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Dissertations / Theses on the topic 'Marginal Zone B Cells'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

You, Yuying. "Cross-talk between marginal zone B cells and marginal zone macrophages." Thesis, Birmingham, Ala. : University of Alabama at Birmingham, 2010. https://www.mhsl.uab.edu/dt/2010p/you.pdf.

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2

MARUYAMA, MITSUO, TADASHI MATSUSHITA, TOMOKI NAOE, et al. "RHOF PROMOTES MURINE MARGINAL ZONE B CELL DEVELOPMENT." Nagoya University School of Medicine, 2014. http://hdl.handle.net/2237/20548.

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3

Gentile, Maurizio 1981. "Role of mTOR in the activation of marginal zone B cells by TACI." Doctoral thesis, Universitat Pompeu Fabra, 2014. http://hdl.handle.net/10803/318167.

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The marginal zone (MZ) of the spleen contains an innate-like subset of B cells that mount rapid protective antibody responses to polysaccharides and lipids from bloodborne viruses and bacteria. These antigens activate MZ B cells by engaging somatically recombined B cell receptors (BCRs) and germline-encoded patternrecognition receptors, including Toll-like receptors (TLRs). MZ B cells receive additional co-stimulatory signals from a proliferation-inducing ligand (APRIL) and B cellstimulating factor of the TNF family (BAFF), two tumor necrosis factor (TNF)-related cytokines released main

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4

Turchinovich, Gleb. "BKLF promotes B cell differentiation towards marginal zone lineage." [S.l. : s.n.], 2007. http://nbn-resolving.de/urn:nbn:de:bsz:25-opus-44233.

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5

Kolan, Shrikant S. "Defining the role of CD47 and SIRPα in murine B cell homeostasis". Doctoral thesis, Umeå universitet, Institutionen för integrativ medicinsk biologi (IMB), 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-107636.

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B cell development is a highly organized process, which commences in the fetal liver during embryogenesis and in the bone marrow (BM) after birth. Surface IgM+ immature B cells emigrate from the BM via the blood stream to the spleen and finally differentiate into conventional mature follicular B (FoB) cells and marginal zone (MZ) B cells. Conversely, some sIgM+ immature B cells can also mature into IgD+ FoB cells in the BM. The ubiquitously expressed cell surface glycoprotein CD47 and its receptor signal regulatory protein α (SIRPα) are members of the immunoglobulin superfamily. Both individua

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6

Bascones, Gleave Sabrina 1985. "B cell ontogeny and stromal regulation of homeostatic antibody responses to commensal antigens in humans." Doctoral thesis, Universitat Pompeu Fabra, 2016. http://hdl.handle.net/10803/664813.

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The spleen contains a unique subset of innate-like marginal zone (MZ) B cells that rapidly mount protective antibody responses to invasive encapsulated bacteria. Here we show that human splenic MZ B cells also mount homeostatic antibody responses to commensal antigens undergoing systemic translocation from mucosal surfaces. This humoral response decreased upon splenectomy and correlated with splenic capture of commensal antigens and with the generation of MZ B cell-derived plasmablasts and plasma cells following GC-independent and -dependent pathways. Remarkably, commensal antigens targeted ma

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7

Barra, Quaglia Carolina M. 1982. "Sinusoid-lining cells are novel myeloid-endothelial innate cells that form splenic niches for marginal zone B cell activation and plasma cell survival." Doctoral thesis, Universitat Pompeu Fabra, 2014. http://hdl.handle.net/10803/328415.

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Los sinusoides del bazo humano promueven la lenta percolación de la sangre, favoreciendo la captura de antígeno por los fagocitos y linfocitos del sistema inmune local. Estratégicamente posicionadas delimitando los vasos, e históricamente conocidas como células retículo-endoteliales, las células que delinean los sinusoides (SLCs), tienen una biología enigmática y podrían ser vistas como un componente desconocido del sistema inmunológico. En esta tesis hemos observado que las SLCs poseían un fenotipo simil-endotelial, eran específicas de humano y expresaban moléculas típicamente asocia

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8

Phan, Tri Giang. "The SWHEL model for studying B cell responses in tolerance and immunity." University of Sydney. Medicine, 2005. http://hdl.handle.net/2123/626.

