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Books on the topic 'Mechanical trauma'

Author: Grafiati
Published: 24 April 2022
Last updated: 29 May 2022

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1

Yan, Hua, ed. Mechanical Ocular Trauma. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-2150-3.

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2

Maul, Timothy M. Mechanical blood trauma in circulatory-assist devices. New York: ASME Press, 2015.

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3

F, Robben Simon G., and Rijn Rick R. van, eds. Forensic aspects of pediatric fractures: Differentiating accidental trauma from child abuse. Berlin: Springer, 2010.

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4

D, Johnson Kenneth, ed. Biomechanics in orthopedic trauma: Bone fracture and fixation. London: M. Dunitz, 1994.

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5

F, Niederer Peter, Walz Felix H, Muser Markus H, and SpringerLink (Online service), eds. Trauma Biomechanics: Accidental injury in traffic and sports. Berlin, Heidelberg: Springer-Verlag Berlin Heidelberg, 2010.

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6

Freivalds, Andris. Biomechanics of the upper extremities: Mechanics, modeling, and musculoskeletal injuries. 2nd ed. Boca Raton, FL: CRC Press, 2011.

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7

Freivalds, Andris. Biomechanics of the upper limbs: Mechanics, modeling, and musculoskeletal injuries. New York: Taylor & Francis, 2004.

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8

Freivalds, Andris. Biomechanics of the upper limbs: Mechanics, modeling, and musculoskeletal injuries. Boca Raton, FL: CRC Press, 2004.

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9

Dynamic bodyuse for effective strain-free massage. Chichester, England: Lotus Pub., 2007.

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10

Yan, Hua. Mechanical Ocular Trauma: Current Consensus and Controversy. Springer, 2018.

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11

Maul, Timothy Michael, and Marina V. Kameneva. Mechanical Blood Trauma in Circulatory-Assist Devices. Momentum Press, 2015.

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12

Maul, T., M. V. Kameneva, and P. D. Wearden. Mechanical Blood Trauma in Circulatory-Assist Devices. ASME Press, 2015. http://dx.doi.org/10.1115/1.860397.

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13

Amato, Marcelo, and Andreas Wolfgang Reske. Ventilator trauma in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0101.

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Ventilator trauma refers to complications of mechanical ventilation, which have an impact on morbidity and mortality. Two major forms of ventilator trauma may be distinguished—an acute form related to rupture of airspaces causing air-leak syndrome and a subacute form causing protracted inflammatory responses. A key feature of mechanically-ventilated lungs is the presence of non-aerated and unstable regions due to atelectasis, oedema, or consolidation. Because of mechanical interdependence, pressures acting in non-uniformly expanded lungs at the boundaries between non-aerated and aerated lung may be a multiple of the apparent transpulmonary pressure. The resulting effects have been reported to precipitate or contribute to ventilator-induced lung injury (VILI). The engineering terms stress and strain were recently proposed for better definition of risk-constellations for VILI. Because the aerated lung volume is positively correlated to compliance, driving-pressure can aid in identifying disproportionate combinations of tidal volumes and compliance.
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14

KLINGMAN, AM. Kligman Chronic Effects of Mechanical Trauma to Th E Skin. John Wiley & Sons Inc, 1986.

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15

Oliver, Charles M., and S. Ramani Moonesinghe. Setting rate, volume, and time in ventilatory support. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0093.

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Ventilator rate, volume, and time parameters are interrelated directly, mechanically, and physiologically, and interactions between intrinsic pulmonary physio-mechanics, pathology and the effects of mechanical ventilation complex. The physiological consequences of mechanical ventilation and risks of ventilator-induced trauma may be exacerbated by lung pathology. Programming of ventilator parameters should be considered within the context of an individualized ventilatory strategy to achieve adequate gas exchange, while minimizing attendant risks of mechanical ventilation. Recommended strategies should be modified within accepted limits to mitigate disease-specific risks. Parameters should subsequently be titrated against blood gas- and ventilator-derived targets, and other clinical variables.
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16

Trauma Biomechanics: Introduction to Accidental Injury. Springer, 2004.

