Leydig em gônadas XY. Nos ovários, o gene FOXL2 atua na foliculogênese, sendo fundamental para a formação dos ovários. Objetivos: Analisar clinicamente e pesquisar anormalidades nos genes CBX2, TCF21, DHH e FOXL2 em pacientes portadores de distúrbios do desenvolvimento gonadal 46, XY e 46, XX. Material e Métodos: Foram estudados 60 pacientes (41 com DDG 46, XY e 19 com DDG 46, XX). A análise molecular foi realizada a partir da amplificação gênica por PCR e sequenciamento direto. Resultados: Várias alterações alélicas foram encontradas nos quatro genes, algumas ainda não descritas na literatura. Uma alteração intrônica no gene DHH foi encontrada em um paciente com DDG 46, XY e não foi encontrada em nenhum dos 360 alelos normais estudados (g.IVS2 +29G>A). Estudamos essa variante através da extração do RNA do testículo do paciente afetado, mas não encontramos alteração no RNA; portanto ela parece não ser uma mutação. No gene TCF21, a variante encontrada foi identificada em controles normais. No gene CBX2, das treze alterações encontradas, uma não foi identificada em 206 alelos normais, e há troca de aminoácidos (p.C132R / g.394 T>C). Trata-se de uma variante que pode ter relação com o fenótipo do paciente, portador de DDG 46, XY. No gene FOXL2, das três alterações encontradas, uma não foi identificada em 206 alelos normais; contudo, não há troca de aminoácidos (p.A181A / g.543 C>T). Conclusão: Esse estudo sugere que mutações nos genes CBX2, TCF21, FOXL2 e DHH são causas raras de distúrbios do desenvolvimento gonadal.
Introduction: Congenital disorders of gonadal development (DGD) include conditions whose gonadal development is atypical. Studies in mice found that some genes such as Cbx2 and Tcf21 interfere in the initial phase of gonadal development, affecting both XX and XY gonads. Dhh gene, in turn, encodes the transcription factor Dhh, produced by Sertoli cells, which is essential for the differentiation of Leydig cells in XY gonads. In the ovaries, genes as FOXL2 act in folliculogenesis, fundamental to the development of the ovaries. Objectives: To analyze patients with disorders of gonadal development (DGD) 46, XY and 46, XX and research mutations in CBX2, TCF21, DHH and FOXL2 genes. Methods: We analyzed 60 patients (41 DGD 46, XY patients and 19 DGD 46, XX patients). The whole coding region of CBX2, TCF21, DHH and FOXL2 genes were amplified by PCR and direct sequenced. Results: Several allelic variations have been found in the four genes, some not even described by literature. One intronic variation in DHH was described in one patient with 46, XY DGD and it wasnt found in any of the 360 normal control alleles studied (g.IVS2 +29G>A). We studied this variant through RNA extraction from the affected patients testes, but we didnt find any alteration in the RNA, so it doesnt seem to be a mutation. In TCF21 gene, the single variant that was found was identified in normal controls. In CBX2 gene, among the 13 alterations described, one wasnt identified in 206 normal control alleles, and there is aminoacid change (p.C132R / g.394 T>C). This is a variant that may be a mutation, causing the patients phenotype that had 46, XY DGD. In FOXL2, among the 3 variations described, one wasnt indentified in 206 normal control alleles, but there wasnt amino acid change (p.A181A / g.543 C>T).Conclusion: This study suggests that mutations in CBX2, TCF21, FOXL2 and DHH genes are rarely causes of disorders of gonadal development.

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Kamtchouing, Pierre. "Ontogenese des profils endocriniens dans differentes situations experimentales chez le rat." Caen, 1987. http://www.theses.fr/1987CAEN2028.

