Academic literature on the topic 'Mammary cancer'

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Journal articles on the topic "Mammary cancer"

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Miller, James L., Alexandra Reddy, Rebecca M. Harman, and Gerlinde R. Van de Walle. "A xenotransplantation mouse model to study physiology of the mammary gland from large mammals." PLOS ONE 19, no. 2 (2024): e0298390. http://dx.doi.org/10.1371/journal.pone.0298390.

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Although highly conserved in structure and function, many (patho)physiological processes of the mammary gland vary drastically between mammals, with mechanisms regulating these differences not well understood. Large mammals display variable lactation strategies and mammary cancer incidence, however, research into these variations is often limited to in vitro analysis due to logistical limitations. Validating a model with functional mammary xenografts from cryopreserved tissue fragments would allow for in vivo comparative analysis of mammary glands from large and/or rare mammals and would impro
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Clavijo-Maldonado, Alejandro, Enio Ferreira, Carlos Vargas-Hernández, and Fredy A. Rivera-Páez. "Canine mammary cancer." Veterinarska stanica 51, no. 4 (2020): 425–39. http://dx.doi.org/10.46419/vs.51.4.2.

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Canine mammary cancer (CMC) is one of the most common neoplasms in intact females in comparison to other species. Several risk factors have been identified, including breed, genetic predisposition, age, reproductive history, hormonal influence, diet, and body condition, in addition to previous lesions to the mammary gland, such as mammary atypical hyperplasia. An understanding of the genetic markers for the disease and a clinical approach are important for establishing a specific therapy that can allow adequate patient survivorship. Overexpression of the HER-2 gene in canines and humans is ass
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Hassan, B. B., S. M. Elshafae, W. Supsavhad, et al. "Feline Mammary Cancer." Veterinary Pathology 54, no. 1 (2016): 32–43. http://dx.doi.org/10.1177/0300985816650243.

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Feline mammary carcinoma (FMC) is similar to human breast cancer in the late age of onset, incidence, histopathologic features, biological behavior, and pattern of metastasis. Therefore, FMC has been proposed as a relevant model for aggressive human breast cancer. The goals of this study were to develop a nude mouse model of FMC tumor growth and metastasis and to measure the expression of genes responsible for lymphangiogenesis, angiogenesis, tumor progression, and lymph node metastasis in FMC tissues and cell lines. Two primary FMC tissues were injected subcutaneously, and 6 FMC cell lines we
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Gestl, Shelley A., Travis L. Leonard, Jessica L. Biddle, Michael T. Debies, and Edward J. Gunther. "Dormant Wnt-Initiated Mammary Cancer Can Participate in Reconstituting Functional Mammary Glands." Molecular and Cellular Biology 27, no. 1 (2006): 195–207. http://dx.doi.org/10.1128/mcb.01525-06.

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ABSTRACT The minimal residual disease foci that beget breast cancer relapse after a period of disease dormancy remain uncharacterized despite their enormous clinical importance. To model dormant breast cancer in vivo, we employed a transgenic mouse model in which Wnt1-initiated mammary cancer is doxycycline dependent. After regression of Wnt-dependent cancers, subclinical disease lesions were propagated in vivo using classical tissue recombination techniques. Surprisingly, outgrowths derived from dormant malignant tissue reconstituted morphologically normal ductal trees in wild-type mammary fa
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Hipp, Elizabeth, Xiaobing Fan, Sanaz A. Jansen, et al. "T2* relaxation times of intraductal murine mammary cancer, invasive mammary cancer, and normal mammary gland." Medical Physics 39, no. 3 (2012): 1309–13. http://dx.doi.org/10.1118/1.3684950.

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Wu, Diana, Lilian U. Thompson, and Elena M. Comelli. "MicroRNAs: A Link between Mammary Gland Development and Breast Cancer." International Journal of Molecular Sciences 23, no. 24 (2022): 15978. http://dx.doi.org/10.3390/ijms232415978.

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Breast cancer is among the most common cancers in women, second to skin cancer. Mammary gland development can influence breast cancer development in later life. Processes such as proliferation, invasion, and migration during mammary gland development can often mirror processes found in breast cancer. MicroRNAs (miRNAs), small, non-coding RNAs, can repress post-transcriptional RNA expression and can regulate up to 80% of all genes. Expression of miRNAs play a key role in mammary gland development, and aberrant expression can initiate or promote breast cancer. Here, we review the role of miRNAs
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Dennison, Kirsten L., Nyssa Becker Samanas, Quincy Eckert Harenda, et al. "Development and characterization of a novel rat model of estrogen-induced mammary cancer." Endocrine-Related Cancer 22, no. 2 (2015): 239–48. http://dx.doi.org/10.1530/erc-14-0539.