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17

Rijn, Rick R. van, Rob A. C. Bilo, and Simon G. F. Robben. Forensic Aspects of Pediatric Fractures: Differentiating Accidental Trauma from Child Abuse. Springer, 2011.

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18

Walz, Felix, Kai-Uwe Schmitt, Peter F. Niederer, Markus H. Muser, Duane S. Cronin, and Barclay Morrison III. Trauma Biomechanics: An Introduction to Injury Biomechanics. Springer, 2019.

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19

Walz, Felix, Kai-Uwe Schmitt, Peter F. Niederer, Markus H. Muser, and Duane S. Cronin. Trauma Biomechanics: An Introduction to Injury Biomechanics. Springer, 2016.

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20

Walz, Felix, Kai-Uwe Schmitt, Peter F. Niederer, Markus H. Muser, and Duane S. Cronin. Trauma Biomechanics: An Introduction to Injury Biomechanics. Springer, 2014.

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21

1916-, Kligman Albert M., Klemme Jay C, Susten Allan S, and National Institute for Occupational Safety and Health., eds. The Chronic effects of repeated mechanical trauma to the skin: Proceedings of a conference. New York: A.R. Liss, 1985.

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22

Thoracic trauma and critical care. Boston: Kluwer Academic Publishers, 2002.

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23

Riyad, Karmy-Jones, Nathens Avery, and Stern Eric J, eds. Thoracic trauma and critical care. Boston: Kluwer Academic Publishers, 2002.

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24

(Editor), Riyad Karmy-Jones, Avery Nathens (Editor), and Eric Stern (Editor), eds. Thoracic Trauma and Critical Care. Springer, 2002.

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25

Head Injury Biomechanics, Volume 1-- The Skull. SAE International, 2011.

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26

(Editor), C. D. Mote, Robert J. Johnson (Editor), Wolfhart Hauser (Editor), and Peter S. Schaff (Editor), eds. Skiing Trauma and Safety (Skiing Trauma & Safety). American Society for Testing & Materials, 1997.

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27

Barral, Jean-Pierre, and Alain Croibier. Trauma: An Osteopathic Approach. Eastland Press, 2000.

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28

Colebourn, Claire, and Jim Newton. Field guide to critical care echocardiography. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198757160.003.0008.

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This chapter is intended as a summary or reference for clinician echocardiographers at the bedside. It gives step-by-step algorithms which address both commonly asked questions and clinical situations which require rapid decision-making using echocardiography in the critically ill. These algorithms cover fluid status and fluid responsiveness, cardiovascular parameters, assessment of the shocked and breathless patient including trauma, assessment of the patient with a clinical diagnosis of pulmonary embolism, and assessment of the unwell obstetric patient and patients who are weaning from mechanical ventilatory support.
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29

1931-, Nahum Alan M., and Melvin John, eds. The Biomechanics of trauma. Norwalk, Conn: Appleton-Century-Crofts, 1985.

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30

Gupta, Tarang, and Daniel Ezra. Oculoplastics. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199672516.003.0001.

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This chapter explores oculoplastics. It first details eyelid and nasolacrimal system anatomy and physiology. It then discusses lash abnormalities, covering trichiasis, distichiasis, madarosis, eyelash ptosis, hypertrichosis, and poliosis. Next, it discusses entropion, covering congenital entropion, involutional entropion, and cicatricial entropion. Next, ectropion is discussed, including congenital ectropion, paralytic ectropion, involutional ectropion, cicatricial ectropion, and mechanical ectropion. Ptosis is covered in two sections, and benign lid lesions, malignant lid lesions, epiphora, acquired nasolacrimal system abnormalities, and congenital nasolacrimal system abnormalities are also discussed. In addition, practical skills in oculoplastics are outlined, such as ptosis examination, syringing and probing, removing sutures, surgical repair of lid trauma, lateral tarsorraphy, and emergency lateral canthotomy/cantholysis.
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31

Siebert, Stefan, Sengupta Raj, and Alexander Tsoukas. Complications of axial spondyloarthritis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198755296.003.0009.

