Relevant bibliographies by topics / MBNH3

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  1. Journal articles
  2. Dissertations / Theses
  3. Books

Academic literature on the topic 'MBNH3'

Author: Grafiati
Published: 25 July 2024
Last updated: 31 July 2025

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Journal articles on the topic "MBNH3"

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Sun, Xueqin, Xinghua Diao, Xiaolin Zhu, Xuexue Yin, and Guangying Cheng. "Nanog-mediated stem cell properties are critical for MBNL3-associated paclitaxel resistance of ovarian cancer." Journal of Biochemistry 169, no. 6 (2021): 747–56. http://dx.doi.org/10.1093/jb/mvab021.

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Abstract Paclitaxel (PTX) is the standard first-line treatment of ovarian cancer, but its efficacy is limited by multidrug resistance. Therefore, it is crucial to identify effective drug targets to facilitate PTX sensitivity for ovarian cancer treatment. Seventy PTX-administrated ovarian cancer patients were recruited in this study for gene expression and survival rate analyses. Muscleblind-like-3 (MBNL3) gain-of-function and loss-of-function experiments were carried out in ovarian cancer cells (parental and PTX-resistant) and xenograft model. Cancer cell viability, apoptosis, spheroids format
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Holm, Frida, Eva Hellqvist, Cayla N. Mason, et al. "Reversion to an embryonic alternative splicing program enhances leukemia stem cell self-renewal." Proceedings of the National Academy of Sciences 112, no. 50 (2015): 15444–49. http://dx.doi.org/10.1073/pnas.1506943112.

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Formative research suggests that a human embryonic stem cell-specific alternative splicing gene regulatory network, which is repressed by Muscleblind-like (MBNL) RNA binding proteins, is involved in cell reprogramming. In this study, RNA sequencing, splice isoform-specific quantitative RT-PCR, lentiviral transduction, and in vivo humanized mouse model studies demonstrated that malignant reprogramming of progenitors into self-renewing blast crisis chronic myeloid leukemia stem cells (BC LSCs) was partially driven by decreased MBNL3. Lentiviral knockdown of MBNL3 resulted in reversion to an embr
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Guendouz, Dj, Z. Charifi, H. Baaziz, et al. "Electronic structure, optical and thermodynamic properties of ternary hydrides MBeH3 (M = Li, Na, and K)." Canadian Journal of Physics 94, no. 9 (2016): 865–76. http://dx.doi.org/10.1139/cjp-2016-0299.

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Electronic band structure, optical and thermodynamic properties of ternary hydrides MBeH3 (M = Li, Na, and K) were studied using ab initio density functional theory (DFT). The effect of the adopted approximation to the exchange-correlation functional of the DFT is explicitly investigated by considering four different expressions of two different classes (local-density approximation and generalized-gradient approximation). The calculated magnitude of B classifies MBeH3 (M = Li, Na, and K) as easily compressible materials. The bonding interaction in these compounds is quite complicated. The inte
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Manesis, Anastasia C., Richard J. Jodts, Brian M. Hoffman, and Amy C. Rosenzweig. "Copper binding by a unique family of metalloproteins is dependent on kynurenine formation." Proceedings of the National Academy of Sciences 118, no. 23 (2021): e2100680118. http://dx.doi.org/10.1073/pnas.2100680118.

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Some methane-oxidizing bacteria use the ribosomally synthesized, posttranslationally modified natural product methanobactin (Mbn) to acquire copper for their primary metabolic enzyme, particulate methane monooxygenase. The operons encoding the machinery to biosynthesize and transport Mbns typically include genes for two proteins, MbnH and MbnP, which are also found as a pair in other genomic contexts related to copper homeostasis. While the MbnH protein, a member of the bacterial diheme cytochrome c peroxidase (bCcP)/MauG superfamily, has been characterized, the structure and function of MbnP,
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Wang, Xianxian, Yanchao Cai, Xiucheng Liu, and Cunfu He. "Quantitative Prediction of Surface Hardness in Cr12MoV Steel and S136 Steel with Two Magnetic Barkhausen Noise Feature Extraction Methods." Sensors 24, no. 7 (2024): 2051. http://dx.doi.org/10.3390/s24072051.

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The correlation between magnetic Barkhausen noise (MBN) features and the surface hardness of two types of die steels (Cr12MoV steel and S136 steel in Chinese standards) was investigated in this study. Back-propagation neural network (BP-NN) models were established with MBN magnetic features extracted by different methods as the input nodes to realize the quantitative prediction of surface hardness. The accuracy of the BP-NN model largely depended on the quality of the input features. In the extraction process of magnetic features, simplifying parameter settings and reducing manual intervention
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Kanadia, Rahul N., Carl R. Urbinati, Valerie J. Crusselle, et al. "Developmental expression of mouse muscleblind genes Mbnl1, Mbnl2 and Mbnl3." Gene Expression Patterns 3, no. 4 (2003): 459–62. http://dx.doi.org/10.1016/s1567-133x(03)00064-4.

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7

Holm, Frida Linnea, Eva Hellqvist, Cayla N. Mason, et al. "Reversion to an Embryonic Alternative Splicing Program Enhances Leukemia Stem Cell Self-Renewal." Blood 126, no. 23 (2015): 1227. http://dx.doi.org/10.1182/blood.v126.23.1227.1227.