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Classical immunoglobulin transgenic (Ig-Tg) mouse models such as the MD4 anti-hen egg lysozyme (-HEL) Ig-Tg line have been used extensively to study B cell responses in tolerance and immunity. This thesis describes a new generation of gene-targeted mice (designated SWHEL mice) whereby the VH10 Ig variable gene encoding the HyHEL-10 specificity of the original anti-HEL Ig-Tg mouse was targeted to the Ig heavy chain locus. B cells in the SWHEL mouse are therefore capable of undergoing class switch recombination (CSR) and somatic hypermutation (SHM), representing a major advance on the original M

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9

Rubtsov, Anatoly V. "Regulation of marginal zone B cell migration in the primary IgM antibody response /." Connect to full text via ProQuest. Limited to UCD Anschutz Medical Campus, 2007.

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Thesis (Ph.D. in Immunology) -- University of Colorado Denver, 2007.<br>Typescript. Includes bibliographical references (leaves 146-169). Free to UCD affiliates. Online version available via ProQuest Digital Dissertations;

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10

Murray-Brown, William. "The study of marginal zone B cell development in the spleens of CD1d-/- mice." Thesis, Imperial College London, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.536029.

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11

Dugge, Rucha [Verfasser]. "Tumor progression in gastric marginal zone B-cell lymphoma of MALT type / Rucha Dugge." Ulm : Universität Ulm, 2021. http://d-nb.info/1238690491/34.

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12

Alyafei, Saud Abdalla Mubarak Mohamed. "Study of the reactivity of a monoclonal antibody that recognises human marginal zone B cells and a subset of germinal centre cells in tissue sections." Thesis, King's College London (University of London), 2018. https://kclpure.kcl.ac.uk/portal/en/theses/study-of-the-reactivity-of-a-monoclonal-antibody-that-recognises-human-marginal-zone-b-cells-and-a-subset-of-germinal-centre-cells-in-tissue-sections(7fcf0edc-b729-4215-ae52-a9e2d2622493).html.

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Marginal Zone (MZ) cells are a subset of B cells. They reside the marginal zone outside the mantle zone of the spleen and circulate the blood in humans. They have also been identified in the crypt epithelium of tonsil, the sub-capsular sinus in lymph node and in the dome area of Peyer’s patches in gut-associated lymphoid tissue of human intestine. A monoclonal antibody (4D12) that recognises an antigen on MZ B cells and a subset of GC cells in tissue sections has been described previously but this has not been investigated in detail and features of the 4D12 antigen and the lymphocytes that exp

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13

Xu, Hui. "Modulation of B cell access to antigen by passively administered antibodies : an explanation for antibody feedback regulation?" Doctoral thesis, Uppsala universitet, Institutionen för medicinsk biokemi och mikrobiologi, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-302780.

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Antibody responses can be up- or down-regulated by passive administration of specific antibody together with antigen. Depending on the structure of the antigen and the antibody isotype, responses can be completely suppressed or enhanced up to a 1000-fold of what is seen in animals immunized with antigen alone. IgG suppresses primary antibody responses against erythrocytes. Suppression works well in mice lacking Fc-receptors for IgG, C1q, C3, or complement receptor 1 and 2 (CR1/2). Here, we demonstrate that IgG anti-NP given to mice together with NP-conjugated sheep erythrocytes, suppresses the

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14

Beattie, Lynette. "The role of the spleen in Malaria : Cellular changes that affect the development of immunity." Queensland University of Technology, 2006. http://eprints.qut.edu.au/16195/.

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Malaria, caused by the apicomplexan parasite Plasmodium, is a major cause of morbidity and mortality throughout the world. This study has focused on the role of the spleen in the control of the blood stage of infection. Three aspects have been examined specifically: the effect of infection on the architecture of the spleen, the role of the spleen in parasite clearance and the formation of B cell memory. Firstly, the effect of infection on the splenic microarchitecture was examined. An essential component of the splenic architecture is the marginal zone (MZ), an area of the spleen that separa

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15

Sakakibaya, Ayako, Kumi Kawai, Tetsuro Nagasaka, et al. "Molecular diagnosis of malignant lymphoma : mantle cell lymphoma, anaplastic large cell lymphoma, and marginal zone B-cell lymphoma of malt." Nagoya University School of Medicine, 2006. http://hdl.handle.net/2237/6127.

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16

Palm, Anna-Karin E. "Function and Regulation of B-cell Subsets in Experimental Autoimmune Arthritis." Doctoral thesis, Uppsala universitet, Kemisk biologi, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-265024.

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B lymphocytes play a significant role in autoimmune arthritis, with their function stretching beyond autoantibody production to cytokine secretion and presentation of autoantigen. However, the involvement and activation of different B-cell subset in the autoimmune response is not fully clear. The main focus of this thesis has been to understand the contribution of marginal zone (MZ) B cells in the induction of collagen-induced arthritis (CIA), a mouse model for rheumatoid arthritis (RA). We show that MZ B cells in the spleen of naïve mice display a natural self-reactivity to collagen type II (

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17

Motta, Vinícius. "The genetic basis of T and B cell contribution to autoimmune diabetes in NOD mice." Doctoral thesis, Umeå universitet, Medicinsk och klinisk genetik, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-728.