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In addition to the well-recognized extra-articular manifestations (EAMs) of ankylosing spondylitis (AS), this condition can also be associated with a number of clinically important complications. While EAMs are considered part of the spondyloarthritis (SpA), the complications are generally a consequence of having the disease. Patients with AS are at increased risk of osteoporosis and spinal fractures. The latter may occur after seemingly minor trauma and may lead to significant neurological compromise. Other potential neurological complications include atlantoaxial subluxation and compressive radiculopathy or myelopathy. Cardiac complications include cardiovascular events, valvular disease, and conduction disturbances. Pulmonary disease in AS relates to parenchymal involvement or mechanical constraint from chest wall inflammation. Renal disease is generally rare in AS.
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32

Warwick, David. Prevention of thrombosis in orthopaedic surgery. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.0006.

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♦ The risk–benefit of thromboprophylaxis in orthopaedic surgery remains unclear♦ Some conditions, such as major trauma, carry a much higher risk than others, such as routine knee replacement♦ Some patients appear to be genetically more predisposed than others♦ In trials of efficacy of thromboembolism, the use of deep vein thrombosis as a surrogate endpoint for death from a pulmonary embolus may not be completely reliable♦ There is a variety of mechanical and chemical methods available, each of which has real and potential advantages as well as real and potential dangers♦ Even the length of time that a patient is at risk after major surgery is unclear♦ Clinicians should adhere to guidelines where possible.
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33

Finn, Patrick C., and Michael C. Reade. Bleeding Emergencies (DRAFT). Edited by Raghavan Murugan and Joseph M. Darby. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190612474.003.0010.

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This chapter is concerned with coagulopathic and non-coagulopathic bleeding in the perioperative period, after trauma, and spontaneously, as a result of hematologic and other disease. The initial assessment and management of all potentially bleeding patients is to stop any obvious bleeding through mechanical first aid measures, then address airway or breathing compromise, and obtain intravenous (or intraosseous) access. Obvious external hemorrhage is easily identified, but most patients with bleeding emergencies who are already hospitalized will have occult blood loss. Physical examination should identify signs of shock and identify or exclude potential bleeding locations. This chapter will cover initial assessment and management, laboratory and bedside testing, as well as disease-specific therapies in the context of rapid response team (RRT) calls.
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34

Harrison, John Henry, and Magdalena Anitescu. Neuraxial Anesthesia in Coexisting Neurologic Conditions. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190271787.003.0041.

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Some patients who need surgery may have coexisting neurologic disorders like multiple sclerosis, amyotrophic lateral sclerosis, peripheral neuropathies (e.g., Charcot-Marie-Tooth disease or Guillain-Barré syndrome), or muscular dystrophies (e.g., Duchenne’s or myotonic dystrophy). When neuraxial analgesia and anesthesia are indicated, the anesthesiologist should be aware of the risks and benefits of the technique. Neuraxial anesthesia is not absolutely contraindicated in nervous system diseases and there are undeniable benefits to ruling out general anesthesia. In patients with coexisting neurologic disorders, prolonged sensory and motor block can be confused with epidural hematoma and abscess when present. Minor nerve injury from local anesthetic cytotoxicity or ischemia and mechanical trauma may cause permanent nerve injury through the double crush phenomenon. Lower concentrations of local anesthetics are generally recommended.
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35

Trauma Biomechanics: Accidental injury in traffic and sports. 2nd ed. Springer, 2007.

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36

Skiing trauma and safety: Tenth volume. West Conshohocken, PA: American Society for Testing and Materials, 1996.

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37

Sabri, Omar, and Martin Bircher. Management of limb and pelvic injuries. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0336.