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The ACI rat model of 17β-estradiol (E2)-induced mammary cancer is highly relevant for use in establishing the endocrine, genetic, and environmental bases of breast cancer etiology and identifying novel agents and strategies for preventing breast cancer. E2 treatment rapidly induces mammary cancer in female ACI rats and simultaneously induces pituitary lactotroph hyperplasia and adenoma. The pituitary tumors can result in undesired morbidity, which compromises long-term studies focused on mammary cancer etiology and prevention. We have defined the genetic bases of susceptibility to E2-induced m
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ULLRICH, R. L., and R. J. PRESTON. "Radiation Induced Mammary Cancer." Journal of Radiation Research 32, SUPPLEMENT2 (1991): 104–9. http://dx.doi.org/10.1269/jrr.32.supplement2_104.

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Miller, WR. "Mammary Development and Cancer." British Journal of Cancer 78, no. 4 (1998): 558. http://dx.doi.org/10.1038/bjc.1998.538.

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Dabydeen, Sarah A., та Priscilla A. Furth. "Genetically engineered ERα-positive breast cancer mouse models". Endocrine-Related Cancer 21, № 3 (2014): R195—R208. http://dx.doi.org/10.1530/erc-13-0512.

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The majority of human breast cancers are estrogen receptor-positive (ER+), but this has proven challenging to model in genetically engineered mice. This review summarizes information on 21 mouse models that develop ER+ mammary cancer. Where available, information on cancer pathology and gene expression profiles is referenced to assist in understanding which histological subtype of ER+ human cancer each model might represent.ESR1,CCDN1, prolactin,TGFα,AIB1,ESPL1, andWNT1overexpression,PIK3CAgain of function, as well as loss ofP53(Trp53) orSTAT1are associated with ER+ mammary cancer. Treatment w
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Dissertations / Theses on the topic "Mammary cancer"

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Boggs, Rene' Michelle. "MicroRNA expression in canine mammary cancer." Diss., Texas A&M University, 2008. http://hdl.handle.net/1969.1/85979.

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MicroRNAs (miRNAs) play a vital role in differentiation, proliferation and tumorigenesis by binding to messenger RNAs (mRNA) and inhibiting translation. To initiate an investigation into the identification of miRNAs in the domestic dog, an emerging model for human disease, a comparison of the human and canine genetic databases was conducted. The bioinformatics work revealed significant conservation of miRNA genes between the two species. Proof of principle experiments, including serial dilutions and sequencing, were performed to verify that primers made to amplify human mature miRNAs can be us
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Quan, Xiaojiang. "Genetic study of mammary cancer development." Doctoral thesis, Universite Libre de Bruxelles, 2001. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/211503.

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Chuah, S. Y. "Prostaglandins and steroids in human mammary cancer." Thesis, University of Strathclyde, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.303867.

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Gillespie, Karen R. "Molecular pathology of mammary neoplasia." Thesis, Glasgow Caledonian University, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.243533.

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Oliver, Joseph James. "Characterizing ErbB2-induced mammary tumourigenesis." Thesis, Kingston, Ont. : [s.n.], 2007. http://hdl.handle.net/1974/687.

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Pascual, Domingo Rosa. "CPEB2 in mammary gland homeostasis and breast cancer." Doctoral thesis, Universitat de Barcelona, 2018. http://hdl.handle.net/10803/586313.

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The mammary gland develops postnatally and is remodeled, during each estrous cycle and pregnancy, through very dynamic expansions and involutions of its polarized epithelial tree. Moreover, the mammary gland is hierarchically organized, where the two main epithelial populations may arise from a common bipotent mammary stem cell (MaSC). The principal epithelial cell types in the mammary gland are luminal cells and myoepithelial cells (also named basal cells), which are found surrounding luminal cells and in contact with the extracellular matrix. Further, the luminal compartment has two main li
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Magub, Stephanie Jane. "The role of HER2 in canine mammary cancer." Thesis, University of Kent, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.580400.

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While the role of the EGF family (in particular the HER2 receptor) has been well documented in human cancers, its role in the progression of canine mammary cancer is less well understood. Dogs have long been used as models of human disease in the development of human therapeutics, however the animal health market is only just beginning to develop animal-specific therapies. This project aimed to survey the expression of the complete family of EGF receptors and ligands in a range of normal tissues, and also in benign and malignant canine mammary cancers. In addition to this, the functional role
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Scully, Jaqueline Susan. "Insertion of oncogenes into mouse mammary epithelium." Thesis, University of Cambridge, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.315287.

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Alford, Deborah Jane. "Morphogenesis of human mammary epithelial cells in vitro." Thesis, University College London (University of London), 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.265150.