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The nonobese diabetic mouse (NOD) is an excellent animal model to study type 1 diabetes. As with some humans, disease in the NOD mouse is effected by a combination of genetic and environmental factors. At least 20 insulin dependent diabetes (Idd) susceptibility loci have been identified so far, both in humans and in the NOD mouse. In this thesis, the overall aim has been to understand the genetic basis of diabetes in the NOD mouse by assessing immunogically-related phenotypes. As lymphocytes are the main players in the onset and progression to overt diabetes, we searched for physiological abno

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18

Wikström, Ingela. "Molecular genetics of B- and T-lymphocyte development." Doctoral thesis, Umeå universitet, Medicinsk biovetenskap, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-802.

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Lymphocytes are essential for the generation of specific immunity. Development of B cells in the bone marrow and T cells in the thymus have several analogous features, and are tightly regulated processes. Even though there is an increasing amount of information concerning lymphopoiesis, a lot of questions remain. The aim of this thesis has been to understand some of the molecular events that contribute to the control of lymphocyte development. Expression of the B cell receptor is an important checkpoint in B lymphocyte development. The Dµ protein is a truncated B cell receptor that can induce

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19

Bun, Mylène. "Etude des mécanismes moléculaires induits par l’activation de la voie de signalisation NOTCH2 dans les lymphomes B de la zone marginale de la rate (SMZL) NOTCH-Induced MYC Expression Is Required for Marginal Zone Lymphoma Cell Survival." Thesis, université Paris-Saclay, 2020. http://www.theses.fr/2020UPASL067.

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Le lymphome B de la zone marginale de la rate (SMZL) est un lymphome non-hodgkinien qui touche principalement les sujets âgés (65 ans). Ce type de lymphome de B de la zone marginale se caractérise par une prolifération aberrante des lymphocytes B de la zone marginale de la rate. Cette sur-prolifération entraîne chez les patients une splénomégalie, une cytopénie, une thrombocytose et dans 15% des cas, une manifestation auto-immune. A ce jour, il n’existe aucun traitement spécifique. Des études basées sur les analyses mutationnelles sur les patients ont permis de mettre en évidence une mutation

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20

Zhang, Lu. "IgG3 Complements IgM in the Complement-Mediated Regulation of Immune Responses." Doctoral thesis, Uppsala universitet, Institutionen för medicinsk biokemi och mikrobiologi, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-316618.

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An intact complement system is essential for the initiation of a normal antibody response. Antibodies can regulate their own production against the antigens that they are specific for. Both IgG3 and IgM are able to enhance the antibody response via complement. Here, we have compared the fate of OVA-TNP (ovalbumin-2,4,6-trinitrophenyl) administered intravenously to mice either alone or in complex with monoclonal IgG3 anti-TNP. IgG3-antigen complexes bind to marginal zone (MZ) B cells via complement receptors 1 and 2 (CR1/2) and are transported into splenic follicles. The majority (50% - 90%) of

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21

Audia, Sylvain. "Etude physiopathologique de la réponse immunitaire au cours de la thrombopénie immunologique (purpura thrombopénique immunologique)." Phd thesis, Université de Bourgogne, 2010. http://tel.archives-ouvertes.fr/tel-00687984.

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La thrombopénie immunologique ou purpura thrombopénique immunologique (PTI) est une maladie auto-immune rare responsable d'une destruction périphérique immunologique des plaquettes associée à une production médullaire inadaptée. Dans la première partie de ce travail, nous exposons les connaissances actuelles de sa physiopathologie ainsi que certaines données concernant la réponse immunitaire T, le rôle des lymphocytes T régulateurs (Treg), l'implication de la rate dans la réponse immunitaire ainsi que les modes d'action d'une thérapeutique anti-lymphocytaire B, le rituximab. Dans une seconde p

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22

Bialecki, Emilie. "Rôle des cellules présentatrices d'antigènes spléniques dans l'activation des lymphocytes T Natural Killer invariants." Phd thesis, Université du Droit et de la Santé - Lille II, 2010. http://tel.archives-ouvertes.fr/tel-00634748.

