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Books on the topic 'Marburg virus disease'

Author: Grafiati

Published: 4 June 2021

Last updated: 28 January 2023

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1

Ebola and Marburg viruses. 2nd ed. New York: Chelsea House, 2011.

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2

Parker, James N. The official patient's sourcebook on Marburg hemorrhagic fever. San Diego, Calif: Icon Health Publications, 2002.

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3

Kuhn, Jens H. Filoviruses: A compendium of 40 years of epidemiological, clinical, and laboratory studies. Vienna: Springer, 2007.

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4

Johnson-Delaney, Cathy A. Topics of clinical interest in nonhuman primates, update bibliographies: Herpes B virus (1989-1991), the filoviridae (1990-1991), simian hemorrhagic fever (1990-1991), and tuberculosis (1990-1991). Seattle, Wash., U.S.A: Primate Information Center, Regional Primate Research Center, University of Washington, 1991.

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5

Johnson-Delaney, Cathy A. The filoviridae: Ebola, ebola-like, and marburg viruses in non-human primates : a selective bibliography, 1965-1990. Seattle, Wash., U.S.A: Primate Information Center, Regional Primate Research Center, University of Washington, 1990.

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6

Kuhn, Jens H. Filoviruses: A compendium of 40 years of epidemiological, clinical, and laboratory studies. Vienna: Springer, 2007.

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7

The hot zone. London: Corgi, 1995.

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8

The hot zone. London: Doubleday, 1994.

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9

Deadly invaders: Marburg virus, bird flu, and other emerging viruses. New York: Houghton Mifflin Co., 2006.

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10

Grady, Denise. Deadly invaders: Virus outbreaks around the world, from Marburg fever to avian flu. Boston, Mass: Kingfisher, 2006.

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11

Richard, Preston. Virus. Paris: France loisirs, 1995.

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12

Virus. Paris: France Loisir, 1995.

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13

Martini, G. Marburg Virus Disease. Springer, 2014.

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14

Klenk, Hans-Dieter. Marburg and Ebola Viruses. Springer, 2011.

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15

Heinz, Feldmann, and Klenk H. D. 1938-, eds. Ebola and Marburg viruses: Molecular and cellular biology. Wymondham, Norfolk, U.K: Horizon Bioscience, 2004.

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16

Feldmann, Heinz, and Hans-Dieter Klenk. Ebola and Marburg Viruses: Molecular and Cellular Biology. Taylor & Francis Group, 2003.

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17

Marburg And Ebola Viruses (Current Topics in Microbiology & Immunology). SPRINGER-VERLAG, 1999.

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18

Simpson, A., E. Aarons, and R. Hewson. Marburg and Ebola viruses. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0038.

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Infection with Marburg and Ebola viruses cause haemorrhagic fevers that are characterized by organ malfunction, bleeding complications, and high mortality. The viruses are members of the family Filoviridae, a group of membrane-enveloped filamentous RNA viruses. Five distinct species of the genus Ebolavirus have been reported; the genus Marburgvirus contains only one species. Both Marburg and Ebola virus diseases are zoonotic infections whose primary hosts are thought to be bats. The initial human infection is acquired from wildlife and subsequent person-to-person spread propagates the outbreak until it is brought under control. Ebola and Marburg viruses are classified as hazard or risk group 4 pathogens because of the very high case fatality rates observed for Ebola and Marburg virus diseases, the frequency of person-to-person transmission and community spread, and the lack of an approved vaccine or antiviral therapy. This mandates that infectious materials are handled and studied in maximum containment laboratory facilities. Epidemics have occurred sporadically since the discovery of Marburg in 1967 and Ebola virus in 1976. While some of these outbreaks have been relatively large, infecting a few hundreds of individuals, they have generally occurred in rural settings and have been controlled relatively easily. However, the 2013–2016 epidemic of Ebola virus disease in West Africa was different, representing the first emergence of the Zaire species of Ebola in a high-density urban location. Consequently, this has been the largest recorded filovirus outbreak in both the number of people infected and the range of geographical spread. Many of the reported and confirmed cases were among people living in high-density and impoverished urban environments. The chapter summarizes the most up-to-date taxonomic status of the family Filoviridae. It focuses on Marburg and Ebola viruses in a historical context, culminating in the 2013–2016 outbreak of Ebola virus in West Africa. Virus biology of the most well-studied member is described, with details of the viral genome and the protein machinery necessary to propagate viruses at the molecular and cellular level. This information is used to build a wider-scale virus–host perspective with detail on the pathology and pathogenesis of Ebola virus disease. The consequences of cell infection are examined, together with our current understanding of the immune response to Ebola virus, leading to a broader description of the clinical features of disease. The chapter closes by drawing information together in a section on diagnosis, ecology, prevention, and control.

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19

Ryabchikova, Elena. Ebola and Marburg Viruses: A View of Infection Using Electron Microscopy. Battelle Press, 2004.

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20

Publications, ICON Health. Marburg Virus: A Medical Dictionary, Bibliography, And Annotated Research Guide To Internet References. Icon Health Publications, 2004.

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21

The Hot Zone: The Chilling True Story of an Ebola Outbreak. Corgi, 2014.

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22

Berger, Stephen, and Inc Gideon Informatics. Marburg Virus: Global Status. Gideon Informatics, Incorporated, 2021.

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23

Berger, Stephen, and Inc Gideon Informatics. Marburg Virus: Global Status. Gideon Informatics, Incorporated, 2022.

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24

Bebell, Lisa M. Ebola and Other Filoviruses. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0002.

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Congenital and pediatric Ebola virus disease (EVD) and Marburg virus disease (MVD) are severe, even lethal infections. Historically, children have been underrepresented in filovirus disease outbreaks, and evidence-based treatment strategies are lacking. Existing data suggest that case fatalities are highest among children under four years of age, which is partially explained by higher virus concentrations in young children. Prevention and aggressive resuscitation, nutrition, and supportive care are the mainstays of management until filovirus-specific therapies can be developed. Differences in pediatric immune and inflammatory responses may necessitate unique approaches to pediatric vaccination and treatment. There are minimal safety or immunogenicity data in children, a crucial knowledge gap that must be addressed in future trials. Studying pediatric survivors of the 2014–2016 West Africa EVD outbreak will provide much-needed data on long-term outcomes and residual effects of filovirus disease while we await effective filovirus-specific vaccines and therapies.

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25

Deadly Invaders: Tracking Today's Global Viruses, from Marburg to the Avian Flu. Kingfisher Books, 2006.

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