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Belobrajdic, Damien. "Dietary butyrate inhibits NMU induced mammary cancer in rats /." Title page and abstract only, 1998. http://web4.library.adelaide.edu.au/theses/09SB/09sbb452.pdf.

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Books on the topic "Mammary cancer"

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Symposium, Biochemical Society. Mammary development and cancer. Portland Press, 1998.

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S, Rudland Philip, ed. Mammary development and cancer. Portland Press, 1998.

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Tavassoli, Fattaneh A. Tumors of the mammary gland. American Registry of Pathology in collaboration with the Armed Forces Institute of Pathology, 2009.

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Medina, Daniel, William Kidwell, Gloria Heppner, and Elizabeth Anderson, eds. Cellular and Molecular Biology of Mammary Cancer. Springer US, 1988. http://dx.doi.org/10.1007/978-1-4613-0943-7.

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Daniel, Medina, and Organ Systems Coordinating Center (National Cancer Institute), eds. Cellular and molecular biology of mammary cancer. Plenum Press, 1987.

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H, Hollmann K., Verley J. M, and International Society against Breast Cancer. Symposium, eds. New frontiers in mammary pathology 1986. M. Nijhoff, 1986.

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Ip, Margot M., and Bonnie B. Asch, eds. Methods in Mammary Gland Biology and Breast Cancer Research. Springer US, 2000. http://dx.doi.org/10.1007/978-1-4615-4295-7.

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Carlyle, Jones Thomas, Mohr U, and Hunt Ronald Duncan, eds. Integument and mammary glands. Springer-Verlag, 1989.

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Uyttenbroeck, Frans. Surgical quality assurance in gynaecological and mammary cancerology. Peeters, 1993.

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Ealey, Kafi N. Role of conjugated linoleic acid in colon and mammary cancer. National Library of Canada, 2002.

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Book chapters on the topic "Mammary cancer"

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Nagao, Minako, and Takashi Sugimura. "Mammary Cancer." In Carcinogenicity. CRC Press, 2021. http://dx.doi.org/10.1201/9781003067641-24.

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Watson, James C., and John P. Hoffman. "Internal Mammary Lymph Nodes." In Breast Cancer. Humana Press, 2002. https://doi.org/10.1007/978-1-59259-161-9_22.

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Vonderhaar, Barbara K., and Maitreyi Bhattacharjee. "The Mammary Gland." In Biological Responses in Cancer. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-1236-9_6.

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Thompson, Henry J. "Mammary Cancer in Rats." In Tumor Models in Cancer Research. Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-968-0_10.

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Thompson, Henry J., and Michael B. Sporn. "Mammary Cancer in Rats." In Tumor Models in Cancer Research. Humana Press, 2002. https://doi.org/10.1007/978-1-59259-100-8_9.

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Smith, Gilbert H. "Mouse Mammary Tumor Virus: Stem Cells and Mammary Cancer." In Stem Cells and Cancer. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-933-8_10.

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Sampayo, Rocío, Sol Recouvreux, María Inés Diaz Bessone, and Marina Simian. "Mammary Gland Organoids." In Cancer Drug Discovery and Development. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-60511-1_3.

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Russo, J., and I. H. Russo. "Mammary Carcinogenesis." In Breast Cancer: Progress in Biology, Clinical Management and Prevention. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-1617-6_12.

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Veronese, Maria Luisa, Florencia Bullrich, and Carlo M. Croce. "Oncogenes and Mammary Carcinogenesis." In Endocrinology of Breast Cancer. Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-699-7_7.

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Black, Maurice M., and Reinhard E. Zachrau. "Stepwise Mammary Carcinogenesis: Immunological Considerations." In Early Breast Cancer. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70192-4_8.

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Conference papers on the topic "Mammary cancer"

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Elliott, Gloria D., and John J. McGrath. "Freezing Response of Mammary Tissue: A Mathematical Study." In ASME 1999 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1999. http://dx.doi.org/10.1115/imece1999-0584.

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Abstract Cryosurgery, the use of low temperatures to devitalize neoplastic tissue, has become an accepted treatment modality for many cancers such as those of the liver and prostate. Recently, the application of cryosurgery to human breast malignancies has been explored (Staren et. al, 1997, Pham and Rubinsky, 1998). Breast cancer will affect 1 in 9 women over the course of their lifetime (American Cancer Society, 1997). Although there are a wide variety of therapies available to treat this disease, the broad pathological spectrum of patients with breast cancer necessitates newer and better tr
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Mazumdar, Abhijit, Jamal L. Hill, Yun Zhang, Frances S. Kittrell, Daniel Medina, and Powel Brown. "Abstract A101: Metformin prevents mammary tumors in p53-null mammary gland mice." In Abstracts: AACR International Conference on Frontiers in Cancer Prevention Research‐‐ Oct 22-25, 2011; Boston, MA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1940-6207.prev-11-a101.