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La zone marginale de la rate apparaît comme un lieu stratégique de détection des antigènes et agents pathogènes véhiculés par le sang. Ces propriétés sont principalement liées à la présence de cellules appartenant au système immunitaire inné parmi lesquelles se trouvent des nombreuses cellules présentatrices d'antigènes (APC), comme les macrophages, les lymphocytes B de la zone marginale (MZB) ou encore les cellules dendritiques (DC). Ces cellules représentent une première ligne de défense contre les pathogènes véhiculés par le sang et sont importantes pour l'initiation des réponses immunes. I

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23

Xue, Xiaoyan. "Identification of retinal stem cells in the ciliary marginal zone of the Xenopus retina." Thesis, University of Cambridge, 2010. https://www.repository.cam.ac.uk/handle/1810/283870.

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24

Baird, Allison Michelle. "Analysis of Low Zone Tolerance in Normal and B Cell-Deficient Mice." eScholarship@UMMS, 1996. https://escholarship.umassmed.edu/gsbs_diss/142.

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This thesis investigates the role of B cells as antigen-specific antigen-presenting cells (APC) in self tolerance to low concentrations of soluble self proteins and in acquired tolerance to low doses of soluble foreign protein antigens. Experiments were performed in normal and B cell-deficient animals, and tolerance induction was measured by T cell proliferation assays. T cell proliferation was reduced in B cell-deficient mice, indicating that B cells may be involved in efficient activation of naive T cells in response to protein antigen both in vivo and in vitro. To study acquired tolerance i

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25

Jallades, Laurent. "Caractérisation moléculaire des délétions du chromosome 7q dans les lymphomes B de la zone marginale splénique." Thesis, Lyon 1, 2012. http://www.theses.fr/2012LYO10342.

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La délétion du chromosome 7q est l'anomalie cytogénétique la plus caractéristique du lymphome de la zone marginale splénique (LZMS). Une étude par hybridation génomique comparative de haute résolution a été conduite sur une série de 27 échantillons de LZMS afin de détecter des micro-remaniements du chromosome 7q. Une région commune de délétion (RCD) de 10,6 Mb a été délimitée sur le chromosome 7q. De plus, une microdélétion somatique du gène AHCYL2 (S-adenosyl-homocystéine hydrolase-like 2) a été détectée au sein de la RCD, définissant la plus petite RCD connue sur le chromosome 7q32 dans le S

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26

Al, Dalahmah Osama Ahmad Odeh. "The molecular regulation of neural stem cell lineage progression in the postnatal subventricular zone by Galectin-3." Thesis, University of Oxford, 2015. http://ora.ox.ac.uk/objects/uuid:cb9020e1-931f-44cd-8409-7d2c58817d6e.

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Neurogenesis continues postnatally in two major neural stem cell (NSC) niches: The subventricular zone (SVZ) and dentate gurus of the hippocampus. SVZ NSCs self-renew and produce transit amplifying progenitor cells that, in turn, divide and give rise to neuroblasts. These neuroblasts migrate to the olfactory bulbs, via the rostral migratory stream (RMS), where they terminally differentiate into mature neurons. The postnatal SVZ (pSVZ) is more gliogenic than its adult counterpart (aSVZ), contributing to robust postnatal astrocytogenesis and oligodendrogenesis in the surrounding brain parenchyma

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27

岸本, 磨由子, and Mayuko Kishimoto. "RHOF PROMOTES MURINE MARGINAL ZONE B CELL DEVELOPMENT." Thesis, 2014. http://hdl.handle.net/2237/20979.

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28

Turchinovich, Gleb [Verfasser]. "BKLF promotes B cell differentiation towards marginal zone lineage / Gleb Turchinovich." 2008. http://d-nb.info/988172089/34.

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29

Chen, Grace Yi-Ying. "Functional Analysis of Adapter Protein c-Abl Src Homology 3 Domain-binding Protein 2." Thesis, 2009. http://hdl.handle.net/1807/17740.

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3BP2 is a pleckstrin homology (PH) domain- and Src homology 2 (SH2) domain-containing adapter protein that has been linked through genetic evidence to a rare human disease called cherubism 146. 3BP2 was originally cloned in a screen to identify c-Abl SH3 binding proteins 23,24. In overexpression studies, 3BP2 has been implicated as a positive regulatory adapter molecule coupled to immunoreceptor on T cells 67,69,70, B cells 68, NK cells 71-73 and mast cells 74,75. It was also evident that 3BP2 forms complexes with a number of signaling molecules, such as Zap-70, LAT, phospholipase C-γ1 (PLC

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30

Whipple, Emily Christine. "Targeted delivery of antigens to follicular dendritic cells via a monoclonal antibody specific for complement receptor 2 on marginal zone B cells : dynamics of delivery and applications towards vaccine development /." 2006. http://wwwlib.umi.com/dissertations/fullcit/3235122.