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Abstract Background Formative research suggests that a human embryonic stem cell-specific alternative splicing gene regulatory network, which is repressed by Muscleblind-like (MBNL) RNA binding proteins, is involved in cell reprogramming. However, its role in malignant reprogramming of progenitors into self-renewing leukemia stem cells (LSCs) had not been established. Methods Whole transcriptome RNA sequencing (RNA-seq) was performed on FACS purified progenitors from normal, chronic phase and blast crisis chronic myeloid leukemia samples and analyzed using Cuff-links, GSEA and IPA software. Sp
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Lee, Kyung-Soon, Kimberly Smith, Paul S. Amieux, and Edith H. Wang. "MBNL3/CHCR prevents myogenic differentiation by inhibiting MyoD-dependent gene transcription." Differentiation 76, no. 3 (2008): 299–309. http://dx.doi.org/10.1111/j.1432-0436.2007.00209.x.

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Grammatikakis, Ioannis, Young-Hwa Goo, Gloria V. Echeverria, and Thomas A. Cooper. "Identification of MBNL1 and MBNL3 domains required for splicing activation and repression." Nucleic Acids Research 39, no. 7 (2010): 2769–80. http://dx.doi.org/10.1093/nar/gkq1155.

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Helbing, Jan. "Spin state transitions upon visible and infrared excitation of ferric MbN3." Chemical Physics 396 (March 2012): 17–22. http://dx.doi.org/10.1016/j.chemphys.2011.04.001.

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Dissertations / Theses on the topic "MBNH3"

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Chieh-Wen and 林介玟. "Identification and functional analysis of zebrafish mbnl3." Thesis, 2009. http://ndltd.ncl.edu.tw/handle/35015125149391021434.

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碩士<br>中山醫學大學<br>生物醫學科學學系碩士班<br>97<br>Muscleblind-like (MBNL) is a family of proteins that participate in regulation of tissue-specific alternative splicing. In Drosophila, the muscleblind protein was shown to regulate terminal differentiation of photoreceptors and muscles. Three MBNL paralogs have been identified in humans and mice. Previously, we cloned three mbnl genes (zmbnl1 – 3) in zebrafish in order to study their function during fish development. In this study, we focus on zmbnl3. Alternative splicing of the zmbnl3 primary transcripts gives rise to at least 5 protein isoforms. In additi
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2

Su, Hung-Chi, and 蘇虹綺. "The Effect of MBNL3 Knockdown on Cell Proliferation and Senescence." Thesis, 2013. http://ndltd.ncl.edu.tw/handle/62014042578290292207.

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碩士<br>國立中正大學<br>分子生物研究所<br>101<br>Muscleblind-like 3 (MBNL3) belongs to MBNL family that has been implicated in the pathogenesis of myotonic dystrophy type 1 (DM1). Mbnl3 expression is abundant in proliferative tissues and greatly decreases during differentiation. Our laboratory previously found that Mbnl3 knockdown interferes with cell cycle progression and induces senescence through cyclin D1-mediated pathway in C2C12 myoblasts. The first part of my study aimed to confirm the effect of Mbnl3 knockdown on C2C12 cells. My results indicated that (1) both restoration of Mbnl3 and cyclin D1 expre
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Ke, Wen-Hsing, and 柯雯馨. "Investigation of Effect of MBNL3 Knockdown on Cell Cycle Progression in C2C12 Myoblasts." Thesis, 2011. http://ndltd.ncl.edu.tw/handle/71769638459090265318.

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碩士<br>國立中正大學<br>分子生物研究所<br>100<br>Muscleblind-like protein 3 (MBNL3) belongs to MBNL family implicated in the pathogenesis of myotonic dystrophy, the most common neuromuscular disease in adults. MBNL3 expression is abundant in proliferating myoblasts and greatly down-regulated during differentiation. In this study, we have investigated the effect of MBNL3 loss-of-function on C2C12 myoblasts by using lentiviral knockdown system. The results indicated that MBNL3 knockdown cells had lower proliferation rate than scramble control as revealed by cell counting and MTT assay. Propidium iodide stainin
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Huang, Shu-Yuan, and 黃淑媛. "Mbnl3 regulates myoblast proliferation and induces senescence through the Sp1/cyclin D1/Rb pathway." Thesis, 2015. http://ndltd.ncl.edu.tw/handle/jx6ny8.

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碩士<br>國立中正大學<br>分子生物研究所<br>103<br>Muscleblind-like 3 (MBNL3) belongs to the MBNL family which has been implicated in the pathogenesis of neuromuscular disease myotonic dystrophy. While MBNL3 is barely detectable in adult muscle, it is expressed in proliferative myoblasts and transiently during muscle regeneration. Our previous study found that Mbnl3 knockdown interferes with cell cycle progression and induces senescence through cyclin D1-mediated pathway in C2C12 myoblasts. The first aim of my study is to confirm the effect of Mbnl3 knockdown on C2C12 cells. The result indicated that the effec
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Books on the topic "MBNH3"

1

Al-Jawaldeh, Ayoub, and Alexa Meyer. Reshaping Food Systems to Improve Nutrition and Health in the Eastern Mediterranean Region. Open Book Publishers, 2023.

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Al-Jawaldeh, Ayoub, and Alexa Meyer. Reshaping Food Systems to Improve Nutrition and Health in the Eastern Mediterranean Region. Open Book Publishers, 2023.

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Health Care in the Information Society Vol. 1: From Adventure of Ideas to Programme for Reform. Open Book Publishers, 2023.

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Health Care in the Information Society Vol. 1: From Adventure of Ideas to Programme for Reform. Open Book Publishers, 2023.

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Health Care in the Information Society : Volume 2: From Anarchy of Transition to Programme for Reform. Open Book Publishers, 2023.

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