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Moreno, Andre, Kimberly Masiero Cola, Larissa Heberle, and Marcelo Moreno. "RELATIONSHIP BETWEEN IMMUNOHISTOCHEMICAL CHARACTERIZATION AND FORM OF DIAGNOSIS OF BREAST CANCER." In Scientifc papers of XXIII Brazilian Breast Congress - 2021. Mastology, 2021. http://dx.doi.org/10.29289/259453942021v31s1008.

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Introduction: Breast cancer is the most incident neoplasia among Brazilian women. According to immunogenetic characteristics, it is possible to verify that malignant breast neoplasms with greater biological activity would be those classified as luminary B, HER2+ and triple-negative, and that the one with the lowest biological activity would be the luminal subtype A. Thus, a mammography would be more likely to detect cancers with a low degree of biological characteristics such as “luminal A”. On the other hand, mammary carcinomas with greater potential for systemic dissemination show faster gro
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Su, Y., K. Shankar, FA Simmen, and RC Simmen. "Regulation of adipocyte lipid homeostasis by genistein alters mammary epithelial cell differentiation: a paracrine mechanism for mammary tumor protection." In CTRC-AACR San Antonio Breast Cancer Symposium: 2008 Abstracts. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-5082.

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Worku, D., WG Jiang, RE Mansel, and K. Mokbel. "SATB1: a 'master genome organizer' in mammary carcinogenesis." In CTRC-AACR San Antonio Breast Cancer Symposium: 2008 Abstracts. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-3070.

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Brady, Nicholas J., Michael A. Farrar, and Kathryn L. Schwertfeger. "Abstract B64: Macrophage-specific deletion of STAT5 disrupts normal mammary gland development and accelerates mammary tumorigenesis." In Abstracts: AACR Special Conference: Advances in Breast Cancer; October 17-20, 2015; Bellevue, WA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1557-3125.advbc15-b64.

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Nair, S., and R. Li. "Critical Role of a Transcription Elongation Regulator in Mammary Gland Development and Mammary Tumorigenesis." In Abstracts: Thirty-Second Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 10‐13, 2009; San Antonio, TX. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-09-1152.

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Brown, Powel, Jamal Hill, Francis Kittrell, et al. "Abstract B56: Rexinoid LG100268 and tamoxifen prevents mammary tumors in p53-null mammary gland mice." In Abstracts: AACR International Conference on Frontiers in Cancer Prevention Research‐‐ Nov 7-10, 2010; Philadelphia, PA. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1940-6207.prev-10-b56.

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Ferreira, Nancy, Darley Ferreira, and Thais Ferreira. "GENETIC EVALUATION OF MICROCALCIFICATIONS AS A PROGNOSTIC FACTOR." In Abstracts from the Brazilian Breast Cancer Symposium - BBCS 2021. Mastology, 2021. http://dx.doi.org/10.29289/259453942021v31s2101.

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Introduction: Breast cancer is the most recurring type of cancer among women, with reduced mortality at an initial stage of lesion. From a radiological perspective, perceived microcalcifications may be associated with histological findings such as proliferative injuries with or without atypical features and ductal carcinoma in situ. Currently, percutaneous and vacuum biopsies allow for the correlation between anatomoradiological and identification of previous lesions and those that offer the risk of cancer. No biomarker has been established to predict the risk of cancer in women diagnosed with
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Izzo, Franco, María C. Díaz Flaqué, Rocío Vicario, et al. "Abstract 2281: GATA3 and progestin interaction in mammary cancer." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-2281.

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Reports on the topic "Mammary cancer"

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Lamartiniere, Coral A. Proteomic Analysis of Genistein Mammary Cancer. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada442989.

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Lipman, Ruth. Genes Controlling Susceptibility to Mammary Cancer. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada396610.

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Lipman, Ruth D. Gene Controlling Susceptibility to Mammary Cancer. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada407281.

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Lipman, Ruth D. Genes Controlling Susceptibility to Mammary Cancer. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada374076.

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Lipman, Ruth. Genes Controlling Susceptibility to Mammary Cancer. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada392321.

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Vandenberg, Ted. Inhibition of Mammary Cancer by Citrus Limonoids. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada398201.

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Donehower, Lawrence A. Mouse Mammary Cancer Models - Mechanisms and Markers. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada404557.

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Donehower, Lawrence A. Mouse Mammary Cancer Models - Mechanisms and Markers. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada408687.

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Lamartiniere, Coral A. Proteomic Analysis of Genistein Mammary Cancer Chemoprevention. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada428933.

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Edwards, John R. Mammary Cancer and Activation of Transposable Elements. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada614053.

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