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Courbes de secretion de lh et fsh et evolution histologique du testicule de rat en fonction de l'age notamment de la puberte. La modification experimentale de la fonction testiculaire (cryptorchidie, irradiation, hemicastration) a permis de mettre en evidence une interrelation entre les cellules de leydig et les cellules seminiferes

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Morel, Frédéric. "Etude experimentale du controle d'une famille de proteines de secretion d'un organe androgeno-dependant : l'epididyme de lezard." Clermont-Ferrand 2, 1987. http://www.theses.fr/1987CLF21074.

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Books on the topic "Male genital diseases"

1

Libby, Edwards, Lynch Peter J. 1936-, and Neill Sarah M, eds. Genital dermatology atlas. 2nd ed. Philadelphia, PA: Lippincott Williams & Wilkins, 2011.

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(Firm), Medicode, ed. Male genitalia. Salt Lake City, UT: Medicode, 1994.

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International, Congress of Andrology (6th 1997 Salzburg Austria). Current advances in andrology: Proceedings of the VIth International Congress of Andrology, Salzburg (Austria), May 25-29, 1997. Bologna: Monduzzi Editore, International Proceedings Division, 1997.

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William, Bonnez, ed. Guide to genital HPV diseases and prevention. New York: Informa Healthcare USA, 2008.

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P, Pryor J., and Lipshultz Larry I. 1942-, eds. Andrology. London: Butterworths, 1987.

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G, Dyment Paul, ed. Male reproductive health. Philadelphia: Hanley & Belfus, 1996.

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R, Barratt C. L., and Cooke I. D, eds. Advances in clinical andrology. Lancaster: MTP Press, 1988.

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Taguchi, Yosh. Private parts: A user's guide. Toronto: McClelland and Stewart, 1988.

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S, Paola Angelo, and Paola Frederick A, eds. Campbell's urology: Review and assessment. Philadelphia: Saunders, 1995.

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Merrily, Weisbord, ed. Private parts: An owner's guide to the male anatomy. 3rd ed. Toronto: M&S, 2003.

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Book chapters on the topic "Male genital diseases"

1

Hall, Anthony. "Genital Granulomatous Diseases." In Atlas of Male Genital Dermatology, 109–11. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-99750-6_33.

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Oriel, J. D. "Genital Warts." In Diseases in the Homosexual Male, 99–109. London: Springer London, 1988. http://dx.doi.org/10.1007/978-1-4471-1634-9_6.

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Dinotta, Franco, Stefano Ramoni, Pompeo Donofrio, and Marco Cusini. "Sexually Transmitted Diseases." In Atlas of Male Genital Disorders, 37–49. Milano: Springer Milan, 2013. http://dx.doi.org/10.1007/978-88-470-2787-9_4.

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Turgut, Ahmet T., Mehmet Ruhi Onur, Erhan Akpinar, Vikram S. Dogra, and Gregory T. MacLennan. "Congenital and Acquired Nonneoplastic Adrenal Diseases." In Genitourinary Radiology: Male Genital Tract, Adrenal and Retroperitoneum, 211–30. London: Springer London, 2013. http://dx.doi.org/10.1007/978-1-4471-4899-9_10.

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Brunner, Helmut. "Mycoplasma Infections of Man: Respiratory and Male Genital Tract Diseases." In Rapid Diagnosis of Mycoplasmas, 39–56. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2478-6_5.

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Hall, Anthony. "Extramammary Paget’s Disease." In Atlas of Male Genital Dermatology, 189–91. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-99750-6_56.

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Hall, Anthony. "Trauma and Artefactual Disease." In Atlas of Male Genital Dermatology, 147–49. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-99750-6_44.

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Hall, Anthony. "Aphthous Ulcers and Behçet’s Disease." In Atlas of Male Genital Dermatology, 141–43. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-99750-6_42.

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Hall, Anthony. "Darier’s Disease and Papular Acantholytic Dyskeratosis." In Atlas of Male Genital Dermatology, 125–27. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-99750-6_38.

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Hall, Anthony. "General Management of Genital Skin Disease." In Atlas of Male Genital Dermatology, 11–12. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-99750-6_5.