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Pelvic ring injuries can be life and limb threatening. The mechanism of injury can often be a good indicator of the type of injury; the Young & Burgess classification deploys that concept to full effect. Early identification based on mechanism of injury and improved prehospital care can play a major role in the outcome following such injuries. Pelvic ring injuries can lead to significant haemorrhage. Mechanical measures to stabilize the pelvis, in addition to modern concepts of damage control resuscitation (DCR), have been shown to be effective in early management of potentially life-threatening haemorrhage. Emphasis is now entirely on protecting the primary clot following a pelvic ring injury. Mechanical disturbance by log rolling the patient or springing of the pelvis are strongly discouraged. Early radiological clearance of the pelvis is encouraged. The lethal triad of coagulopathy, acidosis, and hypothermia should be corrected simultaneously to improve outcome. A traffic light system for monitoring venous lactate as an indicator of the patients’ physiological state can help the intensive care practitioner and the surgeon identify optimum timing for surgery. Pelvic ring injuries are associated with significant concomitant injuries. Limb trauma can also be life or limb threatening. Early identification, splinting, and resuscitation follow the same guidelines as pelvic ring injuries. Open long bone fractures should be managed by senior orthopaedic and plastic surgeons.
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38

Kevin Luk, K. H., and Deepak Sharma. Subarachnoid Hemorrhage. Edited by David E. Traul and Irene P. Osborn. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190850036.003.0024.

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Subarachnoid hemorrhage (SAH) is commonly caused by rupture of an intracranial aneurysm, arteriovenous malformation, or due to trauma. Prompt diagnosis and intervention are required to control intracranial pressure, maintain cerebral perfusion, and prevent rebleeding. Clinical grading of the bleed predicts morbidity and mortality, whereas imaging grading predicts risk of cerebral vasospasm. Hydrocephalus can occur as a result of SAH, which requires treatment with an external ventricular drain. Endovascular and open microsurgical procedures are available for securing the vascular abnormalities. Patients are typically monitored in a neurocritical care unit for up to 21 days post-bleed to monitor for the development of cerebral vasospasm/delayed cerebral ischemia (DCI). Mainstay of treatment for DCI includes induced hypertension, balloon angioplasty, and intraarterial vasodilator therapy. In addition, patient may experience significant derangement in their cardiac, pulmonary, and endocrine systems, requiring inotropic support, mechanical ventilation, or insulin infusion therapy.
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39

van den Boogaard, Mark, and Paul Rood. Delirium in Critically Ill Patients. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199398690.003.0002.

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This chapter addresses delirium in critically ill patients in the intensive care unit (ICU), especially the mixed subtype (alternating hyperactivity and hypoactivity). The Confusion Assessment Method for the ICU and the Intensive Care Delirium Screening Checklist are discussed as useful delirium assessment tools in this setting. Several neurotransmitter pathways have been implicated in delirium, including cholinergic, GABAergic, and serotonergic pathways; cytokines and glucocorticoids also appear relevant. Risk factors for delirium in the ICU include older age, prior cognitive impairment, worse illness severity, recent delirium or coma, mechanical ventilation, admission category (especially trauma or neurological/neurosurgical admission), infection, metabolic acidosis, morphine and sedative administration, urea concentration, respiratory failure, and admission urgency. Prevention and treatment of delirium are discussed, including nonpharmacological interventions (frequent reorientation, providing eyeglasses and hearing aids if needed, promoting nighttime sleep, and early mobilization) and judicious use of opiate, sedative, and antipsychotic medications.
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40

Trauma - An Engineering Analysis: With Medical Case Studies Investigation. Springer, 2007.

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41

Baldwin, Matthew, and Hannah Wunsch. Mortality after Critical Illness. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0003.