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31

Sabourin-Poirier, Catherine. "Étude du niveau de B Lymphocyte Stimulator (BLyS) et de son impact sur les lymphocytes B en relation avec l’infection et la résistance au virus d’immunodéficience humaine (VIH) chez des travailleuses du sexe au Bénin." Thèse, 2016. http://hdl.handle.net/1866/16275.

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L’infection au VIH s’accompagne souvent de dérégulations du compartiment des lymphocytes B qui nuisent à la génération de réponses efficaces. En effet, détectées tôt après l’infection, ces dérégulations perdurent, ne sont pas totalement restaurées par la thérapie, et mènent souvent à des manifestations auto-immunes et lymphomes. Une étude longitudinale de notre groupe, effectuée avec des cellules mononucléées du sang circulant provenant de patients VIH+ avec différents types de progression clinique, a démontré qu’un niveau élevé de BLyS chez des individus VIH+ progresseurs était associé à une

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32

Chagnon-Choquet, Josiane. "L'impact des cellules dendritiques dans la dérégulation des cellules B dans un contexte d'infection au virus d'immunodéficience humaine." Thèse, 2013. http://hdl.handle.net/1866/11212.

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Les cellules dendritiques (DC) sont parmi les premières cellules à rencontrer le virus d’immunodéficience humaine (VIH) au niveau des muqueuses. De plus, le fait que les DC sont, de manière directe ou indirecte par le virus et ses composantes, altérées tant par leur nombre, leur phénotype et leur fonction suggère leur implication dans les dérégulations des cellules B. Selon cette hypothèse, des études longitudinales impliquant des individus infectés au VIH-1 présentant différents profils de progression clinique menées dans notre laboratoire ont démontré que les altérations des cellules B sont

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33

Hou, Szu-Yu, and 侯思羽. "Perinatal development of the corticothalamic neurons and their interactions with marginal zone cells in the rat." Thesis, 2008. http://ndltd.ncl.edu.tw/handle/72850777348049767748.

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碩士<br>國立清華大學<br>分子醫學研究所<br>96<br>The corticothalamic (CT) neurons, residing in the neocortex and sending axons to the thalamus, are important components for cortical processing and other brain functions. The morphology of DiI-back-labeled CT neurons in perinatal rat cortices was studied. The CT neurons are early-generated pyramidal cells in the neocortex. The CT neurons develop from cells having an oval cell body, no basilar dendrites and an apical dendrite with few branches in the marginal zone (MZ) into cells displaying a pyramidal-shaped cell body, 2-3 basilar dendrites and an apical dendri

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34

Fourcade, Lyvia. "L’immunité naturelle contre le VIH-1 est associée à un profil tolérogénique dans la muqueuse génitale des travailleuses du sexe béninoises hautement exposées et séronégatives (HESN)." Thesis, 2020. http://hdl.handle.net/1866/24608.

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La plupart des infections par le VIH-1 sont acquises lors de rapports hétérosexuels. En Afrique subsaharienne on observe 71 % des infections mondiales et 60 % des nouvelles infections par le VIH-1 touchent les femmes. Le tractus génital féminin (TGF) constitue la principale porte d’entrée pour le VIH-1 et joue un rôle important dans la défense de l’organisme contre les microorganismes pathogènes tout en maintenant une tolérance de la flore commensale. On y trouve les cellules épithéliales qui participent à l’élaboration des réponses immunes en collaboration avec les cellules dendritiques (DCs)

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35

Gauvin, Julie. "Étude de la migration des populations de lymphocytes B du sang de patients infectés par le virus d’immunodéficience humaine (VIH)." Thèse, 2015. http://hdl.handle.net/1866/13885.

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La dérégulation du compartiment de cellules B est une conséquence importante de l’infection par le virus de l’immunodéficience humaine (VIH-1). On observe notamment une diminution des nombres de lymphocytes B sanguins ainsi qu’une variation des fréquences relatives des différentes populations de lymphocytes B chez les individus infectés par rapport aux contrôles sains. Notre laboratoire a précédemment démontré l’implication des cellules dendritiques dans la dérégulation des lymphocytes B via la roduction excessive de BLyS/BAFF, un stimulateur des cellules B. De plus, lors l’études menées chez

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El, Aanbouri Mimount. "Überleben und Differenzierung TAT-Bcl-xL-transduzierter transplantierter neuraler Vorläuferzellen nach zerebraler Ischämie der Maus." Doctoral thesis, 2009. http://hdl.handle.net/11858/00-1735-0000-0006-AD87-B.

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