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Conference papers on the topic "Male genital diseases"

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Indriatmi, Wresti, and Giorgio Barnes Komala. "Multiple Genital Ulcer on a Male Patient Due to Fungal Balanoposthitis Suspect of Candida Albicans Infection Mimicking Genital Herpes: A Case Report." In The 2nd International Conference on Tropical Medicine and Infectious Disease. SCITEPRESS - Science and Technology Publications, 2019. http://dx.doi.org/10.5220/0009987903250328.

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Leite, Izabel Feitosa da Mata, Adelina Mouta Moreira Neto, Guilherme de Aguiar Moraes, Lucas Cardoso Siqueira Albernaz, and Matheus de Campos Medeiros. "Neuro-Behçet’s Syndrome: case report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.209.

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Context: Behçet’s Disease (BD) is a multisystem chronic disorder characterized by perivasculitis within several tissues, including the Central Nervous System – Neuro-Behçet’s Disease, which accounts for 3 – 9 % of the BD patients. Neuro- Behçet’s Syndrome may present as brainstem or pyramidal syndromes, myelopathies, meningoencephalitis, intracranial hypertension and movement or psychiatric disorders. The objective of the present work is to report a case of Neuro-Behçet’s Disease, a rare and disabling disorder. Case report: 24-year-old male presenting focal neurological deficits – hemiparesis on the right side and motor aphasia, associated with unstoppable hiccups and visual blurring. His previous pathological history featured several self-limiting episodes of the same neurological presentation, as well as acute exacerbations of oral and genital ulcers. Erythema Nodosum and Folliculitis were his main skin lesions. The ophthalmological evaluation showed bilateral cicatricial chorioretinitis. Laboratorial diagnostic procedures revealed left shift leukocytosis, high ESR, high protein level in the CSF and negative serology for B and C hepatitis, HTLV, HIV and toxoplasmosis. P-ANCA, C-ANCA, ANA and Rheumatoid factor were all found negative. Magnetic Resonance Imaging of the brain showed multiple oval- shaped T2- hyperintensity foci, with adjacent vasogenic edema, in the brainstem and basal ganglia areas – suggesting vasculitis. Treatment involved pulse therapy with Methylprednisolone, followed by Azathioprine and Prednisone. The patient has had full recovery and no other relapses. Conclusions: This case illustrates the importance of investigating Neuro-Behçet’s Disease in patients with neurological symptoms and oral/genital ulcers. The goal is to establish adequate and early treatment to improve the quality of life.

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Rocha, Willker Menezes da, Camila Freze Baez, Larissa Alves Afonso, Fernanda Nahoum Carestiato, Marianna Tavares Venceslau Gonçalves, Rafael Brandão Varella, and Silvia Maria Baeta Cavalcanti. "The use of DNA microarray assay as a diagnostic tool to study penile cancer associated with human papillomavirus." In XIII Congresso da Sociedade Brasileira de DST - IX Congresso Brasileiro de AIDS - IV Congresso Latino Americano de IST/HIV/AIDS. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/dst-2177-8264-202133p159.

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Introduction: The genital infection by the human papillomavirus (HPV) can result in a sexually transmitted disease associated with precursor lesions for carcinogenesis in the genital tract. In recent years, evidence was accumulated defining HPV as the etiologic agent of cervical cancer; however, the etiology of penile cancer is still open and lacks studies. This study aims to contribute to the epidemiologic knowledge regarding the prevalence of this virus in malignant lesions of the male genital tract, using the DNA microarray assay, a technique that allows the simultaneous detection of up to 32 different HPV genotypes. Objective: The aim of this study was to investigate the presence of HPV in penile malignant lesions, to genotype HPV, when present, to correlate the HPV infection and its genotypes with the histopathological data. Methods: A total of 112 penile cancer samples was collected in a cross-sectional study. The detection methodology consisted of (1) detecting the presence of HPV DNA by the polymerase chain reaction (PCR) technique with generic primers, (2) genotyping the HPV using the DNA microarray assay, and (3) correlation of the histopathology, tumor invasiveness, and the dispersion of malignant cells by the lymph nodes with the presence of HPV. Results: The HPV prevalence was 57.1% (64). The most prevalent genotype was the HPV16 (32.8%), followed by HPV6 (23.4%); HPV18, HPV35, and HPV45 (12.5%); HPV31 (10.9%); and HPV70 (7.8%). Of the HPV-positive samples, 25% were mixed infections. Conclusion: The role of the HPV infection was significant within the multifactorial etiology of penile cancer. There was statistical significance between the lesion invasiveness and the presence of high-risk HPV infection. Thus, genotype surveillance can promote a better understanding of the role of HPV genotypes in male cancer development, and the DNA microarray assay proved to be an efficient tool for both the epidemiological study and the diagnostics of the HPV.