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Many critically ill patients now survive what were previously fatal illnesses, but long-term mortality after critical illness remains high. While study populations vary by country, age, intervention, or specific diagnosis, investigations demonstrate that the majority of additional deaths occur in the first 6 to 12 months after hospital discharge. Patients with diagnoses of cancer, respiratory failure, and neurological disorders leading to the need for intensive care have the highest long-term mortality, while those with trauma and cardiovascular diseases have much lower long-term mortality. Use of mechanical ventilation, older age, and a need for care in a facility after the acute hospitalization are associated with particularly high 1-year mortality among survivors of critical illnesses. Due to challenges of follow-up, less is known about causes of delayed mortality following critical illness. Longitudinal studies of survivors of pneumonia, stroke, and patients who require prolonged mechanical ventilation suggest that most debilitated survivors die from recurrent infections and sepsis. Potential biologic mechanisms for increased risk of death after a critical illness include sepsis-induced immunoparalysis, intensive care unit-acquired weakness, neuroendocrine changes, poor nutrition, and genetic variance. Studies are needed to fully understand how the severity of the acute critical illness interacts with comorbid disease, pre-illness disability, and pre-existing and acquired frailty to affect long-term mortality. Such studies will be fundamental to improve targeting of rehabilitative, therapeutic, and palliative interventions to improve both survival and quality of life after critical illness.
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42

Ahuja, Christopher S., and Michael Fehlings. Neuroprotection for Spinal Cord Injury. Edited by David L. Reich, Stephan Mayer, and Suzan Uysal. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190280253.003.0015.

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Traumatic spinal cord injuries (SCI) often have a devastating impact on quality of life for patients and their families. Neuroprotection for spinal cord injury is aimed at improving functional outcomes by limiting secondary injury processes that occur within the first minutes, hours, and days following the primary injury. The primary mechanical trauma initiates a secondary injury cascade where ischemia, inflammatory cell infiltration, and cytotoxic changes in the microenvironment cause further cell death and loss of function. Time-sensitive neuroprotective measures targeting these secondary insults have emerged as key therapeutic strategies. This chapter summarizes current evidence-based neuroprotective treatments, such as blood pressure augmentation, early surgical decompression, and intravenous methylprednisolone, as well as important emerging interventions, including therapeutic hypothermia, sodium channel blockade using riluzole, and the anti-inflammatory actions of minocycline. The chapter concludes by summarizing the current guidelines that all practitioners should be well-versed in prior to providing care for patients with SCI.
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43

1932-, Sances Anthony, ed. Mechanisms of head and spine trauma. Goshen, N.Y: Aloray, 1986.

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44

Skiing trauma and safety, sixth international symposium: A symposium. Philadelphia, PA: American Society for Testing and Materials, 1987.

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45

Greaves, Ian, and Sir Keith Porter. Oxford Handbook of Pre-hospital Care. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198734949.001.0001.

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This handbook is the invaluable guide to providing high-quality care in a pre-hospital environment. Evidence-based and reflecting new developments in regulation and practice, this second edition is designed to provide key information for all immediate care practitioners, including doctors, paramedics, emergency medical technicians, and community responders. The text has been cross-referenced with the Joint Royal Colleges Ambulance Liaison Committee (JRCALC) handbook and appropriate national clinical guidelines to ensure full clinical relevance. Reflecting the major advances in delivery of pre-hospital care, including the greater survival benefits for heart attacks and major trauma when delivering patients directly to higher levels of care, the evolution of the paramedic role into critical care paramedics, roadside rapid sequence induction of anaesthesia, and the introduction of mechanical chest compression devices, this new edition is the ideal companion for those involved in delivering pre-hospital care. It also links to relevant online databases and mobile apps that can assist with calculations, and contains key algorithms and formulae to ensure good care.
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46

Skiing Trauma and Safety: Sixth International Symposium (Astm Special Technical Publication // Stp 938). Astm Intl, 1987.

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47

Freivalds, Andris. Biomechanics of the Upper Limbs: Mechanics, Modelling and Muskoskeletal Injuries. CRC, 2002.

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48

Pritchard, Darien. Dynamic Bodyuse for Effective, Strain-Free Massage. North Atlantic Books, 2007.

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