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Khurana, Anil, Paramjeet Kaur, Ashok K. Chauhan, Yashpal Verma, and Nupur Bansal. "Extra ovarian adult granulosa cell tumor of omentum: A report of a rare entity." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685372.

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Aims: Extra ovarian granulosa cell tumor (GCT) is extremely rare tumor, assumed to arise from the ectopic gonadal tissue along the embryonal route of the genital ridge. A case of extra ovarian granulosa cell tumor of omentum in a 69 year old female presented here. Materials and Methods: A 69 years old postmenopausal, hypertensive female presented with complaints of pain in right lumber and iliac region of one month duration. Pain was off and on and intermittent. The patient had a history of hysterectomy 12 years ago for fibroid uterus. Results: Ultrasound examination of abdomen showed a hypoechoic lesion of size 78.1 mm x 57.3 mm in right iliac fossa with mild thickening of surrounding omentum. Another hypoechoic lesion of size 36.7 mm x 22.9 mm was seen in retroperitoneal region in supero-medial aspect of right kidney. CECT abdomen showed heterogeneously enhanced nodular lesion of size 6.6 x 6.8 cm in right lumbar region, mild thickening of surrounded omentum also seen however there was no evidence of infiltration to bowel loop seen. Uterus was not visualized. PET CT whole body revealed mildly metabolically active enlarged nodes in the bilateral level ib an ii, metabolically active large lobulated heterogeneously enhancing soft tissue density lesion in right lumbar region with non enhancing areas of necrosis. The lesion is closely abutting the anterior abdominal wall musculature antero laterally and small bowel loop medially surrounding mesenty shows increased vascularity and haziness. Colonoscopy findings were normal. Trucut biopsy of mass right lumbar region was positive for malignancy likely Round cell Sarcoma. A provisional diagnosis of retroperitoneal sarcoma of right lumbar region was made. She underwent exploratory laparotomy with excision of tumor. As per Operative findings there was approximately 8 x 7 cm, firm, omental mass present right to midline, arising from under surface of greater omentum. Ovaries were normal. Gross examination of omental mass showed nodular mass measuring 8 x 5 x 6 cm. External surface was multinodular and cut surface was grey brown to grey yellow with solid cystic areas and areas of necrosis. Microscopic examination of specimen showed Extraovarian Adult granulosa cell tumor/metastasis from occult granulose cell tumor. On IHC Vimentin, CK, SMA, Inhibin were positive, Ki67:15%, ER/PR were also positive and are negative for calretinin, thromobomodulin. Extensive necrosis was seen. After that she underwent rexploration and total omenectomy. HPE showed fat necrosis in omentum. All investigation showed no evidence of tumor in ovaries and at any other primary site then the patient finally diagnosed as having Granulosa cell tumor involving only omentum post op stage III C. Then patient was given six courses of chemotherapy with Inj Paclitaxel and Inj Carboplatin three weekly. Now patient is on regular follow up and disease free. Conclusion: Extra ovarian adult granulosa cell tumor of omentum is rare tumor. Multimodal treatment approaches including surgery, multi-agent chemotherapy may provide a survival benefit for patients.

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